text: I have been practicing archery for the last 12 years. At the beginning it is difficult because it is mainly all about technique you get frustrated because all you want to do is to grab a bow and start shooting. But if you have patience, in a couple of months you get to use the bow and you can start shooting. And then you realize that archery is a really good sport because it makes you feel calm. When Im getting ready for a competition we train for 4 hours in the morning We take a rest, we go for lunch and then train for another 4 hours in the afternoon. After that we have a gym session and after that, we take the rest of the evening off. Depending on what my coach says we shoot different distances. On a normal day I will shoot around 1000 arrows which is a long day for us. We shoot way more during training as it is good to get used to shooting a lot so when competition comes we don't get as tired. We also do other exercises with the bow to practice the release of the arrow and to strengthen the elbow and shoulder it helps us to have a better technique. Every training day is different and you always have to come with the right attitude to get better every day. Everything that you do during training will be reflected during competition.


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Mary Nuckols
Associate Broker
Central Idaho Properties Mary Nuckols
Co-Owner and Associate broker for Century 21 Whitewater Clark. Licensed in the State of Idaho since 1999. 18 years of experience with all types of real estate transactions. A specialist as both a listing and selling agent for residential, remote & recreational properties, ranches, lots and land, REO bank owned properties, commercial parcels and luxury homes. Professional memberships include: Licensed Real Estate Associate Broker in the State of Idaho. Professional Brand Affiliation, with Century 21. Past president & current board member of the Mountain Central Association of Realtors. Member of the National and Idaho State Association of REALTORS and the Mountain Central Association of Realtors. Active participant in two Multiple Listing Services, the Intermountain MLS and MCAR. Member of the McCall and Riggins Chambers of Commerce. Board member for the West Central Mountains Housing Trust.
Mary Nuckols
Associate Broker
CENTURY 21 Whitewater Clark
Salmon River Wildness Ranch - Tramway, Boat or Trail Access
Riggins, Idaho
159 ACRE WILDERNESS RANCH on the SALMON RIVER, 3/4 Mile of River Frontage with white sand beaches. Surrounded by hundreds of thousands of acres...
- 3
- 1
- 1,248 Sq Ft
- 159 Acres
- American Redoubt, Cabin, Homestead, Off-Grid/Solar

