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It is that time of year again. Happy Father's Day to all the men who are connected to The Community Fellowship! We will begin a new series at The Community this Sunday that will focus on our homes and encourage men to stand tall and strong through the truth God gives.
The series is called COURAGEOUS and builds off some of the themes found in the recent movie with the same name. Pass the word. Invite friends. Invite men to join us.
There will even be a few surprises and some prizes too. See you Sunday, and join me in praying for men in our community who need Jesus and for those who want to follow Him more.
With GOD as my guide,
Pastor Michael Harrison
James 1:5
for more information about The Community Fellowship or The Community Dream Center, reply to this email or call 276-647-8231. To donate or give online click on this link: E-GIVING


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These tasty stuffed tomatoes are a brilliant idea for alfesco Christmas lunches.
Featured in Christmas sides, Christmas starters
- 12 vine-ripened tomatoes (stem attached)
- Ice for an ice bath
- 1 zucchini, cut into 1cm cubes
- 1 baby eggplant, cut into 1cm cubes
- 1 small red capsicum, cut into 1cm cubes
- 1/3 cup (80ml) olive oil
- 3 garlic cloves, chopped
- 150ml balsamic vinegar
- 3/4 cup (150g) couscous
- 1 cup (250ml) Massel chicken style liquid stock, boiling
- 4 spring onions, thinly sliced
- 2 tablespoons chopped basil
- 2 tablespoons chopped flat-leaf parsley leaves
Step 1Preheat oven to 180°C. Line a baking tray.
Step 2Cut a small cross in the base of each tomato. Plunge each into boiling water for 30 seconds, then remove and refresh immediately in iced water. Peel, then chill for 15 minutes (to firm up). Cut off and reserve the tops. Carefully hollow out tomatoes with a teaspoon, season with salt and pepper, then leave to drain upside-down on paper towel.
Step 3Meanwhile, toss the vegetables in 2 tablespoons oil with garlic. Season, then spread onto the lined baking tray. Roast for 15 minutes, turning once, until slightly charred. Toss with balsamic vinegar.
Step 4Add the couscous, 1 tablespoon oil and 1 teaspoon salt to the hot stock. Cover and stand for 10 minutes. Fluff up with a fork, then stir through vegetables, spring onion and herbs. Season with salt and pepper.
Step 5Fill tomatoes with couscous. Return the tops and drizzle with remaining oil. Serve at room temperature or return to the oven for 10 minutes until warmed through.


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Pediatrics Unit
Pediatrics Unit
News Article | May 8, 2017
Site: www.prnewswire.com
On Thursday, Suárez will tour the local Pediatrics Unit at The University of Kansas Health System, a Children's Miracle Network Hospital, and meet many of the young patients admitted there. While visiting, Suárez and representatives from STANLEY, will present the $100,000 donation to CMN Hospitals on behalf of the Ace Hardware Foundation. The team will also unveil the paint scheme that will run this weekend on the No. 19 STANLEY Toyota. The paint scheme includes names of one child from each state who represents Children's Miracle Network Hospitals, as well as four very special children who will serve as "Honorary Pit Crew Members" of the No. 19 team throughout the weekend. "I've always admired this program," says Suárez. "But I have a whole new level of respect now that I'm partnering with the STANLEY team. I am blown away by the courage and determination of the kids treated at CMN Hospitals and I am more motivated than ever to win this race." This is Suárez's first year racing in the Monster Energy NASCAR Cup Series with Joe Gibbs Racing and the STANLEY race team. The 25-year-old Monterrey, Mexico-native is the first Mexican-born driver to capture a NASCAR national series victory, and the fifth Latino driver to win a NASCAR national series race. Follow Suárez this weekend at #RACINGforMIRACLES. For more information on the "Racing for a Miracle" program, including more ways to support Children's Miracle Network Hospitals, visit http://www.stanleyracing.com Stanley Black & Decker, an S&P 500 and FORTUNE 500 company, is the world's leading provider of tools and storage, the world's second-largest commercial electronic security company, and a leading engineered fastening systems provider, with unique growth platforms in the Oil & Gas and Infrastructure industries. Well-known brands include: STANLEY, BLACK+DECKER, DEWALT, Craftsman, Porter-Cable, Bostitch, Facom, Mac Tools, Proto, Vidmar, Lista, and more. The company's STANLEY Healthcare Solutions division is the North American leader in Infant Security, with its Hugs(TM) Infant Protection System. Learn more at http://www.stanleyblackanddecker.com. Children's Miracle Network Hospitals® raises funds and awareness for 170 