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Chronic Myelogenous Leukemia (CML)
- Author: Emmanuel C Besa, MD; Chief Editor: Koyamangalath Krishnan, MD, FRCP, FACP more...
Practice Essentials
Chronic myelogenous leukemia (CML), also known as chronic myeloid leukemia, is a myeloproliferative disorder characterized by increased proliferation of the granulocytic cell line without the loss of their capacity to differentiate. It accounts for 20% of all leukemias affecting adults. See the image below.
See Chronic Leukemias: 4 Cancers to Differentiate, a Critical Images slideshow, to help detect chronic leukemias and determine the specific type present.
Signs and symptoms
The clinical manifestations of CML are insidious, changing somewhat as the disease progresses through its 3 phases (chronic, accelerated, and blast). Signs and symptoms in the chronic phase are as follows:
Fatigue, weight loss, loss of energy, decreased exercise tolerance
Low-grade fever and excessive sweating from hypermetabolism
Elevated white blood cell (WBC) count or splenomegaly on routine assessment
Early satiety and decreased food intake from encroachment on stomach by enlarged spleen
Left upper quadrant abdominal pain from spleen infarction
The following are signs and symptoms of progressive disease:
Bleeding, petechiae, and ecchymoses during the acute phase
Bone pain and fever in the blast phase
Increasing anemia, thrombocytopenia, basophilia, and a rapidly enlarging spleen in blast crisis
See Clinical Presentation for more detail.
The diagnosis of CML is based on the following:
Histopathologic findings in the peripheral blood
Philadelphia (Ph) chromosome in bone marrow cells
The workup for CML consists of the following:
CBC with differential
Peripheral blood smear
Bone marrow analysis
Blood count and peripheral smear findings
Total WBC count 20,000-60,000 cells/μL, with mildly increased basophils and eosinophils
Mild to moderate anemia, usually normochromic and normocytic
Platelet counts low, normal, or increased
Leukocyte alkaline phosphatase stains very low to absent in most cells
Leukoerythroblastosis, with circulating immature cells from the bone marrow
Early myeloid cells (eg, myeloblasts, myelocytes, metamyelocytes, nucleated red blood cells)
Bone marrow findings
Ph chromosome (a reciprocal translocation of chromosomal material between chromosomes 9 and 22)
BCR/ABL mutation
Hypercellularity, with expansion of the myeloid cell line (eg, neutrophils, eosinophils, basophils) and its progenitor cells
Megakaryocytes are prominent and may be increased
Mild fibrosis in the reticulin stain
See Workup for more detail.
Goals of treatment of CML include the following:
Hematologic remission (normal CBC and physical examination [ie, no organomegaly])
Cytogenetic remission (normal chromosome returns with 0% Ph-positive cells)
Molecular remission (negative polymerase chain reaction [PCR] result for BCR/ABL mRNA
Tyrosine kinase inhibitors for CML
Imatinib mesylate (Gleevec): For chronic, accelerated, and blastic phases; standard treatment of choice
Dasatinib (Sprycel): For chronic phase
Nilotinib (Tasigna): For chronic phase
Bosutinib (Bosulif): For chronic, accelerated, and blast phases
Ponatinib (Iclusig): For chronic or blast phase T315I -positive cases, or in appropriate patients in whom no other TKI therapy is tolerated or indicated
Other medications for CML
Interferon-alfa: Former first-line agent; now combined with newer drugs for refractory cases
Hydroxyurea (Hydrea): Myelosuppressive agent for inducing hematologic remission
Busulfan: Myelosuppressive agent for inducing hematologic remission
Omacetaxine (Synribo): Protein translation inhibitor indicated for chronic- or accelerated-phase CML with resistance and/or intolerance to 2 or more tyrosine kinase inhibitors
Allogeneic bone marrow transplantation (BMT) or stem cell transplantation
Only proven cure for CML
Ideally performed in the chronic phase
Candidate patients should be offered the procedure if they have a matched or single–antigen-mismatched related donor available
Overall survival for allogeneic BMT with matched unrelated donors ranges from 31% to 43% for patients younger than 30 years and from 14% to 27% for older patients
Currently relegated to patients who do not achieve molecular remissions or show resistance to imatinib and failure of second-generation tyrosine kinase inhibitors (eg, dasatinib)
See Treatment and Medication for more detail.
Chronic myelogenous leukemia (CML), also known as chronic myeloid leukemia, is a myeloproliferative disorder characterized by increased proliferation of the granulocytic cell line without the loss of their capacity to differentiate. Consequently, the peripheral blood cell profile shows an increased number of granulocytes and their immature precursors, including occasional blast cells.
CML is one of the few cancers known to be caused by a single, specific genetic mutation. More than 90% of cases result from a cytogenetic aberration known as the Philadelphia chromosome (see Pathophysiology).
CML progresses through three phases: chronic, accelerated, and blast. In the chronic phase of disease, mature cells proliferate; in the accelerated phase, additional cytogenetic abnormalities occur; in the blast phase, immature cells rapidly proliferate.[1, 2] Approximately 85% of patients are diagnosed in the chronic phase and then progress to the accelerated and blast phases after 3-5 years. The diagnosis of CML is based on the histopathologic findings in the peripheral blood and the Philadelphia chromosome in bone marrow cells (see Workup).
CML accounts for 20% of all leukemias affecting adults. It typically affects middle-aged individuals. Uncommonly, the disease occurs in younger individuals. Younger patients may present with a more aggressive form of CML, such as in accelerated phase or blast crisis. Uncommonly, CML may appear as a disease of new onset in elderly individuals.
The goals of treatment are to achieve hematologic, cytogenetic, and molecular remission. Although a variety of medications have been used in CML, including myelosuppressive agents and interferon alfa, the tyrosine kinase inhibitor imatinib mesylate is currently the agent of choice, and other drugs in this category are playing increasingly important roles. However, allogeneic bone marrow transplantation is currently the only proven cure for CML. (See Treatment.)
For more information on CML, see Chronic Myelogenous Leukemia (CML) Guidelines
CML is an acquired abnormality that involves the hematopoietic stem cell. It is characterized by a cytogenetic aberration consisting of a reciprocal translocation between the long arms of chromosomes 22 and 9 [t(9;22)]. The translocation results in a shortened chromosome 22, an observation first described by Nowell and Hungerford and subsequently termed the Philadelphia (Ph1) chromosome after the city of discovery. (See the image below.)
This translocation relocates an oncogene called ABL from the long arm of chromosome 9 to a specific breakpoint cluster region (BCR) in the long arm of chromosome 22. The ABL oncogene encodes a tyrosine protein kinase. The resulting BCR/ABL fusion gene encodes a chimeric protein with strong tyrosine kinase activity. The expression of this protein leads to the development of the CML phenotype, through processes that are not yet fully understood.[3, 4, 5, 6, 7, 8, 9, 10, 2]
The presence of BCR/ABL rearrangement is the hallmark of CML, although this rearrangement has also been described in other diseases. It is considered diagnostic when present in a patient with clinical manifestations of CML.
The initiating factor of CML is still unknown, but exposure to ionizing radiation has been implicated, as observed in the increased prevalence among survivors of the atomic bombing of Hiroshima and Nagasaki. Other agents, such as benzene, are possible causes.
Historically, the median survival of patients with CML was 3-5 years from the time of diagnosis. Currently, patients with CML have a median survival of 5 or more years. The 5-year survival rate has doubled from 31% in the early 1990s to 63% for patients diagnosed from 2005 to 2011.[11] The improvement has resulted from earlier diagnosis, improved therapy with targeted drugs and bone marrow transplantation, and better supportive care.
As treatment improved, the need to stage patients according to their prognoses became necessary to justify procedures with high morbidity and mortality, such as bone marrow transplantation.