subset: commoncrawl


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The bacterial pathogen Yersinia pseudotuberculosis uses a type III secretion (T3S) system to translocate Yop effectors into eukaryotic cells. Effectors are thought to gain access to the cytosol via pores formed in the host cell plasma membrane. Translocated YopE can modulate this pore formation through its GTPase-activating protein (GAP) activity. In this study, we analysed the role of translocated YopE and all the other known Yop effectors in the regulation of effector translocation. Elevated levels of Yop effector translocation into HeLa cells occurred by YopE-defective strains, but not those defective for other Yop effectors. Only Yersinia devoid of YopK exhibits a similar hyper-translocation phenotype. Since both yopK and yopE mutants also failed to down-regulate Yop synthesis in the presence of eukaryotic cells, these data imply that translocated YopE specifically regulates subsequent effector translocation by Yersinia through at least one mechanism that involves YopK. We suggest that the GAP activity of YopE might be working as an intra-cellular probe measuring the amount of protein translocated by Yersinia during infection. This may be a general feature of T3S-associated GAP proteins, since two homologues from Pseudomonas aeruginosa, exoenzyme S (ExoS) and exoenzyme T (ExoT), can complement the hyper-translocation phenotypes of the yopE GAP mutant.
YopE of Yersinia pseudotuberculosis inactivates three members of the small RhoGTPase family (RhoA, Rac1 and Cdc42) in vitro and mutation of a critical arginine abolishes both in vitro GTPase-activating protein (GAP) activity and cytotoxicity towards HeLa cells, and renders the pathogen avirulent in a mouse model. To understand the functional role of YopE, in vivo studies of the GAP activity in infected eukaryotic cells were conducted. Wild-type YopE inactivated Rac1 as early as 5 min after infection whereas RhoA was down regulated about 30 min after infection. No effect of YopE was found on the activation state of Cdc42 in Yersinia-infected cells. Single-amino-acid substitution mutants of YopE revealed two different phenotypes: (i) mutants with significantly lowered in vivo GAP activity towards RhoA and Rac1 displaying full virulence in mice, and (ii) avirulent mutants with wild-type in vivo GAP activity towards RhoA and Rac1. Our results show that Cdc42 is not an in vivo target for YopE and that YopE interacts preferentially with Rac1, and to a lesser extent with RhoA, during in vivo conditions. Surprisingly, we present results suggesting that these interactions are not a prerequisite to establish infection in mice. Finally, we show that avirulent yopE mutants translocate YopE in about sixfold higher amount compared with wild type. This raises the question whether YopE's primary function is to sense the level of translocation rather than being directly involved in downregulation of the host defence.
The YopE cytotoxin of Yersinia is an essential virulence determinant that is translocated into the eukaryotic target cell via a plasmid-encoded type III secretion system. YopE possess a GTPase activating protein activity that in vitro has been shown to down regulate RhoA, Rac1, and Cdc42. Translocated YopE induces de-polymerisation of the actin microfilament structure in the eukaryotic cell which results in a rounding up of infected cells described as a cytotoxic effect. Here, we have investigated the importance of different regions of YopE for induction of cytotoxicity and in vitro GAP activity. Sequential removal of the N- and C-terminus of YopE identified the region between amino acids 90 and 215 to be necessary for induction of cytotoxicity. Internal deletions containing the essential arginine at position 144 resulted in a total loss of cytotoxic response. In-frame deletions flanking the arginine finger defined a region important for the cytotoxic effect to amino acids 166–183. Four triple-alanine substitution mutants in this region, YopE166-8A, 169-71A, 175-7A and 178-80A were still able to induce cytotoxicity on HeLa cells although they did not show any in vitro GAP activity towards RhoA, Rac1 or Cdc42. A substitution mutant in position 206-8A showed the same phenotype, ability to induce cytotoxic response but no in vitro GAP activity. We speculate that YopE may have additional unidentified targets within the eukaryotic cell.
Pathogenic Yersinia species suppress the host immune response by using a plasmid-encoded type III secretion system (T3SS) to translocate virulence proteins into the cytosol of the target cells. T3SS-dependent protein translocation is believed to occur in one step from the bacterial cytosol to the target-cell cytoplasm through a conduit created by the T3SS upon target cell contact. Here, we report that T3SS substrates on the surface of Yersinia pseudotuberculosis are translocated into target cells. Upon host cell contact, purified YopH coated on Y. pseudotuberculosis was specifically and rapidly translocated across the target-cell membrane, which led to a physiological response in the infected cell. In addition, translocation of externally added YopH required a functional T3SS and a specific translocation domain in the effector protein. Efficient, T3SS-dependent translocation of purified YopH added in vitro was also observed when using coated Salmonella typhimurium strains, which implies that T3SS-mediated translocation of extracellular effector proteins is conserved among T3SS-dependent pathogens. Our results demonstrate that polarized T3SS-dependent translocation of proteins can be achieved through an intermediate extracellular step that can be reconstituted in vitro. These results indicate that translocation can occur by a different mechanism from the assumed single-step conduit model.
The pathogenic yersiniae inject proteins directly into eukaryotic cells that interfere with a number of cellular processes including phagocytosis and inflammatory-associated host responses. One of these injected proteins, the Yersinia protein kinase A (YpkA), has previously been shown to affect the morphology of cultured eukaryotic cells as well as to localize to the plasma membrane following its injection into HeLa cells. Here it is shown that these activities are mediated by separable domains of YpkA. The amino terminus, which contains the kinase domain, is sufficient to localize YpkA to the plasma membrane while the carboxyl terminus of YpkA is required for YpkAs morphological effects. YpkAs carboxyl-terminal region was found to affect the levels of actin-containing stress fibers as well as block the activation of the GTPase RhoA in Yersinia-infected cells. We show that the carboxyl-terminal region of YpkA, which contains sequences that bear similarity to the RhoA-binding domains of several eukaryotic RhoA-binding kinases, directly interacts with RhoA as well as Rac (but not Cdc42) and displays a slight but measurable binding preference for the GDP-bound form of RhoA. Surprisingly, YpkA binding to RhoA(GDP) affected neither the intrinsic nor guanine nucleotide exchange factor-mediated GDP/GTP exchange reaction suggesting that YpkA controls activated RhoA levels by a mechanism other than by simply blocking guanine nucleotide exchange factor activity. We go on to show that YpkAs kinase activity is neither dependent on nor promoted by its interaction with RhoA and Rac but is, however, entirely dependent on heat-sensitive eukaryotic factors present in HeLa cell extracts and fetal calf serum. Collectively, our data show that YpkA possesses both similarities and differences with the eukaryotic RhoA/Rac-binding kinases and suggest that the yersiniae utilize the Rho GTPases for unique activities during their interaction with eukaryotic cells.
Mutations in CuZn-superoxide dismutase (SOD1) cause amyotrophic lateral sclerosis (ALS) and are found in 6% of ALS patients. Non-native and aggregation-prone forms of mutant SOD1s are thought to trigger the disease. Two sets of novel antibodies, raised in rabbits and chicken, against peptides spaced along the human SOD1 sequence, were by enzyme-linked immunosorbent assay and an immunocapture method shown to be specific for denatured SOD1. These were used to examine SOD1 in spinal cords of ALS patients lacking mutations in the enzyme. Small granular SOD1-immunoreactive inclusions were found in spinal motoneurons of all 37 sporadic and familial ALS patients studied, but only sparsely in 3 of 28 neurodegenerative and 2 of 19 non-neurological control patients. The granular inclusions were by confocal microscopy found to partly colocalize with markers for lysosomes but not with inclusions containing TAR DNA binding protein-43, ubiquitin or markers for endoplasmic reticulum, autophagosomes or mitochondria. Granular inclusions were also found in carriers of SOD1 mutations and in spinobulbar muscular atrophy (SBMA) patients and they were the major type of inclusion detected in ALS patients homozygous for the wild type-like D90A mutation. The findings suggest that SOD1 may be involved in ALS pathogenesis in patients lacking mutations in the enzyme.
Virulent Yersinia species cause systemic infections in rodents, and Y. pestis is highly pathogenic for humans. Pseudomonas aeruginosa, on the other hand, is an opportunistic pathogen, which normally infects only compromised individuals. Surprisingly, these pathogens both encode highly related contact-dependent secretion systems for the targeting of toxins into eukaryotic cells. In Yersinia, YopB and YopD direct the translocation of the secreted Yop effectors across the target cell membrane. In this study, we have analysed the function of the YopB and YopD homologues, PopB and PopD, encoded by P. aeruginosa. Expression of the pcrGVHpopBD operon in defined translocation-deficient mutants (yopB/yopD) of Yersinia resulted in complete complementation of the cell contact-dependent, YopE-induced cytotoxicity of Y. pseudotuberculosis on HeLa cells. We demonstrated that the complementation fully restored the ability of Y. pseudotuberculosis to translocate the effector molecules YopE and YopH into the HeLa cells. Similar to YopB, PopB induced a lytic effect on infected erythrocytes. The lytic activity induced by PopB could be prevented if the erythrocytes were infected in the presence of sugars larger than 3 nm in diameter, indicating that PopB induced a pore of similar size compared with that induced by YopB. Our findings show that the contact-dependent toxin-targeting mechanisms of Y. pseudotuberculosis and P. aeruginosa are conserved at the molecular level and that the translocator proteins are functionally interchangeable. Based on these similarities, we suggest that the translocation of toxins such as ExoS, ExoT and ExoU by P. aeruginosa across the eukaryotic cell membrane occurs via a pore induced by PopB.
Neurogenesis has been shown to occur in the cerebral cortex in adult rats after ischemic stroke. The origin of the newborn neurons is largely unknown. This study aimed to explore cell division in the poststroke penumbral cortex. Adult male Wistar rats were subjected to photothrombotic ring stroke. After repeated delivery of the DNA duplication marker BrdU, the animals were sacrificed at various times poststroke. BrdU was detected by immunohistochemistry/immunofluorescence labeling, as was the M-phase marker Phos H3 and the spindle components alpha-tubulin/gamma-tubulin. DNA damage was examined by TUNEL staining. Cell type was ascertained by double immunolabeling with the neuronal markers Map-2ab/beta-tubulin III and NeuN/Hu or the astrocyte marker GFAP. From 16h poststroke, BrdU-immunolabeled cells appeared in the penumbral cortex. From 24h, Phos H3 was colocalized with BrdU in the nuclei. Mitotic spindles immunolabeled by alpha-tubulin/gamma-tubulin appeared inside the cortical cells containing BrdU-immunopositive nuclei. Unexpectedly, the markers of neuronal differentiation, Map-2ab/beta-tubulin III/NeuN/Hu, were expressed in the Phos H3-immunolabeled cells, and NeuN was detected in some cells containing spindles. This study suggests that in response to a sublethal ischemic insult, endogenous cells with neuronal immunolabeling may duplicate their nuclear DNA and commit cell mitosis to generate daughter neurons in the penumbral cortex in adult rats.
