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Estate management is as the name suggests all about managing, usually, one estate. The tasks required will depend on what type of estate it is; a private estate in the Scottish highlands will include running field sports (grouse shooting, stalking etc.) whilst managing an estate for a charity (e.g. National Trust) in the south west of England may involve horticulture and visitor management. Estate management covers land surveying, tenant and community relations including lease negotiations and there will be an element of staff management either paid staff or volunteers.
These roles can include elements of conservation, access, land survey, additionally they may include field sports, recreation, woodland management, wildlife, horticulture and visitor management; working in estate management requires a wide range of interests and skills.
Typical jobs include Estate Manager, Estates Officer, Surveyor, Factor, Estates Maintenance, Park Services, Rural Surveyor.
The National Trust stories blog has case studies and stories about Estate Management at the Trust, read them here.
See current vacancies advertised with CJS here.
To gain the experience required volunteering is frequently a good start.
See current volunteering opportunities advertised with CJS here.
There are many different skills and qualifications required to work in estate management from practical land management techniques to managing the office, staff and volunteers.
Practical skills frequently requested include:
Other skills often required include:
For estate management a qualification in landscape or countryside management is usually a prerequisite, agricultural science or farm management qualifications are often acceptable.
Volunteer Management - the Essentials, Volunteer Scotland
Working with Volunteers, Derrick Hale from Heart of England Hedge Laying Group
'Cairngorms – A National Park for All', Cairngorms National Park Authority
Rural skills -their importance and how we can preserve them, Cotswolds AONB
Dry Stone Walling: A Living Craft for the Present Day, Dry Stone Walling Association
Next generation of Countryside Custodians, Yorkshire Dales Millennium Trust
Coppicing - realising the potential of our woodlands, National Coppice Federation
A Career in Conservation Grazing?, Rare Breeds Survival Trust
Hedgelaying, National Hedge Laying Society
Championing Apprenticeships, North York Moors National Park Authority
Volunteer Driven Success at Rutland Water, Leicestershire & Rutland Wildlife Trust
Helping secure a brighter future for people living and working in the countryside, The Princes Countryside Fund
Countryside Ranger Training - The Next Generation, North Ayrshire Council
Fixing the fells - an all-weather task, Fix the Fells
Countryside Management Association
Scottish Countryside Rangers Association


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Light & Easy (From 6 months)
Premium natural white rice and organic millet suitable as baby's first foods from 6 months onwards. Can be cooked as porridge or rice. Naturally aromatic with a delicately sweet flavour, balanced with the earthy flavour of millet. This blend introduces a little more fibre into your little one's diet. Very soft and easy to cook, cooking on a stovetop takes about 15 minutes.
Weight: 520g, 40 servings
Usage Instructions: Store in fridge. Mix well and wash thoroughly before cooking.
Ingredients (Origin): Natural white rice (Malaysia), organic millet (PRC)
31 March 2021 (more than 1 year from now)
What you will get
Light & Easy (premium natural white rice, organic millet) x 1 pack
Recipe with step-by-step instructions on how to cook the porridge
Why Little Baby Grains
Little Baby Grains believes in a gentle and gradual approach to starting solids. We only use specially selected premium grains, which are more nutritious and softer. Our grains are thoughtfully chosen with a focus on digestion and nutrition.
We ship daily on Mondays-Fridays, excluding public holidays. Your order should reach you within 1-2 working days for Peninsula Malaysia and 2-5 working days for East Malaysia.
Just make payment by 11am, and we will ship your order out on the same day itself! For orders which are paid after 11am, we will try our best to ship it out on the same day, otherwise, it will be shipped tomorrow!
You will receive a tracking number via e-mail once your order is shipped.
Shipping charges:
- Orders below RM50: RM7 for Peninsula Malaysia, RM15 for East Malaysia*
- Starter Kits and orders of RM50 and above: FREE for Peninsula Malaysia, RM20 for East Malaysia*
*Shipping charges to East Malaysia are based on weight. Our website will provide you with the final shipping charge before you pay.
Got a special request? Need urgent delivery? Please reach out to us via Facebook or Instagram direct message (DM) and we will assist you promptly!
We put in a lot of effort in ensuring our grains reach your little one in best condition!
However, if you're unhappy in any way, please email us:
We'll do our best to make things right!.

subset: commoncrawl


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My Approach to Helping
I view clients as innately healthy and capable of positive change. I have thirty years of experience as a psychotherapist and I am still excited and enthusiastic about my work with clients and remain endlessly fascinated with people's stories. My approach is holistic; each individual and family is unique. I begin by assessing where the need for development is - on the individual or systemic level, or both. Is it about emotion, cognition, behavior, or all of the above?


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Petit Bateau
Child's Iconic Breton Top
$52 $64
♥ Add to Babylist
The iconic breton top, a wardrobe basic.
• Give in to the irresistible look of this breton top in the thick jersey so emblematic of Petit Bateau.
• This child's garment offers a number of refined accents, including a boat neck, a sailboat badge on the chest and a vintage look.
• An emblematic brand basic.
Material:100 % Cotton


