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"Maiden Lane is a somber but lovely ballad that was composed in memory of the 9/11 tragedy. "Tyner's Corner is a perfect rendering of the great pianist's style, with Zak laying down Tyner-like chords in homage, not imitation. The disc closes with "Below the Rim, where the pianist's deft left hand sets a roadrunner tempo for Foster and Gill to follow. Zak sets off no pyrotechnics here; he and his trio simply opt for conciseness and clarity, hallmarks of all fine composing and playing.
Track Listing: Better Late Than Never; Laughing With RZ; Ugly Beauty; Namely You; Blue Gardenia; Grandpa George; Mamacita; The Worrier; Maiden Lane; Tyner's Corner; Below the Rim.
Personnel: Peter Zak: piano; Paul Gill: bass; Al Foster: drums.
Title: Peter Zak Trio | Year Released: 2005 | Record Label: SteepleChase Records
Oleg Frish: Swing Around The World
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39 Pounds of Love Reviews
November 2, 2010
Can bring a tear to a glass eye.
September 16, 2008
Very good documentary. Ami is an inspiration!
½ May 8, 2008
The "ending" seemed very forced. I doubt that doctor even cared. Fuck that, i KNOW that doctor didn't care... no one did. F-
It's really hard to feel bad for the guy. His animation sucks and he's a total dickhead.
The "ending" seemed very forced. I doubt that doctor even cared. Fuck that, i KNOW that doctor didn't care... no one did. F-
October 17, 2007
English, Hebrew, Spanish
July 7, 2007
The trailer made me laugh and actually made me cry...looks like a great documentary
June 7, 2007
Sounds like an interesting story.
½ December 10, 2006
Extremely boring. Do not bother watching, absolute waste of time.
November 3, 2006
i havent found the storyline yet...
September 28, 2006
This looks really crap.
May 30, 2006
i like this becuase the motercycle is driving supper fas and its a really cool movie and i like it becuase the cars are so cool and hte mottercyle the moterycle goes really fast
½ May 1, 2006
Very touching, very inspirational. Best of all, it's real, baby.
February 5, 2006
it sounds like a touching story!
February 5, 2006
don't like documentaries
January 22, 2006
can't wait to see it....looks quite funny
December 10, 2005
Love it or hate it, you'll never forget it. It is a decision that you really need to make for yourself... See it prepared to laugh and don't forget the kleenex... :fresh:
December 9, 2005
This film made me glad to be alive. Watching Ami overcome his life circumstance made me feel like I could overcome anything. No whimpering allowed!
December 7, 2005
A great uplifting movie. Ami inspires us all to remember our purpose and find gratitude in life and our natural will to life. I encourage all to view the film and be uplifted.


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To determine whether cancer risks for carriers and noncarriers from families with a mismatch repair (MMR) gene mutation are increased above the risks of the general population.
We prospectively followed a cohort of 446 unaffected carriers of an MMR gene mutation (MLH1, n = 161; MSH2, n = 222; MSH6, n = 47; and PMS2, n = 16) and 1,029 their unaffected relatives who did not carry a mutation every 5 years at recruitment centers of the Colon Cancer Family Registry. For comparison of cancer risk with the general population, we estimated country-, age-, and sex-specific standardized incidence ratios (SIRs) of cancer for carriers and noncarriers.
Over a median follow-up of 5 years, mutation carriers had an increased risk of colorectal cancer (CRC; SIR, 20.48; 95% CI, 11.71 to 33.27; P < .001), endometrial cancer (SIR, 30.62; 95% CI, 11.24 to 66.64; P < .001), ovarian cancer (SIR, 18.81; 95% CI, 3.88 to 54.95; P < .001), renal cancer (SIR, 11.22; 95% CI, 2.31 to 32.79; P < .001), pancreatic cancer (SIR, 10.68; 95% CI, 2.68 to 47.70; P = .001), gastric cancer (SIR, 9.78; 95% CI, 1.18 to 35.30; P = .009), urinary bladder cancer (SIR, 9.51; 95% CI, 1.15 to 34.37; P = .009), and female breast cancer (SIR, 3.95; 95% CI, 1.59 to 8.13; P = .001). We found no evidence of their noncarrier relatives having an increased risk of any cancer, including CRC (SIR, 1.02; 95% CI, 0.33 to 2.39; P = .97).
We confirmed that carriers of an MMR gene mutation were at increased risk of a wide variety of cancers, including some cancers not previously recognized as being a result of MMR mutations, and found no evidence of an increased risk of cancer for their noncarrier relatives.
Lynch syndrome is an autosomal dominant inherited disorder of cancer susceptibility caused by germline mutations in the DNA mismatch repair (MMR) genes MLH1, MSH2, MSH6, and PMS2. Estimates of carrier frequency of germline mutations of these genes in the population range widely depending on various assumptions, from approximately one in 3,010 individuals (for MLH1 and MSH2 combined1) to one in 360 individuals (for all four MMR genes combined2). Mutation carriers are at substantially increased risk of cancers of the colon, rectum, endometrium, stomach, ovary, ureter, renal pelvis, brain, small bowel, and hepatobiliary tract, and the diagnoses of these cancers generally occur at younger ages than for the general population.3 Estimates of site-specific cancer risks for MMR gene mutation carriers inform optimal clinical management. Screening colonoscopy,4,5 prophylactic hysterectomy, and bilateral salpingo-oophorectomy6 have the potential to decrease the risk of colorectal cancer (CRC), endometrial cancer (EC), and ovarian cancer, respectively, for MMR gene mutation carriers. All studies estimating the risk of cancer for MMR gene mutation carriers have been retrospective and, therefore, may be biased as a result of differential recall of family history of cancer and failure to adjust for ascertainment of subjects recruited because of a family history of cancer.7 Risk estimates using prospective data of mutation carriers with no prior diagnosis of cancer will not be biased but are challenging because they require long-term follow-up to provide sufficient time for cancers to be diagnosed.
Genetic testing of family members of a mutation carrier will identify relatives who have and who have not inherited the family-specific MMR gene mutation. It is not known whether the cancer risk for noncarriers from families with MMR gene mutations is greater than that of the general population. A family history of CRC increases an individual's risk 1.5- to eight-fold, varying with the number and ages of affected relatives and degree of relationship to the proband.8 In Lynch syndrome families, it is possible that modifier genes participate in the milieu in which cancer manifests.9–16 Noncarriers of MMR gene mutations may share predisposing genetic risk with their mutation-carrying relatives other than that caused by the MMR gene mutation and therefore may be at increased risk compared with the general population. If, however, the MMR mutation accounts for all the excess cancer risks in these families, then noncarriers should be at population risk. In this study, we estimated cancer risks for mutation carriers and noncarriers, who had no prior diagnosis of cancer, from families with MMR gene mutations from the Colon Cancer Family Registry, using a prospective cohort.