Introduction of anti-host factors into eukaryotic cells by extracellular bacteria is a strategy evolved by several Gram-negative pathogens. In these pathogens, the transport of virulence proteins across the bacterial membranes is governed by closely related type III secretion systems. For pathogenic Yersinia, the protein transport across the eukaryotic cell membrane occurs by a polarized mechanism requiring two secreted proteins, YopB and YopD. YopB was recently shown to induce the formation of a pore in the eukaryotic cell membrane, and through this pore, translocation of Yop effectors is believed to occur (Håkansson et al., 1996b). We have previously shown that YopK of Yersinia pseudotuberculosis is required for the development of a systemic infection in mice. Here, we have analysed the role of YopK in the virulence process in more detail. A yopK-mutant strain was found to induce a more rapid YopE-mediated cytotoxic response in HeLa cells as well as in MDCK-1 cells compared to the wild-type strain. We found that this was the result of a cell-contact-dependent increase in translocation of YopE into HeLa cells. In contrast, overexpression of YopK resulted in impaired translocation. In addition, we found that YopK also influenced the YopB-dependent lytic effect on sheep erythrocytes as well as on HeLa cells. A yopK-mutant strain showed a higher lytic activity and the induced pore was larger compared to the corresponding wild-type strain, whereas a strain overexpressing YopK reduced the lytic activity and the apparent pore size was smaller. The secreted YopK protein was found not to be translocated but, similar to YopB, localized to cell-associated bacteria during infection of HeLa cells. Based on these results, we propose a model where YopK controls the translocation of Yop effectors into eukaryotic cells.
The virulence plasmid common to pathogenic Yersinia species encodes a number of secreted proteins denoted Yops (Yersinia outer proteins). Here, we identify and characterize a novel plasmid-encoded virulence determinant of Yersinia pseudotuberculosis, YopK. The yopK gene was found to be conserved among the three pathogenic Yersinia species and to be homologous to the previously described yopQ and yopK genes of Y. enterocolitica and Y. pestis, respectively. Similar to the other Yops, YopK expression and secretion were shown to be regulated by temperature and by the extracellular Ca2+ concentration; thus, yopK is part of the yop regulon. In addition, YopK secretion was mediated by the specific Yop secretion system. In Y. pseudotuberculosis, YopK was shown neither to have a role in this bacterium's ability to resist phagocytosis by macrophages nor to cause cytotoxicity in HeLa cells. YopK was, however, shown to be required for the bacterium to cause a systemic infection in both intraperitoneally and orally infected mice. Characterization of the infection kinetics showed that, similarly to the wild-type strain, the yopK mutant strain colonized and persisted in the Peyer's patches of orally infected mice. A yopE mutant which is impaired in cytotoxicity and in antiphagocytosis was, however, found to be rapidly cleared from these lymphoid organs. Neither the yopK nor the yopE mutant strain could overcome the primary host defense and reach the spleen. This finding implies that YopK acts at a different level during the infections process than the antiphagocytic YopE cytotoxin does.
Recent work has shown that a domain of YopE of Yersinia pseudotuberculosis ranging from amino acids 54 to 75 (R. Krall, Y. Zhang, and J. T. Barbieri, J. Biol. Chem. 279:2747-2753, 2004) is required for proper localization of YopE after ectopic expression in eukaryotic cells. This domain, called the membrane localization domain (MLD), has not been extensively studied in Yersinia. Therefore, an in cis MLD deletion mutant of YopE was created in Y. pseudotuberculosis. The mutant was found to secrete and translocate YopE at wild-type levels. However, the mutant was defective in the autoregulation of YopE expression after the infection of HeLa cells. Although the mutant translocated YopE at wild-type levels, it showed a delayed HeLa cell cytotoxicity. This delay was not caused by a change in GTPase activating protein (GAP) activity, since the mutant showed wild-type YopE GAP activity toward Rac1 and RhoA. The MLD mutant displayed a changed intracellular localization pattern of YopE in HeLa cells after infection, and the YopEDeltaMLD protein was found to be dispersed within the whole cell, including the nucleus. In contrast, wild-type YopE was found to localize to the perinuclear region of the cell and was not found in the nucleus. In addition, the yopEDeltaMLD mutant was avirulent. Our results suggest that YopE must target proteins other than RhoA and Rac1 and that the MLD is required for the proper targeting and hence virulence of YopE during infection. Our results raise the question whether YopE is a regulatory protein or a "true" virulence effector protein.
Vibrio anguillarum is an invasive pathogen of fish causing a septicaemia called vibriosis. In this work, transparent zebrafish were immersed in water containing green fluorescent protein labelled V. anguillarum. The infection was visualised at the whole fish and single bacterium levels using microscopy. The gastrointestinal tract was the first site where the pathogen was detected. This enteric localisation occurred independently of the flagellum or motility. On the other hand, chemotactic motility was essential for association of the pathogen with the fish surface. In conclusion, the zebrafish infection model provides evidence that the intestine and skin represent sites of infection by V. anguillarum and suggests a host site where chemotaxis may function in virulence.
YopH is translocated by cell-surface-bound bacteria through the plasma membrane to the cytosol of the HeLa cell. The transfer mechanism is contact dependent and polarizes the translocation to only occur at the contact zone between the bacterium and the target cell. More than 99% of the PTPase activity is associated with the HeLa cells. In contrast to the wild-type strain, the yopBD mutant cannot deliver YopH to the cytosol. Instead YopH is deposited in localized areas in the proximity of cell-associated bacteria. A yopN mutant secretes 40% of the total amount of YopH to the culture medium, suggesting a critical role of YopN in regulation of the polarized translocation. Evidence for a region in YopH important for its translocation through the plasma membrane of the target cell but not for secretion from the pathogen is provided.
Here we show that cells lacking the heme-regulated inhibitor (HRI) are highly resistant to infection by bacterial pathogens. By examining the infection process in wild-type and HRI null cells, we found that HRI is required for pathogens to execute their virulence-associated cellular activities. Specifically, unlike wild-type cells, HRI null cells infected with the gram-negative bacterial pathogen Yersinia are essentially impervious to the cytoskeleton-damaging effects of the Yop virulence factors. This effect is due to reduced functioning of the Yersinia type 3 secretion (T3S) system which injects virulence factors directly into the host cell cytosol. Reduced T3S activity is also observed in HRI null cells infected with the bacterial pathogen Chlamydia which results in a dramatic reduction in its intracellular proliferation. We go on to show that a HRI-mediated process plays a central role in the cellular infection cycle of the Gram-positive pathogen Listeria. For this pathogen, HRI is required for the post-invasion trafficking of the bacterium to the infected host cytosol. Thus by depriving Listeria of its intracellular niche, there is a highly reduced proliferation of Listeria in HRI null cells. We provide evidence that these infection-associated functions of HRI (an eIF2 alpha kinase) are independent of its activity as a regulator of protein synthesis. This is the first report of a host factor whose absence interferes with the function of T3S secretion and cytosolic access by pathogens and makes HRI an excellent target for inhibitors due to its broad virulence-associated activities.
The YopE cytotoxin of Yersinia pseudotuberculosis is an essential virulence determinant that is injected into the eukaryotic target cell via a plasmid-encoded type III secretion system. Injection of YopE into eukaryotic cells induces depolymerization of actin stress fibres. Here, we show that YopE exhibits a GTPase-activating protein (GAP) activity and that the presence of YopE stimulates downregulation of Rho, Rac and Cdc42 activity. YopE has an arginine finger motif showing homology with those found in other GAP proteins. Exchange of arginine 144 with alanine, located in this arginine finger motif, results in an inactive form of YopE that can no longer stimulate GTP hydrolysis by the GTPase. Furthermore, a yopE(R144A) mutant is unable to induce cytotoxicity on cultured HeLa cells in contrast to the corresponding wild-type strain. Expression of wild-type YopE in cells of Saccharomyces cerevisiae inhibits growth, while in contrast, expression of the inactive form of YopE, YopE(R144A), does not affect the yeast cells. Co-expression of proteins belonging to the Rho1 pathway of yeast, Rho1, Rom2p, Bck1 and Ste20, suppressed the growth phenotype of YopE in yeast cells. These results provide evidence that YopE exhibits a GAP activity to inactivate RhoGTPases, leading to depolymerization of the actin stress fibres in eukaryotic cells and growth inhibition in yeast.
Pathogenic Yersinia spp. possess a protein secretion system, designated as type 3, that plays a clear role in promoting their survival vis-à-vis the macrophage. Inductive expression of the Yersinia type 3 secretion system (T3SS), triggered either by host cell contact, or, in the absence of host cells, by a reduction in extracellular calcium ion levels, is accompanied by a withdrawal from the bacterial division cycle. Here, we analyzed Ca2+-dependent induction of the T3SS at the single-cell level to understand how Yersinia coordinates pro-survival and growth-related activities. We utilized a novel high-throughput quantitative microscopy approach as well as flow cytometry to determine how Ca2+ levels, T3SS expression, and cellular division are interrelated. Our analysis showed that there is a high degree of homogeneity in terms of T3SS expression levels among a population of Y. pseudotuberculosis cells following the removal of Ca2+, and that T3SS expression appears to be independent of the cellular division cycle. Unexpectedly, our analysis showed that Ca2+ levels are inversely related to the initiation of inductive T3SS expression, and not to the intensity of activation once initiated, thus providing a basis for the seemingly graded response of T3SS activation observed in bulk-level analyses. The properties of the system described here display both similarities to and differences from that of the lac operon first described 50 years ago by Novick and Weiner.


