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High Speed Servo Filling Machines Achieve High Efficiency with Small Footprint
Hinds-Bock's servo drive filling machines provide high-speed depositing as well as customizable filling parameter tuning. Customizing the filling also controls the appearance of the fill as well as the velocity. In controlling velocity, the servo filling machines can reach maximum speeds while maintaining clean fills. The machine's speeds are also enhanced through Hinds-Bock's servo driven traveling spout bridge, which moves the spouts with the continuously moving containers.Control technology allows processors to collect data through Ethernet communication. Data such as performance data, up time information, maintenance monitoring and fault alerts enable the food processors to achieve the high filling efficiency yield.
Company Information
Website: www.hinds-bock.com
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First, before I forget, I had a spam comment that said, "why aren't there bullet proof pants?" This is a very good question and should be discussed at great length at another time.
Now, on to the funk.
I am going to start a new section. A review of movies, books and music. This will probably not bring more regular updates but at least they won't just me mindless ramblings.
Ok, here we go.
Eagle Eye with Shia LaBeouf
This was not a terrible movie. It felt like Wanted meets Portal.
Huh, I had a lot more in my head to say about this movie before I sat down to write.
It's a little end of the worldish and you can suddenly think, "Aw jeez, this is predictable." But if you have a list, this is something you can throw on there in between known good movies and unknown complete shots in the dark.
Maybe I was going to go on and on about how I only see LaBeouf as that kid from Even Stevens. Oh well, it's late and I really just want to write about the new Goo Goo Dolls album I purchased tonight. But that will be next on the writing list, since I have to listen to the whole thing first.

subset: commoncrawl


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- Price: $63.00
- Exclusive Price: $2850.00
Agro Agriculture Website Template 27770 Our Services Page
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text: Help Below, you'll find a list of the most common questions and concerns we've encountered from site members. For immediate assistance: Alumni, please contact the Alumni office at firstname.lastname@example.com or (315) 228-7433 Parents, please contact the Parents Fund office at email@example.com or (315) 228-6693 How do I log in or activate my account? I forgot my username or password When I try to log in, I'm told my address can't be found How do I access the Alumni Directory? Where can I find Class Notes? How do I register for Reunion? How do I update my account information? How do I set up e-mail forwarding? Can I change my club affiliation? How does Facebook Connect work? How do I log in or activate my account? Click the link at the top left corner of the page where it says "Sign in". Enter your username and password where indicated. First time users select "First Time Login" to activate your account. 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You must be logged in to view the directory. Where can I find Class Notes? Class notes are posted on the respective class pages. To look up your class page, or the page of another class, go to the Clubs, Classes, and Groups page or type in www.colgateconnect.org/ followed by the year (for example, the address for the Class of 1985 page is www.colgateconnect.org/1985). How do I register for Reunion? The Reunion 2011 registration form will be posted February 1, 201. Until then, check out the Reunion page for more information. How do I update my account information? Click the "Edit My Account" link in the left-hand navigation section. Use the different tabs at the top of the page to update different sections of your account. Make sure to save your changes. How do I set up an alumni e-mail forwarding account? In the Edit My Account page, click the top tab called "E-mail Forwarding." Here you are able to set up an e-mail address (email@example.com) that you can give out to friends and family. However, this is not a "real" e-mail address with an inbox: it is a redirect to an existing e-mail you already have. Your @colgateconnect.org address can forward to any existing e-mail address, so if you ever get a new e-mail address, update your information on the E-mail Forwarding page so you can continue to get your messages. Can I change my club affiliation? Your club affiliation is set automatically based on the address you provide in your account information. To change your affiliation, update your account mailing address on the Edit My Account page or you can send your updated address information to email@example.com. For other questions about your club affiliation, please contact Amanda Kalal, Associate Director of Alumni Relations, at (315) 228-7873 or email@example.com. How does Facebook Connect work? 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This kit accommodates Swanstone 96" high shower wall panel systems. Molding is reversible, Fluted on one side and Butterfly on the other. Rosettes are also reversible, Circle on one side and Star on the other.
Kit includes: Four 3-3/4" x 105" moldings, four 4" x 4" rosettes, one tube of adhesive, and one tube of color coordinated caulk.
- Rosettes: 4"W x 4"H x 5/8"D (102mm x 102mm x 16mm)
- Molding: 3-3/4"W x 105"H x 9/16"D (83mm x 2667mm x 14mm)
- Gorgeous traditional American molding made from solid surface.
- For use with 96" high shower wall systems.
- Also makes beautiful casing around doors, windows, and mirrors.
- Coordinates with Swanstone Solid Surface Walls and Vanity Tops.
Depth | .5625 |
Height | 105 |
Material | Swanstone |
Number Of Pieces | 8 |
Product Weight | 32 |
Width | 3.75 |


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This is an exceptional place - a true retreat from the world -- very secluded andluxurious though totally in touch with nature. With amazing views of the ocean and the hills around. Though quite expensive, we were given a discounted rate as it was the low season. Kelly made every effort to supply us with vegan and vegetarian breakfast options... More
- Also Known As:
- Eagles Nest Hotel Russell
- Eagles Nest Hotel
- Eagles Nest Russell, Bay Of Islands, New Zealand
- Official Description (provided by the hotel):
- Eagles Nest is a multi award winning retreat overlooking the spectacular Bay of Islands. The 75 acre estate offers five world class luxury villas. Each offering; private heated infinity edge lap pool (excl First Light Villa), jacuzzi, air conditioning, home theater system. Talented chefs prepare cuisine featuring regional delicacies matched with New Zealand wines. ... more less
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- TripAdvisor is proud to partner with Expedia, Splendia, Stentorian NV, Agoda, Hotels.com, Venere, Tingo and Priceline so you can book your Eagles Nest reservations with confidence. We help millions of travellers each month to find the perfect hotel for both holiday and business trips, always with the best discounts and special offers.


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Honolulu, Hawaii Bankruptcy Lawyer Greg Dunn answers:
Can creditors sue me after I file for bankruptcy?
Law Firm Info
Greg Dunn, Bankruptcy Attorneyhttp://www.gregdunnhi.com/
841 Bishop Street, Suite 2221
Honolulu, HI 96813
(866) 576-0030
About Our Firm
Honolulu, Hawaii bankruptcy lawyer Greg Dunn is committed to providing effective and efficient legal representation to Hawaii individuals and families in the areas of bankruptcy and debt resolution. Having completed over 8,500 bankruptcy cases since 1996, Greg has the intimate knowledge of Chapter 7 and Chapter 13 bankruptcy law to ensure the best results for each of his clients. Greg has also helped qualifying clients avoid bankruptcy through credit counseling and debt settlements with creditors.
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Microsoft is no longer distributing Product Keys together with the runtime license envelopes.
The new process requires filling out a short form and sending it directly to Microsoft.
To get a product key you need a valid Microsoft Customer License Agreement.
This only affects the following products:
You will receive the requested product keys via email within 48 business hours directly from Microsoft.
Please Note
Product Keys for the products listed above are no longer shipped with the runtime license envelopes.
You can get the product keys via the forms above only!


