text:
Read an Excerpt
<block quote="">
The white wolf ran as fast as she could, lungs burning with the effort, legs stretching out as far as they could. Thank god she was in wolf form, not human, but that brought other problems. She thought she'd scoped out the new territory thoroughly enough, but then she'd run into some other creatures when she was in wolf form who told her a horror story that made her blood freeze.
Trapped and captured, they told her. Their friends disappearing. Rumours of a big preserve on a ranch, land big enough to hide it all. Where the trapped animals were hunted and killed. Sometimes maimed first, then released to be hunted again.
Stay away, they'd told her.
Her brother, John, as usual, had been too curious. Now he'd been gone for two days, and she feared the absolute worst. She needed help, but where was she going to get it? She'd been so sure she'd find the peace she longed for in Texas. And she'd heard about a group of shifters trying to reform a pack. Now she needed to connect with them more than ever.
But her first priority was staying alive.
She heard the voices coming close punctuated by drunken laughter, and the increased thunder of hoof beats as she finally reached the huge oak tree where she'd left her clothes. Praying she had enough time, she paused, forced herself to shift, and scrambled up into the tree. She clung to the thick branch, naked and shivering, while the men passed beneath her. When she was sure it was safe, and movement couldn't be detected, she pulled her clothes back on. But it was nearly dawn before she had the courage to climb down from the tree and stealthily make her way back to her campsite.
* * * *
Drew Noland sat on his back porch cradling a cold beer in his hands, watching the first edge of night creep over the Texas Hill Country. He loved this area of South Central Texas, the mixture of rolling pasture land with tree-dotted hills, copses of trees here and there in the vast areas of emptiness where civilisation had only bumped the edges. Ranch country. Cattle, goats, sheep, all being raised in what Drew considered the closest thing to heaven on earth. He drew in a deep breath, inhaling the mingled scents of nature that never failed to stir his senses.
He'd bought his small ranch ten years ago when he landed in the Hill Country and decided it was a safe place to stay. Running about a thousand head of cattle, he made enough to keep the ranch in the black, needing only a minimum of hands to help and still have time for his work with The Sentinels.
Now he was doing one of his favourite things-watching night lower its blanket over the countryside and the first appearance of the stars in the velvet sky. It gave him a feeling of peace he hadn't known until he came here, peace that had been stolen from him, just like it had from his partners. A sliver of moon drifted into the sky. He heard the soft whinnying of horses in the barn, broken now and then by the distant wail of a coyote.
He'd heard other ranchers discussing the increasing presence of the predator, said there'd never been this many coyotes here before. But what bothered Drew was the talk in the feed store and the diner of the chance spying of other animals not indigenous to the area. Someone even mentioned wolves, which made Drew's guts tie up in a knot. He knew there were no wolves around here except for himself, when he wasn't in human form.
He knew what madness even the whisper of a wolf could do in a community, and a tiny thread of fear unravelled inside him. A human pack was far more dangerous than any group of animals. It was how his pack had been destroyed. How he, himself, had nearly been killed, hiding in the woods in the northern state where they'd lived, woods that he was so familiar with-shifting to his human shape during the day to avoid detection. Men determined to kill wolves at all costs were like rabid dogs, crazed and maniacal.
Few members of his pack had survived, and they'd scattered for safety. Some hand of fate must have led them all to Texas, where he ran into the Spencer brothers working at the Houston Stock Show. The three of them had managed to find the other five remaining members, all gravitating to a small town outside San Antonio.</block>


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Description patchwork players south carolina
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text:
Final Fantasy VII
So is it really the greatest RPG of all time?
Well, Final Fantasy VII is absolutely epic in scope. With a sweeping story line that spans three discs, the game makes the 40-hour completion time suggested by Square seem like an underestimation. A large map, with a wide selection of towns and cities, offers plenty of areas to explore for the intrepid adventurer. Cinematic sequences (including, but not limited to, fully rendered full-motion animation cinemas) advance the complex -- and sometimes surprising -- story. And several mini-games (like arcade fighting, motorcycle racing and snowboarding) offer light-hearted respite from the serious story line. But does it live up to the hype?
Well, as reported, the graphics are nothing short of stunning. The prerendered backgrounds provide some of the richest environments ever seen in a console title. The cinematic sequences segu´ almost seamlessly into actual gameplay, ridding the game of the unpleasant graphic dichotomy found in most cinema-laden CD-based games. And clever graphical tricks, like decreasing the size of the characters as they move from foreground to background, add to the game's visual depth. And the powerful summoning spells acquired as the game progresses are absolutely awe-inspiring, combining polygonal graphics with what appear to be beautifully hand-drawn special effects. But does it live up to the hype?
Well, the combat interface is cleverly done, with a "realtime" engine that is nevertheless turn-based. By turn-based, I mean that characters must wait for a Time meter to fill before choosing their attack, and attacks always occur in the order they are chosen, with no two characters (either friend or foe) attacking at the same time. This adds a level of urgency and excitement not found in most other RPGs.
But does it live up to the hype? Well, that really depends. I have to say that, now that I've been able to put some serious time into the game, I'm a bit disappointed. The most frustrating thing about the game is the surprisingly linear story line. Until gaining access to the game's vehicles (at least 15 hours into the game), players are basically forced to follow a strictly set path. Oh, you'll have the appearance of choices, in conversation and in travel, but explore the alternatives and you'll discover that there is really only one feasible path to take. This is doubtless the case because of the focus on the fully developed story, but it may bother gamers used to more wide-open gameplay. The inclusion of some truly challenging puzzles would have helped a bit, but these are scarce. To make things worse, at some points the translation from the Japanese appears a bit muddy, causing unnecessary confusion and clouding the fine story.
Don't get me wrong, this is a great game -- and I don't mean great like "Hey, great!" but great like Alexander the Great. I guess I was just expecting it to be a lot closer to perfect than it is. It's still a must-buy for any PlayStation owner; just keep in mind that you'll have to check a good deal of your freedom at the door.
See all Final Fantasy VII Reviews >
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More RPG Games
- Game:
- Final Fantasy VII
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- Publisher:
- Sony CEA
- Developer:
- Square Electronic Arts
- ESRB Rating:
- Teen
- Release Date:
- 05/18/1998
- Also Known As:
- Final Fantasy VII
1UP Editor Score: A+
Average Community Score: A
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Although Gust hasn't officially announced the title, which is the fifth in the Atelier series, it's not all that surprising since they did release a PSP port of its prequel, Atelier Judie: Alchemist of Gramnad earlier this year. The Japanese release date given for Atelier Violet's PSP version is February 3rd, 2011. There are also two editions listed: a standard for 5040 yen ($62.48 US), and a 'premium edition' (no details given) for 7140 yen ($88.52).
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text:
Sorry, I am very new to DbGrids.
Should I use the query's field editor and somehow add a new field that captures the TIMEDIFF and then just add that as a column in my DbGrid?
Or can/should I skip the field editor and somehow declare the TIMEDIFFF as a column?
For this table, I want a DbGrid with 4 columns : start time, end time, duration, description (run_id is the primary key & will not be displayed).
I am stumped as to how to get data into a 'duration' column ...
mysql> describe test_runs;
| Field | Type | Null | Key | Default | Extra |
| run_id | int(11) | NO | PRI | NULL | auto_increment |
| start_time_stamp | timestamp | YES | | NULL | |
| end_time_stamp | timestamp | YES | | NULL | |
| description | varchar(64) | YES | | NULL | |
4 rows in set (0.37 sec)
[Update] The query for the datasource is
SELECT start_time_stamp,
TIMEDIFF(end_time_stamp, start_time_stamp) as duration,
FROM test_runs ORDER BY start_time_stamp DESC
and when I execute it manually in MySql, I get
mysql> select TIMEDIFF(end_time_stamp, start_time_stamp) as duration FROM
| duration |
| 00:04:43 |
| 00:00:13 |
| 00:00:06 |
| 00:00:04 |
5 rows in set (0.00 sec)
but the corresponding column in the DB grid remains blank. Can anyone help? Thanks.
[Update] I am using AnyDac, if that helps. The query produces all fields, including the time difference, in MySql and also in the Delphi IDE when I use the AnYDac query editor and execute it.
The only problem is that I don't see it in the DB grid at run time. I double click the DB grid at design time and the columns are correct. The
FielName property is set to
duration, which is retuned by the query shown above. It doesn't exist in the database, but is calculated by the query; could that somehow be the problem??
[Aaaaaaaargh!!!] Someone tried to "improve" my code and set the query's text programatically at run-time (as
SELECT * FROM test_runs), thus overwriting my design time query!! Since the databse table does not have a
duration field, none was shown in the DB grid.
Words were had, voices were rasied and now I must apolgize for wasting your time. Sorry.