This study, using the Colon Cancer Family Registry, included carriers of pathogenic MMR gene mutations and their noncarrier relatives. Details of the Colon Cancer Family Registry have been published previously17 and can be found at the National Cancer Institute Web site (http://epi.grants.cancer.gov/CFR/). Families were recruited between 1997 and 2010 and were ascertained via CRC cases identified from population cancer registries in the United States (Washington, California, Arizona, Minnesota, Colorado, New Hampshire, North Carolina, and Hawaii), Australia (Victoria), and Canada (Ontario) or from family cancer clinics in the United States (Mayo Clinic, Rochester, MN, and Cleveland Clinic, Cleveland, OH), Australia (Melbourne, Adelaide, Perth, Brisbane, and Sydney), and New Zealand (Auckland). Written informed consent was obtained from all study participants, and the study protocol was approved at each center.
At recruitment, baseline information on demographics, personal characteristics, personal and family history of cancer, cancer screening history, history of polyps, polypectomy, hysterectomy, and other surgeries was obtained from all participants. This participant information was updated approximately 5 and 10 years after baseline. Reported cancer diagnoses and ages at which these occurred were confirmed, where possible, using pathology reports, medical records, cancer registry reports, and/or death certificates. The location, histology, and behavior of cancer diagnoses were coded using International Classification of Diseases for Oncology (ICD-O).18 Blood and tumor tissue samples were collected for genetic testing.
MMR gene mutation testing was performed by Sanger sequencing or denaturing high-performance liquid chromatography, followed by confirmatory DNA sequencing. Large duplications and deletions were detected by multiplex ligation-dependent probe amplification.17,19–21 Mutation classification and nomenclature were determined using the InSiGHT Colon Cancer Gene Variant Database (http://www.insight-group.org/mutations), the MMR genes variant database of the Memorial University of Newfoundland (http://www.med.mun.ca/MMRvariants/),22 and the MMR Gene Unclassified Variants Database (http://www.mmruv.info).23 Pathogenic mutations were defined as variants resulting in a stop codon, frameshift mutation, large duplication or deletion, or missense mutation previously reported within scientific literature to be pathogenic.
For this study, we assembled three subcohorts to estimate risk of CRC (CRC risk subcohort), risk of EC (EC risk subcohort), and risk of any other cancer (other cancer risk subcohort; Fig 1). Participants were eligible for all subcohorts if they had undergone genetic testing for their specific family germline MMR gene mutation and were confirmed to be either a carrier or noncarrier of this pathogenic mutation, had been followed up at least once since recruitment, and had no cancer diagnosis before or at the time of recruitment. A total of 81 carriers and 126 noncarriers were excluded from the CRC risk subcohort because they had a polypectomy before or at the time of recruitment. A total of 26 carriers and 46 noncarriers were excluded from the EC risk subcohort because they had a hysterectomy before or at the time of recruitment (Fig 1). For this analysis we did not have information on which participants were aware of their mutation status or when they became aware of their status in part because testing may have been conducted clinically, outside the scope of the study.
Observation time began at completion of the baseline questionnaire and ended at the cancer diagnosis, death, or last follow-up, whichever occurred first. For CRC risk, we censored individuals at the time of polypectomy (except when it occurred within a year before the CRC diagnosis, in which case we assumed polypectomy was for the initial CRC diagnosis), and for EC risk, we censored each woman at the time of hysterectomy.
Observed numbers of cancer diagnoses were divided by the expected numbers of cancer diagnoses to calculate standardized incidence ratios (SIRs). Expected numbers of cancer diagnoses were calculated by multiplying the age-, sex-, and country-specific incidence for the general population by the corresponding observation time in the study cohort. Country-, age-, and sex-specific cancer incidences for the general population were obtained for the period from 1998 to 2002 from Cancer Incidence in Five Continents.24 This 5-year period was selected as the closest available data set with respect to the mean calendar year of cancer diagnoses of the sample, and given that it averaged incidence over the 5-year period, it provides more stable estimates of incidence, especially for less common cancers. For cancer cases from the same family, the jackknife method was used to calculate 95% CIs by allowing for any correlation of risk between relatives from the same family25; otherwise, we used exact methods assuming that observed cancer cases followed a Poisson distribution.
We estimated SIRs for the following cancers observed more than once in carriers and noncarriers: colon and/or rectum (ICD-O C18 to C20), pancreas (ICD-O C25), stomach (ICD-O C16), kidney and renal pelvis (ICD-O C64 and C65), urinary bladder (ICD-O C67), and lung (ICD-O C34) for both sexes; endometrium (ICD-O C54 and C55), ovary (ICD-O C56), and breast (ICD-O C50) for females; and prostate (ICD-O C61) for males.
Kaplan-Meier statistics were used to estimate age-dependent cumulative risk (penetrance) at 5 and 10 years. All reported statistical tests were two-sided and P < .05 was considered statistically significant. STATA version 11.0 (STATA, College Station, TX)26 was used for all statistical analyses.
Selection of eligible carriers and noncarriers of an MMR gene mutation from the Colon Cancer Family Registry for each cancer-specific analysis to estimate risk of CRC, EC, and other cancers is depicted in Figure 1.
This subcohort included 365 carriers and 903 noncarriers from 284 families with MMR gene mutations (Table 1). Of the 365 carriers, 310 (85%) were first-degree relatives, 50 (14%) were second-degree relatives, and five (1%) were third-degree relatives of patients with CRC. Of the 903 noncarriers, 583 (64%) were first-degree relatives, 169 (19%) were second-degree relatives, and 151 (17%) were third-degree relatives of patients with CRC.
Over a median follow-up of 5 years, 16 mutation carriers (MLH1, n = 9; MSH2, n = 4; MSH6, n = 2; PMS2, n = 1) were diagnosed with CRC at a median age of 49 years (incidence rate, 8.84; 95% CI, 5.42 to 14.43 per 1,000 person-years; SIR, 20.48; 95% CI, 11.71 to 33.27; P < .001; Table 2). The CRC SIRs for carriers of specific MMR gene mutations were 39.40 (95% CI, 18.02 to 74.80) for MLH1, 10.76 (95% CI, 2.93 to 27.55) for MSH2, 17.19 (95% CI, 2.08 to 62.10) for MSH6, and 15.47 (95% CI, 0.39 to 86.21) for PMS2.
Five noncarriers were diagnosed with CRC at a median age of 60 years (incidence rate, 1.01; 95% CI, 0.42 to 2.44 per 1,000 person-years). The overall SIR for noncarriers was 1.02 (95% CI, 0.33 to 2.39; P = .97). All of these CRCs were observed in first-degree relatives of patients with CRC. The SIR was 1.43 (95% CI, 0.46 to 3.34) when analysis was restricted to noncarriers who were first-degree relatives of patients with CRC.
Cumulative risks of CRC were 4.14% (95% CI, 2.35% to 7.24%) at 5 years and 8.05% (95% CI, 4.46% to 14.29%) at 10 years of prospective follow-up for carriers and 0.39% (95% CI, 0.12% to 1.20%) at 5 years and 2.04% (95% CI, 0.49% to 8.30%) at 10 years for noncarriers (Fig 2 and Table 3).