Staging of patients is based on several analyses using multiple variate analysis between the association of pretreatment host and leukemic cell characteristics and corresponding survival rates. The findings from these studies classify patients into the following groups:
Good risk (average survival of 5-6 years)
Intermediate risk (average survival of 3-4 years)
Poor risk (average survival of 2 years)
One widely used prognostic index, the Sokal score, is calculated for patients aged 5-84 years by the following equation:
Hazard ratio = exp 0.0116 (age - 43) + 0 .0345 (spleen size [cm below costal margin] - 7.5 cm) + 0.188 [(platelet count/700)2 - 0.563] + 0.0887 (% blasts in blood - 2.1)
Online calculators for the Sokal score are available.
The three categories of the Sokal score are as follows:
- Low risk: score <0.8
- Intermediate risk: score 0.8-1.2
- High risk: score >1.2
The Sokal score correlates with the likelihood of achieving complete cytogenetic response, as follows:
Low-risk patients: 91%
Intermediate-risk patients: 84%
High-risk patients: 69%
A combined prognostic model, incorporating previous models such as the Sokal score, has been devised using the number of poor-prognosis characteristics. Stages in this model are as follows:
Stage 1: 0 or 1 characteristic
Stage 2: 2 characteristics
Stage 3: 3 or more characteristics
Stage 4: diagnosis at blast phase
Poor-prognosis characteristics include the following clinical and laboratory factors:
Older age
Symptomatic presentation
Poor performance status
African-American descent
Negative Ph chromosome or BCR/ABL
Decreased megakaryocytes
Myelofibrosis (increased reticulin or collagen)
The following therapy-associated factors may indicate a poor prognosis in patients with CML:
Longer time to hematologic remission with myelosuppression therapy
Short duration of remission
High total dose of hydroxyurea or busulfan
Poor suppression of Ph-positive cells by chemotherapy or interferon alfa therapy
A German study of 139 low-risk patients with CML, according to the Sokal score, indicated that new therapeutic agents have brought improvement in survival. Median survival according to treatment used was as follows:
Busulfan: 6 years (50 patients)
Hydroxyurea: 6.5 years (55 patients)
Interferon alfa: approximately 9.5 years (34 patients)
Some patients with molecular remissions from interferon alfa may be cured, but this can only be established over time.
The tyrosine kinase inhibitor imatinib has replaced interferon as a first-line therapy, as it is associated with a higher response rate and better tolerance of adverse effects. Long-term follow-up of patients who received imatinib in the treatment of CML and achieved a complete cytogenic response 2 years after the start of treatment demonstrated that their survival was not statistically significantly different from that of the general population.[12]
Patients who develop blast crisis, which has manifestations similar to those of acute leukemia, have a very poor prognosis. Treatment results are unsatisfactory, and most of these patients succumb to the disease. Survival is 3-6 months.
A study by Wang et al addressed the prognostic impact of specific additional chromosomal abnormalities (ACAs) in CML. The concurrent presence of two or more ACAs conferred inferior survival. In patients with a single chromosomal change at the time of ACA emergence, the following three were associated with a relatively good prognosis[13] :
Trisomy 8
An extra copy of Philadelphia chromosome
In contrast, the following three ACAs were associated with a relatively poor prognosis:
-7/7q (-7/del7q)
3q26.2 rearrangements
The American Cancer Society (ACS) estimates that 8220 new cases of CML will be diagnosed in 2016, 4610 in males and 3610 in females. The ACS estimates that 1070 deaths from CML will occur in 2016, 570 in males and 500 in females. The ACS notes that over the past few decades, overall leukemia incidence rates have been slowly increasing. From 2003 to 2012, rates increased by 1.3% per year.[11]
Patient Education
Current patient education information on CML is available on the the American Cancer Society and National Cancer Institute Web sites.[14] For additional patient education information, see the Leukemia Directory.
PDQ Adult Treatment Editorial Board. Chronic Myelogenous Leukemia Treatment (PDQ®): Health Professional Version. March 9, 2016. [Medline]. [Full Text].
Sawyers CL. Chronic myeloid leukemia. N Engl J Med. 1999 Apr 29. 340(17):1330-40. [Medline].
Druker BJ, Sawyers CL, Kantarjian H, et al. Activity of a specific inhibitor of the BCR-ABL tyrosine kinase in the blast crisis of chronic myeloid leukemia and acute lymphoblastic leukemia with the Philadelphia chromosome. N Engl J Med. 2001 Apr 5. 344(14):1038-42. [Medline]. [Full Text].
Kantarjian H, Sawyers C, Hochhaus A, et al, for the International STI571 CML Study Group. Hematologic and cytogenetic responses to imatinib mesylate in chronic myelogenous leukemia. N Engl J Med. 2002 Feb 28. 346(9):645-52. [Medline]. [Full Text].
Merx K, Muller MC, Kreil S, et al. Early reduction of BCR-ABL mRNA transcript levels predicts cytogenetic response in chronic phase CML patients treated with imatinib after failure of interferon alpha. Leukemia. 2002 Sep. 16(9):1579-83. [Medline]. [Full Text].
Talpaz M, Silver RT, Druker BJ, et al. Imatinib induces durable hematologic and cytogenetic responses in patients with accelerated phase chronic myeloid leukemia: results of a phase 2 study. Blood. 2002 Mar 15. 99(6):1928-37. [Medline]. [Full Text].
Kantarjian HM, Cortes JE, O'Brien S, et al. Imatinib mesylate therapy in newly diagnosed patients with Philadelphia chromosome-positive chronic myelogenous leukemia: high incidence of early complete and major cytogenetic responses. Blood. 2003 Jan 1. 101(1):97-100. [Medline]. [Full Text].
Shah NP, Tran C, Lee FY, et al. Overriding imatinib resistance with a novel ABL kinase inhibitor. Science. 2004 Jul 16. 305(5682):399-401. [Medline].
Volpe G, Panuzzo C, Ulisciani S, Cilloni D. Imatinib resistance in CML. Cancer Lett. 2009 Feb 8. 274(1):1-9. [Medline].
Faderl S, Talpaz M, Estrov Z, O'Brien S, Kurzrock R, Kantarjian HM. The biology of chronic myeloid leukemia. N Engl J Med. 1999 Jul 15. 341(3):164-72. [Medline].
Cancer Facts & Figures 2016. American Cancer Society. Available at http://www.cancer.org/acs/groups/content/@research/documents/document/acspc-047079.pdf. Accessed: April 14, 2016.
Gambacorti-Passerini C, Antolini L, Mahon FX, Guilhot F, Deininger M al et. Multicenter independent assessment of outcomes in chronic myeloid leukemia patients treated with imatinib. J Natl Cancer Inst. 2011 Apr 6. 103(7):553-61. [Medline].
Wang W, Cortes JE, Tang G, Khoury JD, Wang S, Bueso-Ramos CE, et al. Risk stratification of chromosomal abnormalities in chronic myelogenous leukemia in the era of tyrosine kinase inhibitor therapy. Blood. 2016 Mar 22. [Medline].
PDQ Adult Treatment Editorial Board. Chronic Myelogenous Leukemia Treatment (PDQ®): Patient Version. 2002. [Medline]. [Full Text].
Barrett AJ, Ito S. The role of stem cell transplantation for chronic myelogenous leukemia in the 21st century. Blood. 2015 May 21. 125 (21):3230-5. [Medline].
Druker BJ, Talpaz M, Resta DJ, et al. Efficacy and safety of a specific inhibitor of the BCR-ABL tyrosine kinase in chronic myeloid leukemia. N Engl J Med. 2001 Apr 5. 344(14):1031-7. [Medline]. [Full Text].
Goldman JM, Druker BJ. Chronic myeloid leukemia: current treatment options. Blood. 2001 Oct 1. 98(7):2039-42. [Medline]. [Full Text].