member hospitals that provide 32 million treatments each year to kids across the U.S. and Canada. Donations stay local to fund critical treatments and healthcare services, pediatric medical equipment and charitable care. Since 1983, Children's Miracle Network Hospitals has raised more than $5 billion, most of it $1 at a time through the charity's Miracle Balloon icon. Its various fundraising partners and programs support the nonprofit's mission to save and improve the lives of as many children as possible. Find out why children's hospitals need community support, identify your member hospital and learn how you can Put Your Money Where the Miracles Are at CMNHospitals.org and facebook.com/CMNHospitals. As the official charitable division of Ace Hardware Corporation, the Ace Hardware Foundation helps enhance the vision of being the "Helpful Place" in local communities across the country and around the globe through charitable giving. The Ace Hardware Foundation provides support and philanthropic opportunities to its consumers, retail store owners, vendors and team members to give back to local communities through fundraising efforts for Children's Miracle Network Hospitals and American Red Cross Disaster Relief. For more than 90 years, Ace Hardware has been known as the place with the helpful hardware folks in thousands of neighborhoods across America, providing customers with a more personal kind of helpful. With more than 5,000 hardware stores locally owned and operated across the globe, Ace is the largest retailer-owned hardware cooperative in the world. Headquartered in Oak Brook, Ill., Ace and its subsidiaries operate an expansive network of distribution centers in the U.S. and also have distribution capabilities in Ningbo, China; Colon, Panama; and Dubai, United Arab Emirates. Its retailers' stores are located in all 50 states, the District of Columbia and approximately 60 countries. For more information on Ace, visit acehardware.com or the company newsroom at newsroom.acehardware.com. KU Pediatrics, in conjunction with The University of Kansas Medical Center and The University of Kansas Health System, provides the medical care and leading-edge research needed to treat children today and find the treatments for tomorrow. Since 1985, KU Pediatrics has been partnered with Children's Miracle Network Hospitals to raise funds and provide families with the best care possible. Through various partners and events, funds raised stay local and are utilized for immediate medical care, equipment and pediatric research initiatives. To view the original version on PR Newswire, visit:http://www.prnewswire.com/news-releases/daniel-suarez-and-no-19-stanley-team-race-for-a-miracle-this-weekend-300453174.html
Rinaldi B.,University of Pavia |
Amarri S.,Pediatrics Unit |
Baldo C.,Galliera Hospital |
Gobbi G.,CNR Institute of Neurological Sciences |
And 9 more authors.
Orphanet Journal of Rare Diseases | Year: 2017
Background: Ring chromosome 14 syndrome is a rare chromosomal disorder characterized by early onset refractory epilepsy, intellectual disability, autism spectrum disorder and a number of diverse health issues. Results: The aim of this work is to provide recommendations for the diagnosis and management of persons affected by ring chromosome 14 syndrome based on evidence from literature and experience of health professionals from different medical backgrounds who have followed for several years subjects affected by ring chromosome 14 syndrome. The literature search was performed in 2016. Original papers, meta-analyses, reviews, books and guidelines were reviewed and final recommendations were reached by consensus. Conclusion: Conventional cytogenetics is the primary tool to identify a ring chromosome. Children with a terminal deletion of chromosome 14q ascertained by molecular karyotyping (CGH/SNP array) should be tested secondarily by conventional cytogenetics for the presence of a ring chromosome. Early diagnosis should be pursued in order to provide medical and social assistance by a multidisciplinary team. Clinical investigations, including neurophysiology for epilepsy, should be performed at the diagnosis and within the follow-up. Following the diagnosis, patients and relatives/caregivers should receive regular care for health and social issues. Epilepsy should be treated from the onset with anticonvulsive therapy. Likewise, feeding difficulties should be treated according to need. Nutritional assessment is recommended for all patients and nutritional support for malnourishment can include gastrostomy feeding in selected cases. Presence of autistic traits should be carefully evaluated. Many patients with ring chromosome 14 syndrome are nonverbal and thus maintaining their ability to communicate is always essential; every effort should be made to preserve their autonomy. © 2017 The Author(s).
