text:
WHAT A TRAVESTY! Am I the only one that does not get Fallon and find his shtick boring?ReplyCancel
I haaaate Jimmy Fallon. He looks like a rotting sea monkey.ReplyCancel
Ugh. Wrote a HeisenBreview and Blogger ate itReplyCancel
I thought it was going to be like that animatronic Chuck-E-Cheese band, but it's worse. Its the mealworm Fallon.ReplyCancel
It went something like:
A group of white men enter a stage dressed like Busey on a bender at Dude Ranch, which later becomes his Heavy Petting Zoo. But I digress…
They then miraculously proceed to further whitewash a song performed by an Aussie blond who many refer to as a slave massa. Admittedly, I stopped halfway through this shitshow, mainly because the harmonica irritated me. Never thought I'd miss the day when Homegirl only rapped about how her Pu$sy be wetter than tha fuckin Amazon!!!!
PS- I hope no fancyscreennames were harmed or offended by this message or the video it refers to.
*** 4 life
@Sugar twins. I came in just to say that.ReplyCancel
The Fallon Method of success.
A) Laugh at your own jokes
B) Mug for the camera constantly
C) Do unfunny crap and have hipsters and beta males say he's a genius.
The cheese has a partner. I like him.ReplyCancel
Love it, too cute.
Everyone's talking about this–PR heaven!ReplyCancel


text:
By PATSY STODDARDEditor
It's not an ordinary sight in Emery County. Two llamas, a man and a dog trailing through backroads, desert and mountains on a quest. Loch Wade is the man, the llamas are Jasper and Chalcy and the dog is McDuff. The journey is part of a dream to walk the Old Spanish Trail from Santa Fe, N.M. to California. Wade is making the trip in increments and last year he walked from Abiquiu, N. M. to Moab. This year he began at Moab and is walking to Cedar City, a journey of approximately 330 miles.
"I've always been interested in the Old Spanish Trail. I've read a lot about it. Probably nobody has packed that trail since John C. Fremont did around 1850. I decided it was about time someone did. I hope to write a book about my adventures.
"I left Moab on Feb. 7 and have been traveling right along. The weather has been great, it's not hot which is good for me and good for the llamas. We traveled through the open desert after we left Moab. We stopped in Green River and then cut across the Swell at Lost Springs. Traveled along Cedar Mountain and then on the road to Castle Dale and followed it all the way to SR-10," said Wade.
Wade plans to travel near the highway and down to Fremont Junction and on to Cedar City. It will probably take another three-four weeks to get there.
Wade said they travel at a leisurely pace and they aren't in a big hurry. He's off work in the winter and his wife "allows" him this time to make the trip. He won't be able to visit the Fishlake cut-off trail because of the weather, and will follow the road past Salina, Richfield and along Highway 89 to Marysvale and into Parowan. He will mainly cut through the open desert along the way if he can.
Wade is from Boulder and he said when he arrives in the desert near Cedar City that will be far enough for this leg of the journey and he'll call his wife in Boulder to come and pick him up with the horse trailer.
Wade travels 12-13 miles a day, but while traveling across the Swell he made good time and was able to log 20 miles. He was happy to see Castle Dale and a bit of civilization, his plans in Emery County included a stay at the local motel, a hot meal and a shower and a trip to the laundromat to catch up on the laundry.
Wade takes good care of the animals and he let's the llamas browse along the way as needed. They begin each day with a healthy breakfast of grains and each night he stakes them out so they can browse as needed. Wade said the animals especially enjoyed the salt brush on the way across the Swell.
Wade thinks what's he's doing might seem a little unusual to some folks, but it's the right thing for him to do and he's enjoying his trek with his friends and spending time in the great outdoors along the Old Spanish Trail.

subset: commoncrawl


text:
Professional Makeup Artist Joanna Schlip has worked with some of the most celebrated people in the entertainment industry. Her work has graced the covers of the most prestigious magazines worldwide. With her tremendous talent and outgoing personality she is consistently booked for beauty commercials, print campaigns, and A-List trendsetters for red carpet events. In addition, she contributes her artistic skill set to cutting edge editorials, runways, and fashion advertising from coast to coast.
At the age of eighteen Joanna left the United States for Paris, determined to start her career. It was a brave step for a young girl who didn't know anyone in Paris or speak a word of French. She just knew that she wanted to work for French Vogue. It took some major perseverance, but she achieved that goal and much, much more. Joanna spent over fifteen years in Europe working throughout Milan, Paris and London. Her experience in the highly visible European fashion industry honed her skills to an elite level taking her work to international status.
When Joanna returned to the U.S. in 1999, Los Angeles proved to be the place for her to be. Since returning to the U.S. she has made over forty television appearances and trained artistry teams for cosmetic giants. She is also a two time keynote speaker at the IMAT's, a beauty author, an international spokesperson and world-wide beauty expert. Joanna's extraordinary work ethic and genuine personality have afforded her an amazing clientele including Eva Longoria, Scarlett Johansson, Ellen Pompeo, Laura Linney, Hilary Duff, Sheryl Crow and Jessica Simpson.
In 2006, Joanna launched her first book, Glamour Gurlz: The Ultimate Step-by-Step Guide to Great Make-up and Gurl Smarts (Random House). This fantastic how-to book of makeup is packed with over 100 beautiful original photos, "Schlip-tips" and tricks. It's layered with inspirational quotes and stories from twenty-five celebrity beauties, thirty models and young women. All of Joanna's proceeds from the book support J-Gurlz, a foundation Joanna created to help young women realize their true potential by making a difference in the lives of teen and transitional-aged girls while building self-esteem through innovative programs.
Joanna has traveled to ninety-three countries around the globe. She places her career first and continues to challenge herself everyday in hopes of sharing her vision of modern beauty with women everywhere.


text:
Mens Fleece Jackets - MensSave on Men's Mens Fleece Jackets at Proboardshop.com! Great selection of snow gear at unbelievably low prices. Proboardshop has been selling Mens Fleece Jackets and outdoor gear for over 30 years and we're proud of it.
Please refine here More in the Outlet Shop.
- Size: X-Small
Mens Fleece Jackets More in the Outlet Shop.
Stormtech Microlight 1/4 Zip Pullover Fleece
Size: X-Small, Small, Medium, Large, XXX-LargeReg: $40.00 On Sale: $16.95