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Trastuzumab has markedly improved the treatment and long-term prognosis of patients with HER2-positive breast cancer. A frequent clinical challenge in patients with relapsing and/or metastatic disease is de novo or acquired trastuzumab resistance, and to date no predictive biomarkers for palliative trastuzumab have been established. In the present study, the prognostic values of factors involved in the HER2-associated PI3K/Akt signalling pathway were explored. The first 46 consecutive patients treated at the Department of Oncology, Linkoping University Hospital between 2000 and 2007 with trastuzumab for HER2-positive metastatic breast cancer were retrospectively included. The gene copy number variation and protein expression of several components of the PI3K/Akt pathway were assessed in the tumour tissue and biopsy samples using droplet digital polymerase chain reaction and immunohistochemistry. Patients with tumours displaying a high-grade ERBB2 (HER2) amplification level of amp;gt;= 6 copies had a significantly improved overall survival hazard ratio [(HR)=0.4; 95%, confidence interval (CI): 0.2-0.9] and progression-free survival (HR=0.3; 95% CI: 0.1-0.7) compared with patients with tumours harbouring fewer ERBB2 copies. High-grade ERBB2 amplification was significantly associated with the development of central nervous system metastases during palliative treatment. Copy gain (amp;gt;= 3 copies) of the gene encoding the tyrosine phosphatase PTPN2 was associated with a shorter overall survival (HR=2.0; 95% CI: 1.0-4.0) and shorter progression-free survival (HR=2.1; 95% CI: 1.0-4.1). In conclusion, high ERBB2 amplification level is a potential positive prognostic factor in trastuzumab-treated HER2-positive metastatic breast cancer, whereas PTPN2 gain is a potential negative prognostic factor. Further studies are warranted on the role of PTPN2 in HER2 signalling.
Funding Agencies|Swedish Cancer Society [17-0479]; Medical Research Council of Southeast Sweden [FORSS-757671]; ALF Grants Region Ostergotland [LIO-795201]; Stiftelsen Onkologiska Klinikernas Forskningsfond i Linkoping [2016-06-21]
The EPH and ephrins function as both receptor and ligands and the output on their complex signaling is currently investigated in cancer. Previous work shows that some EPH family members have clinical value in breast cancer, suggesting that this family could be a source of novel clinical targets. Here we quantified the mRNA expression levels of EPH receptors and their ligands, ephrins, in 65 node positive breast cancer samples by RT-PCR with TaqMan (R) Micro Fluidics Cards Microarray. Upon hierarchical clustering of the mRNA expression levels, we identified a subgroup of patients with high expression, and poor clinical outcome. EPHA2, EPHA4, EFNB1, EFNB2, EPHB2 and EPHB6 were significantly correlated with the cluster groups and particularly EPHB2 was an independent prognostic factor in multivariate analysis and in four public databases. The EPHB2 protein expression was also analyzed by immunohistochemistry in paraffin embedded material (cohort 2). EPHB2 was detected in the membrane and cytoplasmic cell compartments and there was an inverse correlation between membranous and cytoplasmic EPHB2. Membranous EPHB2 predicted longer breast cancer survival in both univariate and multivariate analysis while cytoplasmic EPHB2 indicated shorter breast cancer survival in univariate analysis. Concluding: the EPH/EFN cluster analysis revealed that high EPH/EFN mRNA expression is an independent prognostic factor for poor survival. Especially EPHB2 predicted poor breast cancer survival in several materials and EPHB2 protein expression has also prognostic value depending on cell localization.
Funding Agencies|Swedish Research Council; Stockholm Cancer Society; Oncology Clinics Research Fund of Linkoping
Few biomarkers exist to predict radiotherapy response in breast cancer. In vitro studies suggest a role for Met and its ligand HGF. To study this suggested role, MET and HGF gene copy numbers were determined by droplet digital PCR in tumours from 205 pre-menopausal and 184 post-menopausal patients, both cohorts randomised to receive either chemo-or radiotherapy. MET amplification was found in 8% of the patients in both cohorts and HGF amplification in 7% and 6% of the patients in the pre-and post-menopausal cohort, respectively. Met, phosphorylated Met (pMet), and HGF protein expression was determined by immunohistochemistry in the premenopausal cohort. Met, pMet, and HGF was expressed in 33%, 53%, and 49% of the tumours, respectively. MET amplification was associated with increased risk of distant recurrence for patients receiving chemotherapy. For the pre-menopausal patients, expression of cytoplasmic pMet and HGF significantly predicted benefit from radiotherapy in terms of loco-regional recurrence. Similar trends were seen for MET and HGF copy gain. In the post-menopausal cohort, no significant association of benefit from radiotherapy with neither genes nor proteins was found. The present results do not support that inhibition of Met prior to radiotherapy would be favourable for pre-menopausal breast cancer, as previously suggested.
Funding Agencies|Swedish Cancer Society; Swedish Research Council; LiU Cancer Foundation
Breast cancer is a heterogeneous disease and new clinical markers are needed to individualise disease management and therapy further. Alterations in the PI3K/AKT pathway, mainly PIK3CA mutations, have been shown frequently especially in the luminal breast cancer subtypes, suggesting a cross-talk between ER and PI3K/AKT. Aberrant PI3K/AKT signalling has been connected to poor response to anti-oestrogen therapies. In vitro studies have shown protein tyrosine phosphatase, non-receptor type 2 (PTPN2) as a previously unknown negative regulator of the PI3K/AKT pathway. Here, we evaluate possible genomic alterations in the PTPN2 gene and its potential as a new prognostic and treatment predictive marker for endocrine therapy benefit in breast cancer. PTPN2 gene copy number was assessed by real-time PCR in 215 tumour samples from a treatment randomised study consisting of postmenopausal patients diagnosed with stage II breast cancer 1976-1990. Corresponding mRNA expression levels of PTPN2 were evaluated in 86 available samples by the same methodology. Gene copy loss of PTPN2 was detected in 16 % (34/215) of the tumours and this was significantly correlated with lower levels of PTPN2 mRNA. PTPN2 gene loss and lower mRNA levels were associated with activation of AKT and a poor prognosis. Furthermore, PTPN2 gene loss was a significant predictive marker of poor benefit from tamoxifen treatment. In conclusion, genomic loss of PTPN2 may be a previously unknown mechanism of PI3K/AKT upregulation in breast cancer. PTPN2 status is a potential new clinical marker of endocrine treatment benefit which could guide further individualised therapies in breast cancer.
Funding Agencies|Swedish Cancer Society [13 0435]; Swedish Research Council [A0346701]
Detection of signals in the mammalian target of rapamycin (mTOR) and the estrogen receptor (ER) pathways may be a future clinical tool for the prediction of adjuvant treatment response in primary breast cancer. Using immunohistological staining, we investigated the value of the mTOR targets p70-S6 kinase (S6K) 1 and 2 as biomarkers for tamoxifen benefit in two independent clinical trials comparing adjuvant tamoxifen with no tamoxifen or 5 years versus 2 years of tamoxifen treatment. In addition, the prognostic value of the S6Ks was evaluated. We found that S6K1 correlated with proliferation, HER2 status, and cytoplasmic AKT activity, whereas high protein expression levels of S6K2 and phosphorylated (p) S6K were more common in ER-positive, and low-proliferative tumors with pAKT-s473 localized to the nucelus. Nuclear accumulation of S6K1 was indicative of a reduced tamoxifen effect (hazard ratio (HR): 1.07, 95% CI: 0.53-2.81, P=0.84), compared with a significant benefit from tamoxifen treatment in patients without tumor S6K1 nuclear accumulation (HR: 0.42, 95% CI: 0.29-0.62, Pless than0.00001). Also S6K1 and S6K2 activation, indicated by pS6K-t389 expression, was associated with low benefit from tamoxifen (HR: 0.97, 95% CI: 0.50-1.87, P=0.92). In addition, high protein expression of S6K1, independent of localization, predicted worse prognosis in a multivariate analysis, P=0.00041 (cytoplasm), P=0.016 (nucleus). In conclusion, the mTOR-activated kinases S6K1 and S6K2 interfere with proliferation and response to tamoxifen. Monitoring their activity and intracellular localization may provide biomarkers for breast cancer treatment, allowing the identification of a group of patients less likely to benefit from tamoxifen and thus in need of an alternative or additional targeted treatment.
Funding Agencies|Swedish Cancer Society; Swedish Research Council; Ostergotland County Council; Lions Research Fund
INTRODUCTION: Endocrine therapies targeting cell proliferation and survival mediated by estrogen receptor alpha (ERalpha) are among the most effective systemic treatments for ERalpha-positive breast cancer. However, most tumors initially responsive to these therapies acquire resistance through mechanisms that involve ERalpha transcriptional regulatory plasticity. Here, we identify VAV3 as a critical component in this process.
METHODS: A cell-based chemical compound screen was carried out to identify therapeutic strategies against resistance to endocrine therapy. Binding to ERalpha was evaluated by molecular docking analyses, an agonist fluoligand assay, and short-hairpin (sh) RNA-mediated protein depletion. Microarray analyses were performed to identify altered gene expression. Western blot of signaling and proliferation markers and shRNA-mediated protein depletion in viability and clonogenic assays were performed to delineate the role of VAV3. Genetic variation in VAV3 was assessed for association with the response to tamoxifen. Immunohistochemical analyses of VAV3 were carried out to determine the association with therapy response and different tumor markers. An analysis of gene expression association with drug sensitivity was carried out to identify a potential therapeutic approach based on differential VAV3 expression.
RESULTS: The compound YC-1 was found to comparatively reduce the viability of cell models of acquired resistance. This effect was probably not due to activation of its canonical target (soluble guanylyl cyclase) but instead a result of binding to ERalpha. VAV3 was selectively reduced upon exposure to YC-1 or ERalpha depletion and, accordingly, VAV3 depletion comparatively reduced the viability of cell models of acquired resistance. In the clinical scenario, germline variation in VAV3 was associated with response to tamoxifen in Japanese breast cancer patients (rs10494071 combined P value = 8.4 x 10-4). The allele association combined with gene expression analyses indicated that low VAV3 expression predicts better clinical outcome. Conversely, high nuclear VAV3 expression in tumor cells was associated with poorer endocrine therapy response. Based on VAV3 expression levels and the response to erlotinib in cancer cell lines, targeting EGFR signaling may be a promising therapeutic strategy.
CONCLUSIONS: This study proposes VAV3 as a biomarker and rationale signaling target to prevent and/or overcome resistance to endocrine therapy in breast cancer.
We wish to thank all study participants and their clinicians for their valuable contributions. This work was supported by grants from the Eugenio Rodriguez Pascual Foundation (2012, to MAP), the Government of Catalonia (2009-SGR283, to AV and MAP), the National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health (R01 DK015556, to JAK), the Red Cooperative Research Thematic Network on Cancer (RTICC) (12/0036/0002 to XRB and 12/0036/0008 to XRB and MAP) and the Spanish Ministry of Health, Fund for Health Research-Institute of Health Carlos III (11/00951 to AU and 12/01528 to MAP).
Akt is a signalling modulator for many cellular processes, including metabolism, cell proliferation, cell survival and cell growth. Three isoforms of Akt have been identified, but only a few studies have concerned the isoform-specific roles in the prognosis of breast cancer patients. The aim of this study was to investigate the prognostic value of v-akt murine thymoma viral oncogene homologue 1 (Akt1) and v-akt murine thymoma viral oncogene homologue 2 (Akt2) in oestrogen receptor positive (ER+) and oestrogen receptor negative (ER–) breast cancer with long-term follow-up.
Material and methods
The expression of Akt in tumour tissue was analysed with immunohistochemistry in a cohort of 272 postmenopausal patients with stage II breast cancer. The median follow-up time was 19 years. Hazard ratios (HRs) and 95% confidence intervals (CIs) were estimated using the Cox's proportional hazards model.
The risk of distant recurrence was reduced for patients with ER+ tumours expressing Akt2 compared to patients with no Akt2 expression (HR = 0.49, 95% CI 0.29–0.82, p = 0.007). When adjusting for important clinical tumour characteristics and treatment, Akt2 was still an independent prognostic factor (HR = 0.38, 95% CI 0.21–0.68, p = 0.001) and the association remained long-term. The prognostic value of Akt2 increased with higher oestrogen receptor levels from no effect among patients with ER– tumours to 68% risk reduction for the group with high ER-levels (P for trend = 0.042). Akt1 showed no significant prognostic information.
Our results indicate that Akt2 expression is associated with a lower distant recurrence rate for patients with ER+ tumours and that this association remains long-term. The prognostic value of Akt2 increases with higher oestrogen receptor expression, motivating further mechanistic studies on the role of Akt2 in ER+ breast cancer.
Funding Agencies|Swedish Cancer Society||Swedish Research Council||
The Eph receptor tyrosine kinases mediate juxtacrine signals by interacting "in trans" with ligands anchored to the surface of neighboring cells via a GPI-anchor (ephrin-As) or a transmembrane segment (ephrin-Bs), which leads to receptor clustering and increased kinase activity. Additionally, soluble forms of the ephrin-A ligands released from the cell surface by matrix metalloproteases can also activate EphA receptor signaling. Besides these trans interactions, recent studies have revealed that Eph receptors and ephrins coexpressed in neurons can also engage in lateral "cis" associations that attenuate receptor activation by ephrins in trans with critical functional consequences. Despite the importance of the Eph/ephrin system in tumorigenesis, Eph receptor-ephrin cis interactions have not been previously investigated in cancer cells. Here we show that in cancer cells, coexpressed ephrin-A3 can inhibit the ability of EphA2 and EphA3 to bind ephrins in trans and become activated, while ephrin-B2 can inhibit not only EphB4 but also EphA3. The cis inhibition of EphA3 by ephrin-B2 implies that in some cases ephrins that cannot activate a particular Eph receptor in trans can nevertheless inhibit its signaling ability through cis association. We also found that an EphA3 mutation identified in lung cancer enhances cis interaction with ephrin-A3. These results suggest a novel mechanism that may contribute to cancer pathogenesis by attenuating the tumor suppressing effects of Eph receptor signaling pathways activated by ephrins in trans.
Detecting signals in the mammalian target of rapamycin (mTOR), and the estrogen receptor (ER) pathways for prediction of treatment response may be a future clinical tool in primary breast cancer. Here, we investigated the validity and value of the mTOR targets p70-S6 kinase (S6K) 1 and 2 as biomarkers for tamoxifen sensitivity in vitro and in two independent tamoxifen randomized postmenopausal breast cancer cohorts. In addition, the prognostic value of the S6Ks was evaluated. A simultaneous knockdown of the S6Ks in ER-positive breast cancer cells resulted in G1 arrest, and tamoxifen-induced G1 arrest was in part S6K1+S6K2 dependent, suggesting separate roles in proliferation and in tamoxifen response. We found S6K1 to correlate with HER2 and cytoplasmic Akt activity, whereas S6K2 and phosphorylated S6K were closer connected with ER positivity, low proliferation and nucleic p-Akt. Treatment prediction and prognosis were evaluated by immunohistochemical staining. Nuclear accumulation of S6K1 was indicative of a reduced tamoxifen treatment effect, compared with a significant benefit from tamoxifen treatment in patients without tumor S6K1 nuclear accumulation. Patients with a combination of S6K1 nuclear accumulation and S6K2 cytoplasmic accumulation in the tumor cells had no tamoxifen benefit. Also, S6K1 and S6K2 activation, indicated by p-S6K-t389 expression, was associated with low benefit from tamoxifen compared with untreated patients. In addition, high protein expression of S6K1, independent of localization, predicted worse prognosis. This was not evident for variations in S6K2 or p-S6K-t389 expression.
In conclusion, the mTOR targeted kinases S6K1 and S6K2 interfere with proliferation and response to tamoxifen. Monitoring their activity andintracellular localization may provide biomarkers for breast cancer treatment, allowing for identification of a group of patients less likely tobenefit from tamoxifen and thus in need of an alternative or additional treatment.
INTRODUCTION: mTOR and its downstream effectors the 4E-binding protein 1 (4EBP1) and the p70 ribosomal S6 kinases (S6K1 and S6K2) are frequently upregulated in breast cancer, and assumed to be driving forces in tumourigenesis, in close connection with oestrogen receptor (ER) networks. Here, we investigated these factors as clinical markers in five different cohorts of breast cancer patients.
METHODS: The prognostic significance of 4EBP1, S6K1 and S6K2 mRNA expression was assessed with real-time PCR in 93 tumours from the treatment randomised Stockholm trials, encompassing postmenopausal patients enrolled between 1976 and 1990. Three publicly available breast cancer cohorts were used to confirm the results. Furthermore, the predictive values of 4EBP1 and p4EBP1_S65 protein expression for both prognosis and endocrine treatment benefit were assessed by immunohistochemical analysis of 912 node-negative breast cancers from the Stockholm trials.
RESULTS: S6K2 and 4EBP1 mRNA expression levels showed significant correlation and were associated with a poor outcome in all cohorts investigated. 4EBP1 protein was confirmed as an independent prognostic factor, especially in progesterone receptor (PgR)-expressing cancers. 4EBP1 protein expression was also associated with a poor response to endocrine treatment in the ER/PgR positive group. Cross-talk to genomic as well as non-genomic ER/PgR signalling may be involved and the results further support a combination of ER and mTOR signalling targeted therapies.
CONCLUSION: This study suggests S6K2 and 4EBP1 as important factors for breast tumourigenesis, interplaying with hormone receptor signalling. We propose S6K2 and 4EBP1 as new potential clinical markers for prognosis and endocrine therapy response in breast cancer.