PubMed | Institute Investigacion Biosanitaria Of Granada, University of Granada, University of Cordoba, Spain and Pediatrics Unit
Type: Journal Article | Journal: Pediatric research | Year: 2015
Our aim is to study the prevalence of subclinical celiac disease (CD) and analyze the diagnostic yield of a new rapid test in children aged 2-4.We carried out a cross-sectional study in a sample population of children aged 2-4 from the same metropolitan area. We recruited apparently healthy subjects, and collected clinical, anthropometric, analytical, and serological variables. We also tested for anti-gliadin IgA and anti-transglutaminase IgG and IgA using a rapid immunochromatographic test CD1WB and CD2WB (Operon, Zaragoza, Spain).One hundred and ninety-eight children were recruited, signed the informed consent form, and completed the protocol (mean age 32.39.2 mo, 53% males). CD prevalence according to the serological tests was 3% (CI 95%, 1.4-6.4%). Biopsies were used to confirm the diagnosis in all suspected cases. The sensitivity and negative predictive value of the CD2WB immunochromatographic test strip were 100% and 1, respectively. The sensitivity of CD1WB was 16.6% and its specificity was high (89.1%).The prevalence of subclinical CD in the sample group of 2-4-y old was higher than that found by other authors. The CD2WB immunochromatographic test strip is an excellent diagnostic screening tool with high sensitivity and negative predictive value.
Navarro-Jarabo J.M.,Gastroenterology Unit |
Ubina-Aznar E.,Gastroenterology Unit |
Tapia-Ceballos L.,Pediatrics Unit |
Ortiz-Cuevas C.,Radiology Unit |
And 3 more authors.
Journal of Gastroenterology and Hepatology (Australia) | Year: 2013
Background and Aim: Obesity is an important health-care problem in developed countries. It is considered a multisystemic disease, but it may also affect the liver, thus provoking non-alcoholic fatty liver disease. This disease has been less extensively studied among children than among adults. We propose to analyze the prevalence of hepatic steatosis among a pediatric population within an area in southern Europe besides the variables associated with its development and severity. Methods: Cross-sectional study carried out on a population of children aged 6-14 years inclusive, using abdominal ultrasound as a method to determine the presence and severity of hepatic steatosis; in addition, anthropometric and blood-tested parameters were examined to determine which of these were associated with steatosis. Results: One hundred forty-four children were analyzed, 84 male (58.3%). Steatosis was detected in 50 children (34.7%; 95% confidence interval [CI]: 26.0-42.0%). In six of these cases (12%), elevated aminotransferase levels were recorded. Factors found to be associated with steatosis were body mass index ≥99th percentile (odds ratio [OR] 3.58, 95% CI 1.16-15.6) and the level of alanine aminotransferase (ALT) (OR 1.08, 95% CI 1.03-1.13), while its severity was associated with ALT (OR 1.17, 95% CI 1.09-1.28). A level of ALT <23.5 UI/dL predicted lack of severe steatosis with an area under receiver operating characteristic curve of 0.805 (95% CI 0.683-0.927). Conclusions: Non-alcoholic fatty liver disease is common in the obese pediatric population in our geographical area. High levels of ALT are associated with severe steatosis, although having ALT above the normal range is not common. Also, the lack of severity of steatosis can be predicted in a subgroup of children with obesity. © 2013 Journal of Gastroenterology and Hepatology Foundation and Wiley Publishing Asia Pty Ltd.