text: OverviewBack to top True Natural Cosmetics - All Natural Daily Moisturizer 20 SPF - 3 oz. Item# :113722UPC# :852671003015 Brand:True Natural Cosmetics Size/Form:3 oz. Ship Weight:0.22 True Natural Cosmetics All-Natural Daily Moisturizer SPF 20 3 oz. (84 g) True Natural Cosmetics All-Natural Daily Moisturizer SPF 20 goes on clear and is immediately effective with UVA/UVB Protection and anti-aging hydration. You know how important it is to protect your skin from the sun's damaging (and aging) UV rays. But you can do without the white effect most natural facial sunscreens lend to skin. And what about anti-aging creams? That is important, too. True Natural Cosmetics Intensive Daily Moisturizer SPF 20 is everything you are looking for all in one convenient product. Serious sun protection, goes on clear, and anti-aging ingredients too. True Natural Cosmetics uses natural Zinc Oxide, the most effective sunscreen ingredient available, for the highest protection from both UVA/UVB rays. This Zinc is broad-spectrum, non-nano zinc for immediate protection without the white residue. True Natural Daily Moisturizer soaks into skin immediately - perfect for wearing under makeup. Along with serious sun protection, Daily Moisturizer SPF 20 is packed with anti-aging antioxidant ingredients to hydrate, firm and guard the skin from further damage. Cupuacu butter restores elasticity to skin and soothes irritation and inflammation. Aloe Vera provides a protective layer to skin, shielding it from harmful environmental factors. Cocoa butter is rich in Vitamin E to hydrate and boost collagen. Jojoba is the oil most closely matched to the skin's own sebum, and provides light, balancing moisture. It is perfect for normal and combination skin. True Natural Cosmetics Daily Moisturizer SPF 20 is the first USDA 100% Certified Biobased SPF 20 facial moisturizer on the market, 100% free from harmful chemical ingredients. True Natural Cosmetics products are biodegradable and never tested on animals. True Natural SPF 20 Sunscreen has been listed in the EWG's Skin Deep Sunscreens 2011 list of the "Best Moisturizers With SPF" as a top-rated moisturizer/sunscreen to provide broad-spectrum (UVA and UVB-sunburn) protection with fewer hazardous chemicals that penetrate the skin. What is the PH of this sunscreen? SPF 40 pH range 8.0-9.0 Average pH 8.47 SPF 20 pH range 7.0-8.0 Average pH 7.55 How often should I reapply when swimming? That's a great question. There's been a lot of confusion about the whole water resistance issue for sunscreen. FDA, the regulating body for sunscreens, allows manufacturers to label their sunscreens as water resistant, and very water resistant. Those are the only two allowed claims. Prior to this summer, many other manufactures would use the claim "Water proof" but the FDA has now cracked down on this and made it illegal for anyone to make this claim. In order to use the claim "Water Resistant", a company has to show that the SPF on the sunscreen is still effective after a person has been submerged in water for a period of 40 minutes. IE if you put on the SPF 40 sunscreen, then sit in the pool for 40 minutes, and then retest the effectiveness of the sunscreen — it must have an SPF of 40 or more. If it measures SPF 35 — then you either can label the sunscreen SPF 35, or not make the Water Resistance claim. If a sunscreen has "Very Water Resistant" on the packaging, then that means that the sunscreen would keep its effectiveness even after submersion in water for 80 minutes. True Natural did not test either sunscreen for Very Water Resistant. They did test the SPF 20 on 3 subjects, but the results were so close to the cutoff (results between 19.5—20.5) that they decided to not complete the test and skipped the Water Resistance claim. The SPF 40 passed with flying colors (with a final SPF measuring 41.5). So — does this mean that True Natural sunscreens need to be reapplied after 40 minutes? No — it just means that the sunscreen has been tested to keep the stated effectiveness after full submersion in water for 40 minutes. After that, the effectiveness may decline over time as one continues to stay submerged in water. Reapplication every 1—2 hours is still the recommended course of action. Are your products gluten free? At this time, all but the olive oil is gluten free. True Natural will look for a certified gluten free olive oil for the next production run but at this time, there is a small possibility that gluten exists. How does your self-tanner work? The self tanning lotion utilizes a well known blend of tanning actives that has been proven to work effectively over many years. How do they achieve their purposes? Naturally derived DHA & Erythrulose chemically react with the amino acids on your skin to create a non-toxic brownish pigment called melanoidins, the tan effect. This has been a known affect since the 20's and proven safe of decades of use. How do the products claim to be sunscreens (zinc) as well as being bio-based? True Natural sunscreens are bio-based because zinc oxide, the active ingredient, is a natural mineral that is in effect renewable. The USDA BioBased testing confirms this and ensures it is not petroleum based (non-renewable). You say that your products are 100% Bio-based, does that mean there are no chemicals in them? Bio-based does not mean no chemicals. It means that the ingredients are renewable. The Natural Products Association certification confirms that True Natural products are all naturally sourced and not synthetic. What are the "active" ingredients in your products? In the sunscreens, the active ingredient (as defined by the FDA) is Zinc Oxide. In the Self-Tanner, the active ingredients are DHA & Erythrulose. Is the cream a sunscreen cream because of the properties of the cupuacu butter? The FDA regulates what is considered a sunscreen ingredient — only Zinc Oxide and Titanium Dioxide can be listed as Active sunscreen ingredients. A large number of other ingredients are known to boost spf values but cannot be listed as active ingredients — Green Tea, Buritic Acid and Cupuacu are among those. Hence, the cream is a sunscreen because it uses zinc oxide. The SPF# may be higher because it includes some boosters. How do you preserve your products? There are a variety of proprietary techniques we use for preservation. As far as ingredients are concerned, only naturally sourced preservatives are used. There are no chemicals or artificial additives and preservatives in TRUE products. How is the packaging 100% cruelty-free? Well, the product has never been tested on animals and that's really what the cruelty free applies to. True Natural certainly never tested the packaging on animals. The packaging is plastic so it's not derived from animal source. You say you use organic ingredients — how are these ingredients extracted? All organic ingredients are marked on the label with an *. These ingredients are accompanied by an USDA NOP certificate from each ingredient supplier. The NPA has audited each and every ingredient in our products for natural origin, for extraction process, and required that the NOP certificate be provided for all organic ingredients. The extraction processes for each organic ingredient varies. What is the shelf life for your products? The shelf life for TRUE Natural products is approximately two years. What percentage of the packaging is made from recyclable / renewable materials? What are these materials? Around 40% of the packaging is made from recycled milk containers. For safety/sanitary reasons the tubes must use a virgin inner and outer plastic — but to be as green as possible within regulations, they use a recycled inner layer made from milk containers. The cap needs to be virgin plastic due to regulations (being in direct contact with the product.) That being said, True Naturals is always looking for higher percentage recycled packaging. Is the lemon and orange essential oils in TRUE Natural Cosmetic sunscreens photosensitizing? IFRA recommends that products applied to areas of skin exposed to sunlight, excluding products which are rinsed off (body wash, soaps, etc.) should contain levels of cold-pressed lemon oil not exceeding 2%. Levels for orange oil should not exceed 0.5%. At TRUE Natural they are currently using lemon and orange oils at levels of 0.4% in sunscreen formulations, well below the IFRA recommendations. And remember, even though their essential oil content is well below the safety limit, they use mineral sunscreen zinc oxide which forms a physical barrier on the skin. This barrier prevents UV rays from penetrating to skin's lower layers. So, why use lemon and orange essential oils at all? TRUE Natural Cosmetics utilizes a unique essential oil blend, including lemon and orange oils, for their aromatherapeautic properties. What do you mean by zero chemicals? Yes, everything under the sun is made up of molecules which have a chemical name. For instance, zinc oxide, a natural mineral, has a chemical formula of ZnO. However, when they talk about chemicals in the natural product world, they mean man-made, synthetic compounds. Their slogan of "Zero Chemicals / Zero Harmful Ingredients" means that ALL ingredients are 100% naturally sourced, not created in a lab, or derived from petrochemicals. These ingredients are audited by the NPA, which verified the source and extraction methods that were used to obtain the ingredients. With regards to Glycerin, our organic glycerin is derived from palm, rape and sunflower seed oil. It contains no preservatives, antioxidants or solvents. 1,4-Dioxane is never used. Caprylic/Capric Triglyceride is the industry name for fractionated coconut oil. This simply means a specific density of the coconut oil is extracted, similar to how cream is separated from milk to produce cheese. At True Natural Cosmetics, their mission is to provide you with high performance, chemical-free personal care products that are safe for you, your family & the environment. All True Natural Cosmetic products are formulated & produced in the U.S. using 100% natural ingredients. Their ingredients and packaging are sustainable and they are committed to certified organic ingredients whenever available. True Natural Cosmetics products are safe for the whole family, safely biodegradable and contain no parabens, phthalates, petroleum, synthetic fragrances or dyes of any kind. True Natural Cosmetic ingredients are certified organic whenever possible, of the highest quality and receive the highest certifications, such as being 100% USDA Certified Biobased. Natural Products: At True Natural Cosmetics, they believe that skin care and cosmetics should be 100% natural. Made with pure ingredients that not only offer health benefits, but perform the way you want. They feel that you should never have to sacrifice effectiveness for safety. Natural Ingredients: Only safe natural ingredients are used to make products — never any parabens, PEG, phthalates, petrochemicals, synthetic colors or fragrances, or other harmful synthetics. They constantly work to educate themselves and their customers on the best natural ingredients and latest technologies available to organic cosmetics. Complete Transparency: The ingredients used to make each product will never be a mystery. A complete and clear ingredient listing appears on all True Natural Cosmetics products, making it easy for you to make informed decisions, especially if you have any allergies, sensitivities or concerns. The Environment: True Natural Cosmetics packaging is designed to have the lowest impact on the environment and utilize post-consumer recycled (PCR) content. All True Natural Cosmetics products are formulated and produced locally in the US, and ingredients are biodegradable and come from renewable sources. Humans and Animals: Not only are True Natural products developed to be as safe as possible, they are also committed to fair trade and fair working conditions. True Natural products are never tested on animals, nor do they utilize dead animal byproducts. Giving Back: True Natural Cosmetics is committed to Corporate Social Responsibility, that their activities and products have a positive impact for customers, environment, community and employees. You: True Naturals strives to build your trust by supplying you and your family with safe, nontoxic products that work. Better for you…better for your world. Life is Too Short for Bad Ingredients!


text:
Blammo!! Kate Mara and boyfriend.
Is Mink still dating Chris Evans? Who is the costar?
Are we getting 2 Rumer photos a day now?ReplyCancel
Rumer twice in one day, really???ReplyCancel
Omg, I'm freaking out over the weirdness of Zoe and Taryn's pics…it's the angle, right?ReplyCancel
No Willis girls yesterday means 2 of Rumer today? What's the matter with Scout she looks too good?
Taryn Manning looks strange and that dress….
Woot woot! Pensatucky cleans up well!
Love the picture of Chris Evans and Colbie Smulders!ReplyCancel
Is that Max Minghella in the picture with Kate Mara?ReplyCancel
Love Kiernan! I hope she keeps her head. So far, so good.ReplyCancel
Well, Enty, Stacy IS young compared to the geezers that run the Oscars, for example. LOLReplyCancel
Agent Maria Hill!ReplyCancel
Bella Thorne in Christmas Hershey's chocolate kiss wrapping?ReplyCancel
Minka, Chris & Sebastian Stan?ReplyCancel
Is this the bad hair column? I can't imagine a worse hairstyle for Rumer. If you have a longish face and your dad's forehead, gotta keep that sucker covered.ReplyCancel
@OhMYGod I meant to say covered with bangs. Jaysus.ReplyCancel
Fucking Rumer again. How much is she paying you?ReplyCancel
Keibler looks hard in the face and those granite tits don't help.
Nice tattoo Rumor. Moron.
I guess Cobie Smulders is cute. But not real cute.
Bella needs to eat a hot dog.
Taryn Manning looks bizarre. Ugly legs. And of course a stupid fucking tattoo on her arm.
I think that is the only Jonas that is straight.ReplyCancel
Sally Draper has more style in her pinky finger than people in the business waaaaay longer than her.ReplyCancel
Ugh, these are all hideous!ReplyCancel
Rumer Willis is serving up some serious 'Rocky Dennis' with that hair color. Um, no.ReplyCancel
I know .. I hate to keep ragging on her, but it's hard to think of a worse colour. She has the means to get a really good colorist, but maybe she's one of those people who knows everything.
@Penelope rumer willis
"I carried a watermelon"
I couldn't resist old school quote
weird head collectionReplyCancel