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Timeline for dad list by karora
Photos from #multicoreworld are over this way: http://v.gd/WGA7mI
about 4 months ago from web -
Got a shot of the 'brewer' part of @forkandbrewer the other night. Must go back to check out the 'fork' part too... http://v.gd/4oUHPo
about 4 months ago from web -
@adhoc I learned Java to do this and I don't see why you should get off lightly, but use any android-acceptable language you like :-) e.g. C
Looking for Android coders to help with this kind of thing: http://is.gd/AWZfZP Interested? But can you do it? Well, get in touch then!
@mjollnir so are you in NZ at present? Why have I not seen you?! Enquiring minds want to know...
about 5 months ago from web -
Grumpy at #immigrationNZ today, expressed at length on G+ http://is.gd/immigrationnz - Are you in #Melbourne and want an accountant?
about 6 months ago from web -
Come on #NZ. Five hours to get these guys past $3k: http://is.gd/R3stRs pledge here: http://is.gd/K4tKZe #kids #physics #olympics #IYPT
about a year ago from web -
Something must have happened in this part of 'social' today & suddenly more follow me. Alas, surrounded by medical disasters, I am elsewhere
about a year ago from web -
A New Year email from twitter? Oh yeah: I remember that. I'm only really on G+ now, including #DAViCal's 1.0.0 release, where there's more r
Friday, 06-Jan-12 05:14:30 UTC from web -
If you run an open source project, have you considered going to CeBIT? For free? http://v.gd/cebit_2011
Saturday, 26-Nov-11 02:26:42 UTC from web -
Time to write about who and why I'm voting for on Saturday: http://v.gd/nzelection2011 - and even post something here! #voteNZ #MMP
Thursday, 24-Nov-11 08:38:21 UTC from web -
Flight on @flyairnz announced as boarding in 5 minutes time but still not at gate. #whoops
Some excellent work on #tppa by @normnz and others here http://www.nzcs.org.nz/newsletter/article/143 #nzrise
The @Police_NZ page for paying fines has Westpac favicon, identifies SSL cert as "QValent PTY LTD (AU)" #sovereignty #cloud #security
Pfft! @normnz @elgrodo: A stream of cars flyovering past like ducks at a sideshow will just encourage the batsmen to greater efforts!
I suspect I'm off @normnz's Christmas card list for cheerfully supporting any proposal that will get me past the Basin Reserve faster.
♺ @glynmoody: No Opposition to Software Patent Exclusion from NZ Software Companies - http://bit.ly/m6g10U v. important point #swpats #nz
Trying to watch #nethui @livestream on #android Eee Pad but "Only live streams and mobile compatible clips appear on mobile devices"#fail
.@nigelmcnie, @phirate: Learning to code doesn't include learning humility, of course, but it will increase understanding of the possible.


text:
Video: Scene On the Street Outside Apple iPhone 5 Event
Category: Smartphones
I was walking by San Francisco's Yerba Buena Center for the Arts this morning at 7:45AM local time. The Apple event is scheduled for 10AM local. The satellite trucks and camera people are in force already and some are already waiting in line. Here's a video of the scene.
Follow Larry Seltzer and BYTE on Twitter, Facebook, LinkedIn, and Google+:


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2004 Cardinal by Forest River Fifth Wheel. 36 ft, 3 slideouts, fully loaded, bay window, recliners, beautiful condition. No smoking & no pets. $18,500. 707-292-6725 or 707-331-9050. More info Email a friend |
Wanted all types of RVs Cash buy on the spot 1990 and up Travel trailers 5th wheel motorhomes And boats I pay top dollar call me any time 209-534-9414 or 209-765-5352 More info Email a friend |
1984 24 ft travel trailer. Self-cont., A/C, in good condition. $2,000 (559)665-1735 More info Email a friend |
1991 Nomad 5th wheel 19.5ft, fully self contained, with a/c air and heating on top of roof, in good cond, $4,300 obo 209 667 5532 More info Email a friend |


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Bortolami Gallery Presents Disclosures: On View Through June 22 New Work by Anna Ostoya and Barbara Leoniak
BORTOLAMI | 520 W 20 St | New York, NY 10011 | T: 212 727 2050
Described by The New York Times as an "impressive, conceptually multipronged and curiously self-reflexive exhibition." Click here for the full review.
Anna Ostoya, Kiss 1, 2011/2013, Oil on canvas. 40 x 46 inches, Courtesy of the artist & Bortolami Gallery
Bortolami is pleased to present the exhibition Disclosures, featuring new works by Anna Ostoya and Barbara Leoniak. As the centerpieces of her second show at the gallery, Ostoya presents four...
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Step off the tourist trail and see the real face of Guatemala at our twice weekly Pueblo Tour. Visit Common Hope's family development center, get a tour of a local pueblo, and meet a family who benefits from Common Hope's programs.
Group tours meet in Antigua's central park, next to the fountain.
Mondays at 10:00 a.m. and Thursdays and 2:30 p.m. (Except holidays.)


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Resource Conservation and Recovery Act Cleanup Enforcement
The Resource Conservation and Recovery Act (RCRA) provides a system to safely manage solid and hazardous waste. When solid or hazardous waste is not properly managed and contamination results at RCRA-regulated facilities, the RCRA Corrective Action program monitors and regulates the cleanup of the contamination.
RCRA also regulates underground storage tanks (USTs) that store hazardous substances and petroleum products. When hazardous substances or petroleum products leak from USTs, the UST cleanup program monitors and regulates the cleanup.
The RCRA cleanup enforcement program gets contamination at RCRA facilities cleaned up by negotiating with the facility owner and/or operator to do the clean up themselves, or to pay for the clean up done by another party. EPA may compel an owner and or operator to cleanup a site by:
- enforcing the cleanup requirements in the facility's operating permit,
- negotiating a cleanup agreement with the facility, or
- issuing a corrective action (cleanup) order to the facility.
For information on how EPA administers:
- The RCRA Corrective Action program, visit EPA's Corrective Action website;
- The Underground Storage Tanks program, visit EPA's UST website; and