Mozzillo E.,University of Naples Federico II |
Delvecchio M.,Pediatrics Unit |
Carella M.,Medical Genetics Unit |
Grandone E.,Atherosclerosis and Thrombosis Unit |
And 11 more authors.
BMC Medical Genetics | Year: 2014
Background: Wolfram Syndrome type 2 (WFS2) is considered a phenotypic and genotypic variant of WFS, whose minimal criteria for diagnosis are diabetes mellitus and optic atrophy. The disease gene for WFS2 is . The clinical phenotype of WFS2 differs from WFS1 for the absence of diabetes insipidus and psychiatric disorders, and for the presence of bleeding upper intestinal ulcers and defective platelet aggregation. After the first report of consanguineous Jordanian patients, no further cases of WFS2 have been reported worldwide. We describe the first Caucasian patient affected by WFS2. Case presentation: The proband was a 17 year-old girl. She presented diabetes mellitus, optic neuropathy, intestinal ulcers, sensorineural hearing loss, and defective platelet aggregation to ADP. Genetic testing showed a novel homozygous intragenic deletion of in the proband. Her brother and parents carried the heterozygous mutation and were apparently healthy, although they showed subclinical defective platelet aggregation. Long runs of homozygosity analysis from SNP-array data did not show any degree of parental relationship, but the microsatellite analysis confirmed the hypothesis of a common ancestor. Conclusion: Our patient does not show optic atrophy, one of the main diagnostic criteria for WFS, but optic neuropathy. Since the "asymptomatic" optic atrophy described in Jordanian patients is not completely supported, we could suppose that the ocular pathology in Jordanian patients was probably optic neuropathy and not optic atrophy. Therefore, as optic atrophy is required as main diagnostic criteria of WFS, it might be that the so-called WFS2 could not be a subtype of WFS. In addition, we found an impaired aggregation to ADP and not to collagen as previously reported, thus it is possible that different experimental conditions or inter-patient variability can explain different results in platelet aggregation. Further clinical reports are necessary to better define the clinical spectrum of this syndrome and to re-evaluate its classification. © 2014 Mozzillo et al.
Palumbo O.,Medical Genetics Unit |
Palumbo P.,Medical Genetics Unit |
Delvecchio M.,Pediatrics Unit |
Palladino T.,Medical Genetics Unit |
And 4 more authors.
American Journal of Medical Genetics, Part A | Year: 2015
We provide a detailed clinical and molecular characterization of an 11-year-old female patient presenting with neurodevelopmental delay (NDD), intellectual disability (ID), seizures, stereotypies and dysmorphic features. Chromosomal microarrays analysis (CMA) detected a small, rare de novo deletion on chromosome 12q24.31 encompassing 31 protein-coding RefSeq genes and a microRNA. Phenotypic comparison with molecularly well-defined cases previously reported in the literature harboring an overlapping 12q24.31 microdeletion indicate that these patients shared common clinical features including neurodevelopmental delay, intellectual disability and behavioral problems. Also, seizures and dysmorphic features are frequent and a consistent pattern was recognized. Since there are remarkable resemblance between the patient described here and at least another one previously reported, our report is provides supportive evidence for the existence of an emerging syndrome caused by a microdeletion in 12q24.31. We propose a minimal region shared among patients contributing to the etiology of the common clinical features observed suggesting as candidate, for the first time, the gene SETD1B which is a component of a histone methyltransferase complex. In addition, we speculate on the possible contributive role of the MIR4304 to some clinical features observed in our patient. Evaluation of more patients with well-characterized deletions within 12q24.31, as well as careful clinical assessment of them, is needed to corroborate our hypothesis, to perform a more detailed genotype-phenotype correlation and, finally, to fully delineate this emerging icrodeletion syndrome. © 2014 Wiley Periodicals, Inc.
Franceschi R.,Pediatrics Unit |
Gaudino R.,Pediatrics Unit |
Marcolongo A.,Obstetrics and Gynecology Unit |
Gallo M.C.,Pediatrics Unit |
And 3 more authors.
