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Hotels near Adelaide Casino
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- Most bookedThe Playford Adelaide MGallery by Sofitel
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"Parking was dicey but we managed. Didn't dare move the car out during our stay in case we couldn't get back in again."
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"I had a very pleasant stay - knowlegeable and helpful staff and good facilities"
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"Great hotel, convenient parking at reasonable price. Room well appointed, clean and well presented. All amenities well maintained."
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"Located next to the casino, close to the rail station, sporting & entertainment stadiums within walking distance. Spacious rooms. We had requested an early check in and this was not an issue for the hotel making our weekend much more relaxed. "
A Traveller, May 2016, AU
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"We enjoyed a two night stay over a weekend to negate the need to find a car park when our daughter decided she wanted to celebrate her 30th birthday at KW2. Being Adelaide suburbanites, we chose to go to town by bus and just packed an overnight bag/case. We had booked a city view room, but were u..."
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"Great location 5 minute walk to Rundle mall,10 minutes to Adelaide Ovel,Casino & tram stop just up the road..."
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"The reviews for this hotel have been really harsh and are currently undeserved. My husband and I stayed here recently and either it has improved dramatically or the previous reviewers haven't done much travelling. The property is old, I'll admit that but it was clean and really spacious. Our room..."
A Traveller, Nov 2014, AU
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Map of hotels near Adelaide Casino
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- SA Aquatic and Leisure Centre
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- Victoria Square
- Adelaide Botanic Gardens
- Belair National Park
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- Magill Estate Winery
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That's the word of the day. It means something to savor, knowing that area chefs are taking the bounty of the Central Coast and transforming it into delicious feasts for the senses. New Times is proud to provide a guide to the tastes of the restaurants, shops, and cooks who make this region such an amazing place to eat, drink, and be thankful for the view. Enjoy.
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In developed countries, the lifetime risk of developing colorectal cancer (CRC) is 5%, and it is the second leading cause of death from cancer. The presence of family history is a well established risk factor with 25-35% of CRCs attributable to inherited and/or familial factors. The highly penetrant inherited colon cancer syndromes account for approximately 5%, leaving greater than 20% without clear genetic definition. Familial colorectal cancer has been linked to chromosome 7q31 by multiple affected relative pair studies. The MET proto-oncogene which resides in this chromosomal region is considered a candidate for genetic susceptibility.
MET exons were amplified by PCR from germline DNA of 148 affected sibling pairs with colorectal cancer. Amplicons with altered sequence were detected with high-resolution melt-curve analysis using a LightScanner (Idaho Technologies). Samples demonstrating alternative melt curves were sequenced. A TaqMan assay for the specific c.2975C >T change was used to confirm this mutation in a cohort of 299 colorectal cancer cases and to look for allelic amplification in tumors.
Here we report a germline non-synonymous change in the MET proto-oncogene at amino acid position T992I (also reported as MET p.T1010I) in 5.2% of a cohort of sibling pairs affected with CRC. This genetic variant was then confirmed in a second cohort of individuals diagnosed with CRC and having a first degree relative with CRC at prevalence of 4.1%. This mutation has been reported in cancer cells of multiple origins, including 2.5% of colon cancers, and in <1% in the general population. The threonine at amino acid position 992 lies in the tyrosine kinase domain of MET and a change to isoleucine at this position has been shown to promote metastatic behavior in cell-based models. The average age of CRC diagnosis in patients in this study is 63 years in mutation carriers, which is 8 years earlier than the general population average for CRC.
Although the MET p.T992I genetic mutation is commonly found in somatic colorectal cancer tissues, this is the first report also implicating this MET genetic mutation as a germline inherited risk factor for familial colorectal cancer. Future studies on the cancer risks associated with this mutation and the prevalence in different at-risk populations will be an important extension of this work to define the clinical significance.
Colorectal cancer (CRC) is one of the more familial of cancers, and the presence of a family history of this malignancy is a well established risk factor. Twin studies suggest inherited and/or familial factors contribute to 25-35% of CRC cases [1]. The highly penetrant inherited colon cancer conditions including familial adenomatous polyposis, Lynch syndrome (hereditary nonpolyposis colorectal cancer), Peutz-Jeghers, Cowden syndrome and juvenile polyposis account for approximately 5% of CRCs, leaving greater than 20% without clear genetic definition [2]. An individual's risk of CRC doubles with one affected first degree relative and progressively increases with each additional affected first, second, or third degree relative [3 ,4].
We have previously reported linkage of the 7q31 locus in a cohort of sibling pairs affected with CRC [5]. Other CRC and polyp-affected relative pair studies have also reported linkage to this region of chromosome 7 [6,7]. One candidate gene in this region is the proto-oncogene and tyrosine kinase receptor, MET. MET is expressed mainly on the surface of epithelial cells. In response to binding of the MET ligand, hepatocyte growth factor (HGF), C-terminal tyrosine residues are phosphorylated followed by a cascade of intracellular signals resulting in activation of MAPK and/or PI3K/Akt pathways [8,9] (Figure (Figure1).1). In this way, aberrant activation of MET leads to increased cell proliferation, invasion, and metastasis [10,11]. The MET gene is found to be amplified in approximately 10% of CRCs, and amplification is associated with advanced stages and worse prognoses [12-14]. Additionally, MET gene missense mutations are found in ~3% of CRCs, in particular p.R970C and p.T992I (also reported as p.R988C and p.T1010I) [15]. Specific missense mutations in the tyrosine kinase domain of MET (amino acids 1112 to 1268) lead to hereditary papillary renal carcinoma (MIM ID 164860). Individuals with this condition are usually symptomatic by their fourth decade of life due to multifocal kidney lesions.
To evaluate the possibility that germline changes in the MET gene may lead to colon cancer susceptibility in a familial setting, we scanned and sequenced all coding exons of MET in our sibling pair cohort [5]. Here we report that MET p.T992I, a non-synonymous change suggested to have oncogenic potential, is present in the germline DNA of 5.2% of this cohort and this same change is confirmed in a second familial colon cancer cohort with two affected first degree relatives at a frequency of 4.1%. This substitution is reported in <1% of the general population and has been reported in cancer cells of multiple origins, including 2.5% of colon cancers [15-18].
All aspects of this study were approved by University of Utah's Institutional Review Board for human subject research. Research participants were consented to participate in a study of genetic factors leading to colon cancer risk. Ascertainment and collection of the sibling pair study cohort have been previously described [5]. Briefly, 169 siblings (148 affected with CRC and 21 unaffected) from families with two siblings diagnosed with colorectal adenocarcinoma or a polyp with high grade dysplasia and all of age > 20 years were included. A young onset population, defined as a CRC diagnosis ≤ 50 years (n = 130) and a familial colon cancer cohort, defined as two first-degree relatives diagnosed with CRC (n = 169), was used for confirmation of the MET p.T992I mutation. These populations have been previously described as well [19]. For both of these populations, diagnosis was confirmed with pathology report or state cancer registry data. Known syndromes were excluded through medical record review and/or molecular analysis including analysis for microsatellite instability (MSI) using the "reference marker panel" (BAT25, BAT26, D2S123, D5S346 and D17S250) and sequencing of normal tissue from cancers demonstrating MSI as previously described [5,19]. The presence of polyposis, defined as greater than 10 colonic polyps resulted in exclusion from the study.
Primers were designed using Primer 3 [20] to PCR-amplify each of the coding exons of MET. Amplicons of 250-500 base-pairs in length were generated using LC Green dye mastermix (Idaho Technologies, Salt Lake City, UT, USA), for optimal high-resolution melt-curve analysis (HRM) using a LightScanner (Idaho Technologies). Samples demonstrating alternative melt curves were sequenced using Applied Biosystems 3730XL capillary sequencer.