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Myocardial infarction
Myocardial infarction | |
Classification and external resources | |
Diagram of a myocardial infarction (2) of the tip of the anterior wall of the heart (an apical infarct) after occlusion (1) of a branch of the left coronary artery (LCA). In the diagram, RCA is the right coronary artery. |
DiseasesDB | 8664 |
MedlinePlus | 000195 |
eMedicine | med/1567 emerg/327 ped/2520 |
MeSH | D009203 |
Myocardial infarction (MI) or acute myocardial infarction (AMI), commonly known as a heart attack, results from the partial interruption of blood supply to a part of the heart muscle, causing the heart cells to be damaged or die. This is most commonly due to occlusion (blockage) of a coronary artery following the rupture of a vulnerable atherosclerotic plaque, which is an unstable collection of cholesterol and fatty acids and white blood cells in the wall of an artery. The resulting ischemia (restriction in blood supply) and ensuing oxygen shortage, if left untreated for a sufficient period of time, can cause damage or death (infarction) of heart muscle tissue (myocardium).
Typical symptoms of acute myocardial infarction include sudden retrosternal chest pain (typically radiating to the left arm or left side of the neck), shortness of breath, nausea, vomiting, palpitations, sweating, and anxiety (often described as a sense of impending doom).[1] Women may experience fewer typical symptoms than men, most commonly shortness of breath, weakness, a feeling of indigestion, and fatigue.[2] A sizeable proportion of myocardial infarctions (22–64%)[3] are "silent", that is without chest pain or other symptoms.
Among the diagnostic tests available to detect heart muscle damage are an electrocardiogram (ECG), echocardiography, cardiac MRI and various blood tests. The most often used blood markers are the creatine kinase-MB (CK-MB) fraction and the troponin levels. Immediate treatment for suspected acute myocardial infarction includes oxygen, aspirin, and sublingual nitroglycerin.[4]
Most cases of myocardial infarction with ST elevation on ECG (STEMI) are treated with reperfusion therapy, such as percutaneous coronary intervention (PCI) or thrombolysis.[5] Non-ST elevation myocardial infarction (NSTEMI) may be managed with medication, although PCI may be required if the patient's risk warrants it.[6] People who have multiple blockages of their coronary arteries, particularly if they also have diabetes mellitus, may benefit from bypass surgery (CABG).[7][8] The European Society of Cardiology guidelines in 2011 proposed treating the blockage causing the myocardial infarction by PCI and performing CABG later when the patient is more stable.[7] Rarely CABG may be preferred in the acute phase of myocardial infarction, for example when PCI has failed or is contraindicated.[7]
Ischemic heart disease (which includes myocardial infarction, angina pectoris and heart failure when preceded by myocardial infarction) was the leading cause of death for both men and women worldwide in 2004.[9] Important risk factors are previous cardiovascular disease, older age, tobacco smoking, high blood levels of certain lipids (low-density lipoprotein cholesterol, triglycerides) and low levels of high density lipoprotein (HDL) cholesterol, diabetes, high blood pressure, lack of physical activity and obesity, chronic kidney disease, excessive alcohol consumption, the abuse of illicit drugs (such as cocaine and amphetamines), and chronic high stress levels.[10][11][12]
Contents |
Classification [edit]
There are two basic types of acute myocardial infarction based on pathology:
- Transmural: associated with atherosclerosis involving a major coronary artery. It can be subclassified into anterior, posterior, inferior, lateral or septal. Transmural infarcts extend through the whole thickness of the heart muscle and are usually a result of complete occlusion of the area's blood supply.[13] In addition, on ECG, ST elevation and Q waves are seen.
- Subendocardial: involving a small area in the subendocardial wall of the left ventricle, ventricular septum, or papillary muscles. The subendocardial area is particularly susceptible to ischemia.[13] In addition, ST depression is seen on ECG.
In the clinical context, a myocardial infarction can be further subclassified into a ST elevation MI (STEMI) versus a non-ST elevation MI (non-STEMI) based on ECG changes.[14] The phrase heart attack is sometimes used incorrectly to describe sudden cardiac death, which may or may not be the result of acute myocardial infarction. A heart attack is different from, but can be the cause of cardiac arrest, which is the stopping of the heartbeat, and cardiac arrhythmia, an abnormal heartbeat. It is also distinct from heart failure, in which the pumping action of the heart is impaired; however severe myocardial infarction may lead to heart failure.[7] A 2007 consensus document classifies myocardial infarction into five main types:[15]
- Type 1 – Spontaneous myocardial infarction related to ischemia due to a primary coronary event such as plaque erosion and/or rupture, fissuring, or dissection
- Type 2 – Myocardial infarction secondary to ischemia due to either increased oxygen demand or decreased supply, e.g. coronary artery spasm, coronary embolism, anaemia, arrhythmias, hypertension, or hypotension
- Type 3 – Sudden unexpected cardiac death, including cardiac arrest, often with symptoms suggestive of myocardial ischaemia, accompanied by new ST elevation, or new LBBB, or evidence of fresh thrombus in a coronary artery by angiography and/or at autopsy, but death occurring before blood samples could be obtained, or at a time before the appearance of cardiac biomarkers in the blood
- Type 4 – Associated with coronary angioplasty or stents:
- Type 4a – Myocardial infarction associated with PCI
- Type 4b – Myocardial infarction associated with stent thrombosis as documented by angiography or at autopsy
- Type 5 – Myocardial infarction associated with CABG
Signs and symptoms [edit]
The onset of symptoms in myocardial infarction (MI) is usually gradual, over several minutes, and rarely instantaneous.[16] Chest pain is the most common symptom of acute myocardial infarction and is often described as a sensation of tightness, pressure, or squeezing. Chest pain due to ischemia (a lack of blood and hence oxygen supply) of the heart muscle is termed angina pectoris. Pain radiates most often to the left arm, but may also radiate to the lower jaw, neck, right arm, back, and epigastrium,[7][17] where it may mimic heartburn. Levine's sign, in which the patient localizes the chest pain by clenching their fist over the sternum, has classically been thought to be predictive of cardiac chest pain, although a prospective observational study showed that it had a poor positive predictive value.[18]
Shortness of breath (dyspnea) occurs when the damage to the heart limits the output of the left ventricle, causing left ventricular failure and consequent pulmonary edema. Other symptoms include diaphoresis (an excessive form of sweating),[1] weakness, light-headedness, nausea, vomiting, and palpitations. These symptoms are likely induced by a massive surge of catecholamines from the sympathetic nervous system[19] which occurs in response to pain and the hemodynamic abnormalities that result from cardiac dysfunction. Loss of consciousness (due to inadequate cerebral perfusion and cardiogenic shock) and sudden death (frequently due to the development of ventricular fibrillation) can occur in myocardial infarctions.[7]
Women and older patients report atypical symptoms more frequently than their male and younger counterparts.[20] Women also report more numerous symptoms compared with men (2.6 on average vs 1.8 symptoms in men).[20] The most common symptoms of MI in women include dyspnea (shortness of breath), weakness, and fatigue. Fatigue, sleep disturbances, and dyspnea have been reported as frequently occurring symptoms that may manifest as long as one month before the actual clinically manifested ischemic event. In women, chest pain may be less predictive of coronary ischemia than in men.[21]
At least one-fourth of all myocardial infarctions are silent, without chest pain or other symptoms.[3][22] These cases can be discovered later on electrocardiograms, using blood enzyme tests or at autopsy without a prior history of related complaints. Estimates of the prevalence of silent myocardial infarctions vary between 22 and 64%.[3] A silent course is more common in the elderly,[3] in patients with diabetes mellitus[23] and after heart transplantation, probably because the donor heart is not fully innervated by the nervous system of the recipient.[24] In people with diabetes, differences in pain threshold, autonomic neuropathy, and psychological factors have been cited as possible explanations for the lack of symptoms.[23]
Any group of symptoms compatible with a sudden interruption of the blood flow to the heart are called an acute coronary syndrome.[25]
The differential diagnosis includes other catastrophic causes of chest pain, such as pulmonary embolism, aortic dissection, pericardial effusion causing cardiac tamponade, tension pneumothorax, and esophageal rupture. Other non-catastrophic differentials include gastroesophageal reflux and Tietze's syndrome.[26]
Causes [edit]
Heart attack rates are higher in association with intense exertion, be it psychological stress or physical exertion, especially if the exertion is more intense than the individual usually performs.[citation needed] The period of intense exercise and subsequent recovery is associated with about a 6-fold higher myocardial infarction rate (compared with other more relaxed time frames) for people who are physically very fit.[citation needed] For those in poor physical condition, the rate differential is over 35-fold higher.[citation needed] One observed mechanism for this phenomenon is increased pulse pressure, which increases stretching of the arterial walls.[citation needed] This stretching results in significant shear stress on atheromas, which results in debris breaking loose from these deposits.[citation needed] This debris floats through the blood vessels, eventually clogging the major coronary arteries.[citation needed]