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|Posted by Dan on November 20, 2018 at 5:00 PM
CONGRATULATIONS Going Out To Shihan Benoit This Month On
Receiving His 5th Dan Renshi Certification With The Japanese Karate Federation (JKF).
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ever notice how common "patting oneself on the back" is for those who live in a one-dimensional world?
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How is the high voltage battery in hybrid vehicles differ from the regular low voltage automotive battery?
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Sub-specializing in cataract and lens implant surgery
Los Altos' premier cataract surgeon. Dr. Chang has been regularly voted by his peers into the national edition of Best Doctors in America and is listed by Beckers Review as one of the top 39 ophthalmologists in the United States. In both the 2014 and 2018 surveys, he was voted the 5th most influential ophthalmologist in the world by the international readership of The Ophthalmologist. He has maintained his private practice in Los Altos since 1984 and developed and founded the Peninsula Eye Surgery Center, which opened in 2005. He limits his practice to cataract and intraocular lens implant surgery. Dr. Chang proudly offers his unique eye surgery to the greater San Jose area.Dr. Chang's Bio


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Mangaluru, Dec 27, 2017: Members from Endosulfan Victims' Rehabilitation Centres in Kokkada in Belthangady and Koila village in Puttur, visited Tannirbavi beach, Sulthan Bathery and Kudroli Sri Gokarnanatheshwara Temple in Mangaluru, during a trip organised by Seva Bharati, Mangaluru.
Endosulfan rehabilitation centre inmate Abhishek said that his dream of going to a beach came true. He was seeing the beach for the first time, he added. Satyajit, Narayana, Tulasi, Navin, Sadat, Ramshid and others shared their excitement on the occasion.
One student, Vidya's mother Rajivi and, another student Pradeep's mother Revathi said that their children did not have an opportunity to go to school excursions like other normal children. Mangaluru tour gave them a memorable experience, which would be cherished always.
After visiting the beach, the children travelled to Sulthan Bathery on a boat. They had their lunch in Kodroli Sri Gokarnanatheshwara Temple. The Endosulfan cell of the District Health Department extended their cooperation.
Volunteers from Kalpa Trust, Rangers-Rovers unit members from Government First Grade College, Kavoor, Kokkada-Koila Endosulfan Rehabilitation Centre Manager, also lent their cooperation for the event.
Endosulfan Nodal Officer Dr Arun, Sajuddin, Sumathi Shenoy, Vinod Shenoy, Nagaraj Bhat, Shantaram Pai from the Seva Bharati Trust and Pramila H Rao from Kalpa Trust were present.


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Our members are our greatest asset at ACTP and we love to highlighting the work that they do and the resources they are proud of on a weekly basis. On ACTP Thursdays, we put the spotlight on a member of the week, a resource of the week, or a best practice of the week. We're a community of learning. We work best when we share our passions and practices.
Today, we bring you the work of ACTP Board Member, Justine Chasmar. Justine is an Assistant Professor and Director of Goucher College's Quantitative Reasoning Center. She's been in the world of learning centers for almost ten years, working at both Clemson University and Georgia Tech. Here's Justine!
ACTP: What drove you to the field of academic support and tutoring?
Justine: When I began my job hunt after my master's program, I realized all of my extracurricular activities and jobs dealt with education. I was a Supplemental Instruction (SI) Leader and tutor as an undergraduate. So, my first job after my master's program was a coordinator position in the learning center at Georgia Tech running the one-on-one tutoring program.
ACTP: What do you love the most about your job?
Justine: I love my student leaders and hosting training sessions. I love watching students learn professional skills and build community. In particular, I really enjoy and support Goucher's new curriculum, which requires two data-focused courses for all students. I teach the first data course and love when students transition to being scared of their "math class" at Goucher to the excitement that the data-driven skills they learning will heavily translate to their profession and future.
ACTP: What's a big challenge that you are facing at your center?
Justine: My center is brand new and I was hired to develop programming to support the new data curriculum. I am currently creating a diagnostic that faculty can use in their classes at the beginning of the semester to diagnose quantitative skills deficits and then creating programming to support skill development in those areas.
ACTP: What's one piece of advice you would give to your colleagues?
Justine: Build strong relationships with your colleagues. Provide autonomy to student staff to support deep, professional learning. Deliver comprehensive training to all peer leaders.
ACTP: Please describe the best practice you employ at your center.
Justine: The best practice currently in my center is the comprehensive tutor training that my student tutors complete. The training has been recently approved as a course which will begin this Fall. All tutors on my staff are required to attend a one-day orientation before classes begin and complete a course focused on best practices and pedagogy in quantitative-focused tutoring.
ACTP: Can you share your training resources with us?
Justine: I have a slew of tutor training resources and lesson plans created and am happy to share anything! Please cite my colleagues if I have a citation. Please contact me for resources at [email protected].
ACTP: What's a fun fact (not related to tutoring) that we should know about you?
Justine: I have three cats. One of them is polydactyl (has extra toes). I also love and collect wine, primarily red. I have a BS and MS in Mathematical Sciences and a PhD in Engineering and Science Education, so my current position and my research are very interdisciplinary. I am a huge Clemson fan. Go Tigers!
Are you interested in being highlighted on the ACTP Blog? Simply contact Jen Nguyen, PR Board Member, at [email protected]. All that's required to become ACTP famous is a quick phone conversation. We're looking forward to talking to you!


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This article first appeared in the St. Louis Beacon: There's little argument that downtown St. Louis has experienced a whirlwind of change in the past decade.
"If we want to compare downtown today to where it was 10 years ago, there's no comparison," said St. Louis Mayor Francis Slay. "It's far beyond almost anybody's expectations in terms of the development, the investment, the number of new residents and the vibrancy that we're seeing downtown."
The proof is in the numbers: The number of vacant buildings is down while "boutique" restaurants and retail outlets are up -- as is the residential population.
Many believe that some high-profile projects – such as Saint Louis University's Law School and City Arch River's plans for the Arch grounds – could bring more residents and businesses to the area.
But downtown St. Louis isn't without long-term challenges – and it has suffered setbacks, such as Macy's closing its downtown store. Downtown also faces competition for companies with other business centers in the region.
Over the next few weeks, the Beacon will examine the changes downtown -- its growing residential character, its interplay with business centers in St. Louis County, the impact of the historic preservation tax credit and lingering questions after the departure of Macy's.
What is downtown St. Louis?
Depending on the source, that question is a bit complicated.
The boundaries of the Downtown neighborhood, for instance, are the Mississippi River on the east, Chouteau Avenue on the south, Tucker Boulevard on the west, and Cole Street on the north. Downtown West is bounded by Tucker Boulevard on the east, Cole Street to the north, Jefferson Avenue to the west, and Chouteau Avenue on the south.
But the Partnership for Downtown St. Louis takes a more expansive view of downtown, which it says stretches from Jefferson to the river and Cass to Chouteau. Kevin Farrell, the senior director for economic and housing development for the partnership, says the boundaries encompass "what we call downtown but they generally are consistent with the city's neighborhood boundaries of Downtown, Downtown West, Columbus Square and Carr Square."
Taking the partnership's boundaries into account, here are basic facts that group provided about downtown:
- An estimated 88,000 people work downtown, based on the partnership's survey, which was conducted between 2005 and 2006 and is monitored since then. Farrell said his group plans to hire a full-time research manager "to identify the best third-party source for worker data so we can benchmark and track on an ongoing basis."
- Downtown companies with more than 1,000 employees include: Ameren, AT&T, Blue Cross Blue Shield, Lumiere Place, Nestle Purina, Peabody, Ralcorp, US Bank, U.S. Postal Service and Wells Fargo. Wells Fargo – on the edge of downtown – is the largest employer at 6,000 people.
- Farrell said some of the most prominent industries include law, accounting and architectural firms. Many federal, state and local government workers are downtown, too.
- The sectors on the upswing, Farrell said, include creative and tech firms focusing on media, software and marketing. Financial services companies – such as Wells Fargo and Stifel Nicholas – are also on the rise.
- Downtown's population rose above 14,000 people.
- U.S. Census figures show that Downtown neighborhood's population (remember this is east of Tucker) increased 361.66 percent from 806 in 2000 to 3,721 in 2010. Downtown West's population jumped 78.77 percent during that same time period; with 3,904 in 2010 as compared with 2,204 in 2000. Population growth of both neighborhoods represented the largest increase among the city's 79 neighborhoods.
- Downtown – as defined by the city, not the partnership – had the most total crimes of any neighborhood in 2012. The vast majority – 926 out of 1,299 – in the Downtown neighborhood were larcenies. But there were also 128 aggravated assaults, 81 robberies and two murders.
- Downtown West had 922 total crimes, including 595 larcenies. Carr Square had 246 total crimes, while Columbus Square had 162.
Downtown's future
The announcement that Macy's will close its downtown store is a marker for change. The shopping one finds downtown now is primarily of the boutique variety. The district that led to the second half of the "First in booze, first in shoes …" mantra for the city finds lofts not wingtips in the old warehouses.
Who lives downtown? What businesses are growing? Has the area passed the tipping point, or is its rebirth still fragile? Reporter Jason Rosenbaum looks at these and other questions.