The frequency of colonoscopy screening was not different between individuals without CRC (unaffected) and those with CRC (affected). On average, unaffected carriers were screened once every 2.7 years (95% CI, 2.5 to 3.0 years), whereas affected carriers were screened every 2.5 years (95% CI, 1.4 to 3.5 years; P = .63). Unaffected noncarriers were screened every 3.9 years (95% CI, 3.7 to 4.1 years), whereas affected noncarriers were screened every 3.9 years (95% CI, 0 to 8.6 years; P = .98). Colonoscopy during the follow-up period for carriers and noncarriers is shown in Appendix Table A1 (online only).
This female subcohort included 215 carriers and 523 noncarriers from 229 families with MMR gene mutations (Table 1). Over a median follow-up of 5 years, six carriers were diagnosed with EC at a median age of 53 years (incidence rate, 5.66; 95% CI, 2.54 to 12.59 per 1,000 person-years). The corresponding SIR was 30.62 (95% CI, 11.24 to 66.64; P < .001) for carriers of any MMR gene mutation (Table 2). EC was diagnosed in two MLH1 mutation carriers (SIR, 27.18; 95% CI, 6.80 to 108.66) and four MSH2 mutation carriers (SIR, 44.92; 95% CI, 16.86 to 119.68). Cumulative risks were estimated to be 2.84 (95% CI, 1.06 to 7.46) at 5 years and 9.84 (95% CI, 3.45 to 26.33) at 10 years of prospective follow-up (Table 3). No EC was observed in noncarriers.
This subcohort included 446 carriers and 1,029 noncarriers from 300 families with MMR gene mutations (Table 1). Over a median follow-up of 5 years, for carriers, we observed three ovarian cancers (SIR, 18.81; 95% CI, 3.88 to 54.95; P < .001), three renal cancers (two kidney cancers and one renal pelvis cancer; SIR, 11.22; 95% CI, 2.31 to 32.79; P < .001), two pancreatic cancers (SIR, 10.68; 95% CI, 2.68 to 47.70; P = .001), two gastric cancers (SIR, 9.78; 95% CI, 1.18 to 35.30; P = .009), two urinary bladder cancers (SIR, 9.51; 95% CI, 1.15 to 34.37; P = .009), seven female breast cancers (SIR, 3.95; 95% CI, 1.59 to 8.13; P = .001), and three prostate cancers (Table 2). Cancers observed in only one carrier each included cancer of the esophagus (ICD-O C15.9), biliary tract (ICD-O C24.0), liver (ICD-O C22.1), adrenal gland (ICD-O C74.9), small intestine (ICD-O C17.9), and ureter (ICD-O C66.9).
For noncarriers, there was no evidence of an increased risk of lung, breast, and prostate cancers. In noncarriers, we also observed one case each of pancreatic (ICD-O C25.9), urinary bladder (ICD-O C67.9), thyroid (ICD-O C73.9), and esophageal (ICD-O C15.9) cancer (Table 2).
We confirmed that MMR gene mutation carriers were at increased risk of a wide variety of cancers and found that there was no evidence of an increased risk of cancer for their noncarrier relatives. A major strength of our study is the prospective nature of the design, because observation time for carriers and noncarriers commenced before cancer diagnosis. This design, therefore, avoided ascertainment bias commonly present in retrospective analyses, particularly when estimates of cancer risk are made using relatives who are enrolled in cancer clinics because they have been diagnosed with cancer. A further strength of the prospective analysis is that it provides an estimate, based on empirical data, of future cancer risk at the time they come under clinical care. Our estimated 10-year risk of CRC for carriers with a median age of 49 years (8.05%; 95% CI, 4.46% to 14.29%) is comparable to our previous estimate (using an ascertainment-corrected retrospective analysis) of the 10-year risk for MSH6 mutation carriers at age 50 years (6% [95% CI, 3% to 9%] for men and 3% [95% CI, 1% to 5%] for women).27 It seems to be lower than a previous retrospective estimate of the 10-year estimates for MLH1 and MSH2 carriers (26% for men and 13% for women).28
An unanticipated finding of our study is the confirmation of an increased risk for cancers of the breast and pancreas for MMR gene mutation carriers. Although risk of pancreatic cancer has been investigated in Lynch syndrome,29–33 the evidence was inconsistent. The study showing evidence for an association was that of Kastrinos et al,34 which recently showed an increased risk of pancreatic cancer in Lynch syndrome (hazard ratio, 8.6; 95% CI, 4.5 to 15.7). This result is consistent with our finding (test for difference, P = .79). However, given the limited evidence for efficacy of screening for pancreatic cancers,35 expert opinion has not yet advocated pancreatic cancer screening in the context of Lynch syndrome.
A more controversial association with Lynch syndrome is that of breast cancer. Initially raised by Lynch et al36 several decades ago, the issue of breast cancer risk in Lynch syndrome has been debated with evidence for and against this association. Multiple case reports describing MMR-deficient breast cancers, including cases of male breast cancer, have been published.37–41 Walsh et al40 showed that about half of breast cancers observed in MMR gene mutation carries have loss of expression in the tumor of the gene that is mutated in the germline. However, this and other studies were not able to address whether the MMR deficiency caused breast cancer or was a phenotype of a breast cancer caused by another factor. In this report, we observed an increased risk of breast cancer for MMR gene mutation carriers followed prospectively. Although the numbers are small, the risk was estimated to be increased four-fold above that of the general population, suggesting that MMR mutation carriers may benefit from enhanced screening. Further clarification of age-specific magnitude of risk is needed to determine whether ages at screening or methods such as use of magnetic resonance imaging should be modified in Lynch syndrome.
In addition to defining spectrum and cancer risks for MMR gene mutation carriers, it is important to clarify the risk of CRC and other cancers for noncarrier relatives. Identification of mutation noncarriers is considered to be one of the major benefits of MMR gene testing, providing reassurance that these people are at substantially lower risk than their mutation-carrying relatives and, therefore, not subject to the same costly, frequent, and invasive screening and risk-reductionstrategies. Because no cancer arises, to our knowledge, as a result of a single gene mutation, even highly penetrant disorders such as Lynch syndrome might arise on the background of multiple other genetic variants that modify cancer risks. If that is the case, then relatives who do not carry the family-specific pathogenic MMR mutation may still have a cancer risk that is elevated compared with the general population. In this study, we found no evidence of an increased risk of cancers for the noncarrier relatives, whereas carriers of an MMR gene mutation from the same families were at increased risk of a wide variety of cancers. In a similar scenario, it has been observed that there is no evidence of increased risk of breast cancer42,43 or ovarian cancer44 for noncarriers from families carrying mutations of BRCA1 or BRCA2.