Acute severe infection, such as pneumonia, can trigger myocardial infarction. A more controversial link is that between Chlamydophila pneumoniae infection and atherosclerosis.[27] While this intracellular organism has been demonstrated in atherosclerotic plaques, evidence is inconclusive as to whether it can be considered a causative factor.[27] Treatment with antibiotics in patients with proven atherosclerosis has not demonstrated a decreased risk of heart attacks or other coronary vascular diseases.[28]
There is an association of an increased incidence of a heart attack in the morning hours, more specifically around 9 a.m.[29][30][31] Some investigators have noticed that the ability of platelets to aggregate varies according to a circadian rhythm, although they have not proven causation.[32]
Risk factors [edit]
Myocardial infarction results from atherosclerosis.[7] Smoking appears to be the cause of about 36% of coronary artery disease and obesity 20%.[33] Lack of exercise has been linked to 7-12% of cases.[33][34] Job stress appear to play a minor role accounting for about 3% of cases.[33]
Risk factors for myocardial infarction include:
- Age[10]
- Gender: At any given age men are more at risk than women, particularly before menopause,[35] but because in general women live longer than men ischemic heart disease causes slightly more total deaths in women.[10]
- Diabetes mellitus (type 1 or 2)[36]
- High blood pressure[37]
- Dyslipidemia/hypercholesterolemia (abnormal levels of lipoproteins in the blood), particularly high low-density lipoprotein, low high-density lipoprotein and high triglycerides[37]
- Tobacco smoking, including secondhand smoke[37]
- Short term exposure to air pollution including: carbon monoxide, nitrogen dioxide, and sulfur dioxide but not ozone.[38]
- Family history of ischaemic heart disease or myocardial infarction particularly if one has a first-degree relative (father, brother, mother, sister) who suffered a 'premature' myocardial infarction (defined as occurring at or younger than age 55 years (men) or 65 (women).[10]
- Obesity[39] (defined by a body mass index of more than 30 kg/m², or alternatively by waist circumference or waist-hip ratio).
- Lack of physical activity.[10]
- Psychosocial factors including, low socio-economic status, social isolation, negative emotions and stress increase the risk of myocardial infarction and are associated with worse outcomes after myocardial infarction. Socioeconomic factors such as a shorter education and lower income (particularly in women), and unmarried cohabitation are also correlated with a higher risk of MI.[40]
- Alcohol — Studies show that prolonged exposure to high quantities of alcohol can increase the risk of heart attack.
- Oral contraceptive pill – women who use combined oral contraceptive pills have a modestly increased risk of myocardial infarction, especially in the presence of other risk factors, such as smoking.[41]
- Hyperhomocysteinemia (high homocysteine) in homocysteinuria is associated with premature atherosclerosis,[42] whether elevated homocysteine in the normal range is causal is contentious.[43]
Inflammation is known to be an important step in the process of atherosclerotic plaque formation.[44] C-reactive protein (CRP) is a sensitive but non-specific marker for inflammation. Elevated CRP blood levels, especially measured with high-sensitivity assays, can predict the risk of MI, as well as stroke and development of diabetes.[44] Moreover, some drugs for MI might also reduce CRP levels.[44] The use of high-sensitivity CRP assays as a means of screening the general population is advised against, but it may be used optionally at the physician's discretion in patients who already present with other risk factors or known coronary artery disease.[45] Whether CRP plays a direct role in atherosclerosis remains uncertain.[44] Inflammation in periodontal disease may be linked to coronary heart disease, and, since periodontitis is very common, this could have great consequences for public health.[46] Serological studies measuring antibody levels against typical periodontitis-causing bacteria found that such antibodies were more present in subjects with coronary heart disease.[47] Periodontitis tends to increase blood levels of CRP, fibrinogen and cytokines;[48] thus, periodontitis may mediate its effect on MI risk via other risk factors.[49] Preclinical research suggests that periodontal bacteria can promote aggregation of platelets and promote the formation of foam cells.[50][51] A role for specific periodontal bacteria has been suggested but remains to be established.[52] There is some evidence that influenza may trigger an acute myocardial infarction.[53]
Baldness, hair greying, a diagonal earlobe crease (Frank's sign[54]) and possibly other skin features have been suggested as independent risk factors for MI.[55] Their role remains controversial; a common denominator of these signs and the risk of MI is supposed, possibly genetic.[56]
Calcium deposition is another part of atherosclerotic plaque formation. Calcium deposits in the coronary arteries can be detected with CT scans. Several studies have shown that coronary calcium can provide predictive information beyond that of classical risk factors.[57][58][59]
Many of these risk factors are modifiable, so many heart attacks can be prevented by maintaining a healthier lifestyle. Physical activity, for example, is associated with a lower risk profile.[60] Non-modifiable risk factors include age, sex, and family history of an early heart attack, which is thought of as reflecting a genetic predisposition.[citation needed] To understand epidemiological study results, it is important to note that many factors associated with MI mediate their risk via other factors. For example, the effect of education is partially based on its effect on income and marital status.[40]
Pathophysiology [edit]
Acute myocardial infarction refers to two subtypes of acute coronary syndrome, namely non-ST-elevated myocardial infarction and ST-elevated myocardial infarction, which are most frequently (but not always) a manifestation of coronary artery disease.[14] The most common triggering event is the disruption of an atherosclerotic plaque in an epicardial coronary artery, which leads to a clotting cascade, sometimes resulting in total occlusion of the artery.[61][62] Atherosclerosis is the gradual buildup of cholesterol and fibrous tissue in plaques in the wall of arteries (in this case, the coronary arteries), typically over decades.[63] Blood stream column irregularities visible on angiography reflect artery lumen narrowing as a result of decades of advancing atherosclerosis.[64] Plaques can become unstable, rupture, and additionally promote a thrombus (blood clot) that occludes the artery; this can occur in minutes. When a severe enough plaque rupture occurs in the coronary vasculature, it leads to myocardial infarction (necrosis of downstream myocardium).[61][62]
If impaired blood flow to the heart lasts long enough, it triggers a process called the ischemic cascade; the heart cells in the territory of the occluded coronary artery die (chiefly through necrosis) and do not grow back. A collagen scar forms in its place. Recent studies indicate that another form of cell death called apoptosis also plays a role in the process of tissue damage subsequent to myocardial infarction.[65] As a result, the patient's heart will be permanently damaged. This myocardial scarring also puts the patient at risk for potentially life threatening arrhythmias, and may result in the formation of a ventricular aneurysm that can rupture with catastrophic consequences.
Injured heart tissue conducts electrical impulses more slowly than normal heart tissue. The difference in conduction velocity between injured and uninjured tissue can trigger re-entry or a feedback loop that is believed to be the cause of many lethal arrhythmias. The most serious of these arrhythmias is ventricular fibrillation (V-Fib/VF), an extremely fast and chaotic heart rhythm that is the leading cause of sudden cardiac death. Another life-threatening arrhythmia is ventricular tachycardia (V-Tach/VT), which may or may not cause sudden cardiac death. However, ventricular tachycardia usually results in rapid heart rates that prevent the heart from pumping blood effectively. Cardiac output and blood pressure may fall to dangerous levels, which can lead to further coronary ischemia and extension of the infarct.
The cardiac defibrillator is a device that was specifically designed to terminate these potentially fatal arrhythmias. The device works by delivering an electrical shock to the patient in order to depolarize a critical mass of the heart muscle, in effect "rebooting" the heart. This therapy is time dependent, and the odds of successful defibrillation decline rapidly after the onset of cardiopulmonary arrest.
Diagnosis [edit]
Medical societies recommend that the physician confirm that a patient is at high risk for myocardial infarction before conducting imaging tests to make a diagnosis.[66] Patients who have a normal ECG and who are able to exercise, for example, do not merit routine imaging.[66] Imaging tests such as stress radionuclide myocardial perfusion imaging or stress echocardiography can confirm a diagnosis when a patient's history, physical exam, ECG and cardiac biomarkers suggest the likelihood of a problem.[66]
The diagnosis of myocardial infarction can be made after assessing patient's complaints and physical status. ECG changes, coronary angiogram and levels of cardiac markers help to confirm the diagnosis. ECG gives valuable clues to identify the site of myocardial damage while coronary angiogram allows visualization of narrowing or obstructions in the heart vessels.[67] At autopsy, a pathologist can diagnose a myocardial infarction based on anatomopathological findings.
A chest radiograph and routine blood tests may indicate complications or precipitating causes and are often performed upon arrival to an emergency department. New regional wall motion abnormalities on an echocardiogram are also suggestive of a myocardial infarction. Echo may be performed in equivocal cases by the on-call cardiologist.[68] In stable patients whose symptoms have resolved by the time of evaluation, Technetium (99mTc) sestamibi (i.e. a "MIBI scan") or thallium-201 chloride can be used in nuclear medicine to visualize areas of reduced blood flow in conjunction with physiologic or pharmacologic stress.[68][69] Thallium may also be used to determine viability of tissue, distinguishing whether non-functional myocardium is actually dead or merely in a state of hibernation or of being stunned.[70]