text:
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Insomnia coffee shop & Skyline Chili @ High & 13th. Insomnia was open all night I believe, and had a patio out front that was always packed with scary/interesting people.
As I heard it, both Skyline and Insomnia were pushed out of this building by Campus Partners, who let on that it would be torn down and become some kind of office space or apartments. Instead, the building remained as-is and became a trendy Asian bar/restaurant.
Skyline moved a few blocks down the street, but was never successful and soon closed, leaving OSU's Cincinnatians without a place to get their hometown fix. Insomnia never resurfaced.
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Steven Tyler embarks on a solo tour this summer including a stop at The Chicago Theatre on Saturday, August 13.
Don't miss Jenny Lewis & The Watson Twins as they bring the Ten Year Anniversary Tour of Rabbit Fur Coat to our stage on September 8!
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Visit The Chicago Theatre history page for 90 years of Chicago Theatre memories.


text:
List of Horror Movie Reviews - "D"
This movies list starting with "D" includes reviews for Dark Planet, Dark Reality, Dark Rising, Dark Skies & Dark Star: H.R. Gigers Welt. The first movie on this page is Dark Planet scoring 2/10 stars in our movie review and has an average user score of 5/10 stars from 6 users. Starring in this movie is Harley Jane Kozak, Michael York, Sal Landi, Ed O'Ross, Phil Morris. The second movie listed is Dark Reality scoring 3/10 stars in our movie review list and has an average user score of 5/10 stars from 6 users. We hope you enjoy our list of movie reviews here with more upcoming movies coming soon.
Dark Planet (1997)Our Score: 2/10 IMDB Score: 3.3/10
Harley Jane Kozak, Michael York, Sal Landi, Ed O'Ross, Phil Morris - Yes it is the year 2638; this is the year of the cleansing war; different factions of people such as Alphas (genetically engineered), and the Rebels (natural born) with a few other factions are tossed…
Dark Reality (2006)Our Score: 3/10 IMDB Score: 2.8/10
Alisha Seaton, Eva Derrek, Laila Dagher, Arthur Bullock, Jen Parker - Backpacking across Europe was supposed to be an adventure of a lifetime for Carey. But a different journey begins when she awakes in a cold, dark basement. A nightmarish journey filled with physical torture…
Dark Rising (2007)Our Score: 5/10 IMDB Score: 4.6/10
Landy Cannon, Rogue Johnston, Jason Brown, Chad Allen, Jack Newman - A broken heart, a battle ax, a demon and a lesbian ex-fiancee… Jason Parks' life goes from bad to worse as a series of supernatural events turn his dream of reconciling with his ex Jasmine…
Dark Skies (2013)Our Score: 8/10 IMDB Score: 6.3/10
J.K. Simmons, Brian Stepanek, Josh Wingate, Rich Hutchman, Marion Kerr - Contains Spoilers Dark Skies is another amazing horror movie for 2013. Director Scott Stewart (Legion, Priest, What We Talk About When We Talk About Love) did a phenomenal job with the cast of the movie.…
Dark Star: H.R. Gigers Welt (2014)Our Score: 6/10 IMDB Score: 7.0/10
Leslie Barany, Sandra Beretta, Mia Bonzanigo, Tom Gabriel Fischer, H.R. Giger - Dark Star: H.R. Giger's Welt (2014) is a Switzerland documentary biography that switches back and forth between the year 2014 and archival footages. The documentary is 95 minutes long and was filmed…
Dark Summer (2015)Our Score: 4/10 IMDB Score: 4.2/10
Peter Stormare, Keir Gilchrist, Grace Phipps, Maestro Harrell, Stella Maeve - Dark Summer (2015) is an American low budget thriller horror movie that was filmed that did not live up to my expectations. Director Paul Solet (Grace (2009), Means to an End (2005), Grace (2006)) tried…
Dark Touch (2013)Our Score: 6/10 IMDB Score: 4.8/10
Katie Kirby, Art Parkinson, Richard Dormer, Mark Huberman, Clare Barrett - Dark Touch is about a little girl name Niamh/Neve (Missy Keating - The Sea) who kills every foster/adopted family she lives with. I am not sure if she kills intentionally or not. The movie doesn't really…
Dark Town (2004)Our Score: 2/10 IMDB Score: 3.6/10
Delpaneaux Wills, Ariana Fronti, T'Shaun Barrett, Noah Knight, Lanre Idewu - A quiet suburban town is overrun by vampires! The once idyllic picture of the typical American family, now hosts spectacles of carnage behind every picket fence as the neighbors prowl the streets feasting…
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Reviews beginning with the letter D like Dark Planet, Dark Reality, Dark Rising, Dark Skies, Dark Star: H.R. Gigers Welt.
Displayed in "most viewed" order with titles such as Dark Planet, Dark Reality, Dark Rising, Dark Skies, Dark Star: H.R. Gigers Welt, Dark Summer, Dark Touch, Dark Town.


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Midsomer Murders: Set 13 (Dance with the Dead / The Animal Within / King's Crystal / The Axeman Cometh)
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Set 13 contains the following episodes:
ep 52: Dance With the Dead; orig air date: Nov 2006
ep 53: The Animal Within; orig air date: Jan 2007
ep 54: King's Crystal; orig air date: Jan 2007
ep 55: The Axeman Cometh; orig air date: Feb 2007
SPOILER ALERT!!!: In The Axeman Cometh we meet Simon, Cully's intended. Her wedding is in ep 60 (which I guess will be in set 15). END OF SPOILER ALERT!!!
If you own any MM sets from Region 2, you may already own at least some of the above episodes, as the Region 1 and 2 sets are NOT synchronised. Better check... I read somewhere there were fewer MM episodes released in the US in order to keep the price of each set more affordable. If that was indeed the case, then it is a great shame... I would like to see those missing episodes released at some point in the US as well.Read more ›
First the big review.....These episodes are as wonderful as all the previous shows. The ability of the writers to keep up the quality is nothing short of amazing. What a track record. Whoa!
Now the boring message...these episodes were released on dvd in the UK in September 2008. The entire TEN EPISODES of that season were released as "The Sixth Collection" You can buy it from Amazon UK for about $70 including shipping. This USA release has only four of those episodes issued one year later at about $36.
The one catch here is the UK releases are on the region 2 PAL format which will not play on a lot of the USA region 1 dvd players. BUT you can buy a dvd player that will play the region 2 PAL format right here on Amazon. I did.
Series Note....John Nettles will be retiring at the end of 2010 with one final series of eight episodes.With the way Acorn chops up these programs into little three and four episode packages for the USA it will be 2012 before we get to see any of them.
John Nettles, the lead, settled into character with the very first show and is absolutely brilliant as the stern detective with a strong sense of justice, a good sense of humor. His love and affection for his family lights up his eyes during even the most difficult case. It is nice to see.
We have now bought many of the Midsomer series and will by them all. Can't recommend this series highly enough.
The only drawback is that every now and then we get a disk that sticks. I hate to miss any of the stories due to this. Still giving it 5 stars because we enjoy the series so much.
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I haven't been disappointed by any of the Midsomer Murders series or the characters. They are always entertaining.Published 9 days ago by Lanna Alkazin
Buyer Beware! Do not purchase any Midsomer Murders marked SET. This is not Series 13 of the Midsomer Murder Series. The seller has repackaged the 4 episodes of Season 10. Read morePublished 2 months ago by Bob
I have almost all of the set's, very very great series. I love British villages and settings and thee actors as well.Published 10 months ago by chip fortune
DCI Barnaby is great. he is a woman lover. especially those women with brown/blonde hair. he actually day dreams about them in some of his episodes. Read morePublished 16 months ago by Helen L. Campbell