text:
Skip to comments.Barack Obama Slams John McCain For Opposing Abortion, Activist Judges
Posted on 07/10/2008 11:07:56 AM PDT by wagglebee
Washington, DC (LifeNews.com) -- With pro-life advocates and John McCain's campaign criticizing his position in favor of unlimited abortions, Barack Obama struck back on Thursday. The Illinois senator put forward an absolutist position in favor of abortion and attacked McCain for wanting judges who don't make law from the bench.
"I will never back down in defending a woman's right to choose," Obama said at a breakfast fundraiser with an internal campaign group called "Women for Obama."
"That's what's at stake," Obama added, referring to control of the Supreme Court and whether abortion will remain legal and without limits for another 35 years.
"Senator McCain has made it abundantly clear that he wants to appoint justices like Roberts and Alito -- and that he hopes to see Roe overturned," Obama said, according to an AP report. "I stand by my votes against confirming Justices Roberts and Alito."
Obama is campaigning with his half sister and pro-abortion former presidential candidate Hillary Clinton and orchestrating events with women voters who may have supported his formal rival and are upset that he secured the nomination.
During the event, Obama promised to adopt Clinton's position in favor of abortion and promote it as president.
However, Clinton admitted that, for some of her former presidential backers, it may be difficult to get behind Obama in the general election.
"I understand how challenging it is to turn on a dime, to say, 'Close that chapter now, we're on to the next chapter,'" she said. "It is a process, it does take time for people to just take a deep breath and go forward."
Yesterday, the head of a pro-life women's group said Obama's record on abortion is extreme and that it wouldn't go over well with either women or American voters in general.
Susan B. Anthony List president Marjorie Dannenfelser pointed out how, last week, Obama was trying to make his absolutist position look better.
"Barack Obama knows his extreme record on abortion doesn't resonate with everyday American voters, so now he's trying to soften his image," Dannenfelser told LifeNews.com.
"It won't work. From his active opposition to the Born-Alive Infants Protection Act to his support for the radical Freedom of Choice Act, Barack Obama is anything but moderate on abortion," she said.
She explained that, "Obama's abortion-friendly voting record shows his commitment to abortion on-demand, at any time, for any reason, funded by American taxpayers."
Pro-Life Ping
Freepmail wagglebee to subscribe or unsubscribe from the moral absolutes ping list.
FreeRepublic moral absolutes keyword search
Obama doing the work of Osama.
Any Catholic who now votes for him is defying all that our faith teaches.
"I will never back down in defending a woman's right to choose," Obama said...
That pretty much sums it up.
Wow he really seems to be out to help the black families. Wouldn't that demon Margaret Sanger be proud!
"Women's right to choose"
The biggest red herring in history.
This is what I like about Obama. He stands up for who he really is for all to see - A proud defender of EVIL!!!
there, fixed it...
Well here is one position he won't waffle on.
Hell. That's nothing. Mr. Obama voted for post birth abortion as an Illinois state senator. Yes, you read that right. He voted "present" (no) when it came to protecting babies who survived an abortion.
What a Christian.
We HAVE to stop Obama and if the cure's name is John McCain, so be it!!!
Someone send that to Doug Kmiec. What the hell is he thinking?
That's right Obama! First you come out with your flip-flops that land you in support of Bush's policies and now you are highlighting McCain as a Conservative.
Keep going...
McCain ain't a cure per se, but he can be the ointment that provides some relief from itchy nuts.
How DARE you support judges who actually follow the Constitution!
The line in the sand.
(Apropos of the shifting sand that is Barack Obama.)
"Hit me agin, Br'er Bear!"
"McCain ain't a cure per se, but he can be the ointment that provides some relief from itchy nuts."
Not if Jesse gets to them first...ennnf
"reproductive heath" is what gets me. Other than the death of the child what does health have to do with it in most cases??
"I will never back down in defending a woman's right to choose,"
I never let anyone say this in my presence without adding
"to kill her unborn baby".
It really psses off some folks, but I just tell them that they're not talking about "choosing" which ice cream flavor they prefer.
Barry is an incredible dildo.
more like
"consequence-free sexual behavior choices"
than "reproductive health".
I never let a lib hide behind their callous euphamisms.
Any Catholic who now votes for him is defying all that our faith teaches.
Calling Nancy Pelosi, calling Ted Kennedy....
New Report Indicates Voters Most Interested in Barack Obamas Position on Abortion
Posted on Thursday, July 03, 2008 8:20:41 AM by rhema
I wish we could find a way to make Obama's position on abortion know to all Catholics.
You do have to be careful though because there are some pamphlets out there that are not sanctioned by the church.
Or "reproductive rights." Just another smokescreen to distract from the horrors of liberal positions. "Affirmative action" and "diversity" are a couple of others.
If only all Catholics listened....Obama would be toast.
McCain should answer: "Ahem. Mr. Obama, YOU'VE VOTED TWICE IN FAVOR OF INFANTICIDE. Why the hell should anyone take advice from YOU?"
"Choose what?" asked the rational and imaginary reporter. "Do you mean killing infants in utero? Is that what women have a 'right' to do?"
A woman's right to choose WHAT, Herr Obama?
Maybe we should call him "Steely Dan." It kind of fits.
OTOH, I don't want to associate him with great music.
"Obama's abortion-friendly voting record shows his commitment to abortion on-demand, at any time, for any reason, funded by American taxpayers."
I think that sums it up. He's drawn a line...let's make it clear that this is one of a number issue that place him in the extreme as a candidate for president.
And, never forget, Obama is quite the world traveler. He's spent a lot of time in the jungle and for all anyone knows may have developed a taste for "long pig".
Right on, Obama! You go, girl. Let the people know where you really stand. It's a sure-fire way for a liberal to get elected. /ROFLMAO
They didn't survive the Abortion. They were delivered alive and allowed to die from medical neglect. The attending doctors had to issue Birth and Death certificates.
Actually, the Supreme Court ruled that it was permissible to ban partial birth abortions, which Bush+GOP Congress did.
Roe v Wade is no longer "absolute."
"Hes spent a lot of time in the jungle and for all anyone knows may have developed a taste for long pig."
That is what that Sinclair guy said.
I had to read that one several times.
I wouldn't be surprised if tomorrow Obama came out and declared that everyone should have the right to choose to kill another person if they thought it expedient. I can't stand this man.
but, but, Obama is a man of faith.
Start by writing a letter to the Catholic Digest, or any other religious publication. I've already done so on another issue regarding BHO... to 4 different pubs. Then get a list of parishoners at your church and send them letters informing them of BHO's position on abortion and other issues. If we can change the mind of even a small percentage of folks, it's worth a try.
Disclaimer: Opinions posted on Free Republic are those of the individual posters and do not necessarily represent the opinion of Free Republic or its management. All materials posted herein are protected by copyright law and the exemption for fair use of copyrighted works.