Santos FP, Alvarado Y, Kantarjian H, Verma D, O'Brien S, Mattiuzzi G, et al. Long-term prognostic impact of the use of erythropoietic-stimulating agents in patients with chronic myeloid leukemia in chronic phase treated with imatinib. Cancer. 2011 Mar 1. 117(5):982-91. [Medline].
Sawyers CL, Hochhaus A, Feldman E, et al. Imatinib induces hematologic and cytogenetic responses in patients with chronic myelogenous leukemia in myeloid blast crisis: results of a phase II study. Blood. 2002 May 15. 99(10):3530-9. [Medline].
Tang M, Gonen M, Quintas-Cardama A, et al. Dynamics of chronic myeloid leukemia response to long-term targeted therapy reveal treatment effects on leukemic stem cells. Blood. 2011 Aug 11. 118(6):1622-31. [Medline].
Ibrahim AR, Eliasson L, Apperley JF, Milojkovic D, Bua M, Szydlo R, et al. Poor adherence is the main reason for loss of CCyR and imatinib failure for chronic myeloid leukemia patients on long-term therapy. Blood. 2011 Apr 7. 117(14):3733-6. [Medline].
Hochhaus A, Kreil S, Corbin AS, La Rosée P, Müller MC, Lahaye T, et al. Molecular and chromosomal mechanisms of resistance to imatinib (STI571) therapy. Leukemia. 2002 Nov. 16(11):2190-6. [Medline].
Marcolino MS, Boersma E, Clementino NC, et al. Imatinib treatment duration is related to decreased estimated glomerular filtration rate in chronic myeloid leukemia patients. Ann Oncol. 2011 Sep. 22(9):2073-9. [Medline].
FDA Approval for Dasatinib. National Cancer Institute. Available at http://www.cancer.gov/cancertopics/druginfo/fda-dasatinib. Accessed: October 8, 2015.
FDA Approval for Nilotinib. National Cancer Institute. Available at http://www.cancer.gov/cancertopics/druginfo/fda-nilotinib. Accessed: October 8, 2015.
Bosulif (bosutinib) [package insert]. New York, NY: Pfizer, Inc. April 2016. Available at [Full Text].
Jabbour E, Kantarjian H, O'Brien S, et al. The achievement of an early complete cytogenetic response is a major determinant for outcome in patients with early chronic phase chronic myeloid leukemia treated with tyrosine kinase inhibitors. Blood. 2011 Oct 27. 118(17):4541-6; quiz 4759. [Medline].
Hughes TP, Hochhaus A, Branford S, Müller MC, Kaeda JS, Foroni L, et al. Long-term prognostic significance of early molecular response to imatinib in newly diagnosed chronic myeloid leukemia: an analysis from the International Randomized Study of Interferon and STI571 (IRIS). Blood. 2010 Nov 11. 116(19):3758-65. [Medline].
FDA. Sprycel (dasatinib): Drug Safety Communication – Risk of Pulmonary Arterial Hypertension. US Food and Drug Administration. Available at http://www.fda.gov/Safety/MedWatch/SafetyInformation/SafetyAlertsforHumanMedicalProducts/ucm275176.htm. Accessed: October 8, 2015.
Sawyers CL. Even better kinase inhibitors for chronic myeloid leukemia. N Engl J Med. 2010 Jun 17. 362(24):2314-5. [Medline].
Jabbour E, Kantarjian H, O'Brien S, Shan J, Garcia-Manero G, Wierda W, et al. Predictive factors for outcome and response in patients treated with second generation tyrosine kinase inhibitors for chronic myeloid leukemia in chronic phase post imatinib failure. Blood. 2010 Oct 28. [Medline].
Verma D, Kantarjian H, Strom SS, et al. Malignancies occurring during therapy with tyrosine kinase inhibitors (TKIs) for chronic myeloid leukemia (CML) and other hematologic malignancies. Blood. 2011 Oct 20. 118(16):4353-8. [Medline].
Kantarjian H, Shah NP, Hochhaus A, Cortes J, Shah S, Ayala M, et al. Dasatinib versus imatinib in newly diagnosed chronic-phase chronic myeloid leukemia. N Engl J Med. 2010 Jun 17. 362(24):2260-70. [Medline].
Cortes JE, Jones D, O'Brien S, Jabbour E, Ravandi F, Koller C, et al. Results of dasatinib therapy in patients with early chronic-phase chronic myeloid leukemia. J Clin Oncol. 2010 Jan 20. 28(3):398-404. [Medline]. [Full Text].
Mulcahy N. FDA updates dasatinib label for chronic myeloid leukemia. Medscape Medical News. June 26, 2013. [Full Text].
Jabbour E, Kantarjian HM, Saglio G, Steegmann JL, Shah NP, Boqué C, et al. Early response with dasatinib or imatinib in chronic myeloid leukemia: 3-year follow-up from a randomized phase 3 trial (DASISION). Blood. 2014 Jan 23. 123 (4):494-500. [Medline]. [Full Text].
Shah NP, Guilhot F, Cortes JE, Schiffer CA, le Coutre P, Brümmendorf TH, et al. Long-term outcome with dasatinib after imatinib failure in chronic-phase chronic myeloid leukemia: follow-up of phase 3 study. Blood. 2014 Feb 25. [Medline].
Saglio G, Kim DW, Issaragrisil S, le Coutre P, Etienne G, Lobo C, et al. Nilotinib versus imatinib for newly diagnosed chronic myeloid leukemia. N Engl J Med. 2010 Jun 17. 362(24):2251-9. [Medline].
Kantarjian HM, Hochhaus A, Saglio G, et al. Nilotinib versus imatinib for the treatment of patients with newly diagnosed chronic phase, Philadelphia chromosome-positive, chronic myeloid leukaemia: 24-month minimum follow-up of the phase 3 randomised ENESTnd trial. Lancet Oncol. 2011 Sep. 12(9):841-51. [Medline].
Khoury HJ, Cortes JE, Kantarjian HM, Gambacorti-Passerini C, Baccarani M, Kim DW, et al. Bosutinib is active in chronic phase chronic myeloid leukemia after imatinib and dasatinib and/or nilotinib therapy failure. Blood. 2012 Apr 12. 119(15):3403-12. [Medline].
Chustecka Z. FDA Approves Ponatinib for Rare Leukemias. Medscape Medical News. December 14, 2012. [Full Text].
Cortes JE, Kantarjian H, Shah NP, Bixby D, Mauro MJ, Flinn I, et al. Ponatinib in refractory Philadelphia chromosome-positive leukemias. N Engl J Med. 2012 Nov 29. 367(22):2075-88. [Medline].
Cortes JE, Kim DW, Pinilla-Ibarz J, le Coutre P, Paquette R, et al. A phase 2 trial of ponatinib in Philadelphia chromosome-positive leukemias. N Engl J Med. 2013 Nov 7. 369 (19):1783-96. [Medline].
Mulahy N. Leukemia Drug Ponatinib (Iclusig) Pulled From Market. Medscape Medical News. Available at http://www.medscape.com/viewarticle/813531. Accessed: October 8, 2015.
FDA Drug Safety Communication. FDA asks manufacturer of the leukemia drug Iclusig (ponatinib) to suspend marketing and sales. US Food and Drug Administration. Available at http://www.fda.gov/Drugs/DrugSafety/ucm373040.htm. Accessed: October 8, 2015.
Nelson R. Ponatinib (Iclusig) Returns to Market, With a Few Caveats. Medscape Medical News. Available at http://www.medscape.com/viewarticle/818183?src=wnl_edit_specol&uac=72886PK. Accessed: October 8, 2015.
