From a neurophysiological standpoint, what is known about Alzheimer's disease is that there appears to be a progressive process that is associated with the deposition of β Amyloid. This appears to have a triggering mechanism, which causes the phosphorylation of a TAU protein, which then leads to development neurofibrillary tangles (NFT), which are believed to be responsible for memory loss, especially short-term and other associated problems with Alzheimer's related dementia. It also appears that there may be a selective deposition of the β Amyloid in the hippocampus and the prefrontal regions, which are the major short-term memory regions of the brain.