text:
One of the most important parts of unraveling the AD mystery is finding out what causes the disease. What makes the disease process begin in the first place? What makes it worse over time? Why does the number of people with the disease increase with age? Why does one person develop AD while another remains healthy?
Some diseases, such as measles or pneumonia, have clear-cut causes. They can be prevented with vaccines or cured with antibiotics. Others, such as diabetes or arthritis, develop when genetic, lifestyle, and environmental factors work together to start a disease process. The role that any or all of these factors play may be different for each individual.
AD fits into the second group of diseases. We do not yet fully understand what causes AD, but we believe it develops because of a complex series of events that take place in the brain over a long period of time. Many studies are exploring the factors involved in the cause and development of AD.
Genetic studies of complex neurodegenerative diseases such as AD focus on two main issues—whether a gene might influence a person's overall risk of developing a disease and whether a gene might influence some particular aspect of a person's risk, such as the age at which the disease begins. Slow and careful detective work by scientists has paid off in discoveries of genetic links to the two main types of AD.
One type is the rare, early-onset Alzheimer's disease [1]. It usually affects people aged 30 to 60. Some cases of early-onset disease are inherited and are called familial AD (FAD). The other is late-onset Alzheimer's disease [2]. It is by far the more common form and occurs in those 60 and older. Gaining insight into the genetic factors associated with both forms of AD is important because identifying genes that either cause the disease or influence a person's risk of developing it improves our ability to understand how and why the disease starts and progresses.
DNA, Chromosomes, and Genes: The Body's Amazing Control Center
Enlarge [3]
The nucleus of almost every human cell contains an encrypted "blueprint," along with the means to decipher it. This blueprint, accumulated over eons of genetic trial and error, carries all the instructions a cell needs to do its job. The blueprint is made up of DNA [4], which exists as two long, intertwined, thread-like strands called chromosomes [5]. Each cell has 46 chromosomes in 23 pairs. The DNA in chromosomes is made up of four chemicals, or bases, strung together in various sequence patterns. The DNA in nearly all cells of an individual is identical.
Each chromosome contains many thousands of segments, called genes [6]. People inherit two copies of each gene from their parents, except for genes on the X and Y chromosomes, which are chromosomes that, among other functions, determine a person's sex. Each person normally has one pair of sex chromosomes (females are XX and males are XY). The sequence of bases in a gene tells the cell how to make specific proteins. Proteins in large part determine the different kinds of cells that make up an organism and direct almost every aspect of the cell's construction, operation, and repair. Even though all genes are present in most cells, the pattern in which they are activated varies from cell to cell, and gives each cell type its distinctive character. Even slight alterations in a gene can produce an abnormal protein, which, in turn, may lead to cell malfunction and, eventually, to disease.
Any permanent change in the sequence of bases in a gene's DNA that causes a disease is called a mutation [7]. Mutations also can change the activation of a particular gene. Other more common (or frequent) changes in a gene's sequence of bases do not automatically cause disease, but they can increase the chances that a person will develop a particular disease. When this happens, the changed gene is called a genetic risk factor [8].
In the early days of AD genetics research, scientists realized that some cases, particularly of the rare early-onset AD, ran in families. This led them to examine DNA samples from these families to see whether they had some genetic trait in common. Chromosomes 21, 14, and 1 became the focus of attention. The scientists found that some families have a mutation in selected genes on these chromosomes. On chromosome 21, the mutation causes an abnormal amyloid precursor protein to be produced (see "From APP to Beta-Amyloid Plaques [9]" for more on APP). On chromosome 14, the mutation causes an abnormal protein called presenilin 1 to be produced. On chromosome 1, the mutation causes another abnormal protein to be produced. This protein, called presenilin 2, is very similar to presenilin 1. Even if only one of these genes that are inherited from a parent contains a mutation, the person will almost inevitably develop early-onset AD. This means that in these families, children have about a 50-50 chance of developing the disease if one of their parents has it.
Early-onset AD is very rare, and mutations in these three genes do not play a role in the more common late-onset AD. However, these findings were crucial because they showed that genetics was indeed a factor in AD, and they helped to identify some key cell pathways involved in the AD disease process. They showed that mutations in APP can cause AD, highlighting the presumed key role of beta-amyloid in the disease. Mutations in presenilin 1 and 2 also cause an increased amount of the damaging beta-amyloid to be made in the brain.
While some scientists were studying the role of chromosomes 21, 14, and 1 in early-onset AD, others were looking elsewhere to see if they could find genetic clues for the late-onset form. By 1992, investigators had narrowed their search to a region of chromosome 19. They found a gene on chromosome 19 that they were able to link to late-onset AD.
This gene, called APOE, produces a protein called apolipoprotein E [10]. APOE comes in several forms, or alleles—ε2, ε3, and ε4:
The Hunt for New AD Genes
For some time, scientists have suspected that, in addition to APOE ε4, as many as half a dozen other risk-factor genes exist for late-onset AD, but they have been unable to find them. In 2007, scientists unveiled their discovery of one new AD risk-factor gene.
This AD risk-factor gene is called SORL1. It is involved in recycling APP from the surface of cells, and its association with AD was identified and confirmed in three separate studies. Researchers found that when SORL1 is expressed at low levels or in a variant form, harmful beta-amyloid levels increase, perhaps by deflecting APP away from its normal pathways and forcing it into cellular compartments that generate beta-amyloid.
As AD genetics research has intensified, it has become increasingly clear that scientists need many different samples of genetic material if they are to continue making progress in identifying new risk-factor genes. Genetic material is also essential for identifying associated environmental factors and understanding the interactions of genes and the environment. These advances ultimately will allow investigators to identify people at high risk of developing AD and help them focus on new pathways for prevention or treatment.
In 2003, NIA launched the Alzheimer's Disease Genetics Study to identify at least 1,000 families with members who have late-onset AD as well as members who do not have the disease. All of these family members provide blood samples and other clinical data for the initiative. The material collected allows investigators to create and maintain "immortalized" cell lines—cells that are continuously regenerated in the laboratory. These cell lines are crucial for the exhaustive DNA analysis studies needed to identify risk-factor genes, each of which may have relatively small effects on AD development. More than 4,000 new cell lines are now available for researchers to study risk-factor genes for late-onset AD.
A new initiative, the Alzheimer's Disease Genetics Consortium, was launched in 2007 to accelerate the application of genetics technologies to late-onset AD through collaborations among most of the leading researchers in AD genetics. The ultimate goal of this effort is to obtain genetic material from 10,000 people with AD and 10,000 cognitively healthy people to comprehensively scan the whole genome for the remaining AD risk-factor genes, as well as those for age-related cognitive decline. Some of the genetic material will be drawn from existing samples of blood and tissue; other genetic material will be collected from new participants.
New AD genetics discoveries are possible largely because of close collaboration among scientists, participation of volunteer families, new genetics technologies, statistical and analytic advances, and rapid data sharing. For example, the SORL1 studies involved 14 scientific institutions in North America, Europe, and Asia and the participation of more than 6,000 people who donated blood and tissue for genetic typing. An important part of NIA's efforts to promote and accelerate AD genetics research is to make biological samples and data publicly available to approved researchers.
Genetics explains some of what might cause AD, but it does not explain everything. So, researchers continue to investigate other possibilities that may explain how the AD process starts and develops.
We now know a great deal about how beta-amyloid is formed and the steps by which beta-amyloid fragments stick together in small aggregates (oligomers), and then gradually form into plaques (see "From APP to Beta-Amyloid Plaques [9]" for more on this process). Armed with this knowledge, investigators are intensely interested in the toxic effects that beta-amyloid, oligomers, and plaques have on neurons. This research is possible in part because scientists have been able to develop transgenic animal models of AD. Transgenics are animals that have been specially bred to develop AD-like features, such as beta-amyloid plaques.
Beta-amyloid studies have moved forward to the point that scientists are now carrying out preliminary tests in humans of potential therapies aimed at removing beta-amyloid, halting its formation, or breaking down early forms before they can become harmful.
For example, one line of research by a pharmaceutical company started with the observation that injecting beta-amyloid into AD transgenic mice caused them to form antibodies to the beta-amyloid and reduced the number of amyloid plaques in the brain. This exciting finding led to other studies and ultimately to clinical trials in which human participants were immunized with beta-amyloid. These studies had to be stopped because some of the participants developed harmful side effects, but the investigators did not give up hope. Rather, they went back to the drawing board to rethink their strategy. More refined antibody approaches are now being tested in clinical trials, and additional research on new ways of harnessing the antibody response continues in the lab.
Another important area of research is how beta-amyloid may disrupt cellular communication well before plaques form. One recent study described how beta-amyloid oligomers target specific synaptic connections between neurons, causing them to deteriorate. Other scientists are studying other potentially toxic effects that plaques have on neurons and in cellular communication. Understanding more about these processes may allow scientists to develop specific therapies to block the toxic effects.
Tau, the chief component of neurofibrillary tangles (see "Neurofibrillary Tangles [11]" for more on tau), is generating new excitement as an area of study. The recent focus on tau has been spurred by the finding that a mutant form of the protein is responsible for one form of frontotemporal dementia, the third most common cause of late-life dementia, after AD and vascular dementia. This form is known as frontotemporal dementia with parkinsonism linked to chromosome 17 (FTDP-17). Finding this mutant protein was important because it suggested that abnormalities in the tau protein itself can cause dementia.
New transgenic mouse models of AD have helped tau research make rapid progress. For example, a recent model, the "triple transgenic" mouse, forms plaques and tangles over time in brain regions similar to those in human AD. Another recent transgenic mouse model, which contains only human tau, forms clumps of damaging tau filaments also in a region-specific fashion similar to AD in humans.
These studies of tau also have suggested a mechanism for tau damage that is different from that previously suspected. With these new insights, scientists now speculate that one reason tau may damage and kill neurons is because it upsets the normal activity of the cell, in addition to forming neurofibrillary tangles.
Other studies of mutant tau in mice suggest that the accumulation of tau in tangles may not even be the culprit in memory loss. Rather, as with beta-amyloid, it may be that an earlier and more soluble abnormal form of the protein causes the damage to neurons.
Researchers have found that a number of devastating neurodegenerative diseases (for example, AD, Parkinson's disease, dementia with Lewy bodies, frontotemporal lobar degeneration, Huntington's disease, and prion diseases) share a key characteristic—protein misfolding.
When a protein is formed, it "folds" into a unique three-dimensional shape that helps it perform its specific function. This crucial process can go wrong for various reasons, and more commonly does go wrong in aging cells. As a result, the protein folds into an abnormal shape—it is misfolded. In AD, the misfolded proteins are beta-amyloid (the cleaved product of APP; see "From APP to Beta-Amyloid Plaques [9]" for more on the formation of beta-amyloid) and a cleaved product of tau.
Normally, cells repair or degrade misfolded proteins, but if many of them are formed as part of age-related changes, the body's repair and clearance process can be overwhelmed. Misfolded proteins can begin to stick together with other misfolded proteins to form insoluble aggregates. As a result, these aggregates can build up, leading to disruption of cellular communication, and metabolism, and even to cell death. These effects may predispose a person to AD or other neurodegenerative diseases.
Scientists do not know exactly why or how these processes occur, but research into the unique characteristics and actions of various misfolded proteins is helping investigators learn more about the similarities and differences across age-related neurodegenerative diseases. This knowledge may someday lead to therapies.
Researchers Explore Neurodegenerative "Cousins"
Enlarge [12]
Neurodegenerative diseases like AD, Parkinson's disease, amyotrophic lateral sclerosis (ALS), and dementia with Lewy bodies share more than the basic characteristic of misfolded proteins. They also share clinical characteristics. For example, people with AD have trouble moving, a characteristic of Parkinson's disease. Sleep-wake disorders, delusions, psychiatric disturbances, and memory loss occur in all of these diseases. These diseases also result from a combination of genetic, lifestyle, and environmental causes, and they develop over many years.
This graphic shows one way of thinking about how these diseases may be linked as well as what makes them unique. By investigating the unique characteristics of these diseases as well as the characteristics they share, scientists hope to learn even more than they would if they focused on each disease by itself.
Mitochondria and Free Radicals
Enlarge [13]
Any given cell has hundreds of mitochondria. This illustration shows two—a healthy mitochondrion and an oxidatively stressed and damaged one. The arrows indicate the movement of free radicals, which can spread easily from damaged mitochondria to other parts of the cell.
Another set of insights about the cause of AD comes from the most basic of all risk factors—aging itself. Age-related changes, such as inflammation, may make AD damage in the brain worse. Because cells and compounds that are known to be involved in inflammation are found in AD plaques, some researchers think that components of the inflammatory process may play a role in AD.
Other players in the aging process that may be important in AD are free radicals, which are oxygen or nitrogen molecules that combine easily with other molecules (scientists call them "highly reactive"). Free radicals are generated in mitochondria, which are structures found in all cells, including neurons.
Mitochondria are the cell's power plant, providing the energy a cell needs to maintain its structure, divide, and carry out its functions. Energy for the cell is produced in an efficient metabolic process. In this process, free radicals are produced. Free radicals can help cells in certain ways, such as fighting infection. However, because they are very active and combine easily with other molecules, free radicals also can damage the neuron's cell membrane or its DNA. The production of free radicals can set off a chain reaction, releasing even more free radicals that can further damage neurons (see illustration "Mitochondria and Free Radicals"). This kind of damage is called oxidative damage [14]. The brain's unique characteristics, including its high rate of metabolism and its long-lived cells, may make it especially vulnerable to oxidative damage over the lifespan. The discovery that beta-amyloid generates free radicals in some AD plaques is a potentially significant finding in the quest for better understanding of AD as well as for other neurodegenerative disorders and unhealthy brain aging.
Researchers also are studying age-related changes in the working ability of synapses in certain areas of the brain. These changes may reduce the ability of neurons to communicate with each other, leading to increased neuronal vulnerability in regions of the brain important in AD. Age-related reductions in levels of particular growth factors, such as nerve growth factor [15] and brain-derived neurotrophic factor [16], also may cause important cell populations to be compromised. Many studies are underway to tease out the possible effects of the aging process on the development of AD.
The Brain's Vascular SystemThis image shows the complexity of the human brain's vascular system, particularly large and small arteries that carry oxygen from the lungs to the brain. Although many blood vessels are visible here, this image shows fewer than half of the total number in the brain.
For some time now, hints have been emerging that the body's vast network of small and large blood vessels—the vascular system—may make an important contribution in the development of dementia and the clinical symptoms of AD. Some scientists are focusing on what happens with the brain's blood vessels in aging and AD. Others are looking at the relationship between AD and vascular problems in other parts of the body.
The brain requires a constant and dependable flow of oxygen and glucose to survive and flourish. The brain's blood vessels provide the highways to deliver these vital elements to neurons and glial cells.
Aging brings changes in the brain's blood vessels—arteries can narrow and growth of new capillaries slows down. In AD, whole areas of nervous tissue, including the capillaries that supply and drain it, also are lost. Blood flow to and from various parts of the brain can be affected, and the brain may be less able to compensate for damage that accumulates as the disease progresses.
For some time now, study of the brain's blood vessel system in AD has been a productive line of inquiry. One important finding has been that the brain's ability to rid itself of toxic beta-amyloid by sending it out into the body's blood circulation is lessened. Some scientists now think that poor clearance of beta-amyloid from the brain, combined with a diminished ability to develop new capillaries and abnormal aging of the brain's blood vessel system, can lead to chemical imbalances in the brain and damage neurons' ability to function and communicate with each other. These findings are exciting because they may help to explain part of what happens in the brain during the development of AD. These findings also suggest several new targets for potential AD therapies.
Research also has begun to tease out some relationships between AD and other vascular diseases, such as heart disease, stroke, and type 2 diabetes. It is important to sort out the various effects on the brain of these diseases because they are major causes of illness and death in the United States today.
Much of this evidence comes from epidemiologic studies, which compare the lifestyles, behaviors, and characteristics of groups of people (see "Describing Scientific Findings: The Type of Study Makes an Important Difference" for more information about epidemiologic studies). These studies have found, for example, that heart disease and stroke may contribute to the development of AD, the severity of AD, or the development of other types of dementia. Studies also show that high blood pressure that develops during middle age is correlated with cognitive decline and dementia in later life.
Another focus of AD vascular research is the metabolic syndrome, a constellation of factors that increases the risk of heart disease, stroke, and type 2 diabetes. Metabolic syndrome includes obesity (especially around the waist), high triglyceride levels, low HDL ("good cholesterol") levels, high blood pressure, and insulin resistance (a condition in which insulin does not regulate blood sugar levels very well). Evidence from epidemiologic studies now suggests that people with the metabolic syndrome have increased risk of cognitive impairment and accelerated cognitive decline.
Nearly one in five Americans older than age 60 has type 2 diabetes, and epidemiologic studies suggest that people with this disease may be at increased risk of cognitive problems, including MCI and AD, as they age. The higher risk associated with diabetes may be the result of high levels of blood sugar, or it may be due to other conditions associated with diabetes (obesity, high blood pressure, abnormal blood cholesterol levels, progressive atherosclerosis, or too much insulin in the blood). These findings about diabetes have spurred research on a number of fronts—epidemiologic studies, test tube and animal studies, and clinical trials. The objective of these studies is to learn more about the relationship between diabetes and cognitive problems and to find out in clinical trials whether treating the disease rigorously can positively affect cognitive health and possibly slow or prevent the development of AD.
We know that physical activity and a nutritious diet can help people stay healthy as they grow older. A healthy diet and exercise can reduce obesity, lower blood cholesterol and high blood pressure, and improve insulin action. In addition, association studies suggest that pursuing intellectually stimulating activities and maintaining active contacts with friends and family may contribute to healthy aging. A growing body of evidence now suggests that these lifestyle factors may be related to cognitive decline and AD. Researchers who are interested in discovering the causes of AD are intensively studying these issues, too.
Exercise has many benefits. It strengthens muscles, improves heart and lung function, helps prevent osteoporosis, and improves mood and overall well-being. So it is not surprising that AD investigators began to think that if exercise helps every part of the body from the neck down, then it might help the brain as well.
Epidemiologic studies, animal studies, and human clinical trials are assessing the influence of exercise on cognitive function. Here are a few things these studies have found:
More clinical trials are underway to expand our knowledge about the relationship of exercise to healthy brain aging, reduced risk of cognitive decline, and development of AD. (See "Participating in a Clinical Trial [17]" for more information).
If you want to know more about the benefits of exercise and physical activity and learn ways to be active every day, NIA has free information just for you! Call 1-800-222-2225 or visit www.nia.nih.gov/Go4Life [18].
Researchers have explored whether diet may help preserve cognitive function or reduce AD risk, with some intriguing findings. For example, studies have examined specific foods that are rich in antioxidants and anti-inflammatory properties to find out whether those foods affect age-related changes in brain tissue. One laboratory study found that curcumin, the main ingredient of turmeric (a bright yellow spice used in curry), can bind to beta-amyloid and prevent oligomer formation. Another study in mice found that diets high in DHA (docosahexaenoic acid), a type of healthy omega-3 fatty acid found in fish, reduced beta-amyloid and plaques in brain tissue.
Other studies have shown that old dogs perform better on learning tasks when they eat diets rich in antioxidants, such as vitamin E and other healthful compounds, while living in an "enriched" environment (one in which the dogs have many opportunities to play and interact with people and other dogs).
Scientists also have examined the effects of diet on cognitive function in people. A very large epidemiologic study of nurses found an association between participants who ate the most vegetables (especially green leafy and cruciferous vegetables) and a slower rate of cognitive decline compared with nurses who ate the least amount of these foods. An epidemiologic study of older adults living in Chicago found the same association. The researchers do not know the exact reason behind this association, but speculate that the beneficial effects may result from the high antioxidant and folate content of the vegetables.
Dietary studies, such as the curcumin study in mice or the vegetables study in nurses, generally examine individual dietary components so that scientists can pinpoint their specific effects on an issue of interest. This approach has obvious limitations because people do not eat just single foods or nutrients. Several recent epidemiologic studies have taken a different approach and looked at an entire dietary pattern.
In one of these studies, researchers worked with older adults living in New York who ate the "Mediterranean diet"—a diet with lots of fruits, vegetables, and bread; low to moderate amounts of dairy foods, fish, and poultry; small amounts of red meat; low to moderate amounts of wine; and frequent use of olive oil. The researchers found that sticking to this type of diet was associated with a reduced risk of AD and that the association seemed to be driven by the whole approach, rather than by its individual dietary components. A follow-up study found that this pattern also was associated with longer survival in people with AD.
All of these results are exciting and suggestive, but they are not definitive. To confirm the results, scientists are conducting clinical trials to examine the relationship of various specific dietary components and their effect on cognitive decline and AD.
Many older people love to read, do puzzles, play games, and spend time with family and friends. All these activities are fun and help people feel alert and engaged in life. Researchers are beginning to find other possible benefits as well, for some studies have shown that keeping the brain active is associated with reduced AD risk. For example, over a 4-year period, one group of researchers tracked how often a large group of older people did activities that involved significant information processing, such as listening to the radio, reading newspapers, playing puzzle games, and going to museums. The researchers then looked at how many of the participants developed AD. The researchers found that the risk of developing AD was 47 percent lower in the people who did them the most frequently compared with the people who did the activities least frequently. Another study supported the value of lifelong learning and mentally stimulating activity by finding that, compared with older study participants who may have had AD or who had AD, healthy older participants had engaged in more mentally stimulating activities and spent more time at them during their early and middle adulthood.
Studies of animals, nursing home residents, and people living in the community also have suggested a link between social engagement and cognitive performance. Older adults who have a full social network and participate in many social activities tend to have less cognitive decline and a decreased risk of dementia than those who are not socially engaged do.
The reasons for these findings are not entirely clear, but a number of explanations are possible. Among them:
Describing Scientific Findings: The Type of Study Makes an Important Difference
These days, the media are full of stories about scientific studies. It can be hard to know what to conclude about their findings. Knowing how the study was conducted can help put the results into the right perspective.
One main type of research is the epidemiologic study. These studies are observational—they gather information about people who are going about their daily lives. Study participants follow many behaviors and practices. It is difficult, therefore, to determine the exact benefits or risks of one particular behavior from among all the healthy or harmful behaviors followed by the participants. That is why, in epidemiologic studies of AD, scientists only say that a finding is "associated with" AD, or not. The epidemiologic evidence linking a behavior and AD is, at best, suggestive, but we do not know that the behavior by itself actually helps to cause or prevent AD.
Other types of research—test tube studies and studies in animals—add to the findings from epidemiologic studies. Scientists use them to examine the same issue but in ways in which the various factors that might influence a result are controlled to a greater degree. This element of control allows scientists to be more certain about why they get the results they do. It also allows them to be more definitive in the words they use to describe their results. Of course, showing a cause-and-effect relationship in tissue samples or even in animal studies still does not mean that the relationship will be the same in humans. Clinical trials in humans are the gold standard for deciding whether a behavior or a specific therapeutic agent actually prevents or delays AD (see "Participating in a Clinical Trial [17]" for more on this kind of research).