FDA Drug Safety Communication: FDA requires multiple new safety measures for leukemia drug Iclusig; company expected to resume marketing. US Food and Drug Administration. Available at http://www.fda.gov/Drugs/DrugSafety/ucm379554.htm. Accessed: October 8, 2015.
Synribo (omacetaxine) [package insert]. North Wales, PA: Teva Pharmaceuticals USA, Inc. October, 2012. Available at [Full Text].
Simonsson B, Gedde-Dahl T, Markevarn B, et al. Combination of pegylated IFN-a2b with imatinib increases molecular response rates in patients with low- or intermediate-risk chronic myeloid leukemia. Blood. 2011 Sep 22. 118(12):3228-35. [Medline].
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Book your room at Royal Phala Cliff Beach Resort and Spa
Get an overview of this hotelWaterfront hotel with an outdoor pool
Nestled on the beach, this Ban Chang hotel is within 9 mi (15 km) of Eastern Star Golf Course, Emerald Golf Resort, and Ban Chang Hospital. Plutaluang Navy Golf Course is 10.8 mi (17.3 km) away.
Hotel Features
This hotel features a restaurant, an outdoor pool, and a health club. Other amenities include a coffee shop/café, WiFi in public areas, and conference rooms.
Room Amenities
All 90 rooms offer WiFi, refrigerators, and flat-screen TVs with satellite channels. Other amenities available to guests include minibars, free newspapers, and free bottled water.Would you like to give us feedback? Share it! Opens in a new window
Royal Phala Cliff Beach Resort and Spa241-242 Moo 6 Phala Ban Chang Rayong 21130
Hotel Amenities
Hotel Amenities
The hotel offers a restaurant and a coffee shop/café. High-speed wireless Internet access is available for a surcharge. Royal Phala Cliff Beach Resort and Spa features an outdoor pool and a health club. This beach hotel also offers a garden and dry cleaning/laundry services.
Royal Phala Cliff Beach Resort and Spa has designated areas for smoking.
- 24-hour front desk
- Breakfast available (surcharge)
- Coffee shop or café
- Conference space
- Designated smoking areas
- Dry cleaning/laundry service
- Garden
- Health club
- Luggage storage
- Outdoor pool
- Pool sun loungers
- Restaurant
- Total number of rooms - 90
- WiFi (surcharge)
Available in all rooms: Wireless internet access ( THB 150 )
Available in some public areas: Wireless internet access ( THB 150 )
Room Amenities
- Air conditioning
- Free bottled water
- Free newspaper
- Cribs/infant beds available
- Flat-panel TV
- Hair dryer
- In-room safe
- Minibar
- Pay movies
- Private bathroom
- Refrigerator
- Satellite TV service
- Shower only
- WiFi (surcharge)
Where to Eat
Royal Phala Cliff Beach Resort and Spa has a restaurant on site.
Nearby Things to Do
Recreational amenities at the hotel include an outdoor pool and a health club.
Hotel Policies
Check-in time starts at 2 PM
Check-in time ends at Midnight
Check-out time is Noon
Payment types
- Pets not allowed
You need to know
Extra-person charges may apply and vary depending on hotel policy.
Government-issued photo identification and a credit card or cash deposit are required at check-in for incidental charges.
Special requests are subject to availability upon check-in and may incur additional charges. Special requests cannot be guaranteed.
Optional extras
- Fee for in-room wireless Internet: THB 150 (rates may vary)
- Fee for wireless Internet in public areas: THB 150 (rates may vary)
- Rollaway beds are available for an additional fee
Hotel Name
- Royal Phala Cliff Beach
- Royal Phala Cliff Beach Resort
- Royal Phala Cliff Beach Resort Ban Chang
- Royal Phala Cliff Beach Ban Chang

subset: commoncrawl


text:
Seehotel Töpferhaus, Baltic Sea Coast
- Free WiFi and free parking
Main amenities
- 47 guestrooms
- Near the beach
- Restaurant and bar/lounge
- Full-service spa
- Breakfast available
- Spa tub
- Sauna
- 4 meeting rooms
- Train station pick-up service
- In-room childcare
- Terrace
- 24-hour front desk
What's around
- In a regional park
- Auguste Windmill (4.5 mi / 7.2 km)
- Lohersand Golf Course (6.2 mi / 10 km)
- Dolmen of Lehmsiek (8.5 mi / 13.7 km)
- Rendsburg High Bridge (8.7 mi / 14 km)
- Meerwasser-Wellenbad (10.2 mi / 16.5 km)
For families
- Free cribs/infant beds
- Playground on site
- In-room childcare (surcharge)
- Babysitting or childcare (surcharge)
- Supervised childcare/activities (surcharge)
- Rollaway/extra beds (surcharge)
Seehotel Töpferhaus, Baltic Sea Coast
Hotel Info: 866-539-8117from $84
- Comfort Room (Forest View)
- Standard Room (forest view)
- Junior Suite, Lake View
- Country lodge Double Room
- Comfort Room, Lake View
- Country lodge Single Room
- Standard Room, Lake View
- Country Lodge Room
- Standard Single Room
Also popular with travelers from the USA
At a glance
Key facts
Hotel size
- This hotel has 47 rooms
- This hotel is arranged over 3 floors
- Check-in time starts at 3 PM
- Check-out time is 11 AM
- Express check-in/out
Required at check in
- Credit card or cash deposit required
- Government-issued photo ID required
Travelling with others
- Babysitting/childcare *
- Pets allowed (dogs only) *
- Specific rooms only *
- 1 per room (up to 15 kg)
- Free WiFi in public areas
- Free WiFi in rooms
- Train station pickup *
- Free self parking
Other information
- Resort fee included
In the hotel
- In-room childcare (surcharge)
- Supervised childcare/activities (surcharge)
- Buffet breakfast daily (surcharge)
- Restaurant
- Bar/lounge
- Coffee shop/café
- 24-hour room service
- Coffee/tea in a common area
- Barbeque grills
- Pool sun loungers
- Full-service spa
- Spa treatment room(s)
- Spa tub
- Sauna
- Billiards or pool table
- Number of meeting rooms - 4
- Conference space
- Meeting rooms
- Conference space size (feet) - 110
- Conference space size (meters) - 10
- 24-hour front desk
- Concierge services
- Tours/ticket assistance
- Limo or Town Car service available
- Dry cleaning/laundry service
- Laundry facilities
- Luggage storage
- Wedding services
- Multilingual staff
- Porter/bellhop
- Number of buildings/towers - 2
- Safe-deposit box at front desk
- Designated smoking areas
- Garden
- Picnic area
- Terrace
- Library
- Fireplace in lobby
- Television in common areas
- Accessible bathroom
- Roll-in shower
In the room
- Bathrobes
- Slippers
- Iron/ironing board (on request)
- Hypo-allergenic bedding available
- Pillow menu
- Sofa bed
- Sofa bed size double
- In-room massage available
- Individually decorated
- Individually furnished
- Private bathroom
- Shower/tub combination
- Free toiletries
- Hair dryer
- Flat-screen TV
- Cable TV channels
- Desk
- Free WiFi
- Phone
- Free bottled water
- Daily housekeeping
Special features
Nature Spa has 2 treatment rooms including rooms for couples and outdoor treatment areas. Services include hot stone massages, massages, facials, and body scrubs. A variety of treatment therapies are provided, including Ayurvedic and reflexology. The spa is equipped with a sauna and a spa tub.
The spa is open daily.
DiningToepferhaus - Overlooking the garden, this fine-dining restaurant specializes in Modern European cuisine and serves breakfast, lunch, dinner, and light fare. Guests can enjoy alfresco dining (weather permitting). A children's menu is available. Happy hour is offered.