Estimating penetrance for CRC is particularly challenging in the high-risk family setting because surveillance colonoscopy is likely to be offered to those with a strong family history of CRC or if Lynch syndrome has been diagnosed in a relative. The colonoscopy likely changes the natural history of the disease by removing the precursor lesion at least some of the time. We observed that those who were mutation carriers (although they may not have known about their mutation status at the time) were having frequent colonoscopies (approximately once every 3 years) yet still manifested an appreciable high risk for development of CRC. In contrast, those who were not mutation carriers (although they also may not have known of their mutation status) were also having colonoscopies almost as frequently as the carriers (approximately once every 4 years). Therefore, we cannot dismiss the possibility that noncarriers who are relatives of mutation carriers are at increased risk of CRC, but their frequent colonoscopies may have reduced their risk.
We also found no evidence of increased risk of extracolonic cancers for the noncarriers. It is unlikely that this finding can be attributed to increased screening for these cancers because, unlike CRC, screening does not alter the risk for extracolonic cancers. For example, screening for urothelial, endometrial, and ovarian cancers does not reduce the risk of these diseases, because it is done for early detection only and not for removal of precursor lesions, and therefore, screening would not influence the estimates of these cancer risks.
There are some limitations to our study, most notably the small number of events and a short follow-up duration. We do not have sufficient power to exclude a two-fold increased risk of CRC for the noncarriers. Another possible limitation is that censoring each individual at the age of polypectomy could have resulted in underestimating the true cancer risk.
In conclusion, our prospective analysis provides unbiased estimates of cancer risks for MMR gene mutation carriers, including significantly increased risks of recognized Lynch syndrome–associated cancers (colorectal, endometrial, ovarian, renal, gastric, and urinary bladder) and of breast cancer and pancreatic cancer. For the noncarrier relatives of family-specific MMR gene mutations, we found no evidence that their risks of CRC or other cancers exceed the population risks.
Supported by the National Cancer Institute, National Institutes of Health under Grant No. RFA-CA-95-011 and through cooperative agreements with members of the Colon Cancer Family Registry and Principal Investigators. A.K.W. is supported by a grant from the Cancer Council Victoria and the Picchi Brothers Foundation, Australia. J.L.H. is a National Health and Medical Research Council (NHMRC) Australia Fellow, and M.C.S. is an NHMRC Senior Research Fellow.
Collaborating centers include the Australasian Colorectal Cancer Family Registry (Grant No. U01 CA097735), Familial Colorectal Neoplasia Collaborative Group (Grant No. U01 CA074799) [University of Southern California], Mayo Clinic Cooperative Family Registry for Colon Cancer Studies (Grant No. U01 CA074800), Ontario Registry for Studies of Familial Colorectal Cancer (Grant No. U01 CA074783), Seattle Colorectal Cancer Family Registry (Grant No. U01 CA074794), and University of Hawaii Colorectal Cancer Family Registry (Grant No. U01 CA074806). We thank all study participants of the Colon Cancer Family Registry and staff for their contributions to this project.
|Colonoscopy During Follow-Up
|CRC Present
|CRC Absent
|CRC Present
|CRC Absent
Abbreviation: CRC, colorectal cancer.
The content of this article does not necessarily reflect the views or policies of the National Cancer Institute or any of the collaborating centers in the Cancer Family Registries (CFRs); in addition, mention of trade names, commercial products, or organizations does not imply endorsement by the US Government or the CFR. Authors had full responsibility for the design of the study, the collection of the data, the analysis and interpretation of the data, the decision to submit the manuscript for publication, and the writing of the article.
Authors' disclosures of potential conflicts of interest and author contributions are found at the end of this article.
The author(s) indicated no potential conflicts of interest.
Conception and design: Aung Ko Win, Mark A. Jenkins
Provision of study materials or patients: Joanne P. Young, Noralane M. Lindor, Katherine M. Tucker, Dennis J. Ahnen, Graeme P. Young, Daniel D. Buchanan, Graham G. Giles, Ingrid Winship, Finlay A. Macrae, Jack Goldblatt, Melissa C. Southey, Julie Arnold, Stephen N. Thibodeau, Bharati Bapat, John A. Baron, Graham Casey, Steven Gallinger, Loïc Le Marchand, Robert W. Haile, John L. Hopper,Mark A. Jenkins
Collection and assembly of data: Aung Ko Win, Noralane M. Lindor, Katherine M. Tucker, Daniel D. Buchanan, Melissa C. Southey, Shanaka R. Gunawardena, Mark A. Jenkins
Data analysis and interpretation: Aung Ko Win, Joanne P. Young, Noralane M. Lindor, Katherine M. Tucker, Dennis J. Ahnen, Graeme P. Young, Daniel D. Buchanan, Mark Clendenning, Graham G. Giles, Ingrid Winship, Finlay A. Macrae, Jack Goldblatt, Melissa C. Southey, Julie Arnold, Stephen N. Thibodeau, Bharati Bapat, John A. Baron, Graham Casey, Steven Gallinger, Loïc Le Marchand, Polly A. Newcomb, Robert W. Haile, John L. Hopper, Mark A. Jenkins
Manuscript writing: All authors
Final approval of manuscript: All authors
Aung Ko Win, Graham G. Giles, Ingrid Winship, Melissa C. Southey, John L. Hopper, and Mark A. Jenkins, The University of Melbourne; Ingrid Winship and Finlay A. Macrae, The Royal Melbourne Hospital, Parkville; Graham G. Giles, Cancer Epidemiology Centre, Cancer Council Victoria, Carlton, Victoria; Joanne P. Young, Daniel D. Buchanan, and Mark Clendenning, Queensland Institute of Medical Research; Joanne P. Young, School of Medicine, University of Queensland, Herston, Queensland; Katherine M. Tucker, Hereditary Cancer Clinic, Prince of Wales Hospital, Randwick, New South Wales; Graeme P. Young, Flinders Centre for Cancer Prevention and Control, Flinders University, Adelaide, South Australia; Jack Goldblatt, Genetic Services of Western Australia and School of Paediatrics and Child Health, University of Western Australia, Perth, Australia; Julie Arnold, New Zealand Familial Gastrointestinal Cancer Registry, Auckland City Hospital, Auckland, New Zealand; Bharati Bapat and Steven Gallinger, Samuel Lunenfeld Research Institute, Mount Sinai Hospital; Bharati Bapat, University of Toronto; Steven Gallinger, Cancer Care Ontario, Toronto, Ontario, Canada; Noralane M. Lindor, Stephen N. Thibodeau, and Shanaka R. Gunawardena, Mayo Clinic, Rochester, MN; Dennis J. Ahnen, Denver Veterans Affairs Medical Center, School of Medicine, University of Colorado, Denver, CO; John A. Baron, University of North Carolina, Chapel Hill, NC; Graham Casey and Robert W. Haile, University of Southern California, Los Angeles, CA; Loïc Le Marchand, University of Hawaii Cancer Center, Honolulu, HI; and Polly A. Newcomb, Fred Hutchinson Cancer Research Center, Seattle, WA.