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OS: Opensuse 11.3 with KDE 4.4 x86_64 on AMD
Nvidia graphics with nvidia-driver
everything working very well
Kdenlive Version 0.7.8 with mlt 0.5.10 from Packman
I could make a new project with 66 HD 720/30-files (about 6Gbyte) and HD 720/30 project-properties. Could edit some minutes and saved that project. Today I tried to resume editing - kdenlive loads and after some seconds it crashes with "segmentation fault"
Output from gdb:
File given: true
QWidget::insertAction: Attempt to insert null action
QWidget::insertAction: Attempt to insert null action
Missing separate debuginfo for /lib64/libnss_files.so.2
Try: zypper install -C "debuginfo(build-id)=3c394385f320f548f286d584dfe412a593874bda"
[New Thread 0x7fffdbf2e710 (LWP 19361)]
[New Thread 0x7fffdb72d710 (LWP 19362)]
[Thread 0x7fffdbf2e710 (LWP 19361) exited]
[Thread 0x7fffdb72d710 (LWP 19362) exited]
[New Thread 0x7fffdb72d710 (LWP 19363)]
[New Thread 0x7fffdbf2e710 (LWP 19364)]
[Thread 0x7fffdb72d710 (LWP 19363) exited]
[Thread 0x7fffdbf2e710 (LWP 19364) exited]
[New Thread 0x7fffdbf2e710 (LWP 19365)]
[New Thread 0x7fffdb72d710 (LWP 19366)]
[Thread 0x7fffdbf2e710 (LWP 19365) exited]
[Thread 0x7fffdb72d710 (LWP 19366) exited]
[New Thread 0x7fffdb72d710 (LWP 19367)]
[New Thread 0x7fffdbf2e710 (LWP 19368)]
[Thread 0x7fffdb72d710 (LWP 19367) exited]
[Thread 0x7fffdbf2e710 (LWP 19368) exited]
[New Thread 0x7fffdbf2e710 (LWP 19369)]
[New Thread 0x7fffdb72d710 (LWP 19370)]
Missing separate debuginfo for /lib64/libnss_compat.so.2
Try: zypper install -C "debuginfo(build-id)=131fc800003a4275443f0cb7c9e9eb28e3b702e3"
Missing separate debuginfo for /lib64/libnsl.so.1
Try: zypper install -C "debuginfo(build-id)=14435e6aee08e56dd62ad44bb6726c92da1dbc21"
Missing separate debuginfo for /lib64/libnss_nis.so.2
Try: zypper install -C "debuginfo(build-id)=70284dfc19dd6de044b8ca8ff90a42ff7b2b8599"
[Thread 0x7fffdbf2e710 (LWP 19369) exited]
[Thread 0x7fffdb72d710 (LWP 19370) exited]
[New Thread 0x7fffdb72d710 (LWP 19371)]
[New Thread 0x7fffdbf2e710 (LWP 19372)]
[Thread 0x7fffdb72d710 (LWP 19371) exited]
[Thread 0x7fffdbf2e710 (LWP 19372) exited]
[New Thread 0x7fffdbf2e710 (LWP 19373)]
[New Thread 0x7fffdb72d710 (LWP 19374)]
[Thread 0x7fffdbf2e710 (LWP 19373) exited]
[Thread 0x7fffdb72d710 (LWP 19374) exited]
[New Thread 0x7fffdb72d710 (LWP 19391)]
[New Thread 0x7fffdbf2e710 (LWP 19392)]
[New Thread 0x7fffd9d8a710 (LWP 19417)]
[New Thread 0x7fffd9589710 (LWP 19418)]
[Thread 0x7fffdb72d710 (LWP 19391) exited]
[Thread 0x7fffdbf2e710 (LWP 19392) exited]
Missing separate debuginfo for /usr/lib64/libvdpau.so
Try: zypper install -C "debuginfo(build-id)=9d6abde24a807174aa27b304c7ff7ec3b984a951"
[Thread 0x7fffd9d8a710 (LWP 19417) exited]
[Thread 0x7fffd9589710 (LWP 19418) exited]
[New Thread 0x7fffd9589710 (LWP 19419)]
[New Thread 0x7fffd9d8a710 (LWP 19420)]
[New Thread 0x7fffdbf2e710 (LWP 19421)]
[Thread 0x7fffdbf2e710 (LWP 19421) exited]
[New Thread 0x7fffdbf2e710 (LWP 19422)]
[New Thread 0x7fffdb72d710 (LWP 19426)]
[New Thread 0x7fff97608710 (LWP 19427)]
[New Thread 0x7fff96e07710 (LWP 19428)]
[New Thread 0x7fff96606710 (LWP 19429)]
[New Thread 0x7fff95e05710 (LWP 19430)]
[New Thread 0x7fff95604710 (LWP 19431)]
[New Thread 0x7fff94e03710 (LWP 19432)]
[New Thread 0x7fff94602710 (LWP 19433)]
[New Thread 0x7fff93e01710 (LWP 19434)]
[New Thread 0x7fff93600710 (LWP 19435)]
[New Thread 0x7fff92dff710 (LWP 19436)]
[New Thread 0x7fff925fe710 (LWP 19437)]
[New Thread 0x7fff91dfd710 (LWP 19438)]
[New Thread 0x7fff915fc710 (LWP 19439)]
[New Thread 0x7fff90dfb710 (LWP 19440)]
[New Thread 0x7fff905fa710 (LWP 19441)]
[New Thread 0x7fff8fdf9710 (LWP 19442)]
[New Thread 0x7fff8f5f8710 (LWP 19443)]
[New Thread 0x7fff8edf7710 (LWP 19444)]
[New Thread 0x7fff8e5f6710 (LWP 19445)]
[New Thread 0x7fff8ddf5710 (LWP 19446)]
[New Thread 0x7fff8d5f4710 (LWP 19447)]
[New Thread 0x7fff8cdf3710 (LWP 19448)]
[New Thread 0x7fff8c5f2710 (LWP 19449)]
[New Thread 0x7fff8bdf1710 (LWP 19450)]
[New Thread 0x7fff8b5f0710 (LWP 19451)]
[New Thread 0x7fff8adef710 (LWP 19452)]
[New Thread 0x7fff8a5ee710 (LWP 19453)]
QCoreApplication::postEvent: Unexpected null receiver
..... about 30 times more ....
QCoreApplication::postEvent: Unexpected null receiver
Maximum number of clients reachedQCoreApplication::postEvent: Unexpected null receiver
QCoreApplication::postEvent: Unexpected null receiver
Maximum number of clients reachedMaximum number of clients reachedMaximum number of clients reachedMaximum number of clients reachedMaximum number of clients reachedMaximum number of clients
........about 30 times more ...........
reachedMaximum number of clients reachedMaximum number of clients reachedMaximum number of clients reached
Program received signal SIGSEGV, Segmentation fault.
[Switching to Thread 0x7fffdb72d710 (LWP 19426)]
0x00007fffe743ed44 in av_find_stream_info () from /usr/lib64/libavformat.so.52
My observation:
kdenlive tries to start numerous child-processes
kdenlive tries to hold everything within memory and the mashine is running out of memory (4GB)
I can not even load old projects which I could edit without any crash.
Can anyone help?
Sun, 11/14/2010 - 06:50
The error message you are receiving is "Maximum number of clients reached." This is a redundant post
"kdenlive tries to hold everything within memory"
As the MLT developer, I can tell you that is not correct because I implemented a MRU cache and painstakingly reworked the ffmpeg and image loading plugins to use it. It only holds 10 of the most recently used resources (per plugin class) and automatically restores them on demand.
Please test if disabling vdpau makes a difference. Run kdenlive from a terminal like this:
'MLT_NO_VDPAU=1 kdenlive'
VDPAU needs to do something with X, according to the other thread this appears related to Xlib, and I see you are using vdpau.
Thu, 11/18/2010 - 08:54
Looks like the crash stems from kthumb.cpp.
Try turning off thumbnailing in the Settings > Timeline, restart Kdenlive (just to be sure), and then loading the project.
Sat, 11/27/2010 - 03:14
may I share my experience about a crash:
When I open Kdenlive, it's OK. But in a second, it show an error some of ??? is not readable, as show in attached picture. Could someone help me, please?
p.s. sorry for my english.
Sat, 12/11/2010 - 19:44
jbm recently made a change to thumbnailing to prevent launching a thumbnailing thread for each clip in parallel - limits it to at most # cores. This may help prevent the bug in the future.