On Site
- Sauna
- Spa tub
- Bicycle rentals on site
- Fishing on site
- Playground on site
- Rowing or canoeing on site
- Yoga classes/instruction on site
- Ecotours nearby
- Golf lessons available nearby
- Golfing nearby
- Hiking/biking trails nearby
- Mountain biking nearby
- Sailing nearby
- Windsurfing nearby
Seehotel Töpferhaus, Baltic Sea Coast's small print
Also known as
- Seehotel Töpferhaus
- Seehotel Töpferhaus Alt Duvenstedt
- Seehotel Töpferhaus Hotel Alt Duvenstedt
Pet-friendly rooms can be requested by contacting the property at the number on the booking confirmation.
This property does not have elevators.
For transfers, guests must contact the property 72 hours prior to arrival at the number on the booking confirmation.
Reservations are required for golf tee times and massage services and can be made by contacting the property before arrival at the number on the booking confirmation.
Mandatory fees
Pet deposit: EUR 15.00 per night
The charges below are included in the total price displayed:
- Resort fee
Resort fee inclusions:
- Spa access (may be limited)
- Internet access (may be limited)
- In-room bottled water
- Parking
Inclusions may be listed elsewhere on the page as free or for a surcharge.
Optional extras
Rollaway beds are available for an additional charge
Buffet breakfast is offered for an extra charge of EUR 9 for adults and EUR 0 for children (approximately)
Babysitting/childcare is available for an extra charge
Pets are allowed for an extra charge of EUR 15 per pet, per night
We have included all charges provided to us by this hotel. However, charges can vary, for example, based on length of stay or the room you book.
Recent reviews
Excellent 4.5 /5 from 242 reviews
Excellent stay and break from a long trip on the highway
Charming hotel. Having said that there were some problems that we encountered on our rather short stay. We arrived late in the evening and unfortunately they started drilling in the wall in the morning waking us up. Wifi was unstable going off all the time and staying off 10-20 min before coming back. There was also no pingpong table as advertised. Otherwise very charming in a lovely location but You will need a car to get in and out as it is in the countryside.
A fantastic Hotel for a quiet night in the country side next to a lake. A fantastic place to recommend. We arrived late so we did not have the chance to have dinner there, but can recommend the breakfast. The place to "fill up your batteries" after a stressful journey... I recommend this hotel!!!
Beautiful resort on a lake. Out of the city in a beautiful wooded setting.
This is a lovely hotel, situated in the woods by a lake. The hotel is calm, clean and relaxing. I'd definitely stay here again.
Is the description of this hotel not correct? Tell us


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Measures" 5" x 4" x 5", with a 1/2" perch, holds almost 3/4 cup food, hooks are 2 1/4" apart. Can be used inside or outside the cage depending where you put the hooks. Hand wash, not recommended for dishwasher.
Eliminates 99% of scattered seeds and hulls, with little or no mess.
Raise the feeder door and remove the original feeder. Hook your new Tidy Seed Feeder inside (placing the hooks on the face of the feeder) or on the outside of your bird's cage (placing the hooks on top as shown in the picture).
Your bird enters Tidy Seed and eats, leaving uneaten seeds and hulls behind the retainers; eliminating clean up.
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Recommended for Finches, Budgies, Lovebirds, Ringnecks, Small Conures, Cockatiels, Caiques, etc.
What about the comment I sent on the order...that the
opening is about 3.5". Will the feeder I ordered fit?
The feeder hooks onto the horizontal over the opening so it
should fit unless your feeder door is too low.
The Prevue Clean Life cages have very low feeder doors and
attached seed guards to keep the mess inside the cage so they won't fit those
cages. It would have to go inside.
But most wire cages have higher feeder doors so it likely
won't be a problem.
If you have a large door, you can put the hooks on the back
of the feeder and hang it inside the cage facing into the cage and the bird
goes into it that way.


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|Viewing Single Post From: SOW: Interview with Darin Brooks
|Feb 25 2009, 02:44 PM
But isn´t that normal? I doubt most of guys in their 20´s believes in true love forerver. He is a good looking barman with an open personality and zero trouble to get a date. He is enjoying his life and girls. Not every pairing has to be automatically a supercouple with a years of staying power.
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For Rent
9769 Apricot Ln Beverly Hills, CA 90210 (Beverly Hills) See your commute times
- 3 Bedrooms
- 2 full, 3 partial Bathrooms
- 89 Days on Trulia
- Single-Family Home
- 2,818 sqft
- Built in 1966
Home Details for 9769 Apricot Ln
Report this Listing Listing Reported
For Rent > CA > Beverly Hills > 90210 > Beverly Hills > 9769 Apricot Ln
3 bed, 2.5 bath, 2818 sqft, $8,900
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Ralph Waldo Emerson Middle School
University Senior High School
Listing Info for 9769 Apricot Ln
Information last updated on 06/29/2016 12:00 AM
- Price: $8,900
- 3 Bedrooms
- 2 full, 3 partial Bathrooms
- Single-Family Home
- Refrigerator
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- Status: For Rent
- Modern Architecture
- Pool
- Hot Tub / Spa
- Built in 1966
- Neighborhood: Beverly Hills
- MLS/Source ID: 16972863
- Zip: 90210
- 2,818 sqft
- Patio
Interested in Posting a Listing? Visit Trulia's Post a Rental to learn more.
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TIP 1: Keep your rent within a third of your gross household income.
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Robert Cipolloni
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Harry Potter
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First Chapter
'The Godfather of Silicon Valley'
By GARY RIVLIN
Published: November 4, 2001
The Nasdaq was far from the top of its historic ascent the first time I heard Ron Conway's name. The locale was San Francisco, inside a Mission District café whose purveyor had only recently shared with me a stock tip. The particular Internet stock he mentioned didn't seem nearly as significant as the fact that he was whispering a stock at all. The moment called to mind Joe Kennedy's famous quip that he knew it was time to get out of the market when his shoeshine boy started giving him investment advice. In today's San Francisco the equivalent is the barista in a coffee bar sharing intelligence he overhears while jerking lattes.
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The use of the term "angel" in a business context dates back to the start of the last century, when it was used on Broadway to flatter wealthy benefactors willing to pony up the capital required to stage a theater production. Nowadays the term, at least in less celestial discussions, is most often used to refer to rich individuals who invest in fledgling technology companies. Apple Computer was started with angel funding, as was Amazon.com and a long list of profitless, no-name companies that nonetheless went public and returned an imponderable payout for their patron saints.
"The grassroots of American capitalism have never been better watered," U.S. News declared in 1997 in a lengthy feature article about the angel phenomenon. According to the University of New Hampshire's Center for Venture Research, an estimated 250,000 active angel investors sank a staggering $20 billion into small companies. By 1999, those figures would swell to 400,000 angels investing an estimated $30 billion. The money flowing into venture capital funds was more staggering still. "At the height of the Internet frenzy," says Jeff Bonforte, founder of a company called I-drive.com, "friends of your friends' dads were begging to give you money." At the same time, entrepreneurs such as Bonforte were all competing for the same small group of platinum, well-connected investors, especially those widely regarded as the industry's top-tier venture capitalists.
In 1997, Paul Saffo, a director at the Menlo Park-based Institute for the Future and one of the area's better-known pundits, told me that there was so much money pouring into the Valley that "there aren't enough rat holes to put it down." That year venture firms invested a record $14 billion dollars in startups, according to the National Venture Capital Association (NVCA) and Venture Economics—a fivefold increase over the venture dollars invested just six years earlier. Yet even that record-shattering figure was soon to be dwarfed. VCs invested more than $15 billion in startups in the last three months of 1999 alone, and in the year 2000 venture investments topped $100 billion for the year. College endowments, employee pension funds, foundations, rich individuals: as a group, each dramatically upped the percentage of its holdings earmarked for these highest of high-risk investment vehicles. And why not? In 1994, the venture firm Sequoia Capital had invested $2 million in Yahoo; by 1999, assuming Sequoia and its investors hadn't sold a single share, its Yahoo stake would be worth more than $4 billion. The $6.7 million Benchmark invested in eBay in 1997 was worth $400 million when the company went public in September 1998, just nineteen months later, and an incredible $4 billion by the following spring. By 1999, the average Sand Hill Road venture capital firm specializing in early-stage investments was providing returns of 47 percent a year over the previous five years, according to the NVCA and Venture Economics. The most successful houses, such as Benchmark and Sequoia, were spinning out returns exceeding 100 percent per year.