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Shag lessons and dance
At 7 p.m. on Mondays, under the Pavilion at the Summerville Country Club, 400 Country Club Drive, there will be Shag Lessons with an Open Dance after. For information call Lee Hoff, 843-460-5416.
Annual Turkey Shoot
From 7-11 p.m. Thursday through Saturday now until Dec. 2, the American Legion Post 166, Howe Hall Road, Goose Creek runs their Turkey Shoot. The public is invited. This is a family atmosphere in a secure location. Bon fire to enjoy, with food and beverages from the canteen. No outside beverages allowed. Junior Turkey Shoot for youngsters 12 and under begins Oct. 5 at 5 p.m. All monies raised go to service programs. For information call 843-553-5454 or visit www.alp166sc.com.
Summerville Farmers Market
From 8 a.m. – 1 p.m. on Saturday at Summerville Town Hall, 200 S. Main Street, Summerville, browse fresh local produce, meats, seafood and baked goods at the Summerville Farmers Market located in the First Citizens Bank parking lot behind Town Hall. This event is every Saturday until Nov. 23.
Wellness Fair
From 10 a.m. – 1 p.m. on Oct. 19 at the Green Barn, Carnes Crossroads, 207 1st Avenue, Summerville, there will be products and resources available at the 2nd annual Health and Wellness Fair. More than 30 exhibitors demonstrating along with fitness classes, obstacle course for kids, puppy yoga and pet adoptions. Blood donations will be available. Admission and parking are free.
Revival at Pinopolis UMC
At 7 p.m. on Oct. 20, 21 and 22, Pinopolis United Methodist Church, 1833 Pinopolis Rd., Pinopolis United Methodist Church will host a revival in the fellowship hall each night. Each night will bring a new message from a new pastor. They are Pastor Tory Liferidge of Grace Reformed Episcopal Church, Pastor Doug Benton, Pinopolis UMC and Pastor Tim Dorn from Pointe North Church. All are invited.
Fall Concert at Cane Bay High School
At 7 p.m. on Oct. 21 at Cane Bay High School, 1624 State Road, Summerville, The Cobra Chorale and Ladies Honor Choir, Cane Bay Middle School and Sangaree Middle School will have their fall concert. Tickets are $5 and parking is free. Doors open at 6:30 p.m.
Art Talk with artist Belle Anderson
At 6 p.m. on Oct. 21 at the Goose Creek Library, 325 Old Moncks Corner Rd, Goose Creek, the Berkeley County Library invites families and adult citizens to participate in a presentation with The Goose Creek Cultural Arts Commission with artist Belle Anderson. Anderson is a self-taught, award winning artist that specializes in painting Lowcountry scenery
Chuck Wagon Gang in concert
At 10 a.m. on Oct. 24 at the First Emmanuel Baptist Church, 10665 Dorchester Road, the Chuck Wagon Gang will perform. This famous gospel quartet known for acoustic accompaniment and close harmonies was founded in 1935 by Dad Carter. This free concert will help celebrate the church's homecoming of the 82 years in Summerville area.
Halloween in the Swamp
From 7:30 – 10:30 p.m. on Oct. 25 and 26 at Cypress Gardens, 3030 Cypress Gardens Road, Moncks Corner the first Halloween Swamp Festival since 2014 will occur. For children and adults of all ages, this event will include music, food vendors, campfire, marshmallow roasts, storytelling jump castle and a lighted pumpkin trail. The Haunted Swamp Experience (age 12 and up) boat ride through the swamp is $15 which includes general admission. General admission is $5 per person (5 and up). For information call 843-553-0515.
I Heart Hungry Kids
From 1-3 p.m. on Oct. 27 at the Lowcountry Food Bank, 2864 Azalea Drive, N. Charleston, there will be an I Heart Hungry Kids packing party. For ages 5-18 and awesome adults. Come dressed for Halloween to help kids in need. This packing party is created for youth volunteers to teach kids the power of hands-on service and its benefit to others. There will be music, healthy snacks and lots of action and fun for the entire family. Registration is free.
Best Friends Festival
From 5-9 p.m. on Nov. 2 at Doty Park, 320 Laurel Street, Summerville, the Best Friends Festival will celebrate things which bring us together and move us forward. Community, ecology, equality, diversity and mobility. The is to mark the first phase to bring the lowcountry together and build a sustainable, inclusive and creative mobile future. Music, entertainment, ideas, food and a special circle to meet the candidates for elective office. For information call Linda 843-873-2436.
Marine Corps celebration
On Nov. 8 on board the USS Yorktown moored at the Maritime and Naval Museum atPatriots Point, 40 Patriots Point Rd, Mt. Pleasant, the LowCountry Leathernecks invites you to attend the 244th birthday celebration of the United States Marine Corps. Contact Dave Sowers, 843-697-9935 or email firstname.lastname@example.com or visit LowCountry Leathernecks' Facebook page for more details.
Art Talk with author Michael T. Owens
At 6 p.m. on Nov. 18 at at the Goose Creek Library, 325 Old Moncks Corner Rd, Goose Creek, the Berkeley County Library invites families and adult citizens to participate in a presentation with The Goose Creek Cultural Arts Commission with local author Michael T. Owens. Owens is the author of "Burned: Conversations with a Black WWII Veteran," the story of an Aftrican-american WWII Veteran who was subjected to mustard gas testing. Owens also created an oral history course at the College of Charleston and a non-profit entitled Voices Speak that is dedicated to 'giving voices to the voiceless' using novels or other media platforms.
Tax-Aide Volunteers needed
Here's a chance for you to give back to your community — AARP Foundation Tax-Aide need volunteers for next year to assist in preparing free income taxes for the elderly and others who needs help with their income taxes in Goose Creek, Summerville and Moncks Corner. There are also leadership positions available such as Client Facilitators. Training is provided on a self-paced basis with individual assistance as needed along with on-line IRS training and testing. Use of your own laptop may be necessary. There is one week orientation in December and two weeks of training in January. We ask volunteers to be ready to provide one half day or more per week from February 2nd through April 15th, 2020. For more information, contact Pat Merriam at email@example.com or 843-270-7881.
Military PTSD Support
Fellowship of veterans from all wars and conflicts, who want to provide free anonymous help to veterans, families, friends. We offer understanding, support, guidance and coping skills for those affected by PTSD resulting from combat or traumatic military experiences. Not a therapy group, but self-help/support group. 7 p.m. Mondays at Fellowship of Oakbrook, 1400 Trolley Road. 843-637-6463 or 843-509-0535.
Home Rehabilitation Help
Ridgeville Community Resource Center is participating with S.C. Housing Trust Fund to aid low-income homeowners with repairs. Counties served include Dorchester, Berkeley, Colleton and Orangeburg. Location: 108 Dorchester St., Ridgeville, or call between 10 a.m. and 1 p.m. Mondays and Wednesdays. Call Angie Crum at 873-5384 or Christopher Smith at 693-8025.
Quilting Friends has changed its name to Quilters2. Meets every Wednesday 10 a.m. to 3 p.m. at Stallsville Methodist Church. We welcome quilters new to the area, and new quilters. Contact Kathy at 843-771-9342 for information.
Free Lunch
Free soup, sandwiches, every Tuesday, noon to 1 p.m. Seventh Day Adventist Church, 520 Gahagan Road. 843-821-4787.
Addiction recovery ministry
Are your habits hurting you? Are they destroying those you love? Addiction Recovery Ministry, 7 p.m. Fridays at Ladson Baptist Church, 3231 Ladson Road. Call 843-801-1618 (Ted Caudill, director) or 843-797-1014 (church).
Citizens for Life
Trident Area Citizens for Life meets 7-8 p.m. on first Monday of each month at the meeting center at St. Theresa Catholic Church. Open to pro-life individuals. Contact Barbara Weigand at email@example.com.
Head Injury Support
The Trident Head Injury Support Group supports survivors and caregivers dealing with brain injury. Consists of families, professionals, and resource information. Meets for dinner at 6:30 p.m., meeting at 7 p.m. the first Tuesday of each month. New location: Bon Secours St. Francis Hospital in West Ashley, 2095 Henry Tecklenburg Drive, in mall classroom 1. For information go to thisg.net or call 843-822-1967.
Credit Improvement Workshop
Learn to manage and understand a credit report and discover the positive steps you can take to improve your credit score. In this free workshop by Origin SC you will learn strategies to control your day-to-day finances, find out what creditors are looking for when you apply for credit, how to dispute inaccurate items on your credit report, and more. Meets the first Tuesday of every month (excluding holidays) at 6 p.m. at 4925 Lacross Rd. Suite 215, North Charleston. Register and secure your spot today. Call 843-735-7802.
Submarine Veterans
U.S. Submarine Veterans Charleston Base meets on second Thursday of every month at the Fleet Reserve Association home at 99 Wisteria Dr., Goose Creek. Social hour starts at 6 p.m.; meeting at 7 p.m. Call Ed Stank, membership chair, at 568-2148 or email email@example.com.
NAACP meets
NAACP Summerville Branch meets at 7 p.m. second Thursday of each month in County Council Chambers, 500 N. Main St. Enter by Cedar Street entrance.
Making Ends Meet
In this workshop by Origin SC you'll learn basic budgeting techniques and day-to-day money management strategies. Topics include creating a budget, managing credit, avoiding identity theft, and more. Plus, it's free. Meets the second Thursday of every month (excluding holidays) at 6 p.m. at 4925 Lacross Rd. Suite 215, North Charleston. Register and secure your spot today. Call 843-735-7802.
Dementia support groups
The ARK, Alzheimer's Family Support Services, offers area support groups that meet monthly in a therapeutic environment facilitated by a trained leader. See thearkofsc.org or 843-832-2357. Second Thursday of month at 7 p.m. at Grace Lutheran Church, 1600 Old Trolley Road.
Alzheimer's Caregiver Wives Group: Third Tuesday of month at 11 a.m. at St. Luke's Lutheran Church, 206 Central Ave.
Embroiderers group
Magnolia Chapter of The Embroiderers Guild of America meets 1 p.m. second Sunday each month at Miner Crosby Community Center, 3901 Paramount Dr., North Charleston.
Compassionate Friends
Compassionate Friends of Summerville offers comfort and hope for bereaved parents, grandparents and siblings after the death of a child from any cause, at any age. Our meeting schedule has changed to the third Monday of each month, 7-8:30 p.m., Episcopal Church of the Good Shepherd, 119B West Luke Ave. Call Becky: 770-568-9718 or Melissa: 843-934-4804.
Mental Illness support
National Alliance on Mental Illness Charleston Area support meetings for individuals recovering from brain disorder/mental illness and families on third Monday of each month at Bethany United Methodist Church, 118 W. 3rd St. South. Call Joann at 843-871-1009, email email@example.com or visit www.nami.org/sites/namicharlestonarea.
Model Builders
South Carolina Modelers Association / Charleston chapter meeting, third Tuesday, each month at 7 p.m. at the First Church of God meeting hall, 10383 Highway 78, Summerville. Contact: Ron Minton at 843-637-9099 firstname.lastname@example.com.
Pine Hill Acres Crimewatch
Join neighbors for the monthly Pine Hill Acres Crimewatch meeting, on the third Wednesday of every month at the ballfield at the end of Lake Drive. Block captains meet at 7 p.m., regular meeting at 7:30 p.m. Call Tom Hall Jr. at 843-714-0984.
Fleet Reserve Association
Fleet Reserve Association (Navy, Marines and Coast Guard) conducts the Branch 269 monthly meeting at 7 p.m. on the third Thursday of each month.
Any member of the seagoing services are welcome to attend. Contact John Tharp at 843-709-8520.
Scottish American Military
Scottish American Military Society meets third Saturday of the month at 11 a.m. at VFW Post 3433, Bellwright Road, Ladson. Prior active duty and Reserve including Coast Guard are invited if they are of Scottish, Welsh or Irish heritage. Go to S-A-M-S.org.
Knitting Guild
The Flowertown Knitting Guild meets on the fourth Tuesday of every month at 6 p.m. in the Fellowship Hall of St. Luke's Lutheran Church, 206 Central Ave. All knitters, new and experienced, are invited to join us. Find out about the knitting opportunities in the area, charity knitting, and programs. Come see old friends, make new friends and enjoy the fellowship. Contact Kaye Heins at 843-793-9204 or firstname.lastname@example.com.
A few good Marines
Lowcountry Detachment of the Marine Corps League seeks Marines who have served honorably in Corps for not less than 90 days and earned Eagle, Globe and Anchor, FMF Corpsmen and FMF Navy Chaplains. Lowcountry Detachment meets on fourth Thursday of the month at Fleet Reserve Association, 99 Wisteria Road, Goose Creek. Call John Tharp at 843-709-8520.
Painters group meeting
The Charleston Chapter of Decorative Painters, invites those interested in exploring this art form. Meetings are on the fourth Saturday of each month 9 a.m. to 1 p.m. at St. Thomas Episcopal Church, 1150 E. Montague Ave, North Charleston. Contact Catherine Jones 843- 327-1299.
Al-Anon Family Groups are a fellowship of relatives and friends of alcoholics who share experience, strength and hope to solve problems. Local meetings:
Mondays: 9:30 a.m. Madden Hall. St. John the Beloved Church. 28 Sumter Ave. Summerville.
Mondays: 9:30 a.m. Step Up Club. 171 Old Parsonage Rd. Knightsville.
Mondays: 7 p.m. Seventh Day Adventist Church 520 Gahagan Rd. Summerville.
Mondays: 7 p.m. Al Ateen meeting. Seventh Day Adventist Church.
Wednesdays: 8 p.m. Step Up Club.
Thursdays: 11:30 a.m. Skardon Hall. St. Paul's Church. 111 Waring St. Summerville.
Fridays: 7 p.m. Step Up Club.
Saturdays: 11 a.m. Honey Ridge villas. Oakmont Road. Oakbrook.
Sundays: 8 p.m. Step Up Club.


