The patient is a 64-year-old woman with a history of chest pain and an intraventricular conduction delay, QRS interval of 0.10. In February 1988, an exercise tolerance test showed a left bundle branch block with exercise, and a thallium scan showed no evidence of ischemia. In July 1992, an exercise tolerance test with a maximum heart rate of 167 and maximum blood pressure of 138/60 showed a moderate fixed defect in the apicolateral wall. A cardiac catheterization in 1995 showed no coronary disease, but the patient was told she had cardiomyopathy. On the day of admission, the patient was watching television when she suddenly lost consciousness until she awoke with her grandchildren on top of her. Admission medications included Vasotec 10 mg p.o. q.day, Digoxin 0.25 mg p.o. q.day, and Lasix 20 mg q.day. Discharge medications included Enteric coated aspirin 325 mg p.o., Vasotec 15 mg p.o. q.day, Lasix 20 mg p.o. q.day, and Atenolol 12.5 mg p.o. q.day. Laboratory results revealed a CK of 119, magnesium 2.2, digoxin level 0.7, troponin I 0, electrolytes within normal limits, white blood cell count 10.3, hematocrit 36.5, and platelet count 298, urinalysis 0-1 white cells, 0-1 red cells, 1+ bacteria, and 1+ epithelial cells, chest x-ray revealed no evidence of congestive heart failure or infiltrate, EKG showed normal sinus rhythm at a rate of 93 with intervals of 0.183, 0.15, and 0.417, left bundle branch block, no arrhythmias triggered by exercise, carotid noninvasive studies revealed minimal disease bilaterally, tilt table study was entirely normal, no suggestion of a vasovagal response, ejection fraction 30%-35%, anterolateral wall motion abnormalities, right sided heart catheterization revealed coronary arteries completely clean, EP study was entirely normal, and MRA/MRI of her brain and ECG loop recorder were ordered as outpatient follow up.
MEDS RECORD
Digoxin 0.25 mg p.o. q.day,