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Jeanne's Reviews > sMothering
by Wendy French
by Wendy French
** spoiler alert ** Woman's mother shows up and kick starts her life even though the woman feels like Mom's just interfering (sp?). Her apartment is clean, her job is up in the air after she didn't sleep with her boss, she gets back together with her ex once she realized that her own fears drove him away. Unsatisfying ending with a "happily ever after" account, rather than seeing it work out.
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You are here > Home > Baseball Newsstand
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ContributorsJim Furtado
Founder & Publisher
Editor - Baseball Primer
Baseball Newsstand
— All the News That's Fit to Link
Monday, July 04, 2016
Mike Trout goes big in proposal to longtime girlfriend
Good idea to lock up Trout to a long-term deal.
Read More...
RoyalsRetro (AG#1F) Posted: July 04, 2016 at 07:06 AM | 28 comment(s)
Beats: mike trout
4th of July OMNICHATTER
Gamingboy Posted: July 04, 2016 at 12:18 AM | 60 comment(s)
Beats: mlb, omnichatter
Sunday, July 03, 2016
Wilmer Flores shines as Jose Reyes nears arrival to displace him at third
With Jose Reyes exclusively playing third base during his tune-up in the minors, there is little doubt Flores will be displaced from the position once Reyes is promoted sometime this week. But with Flores on the verge of reverting to his initial backup role, he produced a career game Sunday.
Flores went 6-for-6 with two homers, four RBIs and three runs scored as the New York Mets completed a four-game sweep of the Chicago Cubs with a 14-3 rout at Citi Field.
Flores' six hits matched the ...Read More...
Russlan thinks deGrom is da bomb Posted: July 03, 2016 at 11:26 PM | 14 comment(s)
Beats: jose reyes, mets, wilmer flores
Firing manager John Farrell won't solve problems of the Boston Red Sox - Boston Red Sox Blog- ESPN
Read More...
Jim Furtado Posted: July 03, 2016 at 07:32 AM | 12 comment(s)
Beats: john farrell, red sox
Sunday Notes: Ambidextrous Cowgill, Lawrie's Knuckler, Aussie Oriole, more | FanGraphs Baseball
David Laurila's latest.
Jim Furtado Posted: July 03, 2016 at 07:28 AM | 1 comment(s)
Beats: notes
Is this the end for Clay Buchholz in Boston?
No, when you pitch like crap game after game your manager will lose confidence in you. The Red Sox have put Kelly in the bullpen. It's time they put Buchholz on waivers.
Read More...
Jim Furtado Posted: July 03, 2016 at 07:09 AM | 2 comment(s)
Beats: clay buchholz, red sox
Fort OMNICHATTER
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I bet they learned:
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Beats: cubs
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Beats: general
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Clayton who?
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It doesn't happen every day that you watch a game for a few hours, stop watching for a couple of hours to run errands, and can put the channel back on to watch the end *WITHOUT USING THE DVR*.
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Beats: blue jays, indians
Friday, July 01, 2016
Omnichatter for July 1st.
Putting out an APB for Gamingboy.
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Beats: omnichatter
Bloomberg: Disney Said to Buy Stake in $3.5 Billion MLB Web Unit
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Beats: business, espn, media, mlbam, video
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Beats: international free agents, kevin maitan
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Beats: international free agents, red sox
MLB must permanently retire 'God Bless America,' a song that offends everyone
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Hartman: Pohlad affirms support for Molitor as Twins' woes continue - StarTribune.com
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Beats: paul molitor, twins
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Jim Furtado Posted: July 01, 2016 at 10:41 AM | 7 comment(s)
Beats: domestic violence, jose reyes, mets
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Jim Furtado Posted: July 01, 2016 at 10:30 AM | 0 comment(s)
Beats: mets, steven matz
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Beats: dugout, fights, history
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Beats: bobby bonilla, mets
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Beats: notes, yankees
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Skip to comments.Feds grant eminent domain as collateral to China for US debts.
Posted on 03/09/2009 1:20:28 PM PDT by La Enchiladita
Beijing, China -- Sources at the United States Embassy in Beijing China have just CONFIRMED to me that the United States of America has tendered to China a written agreement which grants to the People's Republic of China, an option to exercise Eminent Domain within the USA, as collateral for China's con More..tinued purchase of US Treasury Notes and existing US Currency reserves!
The written agreement was brought to Beijing by Secretary of State Hillary Clinton and was formalized and agreed-to during her recent trip to China.
This means that in the event the US Government defaults on its financial obligations to China, the Communist Government of China would be permitted to physically take -- inside the USA -- land, buildings, factories, perhaps even entire cities - to satisfy the financial obligations of the US government.
Put simply, the feds have now actually mortgaged the physical land and property of all citizens and businesses in the United States. They have given to a foreign power, their Constitutional power to "take" all of our property, as actual collateral for continued Chinese funding of US deficit spending and the continued carrying of US national debt.
This is an unimaginable betrayal of every man, woman and child in the USA. An outrage worthy of violent overthrow.
I am endeavoring to obtain images or copies of the actual document but in the interim, several different sources both in the US and in China have CONFIRMED this to me.
I'll wait for the actual documents if you don't mind.
The BS meter is pegged on this one.
Legally, Congress would have to ratify such an agreement.
it will be a bloodbath. If that happened, there would be blood in the streets.
This is huge if true. Giant.
But with these Bozos in Washington, who knows?
They can start with all those liberal voter properties that voted for The 0 ..... with O's place first ....
This had dammed well BETTER not be true!!!!!!!
Still rumor, no actually facts or documents yet.
I doubt this is true. About a decade to early.
Nothing personal but this sounds like BS.
Been posted a few times but nobody has been able to confirm it through legit sources so it keeps getting yanked.
Bad news if true, and definitely shooting worthy imo, but I don't believe it.
Scary, Im sure this isnt true. Hell, I dont know what to believe anymore. Im sure MSM wont ask?
Oh, please.
Yes, we don't know if it's true.
It is apparent, however, that Hillary offered them something to take on our debt. What else do we have to offer? China is not going to bail us out without getting more than they deserve. SOP.
I'm reading Edward Gibbon, currently, and so this isn't too surprising.
BS ping
The Federal government does not have the Constitutional authority to conclude such an agreement.
That means there's nothing to stop them.
Not going for this.
I heard about this a while back. I sure would like to get a confirm. It seems beyond belief but considering what now lives in the WH I would not be surprised.
It greatly pains me to have to admit that people on the right lie almost as much as those on the left. As the late Dr Michael Crichton wisely observed, we live in the disinformation age. I need some hard proof to believe this particular story.
Absolutely. That type of agreement has to be ratified.
I'm surprised this thread is still here. The last two or three were pulled quickly.
Maybe it's the disclaimer you used.
Most rumors have some basis in fact. I believe this one does.
It's not.
What's the source, API?
They want to buy our debt. It drives up our currency relative to theirs, helping their export industries.
Hugh, and series.
If true, it would probably lead to Civil War. I doubt O would be so obvious.
I don't think we need to offer the Chinese any incentives to buy our debt.
They have to do something with their dollars, and though it may seem strange, all the other currencies are in worse shape than the USD.
If you see it, let me know.
I may live in OHio but I'm from Missouri...Show Me!