PubMed Central Canada is a service of the Canadian Institutes of Health Research (CIHR) working in partnership with the National Research Council's national science library in cooperation with the National Center for Biotechnology Information at the U.S. National Library of Medicine(NCBI/NLM). It includes content provided to the PubMed Central International archive by participating publishers.


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Gallup redesigned our website to better serve the needs of our clients.
Nearly 150 million people -- or 4% of the world's adult population -- want to move to the U.S.
Americans' feeling of freedom is declining. This drop -- dramatic in the U.S. -- isn't happening in other wealthy democracies.
Gallup's 2017 Global Emotions Report quantifies how the world is feeling.
1 in 5 people worldwide experienced a lot of anger yesterday.


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Revenge of the Mutant Camels
Atari ST - 1992
Also available on: Commodore 64
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Discover Revenge of the Mutant Camels, an excellent game of 1992 runnable on Atari ST system. With 3rd-person, side view perspective, the publisher Llamasoft Ltd. had wonderful ideas around arcade, shooter.
Captures and Snapshots
Screenshots from MobyGames.com
Screenshots from MobyGames.com
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Atari ST ROM
Commodore 64 Version
- Year: 1984
- Publisher: Llamasoft Ltd.
- Developer: Llamasoft Ltd.
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- 12 Jul 2010
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Responses (1)
Stoxil (Idoxuridine); Synonyms: Oftan Idu
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Kazunori is housed right inside the MAZDA Showroom. An interior area can seat as many as 50 pax, a modern looking sushi bar is separated from the rest of the area by wooden posts, and the front area is separated only by glass from the Mazda showroom with a street view.
KAZUNORI in my neighborhood!
I'm really loving this trend where Chino Roces Avenue Extension (formerly Pasong Tamo Ext.) is fast growing into a row of boutique restaurants run by young, creative chefs as well as good, ol' traditional restaurateurs. From city warehouses and car dealers with fancy showrooms, this long strip is now dotted with promising dining and date places spanning from Karrivin Plaza all the way to that popular Little Tokyo near Makati Cinema Square.
If you're a big fan of chawan mushi, you shouldn't miss it here. I always order this for starter and somehow went by a rough rule that if the chawan mushi is good, the rest listed on the menu should be equally so. I wasn't disappointed. The ikura atop the savory egg custard popped in my mouth, in harmony with the spoonfuls of "taho" goodness that closely followed, as if on cue.
We failed to do justice to the chicken salad with creamy tofu. It was good though we found the tastes too overpowering after a solid chawan mushi with its subtle umami-ness. The other thing is the veggie kakiage looking like a bird's nest was such a scene stealer! We marveled about its presentation and mildly panicked that the freshly-fried bird's nest creation should be devoured asap so as not to miss its crunchiness. You can imagine what happened, of course. Some dishes just can't wait!
By the time we were done with the vegetable and prawn kakiage, our order of pork BBQ waited for our attention. Once more, we nearly ignored the skewered meat because the spicy Karai soba and another soba (shoyu-based) dish were simply right up there in terms of soba quality and the Karai so deliciously spicy. We just kept "going back" to this soba for second and third helpings. If I were to head back here -- and there's no IF but rather WHEN here -- I'd definitely order again the chawan mushi, spicy karai soba and veggie kakiage. Oh, it must be mentioned that the soba set comes with a chawan mushi but we still ordered the premium chawan mushi. The complimentary egg custard that went with the soba set went nearly undisturbed 🙄
Next time, when I'm NOT so hungry or when I'm thinking "snacks", I'd likely order the pork skewers to enjoy with a bowl of rice. Yeah, what's wrong with snacking on rice? 😂 And maybe next time, I'd try their gyoza and sushi. Not to forget --- finish off with green tea ice cream. Sorry, no photos. Devoured soon after the dessert cup landed on the table. Happy eating!
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PENSION LARREA in Donostia
It's a pension or guesthouse right above the bars in Parte Vieja or Old Town in Donostia-San Sebastián. If you want to do a serious pintxos bar crawl, locate yourselves in this area. You can eat and drink to death and never mind that the crowd can be noisy especially on weekends.
The rooms are clean, though spartan. But the real attraction here is Amparo. She is a widow, lives alone, tidies up the place squeaky clean, and if she likes you, may even cook white asparagus and artichokes for you. As she did for mi sobrina y nieto. Our friends and family have stayed with Amparo a total of 3 times. And Amparo remembers. 💕
This is Amparo. True Basque, through and through. She hardly speaks English but her hand gestures and facial expressions will suffice to make her understood. If you read the reviews, those who've stayed here were obviously charmed by Amparo. She welcomes you, and easily makes you a friend. Not a guest, but a dear friend. She has a good memory and remembers her guests very well. Tells you how interested she is in people. If you're not looking for luxury but just a modest, tidy, safe and excellently located guesthouse, this is it.
Daci & Daci Bakers (Hobart)
Read about it. Good reviews. So that when we passed it, we went in to check out the place. Oh, there were many choices. And everything looked good!
We settled on a lamb stew pie and some kind of frittata with kale. Both served with green salad. Plus a flat white and a latté. Very filling. Tempting to enjoy some desserts but no space in our jolly bellies!
Honestly? I wouldn't mind eating here again. There's so much to try, each looking so delectable. Each piece served with fresh salad. And I just can't complain about the coffee and the service too. Yeah, check it out. From the Waterfront, just walk up Murray Street. It should be on your right walking up. Buen Provecho!
Restaurant 58 Tour Eiffel
They change the menu twice a year. Picnic lunch is available but a romantic dinner maybe a better deal if you like the idea of an illuminated Eiffel Tower to enjoy the city lights as dusk falls.
A flute of champagne to welcome the entree of either smoked salmon or Foie Gras. There are other choices too. Linger over the menu at your own risk. Agonize. Suffer. But it's hard to go wrong with any from this well-curated menu. It maybe the touristy thing to do but hey, who'd pass up the chance to dine here around a table by the window overlooking the city?
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A ROOM IN THE CITY (San Sebastián)
The last time I was out here with friends, we booked an apartment via Airbnb. One txikiteo night, we also got a room in a Pension in Parte Vieja. Perfect after a pub crawl, but quite noisy too as the Pension is right above the Pintxo bars where a crowd drink txakoli to death till the wee hours of the morning.
I like A Room In The City. The young men behind the Reception Desk may not be the friendliest but they're cool and efficient. I borrowed some photos from the Net here just so I can show you how strategic the location is in a more quiet part of the city. (Calle Easo, 20) It is located right next to a chapel too. In fact, you can spot some of the church pews in the Sun Deck while viewing the pleasant neighborhood which includes the Buen Pastor Cathedral just around the bend.