text:
Velma Lucille Davis, 85, Elizabethtown, died Monday in Harrisburg.
Funeral services are 2 p.m. Thursday at Cox Funeral Home in Rosiclare. Visitation is 6 p.m. to 9 p.m. Wednesday at the funeral home.
She is survived by two sons, a daughter, 10 grandchildren, 18 great-grandchildren and four great-great-grandchildren.
Location, ST | website.com


text:
Written by David Richards
15/05/2013 | 09:17 |
Category: TVS & LARGE DISPLAY
In the USA the 'Value Electronics' TV shootout, which is run by professional sellers and TV tech professionals, is one of the most keenly watched events on the professional installer calendar. By the slimmest of margins a Samsung plasma TV has come out as the winner despite all the hype surrounding new TV technology.
Site Map | Hot News | RSS Feeds | Media info | Environment | Job Opportunities | Contact Us | Terms of Use | Disclaimer
Copyright ©2017 4SqureMedia Pty Ltd. All rights reserved. Level 1, 275 Alfred Street , North Sydney NSW 2060


text:
Nioxin System 7 Scalp Treatment for Medium/Coarse Hair
- Overview
- Features
- About the brand
Nioxin Scalp Treatment System 7 - for medium to coarse, chemically enhanced hair in the early stages of thinning - is used daily to improve the appearance of thinning hair.
System 7 is a 3-Step system for MEDIUM-COARSE, CHEMICALLY ENHANCED HAIR EARLY STAGES OF THINNING that consists of the Cleanser, the Scalp Therapy and the Scalp Treatment.
Use daily. Apply directly onto ENTIRE scalp. Gently and evenly distribute. Comb through. Do not rinse. May cause temporary redness after application.
Nioxin is a pioneer in hair loss, hair growth and hair care products . Nioxin hair products provide treatments for thin, thinning hair and hair loss in men and women. Nioxin continues to create hair solutions and hair care remedies for every hair type at every stage of hair loss. Nioxin offers 8 customized hair systems to thicken hair and promote scalp health without harsh chemicals.