WHO criteria[71] formulated in 1979 have classically been used to diagnose MI; a patient is diagnosed with myocardial infarction if two (probable) or three (definite) of the following criteria are satisfied:
- Clinical history of ischaemic type chest pain lasting for more than 20 minutes
- Changes in serial ECG tracings
- Rise and fall of serum cardiac biomarkers such as creatine kinase-MB fraction and troponin
The WHO criteria were refined in 2000 to give more prominence to cardiac biomarkers.[72] According to the new guidelines, a cardiac troponin rise accompanied by either typical symptoms, pathological Q waves, ST elevation or depression, or coronary intervention is diagnostic of MI.
Prevention [edit]
The risk of a recurrent myocardial infarction decreases with strict blood pressure management and lifestyle changes, chiefly smoking cessation, regular exercise, a sensible diet for those with heart disease, and limitation of alcohol intake. People are usually commenced on several long-term medications post-MI, with the aim of preventing secondary cardiovascular events such as further myocardial infarctions, congestive heart failure or cerebrovascular accident (CVA). Unless contraindicated, such medications may include:[73][74]
- Antiplatelet drug therapy such as aspirin and/or clopidogrel should be continued to reduce the risk of plaque rupture and recurrent myocardial infarction. Aspirin is first-line, owing to its low cost and comparable efficacy, with clopidogrel reserved for patients intolerant of aspirin. The combination of clopidogrel and aspirin may further reduce risk of cardiovascular events, however the risk of hemorrhage is increased.[75]
- Beta blocker therapy such as metoprolol or carvedilol should be commenced.[76] These have been particularly beneficial in high-risk patients such as those with left ventricular dysfunction and/or continuing cardiac ischaemia.[77] β-Blockers decrease mortality and morbidity. They also improve symptoms of cardiac ischemia in NSTEMI.
- ACE inhibitor therapy should be commenced 24–48 hours post-MI in hemodynamically stable patients, particularly in patients with a history of MI, diabetes mellitus, hypertension, anterior location of infarct (as assessed by ECG), and/or evidence of left ventricular dysfunction. ACE inhibitors reduce mortality, the development of heart failure, and decrease ventricular remodelling post-MI.[78]
- Statin therapy has been shown to reduce mortality and morbidity post-MI.[79][80] The effects of statins may be more than their LDL lowering effects. The general consensus is that statins have plaque stabilization and multiple other ("pleiotropic") effects that may prevent myocardial infarction in addition to their effects on blood lipids.[81]
- The aldosterone antagonist agent eplerenone has been shown to further reduce risk of cardiovascular death post-MI in patients with heart failure and left ventricular dysfunction, when used in conjunction with standard therapies above.[82] Spironolactone is another option that is sometimes preferable to eplerenone due to cost.
- Evidence supports the consumption of polyunsaturated fats instead of saturated fats as a measure of decreasing coronary heart disease.[83] In high-risk people there is no clear-cut decrease in potentially fatal arrhythmias due to omega-3 fatty acids.[84] And they may increase risk in some groups.[84]
- Giving heparin to people with heart conditions like unstable angina and some forms of heart attacks reduces the risk of having another heart attack. However, heparin also increases the chance of minor bleeding.[85]
Management [edit]
An MI requires immediate medical attention. Treatment attempts to salvage as much myocardium as possible and to prevent further complications, hence the phrase "time is muscle".[86] Oxygen, aspirin, and nitroglycerin may be administered. Morphine was classically used if nitroglycerin was not effective; however, it may increase mortality in the setting of NSTEMI.[87] A 2009 and 2010 review of high flow oxygen in myocardial infarction found increased mortality and infarct size, calling into question the recommendation about its routine use.[88][89] Other analgesics such as nitrous oxide are of unknown benefit.[6] Percutaneous coronary intervention (PCI) or fibrinolysis are recommended in those with an STEMI.
Prognosis [edit]
The prognosis post myocardial infarction varies greatly, depending on a person's health, the extent of the heart damage and the treatment given. For the period 2005–2008 in the United States, the median mortality at 30 days was 16.6% with a range from 10.9% to 24.9% depending on the hospital.[90] Using variables available in the emergency room, people with a higher risk of adverse outcome can be identified. One study found that 0.4% of patients with a low-risk profile died after 90 days, whereas in high-risk people it was 21.1%.[91]
Some of the more reproduced risk-stratifying factors include: age, hemodynamic parameters (such as heart failure, cardiac arrest on admission, systolic blood pressure, or Killip class of two or greater), ST-segment deviation, diabetes, serum creatinine, peripheral vascular disease and elevation of cardiac markers.[91][92][93] Assessment of left ventricular ejection fraction may increase the predictive power.[94] The prognostic importance of Q-waves is debated.[95] Prognosis is significantly worsened if a mechanical complication such as papillary muscle or myocardial free wall rupture occurs.[96] Morbidity and mortality from myocardial infarction has improved over the years due to better treatment.[97]
Complications [edit]
Complications may occur immediately following the heart attack (in the acute phase), or may need time to develop (a chronic problem). Acute complications may include heart failure if the damaged heart is no longer able to adequately pump blood around the body; aneurysm or rupture of the myocardium; mitral regurgitation, in particular if the infarction causes dysfunction of the papillary muscle; and arrhythmias, such as ventricular fibrillation, ventricular tachycardia, atrial fibrillation and heart block. Longer-term complications include heart failure, atrial fibrillation, and the increased risk of a second myocardial infarction.
Epidemiology [edit]
Myocardial infarction is a common presentation of ischemic heart disease/coronary artery disease. The World Health Organization estimated in 2004, that 12.2% of worldwide deaths were from ischemic heart disease;[9] with it being the leading cause of death in high or middle income countries and second only to lower respiratory infections in lower income countries.[9] Worldwide more than 3 million people have STEMIs and 4 million have NSTEMIs a year.[98]
Rates of death from ischemic heart disease have slowed or declined in most high income countries, although cardiovascular disease still accounted for 1 in 3 of all deaths in the USA in 2008.[99] In contrast, ischemic heart disease is becoming a more common cause of death in the developing world. For example in India, ischemic heart disease had become the leading cause of death by 2004 accounting for 1.46 million deaths (14% of total deaths) and deaths due to ischemic heart disease were expected to double during 1985–2015.[100] Globally it is predicted that disability adjusted life years (DALYs) lost to ischemic heart disease will account for 5.5% of total DALYs in 2030, making it the second most important cause of disability (after unipolar depressive disorder), as well as the leading cause of death by this date.[9]
Legal implications [edit]
At common law, in general a myocardial infarction is a disease, but may sometimes be an injury. This can create coverage issues in administration of no-fault insurance schemes such as workers' compensation. In general, a heart attack is not covered;[101] however, it may be a work-related injury if it results, for example, from unusual emotional stress or unusual exertion.[102] In addition, in some jurisdictions, heart attacks suffered by persons in particular occupations such as police officers may be classified as line-of-duty injuries by statute or policy. In some countries or states, a person having suffered from a myocardial infarction may be prevented from participating in activity that puts other people's lives at risk, for example driving a car or flying an airplane.[103]
Research [edit]
Patients who receive stem cell treatment by coronary artery injections of stem cells derived from their own bone marrow after a myocardial infarction (MI) show improvements in left ventricular ejection fraction and end-diastolic volume not seen with placebo. The larger the initial infarct size, the greater the effect of the infusion. Clinical trials of progenitor cell infusion as a treatment approach to ST elevation MI are proceeding.[104]
There are currently three biomaterial and tissue engineering approaches for the treatment of post-MI conditions, but these are in an even earlier stage of medical research. Many questions and issues must be addressed before they can be applied to patients. The first involves polymeric left ventricular restraints in the prevention of heart failure. The second utilizes in vitro engineered cardiac tissue, which is subsequently implanted in vivo. The final approach entails injecting cells and/or a scaffold into the myocardium to create in situ engineered cardiac tissue.[105]
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External links [edit]
Find more about Myocardial infarction at Wikipedia's sister projects | |
Definitions and translations from Wiktionary | |
Media from Commons | |
Learning resources from Wikiversity | |
News stories from Wikinews | |
Quotations from Wikiquote | |
Source texts from Wikisource | |
Textbooks from Wikibooks |
- Cardiac disorders - Open Directory Project
- American Heart Association's Heart Attack web site — Information and resources for preventing, recognizing and treating heart attack.