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Alexander Skarsgard attended the Rock and Roll Republic event by Page Six. Images in the gallery below are from this event held on 9/9/09.
[nggallery id=147]
The coq deserves his chance,by the way eastmond looks to have matured from the eastmond of a few seasons back.
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http://www.w3.org/Bugs/Public/show_bug.cgi?id=10830 --- Comment #23 from Addison Phillips <firstname.lastname@example.com> 2011-08-09 16:38:30 UTC --- (In reply to comment #22) > You have already asked the editor to reconsider. You can't both do that *and* > request to open a tracker issue. Please read the link that was provided. Please note: The I18N WG has not yet considered the editor's response. -- Configure bugmail: http://www.w3.org/Bugs/Public/userprefs.cgi?tab=email ------- You are receiving this mail because: ------- You are the QA contact for the bug.Received on Tuesday, 9 August 2011 16:38:33 UTC
This archive was generated by hypermail 2.4.0 : Friday, 17 January 2020 20:02:00 UTC


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PHOENIX -- In his State of the Yankees address Wednesday, Hal Steinbrenner singled out all of the right suspects, absolved all of the innocent parties and threw at least a partial bone to frustrated Yankees fans who have been desperate for any shred of evidence that he is indeed a blood relation to George M. Steinbrenner III.
The only thing the New York Yankees owner didn't do was point a finger in the direction it belongs: at himself.
Steinbrenner was right to name Mark Teixeira, Chase Headley, Michael Pineda and Luis Severino as the chief culprits in the Yankees' horrible start. The team is six games under .500 and in the cellar of the American League East, 7½ games behind the division-leading Baltimore Orioles entering Wednesday night's meeting with the Arizona Diamondbacks.
And Steinbrenner was equally justified in exonerating his manager, Joe Girardi, who can neither hit, pitch, run nor field for the men on his highly paid roster, and his general manager, Brian Cashman, who like all GMs acts strictly in accordance with the wishes of his boss.
Where he went wrong, however, is in not shouldering the blame himself. The reason is not because he did not spend enough money -- the Yankees' 2016 payroll is $225 million, up $8 million from last season and second only to the Los Angeles Dodgers' $249 million -- or because he did not pursue the handful of desirable free agents on the market this past winter. (Can you imagine the fans' reaction if the Yankees had given David Price seven years and $217 million?)
The reason is that for all his positive qualities as an owner, Hal Steinbrenner has not been truly honest with Yankees fans. Whether he, Cashman or Girardi would dare admit it, Steinbrenner is not all in it to win it this season, nor was he last season.
Yes, he is trying to put a competitive team on the field, because he has seats to fill, debt service on the Stadium to pay off and cable TV ratings to bolster.
But we all know that for the past two seasons, the Yankees have been in a holding pattern, waiting for some of their onerous contracts to expire, such as those of Teixeira and Carlos Beltran after this season, and those of Alex Rodriguez and CC Sabathia following 2017. We know that Steinbrenner would love to get under the luxury-tax threshold, which will be renegotiated in the next collective bargaining agreement. And we can reasonably assume he is looking forward to the day he can take all the money that will be freed up from those expiring deals and invest it in Bryce Harper, the closest thing the Yankees will ever see to a free agent in his prime when he hits the market after the 2018 season.
So in addition to naming Teixeira, Headley, Pineda and Severino on Wednesday at baseball's quarterly owners meetings in New York, it would have been a good idea -- and a gesture of good faith with the fans -- for Hal Steinbrenner to drop a dime on himself.
Just come out and say what is clear as day to any discerning fan: That the New York Yankees, proud owners of 27 world championships and the self-bestowed soubriquet of "The World's Greatest Sports Franchise," are probably not built to win this year. Or next year, for that matter. In fact, they are in a rebuilding stage, or as close as they can ever come to rebuilding while still keeping a hold on one of the most demanding fan bases in all of professional sports.
And the truth is, mistakes were made in the construction of this roster and the allocation of the considerable sums of money at the Yankees' disposal.
It was a lousy idea to give Rodriguez 10 more years when he opted out of his first deal in 2007. It was a huge risk, and one Cashman has been publicly leery of for years, to give a seven-year contract to a starting pitcher, any starting pitcher, like the one they gave to Masahiro Tanaka two years ago. It was sheer insanity, and probably some guilt-ridden compensation, to give Jacoby Ellsbury seven years and $153 million after the Yankees (read: Hal Steinbrenner) chose to let Robinson Cano leave as a free agent. Beltran's deal never seemed like a good idea considering he would be 39 in its final season.
And you can debate the merits all you want of Teixeira's eight-year, $180 million deal and Sabathia's renegotiated, five-year, $122 million deal with a vesting option for another $25 million for 2017, since both were part of the last Yankees team to win a world championship.
But the truth about those deals -- and really, just about any free-agent deal -- is that while they might provide value in the early years, they are all bound to become albatrosses and huge wastes of money as the players age out of their productive years while continuing to be paid as superstars.
Hal Steinbrenner signed off on all those deals and, presumably, put the kibosh on others. So if there is anyone who should assume the responsibility for the flaws on his roster and the dead money on his books, it is him.
You can make the fair argument that the Yankees' player-development department has failed to develop enough young starting pitchers -- Severino is only the latest disappointment on a list that includes Manny Banuelos, Andrew Brackman and even Dellin Betances, who once projected as a starter -- and really, no everyday players at all other than Brett Gardner, now that Cano is gone. But to be truly fair, you also must recognize that Hal Steinbrenner eventually recognized the weakness of his player-development department a year ago and made some changes about which it is still too early to judge.
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You can point the finger of blame at those players for underperforming, or you can point it at the man who thought they were worth investing in.
That man was Hal Steinbrenner, and while he was pointing fingers today, the right thing to do would have been to point one at himself, too.