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SKU: not available till further not
per item
*Due to unavailability of the 'diamond' screen material we can no longer provide product with the "D" designation. This configuration light display has been suspended.
The M18-3D is fitted with the popular old school diamond type of front screen. M-18-3D color organ light display.Features a three channel control system to produce lights that flash to the beat of your music. Connects via hard wire to RCA connector on back .
The features of our displays and the lighting patterns are like no others. Plus these are not made from some easily damaged vinyl covered particleboard. Cabinets are upgraded to black satin finish on plywood that features easy to remove front screen. You can also change the entire look of your display, by sliding another type of screen in or easily rearranging the light bulbs to your your liking...how cool is that?
Features 13 lights that respond to your audios low, mid and high frequency's, real wood cabinet, transformer isolated solid state main board. Ready to connect to any analog high level audio source with the RCA connector on back of unit. Connects in parallel to your speaker leads on most any sound source (stereo receiver, boom box, etc.) can be connected right along with speakers on most any sound source. Measures 18x12x5 inch weight 7 lbs.


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The Key Worksheet scan be used for a Bible Chapter or passage. Document Key People, Key Subject, Key Words (Inductive), Application, and Conclusion. Bible study should involve note taking. Whether it is answering questions, rewriting a verse, or making a list; writing helps us to slow down and focus.
Bible Journal Key Worksheet
Subscribe at the bottom of this page to get the 5 PDFs: One black and white for coloring and 4 colored versions.
Bible Journaling in a Travelers Notebook
Since you like crafts you may be interested in the Proverbs 31 & Bible Women Online Class! Here are sample pages:
Includes 22 Lessons with Over 75 Printables!
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The download includes 4 color versions and black and white version for coloring. Five PDFs in a zip file. Double click on the zip file to unzip it. Subscribe and receive the 5 Key Bible Worksheets.
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I am happy to anounce the first stable version of x2svg, a tool to
render tree like structures in scalable vector graphics (SVG).
Currently the following three input formats are supported:
- Document Type Definition (DTD)
- Java properties files
- ant build-files
Please check out the release information at Freshmeat.
And as always: please provide me with feedback, bug reports etc.


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Therapeutic Technologies
A Hallmark Research Initiative
The Therapeutic Technologies Hallmark Research Initiative was announced in 2015 with funding from the Deputy Vice-Chancellor Research for three years. Professor Alastair Stewart and Dr Susan Northfield, former chair and Academic Convenor respectively, remain involved with an active community of researchers in this field at the University.
This Initiative was vital to pave the way for a new ARC-funded Industrial Transformation Training Centre (ITTC) focused on Personalised Therapeutics Technologies. This ITTC will foster a vibrant, sustainable, growing industry of highly skilled researchers using organ-on-a-chip and bioprinting technologies that will support improved selection of therapeutics for investment in clinical trials.
The Therapeutic Technologies Hallmark Research Initiative was established to bring together a unique, critical mass of highly experienced scientists from across a range of disciplines (including pharmacology,engineering, physics, chemistry) and faculties to engage with industry to develop mechanistic and discovery studies requiring phenotypic screening in 3D chronic disease models. In addition to the quality research supported by the Initiative through generous seed-funding grants, numerous seminars, symposia and workshops for university staff and students were also offered.
The Initiative has handed over an established portfolio of connections and activities to the new Centre for Personalised Therapeutics Technologies, which is maintaining and growing the local community of researchers in the field.
Activities of the Therapeutic Technologies Hallmark Research Initiative focused on new applications of mechanopharmacology and organ-on-a-chip technology to transform drug screening processes. The Initiative selected three research themes as areas of focus:
- Cell/Tissue/Organ-on-a-chip drug screening technology - Drug Evaluation
- Cellular biomechanics - Mechanopharmacology
- Stem cells and disease modelling.
Key outcomes
- Establishment of our ARC-funded Centre for Personalised Therapeutics Technologies
- Annual science communication workshop for research students
- Development of an extensive network of researchers in the therapeutic technologies field
- Engagement with industry partners.
Modelling retinal inflammation in Alzheimer's disease using iPSC-derived RPE and microglia
- Lead investigator: Grace Lidgerwood. Co-investigators: Alice Pébay, Rosie Watson
- This project aims to assess the interaction and response of retinal cells with immune cells in a model of Alzheimer's disease (AD), using patient iPSC-derived retinal cells and microglia. It has real potential to develop a new and accurate model to functionally assess AD pathology, and a predictive model.
Quantitative proteomic analysis of drug response in metastatic colorectal cancer
- Lead investigator: Jonathan Mangum. Co-investigators: Frédéric Hollande
- This project aims to address the current limitations in colorectal cancer (CRC) by using tumour organoids, which are hypothesised to represent a great model to combine molecular and phenotypic analyses of CRC liver metastases.
Patient-derived liver cancer organoids to personalise anti-cancer treatment
- Lead investigator: Dale Christiansen. Co-investigators: Elizabeth Vincan, Joseph Torresi, Manh Bang Tran
- In this project researchers adopt organoid technology to establish a high-throughput platform to test their sensitivity to agents currently used to treat liver cancer, eradicate hepatitis B virus, and new emerging therapies, both in isolation and in combination. Using patient-derived organoids, they aim to develop personalised therapies for the patient.
Molecular tension probes to measure cell migration – a proof of concept study
- Lead investigators: Susan Northfield and Daniel Heath. Co-investigators: Brian Gao, John Karas
- In this project researchers aim to develop a mechanical biosensor that can be applied in mechanopharmacology research to measure mechanical stimulation resulting from cell migration. The novelty of this work is the application of the biosensors to assays with synthetic extracellular matrices of varying 'stiffness' allowing us to measure the impact of stiffness on mechanical migration of cells.
Development of Biocompatible Hydrogel with Tuneable Stiffness for 3D Cell/Tissue Culture
- Lead investigator: Bryan Gao. Co-investigators: Susan Northfield, Alastair Stewart, Daniel Heath, Meina Li
- This project aims to establish a workflow for synthesis and characterization of polyethylene glycol (PEG)-based biomimicry hydrogel for long-term culturing of cells and spheroids/organoids. Collectively, we will be able to manipulate both physical and biochemical parameters of the 3D culture environment to more closely recreate the physiological conditions.
Biodegradable and biocompatible magnetic materials for mechanotransduction in vitro
- Lead investigator: Andrea O'Connor. Co-investigators: David Simpson, Javad Jafari
- This project will develop methods to synthesise and characterise advanced magnetic nanobiomaterials to allow more precise control of the forces applied to cell and tissue constructs in vitro. It will have benefits in creating systems that enable spatial and temporal control of both active and passive mechanical stimuli on cells, which can be applied for improved mechanotransduction in tissue engineering and in vitro drug screening.
Characterising pluripotent stem cell reprogramming using fluorescent barcoding
- Lead investigator: Frédéric Hollande. Co-investigators: Christine Wells, Alice Pébay, Davide Ferrari
- Induced pluripotent stem cells (iPSCs) represent an ideal model to investigate the mechanisms of self-renewal and differentiation during human embryonic development. This project combines an innovative fluorescent barcoding technology with systems biology and mathematical modelling to monitor, identify and analyse the molecular characteristics of cells that fail or complete the reprogramming process.
Design of an air-liquid-interface microfluidic chip with an array of electrode
- Lead investigator: Mirella Dottori. Co-investigators: Sophie Payne, Owen Burns, Tania Kameneva
- Transepithelial electrical resistance (TEER) is a commonly accepted non-invasive technique to measure electrical resistance across a cellular layer. This project incorporates TEER measurement into an on-a-chip microfluidic platform for continuous monitoring of cell mechanical properties and cell integrity in response to drugs.
Using stem cell models to investigate mitochondrial protein transport in disease
- Lead investigator: Diana Stojanovski. Co-investigators: Ann Frazier, David Elliott, David Thorburn
- Mitochondria are "power stations" that provide our cells with energy in the form of ATP, but also play important roles in metabolism, oxidative stress, calcium homeostasis, cell signalling and death. This project involves generating human stem cell (hESC) knock out lines for a panel of mitochondrial disease genes. These stem cells will be differentiated to disease relevant cell types for further investigation, which will provide us with a powerful opportunity to dissect the pathomechanisms of specific mitochondrial diseases in a tissue specific manner.
A role for C. elegans in high-throughput drug discovery: Bridging the innovation gap
- Lead investigator: Raymond Dagastine. Co-investigators: Simon James, Gawain McColl
- The stubborn resistance of Alzheimer's disease (AD) to effective therapeutic intervention, coupled with the increasing costs of drug development, demand new & cost-effective technologies better able to identify which of the "hits" emerging from discovery pipelines will show efficacy in the clinic. This project involves developing an automated microfluidic platform to undertake both high-throughput and high-resolution imaging.
Microfluidics device for automated preparation and maintenance of air-liquid interface epithelial cell culture
- Lead investigator: Dr Christine Keenan. Co-investigators: YuXiu (Connie) Xia, Vijay Rajagopal
- The cells that line our lungs (epithelial cells) sit at the interface between the air we breathe and our blood supply carrying vital nutrients for cellular health. Recapitulating this environment in the laboratory means we can grow cells that physically resemble the airway surface. In this project, we are developing a microfluidics device to automate air-liquid interface culture of epithelial cells., which will allow live measurements of cellular function and will comprise a transformative addition to the field of organ-on-a-chip technology.
A diagnostic device for studying rare cell types in patient samples for laboratory and clinical use
- Lead investigator: Dr Daniel Heath. Co-investigators: Bill Kalionis, Shaun Brennecke
- Rare cells such as stem cells or metastatic tumour cells are critically important to human health. However, the rarity of these cells makes them difficult to isolate and study. This project involves developing a new cell culture platform that will enable these rare cells to be isolated and cultured individually in order to assess their true prevalence in patient samples as well as determining the heterogeneity within the cell population. This cell culture platform will enable unprecedented insight into the biology of these cells and has the potential to be used in disease diagnostics.
Controlling the 'Master': Generating more relevant models to facilitate discovery of novel mast cell regulators
- Lead investigator: Dr Graham Mackay. Co-investigators: YuXiu (Connie) Xia, Paul McMillan
- Mast cells are found everywhere in the body and whilst their pariah status in allergic disease is well known, there is increasing evidence that these cells can play both deleterious and protective roles in many other diseases and are thus truly 'master' cells. In this project we identify extracellular matrix compositions that permits optimal adherence of human mast cells and examine the effects of these matrices on mast cell phenotype and activation.
Identifying novel inhibitors using an FDA-approved drug screening library on a drosophila brain tumor model
- Lead investigator: Dr Rodney Luwor. Co-investigators: Leonie Quinn
- Surgery to remove brain cancer tumours in almost all cases leaves residual tumour cells that continue to divide uncontrollably, leading to tumour recurrence and patient mortality. There is an urgent need to discover novel therapeutic agents that will prolong the survival times of patients with glioblastoma. This project involves screening a large number of potential FDA-approved agents for their efficacy in established Drosophila brain tumour models to identify novel agents that can inhibit brain tumour proliferation and invasion.
Modelling retinoblastoma using human induced pluripotent stem cells (iPSCs)
- Lead investigator: Dr Raymond Wong. Co-investigators: Sandy Hung, Sandra Staffieri, Alice Pébay, Alex Hewitt, David Mackey
- Retinoblastoma (RB) is the most common malignant tumour of the eye in children caused by mutations in the RB1 gene. This project involves using RB patient-derived iPSCs with known mutations, to model this disease. Studies from this project will build a better understanding for RB disease with the ultimate aim to drive advancements in better treatment for patients.