text:
Fibonacci Sequence Use the result of Problem to do the following problems:
(a) Write the first 11 terms of the Fibonacci sequence.
(b) Write the first 10 terms of the ratio .
(c) As n gets large, what number does the ratio approach? This number is referred to as the golden ratio. Rectangles whose sides are in this ratio were considered pleasing to the eye by the Greeks. For example, the façade of the Parthenon was constructed using the golden ratio.
(d) Write down the first 10 terms of the ratio .
(e) As n gets large, what number does the ratio approach? This number is referred to as the conjugate golden ratio. This ratio is believed to have been used in the construction of the Great Pyramid in Egypt. The ratio equals the sum of the areas of the four face triangles divided by the total surface area of the Great Pyramid.
Fibonacci Sequence Let
define the nth term of a sequence.
(a) Show that u1 = 1 and u2 = 1.
(b) Show that un+2 = un+1 + un.
(c) Draw the conclusion that {un} is a Fibonacci sequence.


text:
Installed new headgasket, had head cleaned, resurfaced and tested, installed new water pump and timing belts, thermostat and few hoses, bled system, ran car with no leaks, timed car to 15 degrees and is overheating. Have no clue what could be causing this other than possible stuck thermostat but seems to be good.
Friday, May 18th, 2012 AT 4:51 PM


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Showing Results: Most Recent
This review is from: AMD FX-8350 Black Edition Vishera 8-Core 4.0 GHz (4.2 GHz Turbo) Socket AM3+ 125W FD8350FRHKBOX Desktop Processor
Pros: Roughly 6-7% increase in architectural improvement over bulldozer at the same clock speed and lower power consumption at stock and oc than bulldozer.
Cons: The end of the line for AM3+. Still much higher power consumption than intel.
Other Thoughts: This coupled with faster ram netted me an 18% increase over my fx-8150 with roughly the same percentage overclock. Ill be hanging onto this chip until a new socket and cpu with ddr4 support is available.READ FULL REVIEW
This review is from: SAPPHIRE Radeon R9 290 DirectX 12 100362-3L 4GB 512-Bit GDDR5 PCI Express 3.0 CrossFireX Support Tri-X OC Version (UEFI) Video Card
Pros: Card is extremely fast. Upgraded from MSI 7950 and its night and day faster.
Cons: noneREAD FULL REVIEW
This review is from: G.SKILL 4GB 204-Pin DDR3 SO-DIMM DDR3 1600 (PC3 12800) Laptop Memory Model F3-12800CL11S-4GBSQ
Pros: Excellent quality and great price. Took my laptop to 8 gig and i love it.
Cons: noneREAD FULL REVIEW