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The corporate refinancing boom help drive Morgan Stanley's third-quarter earnings, which handily beat expectations.
During the third quarter, the bank's revenue from underwriting bonds more than doubled from a year earlier to $431 million.
"The rebound in fixed income & commodities sales and trading indicates that clients have re-engaged after the uncertainty of the rating review in the previous quarter," said Morgan Stanley CEO James Gorman in a statement.
Back in June, Moody's downgraded all major banks, but Morgan Stanley's rating took the biggest hit, falling two notches.
Overall, Morgan Stanley earned $561 million, or 28 cents per share, on revenue of $7.6 billion. That's better than the 24 cents a share and $6.4 billion of revenue that analysts had predicted.
Alongside its jump in revenue, the bank increased its compensation expenses by 8% to $3.9 billion.
Morgan Stanley's earnings were adversely affected by accounting quirks that force banks to adjust both revenue and profits to reflect how the market values its debt. In a stronger market, when a bank's credit is considered to be higher quality, the so-called debt valuation adjustment weighs on both earnings and profits.
Including DVA, Morgan Stanley reported a net loss of 55 cents per share and $5.3 billion of revenue.
Bank executives said the slow M&A environment weighed on revenue and profits. On a conference call Thursday morning, CFO Ruth Porat said that merger activity isn't rebounding with the broader markets because companies fear the U.S. fiscal cliff and potential developments related to Europe's sovereign debt crisis.
"We need to have more clarity about the changes here in the U.S. for activity to move forward," she said, while noting that "conversations are ongoing and the pipeline is healthy."