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!! History Commons Alert, Exciting News
This is a scalable context timeline. It contains events related to the event 1998 and Before: CIA Funds Egyptian Counterterrorism Hit Squad, but Gets 'Nervous' and Eventually Closes Program. You can narrow or broaden the context of this timeline by adjusting the zoom level. The lower the scale, the more relevant the items on average will be, while the higher the scale, the less relevant the items, on average, will be.
The CIA begins a program to track Islamist militants in Europe. The program is operated by local stations in Europe and CIA manager Michael Scheuer, who will go on to found the agency's bin Laden unit in early 1996 (see February 1996). The program is primarily focused on militants who oppose the Egyptian government. It traces the support network that supplies money and recruits to them and that organizes their propaganda. US Ambassador to Egypt Edward Walker will later say that the operation involves intercepting telephone calls and opening mail. Suspects are identified in Egypt and in European cities such as Milan (see 1993 and After), Oslo, and London (see (Late 1995)). [Grey, 2007, pp. 125] The intelligence gathered as a part of this operation will be used for the CIA's nascent rendition program (see Summer 1995).
Entity Tags: Michael Scheuer, Edward Walker, Central Intelligence Agency
Timeline Tags: Complete 911 Timeline
Diplomats at the US embassy in Egypt are not informed of the CIA's rendition program. At this time the program is primarily aimed at locating enemies of the Egyptian regime and bringing them back to Egypt, where they are tortured (see Summer 1995 and Before Summer 1995). The only exception to this is US ambassador to Egypt Edward Walker, who is read into the CIA program although he is actually a State Department employee. One of the diplomats' jobs is to report on Egypt's extremely poor human rights record, including its torture methods. Walker will later comment, "It wasn't a question of mincing words… I think the human rights reports were correct." He will add that there are Chinese walls at the embassy to keep the CIA program secret from the diplomats: "The walls were huge and they only come together at the ambassador level… [The diplomats working on human rights] might have been a little upset if they knew what was going on." [Grey, 2007, pp. 126]
Entity Tags: Central Intelligence Agency, Edward Walker
Timeline Tags: Torture of US Captives
As a part of co-operation with Egyptian authorities against Islamic militancy, the CIA funds what is supposed to be a program to train Egyptian special operations forces in counterterrorism arrests. However, according to US ambassador to Egypt Edward Walker, a problem emerges: there are "too many people that die[…] while fleeing" their arrest. Author Stephen Grey will comment, "It was more of a hit squad than an arrest squad." The funding for the program is cut off in 1998, because, according to Walker, "It got to be a little too obvious and the Agency got very nervous about this." [Grey, 2007, pp. 126-127]
Entity Tags: Stephen Grey, Central Intelligence Agency, Edward Walker
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|Virtual Tour - Spherical 3D
Click on the thumbnails below to access the virtual tour of each type of houses. These are spherical photographs that allow you a view of the parts to 360 °. Now you are heading in the left click of the mouse and moving well in the three-dimensional photography. Click on the links to a different room.
Realisation Traezh Prod
New! Location visits on domain area
View of the center of the domain


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Garrett IT Services is responsible for the maintenance and operational support of technology in classrooms on campus as well as providing training. G-ITS also provides a full range of Audio/Visual equipment for faculty and staff to use in classrooms. Equipment can be reserved by emailing firstname.lastname@example.com.
G-ITS staff is available to instruct users in the operation of various equipment and technologies:
Multimedia control systems in classrooms that may include sound systems, computers, video projection, VCR and document cameras.
G-ITS can repair most types of Garrett owned audiovisual and electronic equipment. We also provide repair and preventative maintenance of installed multimedia systems in the classrooms on campus. E-mail firstname.lastname@example.com if you find equipment in need of repair.
Follow the link and watch the video to see how to make a group video call in just under a minute. A step-by-step guide is also available below the video under the heading "Making a group video call"
To talk with friends and family, you'll first need to share contact details. If you send someone a contact request, here are the directions you may need to help them accept you request before you can talk to them.


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Gerd Hatje, the influential publisher of numerous art and photography books has died aged 92.| Industry News
Press Release:
Hatje was born in Hamburg in 1915. After an apprenticeship as a typesetter in Stuttgart, he started the Humanitas Verlag (publishing house) when he was thirty. The publishing house was renamed Verlag Gerd Hatje in 1947. His varied interests were reflected in his publishing program; in addition to classics and contemporary literature, he also produced books on jazz. One of his earliest publications was a catalogue for a Max Beckmann exhibition and an edition of the German classic The Book of Le Grand by Heinrich Heine.
In the fifties and sixties the great masters of painting, sculpture, and architecture gained importance. These years were marked by Hatje's contact and friendships with artists, architects, and art historians, including Willi Baumeister, Hans Arp, Georges Braque, Alberto Giacometti, Joan Miró, Marc Chagall, Pablo Picasso, Max Ernst, Christo, Joseph Beuys, Max Bill, Le Corbusier, Marcel Breuer, Ludwig Mies van der Rohe, Walter Gropius, and James Stirling, as well as collaboration with notable international publishers such as Harry N. Abrams, Thames and Hudson, Gustavo Gili, and Eric Hazan. Gerd Hatje concentrated on realizing his dream of publishing art books of the highest quality. In his words, publishing is the process by which "intellectual spaces are made accessible."
Hatje was motivated by a strong personable conviction, embarking on intellectual adventures with artists and authors, and was always ready to take risks in the interest of a beautiful book. Among the books he published are some of the best designed art, design, and architecture books of the twentieth century, and many were awarded prizes. Hatje was always concerned with quality, both in books and art. He once said, "For me, there is neither a past nor a future in art. A work of art that cannot always exist in the present is not worth talking about."
In 1990, at the age of 75, Gerd Hatje sold his publishing house to the Dr. Cantz'sche Druckerei, the renowned Stuttgart-based printer of art books. In the beginning, the publishing division Edition Cantz and Verlag Gerd Hatje continued to work independently, but under the management of Annette Kulenkampff the two companies merged as Hatje Cantz in 1999. In the new company, now based in Ostfildern, near Stuttgart, Senior Publisher Gerd Hatje continued his daily routine until almost the very end, coming into the office every day and keeping up on trends in contemporary art. He received many awards for his achievements, including the Medal of Outstanding Merit from the German state of Baden-Württemberg, and was appointed Honorary Senator of the Stuttgart Academy of Arts. Annette Kulenkampff, who has been director for over ten years, and her team will continue publishing in the tradition of Gerd Hatje.
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ILO-IPEC, World Scout Movement extend cooperation to fight child labour
Ms. Michele Jankanish, Director of the ILO's International Programme on the Elimination of Child Labour (IPEC ) and Mr. Luc Panissod, Acting Secretary-General of the World Organization of the Scout Movement (WOSM), signed a new Memorandum of Understanding (MoU) today, to extend their cooperation in the fight against child labour for a further 3 years.
The cooperation between the two organizations will be based on a shared vision in pursuing social justice and peace, the empowerment of young people, and promoting the social dimensions of globalization.
Over the last 3 years, the enthusiastic and vibrant participation of youth in jointly planned ILO-WOSM initiatives and events created local and global interest and developed new networks involving IPEC field offices and national scout organizations. As the partnership moves forward in this important year which marks the 90th anniversary of the ILO and the 10th anniversary of the adoption of the ILO's Worst Forms of Child Labour Convention (No.182), an increasing number of scouts will be involved through raising awareness on child labour and promoting interventions to assist the most disadvantaged children.
Seminars and projects led by scouts and involving university students, mayors, government and workers representatives already took place in many places around the world, notably in Colombia, Egypt, Kenya, Ukraine, Russia and Zambia, and offered youth the possibility to gain knowledge and take action on important global issues.
"Young people are a force for change when armed with knowledge and consciousness to fight social injustice. Their energy, creativity and determination to end child labour makes a difference in their communities and influences policy makers. This gives great hope for the future and it is with this spirit that we are delighted to renew the MoU with the World Organization of the Scout Movement", said Michele Jankanish, director of ILO-IPEC.
Luc Panissod, Acting Secretary General of the WOSM rejoined her saying that "the World Organization of the Scout Movement is happy to renew the MoU with ILO/IPEC to consolidate all the good work we have achieved together so far, especially in promoting the wonderful SCREAM Education Pack and the World Day Against Child Labour. Through this partnership, our National Scout Organizations have advocated children's rights to their young members and extended non-formal education to children and young people in difficult circumstances, to help them build a better future for themselves."
The ILO's International Programme on the Elimination of Child Labour (IPEC) is active in over 90 countries and its SCREAM – Supporting Children's Rights through Education, the Arts and the Media - programme, which is a main tool to promote children's participation and youth empowerment, has been translated into 19 languages.
World Scouting is the largest global youth movement with more than 28 million members, youth and adults, boys and girls, in 160 countries. Their mission is to contribute to the education of young people through a value system based on the Scout Promise and law, to help build a better world where people are self-fulfilled as individuals and play a constructive role in society.
Useful links:
ILO: International Labour Organization
IPEC : International Programme on the Elimination of Child Labour
SCREAM: Supporting Children's Rights through Education, the Arts and the Media,
Picture: A renewed cooperation with the International Labour Organisation
You should not see this