Tumor DNA, micro-dissected and extracted from formalin-fixed paraffin-embedded (FFPE) blocks, was assayed for the presence of the MET c.2975C >T mutation (NM_000245.2, NP_000236.2), which results in p.T992I in the protein, using the Applied Biosystems probe assay for rs56391007 and TaqMan Genotyping Master Mix according to manufacturers' specifications (Applied Biosystems, Carlsbad, CA, USA). The major "C" allele was labeled with VIC (517 nm emission) and the minor "T" allele was labeled with FAM (554 nm emission). Products that peaked before 35 cycles using the BioRad CFX96 Real-time PCR machine with a signal of >500 RFU on the respective VIC or FAM channel were designated for that allele. When the MET c.2975C > T mutation was identified in tumors, normal DNA from the same patient (blood when available, otherwise FFPE micro-dissected normal) was tested for the mutation. Allelic imbalance was evaluated using cycle-time (CT) data generated by real-time quantitative PCR with the TaqMan assay. Where ΔCT = cycle time of VIC (c.2975C, wild type allele) - cycle time of FAM (c.2975T, mutant allele), with the fold excess mutant alleles = 2(Δ CT tumor-Δ CT normal).
Because the MET gene resides within the linkage peak previously reported on chromosome 7q31 [5] and because it is an oncogene amplified in CRCs, the MET exons were scanned for variation using high-resolution melting (HRM) followed by sequencing of samples with different melt curves in our study population. The sibling pair population was composed of 169 subjects from 77 families including 148 affected with CRC and 21 unaffected [5]. The population is 86% Caucasian, 5% Black, 1% Native American, and 8% other. All known MET coding SNPs reported in dbSNP with population frequencies as well as coding variants identified in germline DNA of the sibling pair population but not present with population frequencies in dbSNP are listed in Table Table1.1. The frequency of 9 of the SNPs in the sibling pair population are representative of the allelic frequencies reported in dbSNP and one unreported SNP results in a synonymous change (Table (Table1).1). Three non-synonymous MET gene changes were identified in the germline DNA of affected individuals whereby a population frequency was either not available or the minor allele frequency was less than 1% in dbSNP build 132 (http://www.ncbi.nlm.nih.gov/projects/SNP) [21]. Two individuals each had a single change in the extracellular domain of the MET receptor; p.V136I and p.E168D. However their affected siblings did not harbor the respective changes and the changes are not predicted to disrupt protein function with both Polyphen and SIFT analysis tools [22,23]. Seven of the 148 affected individuals, and none of the unaffected siblings, have the p.T992I germline change, which resides in the tyrosine kinase domain of MET. These 7 individuals represent 3 affected sibling pairs and 1 affected individual with an unaffected sibling or 4 of 77 families (5.2%). The consensus threonine amino acid at position 992 is completely invariant across vertebrates to zebrafish and both Polyphen and SIFT analyses predict that this change is damaging. This mutation has been previously reported in 2.5% of colon tumors [15]. It is reported in dbSNP at 0.7% based on low coverage sequence of ~600 individuals from the 1000 Genomes project (4 individuals). There are no clear pathologic characteristics of the individuals with p.T992I germline mutation (Table (Table2).2). The average age of CRC diagnosis in the sibling pairs with p.T992I germline mutation was 61.9 years with a range of 44 to 75 years, whereas the average age of the study population was 58.7 years (range 28-91).
Because each of the families in the sibling pair cohort is of Caucasian descent, we were interested to determine whether there was evidence of a founder mutation. Using polymorphic markers surrounding the MET locus (D7S2418, D7S486 and D7S648), the haplotype segregating with the mutation in each of the 4 families was identified and found to be distinct. This suggests that this mutation arose independently.
Using tumor DNA from individuals diagnosed with CRC ≤50 years (n = 130) or individuals diagnosed with CRC and having a first degree relative with CRC (n = 169), a TaqMan assay was used to screen for the p.T992I mutation. We found that 9 of 299 tumors (3.0%) harbored the change, 1 from diagnosis ≤ 50 years (0.8%) and 8 from the two affected first-degree relative probands with CRC (4.7%). Interestingly, two of the 8 are a second-degree relative pair from a large family with excess colon cancer; one ascertained as a member of a first-degree relative pair (hr10-D on Table Table2)2) and the other ascertained as a member of a parent-child pair also diagnosed ≤ 50 years (hr16-D on Table Table2)2) [24]. Since these two are known to be closely related, the frequency is adjusted to 4.1% (7 of 168 families). DNA from normal tissue was available from all cases except hr16-D, and this individual was found to be an obligate carrier based on children's genotypes, thus the mutation was present in all 9 suggesting it was germline in origin. None of the 16 colorectal cancers arising in individuals with the germline p.T992I had microsatellite instability, a feature of defective mismatch repair pathway.
Because amplification of the MET gene occurs in colon cancers, we hypothesized that the allele harboring p.T992I would be preferentially amplified in colon tumors. Using cycle time from the TaqMan assay, we compared the difference of copy number of the mutant (c.2975T) from wild type (c.2975C) in tumor versus normal. Six of the 15 cases examined showed an excess of the mutant c.2975T allele over the wild type allele (Table (Table2),2), but this was not universal. This finding suggests that, in the presence of this mutation, additional amplification of the gene is not required for establishment of cancer.
Ten percent of all colon cancers arise in a familial setting when defined as two or more affected first-degree relatives [19]. There is a also two-fold increase of developing colon cancer with an affected first-degree relative [3]. Specifics of the genetic etiology of this group are not defined, leaving a gap in our knowledge of moderate risk genetic variants. A germline mutation in the MET gene, p.T992I, was identified in ~4.5% of colon cancers arising in first-degree relative pairs from two separate cohorts. The mutation was observed in less than 1% of colon cancer cases that occurred ≤ 50 years, suggesting that it does not promote very young CRC. MET p.T992I is also reported in the germline DNA of 4% of thyroid cancers [17], one endometrial and two melanoma cancer cases, and one normal individual [16]. In cancers, the p.R970C and p.T992I mutations are thought to affect phosphorylation of the serine residue (p.S985) that negatively regulates MET kinase activity [8,25]. By comparison, the specific missense mutations reported in hereditary papillary renal carcinoma (HPRC) surround two tyrosine residues within the catalytic site (p.Y1234 and p.Y1235) that positively regulate kinase activity (Figure (Figure1).1). The phenotypic differences between p.T992I and HPRC MET mutations could be explained by the difference between factors driving negative versus positive activation of MET kinase.
We propose a model whereby the p.T992I mutation functions as a progression factor rather than an initiation factor in the canonical colon cancer model [26]. Specifically, we hypothesize that an adenoma is initiated through somatic mutations in the canonical APC pathway, then the adenoma acquires other proliferative mutations, and in the presence of an underlying MET p.T992I mutation, is then able to move beyond the mucosal layer to become invasive colon cancer. This hypothesis is supported by the following observations. The individuals we identified with the MET p.T992I germline mutation do not have the hallmarks of inherited mutations in initiating factors such as multiple adenoma formation (APC gene) or microsatellite instability (mismatch repair genes). Additionally, CRC diagnosis under age 50 is infrequent. In fact, the chromosome 7q genetic locus is associated in affected relative pair studies when CRCs are included and adenomas are excluded [5-7]. The average age of CRC diagnosis in the general population is ~71 years, and it is estimated that 10 years are needed for a small polyp to progress to invasive CRC [27]. A model of rapid progression of polyp to cancer in the presence of MET p.T992I is supported in that individuals with the p.T992I mutation are diagnosed with CRC at an average age of 63 years. This would be when the general population, on average, is developing adenomas that will progress to cancer. This mutation also occurs in a variety of cancers including colon, melanoma, endometrial, thyroid, and mesothelioma [15-18] with germline confirmation in colon, thyroid, uterine, and melanoma [16,17] suggesting that it is not a tissue-specific mechanism. MET p.T992I mutation is proposed to function through inhibition of phosphorylation of Ser985, which, when phosphorylated, corresponds with reduced MET signaling (Figure (Figure1)1) [28,29]. In cell models, the specific mutation reported here generally affects invasive behaviors including changes in cell morphology, adhesion, motility, migration and anchorage-independent growth but not proliferation, such as IL-3 independent growth in Ba/F3 cells [18,29]. Based on these reports, it is reasonable to predict that MET p.T992I requires a growth signal (activation) but then is disabled in its ability to turn off the activation through phosphorylation of p.S985. It has also been shown that over expression of MET is an early event in the colorectal adenoma-carcinoma sequence [30]. In the context of a proliferating precancerous colonic adenoma, over expression MET p.T992I and the inability to turn off activation could allow the invasive behaviors to take place.