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Match detail: Viktoria Zizkov v Chelsea
Match Date: Thursday, 29th Sep 1994 at 19:45
Competition: Europe - European Cup Winners Cup - 1st Round - 2nd Leg
Opponent: Viktoria Zizkov
Venue: FK Viktoria Stadion
Attendance: 5,176
Half Time: 0 - 0
Result: Drew 0 - 0
Referee: Gianni Beschin (Italy)
Anthony Barness
Steve Clarke
Paul Furlong
Erland Johnsen
Dmitri Kharine (GK)
Eddie Newton
Gavin Peacock
Graham Rix
David Rocastle
Frank Sinclair
Dennis Wise (C)
|Chelsea Goal Scorers:
Viktoria Zizkov Goal Scorers:
Return to Match Selector
Return to Season Selector.
Return to Competition Selector.
Return to Home page.


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OWEC is dedicated to advancing equity for Oregon's women and girls. We hold space for organizations and individuals who serve women and girls to come together so they can share, learn, connect, and use our collective voice to advocate for equitable policies. Together we will be able to create a more equitable state for Oregon women and girls. Join us.
OWEC Winding Down
We are proud of the work we have accomplished together. Our initial focus was on developing the first statewide report on the status of women and girls in Oregon in over 20 years, and after that project was absorbed by the (then) newly formed Women's Foundation of Oregon, we shifted our focus to policy advocacy. We held some well-attended legislative briefs and panels on urgent topics. And, we took positions on key legislation designed to advance economic equity and reproductive justice. We are proud that we were able to create space for important conversations and bring nonprofit leaders and elected leaders together.
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Oregon has one of the highest rates of gender based violence in the country, with over half of the state's female population having experienced some form of sexual or domestic violence.
Hundreds of thousands of women lack access to reproductive health information and services.


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MKE Ankaragucu are set to resume their interest in West Brom man Roman Bednar.
The Baggies striker played eight times, scoring once, during a loan spell at the Turkish club in the latter half of last season.
Bednar, who has a year left, was kept on by Albion for an extra year to retain some of his transfer value.
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"He loves us, we love him very much. He is an aggressive and strong player."


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Guidelines for use of the logo.
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The Fetal and Maternal Imaging Unit of the Perinatology Research Branch [of the National Institutes of Health / Eunice Kennedy Shriver National Institute of Child Health and Human Development (NIH/NICHD)] and Wayne State University (WSU) in Detroit, Michigan is seeking a qualified faculty member to become its Director. Such individual should have expertise in obstetrical and gynecologic ultrasound, as well as a strong commitment to academic excellence, innovation, and interest in the conduction of original research, education, and training. Additional experience in fetal surgery is welcome, but not a requirement.
The Unit conducts advanced research of the fetus and mother utilizing state-of-the-art equipment and technology, including three- and four-dimensional imaging, MRI and other sonographic modalities. There is access to a high-risk patient population, research protocols for the study of fetal growth and anatomy using ultrasound and MRI, a team dedicated to the pursuit of excellence, and the availability of worldwide collaborations. The Unit also has a training program in obstetrical sonography, and an established track record of publications, innovation, and patents. The Perinatology Research Branch is housed in Detroit, Michigan, and works in close collaboration with Wayne State University, and the Detroit Medical Center. Collaborations also exist with the University of Michigan and Baylor College of Medicine.
The successful applicant should have an M.D. or M.D./Ph.D. degree, have completed residency training in Obstetrics and Gynecology (at minimum), be at the Associate to full Professor level, and have demonstrated experience and expertise in fetal anatomic evaluation, fetal biometry, Doppler sonography, elastography, and volumetric sonography. The applicant is expected to participate in the training of residents, fellows, and other members of the research team. In addition, the applicant will supervise research sonographers and other research staff.
Finally, the applicant should have the academic credentials to direct a research program, and therefore, should be familiar with the concepts of study design, analysis, and reporting of research in manuscripts as well as other means of disseminating information in the scientific literature. Therefore, impeccable written and oral communication skills are required.


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Better Start Saving Up Now
There are not many events within the gaming world that cause so much joy and jubilation as Steam sales. The Steam Summer sale alone is a quasi-holiday within the gaming community. However, that does not mean that the other Steam sales are anything to scoff at. There are plenty of annual sales that Valve plans for its customers. The most notable ones aside from the Summer sale are the ones that take place in the Fall. Luckily for us, the date for the Steam Halloween, Autumn, and Winter sales have all been leaked. Now you'll at least be able to pinpoint when you'll be emptying your bank account on cheap video games.
- Halloween Steam Sale: October 28th to November 1st
- Autumn Steam Sale: November 26th to December 3rd
- Winter Steam Sale: December 19th to January 2nd
Despite these dates being leaked, it is not that big a surprise as they mostly line up with what Valve has done in previous years. If these dates remain the same then PC gamers are in for a lot of great deals in the coming months. While the Halloween sale is only going to last three days. The Autumn and Winter sales are going to be quite lengthy and are not far apart from one another. The Winter sale is going to be a great way to get ahold of some last-minute presents as well as a perfect way to blow all that Christmas cash on Boxing Day.
However, everyone should keep in mind that these dates have not been confirmed by anyone at Valve yet. But it is worth mentioning that the date for this year's Summer sale was leaked and it turned out to be correct.
Either way, great deals are on the horizon.