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Previous work has shown the ability of waveform LiDAR sensors to accurately describe various land cover types [1] and biomass estimates made in the field [2]. What is lacking, however, is a way to describe the different structural components that are embedded in the digitized backscattered energy from the LiDAR pulse. This study aims to extract structural components from waveform LiDAR data in terms of woody, herbaceous, and bare ground components from data collected over a savanna environment in and around Kruger National Park (KNP), South Africa. These components are comprised of metrics extracted from the waveforms and validated using biomass measurements made in field plots. Different size windows around plot centers, 3 × 3 pixels and 9 × 9 pixels (resulting in 1.5m and 4.5 m footprint, respectively), were used to examine scale effects of larger footprints. It was found that composite waveforms resembling plot sizes (9 × 9) most often are able to describe more than 80% of the woody biomass variability across the entire study site, and individually for two of the three land uses within the area. However, the herbaceous component of the waveform did not correlate well with the field measurements, while the bare ground component was verified visually in a side-by-side comparison with optical imagery.
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Human faces present crucial visual information for social interaction. Specialized brain regions are involved in the perception of faces, with the fusiform face area (FFA) a key neuronal substrate. Face processing is genetically controlled, but by which specific genes is unknown. A genome-wide approach identified common single nucleotide polymorphisms (SNPs) associated with areas of increased brain activity in response to affective facial expressions, measured with functional magnetic resonance imaging. SNPs in 20 genetic regions were linked with neural responses to negative facial expressions in a Norwegian sample (n=246), which included patients with mental illness. Three genetic regions were linked with FFA activation in a further discovery experiment using positive facial expressions and involving many of the same individuals (n=284). Two of these three regions showed significant association with right FFA activation to negative facial expressions in an independent North American replication sample of healthy Caucasians (n=85, 3q26.31, P=0.004; 20p12.3, P=0.045). The activation patterns were particularly striking for the SNP in 3q26.31, which lies in a gene TMEM212; only the FFA was activated. The specialized function of this brain region suggests that TMEM212 could contribute to the innate architecture of face processing.
Face processing is a crucial cognitive ability. Rapid recognition of faces identifies individuals and informs us about basic properties such as age, gender and race, as well as complex processes such as direction of attention, mood and intent. Thus, the ability to extract such information from even a momentary viewing of a face is critically important for normal social interactions.
Several lines of evidence indicate that face processing is unique and qualitatively different from processing of other visual stimuli.1, 2 Studies of neurological disorders suggest a dedicated circuitry exists in the human brain for face processing. Converging evidence now suggests that a network of multiple brain regions, in particular the right fusiform gyrus, supports face perception.3, 4, 5, 6
Similar specialized processing of faces has been reported in macaques,7, 8 and it has been suggested that specialized pathways for face processing were critical for the survival of our primate ancestors.9 This implies that face processing is under some genetic control, and findings in infants show that face recognition is an innate human capability.10, 11 In fact, using behavioral tasks several twin studies have detected a high degree of heritability in face processing12, 13 (one study found that correlation of one particular score of face recognition for monozygotic twins (0.70) was more than double that of dizygotic twins correlation (0.29)13). However, the specific genes involved in face processing are unknown, as is the effects of such genes on the neural networks supporting this specialized behavior.
The aim of the current study was to identify common genetic variants associated with neural activation during face processing. Using genome-wide genotype microarray technology together with functional magnetic resonance imaging (fMRI) in an ethnically homogenous sample of healthy controls and psychiatric patients from Norway,14 and an independent replication sample of Caucasians from North America,15 we discovered two genomic regions associated with neural activation during face processing.
The Norwegian TOP sample consisted of 246 individuals after quality control on the genotype and imaging data. The fMRI protocol consisted of a widely utilized emotional faces paradigm,15, 16, 17 involving the perceptual processing (matching) of emotional facial expressions (experimental condition) or geometric shapes (control condition). Subjects completed two sessions, one with all negative (angry or afraid) and another all positive (happy) expressions, in a counterbalanced order. We will refer to these experiments as the negative and positive faces experiments. No one was excluded based on performance as it was a very easy task and subjects practiced it to demonstrate understanding before entering the scanner. In the negative faces experiment, the age was 34±10 (mean±s.d.) years and there were 130 men and 116 women. There were 138 participants with mental disorder, whereas 108 were healthy controls. For the positive faces paradigm, we included 284 individuals, age 34±9 years; 157 male, 127 female, 160 with mental disorder and 124 healthy controls. Single nucleotide polymorphism (SNP) genotyping was performed using the Affymetrix Gene Chip Genome Wide SNP 6.0 array (Affymetrix Inc., Santa Clara, CA, USA, http://www.affymetrix.com). Individuals with discrepancies between reported and genotype-inferred gender and those whose ancestry differed from the majority of the sample were removed. Mitochondrial and X-chromosome SNPs were also removed, and we excluded individuals and SNPs with >3% missing data, and SNPs with a minor allele frequency of <0.05.
The fMRI data from both tasks were processed using the SPM2 software18 to produce contrast maps. For both negative and positive faces experiments, we focused on the 20,000 voxels with most evidence of differential activation to reduce multiple testing. A further prescreening stage rejected voxels in clusters of <50. In the negative faces experiment, every voxel was tested for association with every SNP using an additive model of genetic effect. For each voxel, the P-value for the most significant SNP was adjusted for multiple testing across the SNPs using the Šidák correction.19 Multiple testing across the voxels was accounted for by controlling the false discovery rate (FDR) under the assumption of positive dependencies using a common adjustment method.20 We validated our associations in the positive faces experiment by testing each voxel for association with 35 SNPs suggested by the negative faces experiment; multiple testing across voxels was controlled for each SNP separately.20 In both analyses, voxels were only included in the results if they lay within clusters of >10.
The North American replication sample consisted of 85 healthy middle-aged Caucasian subjects, 41 males, 44 females; age 45±7 years). All fMRI data preprocessing and single-subject analyses were completed using SPM2.18 These participants were genotyped using the Illumina 610-Quad BeadChip (Illumina Inc., San Diego, CA, USA). Perfect proxies were available on this array in two of the genetic regions under study, while the third region had no proxy and so was separately genotyped using fluorescence polarization methods. Single-subject contrast images were entered into separate regression analyses with the rs12485367, rs6038686 and rs6081495 genotypes, including gender and age as nuisance covariates; these analyzes were conducted in SPM818 (http://www.fil.ion.ucl.ac.uk/spm).
More details, including a full description of the subjects, and the processing and analysis of the data can be found in Supplementary material available online.
We carried out genome-wide analyses using the Affymetrix 6.0 Array on the subjects who had successfully completed the fMRI protocol. Clusters of voxels exhibiting significant differential activation in the direct comparisons of the experimental and control conditions were used. The 20,000 most significant voxels were identified and any clusters of <50 voxels were discarded, in order to choose groups of voxels able to contain diffuse associations. After this procedure, 19,742 voxels remained for analysis from the negative faces scan, and 19,864 from the positive faces scan. For both the negative and positive faces, voxels exhibiting a significant main effects of task (that is, experimental>control condition) were principally located in ventral visual cortices as well as frontal and subcortical regions. There is highly significant evidence of differential activation for these voxels (Bonferroni-corrected family-wise error (FWE) rate for the voxels in the negative faces is 1.2 × 10−8). The exact location of the voxels selected for both scans is provided in Supplementary Figure S1 online.
Initially, fMRI data from the scan with negative facial expressions were analyzed as these stimuli have been more commonly used than positive expressions in the larger neuroimaging literature.21 Each of the voxels as chosen above was tested for an association with each SNP (a total of 549,640 SNPs, meaning approximately 1010 tests) using an additive model of genetic effect. The P-values were adjusted to account for multiple testing: first using the Šidák correction19 to produce P-values, adjusted for multiple testing across the SNPs. Then the Benjamini Hochberg procedure20 was applied to these adjusted P-values to control the FDR (the procedure controls the expected proportion of false positive results), accounting for multiple testing across both SNPs and voxels. With the exception of the preselection of task-related voxels described above, this is a similar strategy to one previously presented applied in a genome-wide association study of structural imaging phenotypes.22 In that paper, identical procedures for controlling for multiple testing across SNPs and phenotypes (vertices as opposed to voxels) were followed, with the exception that we applied a more conservative procedure to control for multiple testing across SNPs (ignoring linkage disequilibrium structure and correcting for every SNP rather than calculating an 'effective number of tests'). Similarly, a set of phenotype–genotype associations is presented, with the appropriate estimate of the FDR. More details can be found in Supplementary material, including Supplementary Figure S2 available online, a visual representation of the correction procedure.
Evidence that the activation of 455 voxels had a genetic component was found, with an associated FDR of 0.5. This is a highly liberal threshold, but as this first part of the study was intended to contribute to the initial discovery of potential SNPs and significance will be assessed in the replication experiments, a higher proportion of false positives was tolerated to avoid discarding interesting results.
Only clusters of at least 10 voxels were selected. This is a smaller threshold than that used for preselection of the 20,000 voxels (which insisted clusters had at least 50 voxels), as here we simply wished to discard spurious findings. There were 10 such clusters, totaling 226 voxels, located in the cuneus and occipital gyrus, both known to be involved in visual processing, and bilateral fusiform gyrus, which is implicated as central to face processing specifically.3 These clusters were associated with 37 SNPs located in 20 genomic regions <40kb. These 20 genetic regions and the associated brain regions are listed in Supplementary Table S1 available online. Supplementary Figures S3–S5 available online show Manhattan and Q-Q plots for three voxel phenotypes, those most significantly associated with SNPs rs12485367, rs16992973 and rs2208796, these are SNPs we will develop further. There is little evidence that any of these SNPs are not in Hardy Weinberg Equilibrium (for rs12485367, P=1.00; rs16992973, P=0.14; and rs2208796, P=0.13). We see little evidence of genomic inflation for these phenotypes, for example, λ=0.997 for the phenotype associated with rs12485367. Supplementary Figures S6–S8 available online shows in more detail the P-values for these phenotypes in the genetic regions where they show most significant associations. In Supplementary Figure S6 available online, we see that the association relates to a single signal, as no other SNPs in high linkage disequilibrium with rs12485367 were available in this dataset. This increases the chance that the association could be a false positive induced by genotyping errors; to rule this out, we have explored the population genetics of this SNP in comparison with the North American sample and the CEU sample from the 1000 genomes pilot project23 for evidence of biased ascertainment. This information is contained in the Supplementary Material. We also note that in Supplementary Figure S6 available online, we see that a number of SNPs closest to rs12485367 show P-values between 10−2 and 10−4 despite being in only weak linkage disequilibrium with rs12485367, some evidence that they may be weakly tagging the causal variant.
We plot genotype against peak phenotype for these three SNPs in Supplementary Figure S9 available online. All three SNPs seem to show a consistent trend across genotype; there is little evidence these associations are induced by a few extreme outliers. Finally, Supplementary Figure S10 available online shows Manhattan and Q-Q plots for the peak voxel associated with each of the 20 genetic regions listed in Supplementary Table S1 online. Phenotypes from certain brain regions, in particular the cuneus, show more evidence of genomic inflation. This could be because these regions show greater variation in brain activation, possibly breaking the normality assumptions of the model.
The 37 most significant SNPs from the negative faces scan were tested in the positive faces scan. This is not an independent replication in a new population, as the experiments were performed on mostly the same people during the same scanning occasion performing a similar task and thus the P-values reported are biased towards significance. However, recruitment into the study continued after data for the negative faces scan were collated and analyzed and so genotype data on 57 new individuals were available for the positive faces scan. Two SNPs were discarded as their minor allele frequency was <0.05 in this new population, leaving 35 SNPs. Again using an additive model of genetic effect, associations between SNPs in seven genetic regions and neural activation were identified in 26 clusters of >10 voxels, located in the cuneus, lingual and superior occipital gyri as well as in the bilateral fusiform gyrus (the associated voxel-wise FDR was 0.05, the associations with regions of the fusiform gyrus, a structure particularly involved in face processing, are listed in Table 1, all associations discovered are listed in Supplementary Table S2 online).
The neural regions associated with the seven genetic regions across both the negative and positive faces data are all known to be involved in visual processing. Some of these regions, such as the primary occipital gyrus, are involved with general visual processing. Others overlap with the so-called occipital face area which, together with the fusiform gyrus, is considered necessary for normal face processing.24 For the fusiform gyrus, the right hemisphere is known to be preferentially involved in face processing.3 The results in the negative faces scan are consistent with this, a greater number of associations were found with fusiform gyrus voxels in the right hemisphere than the left hemisphere (36 voxels in the right fusiform gyrus were associated with genetic variants, as opposed to 19 in the left fusiform gyrus). The pattern of activation in the Norwegian sample is particularly striking in the case of rs12485367; significant associations with the fusiform gyrus, only in the right hemisphere in the negative faces scan (16 voxels), and more pronounced in the right than left hemisphere for the positive faces scan were discovered (linked to 47 voxels in the right hemisphere and 43 in the left). This SNP was not associated with any other brain regions in either experiment. In addition, the peak activation in the right hemisphere to negative faces is in almost exactly the same location as the activation associated with this SNP in the positive faces scan (see Figure 1). In the positive faces experiment, the rs16992973 variant was also associated with bilateral fusiform area activation (though more pronounced in the left hemisphere); the remaining genetic region was associated only with activation in the left fusiform gyrus.
Our initial analysis ignored disease status, as analyses of pilot data found no trend effects on activation. However, as others have found associations between regional brain activation and mental illness,25 it was important to investigate whether the present results could be caused by confounding with this factor. We found no significant associations between disease status and the activation of the 20,000 preselected voxels in either experiment. We have also investigated how much controlling for disease status, using indicator variables coding for schizophrenia, bipolar and other psychosis, would affect the results. Supplementary Figure S11 available online shows the P-values declared significant in both experiments against the same P-values calculated when diagnosis is included in the model (for the negative faces experiment, all associations listed in Supplementary Table S1 available online were reanalyzed, this time regressing on SNP dose and disease status; for the positive faces experiment, all associations listed in Supplementary Table S2 available online were analyzed similarly). We see little change; for both experiments, results were actually slightly improved when diagnosis was included in the model, with a small majority of associations more significant (in the negative faces experiment, 207 associations were more significant including diagnosis, 197 were more significant ignoring it; for the positive faces experiment, these numbers were 627 and 615, respectively).
We have also repeated the full analysis performed on the positive faces experiment, but again, controlling for diagnosis. Similarly, we found that the results were largely unchanged, indeed slightly improved. Considering diagnosis makes very little difference to the conclusions we draw and would not affect the choice of SNPs to be investigated further in the replication experiment described below. This agrees with previous work, which found preserved function of the fusiform face area (FFA) in schizophrenia.26 These results are covered in more detail in the Supplementary material online.
Associations across the negative and positive faces datasets were identified. However, in the first negative faces dataset, we expect half of the associations to be false positives, and in the second positive faces dataset, the estimates of significance are difficult to interpret as the SNPs were selected using related data. Therefore, and in line with recent guidelines for replicating genotype–phenotype associations,27 a subset of the significant SNPs was investigated in an independent sample of Caucasians from North America. The replication experiments were designed to concentrate on those SNPs most likely to be involved in face processing. The different levels of complexity of the stimuli could cause activations; viewing faces could also inspire higher levels of attention than geometric figures as could the emotional content. However, there is a substantial body of literature pointing to a specialized role for the FFA in face processing3, 4, 5, 6 and thus we concentrate our focus on SNPs linked to this brain region, in particular in the positive faces paradigm (one SNP of particular interest, rs12485367, was associated with FFA activation in both negative and positive faces paradigms). We use the literature to argue that activations in this region are related to face processing rather than any other cognitive subtractions. For SNPs associated with other regions, a replicated finding would not have this body of work to aid in the interpretation.
The SNPs that were identified in the negative faces dataset and associated with FFA activation in the positive faces dataset were selected (there were five such SNPs, located in three regions). The analyses on activations in the fusiform gyrus used an anatomical region of interest approach, using standard fMRI analyses, which are impractical in the genome-wide association study context due to the large number of tests. The NCI-NHGRI Working Group guidelines state that replication experiments and analyses should be as similar as possible, though this is chiefly in order to remove study differences as a reason for failure to replicate.
The replication sample consisted of healthy middle-aged volunteers who completed a similar fMRI task15, 16, 17 from which the Norwegian paradigms were derived. These participants were genotyped using the Illumina platform. The SNPs upstream of SLC24A3 (20p11.23), had a proxy on the Illumina platform (rs6081495). Another SNP, rs16992973 near RP4-764O22-001 (20p12.3), had a proxy in rs6038686. All proxies were in complete linkage disequilibrium (R2 and D′ equal 1 in the European ancestry population of the HapMap project28). The final SNP, rs12485367 in TMEM212 (3q26.31), did not have a proxy on the Illumina platform and was individually genotyped.
A significant association between rs12485367 (TMEM212) and right fusiform gyrus activation was obtained (x=20, y=−68, z=−14, t=4.77, P=0.004, FWE-corrected, kE=92; see also Figure 2). As in the original experiments, participants carrying the minor (G) allele showed higher fusiform gyrus activation relative to C homozygotes. Significant association between rs6038686 (RP4-764O22-001) and right fusiform gyrus activation (x=26, y=−52, z=−14; T=4.07, P=0.043, FWE-corrected, kE=80; see Figure 3) was also found; such that participants carrying the minor (G) allele showed relatively heightened activation. Again, this matches the result from the Norwegian sample. As in the rs12485367 analyses, the right fusiform gyrus activation remained significant after FWE correction over the search volume of an anatomical fusiform gyrus region of interest. Finally, rs6081495 did not show a significant association with fusiform gyrus activation.
The present findings represent a demonstration of an association between common gene variation and neural function associated with face processing. Validation of some of the initial results using a parallel experiment and replication of the most promising candidates in an independent population was used. It is also important to note that the original associations with fusiform gyrus activation in the Norwegian discovery sample were identified when searching across all neural regions sensitive to the task and not through the narrow window of an anatomical region of interest approach.
There is little knowledge about the function of the genes that were identified, but there are some indications that TMEM212 is expressed in the brain. Affymetrix provides a test of whether a transcript is present or absent within a sample,29 and TMEM212 is present in five out of seven samples of the whole brain tissue collated in the Gene Enrichment Profiler30 (http://xavierlab2.mgh.harvard.edu/EnrichmentProfiler/index.html), and in one out of two samples from the occipital lobe.
Interestingly, both genes replicated in the North American sample lie within regions for which associations have been proposed to disorders involving impairment of social functioning.31, 32, 33, 34, 35 But, as no association to diagnosis was found, and the replication sample excludes any individuals with a current DSM-IV (Diagnostic and Statistical Manual for Mental Disorders) Axis I disorder, we believe these genes to be involved in regulating fundamental brain functions related to face processing unrelated to psychiatric disorders. Further, as these variants were identified and validated using faces expressing negative and positive emotions, these pathways may be broadly important for face processing independent of emotional expression.
The current findings are in line with recent evidence of high heritability of the ability to process face information,12, 13 which suggests a strong biological control of this important human function. Previous studies indicate that the ability for face recognition has little overlap with other cognitive abilities,12, 13 which suggests that the face processing ability is specific. This is supported by the present findings of specific activation patterns related to face processing associated with distinct gene variants, indicating a specific genetic basis. Particularly TMEM212 seems to be specifically involved in the innate architecture of face processing, as it was associated with activation in the FFA only.
This supports the position that distinct cognitive and neuronal mental processes may have specific genetic determinants, as has been seen with language disability.36 Thus, the genes identified in the current study would be interesting candidate genes to investigate for association with developmental prosopagnosia,37 a disorder characterized by impairments in face recognition that are unaccompanied by brain lesions; multiple cases of this disorder have been seen in the same family.38, 39 Investigations into pedigrees has suggested that forms of congenital prosopagnosia can have a dominant, autosomal mode of inheritance.40 Although we have looked for common variation, these genes could be investigated to find rare variants causing pathological results in such families. However, the development of such distinct mental processes probably depends on gene–environment interactions.
To conclude, specific genes that may be important for the modulation of basic neurobiological processes, which underlie human social interactions, have been identified. In addition, we have demonstrated how the synthesis of fMRI and genome-wide data can be used to discover and replicate links between common genomic variation and behaviorally relevant brain function.
We thank the study participants and the members of the TOP study group involved in data collection. This work was supported by Oslo University Hospital, University of Oslo, South-Eastern Norway Health Authority (grant # 2004-123), and the Research Council of Norway (#167153/V50, #163070/V50). Eli Lilly supported parts of the genotyping costs. We would like to thank Eivind Bakken and Christine Lycke Brandt for their work in processing the data and providing background context, Statistics for Innovation for fruitful discussions and Anders M Dale for his very useful advice and guidance.
The authors declare no conflict of interest.
Supplementary Information accompanies the paper on the Translational Psychiatry website (http://www.nature.com/tp)
PubMed Central Canada is a service of the Canadian Institutes of Health Research (CIHR) working in partnership with the National Research Council's national science library in cooperation with the National Center for Biotechnology Information at the U.S. National Library of Medicine(NCBI/NLM). It includes content provided to the PubMed Central International archive by participating publishers.