text:
OGDEN/WELLINGTON-Tarina Siaperas Shepherd, age 36, had her life cut short in a tragic automobile accident on June 2, 2009.
She was born Sept. 17, 1972 in Price, Utah to John and Patty Siaperas Burr.
Married Kelly Shepherd in Ogden, they later divorced.
Tarina loved the outdoors, fishing, and camping with her sons Scott and Beau. She had a great imagination. She loved hiking, rock-n-roll and 70's music. She loved to help others, especially her grandma Ileen. The most important part of her life were her two sons.
Tarina was currently employed with Hidden Splendor Resources, and will be dearly missed by all.
Tarina is survived by her parents, John and Patty Siaperas Burr, Layton; loving sons, Scott (20), Christopher (17), both of Ogden; brothers, Jesse Seabolt, Landun Burr, of Layton; grandmother, Ileen Siaperas, Wellington; and many loving aunts and uncles.
She was preceded in death by her grandfather, Nick Pete Siaperas.
Graveside services were held, Friday, June 5, 2009, 11:30 a.m., at the Wellington City Cemetery. Family received friends at Mitchell Funeral Home, 233 East Main Street in Price Friday morning from 9:30-11 a.m. Friends are welcome at Mitchell's daily.


text:
At first glance this seems like a strange question. Isn't science precisely the quantification of observations into a theory or model and then using that to make predictions? Yes. And are those predictions in different cases then tested against observations again and again to either validate those models or generate ideas for potential improvements? Yes, again. So the fact that climate modelling was recently singled out as being somehow non-scientific seems absurd.
Granted, the author of the statement in question has little idea of what climate modelling is, or how or why it's done. However, that his statement can be quoted in a major US newspaper says much about the level of public knowledge concerning climate change and the models used to try and understand it. So I will try here to demonstrate how the science of climate modelling works, and yes, why it is a little different from some other kinds of science (not that there's anything wrong with that!).
Climate is complex. Since climatologists don't have access to hundreds of Earth's to observe and experiment with, they need virtual laboratories that allow ideas to be tested in a controlled manner. The huge range of physical processes that are involved are encapsulated in what are called General Circulation Models (or GCMs). These models consist of connected sub-modules that deal with radiative transfer, the circulation of the atmosphere and oceans, the physics of moist convection and cloud formation, sea ice, soil moisture and the like. They contain our best current understanding for how the physical processes interact (for instance, how evaporation depends on the wind and surface temperature, or how clouds depend on the humidity and vertical motion) while conserving basic quantities like energy, mass and momentum. These estimates are based on physical theories and empirical observations made around the world. However, some processes occur at scales too small to be captured at the grid-size available in these (necessarily global) models. These so-called 'sub-gridscale' processes therefore need to be 'parameterised'.
A good example is related to clouds. Obviously, in an actual cloud, the relative humidity is close to 100%, but at a grid box scale of 100's of km, the mean humidity – even if there are quite a few clouds – will be substantially less. Thus a parameterisation is needed that relates the large scale mean values, to actual distribution of clouds in a grid box that one would expect. There are of course many different ways to do that, and the many modelling groups (in the US, Europe, Japan, Australia etc.) may each make different assumptions and come up with slightly different results.
It's important to note what these models are not good for. They aren't any good for your local weather, or the temperature of the water at the nearest beach or for the wind in downtown Manhattan, because these are small scale features, affected by very local conditions. However, if you go up to the regional scale and beyond (i.e. Western Europe as a whole, the continental US) you start to expect better correlations.
One of the most important features of complex systems is that most of their interesting behaviour is emergent. It's often found that the large scale behaviour is not a priori predictable from the small scale interactions that make up the system. So it is with climate models. If a change is made to the cloud parameterisation, it is difficult to tell ahead of time what impact that will have on, for instance, the climate sensitivity. This is because the number of possible feedback pathways (both positive and negative) is literally uncountable. You just have to put it in, let it physics work itself out and see what the effect is.
This means that validating these models is quite difficult. (NB. I use the term validating not in the sense of 'proving true' (an impossibility), but in the sense of 'being good enough to be useful'). In essence, the validation must be done for the whole system if we are to have any confidence in the predictions about the whole system in the future. This validation is what most climate modellers spend almost all their time doing. First, we look at the mean climatology (i.e. are the large scale features of the climate reasonably modelled? Does it rain where it should, is there snow where there should be? are the ocean currents and winds going the right way?), then at the seasonal cycle (what does the sea ice advance and retreat look like? does the inter-tropical convergence zone move as it should?). Generally we find that the models actually do a reasonable job (see here or here for examples of different groups model validation papers) . There are of course problematic areas (such as eastern boundary regions of the oceans, circulation near large mountain ranges etc.) where important small scale processes may not be well understood or modelled, and these are the chief targets for further research by model developers and observationalists.
Then we look at climate variability. This step is key, but it is also quite subtle. There are two forms of variability: intrinsic variability (that occurs purely as a function of the internal chaotic dynamics of the system), and forced variability (changes that occur because of some external change, such as solar forcing). Note that 'natural' variability includes both intrinsic and forced components due to 'natural' forcings, such as volcanoes, solar or orbital changes. A clean comparison relies on either being able to isolate just one reasonably known forcing, or having enough data to be able to average over many examples and thus isolate the patterns associated solely with that forcing, even though in any particular case, more than one thing might have been happening. (A more detailed discussion of these points is available here).
While there is good data over the last century, there were many different changes to planet's radiation balance (greenhouse gases, aerosols, solar forcing, volcanoes, land use changes etc.), some of which are difficult to quantify (for instance the indirect aerosol effects) and whose history is not well known. Earlier periods, say 1850 going back to the 1500s or so, have reasonable coverage from paleo-proxy data, and only have solar and volcanic forcing. In my own group's work, we have used the spatial patterns available from proxy reconstructions of this period to look at both solar and volcanic forcing in the pre-industrial period. In both cases, despite uncertainties (particularly in the magnitude of the solar forcing), the comparisons are encouraging.
Recent volcanos as well have provided very good tests of the model's water vapour feedbacks (Soden et al, 2002), dynamical feedbacks (Graf et al., 1994; Stenchikov et al., 2002), and overall global cooling (Hansen et al, 1992). In fact, the Hansen et al (1992) paper actually predicted the temperature impact of Pinatubo (around 0.5 deg C) prior to it being measured.
The mid-Holocene (6000 years ago) and Last Glacial Maximum (~20,000 years ago) are also attractive targets of model validation, and while some successes have been noted (i.e. Joussaume et al, 1999, Rind and Peteet, 1985) there is still some uncertainty in the forcings and response. Other periods such as the 8.2kyr event, or the Paleocene-Eocene Thermal Maximum are also useful, but clearly as one goes further back in time, the more uncertain the test becomes.
The 20th Century though still provides the test that appears to be most convincing. That is to say, the models are run over the whole period, with our best guesses for what the forcings were, and the results compared to the observed record. If by leaving out the anthropogenic effects you fail to match the observed record, while if you include them, you do, you have a quick-and-dirty way to do 'detection and attribution'. (There is a much bigger literature that discusses more subtle and powerful ways to do D&A, so this isn't the whole story by any means). The most quoted example of this is from the Stott et al. (2000) paper shown in the figure. Similar results can be found in simple models (Crowley, 2000) and in more up to date models (Meehl et al, 2004).
It's important to note that if the first attempt to validate the model fails (e.g. the signal is too weak (or too strong), or the spatial pattern is unrealistic), this leads to a re-examination of the physics of the model. This may then lead to additional changes, for example, the incorporation of ozone feedbacks to solar changes, or the calculation of vegetation feedbacks to orbital forcing – which in each case improved the match to the observations. Sometimes though it is the observations that turn out to be wrong. For instance, for the Last Glacial Maximum, model-data mis-matches highlighted by Rind and Peteet (1985) for the tropical sea surface temperatures, have subsequently been more or less resolved in favour of the models.
So, in summary, the model results are compared to data, and if there is a mismatch, both the data and the models are re-examined. Sometimes the models can be improved, sometimes the data was mis-interpreted. Every time this happens and we get improved matches between them, we have a little more confidence in their projections for the future, and we go out and look for better tests. That is in fact pretty close to the textbook definition of science.


text:
Print this page
Return to Article
I found a great way to get in shape that involves my children, can be fit around my 12-hour work shifts and relieves stress as well.
I have a son, Brent, and a daughter, Addison. Once a day, I turn the radio to a station playing upbeat music, lift up one of the children and start dancing. After a few minutes, the other child gets a turn.
I swing the kids from side to side and bounce up and down, holding my stomach tight and squeezing my leg muscles in time to the music. Before I know it, the three of us are giggling and singing, and I can feel the workout taking effect.
At times, the dancing turns into a game of chase, but that's good exercise, too. And if my husband walks in, I'm sometimes able to snag him for a slow dance that I use as a cool-down routine.
Not only did this help me tone up and lose weight, but it saved me from dull workouts and gave me another opportunity to have fun with my children.
—Meg Hibbler, Farrandsville, Pennsylvania


text:
Spoiler!That's what you get for underestimating your opponent Natsu. Jeez, winning the magic games has really got to their heads. First Laxus, now Natsu.
So So
Spoiler!That's what you get for underestimating your opponent Natsu. Jeez, winning the magic games has really got to their heads. First Laxus, now Natsu.
Aggravate, Anger, and Piss off
I think it be cool if Gildarts came in, the next chapter is called "The Two Bombs" could be a pun, on making the council member, and his grand daughter bombs. Reason why I said Gildarts.... his magic blows shit up. I think this would cool way for the plot... where Lucy, and Wendy have to find Igneel to save Natsu's life. xD
It's funny how Tartaros seem to be weak as hell...they just have many lives...you know like when playing online games, noobs die a lot, but they still have infinte amount of respawns lol.
Anyway, I thought Natsu would be able to take it, since bomb produces fire and he's fire. Can't really see where this is going, too many variables here that cold affect the plot.
He can't take the force of the bomb. It was a like a truck hitting Natsu. I wish Gildarts would come in and save his ass, and teach him a thing or two, and Tartaros does seem weak, but they all have curses, I just wonder how Strong Zeref is.
His dragon skin is going to play a big part in this arc I think. Hope he goes into dragon force now like he did with Etherion mmmmmm
Either way Jaguar is a bad ass to get all those hits and still bomb Natsu lol
A pie in the sky?
Yeah, those are power ups we know Natsu is going to have, and most likely pull them out of his ass. xD


text:
Verified Purchase
Excellent creatine, mixes easier than other creatines.
Too much bloating
Posted by Reviewer3334648 on Dec 25, 2011
2 weeks and I have gained 3kgs. Good dissolving. Too much bloating.!!!!!
Excellent creatine, mixes easier than other creatines.
Fast Shipping
Posted by Reviewer3195679 on Feb 23, 2011
Arrive at Brazil in 20 days.... I'll see as it work´s
creatine is creatine
Posted by Reviewer2741350 on Dec 13, 2011
better than most of the creatine i have tryed.
Very good
Posted by Reviewer1101868 on Jun 29, 2011
Good product. No taste. Mixes very well.
The best!
Posted by Reviewer3052015 on Mar 14, 2012
The best!
One of the best
Posted by Reviewer1569422 on May 08, 2012
Very good creatine, at a low price
Posted by Reviewer2772212 on Jun 15, 2012
excellent quality and price on this micronized pure creatine
Posted by Reviewer2082378 on Sep 25, 2012
Excellent product!
The best creatine
Posted by 5605911921701114931 on Feb 20, 2014
The best Creatine.