Although, the MET p.T992I genetic mutation has been commonly found in somatic colorectal cancer tissues, this is the first report also implicating this MET genetic mutation as a germline inherited risk factor for familial colorectal cancer. A strength of this study is the use of colorectal cancers enriched for a hereditary component. One of the limitations, however, is the small sample size and lack of a large unaffected cohort. Future independent studies on large case and carefully selected control sets are needed to replicate these results, confirm the conclusions, and provide an accurate estimate of the prevalence of this mutation in the cancer and normal populations.
We estimate that the specific germline mutation of this investigation is responsible for ~4.5% of CRCs that occur in a familial setting, defined here as two first-degree relatives with CRC. Since 10% of all CRCs occur within this definition, this would translate to 0.45% of all CRCs. Future work should be focused on measuring the precise prevalence of this mutation in a large set of cases and controls and estimation of CRC risk associated with the germline mutation. Because MET p.T992I has also been found in germline DNA of individuals diagnosed with other cancers, a large-scale study to examine the penetrance and risks of all cancers in mutation carriers will be an important advance in our understanding. Specific inhibitors of the MET protein are currently in use in human clinical trials and may have a specific utility in preventing invasion and the metastasis of early-stage cancers in individuals with the MET p.T992I mutation [8].
The authors declare that they have no competing interests.
DN conceived of the study, its design, implementation, obtained funding and drafted the manuscript; MD designed and carried gene scanning and sequencing and assisted with manuscript; NS assisted with gene analysis, designed the TaqMan assays and analysis of tumors, and assisted with the manuscript; AS, HA, CG, DA, JS, GT, LS, AM, JS contributed samples and assisted with revision of the manuscript; RB participated in study design, obtained funding, and helped to draft the manuscript. All authors read and have approved the final manuscript.
The pre-publication history for this paper can be accessed here:
This project has been funded with federal funds from the National Institutes of Health, National Cancer Institute under contract RO3-CA150067, RO1-CA040641, PO1-CA073992 and sample collection was supported by Detroit Cancer Registry SEER program grant number N01-PC35145, and the Cancer Genetics Network, RFA CA-97-004, RFA-CA-97-019, and RFP N01-PC-55049-40; grants numbers U24-CA78134, U24- CA78156, U24-CA78148, U24-CA78174, U24- CA78157, U24-CA78142. Additional support was provided by a Cancer Center Support Grant P30-CA42014, Utah Cancer Registry grant HHSN 261201000026C from the National Cancer Institute's SEER program with additional support from the Utah State Department of Health, and Utah Population Database supported by the Huntsman Cancer Foundation.
This project could not have been done without the continued support from the Cancer Genetics Network who shared DNA samples and data. We acknowledge University of Utah Sequencing and Genotyping Cores. We thank Mark Yandell, Barry Moore and Hao Hu for assisting with 1000 genomes pilot data, Therese Tuohy for critical review of the manuscript, and James P. Evans, Jan T. Lowery, and Al Ziogas for their participation in CGN sample collection.
PubMed Central Canada is a service of the Canadian Institutes of Health Research (CIHR) working in partnership with the National Research Council's national science library in cooperation with the National Center for Biotechnology Information at the U.S. National Library of Medicine(NCBI/NLM). It includes content provided to the PubMed Central International archive by participating publishers.


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Mark Rivera, Customer Retention Manager and CJIS Security Compliance Officer with Vigilant Solutions, served for sixteen years with the Maryland State Police, retiring at the rank of First Sergeant with thirteen of those years at the supervisory and command level. He holds a Master of Science Degree in Management from The Johns Hopkins University and Secret clearance through the FBI, Baltimore.
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While all components of crime scene investigation are important, visual documentation stands out as the most effective tool for describing and recreating a crime scene.
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A wide variety of new or refined police products was the highlight of the TREXPO East trade show and conference held Aug. 12-15 at the Baltimore Convention Center.
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To anyone casually tuning into American TV or perusing a major U.S. newspaper, it looked like the country had been beset by an epidemic of kidnapped children.
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Cops have to deal with all sorts of silly fights people people get into, and some are dang near as deadly as shoot-outs. Just imagine how the cops in Calgary, Alberta, Canada, felt when they got called to a riot involving "softballers and croquet players."
By Al Valdez
Although other types of racism certainly exist, we will focus on this pervasive form of bigotry often called 'white supremacy.'
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Emotionally disturbed persons, or EDPs, present a major challenge to the law enforcement officer sent to deal with their sometimes bizarre and frequently unpredictable behavior.
Police officers swept up in a high-profile case involving accusations of racism and excessive force probably sympathize with those poor swimming witches. Except their trial is not by water, it's by media.
No department would dare let said officer hit the street without training and instruction in policies governing the use of a pistol. Many PDs even have policies regarding the use of flashlights. But in most departments an edged weapon/cutting tool clipped to the pocket of the officer's duty pants flies under the radar.
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At Columbine, the officers, deputies, firefighters, and emergency medical personnel did an incredible job in their response to an unbelievably bad situation. But through no fault of their own, the tactics they were trained to use were not suited to the nature of the incident.


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December 05, 2013
Love's Travel Stops opened a new location in Dayton, Ohio, off I-75, expanding its presence in the Buckeye State.
The new Love's is open 24 hours a day and features 98 truck parking spaces, a Hardee's restaurant, coffee, name-brand snacks, fresh fruit, gift merchandise and various other things.
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A Love's Truck Tire Care center will open there later this month.
The travel center is located at 2217 S. Edwin C. Moses Blvd.
Love's now operates eight travel stops in Ohio and has more than 300 locations and 180 Love's Truck Tire Care centers in 39 states,
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The expert, Brian Fletcher from Eaton, will answer your questions
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Kenworth Launches Essentials App for Mobile Devices
EZ Drain Valve Made For Vaporizer Drain Systems
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Enter your stay dates and any discounts you qualify for to see Shoreham-by-Sea, United Kingdom rates found only on HotelPlanner!
Find the perfect hotel in Shoreham-by-Sea using our hotel guide provided below. Find cheap and discounted hotel/motel rates in or nearby Shoreham-by-Sea, United Kingdom for your personal leisure or group travel. We list the best Shoreham-by-Sea hotel rooms. Review the Shoreham-by-Sea hotel list below to find the perfect lodging. We also have a map of the Shoreham-by-Sea hotels.
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"Our clients are true artist, being able to learn from our fabulous instructors and grow with in themselves is an honor. It is also an incredible honor to experience that growth with their families and care providers."
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AFL Cats overpower Adelaide in second half
Geelong have snapped Adelaide's eight-game winning streak with an impressive 30-point victory at Simonds Stadium.