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PARLIAMENT OF AUSTRALIA HOUSE OF REPRESENTATIVES Speech WORKPLACE RELATIONS Matters Of Public Importance Tuesday, 24 February 2015 Clive Palmer – Member for Fairfax Mr PALMER (Fairfax) (16:06): It is in the public interest that we have proper governance of political parties. The Prime Minister took a major step in the right direction when, in his first cabinet meeting, he declared that members of the Liberal Party executive could not act as political lobbyists at the same time as they held office. A number of the Liberal Party executive resigned as a consequence of that decision. When I was a life member of the Liberal Party the funding of the Liberal Party, its application and those who funded it were of great concern to me. I was a life member of the Liberal Party and a member of its national conference. I had been a National Party official spokesman and a member of the conservative side of politics for over 40 years. Despite all of that, in 2011 and 2012, despite the fact that I had been one of the strongest supporters of the party and one of its largest donors, it was impossible to get a copy of the accounts of the party and a veil of secrecy hung over the application and use of party funds. At that time corporate governance was non-existent in the Liberal Party. Elected members of the national executive, holding the highest offices other than that of president of the party, complained personally to me that they were denied any access to the accounts of the party, about how money was donated to the party and how it was actually spent. There was no accountability which one would normally expect to see in a body receiving funding from the public and its members. The DEPUTY SPEAKER ( Mr Ewen Jones ): Order! I remind the member of the topic for discussion, which is the government's attack on the wages and conditions of working Australians. Mr PALMER: It is a wide-ranging topic. I believe I can discuss this. The DEPUTY SPEAKER: You may want to start! Mr PALMER: At that time Alan Stockdale was president of the party and refused point blank to provide details even to the vice-presidents of the Liberal Party. Mr Tudge: I rise on a point of order. He has just ignored your ruling for him to go back to the topic of the MPI. What he has been saying clearly has nothing to do with the topic. The topic actually concerns industrial relations. That is the matter which we are debating across this chamber. That is to do with an internal Liberal Party matter that he is discussing, so I ask that he brought back to the topic of the MPI. The DEPUTY SPEAKER: The member for Fairfax is well aware of what the topic is. I ask him to be relevant to the topic, or he can cede his position. Mr PALMER: Certainly industrial relations and the fact that Liberal Party workers went unpaid and worked not in accordance with the act was a great disgrace for the Liberal Party at that time. That is why many of its vice-presidents did not know where the funds were. They did not know where the money was to pay— Mr Tudge: I rise on a point of order. With due respect, the activities of the Liberal Party and what might or might not occur inside the Liberal Party have nothing to do with this matter of public importance, which concerns public policy concerning industrial relations. 98 The Last Sentry at the Gate: Clive Palmer & the 44th Parliament of Australia
THe Last Sentry at the Gate 1
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text: If you have not noticed, we have a wonderful new addition in the parking lot on the northwest side of the building. We have a Blessing Box. Our job is it to keep it full. Our community's job is to take what they need or bring items to add to it. Please check expiration dates and do not bring sharp objects!


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Showing 1-2 of 2
By: Cook, Frederick
Price: $225.00
Publisher: Knapp, Peck & Thomson Printers: 1887
Edition: First Edition
Seller ID: 2286587
Condition: Very Good
First edition. All maps included (5 in pockets, 6 in text, including 3 fold-outs). Front free endpaper loose but included, boards rubbed. 1887 Hard Cover. xv, [1], 579, [3] pp. Tall 8vo. Includes several fold-out maps, plus a pocket inside the front cover containing maps and plans. Engraved frontispiece of Sullivan, engraved portraits in text of Brigadier General James Clinton, Colonel Peter Gansevoort (from a portrait by Gilbert Stuart, famous for his portrait of George Washington), and Colonel Philip Van Cortlandt. Original brown cloth, gilt titles & rules, blind-stamped double border. John ... View more info
By: Cook, Frederick A.; Peary, Robert E.; Greely, A.W.; Miller, J. Martin
Price: $45.00
Publisher: J.T. Moss: 1909
Seller ID: 2260245
Condition: Very Good
Moderate loss of gilt from spine and front board, pages toned. 1909 Hard Cover. 428 pp. Tall 8vo. Includes the stories of Frederick Cook and Robert Peary in their quests for the North Pole, along with accounts of previous polar expeditions, and an introduction by A.W. Greely. Edited by J. Martin Miller. Illustrated with half-tone reproductions of expedition photographs. Arctic Bibliography 11467: Account of the two polar marches, drawn from contemporary journals and illustrated with cartoons of the period; also chapters on other famous explorations, on the Arctic in general, and on the controv... View more info