Apart from the Sun Deck, the dining hall and sitting lounge are pleasant additions. Much better than sitting in hotel lobbies. In the dining hall, one can partake of free coffee, tea, biscuits, nuts. There is also a vending machine where you can buy chips, chocolate bars and drinks. Then there's the kettle and microwave to heat up water and food. All in a very spacious hall where everyone washes the dishes they use and wipe the tables clean. Neat!
Next time I get here with family and friends, I'd likely book a dormitory good for 4 or 6 pax on bunk beds. Sounds like fun especially with young ones. Then we'd spend more time in the lounge or the Sun Deck and laugh at each other's jokes. Some croissants bought from the nearby boulangerie cum patisserie, warmed briefly in the microwave, along with my favorite morning brew. Swell. And before we hit the Pintxo bars at night, we'd have some cerveza on the deck with some aceituna and chistorra bought from the nearby deli. Oh, life is good. 💕
Imagine how my heart broke, going to La Viña in San Sebastián and finding it closed for 4 consecutive days. 😭😭😭Imagine how I grew desperate checking on other bars and restos in San Sebastián, hoping their version of tarta de quezo comes close to La Viña's. Imagine how I felt upon learning La Viña has reopened as soon as I've left San Seb and arrived back in Madrid! 😡
Thank God for friends who visited San Sebastián soon after we've left and brought back --- drumroll, please --- an entire cake for us to devour in Madrid. 🤗😋😀 All that creamy, yummy, burned on top 🍰🍽🎂goodness packed in a red box! You don't know happiness until that first bite.
When in San Sebastián, you'd do your txikiteo to complete your Basque experience. In Parte Vieja along Calle 31 Agosto, just a stroll away from Yglesia de Santa Maria, Atari Gastroteka, La Cepa and La Cuchara de San Telmo, is this culinary gem. Your Basque guide won't be lying if he says it's the world's best cheesecake! Seriously.
The first time I was here, I was too full from a La Cepa dinner to do a serious Pintxo bar crawl. I knew better this time. I started with Bar Zeruko. And almost got stuck here. The '2 pintxos and a drink' rule can't, won't apply. Not content with what's on display at the bar, we ordered from the kitchen. Must be the txakoli. So white and clear and young and Basque!
The fruits from the sea were great starters. La Hoguera literally translates to "bonfire" and La Hoguera de Bacalao is a special Pintxo here where your smoked cod is served "smoking" until it's ready to rest on a toast smothered with some herbal cream and a vial of warm, liquid salad dressing. Such effort in presentation! And how about this bowl of baby eels and creamed uni with a dash of perejil sauce? Yum.
I chose another bacalao dish -- this cod being one of my personal favorites. It looked interesting on display at the bar, bottled with a green pepper and some cheese at the top. Plated, it looked kinda ordinary, methinks. But it was full of flavor and I had to stop myself from having seconds.
The chistorra came wrapped and was served with a mildly spicy tomato sauce. The mushroom pintxos were good but not what I'd order next time. I was likewise intrigued with the soft boiled egg encased in a gelatinous meat stock and served on a bed of Migas or bread crumbs flavored with chorizo. Every dish is so creatively done. How delightful!
My niece and I shared these pintxos and decided we should start moving. Txakoli on hand, we cheered we had a good start. Bar Zeruko rocks!
Restaurant Iruñazarra
There are many dining options in Pamplona's Plaza Del Castillo and the streets and alleys around it. We found this one.
The pig's cheeks and white asparagus salad were superb. I'm a sucker for white asparagus and will have them whenever they're in season. The pig's cheeks dish was cooked a la estofado but not quite. Served with steaming rice!
If you're here, you have the choice of a sit-down meal or a bar set-up where you can enjoy your Rioja and some pintxos. Both are very good. I was intrigued by many tapa or Pintxo varieties but only tried one. No room for more. 😭
But what I had, I liked! The Iberico con quezo with honeyed walnuts was sooo good. Somehow, I regretted not sitting at the bar. I could have tried some others I haven't had the chance to figure out.
Buen Provecho!
We only stayed 2 nights here. Both nights we dined in this place. No regrets. The variety may be limited but with only 2 people running the restaurant, I'm amazed how they manage to keep customers happy with prompt service and food quality.
My sister is a meat person while I prefer vegetables and fish. The Entrecôte, served with salad and potatoes made my sister happy. Another time she had chicken roasted in its juice which was equally good. I liked the chicken too, savory and very tender.
But we both agree the soup is the best. I was happy with their generous portion in a big bowl, filled to the brim with potatoes, carrots and leafy veggies. The beef and vegetable broth was served piping hot, perfect with the bread.
Btw, Hurrup Eta Klik translates to SUCK ON. How about that, eh? 🤣🤐😳
Can't resist putting in a few words on our charming hotel in Saint Jean Pied de Port. It wasn't my first choice, but have absolutely no regrets booking 2 nights here where pilgrims start their Camino Frances.
Hotel Itzalpea is right outside the walled area, just a few steps from the gate leading to Rue de France. You can view it from the slits if you're walking on the ramparts of the muralla.
The interiors are very simple. After all, it only has 3 floors. We were lucky to have the room with a view of the street and the walls. Not noisy like other hotel rooms on the street side. Breakfasts can be had for €9 a set , which was quite enormous! No hot meals but ham, marmalade, jams, yogurt, bread basket, pie, pots of coffee, tea or chocolate.
The rooms go for about €70 a night, twin. Good-sized, clean and well-appointed. It wasn't pilgrim season yet when we arrived, so expect a crowd by the front where they set up tables under the sun.
I'd book this hotel again when I head back. Very charming.
November 2015


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Should I add liquid if my compost looks dry
Friday, 6 January 2012 | HOTBIN Composting
As a rule, do not add liquids (water, urine, soups, gravy) to the HotBin.
Occasionally, small areas of the compost in the centre of the bin may appear dry with a white/powdery fungus on. This is a sign of 'preferential' drying through oversize air channels. Rake new waste into the dry area and then gently push down the waste to close the air channels. After adding more waste, recheck the temperature and waste after 48 hours, if the waste still appears dry, add no more than one cupful of hot water (the hotter the better).
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A classic teaching aid with a fun twist!
Young swimmers will be motivated to power through the pool with Spark Shark or Dart Dolphin to help! Beginners can hold onto this tough, fabric-coated kickboard as they gain confidence; older swimmers will love using it for games and exercise in the water. Both beginner and advanced swimmers with improve their kicking technique and strength.
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Job Posting: Hartmann Fine Art Conservation Services, Inc.
JOB TITLE: Senior Paintings Conservator
PRIMARY LOCATION: Hartmann Fine Art Conservation Services, Inc. conservation facility in Carlisle, PA
REPORTS TO: Chief of Conservation
STATUS: Full-time
APPLICATION PERIOD: Will remain open until filled
Hartmann Conservation is seeking a driven, self-motivated professionally trained Paintings Conservator with strong management, leadership, oral and written communication, artifact assessment, and advanced conservation treatment skills.