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Light RequirementSet your living holiday tree in a bright sunny location, but not directly in sunlight. Display your European Tree® near a north-, east- or west-facing window that is bright, but sunless.
Water NeedsAnother important step to keeping European Trees® looking good is providing the correct amount of water. The soil should always be kept slightly moist, but not soggy. Test the surface with your finger and water when it's just dry to the touch. If left unchecked, over- or under-watered plants will lose their needles.
Pro Tip: After each watering, dump out the extra water that runs into the saucer or pot wrap. It's not good to leave a plant sitting in water.
PlacementNever display a living holiday tree on top of a radiator or appliance. The heat given off will dry the plant out quickly and likely result in an unhealthy-looking plant.
FertilizerApply a balanced liquid fertilizer monthly at half the usual rate recommended in the package directions.
After the HolidaysIf you live in USDA Zones 5 through 9, then you can plant your holiday tree in your garden rather than disposing of it (find your USDA Zone here). Your European Tree® should be repotted into a larger container in early January and grown indoors until the soil thaws in spring.
Cypress trees are native to temperate, coastal areas where the summer temperatures rarely exceed 80° F (27° C ). European Tree® is a short variety that will grow six to ten feet tall if grown in the right conditions in the landscape.
Visit our Plant Library to learn more about European Tree® Indoors.


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Hospital Readmissions within 30 Days after Discharge: A Review of All Readmissions to Hospitalists in One Year
1Wake Forest University, Winston Salem, NC
2Wake Forest University, Winston Salem, NC
3Wake Forest University, Winston Salem, NC
4Wake Forest University, Winston Salem, NC
5Wake Forest University, Winston Salem, NC
6Wake Forest University, Winston Salem, NC
7Wake Forest University, Winston Salem, NC
Meeting: Hospital Medicine 2009, May 14-17, Chicago, Ill.
Abstract number: 49
Readmission to hospital soon after discharge is a significant burden to patients and our health care system. Centers for Medicaid and Medicare services are proposing to add hospital readmissions as a quality measure by 2010. We aimed to study the characteristics of all readmissions within 30 days of discharge from our hospitalist service in 1 year,
This was a retrospective chart review of all readmissions to our hospital within 30 days after discharge from the hospitalist service during the year 2007. Patients readmitted to our service were identified from the University Health Consortium database. The review was done by 7 physicians. We defined the factors for readmission as: patient factor (for example, noncompliance with treatment), physician factor (for example, inadequate treatment), disease factor (for example, end‐stage COPD disease), and system issue (for example, lack of primary care physician).
The total number of readmissions in year 2007 was 239. Fifty‐five percent of all readmissions were men, 54.4% were white, 43.9% were African American, and 1.7% were Hispanic. Seventy‐one percent of readmissions were for the same diagnosis as the original admission, and 37.1% of readmissions were thought to be preventable by the reviewing physicians. Fourteen percent of readmitted patients were discharged to a skilled nursing facility, whereas most patients were discharged home (59.8%) or home with home health services (13%). A new medication was introduced in 15.9% of patients before discharge. The most common reasons for readmission were acute exacerbation of chronic illness (32.3%), medical noncompiiance (19.5%), new diagnosis or problem (18.2%), end‐stage illness (8.2%), failure of outpatient regimen (7.7%), substance abuse (6.4%), medication overdose or side effect (3.6%), and patient leaving against medical advice (2.7%). Patient factor caused 18.8% of readmissions, physician factor caused 2.9% of readmissions, disease factor caused 36.4% of readmissions, and a combination of more than 1 factor caused 33.8% of readmissions.
In our study, more than one third of our readmissions were thought to be preventable; however, a combination of factors contribute to most readmissions. Interventions targeting multiple levels in the discharge process are necessary to reduce this readmission burden to our patients and health care system.
Author Disclosure:
V. Jeevanantham, none; G. Jao, none; R. Vadlamudi, none; B. Gadi, none; M. Eapen, none; S. Stefanescu, none; P. Agborbesong, none.
To cite this abstract:Jeevanantham V, Jao G, Vadlamudi R, Gadi B, Eapen M, Stefanescu S, Agborbesong P. Hospital Readmissions within 30 Days after Discharge: A Review of All Readmissions to Hospitalists in One Year. Abstract published at Hospital Medicine 2009, May 14-17, Chicago, Ill. Abstract 49. Journal of Hospital Medicine. 2009; 4 (suppl 1). https://www.shmabstracts.com/abstract/hospital-readmissions-within-30-days-after-discharge-a-review-of-all-readmissions-to-hospitalists-in-one-year/. Accessed April 3, 2020.