text:
Since you guys removed the "Thanks" button, where we thanked anyone who came with a useful button.. How do you mods expect we should "thank" Or Show our appreciation's to the people who speak the truth? It could get messy if we posted in a thread about thanking and stuff..
I think a reputation button would be cool.. Where we can say short about negative or positive stuff about threads or posts without having to publicizes it.. Mods? Anyone? XD
If something like that already exists, I apologize for I didn`t notice... If not, get busy! XD


text:
Welcome to Tesco Wine by the case.
We hope you enjoy your visit.
Sorry, this product is currently unavailableWhy not try Blason de Bourgogne Chablis La Réserve 75cl?
Reference number 050339923
£60.00 per case
Equivalent to £10.00 per bottle
60 Clubcard Points
A truly classic Chablis, made by one of the best producers in this northerly region in France.
Characterised by the rich soils, the wines are known for their pure fruit and rarely see an oak barrel.
This one included - it is practically quivering with crisp green apple fruit and really zippy freshness.
Serve chilled, with simply grilled fish, or try with fish pie.
This wine is ideal for drinking now but can be kept for up to 1 year
Extended product information
If you require further product information, including information about allergens in this wine,
Enjoy Responsibly
In the early days, Union des Viticulteurs de Chablis, the flagship of the Chablis appellation, was a core of pioneer wine growers. It is now a group of several hundred dynamic owners many of whom have been tending their vines for generations. Between them they cultivate more than 1,000 hectares of wine which makes up a quarter of the Chablis vineyard area.
Cyril Gros
Chablis is near Auxerre in the Yonne département and is one of the northern-most wine growing areas in Burgundy. It has the particular characteristic of only producing white Chardonnay wines with distinctive minerality owing to the kimmeridgian clay slopes. These particular Chardonnay grapes come from 20 year old vines.
£75.00 per case
Equivalent to £12.50 per bottle
75 Clubcard Points
£75.00 per case
Equivalent to £12.50 per bottle
75 Clubcard Points
£96.00 per case
Equivalent to £16.00 per bottle
96 Clubcard Points


text:
By RON LOWENevada CityCalif.
Coverage of the Republican convention revealed the astonishing lack of diversity among GOP delegates. As the cameras swept over the crowd, we rarely caught a glimpse of anyone of color. It's as if the Republicans have given up even the pretense of the "big tent" they claimed to represent in recent years.
The contrast with the Democratic convention, where every shot of the attendees captured the full spectrum of America's rich heritage, is striking. It conveyed a much more powerful message about true inclusiveness and representation than any speaker at the podium.
White, conservative Republicans are locked into the past while Democrats are seeking true change and a new direction compatible with the diversity of an ever-evolving America.


text:
Issues in Innovation
December 16, 2013
November 5, 2013
November 4, 2013
October 31, 2013
October 25, 2013
October 23, 2013
October 23, 2013
October 16, 2013
October 2, 2013
September 30, 2013
- ‹ previous
- 17 of 36
- next ›
Most people assume that consumers have a fair use right to time shift television to watch at a later time.
In Arista v. Lime Wire the recording industry plaintiffs seek to hold Lime Wire liable for acts of copyright infringement by users of its software.
At issue in this case was whether three software programmers who created the BnetD game server -- which interoperates with Blizzard video games online -- were in violation of the Digital Millennium Copyright Act (DMCA) and Blizzard Games' end user license agreement (EULA).
In a legal battle over Internet music storage that could impact innovation and free expression on the Internet EFF Public Knowledge and other public interest groups asked a federal judge in an amicus brief to protect the "safe harbor" rules for online content in EMI v. MP3Tunes.
Junger sought an injunction against the enforcement of provisions of the International Traffic in Arms Regulations that require him to get the permission of the State Department's Office of Defense Trade Controls (the "ODTC") before he can communicate information about cryptographic software to f
"Digital Betamax": Replay TV Owners Stand Up to Hollywood to Defend Digital VCRs
View Newmark v. Turner frequently asked questions.
What Is the ReplayTV Case About?
EFF filed suit against Apple Inc. to defend the First Amendment rights of an operator of a noncommercial public Internet "wiki" site known as Bluwiki.
Adult entertainment publisher Perfect 10 sued Google's Image Search service arguing that Google violates copyright law by indexing Perfect 10 photos posted on unauthorized websites then making and delivering thumbnail images of those photos in its search results.
EFF and a coalition of academics and public policy groups are urging a federal judge to dismiss a criminal indictment that could give websites extraordinary power to dictate what behavior becomes a computer crime.
In 2005, Macrovision sued Sima to block the sale of the Sima CopyThis! (CT-1, CT-Q1, CT-100, CT-2, CT-200) and GoDVD (SCC, and SCC-2) products, which are designed to digitize analog video, such as the analog video outputs of DVD players and analog VCRs.
In September 2008, the motion picture industry sued RealNetworks over its RealDVD software, which was designed to allow consumers to copy their DVDs to their computers for later playback.
EFF defended the right of innovators to build new technologies without begging Hollywood's permission first.
Hollywood has hoped to control innovation by overturning the "Betamax doctrine" - the bedrock principle that the developer of a technology with substantial legal uses cannot be held liable for users' copyright violations. In the Grokster case, 28 of the world's largest entertainment companies sued the distributors of peer-to-peer (P2P) file-sharing software. EFF defended one of the software companies all the way to the U.S. Supreme Court, which refused to overturn the Betamax doctrine or to force technology companies to redesign multipurpose technologies. Meanwhile, EFF helped block the INDUCE Act, a bill that would have severely undermined the Betamax doctrine.
EFF helped convince the U.S. Supreme Court to invalidate a dangerous patent law precedent that threatened free speech and consumers' rights.
The court unanimously held that issuing automatic injunctions in patent cases improperly removed discretion from trial judges to weigh competing factors, including the effect that enforcing the patent would have on the public interest. This followed the reasoning outlined in a friend-of-the-court brief filed by EFF, which urged the justices to overrule the lower court and protect the public interest in free speech, innovation, and education.
EFF urged a federal appeals court to protect the rights of start-up innovators working to improve TV viewing and other entertainment experiences, arguing that big content companies should not be allowed to block add-on technology with baseless copyright claims.
In October 2013, NY State Attorney General Eric Schneiderman issued a sweeping subpoena seeking identification, financial, and other information on effectively all New York Airbnb "Hosts," those users offering living space for rent.
EFF fought for open, transparent governance of the domain name system.
Karl Auerbach began asking for Internet Corporation for Assigned Names and Numbers's (ICANN) corporate records in November 2000, shortly after he was voted as the North American Elected Director of ICANN. ICANN management proceeded to obstruct his access to these records. Represented by EFF, Auerbach successfully sued to gain his rightful access under the law.
The Supreme Court's ruling in Sony v. Universal Studios (aka the Betamax case) is a landmark copyright precedent that has sheltered a wide array of technology innovators from lawsuits at the hands of the entertainment industries.
The Digital Millennium Copyright Act (DMCA) limits the circumvention of software that's designed to restrict access to copyrighted works. Unfortunately, such a blanket restriction can chill competition, free speech, and fair use.
EFF established that computer code is speech and shielded the developers of privacy-protecting software from government censorship.
In 1995, researcher Dan Bernstein planned to distribute an encryption program he had written that could help prevent strangers from snooping on online communications, discovering passwords, and stealing credit card numbers. But draconian federal laws restricted the publication of his program, treating privacy protection as a potential threat to national security. EFF successfully sued the government on behalf of Bernstein, and a federal court affirmed, for the first time, that software code deserves First Amendment protection.
Like many other companies that host content on behalf of users, video-hosting service Veoh has been bedeviled by copyright lawsuits.
In the U.S., two copyrights generally exist for every song you hear on the radio or online. One relates to the underlying musical work (think sheet music). It typically belongs to a music publisher or songwriter.
Note: There are two parts for the case page for Authors Guild v.
Aereo was a company that let users watch local channels by renting their own small antenna located at the Aereo facility, with the signal from the antenna sent over the Internet to that single user. In a lawsuit, TV networks argued that this constituted a public pe


text:
Dynasties: The Elliotts, Books 1-6
and and and and
Sign Up Now!
Already a Member? Log In
You must be logged into Bookshare to access this title. Learn about membership options, or view our freely available titles.
- Brief Synopsis:
- Missed out on Dynasties: The Elliotts? Now's your chance to catch up with this miniseries with our 6-book bundle!
- Long Synopsis:
- Missed out on Dynasties: The Elliotts? Now's your chance to catch up with this miniseries with our 6-book bundle! This wealthy Manhattan family is suddenly at odds as the family patriarch has pitted sibling against sibling to see who will win control of the Elliott publishing dynasty. As the family clashes, secrets are revealed, scandals are ignited and strong passions are unleashed. Bundle includes: Billionaire's Proposition by Leanne Banks, Taking Care of Business by Brenda Jackson, Cause for Scandal by Anna DePalo, The Forbidden Twin by Susan Crosby, Mr. and Mistress by Heidi Betts, and Heiress Beware by Charlene Sands.
- Book Quality:
- Publisher Quality
- 9781552547007
- Publisher:
- Harlequin Enterprises Ltd.
- Date of Addition:
- 01/28/13
- Copyright Date:
- 2006
- Copyrighted By:
- Harlequin Books S.A.
- Adult content:
- No
- Language:
- English
- Categories:
- Romance, Literature and Fiction
- Submitted By:
- Bookshare Staff
- Usage Restrictions:
- This is a copyrighted book.
Reviews of Dynasties: The Elliotts, Books 1-6 (0 reviews)
0 - No Rating Yet


text:
The Federal Government will Become America's Model Employer for the 21st Century.
Recruit, Retain and Honor a World-Class Workforce to Serve the American People.
Human Resources and Security Specialists should use this tool to determine the correct investigation level for any covered position within the U.S. Federal Government.
Visit this federal site to search for our regulatory notices, proposed and final rules.
See the latest tweets on our Twitter feed, like our Facebook pages, watch our YouTube videos, and page through our Flickr photos.
The URL is either incorrect, or the video is no longer available.