It sounds ridiculous, but this is a time for ridiculous measures like a 3.6 trillion budget with a 1.8 trillion deficit that obama proposed.
Basically, our treasury notes may be worthless and the chinese may want to dump them if not for some guarantee.
Dumping the notes by the chinese would cause a worldwide run on the dollar effectively "bombing the world back to the stone age".
Back in the days of the Constitution you would have been correct.
First, legality means nothing to this Congress. Second, it might be buried in the porkulus package. But, how would we know, no one read it!
House Vote:
H.R. 1105 Omnibus Appropriations Act, 2009
February 24, 2009
H.R. 1105 Omnibus Appropriations Act, 2009
Managers: McGovern / Sessions
Motion offered by Foxx
To make in order and provide the appropriate waivers for amendment #6, offered by Rep. Goodlatte (R-VA), which would prevent funds in the bill from being used to exercise the power of eminent domain to take private property from a private entity and give that property to another private entity, while exempting certain enumerated uses, such as highways, prisons, public utilities, and in cases of national emergencies or national disasters declared by the President.
Defeated 4 to 7*
See post 41
To them, the thought of taking orders from a Communist commisar is probably a wet dream.
"It is apparent, however, that Hillary offered them something to take on our debt. What else do we have to offer? China is not going to bail us out without getting more than they deserve. SOP"
For the love of god, people, please research this topic. Not on Sinophile sites or Sinoscare sites. Google some monetary econ blogs and read a little bit. China doesn't need to be "offered something" to keep "taking on" our debt, China buys our debt because it has to for many reasons, all of which relate to its own economic survival.
Repeating, China buys our debt for the same reason it has ALWAYS bought our debt and will continue to buy our debt at varying levels - because it NEEDS to.
It is not complicated to understand this.
You may have to wait a long time, even if it's true. This admin. can hide birth certificates, school transcripts, theses, and close relatives for indefinite periods of time.
Crackpots and FR - it's getting tiresome.
I think hiding something like this would be even more difficult...at least I would hope.
Why wouldn't this be true? The SCOTUS already said it was ok to take land so someone else could make more money from it(Kelo). If China, or anyone else bought our debt, what other collateral would they have? Our worthless money?
The sad thing is that many people would believe this or not be surprised if it turns out to be true. I don't believe it now, but I wouldn't be surprised.
Time for affirming your state's rights in your state legislature. Be prepared to back it up.
Also, buy more ammo.
So the day will come when Wun-hung-lo comes to my door and orders me from "his" house? My internal "caca grande" meter is buzzing like hell. Not that I would put it past der Fuhrer, but I think that this would be the type of news even his wholly owned and operated MSM couldn't keep quiet. And woe to him and his brownshirts if it should get out and be proven. Nope. Don't buy it. But then I was wrong about the Jets/Colts Super Bowl too!
Disclaimer: Opinions posted on Free Republic are those of the individual posters and do not necessarily represent the opinion of Free Republic or its management. All materials posted herein are protected by copyright law and the exemption for fair use of copyrighted works.


text:
Results 1 to 10 of 10
Thread: B&W G3 LARGER HARD DRIVE
07-20-2005, 11:28 AM #1Racing666GuestB&W G3 LARGER HARD DRIVE
my b&w g3 450 has a 9.1gb IBM SCSI hard drive in it right now..i have a DVD Writer and want to write dvd's but i dont think the HDD is large enough, as i only have about 4gb free..i know with Rev. 1 b&w's, there was a data corruption issue, but i have the Rev. 2 board and wish to put a larger hard drive on the IDE bus and get rid of the SCSI setup altogether. My few questions are:
1) what is the hard drive limit in terms of size i can use?
2) i read something about the OS has to be in the first 8 gb of the drive, but can I just use 1 partition if say i get a 40-80-120 gb drive? Or what would be the best way to do it?
3) will i lose performance? i know scsi is supposedly faster than ATA, but if using a PCI adapter card, a scsi hard drive is limited by the 66mhz pci bus..
i just need some guidance i think... and some recommendations.. :dummy:
Originally Posted by Racing666
- Member Since
- Oct 18, 2004
- Location
- Norfolk, VA, USA
- Posts
- 629
- Specs:
- MacBook: 2GHz, 2GB RAM, 120GB HD; PowerTower Pro (300MHz G3 clone): 608MB RAM, 10GB HD
The B&Ws have either ATA33 or ATA66 built in that's the only reason SCSI is faster. If you use ATA133 adapter and drives or SATA (transfers~150mb/s) they will be faster. ATA100 and ATA133 drives are pretty cheap right now too, so if you got one that was not over the size limit you could hook it up the built in bus and get the host adapter later to speed things up.
Good luck
07-20-2005, 12:01 PM #3Racing666Guest
even if using an ata controller card, wouldnt it have to slow down to the PCI's 66mhz bus anyway? kinda defeats the purpose...if i could find a cheap SCSI hard drive around 40-80 gb, i'd be all set..its got the scsi that has a trapezoid shaped type connector, not the regular scsi that looks like ata..not sure if its called SCSI2 or Ultra Scsi or what...
Originally Posted by Racing666
- Member Since
- Oct 18, 2004
- Location
- Norfolk, VA, USA
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- 629
- Specs:
- MacBook: 2GHz, 2GB RAM, 120GB HD; PowerTower Pro (300MHz G3 clone): 608MB RAM, 10GB HD
The 66 or 33 in ATA33 or ATA66 is the transfer speed in MB/s not a MHz speed.
The PCI bus is plenty fast to handle the throughput of up to 4 hard drives even at RAID speeds.
I have a PCI ATA Host on my B&W because it has a Rev 1 MoBo, but I think even if I had a Rev. 2, I would keep the Host adapter because of the performance advantage.
In any case ATA100/133 drives are backwards compatible with the ATA33/66 bus on your MoBo so (except for the size restriction) you can install a much bigger drive (i.e. 80gig) for relatively cheaply. And I can't think of a reason not to do it.
Originally Posted by Racing666
- Member Since
- Oct 18, 2004
- Location
- Norfolk, VA, USA
- Posts
- 629
- Specs:
- MacBook: 2GHz, 2GB RAM, 120GB HD; PowerTower Pro (300MHz G3 clone): 608MB RAM, 10GB HD
07-20-2005, 08:48 PM #6Originally Posted by Racing666
You will need to make at least two partitions. One should be 8 GB or less, (7.5 GB is a good size), and you can use the rest for storage.
You will have to install OS X on the 8 GB volume.
If you plan on using Classic, I find it easier to make 3 partitions. One 8 GB for OS X, one 2 GB for OS 9 and the rest for storage/apps/etc.
This makes it easier if you should ever run into any problems with Classic, as you just have to wipe that partition and install again if necessary.__________________________________________________
Posting and YOU|Forum Community Guidelines|The Apple Product Cycle|Forum Courtesy
mac: a waterproof raincoat made of rubberized fabric
MAC: a data communication protocol sub-layer, also known as the Media Access Control
Mac: a brand name which covers several lines of personal computers designed, developed, and marketed by Apple Inc.
07-20-2005, 10:33 PM #7Originally Posted by D3v1L80Y
Not true. I have a 120 gig ATA drive in my 400 with Tiger installed and it works fine. It is not partitioned, I have OS 9 on another drive (6 gig). Of course I'm using a Sonnett Tempo PCI card for the drives because mine is a rev. 1 and can't handle larger drives on the moboard controller.
I dont think the B&W suffers from the rom restriction that the older iMacs did. On my rev. D iMac I had to partition the drive (40 gig) and install X and 9 on the first 8 gig partition.
How or if you partition depends on how you use the drive.Delusions are often functional. A mothers opinions about her childrens beauty, intelligence, goodness, et cetera ad nauseam, keep her from drowning them at birth.