Geelong have snapped Adelaide's eight-game winning streak with an impressive 30-point victory at Simonds Stadium.
Melbourne have taken the NRL ladder lead with a 26-10 win over the Sydney Roosters although Cronulla can reclaim top spot with victory over Newcastle.
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Sydney have held off a spirited Carlton side to score a six-point AFL win at the SCG.
The Hurricanes have stormed to a 41-0 Super Rugby quarter-final win over the Sharks, but a serious rib injury to skipper Dane Coles has muted their joy.
Canberra have leapfrogged Canterbury into the NRL's top four with a 26-22 golden-point win over the Warriors.
Robbie Farah has ridiculed Jason Taylor's reasons for dropping him to reserve grade, but has conceded his NRL career could be over.
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Sports scientist Stephen Dank, who suffered minor injuries when his house was shot at, says he has his suspicions who would do such a thing.


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George H. Bon Salle
Position: Power Forward ▪ Shoots: Right
Height: 6-8 ▪ Weight: 220 lbs.
Born: July 1, 1935 in Chicago, Illinois
Died: July 20, 2015
High School: Loyola Academy in Wilmette, Illinois
College: University of Illinois at Urbana-Champaign
Draft: Syracuse Nationals, 1st round (7th pick, 7th overall), 1957 NBA Draft
NBA Debut: October 28, 1961
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April 17, 1957: Drafted by the Syracuse Nationals in the 1st round (7th pick) of the 1957 NBA Draft.
June 22, 1961: Signed as a free agent with the Chicago Packers.
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United States Army Division, 82nd
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- The All-Americans at war : the 82nd Division in the Great War, 1917-1918 by James J Cooke( Book )
- 82d Division, summary of operations in the World War by American Battle Monuments Commission( Book )
- Official history of the 82nd Division, American Expeditionary Forces : "All American" Division by United States( Book )
- On time, on target : the World War II memoir of a paratrooper in the 82nd Airborne by John D McKenzie( Book )
- On to Berlin : battles of an airborne commander, 1943-1946 by James M Gavin( Book )
- US 82nd Airborne Division : 'all American' by Mike Verier( Book )
- Night drop : the American airborne invasion of Normandy by S. L. A Marshall( Book )
- 82d Airborne Division : historical summary, 1917-present( Book )
- Night drop; the American airborne invasion of Normandy. With a pref. by Carl Sandburg. Illustrated by H. Garver Miller by S. L. A Marshall( Book )
- 82nd "All American" Division A.E.F. : 157th F.A. Brigade( Book )
- 82d Division AEF : WWI battle citation General orders, 1918-1919( Book )
Most widely held works by United States
Official history of the 82nd Division, American Expeditionary Forces : "All American" Division by United States( Book )
6 editions published between 1919 and 2006 in English and held by 39 WorldCat member libraries worldwide
6 editions published between 1919 and 2006 in English and held by 39 WorldCat member libraries worldwide
Audience Level
Related Identities
- Cooke, James J. Author
- American Battle Monuments Commission
- McKenzie, John D.
- Gavin, James M. (James Maurice) 1907-1990 Author
- Marshall, S. L. A. Author
- United States Army Airborne Division, 101st
- Verier, Mike Author
- United States Army Airborne Division, 82nd
- 82d Airborne Division War Memorial Museum
- United States Army Field Artillery Regiment, 321st
Associated Subjects
Armed Forces--Awards Europe France--Normandy McKenzie, John D Military campaigns Military operations, Aerial--American Regimental histories Soldiers United States United States.--Army United States.--Army.--Airborne Division, 101st United States.--Army.--Airborne Division, 82nd United States.--Army.--Division, 82nd United States.--Army.--Parachute Field Artillery Battalion, 456th United States.--Army.--Ranger Battalion, 4th Western Front (World War (1914-1918)) Western Front (World War (1939-1945)) World War World War (1939-1945)
Alternative Names
United States. Army. 82d Division
United States. Army. American Expeditionary Forces. Division, 82nd
English (26)


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News Archive
Home Invasion and Assault
Two men were arrested Wednesday by the St. Cloud police after allegedly breaking into a woman's home in the middle of the night and holding her and her infant child at gunpoint. The report came in at 6:45am from a residence in the 700 block of 18th Avenue South. The 27 year old victim told police she woke up and found two masked men with handguns at the foot of her bed.
She was assaulted, bound with duct taped and threatened several times with a handgun. Her 9 month old child was lying on the bed next to her and also threatened. The assailants ransacked the home for two hours trying to apparently find property of value. The stole cash and other items then fled.
The St. Cloud Metro Gang Strike Force and police investigated and arrested 26 year old Eric Grant Andrews of St. Cloud Wednesday night. The Brooklyn Park police department arrested the second suspect, 29 year old Corey Dell Andrews of St. Joseph Thursday morning. Both men are facing several charges including first degree burglary, aggravated robbery, assault and false imprisonment.
Latest News
Detours Will Be Posted Along The Lake Wobegon Trail Early Next Week Near Melrose
Motorcyclist Hospitalized In Serious Condition In St. Cloud After Crash Saturday
K-9 Retires From Benton County Sheriff's Office Wednesday After ACL Injury
St. Cloud Area Schools Receive Grant Support From MnDOT For Bicycle Safety Education
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Save Big On Open-Box & Pre-owned: Buy "Just Dance 4 - Nintendo Wii" from Amazon Warehouse Deals and save 50% off the $19.99 list price. Product is eligible for Amazon's 30-day returns policy and Prime or FREE Shipping. See all Open-Box & Pre-owned offers from Amazon Warehouse Deals.
Just Dance 4 - Nintendo Wii
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About the Product
- Top Of The Charts - Just Dance 4 has over 40 all-new songs, ranging from current Billboard hits, legendary favorites, and classic dance party tracks!
- Just Dance-Off! - Challenge your friends in 6 rounds of dance battles with the all-new battle mode!
- Bring Your Crew - Each player can get his own moment of fame with unique choreographies and moves for each member of your dance crew.
- Just Sweat It Out - The popular Just Sweat mode gets a major upgrade with brand new workout sessions, personalized programs, live feedbacks and a real calorie counter.
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Product Description
Product Description
From the Manufacturer
Throw the ultimate party with Just Dance 4!
The latest edition of the record-breaking franchise takes the party to a whole new level of fun, with over 40 all-new chart-topping hits, the coolest dances and unique features for every platform.
Just Dance 4 is your ticket to the best new music with over 40 tracks from a variety of top artists, popular genres and hot billboard songs like "Good Feeling" by Flo Rida, and "Moves like Jagger" by Maroon 5 ft. Christina Aguilera. The tracklist also features legendary artists such as Barry White and The B-52's to get the whole family up and moving.
Just Dance 4 is the perfect catalyst for the best parties, no matter what the occasion, thanks to innovative new modes like the all-new Battle mode, an upgraded Just Sweat mode, and even more fun, exclusive modes for each platform. Everything you know and love about Just Dance gets even better with Just Dance 4—hot new tracks, fun irreverent dances, and more. With Just Dance 4, the party never ends! Have a blast dancing, laughing, and breaking a sweat with friends and family all night long.
Track listing
- 2 Unlimited "Tribal Dance"
- A.K.A – "Hot For Me"
- Anja – "Crazy Little Thing"
- Rick Astley – "Never Gonna Give You Up"
- Army of Lovers – "Crucified"
- The B-52's – "Rock Lobster"
- Justin Bieber feat. Nicki Minaj – "Beauty and a Beat"
- The Blackout Allstars – "I Like It"
- Boys Town Gang – "Can't Take My Eyes Off You"
- Bunny Beatz – "Make The Party (Don't Stop)"
- Blu Cantrell – "Hit 'Em Up Style (Oops!)"