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County and state officials alike said they were relieved last week when the East-West Gateway Council of Governments board passed its Transportation Improvement Plan.
John Griesheimer, Franklin County presiding commissioner and a member of the EWG board, said there was a risk the plan wouldn't get passed after Illinois officials objected to plans to remove a ramp on Interstate 55 heading over the Poplar Street bridge.
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i aint got no worries
Face Tags
wish I was in Georgia to watch my boy play in the GSC Championship game!! good luck @tyaldridge! #gostatesmen
nowhere I'd rather be than in Georgia watching my boy play for the GSC championship!! good luck @tyaldridge!!! Good luckkkk #gostatesmen
And I'm all like "be my boyfriendddd" @alliejmartin #photobooth
We rented out the movie theater for This is 40 if anyone would like to join. @mbhanna4
I can finally say congrats to my best friend @c_gilbs on getting engaged! so happy for you & @HotspurRackem
I can finally say congrats to my best friend @c_gilbs on getting engaged. ! so happy for you and @HotspurRackem #yayyy
good luck to my favorite Seahawk in his first playoff game today!!! play hard buddy!! #goseahawks @KJ_WRIGHT34
They would give us the most precious but rachet wild puddle jumpin in dog to play with at the shelter @mbhanna4
I blame you for how I feel today #nyeinmemphis @NativeFlash22
we are gonna be new besties #getready @SeanKelley7
my kidden #goodnight @mbhanna4
well my dad's in Stewie pajamas about to fly his new remote helicopter my mom bought him for Christmas. #MerryChristmas
I'm sorry what??? #152
Hell yeahhh #takecare #152
love that we are all in town together for the first time in a year!! #besties @mbhanna4 @carsonbyrd
I told you I'd watch you tonight boyfriend @NativeFlash22 #gorebs ??
I would miss 12:12 on 12/12/12 because I was googling pictures of Tom Brady #whoops #HappyBradyDay
"RT @mbhanna4: off to find the mythical marge @c_melle3 @cpaigechandler" found her @margievia
congrats @JBANKS_13!!! #hailstate
"Those weren't as big of a party hit as I thought they'd be now were they Carl..."- @Hilesj #partyfoods
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Camaro All Weather Jacket - Black$89.00Looking for ultimate comfort? This impressive full-zip jacket combines good look with wind resistant technology. Made of 100% polyester, this jacket features a bonded interlock face with a fleece back, adjustable hook and loop cuff with hidden fleece storm cuff detail inside, chest and side zip pockets. Imported.
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Camaro Fifty Half Zip Pullover$65.00Wear the style of Fifty years of Camaro excellence in this soft-brushed half-zip, long sleeve pullover. Learn More
Camaro Fifty Drivers Jacket$85.00Commemorate Camaro's 50th with a jacket that's perfect when you need something light that looks great! Learn More
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Camaro SS Jacket - All-Weather$89.00Camaro SS Jacket - All-Weather Learn More