-Lazarus Long
07-21-2005, 02:00 PM #8Racing666Guest
cradom, how is the performance of the machine? and what 120 gb hard drive do you have? i wonder if i put the 120 on the mobo controller (i have rev 2), if it will run ok or be a slug...i only want to upgrade because i tried burning a DVD and it errored on me saying not enough space on hard drive...i only have a 9gb scsi hard drive, and 4.5gb is taken up by programs/OS 10.3.9..cheapest route for now would be good.. about $100 or less is what i got to spend on a new hard drive..i'll buy a controller card later since their about $50-$100 for a decent one..
Well, it's a little slow to boot because the pci card has to power up and then spin up the drives. The drive connected to the motherboard controller spins right up, but it's not a boot drive.
I have a Maxtor 6Y120LO drive. The system runs fine. Not as fast as 9.2.2 but very usable. Faster than earlier versions of X. At one time I had a triple boot...X 10.4.2, 9.2.2, and Yellowdog Linux. (That gets hairy if you dont know what you're doing :doctor: ).
I dont remember about the rev. 2's. Go here: http://www.xlr8yourmac.com/G3-ZONE/
That Sonnett controller can handle 4 drives, tho you probably can't put 4 in the case. Mine has 3, the 120 and 2 6gigs.Delusions are often functional. A mothers opinions about her childrens beauty, intelligence, goodness, et cetera ad nauseam, keep her from drowning them at birth.
-Lazarus Long
07-22-2005, 05:38 PM #10spongGuestOriginally Posted by jhelm007
I'm in Brazil, so it's hard to compare prices, but these 33gb units cost roughly 4 x the price of an equivalent 120gb IDE unit.
So, it's around 15 x the price per GB, if that makes sense.
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|Miro 5.0.4 10/10/12
|Free (GPL)
|Miro 2.5.3 19/06/13
|Miro 2.5 19/06/13
|Miro 2.0.2 19/06/13
|Miro 2.0 19/06/13
Apps & Games content ratings on Softonic
Watch internet videos like you watch TV
Miro is defined by its developers as "the open-source Internet TV platform". In fact, it aims to bring together a community of broadcasters to create, what it calls, an 'Internet video ecosystem'. But besides all these elaborate definitions, Miro is simply a great application to watch online videos.
Miro uses open protocols to let you subscribe to video channels and video blogs that are fed in via RSS, and uses BitTorrent to download the files. You can also watch videos from Youtube and Dailymotion, or use it to subscribe to a Youtube channel you like, for example. Miro supports video playlists and can resume playback just where you stopped. You can even watch those videos on a separate window while you keep on browsing Miro's Guide
This guide is actually a welcome one, as it helps you get started with Miro and also lets you search and browse the various channels effortlessly. Not only can you browse through the existing channel list, but you can easily add new channels to the sidebar simply by clicking a button to subscribe. There are more than two thousand channels available, and all of them are free. Beware, however, that the video quality may vary from one channel or video source to another, and that there may be video services that are not available in your country.
Miro is a perfect example of how video content from different sources can be integrated into one single application and served directly to your PC in a fast, easy and elegant way.
Recent changes
- * Bug fixes
- - (all) crash on database upgrade: OperationalError: no such table: dtv_variables
- - (OS X) Vimeo "likes" are not downloaded
- - (all) Intermittent Vimeo download failure
- - (Windows) Monitor goes into power saving mode while playing back
- - (Windows) Mouse doesn't disappear in fullscreen playback mode on windows
- - (all) clicking yes to donate resets the counter to 100 but doesn't wait 6 months before displaying the ask again
- - (all) Re-fetch 7-digital data
- - (all) Crash in check_subprocess_hung
- - (all) n _scrape_vimeo_download_errback TypeError: sequence index must be integer, not 'str'
- - (all) AttributeError: 'NoneType' object has no attribute 'group'
- Thousands of free video channels available
- An easier, faster way to watch videos online
- Support for many video formats and video playlists
- Resumes playback right where you stopped
- You can keep on browsing while watching videos on a separate window
- Some video services may not be available in your country
- Video quality may vary
"Great program!"
I've been using Miro for several months now and I have to say that the overall ease-of-use and speed of this program is tops!
- Fast program, ease-of-use, no spyware
- Reminds me of Apple
- Views:
- 455
- Date:
- 22 May 2012
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text:
Not sure if I feel comfortable with him shorting the long bond, the housing market and job market still in a crisis. I still say he is to early?
I didn't get him shorting the long bond either, I'm no pro like Cass is but I'm smart enough to figure out with the housing market still in shambles and the unemployment rate still high and Europe in crises that only a moron would buy TBT at $22 or even $20. I like Cass think the Long Bond is a bubble also, but irrational markets can stay solvent longer than I can trying to chase it lol Any one seeing it improving any time soon? I don't, TBT could go to $15 no?


text: On Thursday, 24 May 2007 00:53, anthony baldwin wrote: > >I'm not talking about backing up, but rather making a separate partition > >so the folder won't be erased at all during reinstalls. > > > >Matt Flaschen > > I should do that, too... > I have to say, I used other distros for about 6 yrs before coming > recently to Ubuntu, > and I had never heard of this idea. Now, that I have, it makes so much > sense, and I have to wonder > why I never saw the suggestion on the rh/fedora lists. Never used Fedora, but before switching to Warty, I used RedHat 5.2 - 7.3. The install guides always suggested separate partitions for /home /usr/local /boot /swap /opt The problem in those days was that drives were smaller so, unless you accurately predicted the partition size needs, you could end up out of space on a partition while having surplus space on another. Ubuntu's desire (as of warty) to only partition / /boot /swap seemed great at the time. It does, however impede clean installs. So, as to the original question, he may also want to back up /usr/local /opt -- _____________________________________________________________ Art Alexion PGP fingerprint: 52A4 B10C AA73 096F A661 92D2 3B65 8EAC ACC5 BA7A Keyserver: hkp://subkeys.pgp.net The attachment - signature.asc - is my electronic signature; no need for alarm. Info @ http://mysite.verizon.net/art.alexion/encryption/signature.asc.what.html _____________________________________________________________ -------------- next part -------------- A non-text attachment was scrubbed... Name: not available Type: application/pgp-signature Size: 309 bytes Desc: not available Url : https://lists.ubuntu.com/archives/kubuntu-users/attachments/20070607/fa31c2c5/attachment.pgp


text:
Acorn Squash Soup Recipe
"The recipe for this thick and creamy soup was given to me by a fellow squash lover," reports Dorrene Butterfield from her home in Lincoln, Nebraska. "The attractive rich yellow soup is especially enjoyable during the cool nights of Indian summer."
- 1 small onion
- 1/4 cup chopped celery
- 2 tablespoons butter
- 2 tablespoons all-purpose flour
- 1 teaspoon chicken bouillon granules
- 1/2 teaspoon dill weed
- 1/4 teaspoon curry powder
- Dash cayenne pepper
- 2 cups chicken broth
- 1 can (12 ounces) evaporated milk
- 3 cups mashed cooked acorn squash
- Salt and pepper to taste
- 5 bacon strips, cooked and crumbled
- 1. In a large saucepan, saute onion and celery in butter. Stir in flour, bouillon, dill, curry and cayenne until blended. Gradually add broth and milk. Bring to a boil; cook and stir for 2 minutes. Add the squash, salt and pepper; heat through.
- 2. In a blender, process the soup in batches until smooth. Pour into bowls; sprinkle with bacon. Yield: 6 servings.
Nutritional Facts
1 cup: 0 calories, 0g fat (0g saturated fat), 0mg cholesterol, 0mg sodium, 0g carbohydrate (0g sugars, 0g fiber), 0g protein
Reviews for Acorn Squash Soup
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