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10th Annual Fair Haven Community Parade
May 29th, 2018
Thank you to the 700+ participants who joined Mary Wade for the 10th Annual Fair Haven Community Parade hosted by Mary Wade. A special note of thanks to the media who covered the event and captured the spirit and energy of the day!
Here are a selection of photos from this year's parade. There is a link to an expanded album at the bottom for more photos.
Good Morning @WQUN!
May 18th, 2018 by Kara Hunter
Good Morning!! It's Friday Marketplace Show on WQUN AM1220 with David V. Hunter,President &CEO and Lisa Hottin, Director of Development.#wqun #marywade #itsfriday #nhv #adultdaycare#assistedliving #seniorliving #shorttermrehab#vacation #volunteer #transportation #pulmonary#beautybarbers — at The Mary Wade Home.
The Art of Volunteerism!
May 11th, 2018 by Kara Hunter
Sending a big thank you to TRINITY LUTHERAN [email protected] for their voluntary service to our residents and they always bring the most beautiful hand made gifts. #nhv #seniorliving#engageateveryage #marywade #assistedliving#thearts
Coffee Cup Solute
May 10th, 2018 by Kara Hunter
This morning we're celebrating The Mary Wade Home in New Haven. It began in 1866 to provide a home and job training for young women in need. Today, it provides a continuum of care of our senior community, including adult day care, rehab services, residential care, and skilled nursing. It's expanding its memory care services with a partnership with the Alzheimer's Association of Connecticut. There are 300 employees at the Fair Haven home, making it one of the largest employers in Fair Haven.
2018 Nightingale Award s for Excellence in Nursing
May 9th, 2018
Congratulations to Tyisha Barrett, LPN, Director of Adult Day Center and Transportation at the @MaryWadeHome is a 2018 Nightingale Award Winner.
Game Night!
March 16th, 2018 by Kara Hunter
Thursday Night Card Game!
This weekly card game The Mary Wade Home is so much fun, the laughter is contagious!
Celebrating St. Patrick's Day
March 16th, 2018
Our Director of Recreation, Tiffany Burnham, bringing smiles to our residents today. Tiffany is overseeing the Irish Soda Bread baking class today using one of Mary Wade's "families" recipe!
Wine Spectator's Legende Collection and Tom Matthews
March 15th, 2018 by Kara Hunter
An Elegant Evening Filled with Friendship, Fine Food, & Endless Inspiration!
The Mary Wade Annual Wine Dinner, now in its 13th year, has evolved to become one of the more popular events in the New Haven area.
Thomas Matthews, Executive Editor of Wine Spectator, returns as host again this year. Esprit du Vin Wines will be our 2018 wine sponsor. Together, they will present an exceptional evening of delicious education. Thomas began his love affair with wine as a grape picker in Bordeaux during the harvest of 1979. He has almost 40 years of experience and will present a fascinating tour of selected wines and their characteristics.
This year's dinner will take place on Saturday, April 21 at the Omni New Haven Hotel at Yale. We look forward to seeing you for this educational and entertaining evening.
Signs of Spring
March 14th, 2018 by Kara Hunter
Goodbye winter, with your bitter winds and mounds of snow, and hello spring. Spring, of course, marks the beginning of warmer weather and longer daylight hours, the blooming of flowers and the budding of leaves. Here, our amazing garden displays visible signs that spring is arriving!
We're Hiring
March 14th, 2018 by Kara Hunter
Join the Mary Wade Team!
We are recruiting for an MDS Coordinator to join our team.
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When did your interest in the RAF begin?
I have always been interested in aircraft, starting as a young boy during the Second World War when my friends and I used to carve blocks of balsa wood into something that approximated to the shape of the combatant aircraft of the day. I later became a Technical Apprentice at Boulton Paul Aircraft at Wolverhampton for five years, and this in turn led to a working relationship with several of the Museum's exhibits. I flew a couple of times as an Observer in the Boulton Paul Balliol, I have fired guns from a gun turret similar to that in the War in the Air Hangar at Cosford, and worked on the Vickers Wellington bomber when Boulton Paul were converting them to training aircraft. After my National Service, in the RAF of course, I returned to Boulton Paul and worked in the Inspection Department checking powered flying controls units manufactured for various aircraft, including the Vulcan. One of my claims to fame is that I managed to ground the entire fleet of Vulcan bombers after finding an error in one of the units on test.
How long have you been a Volunteer and why did you decide to join?
Nineteen years ago, when I finally retired from work, I brought my grandson for a visit to the Cosford Museum and was amazed to find the Fun 'n' Flight section with its hands-on exhibits. About three weeks after the visit, there was an article in the local paper asking for people interested in volunteering at the Museum to make contact, and that, as they say, was that. My application was in the next day's post with my C.V. and I was duly invited to become a Volunteer.
How has your role changed since you first started?
After several years as a Volunteer on Fun 'n' Flight, the Museum set up the Access and Learning Department and started to advertise guided tours of the Museum and so I applied for and duly became a Guide in addition to being an Explainer on Fun 'n' Flight. Some years ago, the Second World War became a subject in the primary schools' education curriculum, and RAFM Cosford started doing oral history talks, and I was one of the volunteers invited to give talks to children on what it was like to be a child growing up in the war. Blackout experiences, rationing, entertainment, and bombing were all explained. There was always a gasp of horror when the children were told the sweet ration was 57 grams per week for an adult, and children got half of that. I show children what that was in real terms, and it is four chocolate éclairs per week.
What do you find most enjoyable about your role?
All three activity bases are enjoyable in different ways. When you are talking to a Year 6 pupil in Fun 'n' Flight and explaining why an 'H' girder is stronger but lighter than a fl at strip of the same width and the child suddenly grasps the concept I have a sense of achievement. Being a Tour Guide is enjoyable because it is often a two-way communication. You can virtually guarantee that you will end the day knowing something that you didn't know when you started the tour which makes being a Volunteer satisfying.
Would you recommend being a Volunteer to others?
Being a Volunteer brings you into face to face contact with people from all walks of life and they all have a story to tell, and that is what makes being a 'Front of House' Volunteer so enjoyable. I can thoroughly recommend volunteering at the RAF Museum to anyone


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|★ AMSTRAD CPC ★ GAMESLIST ★ SNOW JOKE (c) DELBERT THE HAMSTER SOFTWARE/THE ADVENTURE WORKSHOP ★
The idea behind this game interested me. The whole adventure takes place in once location — fans of map-making will, of course, find this to be something of a disappointment. The story is quite simple. You are trapped inside your car in a snowstorm, the doors have frozen solid and your task is to escape from the car.
Sounds quite simple: and in fact it is. The game took me just twenty minutes to complete. There are one or two nice touches where objects have been cunningly hidden, or you have to think of how to obtain them.
Although the idea of having a one-location adventure is not a new one, I was quite looking forward to seeing how much of a challenge the game would be.
It has been converted to the Amstrad format by Simon Avery using Adlan. There is nothing wrong with Simon's conversion, but I might want to forget that he converted it.
Snow Joke didn't live up to my expectations. Writing a game of this nature cannot be easy because you don't want to make your problems unsolvable. Yet I got the distinct impression that the game could have been much better.
As an example of what I mean... there is a box of matches. The ignition key is frozen into the ignition, I EXAMined the box and was told it is just an ordinary box of matches. I tried OPEN BOX (you can't). I tried to STRIKE MATCH (you can't). I even tried GET MATCH on the chance that BOX and MATCH were defined as different nouns. I still couldn't get the match out of the box.
As it happens, heating the key with the match is not the way to free it. However, there was no trapping of input to allow you to make the attempt. With such a small game there must be oodles of free memory and using Adlan, which allows almost anything to be checked for, I would have expected more catering for whatever input the player might make in such circumstances.
I was left with the belief that, whatever the game might say to the contrary, this was not an ordinary box of matches.
Snow Joke didn't live up to my expectations but nevertheless it was different from most other games I've played. For that, if nothing else, I have to give it credit. It shouldn't take you too long to solve and is worth playing as a warm-up exercise before you load in a more challenging adventure.
Reviewed by Phill Ramsay, played on the Amstrad
CPCrulez[Content Management System] v8.7-desktop/cache
L'Amstrad CPC est une machine 8 bits à base d'un Z80 à 4MHz. Le premier de la gamme fut le CPC 464 en 1984, équipé d'un lecteur de cassettes intégré il se plaçait en concurrent du Commodore C64 beaucoup plus compliqué à utiliser et plus cher. Ce fut un réel succès et sorti cette même années le CPC 664 équipé d'un lecteur de disquettes trois pouces intégré. Sa vie fut de courte durée puisqu'en 1985 il fut remplacé par le CPC 6128 qui était plus compact, plus soigné et surtout qui avait 128Ko de RAM au lieu de 64Ko.


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We are a family business and we love mountain, rock climbing and traveling. We know that children in touch with nature develop healthier physically, emotionally and mentally; so we are constantly looking for the best way to enjoy nature with our kids.
Our goal is to specialize in products for travel and for sports and leisure outdoor activities in nature with children. We go slow in our search because we combine our profession, but we hope to gradually filling the store with beautiful, useful, fun and quality products to enjoy nature and travel with the kids.
+34 687 743 938


text: Home » Archive for 04/20/19 App to See who Looks at Your Facebook — Who Looks At Your Facebook The short answer is: No, it's just not feasible to see Who has seen your Facebook profile. Below's why it can't be done - App To... How Do I See My History On Facebook — View Facebook Search History How Do I See My History On Facebook - Below's something you may not know. Facebook tracks each and every single thing you kind into its ... Posting Videos to Facebook — Post Facebook Video Posting Videos To Facebook: As Facebook increases, there are extra alternatives for sharing web content with others. And now, sharing videos... Facebook.com Email Login — Facebook Login By Email Facebook.com Email Login ~ The Facebook.com login register page will certainly offer you accessibility to your entire Facebook.com account. ... My Activity Log On Facebook — Facebook Activity Log Whenever you search for something on Facebook, your search terms get saved in your Activity Log. Right here's just how to My Activity Lo... Online Make Facebook Cover Photo — Make Facebook Cover Photo Online Make Facebook Cover Photo: For a growing brand name, social media sites look matters a lot. Every little thing that they make use of ... Facebook Videos Download — Download Facebook Videos Over the past year, Facebook Watch has seen some substantial success - because the begin of 2018, total time invested watching videos in Wat... I Want to Start A New Facebook Account — Create Facebook Account I Want To Start A New Facebook Account: You have actually ultimately given up to the power of peer stress and also have actually made a deci... Who Viewed My Facebook Page — Who Views My Facebook Facebook remains in full situation management mode. Users are leaving the platform following the discoveries that individual data was shared... Www.facebook.com Log In Sign Up — Facebook Login Sign Up Facebook is the world's most preferred social media which permits individuals to share their feelings, update condition, send free text,... Photo Likes On Facebook — Get Like On Facebook Picture Photo Likes On Facebook - Facebook is the largest social networks platform on earth! It has over 1 billion energetic users each day. As an e... How Do Copy and Paste On Facebook — Copy And Paste Facebook If you think about which tool you kind the most on in a day, it's most likely your phone, and that's fine. Yet sometimes keying on a... Facebook Login now — Facebook Login Facebook prides itself as the leading social media sites website having actually grown preferred globe wide. Facebook is an on the internet ... How to Retrieve My Facebook Password — Retrieve Facebook Password How To Retrieve My Facebook Password: So you just got hacked and also shed accessibility to your Facebook account. What now? Initially, allo... How to Put Facebook On Private — Make Facebook Private We've all done it: set up an account on social media sites or for a new gadget in our residence and simply clicked through all the setti... How Do You Create A Facebook Page — Create Facebook Fan Page A Facebook Business Page is a free chance for businesses to increase Brand understanding and also create sales on Facebook. How Do You Creat... ← Newer Posts Older Posts → Home Subscribe to: Posts (Atom)


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Orbis fundraiser Simon Speirs' family cycle London to Paris in his memory
August 2018
The family of Orbis UK supporter Simon Speirs is taking on a London to Paris cycle challenge in his memory this weekend
Simon was a keen Orbis fundraiser who sadly passed away in 2017 during the Clipper Round the World Race. The team of six has designed their own cycle route, and will be setting off this Friday from London and arriving in Paris on Sunday.
Simon's son Mike told us it feels appropriate as Simon loved cycling. Simon himself took on the London to Paris cycle challenge in 2008, and 10 years and one month later his four children and their partners will be following in his footsteps.
Mike explains: "We're doing it in honour of him as he loved a challenge."
The team has been training around London and Bristol to prepare for the more than 200 mile journey.
"I'm definitely excited – I love cycling anyway!" Mike says. "Some are a little nervous but it's all within energy levels and fitness levels."
And perhaps most importantly, what are they looking forward to most when they reach Paris? "A beer or glass of wine in the sun!"
They'll certainly deserve it after all of their effort. If you want to support the team's effort, please visit their VirginMoneyGiving page.
All of the money raised will be split between Orbis UK and Practical Action.