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Net Positive
Kingfisher Net Positive is a new approach to doing business. To succeed, business must do more than minimise its negative impact – it must be designed to have a more positive one.
Net Positive is a big ambition: to go beyond neutrality and seek to make a positive contribution to the world's future.
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To ensure that every Kingfisher product will enable a more sustainable, and ultimately net positive, lifestyle.
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The effort comes as top Republicans unveil a huge spending measure to fund the government through September.
The broad new measure would leave in place cuts of 5 percent to domestic agencies and 7.8 percent to the Pentagon ordered by President Barack Obama Friday night after months of battling with Republicans over the budget.
But the measure would award the Defense and Veterans Affairs departments their detailed 2013 budgets while other agencies would be frozen at 2012 levels — and then bear the across-the-board cuts.


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Sponsored with the Macaronis Institute of Trial and Appellate Advocacy
Date: Thursday, March 27, and April 3, 10, 17 & 24 2003 Location: Suffolk University Law School, 120 Tremont St., Boston, MA Time: 04:00 PM - 06:30 PM
All litigators, no matter how experienced, grapple with evidentiary issues. Even if you are confident about all the Federal and Massachusetts rules, do you understand the various strategy considerations? Avoiding Evidence Pitfalls is designed to refresh your recollection of the rules of evidence, but also to expose you to the various tactical concerns and to provide demonstrations of how, most effectively, to handle character, impeachment, hearsay, computer/demonstrative and scientific evidence in the courtroom. Each of the five sessions will be presented in a different format to best teach you how to apply the law in that area and to make you aware of its potential impact at trial.
Attend this program and hear judges, professors and leading litigators discuss how they deal with difficult evidentiary problems. This intermediate level course will expose you to the latest cases and developments. Learn the substantive law and practical skills to enable you to get your evidence in and keep the other side's out.
How to get in both bad and good character evidence When you can you impeach with pending cases or cases that have been dismissed When to use hearsay exceptions What is the distinction between computer evidence that is a representation and that which is an interpretation Whether any expert is immune from Daubert standards
Character evidence may be important for winning over the hearts and minds of jurors. However, there are challenges for both sides in how to handle this type of evidence to most effectively help their case. While character evidence can be a powerful tool at trial, there are also risks involved. You need to know the Federal Rules as well as the rules of Massachusetts. Using a scenario involving abuse, we will demonstrate how to conduct a direct and cross examination. Know how to make the most of this valuable evidentiary tool, no matter which side you represent.
Many times in the course of a trial, statements are made that are inconsistent with prior statements. But should you always pounce on this potentially harmful evidence for impeachment purposes? Do you know how to put in evidence of prior convictions? What about promises concerning sentencing? Through a cross examination of a government witness, Attorney Norm Zalkind will show you how to impeach and the panel will discuss the law and strategies involved. Federal rules and Massachusetts practice will be compared. In-depth discussion will include:
Take the hearsay quiz. As a group, participants will work classic hearsay problems taken from Professor Morgan's Harvard Law School exam and elsewhere. These problems are designed to sharpen your skills in recognizing the difference between hearsay and non-hearsay, and in working with hearsay exceptions. The quiz format provides an entertaining vehicle to review your knowledge of these important evidence issues.
Professor Michael Avery, Suffolk University Law School, Boston Honorable Maria I. Lopez, Massachusetts Superior Court
Although computer evidence is becoming more common in the courtroom, not everyone knows how it fits into a trial strategy or even how to categorize it. Do you know how to challenge its admissibility? Do you know what you are required to share with the other side and when? Through a combination of demonstrations of what's available and discussion of the best practices, you will learn how to effectively use and challenge the use of computer evidence. Attend and learn the law and possible objections and practical strategy issues.
Honorable John J. Irwin, Jr., Director, Macaronis Institute for Trial & Appellate Advocacy, Suffolk U. Law School, Boston Honorable Stephen E. Neel, Massachusetts Superior Court Steven M. Bauer, Esq., Testa, Hurwitz & Thibeault, LLP, Boston Brian J. Carney, Esq., President, WIN Interactive, Plymouth
Lawyers continue to grapple with the law regarding experts and how to satisfy the gatekeeping requirement. How do you measure reliability? What kinds of evidence raise Daubert issues? You will learn how to approach these issues through a demonstration of a direct and cross examination of a forensic psychologist in the area of psychological interrogation techniques. Attend and learn:
Thursday, March 27, and April 3, 10, 17 & 24 2003
Tuition is $249; $199 for attorneys admitted to the Bar after 2000, or $99 per session. The course book is included in the tuition charge. A limited number of partial scholarships are available. Please submit a written request via fax 617-305-3099.
Space is limited. Registrations at the door are welcome, but please register in advance to reserve a seat and your written course materials or call to confirm space availability.
Written requests for cancellations received via fax or email 24 hours prior to the program will be granted a refund, minus a $15 charge. If you cannot attend, you can send a substitute, otherwise you will receive the written course materials.
Suffolk University Law School, 120 Tremont St., Boston, MA
Approved for CLE Credit in RI, NH, VT, & ME.
If you have special needs addressed by the Americans with Disabilities Act, please notify us as soon as possible.