- Dancing Bros. – "Everybody Needs Somebody To Love"
- Kat DeLuna ft. Busta Rhymes – "Run The Show"
- Europe – "The Final Countdown"
- Flo Rida – "Good Feeling"
- Nelly Furtado – "Maneater"
- The Girly Team – "Oops!... I Did It Again"
- Selena Gomez and the Scene – "Love You Like A Love Song"
- Halloween Thrills – "Time Warp"
- Hit The Electro Beat – "We No Speak Americano"
- Carly Rae Jepsen – "Call Me Maybe"
- Las Ketchup – "Asereje (The Ketchup Song)"
- Jennifer Lopez ft. Pitbull – "On The Floor"
- Marina and The Diamonds – "Oh No!"
- Maroon 5 ft. Christina Aguilera – "Moves Like Jagger"
- Ricky Martin – "Livin' La Vida Loca"
- Bill Medley & Jennifer Warnes – "(I've Had) The Time of My Life"
- Sergio Mendes ft. The Black Eyed Peas – "Mas Que Nada"
- Nicki Minaj – "Super Bass"
- One Direction – "What Makes You Beautiful"
- Panjabi MC – "Beware Of The Boys (Mundian To Bach Ke)"
- P!nk – "So What"
- Elvis Presley – "Jailhouse Rock"
- Rihanna – "Disturbia"
- Rihanna ft. Jay-Z – "Umbrella"
- Sammy - "Some Catchin' Up To Do"
- Skrillex – "Rock N'Roll (Will Take You To The Mountain)"
- Will Smith – "Wild Wild West"
- Alexandra Stan – "Mr. Saxobeat"
- They Might Be Giants – "Istanbul"
- Carrie Underwood – "Good Girl"
- Barry White – "You're The First, The Last, My Everything"
- Stevie Wonder – "Superstition"
Key Game Features
- Top Of The Charts - Just Dance 4 has over 40 all-new songs, ranging from current Billboard hits, legendary favorites, and classic dance party tracks! With a wide variety of songs, artists and genres such as "Good Feeling" by Flo Rida, Nicki Minaj's "Super Bass", "Disturbia" by Rihanna, and Pink's "So What," everyone is sure to find their groove. Want more tracks? Download from an expanding catalogue of new songs from the in-game store.
- New Battle Mode - Challenge your friends in 6 rounds of dance battles with the all-new battle mode! Go head to head to unique choreographies and music to be crowned the dance battle champion!
- Bring Your Crew - With songs that feature 4 concurrent dance routines, it's your time to shine! Each player can get his own moment of fame with unique choreographies and moves for each member of your dance crew.
- Just Sweat It Out - The popular Just Sweat mode gets a major upgrade with brand new workout sessions, personalized programs, live feedbacks and a real calorie counter. Stay fit, and have fun playing Just Dance!
- Alternative Choreographies - Unlock alternative choreographies for your favorite tracks including ultra-challenging and outlandish routines.
- Dance Quests - Discover and win up to 6 challenges per song and unlock more rewards.
- Dancer Card - Create and personalize your own dancer card to check out all your awesome dance stats (e.g., favorite tracks, best scores, and challenges).
- Just Can't Get Enough - Just Dance 4 still has everyone's favorite modes like Duet Mode, Mash-up Mode with 45+ mash-ups, and more!
Kinect for Xbox 360 Exclusive Features
- Immortalize your best moments with Autodance - Record your friends dancing and Autodance will sync their movements into an instant dance routine, creating fun souvenir videos!
- Just Dance TV - Watch and share souvenir pictures and videos of your best Just Dance parties with your friends! Also check out the funniest videos from the worldwide Just Dance community!
Customer Reviews
Top Customer Reviews
Today I had 5 classes play Just Dance 4 for the first time. We played the "Just Sweat" mode using the 25 minute option using the cardio-boxing style workout.
It was TERRIFIC!
It started with a 2 minute warm-up to a gentleman that sounded like the karate instructor from Exerbeat. My students were all engaged with the various movements.
After the warm-up, the game would randomly pick a song. They were all fantastic but the student's faves were Skrillex (off the charts choreography), Mr. Saxobeat, Good Feeling, Moves Like Jagger and the J-Lo song.
After each song, another 2 minute program with cardio boxing, kickboxing and other aerobic style movements were done, all engaging. The actual program usually took 28 minutes with around 5 songs interspersed between the workouts. It even ended with a 2 minute cool down with good stretches. Ubisoft could make a separate game just using the workout segments, it is that good.
Between the 5 classes, we made it to Level 9 and unlocked various goodies such as a song (Umbrella), new avatars, game modes, 60's just sweat workout, etc.
I wish I could post a short video I took of my 38 sixth graders singing loudly and dancing to "Good Feeling", it was awesome!
A couple of classes also performed the quartet of "Instanbul" and many already loved it because our social studies teacher plays that song when they study ancient civilizations. To cool......and a great follow up to last years fav, "Night Boat to Cairo"
Great job Ubisoft!!!!!!!!
Small nitpick, I wish I could make a custom playlist for the just sweat workouts.
Customer Images
Most Recent Customer Reviews
I got this game specifically for one song but the whole song selection is great!Published 11 days ago by Amazon Customer


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1) Project Details: (be as specific as possible):
We organise flea markets and we like our vendors to be able to book and pay the tables online. If you have an existing project that can be recylced, that is also ok.
2) Payment method/ details (Paypal, check? Timeline?):
Paypal, how fast can it be?


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Please explain the following line from the "top" command in the bold :
Mem: 1026228k total, 449640k used, 576588k free, 44812k buffers
Swap: 1052248k total, 2020k used, 1050228k free, 189092k cached
Thanks Arpit
Please explain the following line from the "top" command in the bold :
Thanks Arpit
migrated from stackoverflow.com Jan 18 '10 at 14:41
This question came from our site for professional and enthusiast programmers.
The "buffers" entry indicates how much of the memory in use is currently being used for disk buffering.
Swap Cache
So long as these pages are not modified after they have been written to the swap file then the next time the page is swapped out there is no need to write it to the swap file as the page is already in the swap file. Instead the page can simply be discarded. In a heavily swapping system this saves many unnecessary and costly disk operations.
Linux automatically caches files read from the disk. That cache is called buffers. The buffers will be released, if your program wants more memory and no more is free.
I don't know what the cached swap means.
EDIT: I googled shortly for the swap and saw the following: http://www.linux-tutorial.info/modules.php?name=MContent&pageid=314
Seems that the cached swap means all the pages in the swap, that are reloaded into memory but unchanged since. These pages in the swap are already valid and if the page is swapped out later again Linux doesn't need to write them actually to the disk.


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Decorate your table with shimmering style that will bloom with a little bit of candlelight. Two crystalline flowers are ready for the candles of your choice, and the gorgeous iron base features a circular stand and detailed leaves along with crystalline flower buds. Candles not included.
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Sparkle, shine and glow! This glamorous candle holder features faceted clear crystals that capture and enhance candlelight. Your tabletop will dance with candlelight when you place the candle of your choice inside this stunning candleholder. Rows of faceted glass gems refract the light from inside, and the silver tone metal framework enhances the rich shine.
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Objective and Practice Questions for Economics
Objective type & multiple choice questions on Economics for interview and Competitive Exams preparation.