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CI Games has announced that its upcoming open world first-person tactical shooter Sniper Ghost Warrior 3 will have a Season Pass. While the Season Pass will be available to purchase for $29.99/£24.99/€29.99, those who pre-order the game will receive the Season Pass at no additional cost.
The Season Pass includes the following:
- Two major single-player expansions: 'The Escape of Lydia' and 'The Sabotage'
- Two multiplayer maps
- An exclusive all-terrain vehicle
- Two exclusive weapons:
- The McMillan TAC-338A Sniper Rifle
- Compound Bow
While PS4 and PC players have the option to get the "Season Pass Edition", it's unclear whether or not it will also be available on Xbox One as only a "Limited Edition" is currently available. Unlike, PS4 and PC players, Xbox One players who pre-order the game will only receive 'The Escape of Lydia', an exclusive all-terrain vehicle and the compound bow.
Sniper Ghost Warrior releases on the 4th of April on PS4, Xbox One and PC. PC players can start playing the game early via the open beta, which starts tomorrow.
Source: Official Website


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No Dig Ground Screw, there is no digging. Load bearing can be immediate and there is certainty as to the very high quality. The screws are also removable for more portable projects.
No Dig Ground Screw
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Engineered with both structural and geotechnical engineers for all applications available. Statically tested for pressure, extraction and lateral strain according to strict regulations.
● No dig, No concrete pouring, wet trades, or landfill requirements.
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Vodafone Customer Care Number for all States in India
Vodafone is a multinational telecom network service provider company that provides GSM and broadband ...Jan 17, 20
Tech News and Gadget Buying Guide
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Our aim to provide you with the latest news and tips on the best Gadget available in the market at present … more.
We're always on the lookout for passionate people who love technology as much as we do. We are looking for … more.


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Why it is Very Important to Differentiate Between the INITIATION of Violence and RESPONDING to this Initiation of Violence by Standing Up to Protect Your Rights and the Rights of Your Children
Do you think it is important to differentiate between the INITIATION of violence and when people RESPOND to this initiation of violence by standing up to protect their rights and the rights of their children?
Using lies, threats, false stories and other acts to intimidate or betray the trust of children of any age and force or threaten them to agree to treat their loving Mother (Father) disrespectfully and agree to stop communicating and spending time with them is an INITIATION of violence. It is also very evil and horrible beyond words as millions of us know.
***It is NOT REASONABLE or rational to destroy the parent/child bond UNLESS you are a TERRORIST and want to use the children to carry out acts of corruption which you know the loving Mother (Father) would never allow.
***An alienating terror parent has to use ACTS OF VIOLENCE to separate the loving Mother (Father) before he can USE the children in corrupt and evil ways and also destroy the loving parent so she (he) is unable to help the children; because the loving Mother (Father) would never allow the children to be used and abused.
This is what is happening in millions of situations of parental alienation as many psychologists and other divorce experts have shown. [www.PAlienation.org] Aside from causing these children and their loving Mother (Father) great distress and emotional and physical problems; this also deprives these children and their loving Mother (Father) of many of their fundamental rights.
A parent's right to care and have the companionship of her (his) children is fundamental, and guaranteed protection under the First, Ninth, and Fourteenth Amendments of the United States Constitution as many cases have shown.
Consequently, a person OWNS his own mind and has a RIGHT to make his or her own choices especially when it comes to having a relationship with his or her own loving Mother (Father).
***Thus, standing up against the INITIATION of violence called Parental Alienation and other corrupt acts that commonly go along with it ENFORCES our fundamental rights.
Neither a loving Mother (Father) nor their precious children of any age are expendable. They are very special and cherished for their own special talents, capabilities and personality especially by their loving parent. This is why the Constitution protects the rights of parents and children of all ages to be able to care and support one another and have a special and important companionship.
*** Any parent who destroys this companionship is not only unreasonable, irrational and evil but is also in violation of our Constitution and other laws.
***I hope this helps many understand why it is so important to be strong and stand up to those who create parental alienation.
***If we don't we sacrifice our way of life and our freedom as well as those of our children. This is how a person sells his heart, soul and mind since they have chosen to stop using it to help others and improve our world and instead have chosen to become part of the corruption.
This is not legal advice; just a perspective I thought would be helpful to share to understand why I and many will never give up trying to help our children and help end the form of abuse called Parental Alienation. I believe in freedom for ALL not just some. "All" includes women and children and others.
I hope this also provides motivation for many too as we work together in our own communities bringing awareness and understanding to the emotional torture and terror called Parental Alienation so millions of children can feel free to reunite with their loving Mother or Father. Families should help and care for each other, not destroy each other-only an evil person would do this.
***Standing up for your rights, especially the right for children and parents to have a companionship and communicate which is vital to our freedom and way of life.
***If not we return to slavery.
-By Sara Hassman, Parental Alienation Solutions, Founder, www.PAlienation.org
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Senior Director of Program, Bay Area (Prior role: Academic Director)
As an educator, I often found myself challenged with issues that were outside of the education realm. When I found Year Up, I was excited to work with young adults who were eager to learn and chose to be a part of the program. I appreciate the idea of seeing immediate outcomes from my work and believe that the most successful people at Year Up are those who live out the mission – people who are truly committed to closing the Opportunity Divide and are able to creatively innovate to make that happen.


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Pioneer Zephyr
Discussion relating to the BN, and its predecessors GN, NP, CB&Q, SP&S, SL-SF, and their subsidiaries. Visit the Friends of the Burlington Northern for more information.
2 posts • Page 1 of 1
- Posts: 403
- Joined: Thu Mar 11, 2004 9:09 pm
Pioneer Zephyr
I'm trying to find the routing used by the Pioneer Zephyr when it made its 33 state tour before entering revenue service. I'd particularly like to know how far east it came. Any assistance would be appreciated.
- Posts: 159
- Joined: Wed Dec 10, 2008 1:33 pm
Re: Pioneer Zephyr
Zimmerman, Karl (2004). Burlington's Zephyrs. MBI Publishing Company, St. Paul, Minnesota; reprint by Andover Junction Publications. ISBN 0-7603-1856-5. p.40
2 posts • Page 1 of 1


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With a Folkmanis finger puppet, you'll always have a friend nearby. All fluffy like he just popped out of his shell, this baby Emperor Penguin is looking for a little warmth and love. Ideal for use in interactive stage and puppet theater, Folkmanis Puppets are also great for teaching, daycare, school, pretend play, games and parties, or to pair with a book for storytelling. Easy to feed and care for, just add lots of love and snuggly fun. This puppet measures 2 x 2 x 4 inches (LxWxH). Folkmanis has been encouraging young imaginations to blossom since 1976 with its innovative and engaging specialty puppets, promoting creativity and discovery while winning nearly every industry, child development, and kid-tested award. Folkmanis has grown to become the premier manufacturer of plush puppets worldwide, offering over 200 of the most heartwarming and realistically designed creatures--both exotic and familiar--on the market today. (Birthday: January, 2006).
Folkmanis Puppets - 2680 - Marionnette et Théâtre - Mini Baby Emperor Penguin - B000EF5JC0


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It's National Mentoring Month, a campaign held each January to promote youth mentoring in the United States. Here at Denver Kids, all throughout the month we will be highlighting the incredible, long-term benefits that mentors have on children and teens experiencing situations of higher risk. Head over to our Facebook and Twitter pages to stay connected and learn more.
More than 200 Denver Kids students receive the support and guidance from a caring volunteer mentor. These meaningful relationships can be life-changing for both the mentor and mentee – and responsible for myriad positive youth outcomes. In fact, with a mentor, children and teens facing adversity are:
- 130% more likely to hold leadership positions
- 81% more likely to report participating regularly in sports or extracurricular activities
- 78% more likely to volunteer regularly in their communities
- 55% more likely to be enrolled in college
- 52% less likely than their peers to skip a day of school
- 46% less likely than their peers to start using drugs
Still, there are several eligible students in our program waiting to be matched. If you've considered becoming a mentor, we hope you'll take action this month.
Your relationship with a Denver Kids student can help them navigate new experiences, empower them to reach for their dreams, and transform their futures. Plus, it's a lot of fun! Go for a hike. Catch a ball game. Check out a museum. Hang out and talk. There are so many ways to engage with your mentee each month. Whatever you do, the experience is sure to be rewarding.
As a Denver Kids mentor, you'll recieve hands-on support from our dedicated Volunteer Mentor Program Manger and your mentee's Denver Kids Educational Counselor. Throughout the year, you'll also receive access to ongoing trainings, invitations to mentee/mentor mixers, and lots of additional enrichment opportunities to ensure your match is a successful one.
Click here to apply and learn more about becoming a mentor.
"I have been matched with my mentor for 12 years and there's no end to what he has helped me accomplish. He has helped me grow into the young man I am and influenced me as if he were a father figure."
– Denver Kids Mentee


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Jesse Tree Journey VBS Tees
These colorful tees will help children and volunteers remember their amazing time at Jesse Tree Journey VBS! Our tees' imprints are paint pressed so they wash better than iron-ons. Made of comfy 100% or 50/50 preshrunk cotton, you'll see participants and leaders sporting this shirt all year long. Ladies, we offer options for your specific sizing, too!
Allow 2 weeks for paint pressing and shipping.


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New Year Training Timetable
🎉🎉 Happy new year everyone 🎉🎉
🏑Hockey training starts back Monday 9th Jan 🏑
Here is a quick glance at the training times and locations for Ross on Wye Hockey Club. If you are new to the team or fancy giving hockey a go then please come along to a training session and meet the squad and coaches. If you need any more information then email us via our contact page.
Monday– Monmouth Leisure Centre
7-8pm Juniors 7-16 years
8-9pm Adults
Tuesday- Monmouth Boy School Sports Club
8-9pm Ladies training
Wednesday- John Kyrle High School, Ross on Wye
7-8pm Juniors 7-11 year olds
8-9pm Juniors age 11+