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15th/16th Scale
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Etched Brass Sets
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WW2 Vehicle KITS
A complete 1/35th scale kit of the 20mm Oerlikon cannon on a typical Naval mounting. This mounting was fitted to a multitude of ships, smaller vessels, landing craft. The same mounting was also used in coastal concrete/brick positions and also mounted on trucks. (See our Bedford kit K147, as an example).
This all resin model includes etched brass shield, gunsight and gunners strap.


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Two presidents and a funeral
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Wednesday: What will be the legacy of the African Union? Record a video for the show.


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Matching 1 Tags
Scientists Tell Their Stories: George Murphy
During his seminary education, Dr. Murphy also gained a deeper understanding of Luther's theology of the cross, and he realized that it's really the best way to approach the science and theology dialogue.
Hominids Lived Millions of Years Ago, but How Can We Tell? (Videocast)
This BioLogos videocast addresses the age of recently discovered hominid fossils and how scientists are able to obtain those dates.
Does the Slippery Slope Always Go to the Left?
In this video Conversation, N.T. Wright responds to a question from a BioLogos Forum reader who notes that in the world of evangelical Christianity there seems to be a great suspicion about becoming too politically liberal, but few worries about becoming too conservative.
The Danger of Preaching on Genesis
In this video Conversation, Joel Hunter acknowledges the risk that pastors take when preaching on Genesis—and in particular, when they approach it with an attitude of humility, allowing the possibility that the text was not meant to be understood in literal terms.
On What It Means To Be An Image Bearer
In this video conversation, N.T. Wright suggests that what the book of Genesis and the apostle Paul mean by humans "bearing the image of God" is less a static picture and more of a creative, dynamic proposition-- specifically, how we "reflect" God into the world.
Getting Back to Basics
In today's video Conversation, Dr. Greg Boyd discusses the basic truths that provide the framework for Christian belief and distinguishes them from the potentially divisive issues that do not compromise what he refers to as the "non-negotiable" truths.
Science and Faith in the Front Lines of the Culture War
In this brief video conversation, Os Guinness, author and founder of the Trinity Forum, suggests that the religious right might have largely created the current culture war that has science and faith as its core antagonists.
Mystery and Faith
In today's video, Michael Ramsden discusses the importance and meaning of mystery in the Bible.
An Unfolding Creation
When we get stuck on the idea of having biological ancestors, we can miss the point that being made in the image of God is a relational quality, not a biological trait. We can communicate and have a relationship with God, and we can reflect his character and represent him to the rest of creation.
Beginning with the End in Mind
In today's video, Oxford physicist Ard Louis discusses the famous debate between renowned evolutionary biologists Stephen Jay Gould and Simon Conway Morris over the idea of evolutionary convergence.
The Cambrian 'Explosion', Transitional Forms, and the Tree of Life
Geologist Keith Miller examines the "Cambrian Explosion", a period of rapid evolutionary diversification approximately 575 million years ago, and whether it poses a challenge to evolutionary theory.
Creation, Evolution, and Christian Laypeople
In this paper, Keller considers three main clusters of questions lay people raise when they learn of anyone teaching that biological evolution and biblical orthodoxy can be compatible. Keller offers some ideas on how to provide responses that take these concerns seriously.
America's Culture Wars: A Different Perspective
In this video Conversation, Rev. N.T. Wright responds to the controversy in evangelicalism about evolution. Is this a "culture war" issue?
Pastors Dealing With Controversy
In this video Conversation, Joel Hunter offers his thoughts on the challenges of preaching on controversial topics and offers advice to pastors who consider introducing these complicated discussions into their own congregations.
On Engaging in Difficult Conversations
In this video Conversation, Joel Hunter points out that when issues of faith become politicized and polarized, there will always be people who leave the churches that tackle these issues head on.
Confidence and Slippery Slopes
In today's video, Pastor Brian McClaren notes that the metaphor "slippery slopes" is problematic, because we often assume that we are on the top of the slope to begin with, when in fact changing our views may help us ascend the slope, or to reach a new peak of understanding on the other side.
A Pastor Reflects on the Scientific Method
In today's video, Daniel Harrell reflects on the rigors of the scientific method, reminding us that what gives scientific discoveries their weight is not the individuals who make them, but the methods of science themselves, which instill in us a level of confidence.
Hydrology of the Bow River
There's a word beneath the water, and the Bow River belongs to God. Have you been listening?
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Following are the reports and research we have on multichannel marketing. Some of the reports specifically address multi-channel marketing, others refer to multichannel and we've included those too. Let us know if you can't find what you're looking for on multichannel marketing and we might be able to point you in the right direction.
Christmas 2012 Online Shopping Survey Report
The Christmas 2012 Online Shopping Survey Report, free to registered Econsultancy users, is based on a survey of 1,000 consumers in the UK and another 1,000 in the US, carried out using Toluna QuickSurveys, in January 2013. The findings not only confirm the growth of online retail, but also show how consumers' research and purchase patterns are changing thanks to the influence of mobile, as well as services offered by retailers such as Click and Collect.
South-East Asia Digital Marketing Trends Briefing: Key takeaways from Digital Cream Singapore 2012
Econsultancy's South-East Asia Digital Marketing Trends Briefing focuses on the key trends, challenges and solutions for digital marketers in South-East Asia. Based on roundtables from Digital Cream Singapore, held on 29th November 2012, the briefing has been co-authored by ten independent marketing experts who also acted as moderators for the roundtables at Digital Cream.
Quarterly Digital Intelligence Briefing: Digital Trends for 2013
Econsultancy's latest Quarterly Digital Intelligence Briefing, produced in partnership with Adobe, aims to distil a plethora of data and discussion points into some key digital trends, challenges and opportunities which organisations are (or should be) paying close attention to during 2013. The report is based on a survey of around 700 business respondents.
Infographic: Key Digital Trends for 2013
The Key Digital Trends for 2013 infographic looks at what marketers see as the most important trends in the next 12 months. The infographic sums up the key findings of the eighth Quarterly Digital Intelligence Briefing, produced in association with Adobe.
Digital Cream Singapore 2012 Presentations
Two keynote presentations from Econsultancy's Digital Cream Singapore 2012 event on 29th November 2012, where Asia's most senior client-side digital marketers gathered to discuss and explore the latest best practices on e-marketing procurement, business cases, investment, ROI, and suppliers selection. Delegates also discussed their future online stratgies and compared them with like-minded peers.
Talk at AdTech 2012; Email in the Multi-channel
From a November 2012 presentation by Stefan Tornquist (VP of research, US for Econsultancy) at AdTech New York.
Infographic: Making Sense of Marketing Attribution
The Making Sense of Marketing Attribution infographic shows how companies are using marketing attribution, what the barriers to success are and the five steps companies need to take to master attribution. The data in the infographic is taken from the seventh Econsultancy / Adobe Quarterly Digital Intelligence Briefing.
Quarterly Digital Intelligence Briefing: Making Sense of Marketing Attribution
Econsultancy's seventh Quarterly Digital Intelligence Briefing, produced in partnership with Adobe, is dedicated to Making Sense of Marketing Attribution. The briefing looks in detail at how organisations can overcome obstacles to success, and how they can work towards best practice. The research is based on a survey of more than 700 digital professionals working for both client-side marketing teams and agencies.
FUNNEL 2012 Presentations
Presentations from Econsultancy's FUNNEL event which took place on 13th November 2012, where over 600 of the UK's most senior marketing and sales strategists attended to hear from those leading the way in marketing automation, lead nurturing, demand generation, revenue performance & beyond.
Marketing Automation Buyer's Guide 2013
The Marketing Automation Buyer's Guide is an invaluable resource for those investigating the marketing automation sector, with profiles of 22 leading vendors, the latest market trends and tips and pitfalls for buyers. The 163-page report provides details on the issues and trends affecting this sector, as well as information about best practice and tips for successful marketing automation implementations.


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The Strange Case of Dr. Jekyll and Mr. Hyde - Tags - hyde
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Donald Dobson Birthday Celebration
Happy 38th Birthday to Donald Dobson This was a FABULOUS private party thrown together by his loving wife Noelene and WHAT a shindig it was! Valet parking for guests, bartender service, caterers, more delicious food that anyone could possibly eat! I provided complete coverage for the night plus put up a photobooth so everyone there
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February 23, 2011 Bryan Glynn Leave a comment Blog
Anyone, bartender, Birthday, birthday celebration, blast, Celebration, coverage, delicious food, dobson, everyone, FABULOUS, food, fun, gallery, Happy, loving wife, night, Noelene, parking, party, photobooth, portraits, private party, service, shindig, valet parking, wife


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Articoli in riviste
On lower bounds of the solutions of some simple reaction-diffusion equations |
Henkel M. |
ArXiv : cond-mat/0309027 [PDF] |
The mean-field reaction-diffusion equations of the diffusive pair-annihilation and triplett-annihilation processes are considered. A direct lower bound on the time-dependent mean particle-density is derived. The results are applied to the mean-field theory of the diffusive pair-contact process. |