text:
Mary Parise, D.D.S.
Why you are going to love our program
At UDM's School of Dentistry you have great opportunities to learn about every aspect of dentistry while you receive a stipend. We like to say, "you are being paid to learn." Our one-year general dental residency program (with an optional second year) will provide you more training and experience following your graduation from dental school. Through your program rotations, you will have clinical time in our Corktown clinic and the St. John Providence Riverview Clinic. Your residency training will include multiple learning environments outside of clinic. You will have seminars within your program and with faculty and residents in specialty programs, in-service training with the dental school faculty as a whole and continuing education courses that allow for networking with alumni for potential practice opportunities.
Great things for you at UDM
As a general dentistry resident, you will be able to follow patients through their plan of treatment and evaluate treatment completed in previous years to learn what has worked out well and what has not. You will see more patients a day with more difficult treatment needs. You will have experience in restoring implants, performing periodontal surgery and working with endodontic and periodontic residents. You will have a one month oral surgery rotation with the St. John Providence residents. These specialty interactions prepare you for your practice by providing criteria to select your specialty referrals and knowledge regarding the type of relationships you would like to establish with specialists.
Great things in store after UDM
Because of the broad range of experience you gain at UDM's dental school, you learn every aspect of dentistry. If you can plan treatment for some of the difficult patient situations we see in our clinic, then you will have a sound basis to undertake a treatment plan anywhere.
My favorite part about teaching you
I love teaching especially on the clinic floor. I teach in the resident clinic two full days a week. We see a lot of challenging patients from a medical and dental perspective; they have a lot of needs. I enjoy working with the residents as you develop different ways to communicate with patients and techniques to use to provide positive outcomes. It's rewarding to see your professional growth.
I may change your mind
If a patient has lived with missing or infected teeth for a long time and you can provide that patient with teeth or relieve their infection, you will be deeply moved by that experience. You will see what a difference you can make in your patients' lives.
You might like to know
I was a nurse prior to becoming a dentist. I can evaluate a patient walking in the door and pick up on some subtleties of the patient's general appearance that will give clues to the patient's overall health. Many times the patient's overall health affects our plan of treatment.


text:
Accounts Receivable Solutions
Speed collection of accounts receivable and access to working capital while increasing your company's productivity. For any business, in any industry, our Treasury Management team can help you achieve results by delivering efficient, cost-effective solutions to suit your specific needs.
Get Started
By Phone 1-888-558-7568
By Email Online contact form


text:
Paris Hilton Gives the World's Worst Tour of the Waldorf-Astoria
Paris Hilton took the Wall Street Journal on a tour of her childhood home, the Waldorf-Astoria hotel. She manages to do this without stepping foot in the building—and talking only about herself.
Hilton was the subject of the paper's "Walkabout" video feature, wherein notable New Yorkers take a reporter on a tour of their neighborhood. Setting aside the fact that Paris is far more famous for being a Los Angeleno than a Manhattanite, we don't get to see anything inside the Waldorf. Instead we hear that as children she and sister Nicky used to like to go to FAO Schwarz (duh) and used to crash all the parties at the hotel (duh, squared). Then reporter Lee Hawkins takes the celebutante—sorry, "celebrity entrepreneur"—to Central Park, a place that she says she never goes to unless it's for a "photo shoot, or shooting a movie, or a business meeting." Oh, speaking of business, her perfume line brought in $100 million last year. Thanks for that, Paris.


text:
|Posted by hj on February 28, 2004 at 09:51:15:
|In response to Re: washing machine hookup
You have a "Mickey Mouse" drain connection. There is not supposed to be a "clamp or grip to keep spillage in the drain". If you have one, then it must be because the drain in the wall is defective and cannot accept the normal water discharge from the washing machine. Rather than trying to make the drain tighter, which will overload the washing machine pump and possibly cause it to fail, have a plumber check the drain line to see what its problem is.
: : My washer has a plastic hose that goes into the "drain" or "pipes" on the wall in my basment. The water should spin out and go through the hose and into the pipe. However, the "clamp" or "grip" that should leave no room for "spillage" isn't working properly. I changed it by getting a rubber hose and two small clamps in an attempt to force the water to go straight into the drainage pipe and not overflow or spill out, but it still isn't working. What should I do?
: Reply:
|Replies to this post
|There are none.


text:
Bible Reference Books
Use a Bible reference book to get more out of your Bible study, including complete Bible concordances and Bible dictionaries. Reference Bible handbooks help answer common questions and bible companions are helpful aids as you seek deeper understanding of the text.
Layman's Bible Survey: An Illustrated, "Big Picture" Guide to All of God's Word
George W. Knight, George W. Knight, George W. Knight$16.99Add Your ReviewGet the most from your Bible study with this "big picture" resource! Taking you from "In the beginning" to John's Revelation, Knight's survey is packed with entries exploring major periods of biblical history. Color illustrations, maps, sidebars connecting the Old and New Te... Learn More
Insights on Mark
Charles R. Swindoll, Charles R. Swindoll$25.99Add Your ReviewMark's Gospel is fast-paced and action-packed. In this volume, Chuck unpacks both Jesus' story and how Mark wants his readers to see themselves in this story. From beginning to end, we will see the Master preparing His disciples and then propelling them forward to encoun... Learn More
The Baker Book of Bible Charts, Maps, and Timelines
John A. Beck$24.97Add Your ReviewBible charts, maps, and timelines enrich our understanding of God's Word and support effective Bible teaching, but clear and reliable information is difficult to find. To put this information into the hands of teachers and students of the Bible, The Baker Book of Bible Chart... Learn More
Greek for Everyone: Introductory Greek for Bible Study and Application
A. Chadwick Thornhill, A. Chadwick Thornhill$24.99Add Your ReviewReading the New Testament in its original language is one of the most effective ways to gain a greater understanding of the message of the Bible. Even though Greek is important to preparation for preaching and teaching, many who are called to ministry will not be able to ded... Learn More
Unlocking the Bible: What It Is, How We Got It, and Why We Can Trust It
Jeff Lasseigne, Jeff Lasseigne, Greg Laurie, Jeff Lasseigne$16.99Add Your ReviewWho decided which books should---and shouldn't---be in the Bible? Why isn't it in chronological order? Which translation is best? Believers and skeptics have questions, and Lasseigne provides clear answers. Discover the Bible's significance, origin, historical transmission, ... Learn More
The Baker Illustrated Guide to the Bible: A Book-By-Book Companion
J. Daniel Hays, J. Daniel Hays, J. Scott Duvall, J. Scott Duvall$14.99Add Your ReviewEnhance Sunday school lessons, small-group sessions, or personal Bible study with this reliable companion! It features full-color photos, maps, timelines, and illustrations including "Old Testament Messianic Prophecies," "The Miracles of Jesus," and many more. Sections for e... Learn More
Holman QuickSource Bible Atlas: With Charts, Maps, and Biblical Reconstructions
Dr Paul H. Wright, Paul H. Wright$14.99Add Your ReviewHolman QuickSource Bible Atlas with Charts and Biblical Reconstructions packs an amazing amount of information about the Bible into a book that's easy-to-carry and easy-to-use. The book opens with an introduction to the physical context in which the events of the Bible took ... Learn More
Holman Bible Atlas: A Complete Guide to the Expansive Geography of Biblical History
Thomas V. Brisco$29.99Add Your ReviewExplore the world of the Bible through this visual feast and increase your knowledge of God's Word. The Holman Bible Atlas illuminates the Bible for a whole new generation of students and teachers by introducing them to teh physical, cultural, and historical settings of t... Learn More
Holman Illustrated Pocket Bible Dictionary: Pocket Reference Edition
Holman Reference$9.99Add Your ReviewAn updated package on the Bible Dictionary for the popular Holman Pocket Reference series, this portable Bible reference basic allows the reader to quickly find entries on thousands of the most common Bible people, places, terms, and events. The user-friendly edition also fe... Learn More
God's Word, Our Story: Learning from the Book of Nehemiah
D. a. Carson, Kathleen B. Nielson, Paige B. Brown$16.99Add Your ReviewWith contributions from popular Bible teachers, this collection of nine biblical expositions looks to the Book of Nehemiah for truth about God's redemptive plan for his people and the world. Contributors include Tim Keller, John Piper, Nancy Guthrie, Paige Brown, Kathy Kelle... Learn More


text:
With the spring weather comes the fire season and Emery County has been flaming. There have been several recent fires including one on Bott Lane, one in Ferron, one on farm in Elmo, a brush fire in Green River, house fire in Castle Dale and a field fire in Huntington that threatened a house.
The open burn season in the cities began on March 27 and will continue through May 2. Each of the communities have their own set of rules governing burning, but most require obtaining a burn permit. The fire departments in each of the communities like to know when and where a burn is taking place so they can be on the alert should the blaze become out of control.
Children playing with matches in the Huntington fire is believed to have started the blaze which blackened a nearby field and hillside. The fire threatened the home of Nancy Martinez and blackened the retaining wall in the backyard of the home.
The Hayward home in Castle Dale also suffered damages in a fire.