text: iPad Mini To Be Announced October 23rd, Says All Things DJamie Condliffe10/12/12 7:54amFiled to: ipad miniRumorsipadApple22EditInvite manuallyPromoteDismissUndismissHideShare to KinjaToggle Conversation toolsGo to permalink AllThingsD is reporting that "people familiar with Apple's plans" say that the iPad Mini will be unveiled at an event to be held on October 23rd.It's tough to say how accurate the rumor is: it's a Tuesday, not a Wednesday, so it would break with tradition, but it could be timed to steal the thunder of the Microsoft Surface (which officially launches just three days later). And AllThingsD has a sparkling track record with these things, so we feel confident backing this one. We still, though, don't know where the event might be held. For full details about what we're expecting from the new iPad Mini, read our full rumor round-up. [AllThingsD] Reply22 repliesLeave a reply


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With the weather forecast predicting temperatures in the high 90's on Sunday, the City of Toledo is expanding its municipal pool schedule. In addition to Willys and Roosevelt Pools that are normally …
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MBE/WBE Contractor Event
Thursday, March 31, 2016
5:30 to 7 pm
McMaster Center, Main Branch
Toledo Lucas County Public Library
325 N. Michigan Street
The City of Toledo invites Minority Business Ent…Read More →
Downtown Toledo Skyline
& Maumee River
One Government Center, Suite #1800Toledo, OH 43604Phone: 419.245.1400Fax: 419.245.1192
The Department of Neighborhoods provides and assists with the development of quality affordable housing, collaborates with community partners to support quality of life initiatives and programs. We strive for code compliance on properties in our community and work to eliminate blight affecting our neighborhoods.
TOLEDO WHEELS Program
If so, you may be eligible for the TOLEDO WHEELS Program. Having adequate transportation can be a significant barrier to a person's employment success and general quality of life. The TOLEDO WHEELS Program addresses this gap by assisting already working individuals/families in acquiring more reliable transportation to gain access to better employment and become self-sufficient.
Please click here TOLEDO WHEELS Program for more information!
The Division of Housing's Owner-Occupied Rehabilitation Program is CURRENTLY CLOSED and is not accepting applications for housing code repairs at this time. A notice will be posted on this website when the program will begin accepting applications.
Charitable Donation Container – REGISTRATIONS NOW REQUIRED
The City of Toledo enacted Toledo Municipal Code Section 962 which defines the City's "Charitable Donation Container Registration" process. The Ordinance establishes rules and regulations pertaining to the approval and placement of containers along with a required yearly registration fee of $100 per location. The registration fee is due by January 1st of each year and is valid for one (1) calendar year or the remaining portion of the calendar year in which the registration was initially required. For questions or more information contact 419-245-1400.
Please click the link below to view registration form.
Charitable Donation Container Registration Form
To view the 2015 Analysis of Impediments to Fair Housing Choice, please click here!
A tree-care specialist can help keep your trees looking healthy and beautiful. From basic pruning to major tree removal and beautification projects, a professional has the experience to handle any tree-related task. Click here for more information about these affordable services!
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To see highlights of the Department of Neighborhoods service to the community, please Click Here to view.
For full listing of reports please visit the Reports & Information Page


text:
Your skill will accomplish
what the force of many cannot
tommywby tommyw (Hermit)
|on Sep 14, 2001 at 12:32 UTC ( #112380=user: print w/ replies, xml )
|Need Help??
I was born at an early age, went to school, discovered computers, and left.
I became a geek. I'd always been a geek, but never knew the term. I went to university, spent far to much time role-playing (or rope-layin as it was infamously misheard), and left.
I got a job, a new job, a mortgage,and left.
I now work for the Oxford English Dictionary.
No, I'm not answering comments about <cite>D'oh!</cite>


text:
Vowing to guide you on the road to spiritual and philosophical maturity, MINGS hopes to help you understand how humankind and its surroundings are inextricably linked. And the role the five elements - metal, wood, fire, water and earth – play in this bonding.
As many associate Feng Shui to Chinese folklore, myths and beliefs and ancient anecdotes, it is MINGS intention to change this perception; to introduce Feng Shui as a science and review its positive integration into the daily highs and lows of an individual.
After a rambunctious 2015 - battered by economic and geopolitical events, we turn our eyes to the cheeky Monkey in 2016. The highly energetic Monkey brings more uncertainty - and opportunity for the prepared.
Discover what is in store for you and the world in 2016 in the bilingual MINGS Outlook 2016 forecast book.
Purchase a copy today!
We are proud to announce our series of members meetings for the coming months.
Come join us to discuss Feng Shui matters of interest and learn authentic Feng Shui principles from MINGS.