- Economics Awareness Solved Questions (Download PDF)
- Economics Mock Test 1 (Download PDF)
- Economics Mock Test 2 (Download PDF)
- Economics Mock Test 3 (Download PDF)
- Economics Quantitative Questions (Download PDF)
- Economics Sample MCQ Part 1 (Download PDF)
Examrace- Latest Exam Updates & Current-Affairs
Union Cabinet Increases Limit for Foreign Investment in Stock Exchanges from 5% to 15% [ Current Affairs ](on )
Union Cabinet has given its approval for raising foreign shareholding limit from current 5 % to 15 % in Indian Stock Exchanges. Decision carries the investment limit of foreign units at par with that of domestic organization.
CBSE NET Psychology Paper 3 10th July 2016 Solved Paper3 Solutions at Youtube Examrace Que.51 to 75 [ CBSE NET-JRF Updates ](on )
CBSE NET July 2016 psychology paper-3 detailed solutions will be made available at Youtube Examrace channel and for full Examrace CBSE (UGC) NET-JRF Psychology Series visit www.examrace.com. For complete detailed solutions for July 2016 paper you can refer www.doorsteptutor.com.
51)… (1920 more words) …
IBPS CWE PO/ MT – VI 2016 - Prelims in Oct and Mains in Nov [ Bank-PO Updates ](on )
Institute of Banking Personnel Selection (IBPS) going to be conduct the online examination for the next Common Recruitment Process for selection of personnel for Probationary Officer/Management Trainee (CWE PO/MT-VI) vacancies in the Participating Organizations. Interested candidates may register online from… (745 more words) …
NEET Phase 2 July 2016 Solved Paper Part 3 - Question 121 to 180 [ NEET Updates ](on )
Solutions for NEET Phase 2 July 2016 - Questions 121 to 180. Detailed solutions to these questions are available at - www.doorsteptutor.com.. Subscribe to Youtube Examrace Channel to view our evergrowing collection of NEET videos at www.youtube.com Also dont… (3698 more words) …
Less than a month to Go! SSC CGL exam to be conducted on 27th August 2016 [ SSC CGL Updates ](on )
The Staff Selection Commission (SSC) is a GOVT OF INDIA organization holds Combined Graduate Level Examination, for recruitment to different posts for which minimum requirement is Graduation from a recognized University. The Examination held in two Tiers written Objective Type… (542 more words) …
KVPY Will Be Held ONLINE on 6th November 2016 [ KVPY Updates ](on )
Kishore Vaigyanik Protsahan Yojana (KVPY) is National level Fellowship program started by the Department of Science and Technology (DST) in 1999 with aim to inspire those Candidates who are studying in Basic Science for getting interest in research career in… (943 more words) …


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The proposed budget cuts that would shutter Data.gov sparked a debate about what would be lost
The latest budget battles are putting an existential squeeze on federal websites such as Data.gov and USAspending.gov, which has drawn supporters and critics into a debate about the goals, value and effectiveness of sites intended to promote government transparency.
In the face of a proposed $27 million reduction in the E-Government Fund that pays for the sites, Rep. Darrell Issa (R-Calif.), chairman of the House Oversight and Government Reform Committee, made a personal pledge to keep them operating.
"The specific funding goes away, but reprogramming authority would still be available," Issa said during a panel discussion hosted by the Association of Government Accountants, as reported by Federal News Radio. "Our view is, on a case-by-case basis, we will be able to keep them open."
Issa is hardly a fan of every Obama-era Web initiative. In August 2010, he accused the administration of using federal websites such as Recovery.gov as propaganda to advance the president's political agenda.
But Issa's across-the-aisle support for Data.gov and USAspending.gov drew praise from John Wonderlich, policy director at open-government advocacy group Sunlight Foundation. Wonderlich wrote on the organization's blog that few lawmakers have been willing to support the sites during the recent budget fights.
"If we entirely defund USAspending or Data.gov, we wouldn't be erasing an 'Obama initiative' as much as we'd be removing the only portal to federal grants and contracts or shutting down the clearinghouse for federal bulk data access," Wonderlich writes. "Transparency reform often functions through political incentives, but it isn't a political phenomenon. It's a democratic phenomenon."
Laudable intentions and lofty language aside, others have questioned whether the federal transparency sites are accomplishing much.
A website called Nerd Collider hosted a discussion about Data.gov earlier this month and kicked it off by pointing out that the number of unique visitors to the site has plateaued at about 10,000 per month.
Kate Ray, Nerd Collider's co-founder, summarized the results of the online discussion by highlighting questions raised about who the site's users are, why it's so hard to use and how the site's supporters measure success. "It's unclear whether Data.gov is aimed at journalists, academic researchers, entrepreneurs or special-interest groups," Ray writes.
James Hendler, a professor and an artificial intelligence researcher at Rensselaer Polytechnic Institute and an adviser to the Data.gov team, defended the site during the Nerd Collider discussion. However, he said there is room for improvement. He said tracking the number of unique visitors is misleading. The more important measurement is how often data is downloaded from the site, and that occurred 100,000 times in February, the most recent month for which figures were available.
The real value of Data.gov is what those 10,000 visitors per month don't see, and that could be lost if the sites go dark, writes Harlan Yu on the "Freedom to Tinker" blog, hosted by Princeton University's Center for Information Technology Policy.
Data.gov "plays a crucial behind-the-scenes role, setting standards for open data and helping individual departments and agencies live up to those standards," Yu writes. He said the loss of the program would negatively affect the quantity and quality of government data posted in the future.
About the Author
John Zyskowski is a senior editor of Federal Computer Week. Follow him on Twitter: @ZyskowskiWriter.
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The latest version of the Defense Department-wide Enterprise Service Management Framework places more emphasis on managing IT risk.
Despite GSA's efforts to accelerate the FedRAMP approval process, the lack of agency reciprocity puts the program's central goals at risk.
Got some early-career colleagues who are doing great things in federal IT? Nominate them for FCW's 2016 Rising Star awards.
The agency software inventories required under the draft category management policy will be treasure maps that lead to efficient, effective, streamlined buying and unprecedented savings.
As experts warned of the "dire" threats posed by outdated federal technology, lawmakers grilled top feds, debated workforce issues and inched closer to backing a $3.1 billion fix-it fund.
The agency is making strides in developing and deploying border technologies but still has some weaknesses.
Agencies have spent almost $23 billion on legacy IT over the past three years, according to reports to Congress. Is a revolving fund the answer?
The United Kingdom will go live with its governmentwide digital identity platform, GOV.UK Verify, in the coming days. The U.S. government will need a little more time.
What contracting officer representatives really think is needed to fix contract management.
The Census Bureaus Annual Statistical Abstract has been around since 1878 and contains the high-value, high-quality data we really want - see http://en.wikipedia.org/wiki/Statistical_Abstract_of_the_United_States - for data science.
It contains about 1300 tables with their metadata ready to be used in statistical analyses, mashups, and visualizations. See http://www.census.gov/compendia/statab/
I suggest you ask the Census Bureau how many books, CDs and data downloads they have received to see what the taxpayer is getting for funding that all of these years.
I make their data available at http://semanticommunity.net/StatAbs2011/in support of the Open Government Data Initiative and have produced data science products to preserve and apply these data at http://semanticommunity.info/#Data_Science_Products.