Job Posting: Hartmann Fine Art Conservation Services, Inc.
JOB TITLE: Assistant Paintings Conservator
PRIMARY LOCATION: Hartmann Fine Art Conservation Services, Inc. conservation facility in Carlisle, PA
REPORTS TO: Chief of Conservation and Senior Paintings Conservator
STATUS: Full-time
APPLICATION PERIOD: Will remain open until filled
Hartmann Fine Art Conservation Services, Inc. ("Hartmann Conservation") is seeking a driven, self-motivated, experienced, professionally trained Conservator with 3 – 5 years of working experience in the field of Paintings Conservation.
Job Posting: Artex, Painting Conservator
Painting Conservator, 0030-17
Department: DC – Office
FLSA Status: Hourly
Artex is the nation's largest and most comprehensive fine art service provider to museums, galleries, auction houses and private collectors. We specialize in the safe handling of fine art, antiques and museum objects of all kinds. We provide transportation, crating and packing, storage, installation, rigging, and conservation services to museums, galleries, and auction houses as well as private and corporate collections.
Kress Fellowship- UVa Library
The University of Virginia Library seeks a thoughtful and talented candidate for a nine month Graduate Conservation Fellowship at the University of Virginia Library. The Fellowship is made possible by a generous grant from the Kress Foundation.
Post Doc on Detection of Unstable Glass
The George Washington School of Engineering and Applied Science Department of Biomedical Engineering (BME) is seeking a full-time Post-Doctoral Scientist to help with an NEH-funded research project entitled, "Glass at risk: simple tools for detecting unstable glass in 19th century cultural heritage collections," to develop, apply, and simplify a set of imaging methods to characterize glass and its degradative processes.
Job Posting: US Senate
The Office of Senate Curator has an opening for a collections specialist. This is professional, technical, and administrative work supporting the registration and collections management programs for the Office of Senate Curator on behalf of the Senate Commission on Art. Responsibilities include implementing standards, policies and procedures for the registration, inventory, movement, placement, protection, storage and care of the Senate's permanent collections of art, historical furnishings, and memorabilia.
Temporary Position at the MAC Lab
Conservation Project at the Maryland Archaeological Conservation Laboratory
Maryland Department of Planning/Maryland Historical Trust/Jefferson Patterson Park and Museum
Opening Date: March 20, 2017
Closing Date: April 10, 2017
Salary: Not to exceed $7,200; $15/hour
Employment Type: Temporary
Work Location: St. Leonard, Calvert County, MD
Conservator Position at the US Department of State
The State Department is looking for a conservator with a broad background who can do some hands on work, but mostly be able to identify conservation needs, write scopes of work, work with conservators around the world to complete conservation work, etc.
Photo Conservator Position at NMAAHC
Full-Time, Two- year Term with possible extension up to four years
Salary range: $64,650.00 to $84,044.00 / Per Year
Job Announcement Number: 17R-YH-302361-DEU-NMAAHC
Term Positions at Winterthur
Winterthur Museum, Garden & Library seeks two part-time conservation assistants for a two year grant-supported project to clean and lacquer or re-lacquer approximately 500 pieces from its extensive collection of early American silver. Working under the supervision of Winterthur staff in objects conservation, both conservation assistants will first receive training in the appropriate procedures used to remove failed coatings from silver and silver plated objects, gently clean the surface, remove all polish residues, and then lacquer the silver objects through brush or spray application.


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Ministers sent violent inmates to open jails
Tony McNulty, the beleaguered minister at the centre of the foreign convict scandal, admitted in a letter that overcrowding in secure prisons had led to "open prisons being asked to take prisoners who would perhaps have not previously been allocated there".
McNulty was last week stripped of his responsibility for immigration and switched to handling police affairs.
Prison governors also revealed this weekend that they had come under "inappropriate pressure" from Home Office officials to send prisoners to open jails, even though they were judged likely to escape.
Those who subsequently absconded have been responsible for a series of crimes — including violent robberies, sexual assaults and false imprisonment.
The shift in practice by the Home Office has never previously been publicised and only came…


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Jon Fjortoft: Photographs
Jon Fjortoft: Photographs
78 E. Washington St.
Chicago, IL 60602
Chicago, IL 60602
April 3rd, 2010 - June 27th, 2010
Opening: April 9th, 2010 6:00 PM - 8:00 PM
This exhibition looks at two bodies of work by Jon Fjortoft, a largely self-taught, Chicago-based photographer.
First, his landscape portraits of manufacturing plants in the suburbs show pared down compositions and strange landscapes that emphasize the surreal quality of these relics. These are accompanied by Fjortoft's "street photographs" that use his strong sense of formal composition to reveal keen observations of people in downtown Chicago.


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Hello and Welcome to Jes Hooper's Monthly Maker: THE place to discover artistic innovators and creative entrepreneurs! Every first Monday of the month I'll be introducing a new artist to my website blog to share their craft, inspirations, and aspirations. Why? To showcase the talented small business' who are paving the way to success in our creative communities.
This month is the second installment of Monthly Maker and I've been chatting with Julia Smith from Julia Smith Ceramics all about her love of clay, the thought process behind building a innovative home studio, and what she has in the pipeline for 2017.
I immediately fell in love with Julia's Ceramics, her use of glass-like glazes and earthy tones brings a richness to her work unlike any other. I also truly admire her unique illustrations which she applies sparingly to give a subtle yet wholesome meaning to each piece; I hope you all enjoy her work as much as I do.
What first inspired you to create ceramics over other artforms?
I was attracted to the raw material and was interested in the changes clay goes through from a soft wet malleable substance to dry and brittle then stone like after firing. The alchemy of the glazes intrigued me too, seeing dry powdery pastel coatings transformed to shiny colourful glassy surfaces when fired in the kiln, it seemed like magic to me.
My studio looks out over the Moray Firth and the changing weather, light on the water, wildlife, horizon, mountains across the water, pebbles and driftwood on the beach all have influenced my work since I moved here 6 years ago. My garden, bike rides, forests and winter walks also give me ideas.
I studied ceramic design at Glasgow School of Art and graduated in 1996. I feel self taught as most things I learn through trial and error in the studio.
My parents were very creative when me and my siblings were growing up, possibly for economic reasons. My Dad built both the houses we lived in, designed and built his own racing car and my Mum would buy furniture from the auction rooms and fix it up to sell on and make wonderful costumes for fancy dress parties. It took me a while to figure out what I wanted to do but I knew I wanted to go to art school and this wasn't discouraged thankfully.
What is your favourite and least favourite aspects of your creative process?
My favourite part of my creative process is when I discover a new glaze or combination of glazes that I really like. I usually have to do a lot of experimenting before I find a keeper so it is satisfying when it works. My least favourite aspect is paper-work and cleaning out the waste trap below my sink.