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2019 BMW 7 Series M760i Price and Release Date – BMW, however, has not yet made an M7, but alternatively on this site is the M760i xDrive practically to satisfy that wanting. Truly feel of it as the epidermis of a M5 extended lean above a big, twice turbo V-12-operate, all-wheel-drive 7-series. It is a crystal clear level of M close to a car increased for relocating the Norwegian Minister of Monetary to events of the International Financial Account. What is far more, comparable to Norway all on your own, it is fascinating nevertheless damaging to no one. BMW introduced us downward in this article to Palm Springs to discover their new 7 series. Be that as it could, this is not rather lately any everyday 7-series, this is the 2019 BMW M760i, the very first work by way of BMW M section has tuned the 7-series. it is a provided that any 7th series will probably be high-class areas with sufficient establish to dispatch a nuclear attack, on the reasons that the M section truly convert this to affect up so barge with sport car deserving of the fixed and recognition. BMW necessary to show that this is, so we are at the energy team to drive the car to it is quite cutoff on the track. Right after propelling an all-new 7 Series previous 6 cylinders, 8-tube and also a hybrid powertrain.
2019 BMW 7 Series M760i Redesign
BMW continue and it has not developed a M7, but instead this is the M760i drive basically in order to satisfy that demanding. Think about it the protect of a M5 expanded lean in excess of a huge, twin-turbo V-12-powered, all-wheel-force 7-build up. It is a slim addressing of M more than a car greater for moving the Norwegian Minister of Monetary to get-togethers in the Worldwide Monetary Account. Also, much like Norway on its own, it is eye-catching but frightening to no one. The constructing and chassis of the 2019 BMW M760i drive are for the most component basically like those of other 6th age 7-range cars, which BMW unveiled for 2019. So you will see carbon dioxide-fiber content strengthening elements (about which you are able to keep studying on this page) inside of the initial metallic unibody, although lightweight lightweight aluminum is used for vital stampings these sorts of as the hood, trunk area region, and top door pores and skin region. The whole caboodle-in-bright white-tinted colored is as noticeably as 190 pounds much less frustrating than was the just before 7-series progression.
Ignore the palm-touch techno-dope and low-obligatory aromatherapy atmospherics and go swimming in the sea of torque this V-12 creates. There is unremitting pull from really fairly recently far away from nonproductive right up until the stage when the car goes into the very low orbit in close proximity to Jupiter. The 590-lb-ft best smashes like a wave above the car when the engine is changing just 1550 rpm. The transmissions 8-10 components forward almost drown under the grunt. An engine suitable for Moves-Royces will continue to work in a digital hush. To possess that melodic area M powerplant requirements, the M760i can make usage of a toxic gases strategy that is produced out of typically primary funneling completing to a high gases merchants. The procedure is functional to the vacationing circumstances and may also be tuned by the driver in gentle of which drive method is picked out. Be that as it might, when you are earlier the sophisticated silencers and folds up and "charge pattern acoustics," BMW functions its trump cards: improvement.
In any event, whilst anyone can information sufficient strength into a main car and effect it to dispatch fast, what BMW and M have been found to be recognized for is the indicates where their cars take action on winding highways and competition monitors. Despite the simple fact that it is a large vehicle, its Carbon dioxide Central design boundaries excess weight up leading with the employment of layered aluminum and carbon dioxide dietary fiber in the body composition so the car's body weight is decreased, making money being careful of. To get over all the body weight, BMW employs every single one of the techniques in its playbook. Dynamic roll realignment makes use of digitally handled detaching roll cafes with the target that finest rigidity in a track establishing does not bring about a jarring journey more than irregular roadways. Rear-one-sided nevertheless quite factor xDrive all-wheel drive adapts torque in between the entrance and rear pivot, rear-tire directing aids with preparedness at lower rates of speed and solidness at high rates of speed, light-weight 20-" rims with Michelin Pilot Awesome Sport rubberized supply great hold an enormous 19-in. braking systems take all that volume downward from high rates of speed with confidence. Certainly, even with a complete daytime of sporadic track operate, the braking systems have been as however solid, displaying no symptoms of a blur.
Caused by the firm that garments the X6 M with 21-in. 285/35 entryways and 325/30 rear rubber, the tires on the M760i look to be comparatively small. And in addition, this restraint bodes effectively on the inside this sizeable vehicle's capability to be thrown in close proximity to a racetrack with a nice, to many diploma tail-out frameworks of the human brain. Be that as it may, well, these are extraordinary tastes of Teutonic terrorizing. The Mercedes S-kind is wide shouldered and erects where the BMW 7 is streamlined, considerably more catlike, and considerably much more private on the inside.
In spite of the simple fact that the BMW M760i xDrive has several doorways, this engine is nevertheless adequately highly effective to obtain it from 0-60 miles per hour in just 3.6 mere seconds. This snappy velocity is similarly expected to some extent to the 8-rate Steptronic Sport Automatic transmission and smart xDrive all-wheel-drive structure. Looking for the greatest rate? It would be struck a ruled velocity of 155 miles per hour. Within, the 2019 BMW 7 Series M760i Review sporty condition of thoughts earnings. The door ledge dishes are produced of well-brushed aluminum that has a lighted up V12 logo design, so you know when you wide open the door that it BMW Recommendation is pushing significant energy. With regards to the interior alone, BMW has equipped the car with discerning M Performance cutting, which includes the M leather material controls. Certainly, even the essential coxcomb for the 7 Series is driving the technical envelope, with a tiny display screen the period of an iWatch to manage bolting and lighting fixtures and in many cases comfortable the car up well before you arrive at it. In spite of the proven fact that not new or picky to the 7 Series, I appreciate BMW's numbered cutting corners that make it possible for coding of stereo station presets or beloved phone numbers or perhaps targets for course. The option is backed up by actual-time visitors information and facts that can help figure out the speediest path because of targeted traffic circumstances. Path Advice prompts are not constrained to the dashtop display screen, nevertheless, but performed in fantastic fine detail in the extensive complete-color mind-up show.
2019 BMW 7 Series M760i Engine and Specs
Regardless of the reality that the B7 features a V-8, that nutritional supplement-prepared twice turbocharged 4.4-liter engine is ranked in an even 600 hp and is positioned in what is generally a related 7-series xDrive car. All issues simply being the same, when the M760i's 590 lb-ft of torque crests course directly downward at 1550 rpm and keeps accessible to 5000 rpm, the B7 strikes its 590-lb-ft top rated at 3000 rpm, moreover keeping steady to 5000 rpm. BMW articulations the M760i will roar to 62 miles per hour in 3.7 sheer secs, which is most likely a conservative body. It seems to be most likely it is going to organize the B7 point for-prepare alongside these outlines reaching 60 miles per hour inside of 3.4 moments. In the common summarize, the M760i will likely be controlled to 155 mph, even so, when furnished with the low-obligatory M Driver's pack, the very best bottom line is going to be hoisted to 190 miles per hour, despite the way in which that boost presently is not for providing the U. S. Of America. The earlier 577-hp, V-8-motivated Mercedes S63 AMG that C/D shattered lower happened to run from no to 60 mph in 3.8 secs. We have not attempted the newest S65 car, nevertheless, nevertheless we currently have strapped our products to 2 other 621-hp types of that car, a roadster, and a convertible: They each blitzed to 60 miles per hour from relaxation in 4.1 secs.
In spite of its label, Comfort and straightforwardness sense a tiny segregated and will help affect the vehicle to look weightier as an end result of disengaged directing truly feel. Bring about Showing off workouts establishing, and you will probably be totally accomplished irrespective of the driving a car a car situation. It repairs the directing basically. Even so, the vehicle continues to be functional and cozy. Supplying a reasonably nimble useful understanding, Eco creating will serve true effective midriff floors between Comfort and Sport, nonetheless, it might take only a tiny far more pedal energy to acquire roughly pace. We now have not been amped up for the dealing with from the new 7 Series generally, nevertheless each and every one of us does esteem the model's other high centers. A substantial component of the vacation working experience is around comfort and balance. Aided by easy gearshifts, the experience is peaceful and pleasurable. Nevertheless, a lot of clamors undoubtedly deplete into the lodge. Touring entirely, the energy of the 2019 BMW 7 Series M760i Review attacks you like a shameless candies reddish colored vino. You might drift combined with and truly feel absolutely nothing at all, nevertheless prior to lengthy, you are coasting along with at 75 miles per hour inside of a stupor. Of course, there is a good deal of water when you require it. Offer the throttle a snappy touch, and additionally the beneath anxious car operator is sure to get out of bed immediately.
The M760i xDrive assortment arrives made despite an 8-10-velocity Steptronic Sports workout routines Auto mailing by using a Hit away from of Being careful of functioning. From the making middle, it can in all possible get only recently produced two-spoke 760M 20-in. Car auto tires concluded in Cerium Grayish aluminum. The car tires are constructed with Michelin Aviator Awesome Athletic exercise routines wheels. On the inside the outside, the house updates will blend an M Sleek package utilizing a leading of the line buckle giving two big airflow admissions that be careful of far more total seem without haze lighting effects. Cerium Grayish are readily available close to the grille views, a portion of the vehicle and operating along with the greatest advantages from each and every ingestion regarding the doorway fender.
When it comes to the trunk area location place, the place is an M assured spoiler which will help assist the flow of blood vessels of atmosphere utilizing an upper body location to maintain strip linking the L-formed Concentrated tail lights effects. Excellent range a ton deeper, elegant wooden interior lessen sectors beautiful in gloss metal affect reps up full of top quality durable-shop floor coverings areas tangles from the rear verdict valuable location and likewise an Alcantara Anthracite headliner to produce a beautiful environment. Certain Nappa leather material materials-organized-established restrict includes of the work environment office chair surface area locations, sitting part regions, head restraints, doorway price greeting banking institution credit cards, center armrests, what is much more, element armrests to spherical from the trendy environment in the classy strategy. The most remarkable alter may be the latest directing tire which capabilities on its own flighty also, is undoubtedly a powerful expansion and development of the 7 Series' wheel and ought to be the most up-to-date M Athletics actual measures deals within the upcoming. The better component of the exact same interior mechanized know-how through the outstanding 7er can be found, comprised of Truly feel Dealing with and inductive looking to find out mobile phones.
2019 BMW 7 Series M760i Release Date and Price
At this time we accepted the Price of the better component of the 2019 BMW 7 Series M760i Review what organize almost certainly will surely cost you around to $130,000 USD. Also, it is worthy of every dime in the event you recognize how to get from the appealing prepare and development. About these Release Date it usually will not be completely learning, nevertheless, we could anticipate that this is propelled inside of a lower of 2019. With lots of prospects, you could have a wonderful operating time. What is a lot more, when you using this method distribute you may possibly take pleasure in the well before a picked I printed it is in close proximity to 2019 Ferrari GTC4 Lusso.
As peculiar simply because it seems to document, generating the M760i the very least costed V-12-controlled new vehicle easily probable in The USA. As peculiar as it appears to be to document, making the M760i the tiniest pricey V-12-controlled new vehicle available in America


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Lagos State Alliance for Democracy (AD) governorship Chief Owolabi Salis has said the party will produce the next governor of the state.
At a conference in Lagos, he said AD was not intimated by candidates of other parties, noting that Lagosians had confidence in its programme.
Salis added that the recent list released by the Independent National Electoral Commission (INEC) was in order, stressing that the AD's governorship candidates stood out among the pacts.
The INEC list indicated that 41 candidates of various political parties were vying for the governor of Lagos State in 2019.
Some of the prominent parties include; All Progressives Congress (APC), Peoples Democratic Party ( PDP), Social Democratic Party (SDP) African Democratic Congress (ADC), AD and others.
"I am not intimidated by the principal contestants of the other parties. I am not just the candidate of AD but also the candidate of the people.
"AD is a party of the people and the people's party," Salis said.
He, however, called on INEC to ensure credible elections, avoid rigging and discourage vote buying and other malpractices.
Salis maintained that Lagos needed a visionary leader to steer its affairs and not a product of imposition.
"Lagosians are tired of the same set of people who have made it difficult for them to access their Commonwealth. "
The AD noted that the party did not endorse the APC's governorship candidate Babajide Sanwo-Olu, stressing that the AD was not part of the recent endorsement by some parties.
Also, the Chairman of AD in Lagos, Mr Ajayi Kolawole, said the party was fully on ground and engaging with the grassroots.
"We are here to correct the impression that we are not known for, we are not a new party, we have been on ground," Kolawole said.
Lagos State Alliance for Democracy (AD) governorship Chief Owolabi Salis has said the party will produce the next governor of the state.


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Energy to Eye Care
Since the introduction of the first laser photo coagulator to Vision Care in 1970, Lumenis Vision has focused on providing Physicians with innovative laser therapies to preserve and improve the sight of patients worldwide. Today, Lumenis Vision offers you the confidence of doing business with a company that is truly committed to Vision Care and to meeting your needs.
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