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text: ProtectingOhio Since- 1884 - Reporting Gabapentin Products to OARRS – Effective 12-1-2016Pharmacy Intern Authorization to Test InformationClick Here to Begin Your Ohio License RenewalOhio Pharmacies Dispensing Naloxone without a Prescription Important Notices Welcome to The State ofOhio Board of Pharmacy Thank you for visiting our website. The State of Ohio Board of Pharmacy is the single state agency in Ohio responsible for administering and enforcing laws governing the practice of pharmacy and the legal distribution of drugs. Here you'll find information and resources to assist healthcare professionals, law enforcement and the public in understanding Ohio's drug laws. -- Steven W. Schierholt, Esq., Executive Director Do You Know the Red Flags of prescription drug diversion? > Watch video Click here to subscribe to updates! Click here for meeting dates! Recent Updates E-News Update (July 2016)Notice of Public Hearing - 08.23.20164729-16 - BIA - Comments Due 8.5.20164729-5, 7 & 9 - BIA - Comments Due 8.5.2016Compounding in OhioCompounding in OhioEmergency Medical Services - Frequently Asked QuestionsTerminal Distributor's License for Prescribers Compounding Dangerous Drugs (Updated 7.18.2016)2016 Roundtables201606 - Minutes (Jun 2016)Short-term Weight Loss Anorexiants FAQReporting Gabapentin Products to OARRS – Effective 12-1-2016Reporting Gabapentin Products to OARRS – Effective 12-1-20162016 Continuing Education Law PresentationsZika E-UpdateAgenda (Jul 2016)New and Updated Rules – Q1 and Q2 of 2016 Follow @ohiorxboard Licensing Information: Pharmacists Pharmacy Technicians Pharmacy Interns Terminal Distributor Wholesale Distributor Veterans and Military Families


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Redshirt freshman guard Brandone Francis Ramirez is transferring from the Gator basketball program. Francis Ramirez announced his decision on Twitter Wednesday afternoon. Although he'll play elsewhere in the future, he plans to stay in Gainesville to complete a summer class before moving on. I thought about it long and hard …Read More »
A Full-Court Press On Gator Women's Basketball Head Coach Amanda Butler
GAINESVILLE, Fla—The half-time buzzer sounds off. She looks up at the scoreboard one more time, walks towards the tunnel as she high-fives Gator fans of all ages who are eagerly leaning over the rails screaming, "Go Gators" and then she's headed to coach up her team for second-half play. This …Read More »
Hornet's Starting Lineup Adjustment Baffles Miami, Even Series
Going forward with a commanding 2-0 series lead is a very easy way to get over confident. And as the team who is down considerably, desperation starts to ring, and quick, drastic changes can be made. Some work out, others don't. Luckily for the Charlotte Hornets, their desperation has worked …Read More »
UConn Defeats Syracuse 82-51, Continues to Make History
Few collegiate athletes are lucky enough to call themselves champions at least once in their collegiate career. Fewer are lucky enough to call themselves champions twice during their collegiate careers. Even less can call themselves champions four times in their college careers. Then there are UConn women's basketball players Breanna Stewart, …Read More »
2016 Naismith Basketball Hall of Fame Inductees Announced
Shaquille O'Neal, Allen Iverson, and Yao Ming were among the names inducted into the Naismith Basketball Hall of Fame on Monday. The most notable 2016 Hall of Fame inductees perhaps are O'Neal and Iverson. O'Neal spent 19 years in the NBA, playing for six different teams. One of the most commanding centers in NBA history, O'Neal …Read More »
NCAA Women's Basketball Championship Preview
Top overall seed Connecticut and No. 4 seed Syracuse will battle for the national title in Indianapolis tonight at 8:30 p.m. Holding a perfect record of 37-0 the UConn girls will compete for an NCAA-record 11th national championship (4th straight). Syracuse (30-7) clawed their way to the national championship game by beating the Washington …Read More »
Gators' Robinson Declares for NBA Draft
Florida Gators sophomore forward Devin Robinson is declaring for the NBA Draft, according to a post on his Instagram from Monday. However, this isn't necessarily the end of Robinson's time in the orange and blue. New NCAA rules allow players to declare for the draft and participate in the NBA …Read More »
Kris Jenkins' Buzzer-Beater Lifted Villanova Over North Carolina In National Championship Game
On Monday night, Villanova stunned North Carolina and the Wildcats earned themselves its first national title since 1985. With 4.7 seconds left in the National Championship game, the North Carolina Tar Heels and the Villanova Wildcats were all knotted up at 74 points apiece. But in a blink of an eye, confetti showered …Read More »
NCAA Women's Championship Preview: Connecticut Continues Historic Run Against Syracuse
With the end of March it means the end of the madness…almost. The NCAA Women's Tournament comes to an end Tuesday night as powerhouse UCONN takes on underdog Syracuse. Both teams come into this championship game after dominating in every game thus far; the lowest margin of victory for either …Read More »
National Championship Preview: North Carolina to Face Villanova
The National Championship game is set. No. 1 seed North Carolina will take on No. 2 seed Villanova. The Tar Heels will be playing in their 10th national championship and first since winning it in 2009 while the Wildcats will be playing in their third national championship and first since winning it in …Read More »


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A Love Story In 22 Pictures
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- The Democratic National Committee on Monday apologized for remarks sent over email criticizing the Bernie Sanders campaign 🇺🇸
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Recent recon of two radiators and fluid refill has left the heater not blowing hot air through, with dials on max-red, till the car goes above 25000 revs, then becomes devent. The fan is working and the temp guage reaches temp 1/2 way on dial.
What would be your opinion please.
Tuesday, January 29th, 2013 AT 9:08 PM


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Portland Pain Management Physician Doctors for Chronic Pain
Type of Physician: Pain Management Physician
What is a Pain Management Physician? An interdisciplinary subspecialty certification by the Boards of Anesthesiology, Psychiatry & Neurology, and Physical Medicine & Rehabilitation; practitioners provide a high level of care either as a primary physician or consultant for patients experiencing problems with acute or chronic pain in both inpatient and outpatient settings, and coordinate a multidisciplinary approach toward pain management.
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Portland, Oregon
Portland is a city located in the Northwestern United States, near the confluence of the Willamette and Columbia rivers in the state of Oregon. It has been referred to as the most environmentally friendly or "green" city in the United States, and the 2nd most in the world. The city and region are noted for strong land-use planning and investment in light rail, supported by Metro, a distinctive regional-government. Portland is known for its large number of microbreweries and microdistilleries, and its coffee fanaticism.
(Source: http://en.wikipedia.org/wiki/Portland,_Oregon)
Chronic Pain
Chronic pain facts*
*Chronic pain facts by John P. Cunha, DO, FACOE
- Pain is an unpleasant sensory and emotional experience.
- Acute pain results from disease, inflammation, or injury to tissues and comes on sudde...