I have lived here for 6 years and each year I have added or renovated a new space for my ceramics and now I finally have enough space (I'm not allowed any more). I work at home and have a main studio in the garden where I do all the clay work, a small hut for storing and recycling clay, a kiln room in the garage, a glaze studio and office/ packing room in the house and a small wood workshop outside for building displays and maintenance jobs.
For my main studio space I wanted natural light, running water and lots of shelving to move work around through the stages of making. I have a small kiln in my studio which keeps me warm through the winter and dries pots out ready for firing.
It is such a big part of my life, when I am not making I am thinking about what I am going to make. The highlight for me is having fantastic customers who allow me to do this everyday.
I have had insightful and helpful business support from The Design Trust (http://www.thedesigntrust.co.
uk) and have been lucky to have access to mentoring and training through Emergents (http://www.emergents.co.uk) in the Highlands. I have learned a lot about marketing, social media, working out pricing and thinking about long term plans with their help and they also helped me feel more confident about my work.
I am getting work ready for an exhibition at the Craft & Design Centre in Leeds, 'Branching Out", which opens in the spring then I am off to Iceland in March for the DesignMarch festival. There are a group of makers going from the Highlands and we will be meeting up with Icelandic makers to create collaborative pieces for an exhibition in Iceland next year. In April I will be running my first block of evening classes in the new studio so looking forward to that. I have just been applying for shows for later in the year so will wait to hear back, I will add information to my website as news come in.
A huge thank you to Julia for taking part in my MONTHLY MAKER blog project. As someone who has not worked with clay before (and someone who is obsessed with tactile art forms) I found Julia's approach to ceramics and her appreciation for the medium really inspiring. I can't wait to see what her new trip to Iceland will bring to her works!
You can find Julia online here:
Next month on MONTHLY MAKER I speak to Paul Johnston of PJWoodcraft. Paul specialises in wood turning (another artform that I am desperate to try in the near future!).
We speak all about our shared love for raw timber and working from logs to reveal the hidden mysteries inside! Make sure to check back on Monday 3rd April to read more.


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BRONSON - Many Levy County workers enjoy a form of compensation, which cushions the impact of rising fuel prices. They drive county vehicles to and from home, and the gasoline or diesel is included.
Not all employees are willing to relocate here, County Coordinator Freddie Moody said. For the county to hire qualified staff, Moody said, an offer including transportation is part of the pay. Two County Commission workers received this incentive, Moody said. They are County Planner Shenley Neely and Nature Coast Transit Director Desiree Painter. Neely lives in Alachua County and Painter lives in Taylor County, he said.
There are 53 people who either drive or ride to and from home in county vehicles on the County Commission staff, according to Human Resources Manager Jacqueline Martin. Some live near to the courthouse in Bronson. Others live in Yankeetown, Inglis, Chiefland and other parts of Levy County, and in Alachua, Marion, Taylor or Gilchrist counties, Moody said.
Only four department heads do not use a vehicle to get between home and work. They are Veterans Service Office Director Michael Engel, ibrary Director Bonnie Tollefson, Levy County Visitors' Bureau Director Carol McQueen and County Attorney Anne Brown, Moody said.
Moody recently sent a memorandum to department heads asking them to consider if any of them or their workers could relinquish this benefit to reduce costs for county taxpayers.
Workers for the Levy County Board of County Commissioners are not the only group with this allowance. The Levy County Sheriff's Office has a higher number of employees going home in county vehicles, although their circumstances for using cars differs from most other county satff.
Sheriff Johnny M. Smith Jr. uses a county vehicle to go to and from home, as well as in his duties of preventing and fighting crime. A total of 76 LCSO vehicles go home, Lt. Evan Sullivan said. He was quick to note the nature of business by law enforcement requires cars.
Deputies are on call for Special Weapons And Tactics missions, dive team duties, and they can be called out instantly for any natural disaster or civil disorder, Sullivan said. If an off-duty officer sees a crime on his way to begin his or her shift or on his or her way home, Sullivan said, he or she could take action as well.
One constitution officer has no county vehicles in her department. Tax Collector Linda Fugate does not have any county vehicles.
One constitutional officer has access to a county vehicle for travel between home and work, but he chooses to walk to and from work instead.
Property Appraiser Francis Akins' would be allowed to use a 1997 Isuzu Rodeo to drive to and from home, he said. Akins lives right across the street from the Levy County Courthouse, however, so he sees no need to drive the vehicle except on official business.
Clerk of Court Danny Shipp has a county vehicle that he takes home, as well as uses for official business. Supervisor of Elections Connie Asbell uses a county vehicle to take her to and from her home, as well as all over the county for election duties.
Superintendent of Schools Cliff Norris takes his vehicle home, School Board Finance Director Robert Clemmons said. The superintendent is on call 24-hours-a-day, seven-days-a-week, Clemmons added.
School Board Maintenance Director Ted Alexander said four vehicles go home with workers, because they are on call. Men in Chiefland, Bronson, Williston and Cedar Key on call to respond to maintenance needs from break-ins, ruptured water or sewer lines, and other maintenance duties after daytime business hours.
School Board Transportation Director Steve Tyson said 75 buses go home with their drivers because it is the most efficient method to meet the transportation needs for the district. Of those 75 bus routes, they all begin at the drivers' homes, he said. Four of the 79 total buses in the district are stored at schools, because drivers for those routes live too far off of them to park at their homes, Tyson said.
Employees who are allowed to use county vehicles for personal transport to and from home are given a value added to their tax return, Clemmons said. It is relatively small, about $3 per day. Moody said the same is true for County Commission workers who receive this benefit.
To put a perspective on the increasing value of having a vehicle to transport a person to work, without an increasing expense to the person from the cost of gasoline going up, one need only look at the U.S. Department of Energy's record of weekly retail gasoline prices.
On March 17, 2007, according to the Department of Energy (DOE), the cost of one gallon of regular grade gasoline on the East Coast was $2.57. On March 18, 2008, the same quality and quantity of gasoline sold in the same region of the United States for $3.28, according to the DOE.
The upsurge in gasoline prices over those 12 months is almost a 28 percent increase.
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text: Taxon DOI 10.1601/tx.8745 Name Nocardiopsis salina Li et al. 2004 emend. Li et al. 2006 Taxonomic Rank species Methodology Nomenclatural Taxonomy [1980-2016] Incertae sedis taxa are placed using SOSCC (Garrity and Lilburn 2008) Parent Taxon RecordName Record genusNocardiopsis (Brocq-Rousseau 1904) Meyer 1976 (Approved Lists 1980) Type Strain representative organism YIM 90010T (=KCTC 19003 =CCTCC AA 204009) Digital Object Identifier https://doi.org/10.1601/tx.8745 View the complete NamesforLife Abstract Viewing the full Abstract requires login with a valid NamesforLife account and agreement to the NamesforLife Terms of Use. By signing in, I confirm that I have read and agree to the NamesforLife Terms of Service and Privacy Policy. e-mail address password Don't have a NamesforLife account? Create one for free here. Forgot your password?