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Yellow Watermelon
The yellow watermelon has a canary yellow flesh, often seedless, with occasional black seeds. Tasting no different from the common red watermelon, when ripe, yellow watermelons have the same signature two-toned green skin.
Yellow Corn
Yellow corn is a variety of sweet corn. Its ears are wrapped in tightly bound lime hued husks with silks and a tassel that extend out from the tip. The yellow kernels are packed in tight almost uniform rows.
Fresh Green Almonds
Inventory, lb : 0
This item was last sold on : 06/19/16
|Weiser Family Farms
Green almonds are distinguishable by their fuzzy, green outer hull. Within the green hull, the flesh offers a soft jelly consistency within a skinless nut. If the Green almond is left on the tree to grow and dry out it will eventually harden, split, and reveal a conventional almond. The Green almond can be eaten whole when fresh, green skin and all. When the Green almond is slightly older, the green hull will become tough and bitter and should be removed and the inner almond eaten on its own. The Green almond offers a complex flavor with mild floral and grass-like notes and an overall sweet-tart flavor that is compared to that of green apple, pea pods, and uncured olives.
Green almonds have a short window of availability during the first few weeks of spring.
Current Facts
Green almonds, botanically speaking, are not a nut but rather a type of stone fruit and officially listed scientifically as a member of Prunus dulcis. The almond we consume today as a snacking nut is the seed of the stone (pit) located within the fruit of the almond tree. You can start to see the familial similarities when you note the fuzzy exterior of the Green almond which is similar in texture to another well-known stone fruit, the peach. Green almonds are a spring delicacy, one of the first fruits to appear of the season, and they have a short shelf life. They can found at select farmers markets and Middle Eastern markets during their very brief season.
Nutritional Value
Green almonds offer the same nutritional benefits as dry almonds including vitamin C, vitamin E, iron, calcium, magnesium, riboflavin, and L-carnitine. Green almonds also offer protein and fiber and have a low glycemic index.
Tender Green almonds can be used whole when young and when more mature the husk should be removed, and the inner almond used. Serve simply as is or sprinkle lightly with sea salt to cut its tart flavor. Slice thin and add to salads or use to make salsa or pickled condiments. Sauté Green almond slices simply in olive oil, sea salt, and lemon or poach whole in olive oil. Use sliced atop fruit tarts, ice cream, and other desserts. When Green almonds are a bit more mature, typically towards the middle or end of summer, they can be used to make almond milk. The Green almond pairs well with the tart flavors of arugula, watercress, and lemon; with lobster, white fish, rabbit, and lamb; and with rhubarb, apricot, asparagus, artichoke, capers, nectarines, and berries. To store, keep Green almonds refrigerated and use within three weeks.
Ethnic/Cultural Info
Almonds have been a food crop in nearly every ancient civilization. The shape of the almond can be found featured prominently in art throughout history. The oval shape of a halved Green almond can be seen in the halo of figures in religious paintings, stained glass windows, and book covers. In Italy, this halo was referred to as the Italian word for almond, "mandorla". Green almonds are popularly used in Persian cuisine to make a traditional stew. In France, Green almonds are often served before spring dinners paired with an aperitif. In Portugal whole, Green almonds are candied. In Iran, the Green almond is sold whole and salted as a popular snack food.
Both peaches and almonds are thought to have evolved from the same ancient rootstock and as the terrain of the continents changed and mountains between China and Central Asia developed, peaches spread into eastern China, and almonds westward. As nomads discovered the fruits of the almond tree and took them with them as they traveled the almond spread rapidly. Evidence of this can be found in the existence of wild trees along trade routes and population centers near many ancient cities. Almond trees first cultivated in the Mediterranean climate regions of Iran, Spain, Greece, Israel, and Rome. In the 1700's the almond tree first made its way from Spain to the United States via Franciscan Padres. Almond trees did not have success right away and it was not until 1850 that pioneers in California would pinpoint the ideal growing conditions that would put the almond on its path to becoming the most popularly produced nut in the United States.
Recipe Ideas
Recipes that include Fresh Green Almonds. One is easiest, three is harder.
Recently Spotted
People have spotted Fresh Green Almonds using the Specialty Produce app for iPhone and Android.
Produce Spotting allows you to share your produce discoveries with your neighbors and the world! Is your market carrying green dragon apples? Is a chef doing things with shaved fennel that are out of this world? Pinpoint your location annonymously through the Specialty Produce App and let others know about unique flavors that are around them.
North Park Produce
Near San Diego, CA, United States
About 10 days ago, 7/19/16
Spotter's comments : Fresh Green Almonds spotted at North Park Produce.
Ansar Gallery International Supermarket Near Tustin, California, United States
About 84 days ago, 5/06/16
Liberty Public Market Near Coronado, California, United States
About 84 days ago, 5/06/16
Spotter's comments : Fresh Green Almonds spotted at Liberty Public Market.
Santa Monica Farmers Market Near Santa Monica, California, United States
About 107 days ago, 4/13/16
Spotter's comments : Fresh Green Almonds spotted at Santa Monica Farmers Market.
Santa Monica Farmers Market
Nate SiemensNear Santa Monica, California, United States
Fat Uncle Farms
912 7th Street Suite B , Wasco 93280
About 121 days ago, 3/30/16
Spotter's comments : Fresh Green Almonds spotted at Santa Monica Farmers Market.
Santa Monica Farmers Market
Nate SiemensNear Santa Monica, California, United States
Fat Uncle Farms
912 7th Street Suite B , Wasco 93280
About 499 days ago, 3/18/15
Spotter's comments : Fresh Green Almonds spotted at Santa Monica Farmers Market.


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^Fingers crossed!
Looking forward to the figure so hoping that is indeed the case.
Type: Posts; User: retcon
^Fingers crossed!
Looking forward to the figure so hoping that is indeed the case.
I am pretty sure not, but have we seen what Darius' finished ankles will be? The old or the "new and improved" version. I know my decision to purchase hinges on the final ankles.
I decided not to get it in the end. With shipping and exchange rate it came to $78...
So he appears to come with thee Alcala sword?
There is quite gap on the neck artic? Or does the camera accentuate it?
Torn. I sold my 200x Hsss set but this would look good with vintage Hsss as the original version is a little "soft".
^ Some amazing pics! Awesome.
Don't know what a Skelecon is so as usual I will be selling/chucking the figure that comes with the vehicle.
Maybe that's what the stone block is for?
To stand in front of the figure and block the view of the ankles?
I kept the Teela, used the original Snake Armour head and threw out the included heads. Not approval from me either.
I think the bigger/sadder change from proto to figure are the overly stubby "horns"....
Interesting as didn't they give 200x subbers EA earlier than the sub beginning?
Wow, I sold off a lot more than I realised. I liked what I kept though:
Point Dread
I don't know - I think it just frustrated me more than anything? Thanks though for taking the time to answer the questions here - it is actually appreciated.
Anyway, sorry, please just ignore me -...
Well I stand corrected thanks to Microsoft Word. My apologies. I even ran a search on "Gray" in my grab of the Bios and it didn't come up...
That said, I still found the "keep reading...
While I will admit it's been great having TG answer some questions here (mine included) I must say the whole "keep reading the bios" thing is really tiresome. Especially as there continue to be...
Hey Irian - I was wondering if you were planning to update this awesome timeline now that the final bios and mini comics are out? There was also that cool Q&A from Toyguru which filled in a few gaps....
I think you have the hood on wrong. It looks like Little Bo Peep.
I'd love it. As per the other vehicles - stick with the vintage design too. It's the missing link of the original MOTU range in Classics.
Could we please get a clarification on Adam's early timeline?
When did he become He Man? Was he adventuring as Adam and wearing OoLarr's vest/harness and carrying the electronic sword? If so, what...
Who was "Ka" - as in Staff of and Shield of.
What is the story behind Teela's snake armour?
Did He Man really wear the laser light armour for 10 years of adventures?
Are there other Time...
Sorry - the old guy killed the queen?
I never quite understood how she died as it was off panel. Asking now as I am doing a re-read of all the minis upon the arrival of the final 2.
Scott - a mini comic question from The Secret Origin of Skeletor.
After the death of the queen, an old man in a hood (who may be bleeding or missing an eye-it's hard to tell) says "W-Where am I?"...
Awesome. Why not switch Scareglow and the Spinning Top guy above? Scareglow's weapon looks too tall for the shelf and the is a bigger space above...?
Dan's back with a new review! This time he takes a look at Multi-Bot from MattyCollector. Check it out!
Just having the figures turn up looking like the prototypes, without paint issues, molding issues, derpy eyes etc. would be "bonus" enough for me.
So essentially I will be paying around $70 for a little plastic sword...?
I think it's another one of these laws/regulations that only Mattel seems subjected to.
In the garbage bin?
The early prototype toys she was blonde too wasn't she? Not sure when the hair changed to red.


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This year Thaipusam committee for KRT Taman-Taman Muhibbah, Sg. Siput was headed by Mr Saravanan, the Treasurer. Mr Saravanan took over the responsibilities from the late Mr Sagaran. He did a good job. All the Indian community in Taman Muhibbah Jaya 2, Taman Muhibbah Aman and Kg. Muhibbah Fasa 2B came together for the event. However, we missed our banner!
The Late Mr Sagaran has left us almost a year. The KRT Taman-Taman Muhibbah, Sg. Siput missed him very much. All the jokes and his easy going character make us enjoy having him in our committee and events. RIP and condolence to his family.
As usual KRT Taman-Taman Muhibbah contributed RM500 for the Thaipusam program by giving out foods and drinks kiosks during the procession. Hopefully this little contributions could unite all the community in Kg. Muhibbah Fasa 2B, Taman Muhibbah Aman, Taman Muhibbah Jaya and Taman Muhibbah Jaya 2.