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Emergency Contact for Portland
- In case of Emergency, call 911
- Police (503) 823-3333
- Fire & Rescue (503) 823-3700
Nearby Portland Hospitals results
- 1Shriners Hospital For Children
3101 SW Sam Jackson Park Rd
Portland, Oregon 97239
(503) 241-509
- 2Doernbecher Children's Hospital
3181 SW Sam Jackson Park Rd
Portland, Oregon 97239
(503) 494-831
- 3Oregon Health Science Univ Hosp
3181 SW Sam Jackson Park Rd
Portland, Oregon 97239
(503) 494-831
- 4VA Medical Center-Portland
3710 SW US Veterans Hospital Rd
Portland, Oregon 97239
(503) 220-826
- 5Oregon State Hospital-Portland
1121 NE 2nd Ave
Portland, Oregon 97232
(503) 945-280
- 6Legacy Good Samaritan Hospital
1015 NW 22nd Ave
Portland, Oregon 97210
(503) 413-771
- 7Rehab Institute Of Oregon
1015 NW 22nd Ave Fl 6
Portland, Oregon 97210
(503) 413-771
- 8Children's Hosp Legacy Emanuel
2801 N Gantenbein Ave
Portland, Oregon 97227
(503) 413-250
- 9Emanuel Medical Center
2801 N Gantenbein Ave
Portland, Oregon 97227
(503) 413-220
- 10Providence Portland Medical Ctr
4805 NE Glisan St
Portland, Oregon 97213
(503) 215-111
- 11Providence St Vincent Med Ctr
9205 SW Barnes Rd
Portland, Oregon 97225
(503) 216-123
- 12Providence Milwaukie Hospital
10150 SE 32nd Ave
Portland, Oregon 97222
(503) 513-830
- 13Adventist Medical Center
10123 SE Market St
Portland, Oregon 97216
(503) 257-250
- 14Kaiser Sunnyside Medical Center
10180 SE Sunnyside Rd
Portland, Oregon 97015
(503) 652-288
- 15Portland VA Med Ctr-Vancouver
1601 E Fourth Plain Blvd
Portland, Oregon 98661
(360) 696-406
- 16Peacehealth Southwest Med Centr
400 NE Mother Joseph Pl
Portland, Oregon 98664
(360) 514-200
- 17Memorial Health Center
3400 Main St
Portland, Oregon 98663
(360) 696-504
- 18Legacy Meridian Park Hospital
19300 SW 65th Ave
Portland, Oregon 97062
(503) 692-121
- 19Willamette Falls Medical Center
1500 Division St
Portland, Oregon 97045
(503) 656-163
- 20Legacy Mount Hood Medical Ctr
24800 SE Stark St
Portland, Oregon 97030
(503) 667-112
- 21Legacy Salmon Creek Medical Ctr
2211 NE 139th St
Portland, Oregon 98686
(360) 487-100
- 22Tuality Community Hospital
335 SE 8th Ave
Portland, Oregon 97123
(503) 681-111
- 23Providence Newberg Med Ctr
1001 Providence Dr
Portland, Oregon 97132
(503) 537-155
- 24Tuality Forest Grove Hospital
1809 Maple St
Portland, Oregon 97116
(503) 357-217
- 25Willamette Valley Med Ctr
2700 SE Stratus Ave Unit 405
Portland, Oregon 97128
(503) 472-613
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"Your Nation is about to Sin" [Posted 11/4/12] Says The Lord Jesus Christ "Too Many Liberals" [Posted 11/1/12] FULFILLED
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It is one interpretation. However, if you examine the prophecies and the identity of the "king of the north", obama fits perfectly. America dictates global policy and extends as far north as Alaska. It is the most powerful nation in the northern hemisphere (North America). Daniel 11:37 and Daniel 11:43 are prime examples.
What does it mean? It means that there will soon be great tribulation like never before. If you are not prepared, you will indeed face the "man of lawlessness" and you will be forced to accept a new monetary system requiring you to have an implantable device inserted into your right hand or forehead. Interestingly, 666 is also representative for the bismillah, or the islamic proclamation "there is no god but allah". If you do not agree.. [
link to www.youtube.com
] Those that have offered their support and admiration to the "man of sin" are going to face an angry God.
The "antichrist", or the "beast" will face Jesus Christ in the valley of Jehosophat (Israel). He will arrogantly try to stop Jesus, and will be destroyed along with the armies of the earth who are participating in this assault.
Read the scriptures. There is NO OTHER WAY.
Anonymous Coward 15537797
Directions in Bible prophecy are from Jerusalem, not just anywhere. If you make "north" mean anyplace in the norther hemisphere, you have to allow extreme latitude with every other detail of prophecy, and then it's whatever you want it to be. That's too sloppy for me. And N. America is not equated with the northern hemisphere; Russia at least fits some of the prophecies, and they are very much north.
As for preparation for the Tribulation, have you forgotten what Jesus said? It will be "a time such has never been, and will never be again". Christians have always suffered persecution, some of it going on today in the extreme. If anyone teaches that we are not going to suffer much in this life, they are the poorest teacher. And I would never teach such a thing, nor would any pre-trib Rapture teacher.
And even if I'm wrong about the timing of the Rapture, I would like very much to see how bravely and preparedly you go through it. Such bravado deserves an audience.
ADDED: I am SO VERY FED UP with people spreading these ridiculous straw men about us who believe the pre-trib Rapture! "Oh, you're not prepared! Be prepared like ME, I am so very PREPARED! You will be fooled into taking the mark of the Beast! Hahahahaha!"
Did I mention I'm fed up?
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Wally Hall is the managing sports editor for the Arkansas Democrat-Gazette. A graduate of the University of Arkansas-Little Rock after an honorable discharge from the U.S. Air Force, he is a past president and member of the Football Writers Association of America, member of the U.S. Basketball Writers Association, past president and current executive committee and board member of the Arkansas Sports Hall of Fame, and voter for the Heisman Trophy.
Malik, like Marcus, looks like a real deal
Wings Elite's Malik Monk looks for an opening pass during their game against RL9 at the Real Deal tournament in Little Rock, April 18, 2014.
People were circling around looking for a parking place.
P.A.R.K. was packed, and it was still an hour before the Wings Elite would play.
Pool play was finishing up in Real Deal in the Rock and that always creates interest, but most people were wanting to get a glimpse of Malik Monk, the player who is finishing up his sophomore year at Bentonville.
By all accounts he is the type of player who will be recruited by every college that wants a player who can make an impact as a true freshman.
Monk has the good fortune to have a big brother, Marcus Monk, who knows about recruiting and college. Marcus Monk is the Arkansas Razorbacks' all-time leader in touchdown receptions with 27, and he played most of his final season while recovering from a knee injury, which hurt him in the NFL Draft.
He also played some basketball for the Hogs, lettering in 2004-2005, and giving the team a big boost for eight games in 2008-2009, helping them win games against Oklahoma and Texas before surrendering his spot on the team after taking a loan from a lifelong friend to pay his rent.
If he had taken on the NCAA he probably would have won, but it was decided it would be easier on everyone if he just stepped aside, which he did without complaint.
Marcus Monk was always one of those guys who could have easily gone to college on an academic scholarship but loved sports. He is finishing work on his master's degree.
In his senior year at East Poinsett County High School in Lepanto, he was named Mr. Basketball by the Arkansas Democrat-Gazette, and two months later he was honored as the Arkansas Democrat-Gazette Male Athlete of the Year.
Now comes his little brother, a natural guard who has great quickness, vision and understanding of the game. He scores easily and gets assists even easier.
By Saturday morning's 11:30 tip off at P.A.R.K. in Little Rock, there was standing room only to watch Malik Monk and the Wings Elite complete pool play in the tournament.
He's not the only great player in the tournament: Several others will also be headliners in college and the NBA, but Monk is an Arkansan and everyone wants to seethe guy who can help turn the Razorbacks - if that's where he decides to go - into a national powerhouse again.
Bill Ingram, founder of Real Deal, was looking a little tired Saturday - technically it was the tournament's third day - and the light at the end of the tunnel was still a long way away.
As the tournament director, Ingram makes himself available to coaches, officials and anyone who might think they have a problem, which generally is about parking, but that's a good problem to have.
In the weight room area above the playing courts, which is restricted, of course, stood Dr. Jimmy Tucker and his son.
From their vantage point they could see all three games at once, and as die-hard basketball fans it was a great spot to have, but one they have earned.
When Ingram was first thinking about starting a tournament, one of the people at the top of his list to be a sponsor was Tucker, and the doctor came on board immediately and has continued to be one of the main cogs in the wheels of Real Deal.
Others, including the Arkansas Democrat-Gazette, have also been there since the beginning as this tournament has grown to be one of the nation's most successful. It has done so because of the way Ingram and his support staff operate it from the day they start accepting entries until the last game today.
Everyone is treated the same, with respect. There are no favorites, and that's why teams sponsored by every shoe company will travel to central Arkansas. They know the games are going to called honestly and without bias by the officials and that someone has an answer to every question.
This afternoon a great tournament will end, most likely on a great note again.
Sports, Pages 23 on 04/20/2014


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