Fertility and Sterility | Year: 2010
Objective: To assess the prevalence of polycystic ovary syndrome (PCOS) in a cohort of young women with previous idiopathic central precocious puberty (ICPP) at least 3 years after menarche, and to look for any predictive factors of PCOS at the time ICPP was diagnosed. Design: Longitudinal study. Setting: Pediatrics unit, Verona, Italy. Patient(s): Forty-six young women (18.1 ± 3.0 years) who had been treated with GnRH analogues during childhood, observed at gynecologic age of 6.23 ± 3.3 years. Intervention(s): Semistructured interview concerning cycles, physical exam, blood sampling, and transabdominal pelvic ultrasound. Main Outcome Measure(s): Oligomenorrhea, LH, FSH, E2, T, DHEAS, free T, delta4-androstenedione, 17-OHP, P, polycystic ovary morphology (PCOM). Result(s): Fifteen percent of the young women had oligomenorrhea, 28% clinical hyperandrogenism, 48% biochemical hyperandrogenism, and 37% PCOM. A total of 32% of the patients had PCOS according to the Rotterdam definition and 30% had PCOS according to the Androgen Exess Society. The prevalent phenotype of PCOS was characterized by clinical and/or biochemical hyperandrogenism and PCOM. We did not find any predictive factors for PCOS at the time ICPP was diagnosed. Conclusion(s): Patients with ICCP are prone to developing PCOS. The prominent phenotype in this cohort was PCOM associated with clinical and/or biochemical hyperandrogenism. Further follow-ups of these young adult patients will clarify whether this phenotype persists and if it will have important long-term implications regarding increased risk of infertility or metabolic complications. © 2010 American Society for Reproductive Medicine.
Delvecchio M.,Pediatrics Unit |
Faienza M.F.,University of Bari |
Lonero A.,University of Bari |
Rutigliano V.,University of Bari |
And 2 more authors.
Hormone Research in Paediatrics | Year: 2014
Background/Aims: Prolactin (PRL) is produced by the anterior pituitary gland. It exerts its role on the breast gland but also plays a modulatory role in autoimmune mechanisms. Celiac disease (CD) is a gluten-sensitive autoimmune enteropathy sometimes associated with autoimmune endocrinopathies. No data on PRL levels in CD patients are available at diagnosis, and no conclusive data are reported. Methods: We aimed to evaluate PRL secretion in newly diagnosed CD pediatric patients and, in the case of hyperprolactinemia, any changes in its levels while the patients were on a gluten-free diet (GFD). We recruited 67 patients and 39 healthy controls. Results: PRL was statistically higher in the CD patients (13.5 ± 9.2 ng/ml) than in the controls (8.5 ± 5.0 ng/ml). In the CD group, PRL was inversely correlated with the age at diagnosis (r = -0.326; p = 0.007). In patients with hyperprolactinemia at diagnosis, PRL decreased after 6 months of GFD. Conclusion: This paper confirms that PRL may be increased at diagnosis of CD and shows, for the first time, that it decreases after a short course of GFD. Changes in the levels of inflammatory cytokines in CD may account for changes in PRL levels. Younger patients seem more prone to develop hyperprolactinemia than older ones. © 2014 S. Karger AG, Basel.
Sanchez-Solis M.,University of Murcia |
Garcia-Marcos L.,University of Murcia |
Bosch-Gimenez V.,University of Murcia |
Perez-Fernandez V.,Pediatrics Unit |
And 2 more authors.
Pediatric Pulmonology | Year: 2012
Objective Both healthy preterm infants and those with bronchopulmonary dysplasia (BPD) have poor lung function during childhood and adolescence, although there is no evidence whether prematurity alone explains the reduction in lung function found in BPD infants. Our study seeks to know if lung function, measured in infancy by means of rapid thoracic compression with raised volume technique, is different between preterm infants with and without BPD. Methods Lung function was measured in 43 preterm infants with BPD and in 32 preterm infants without BPD at a chronological age range of 2-28 months. Forced vital capacity (FVC), forced expiratory volume at 0.5âsec, and forced expiratory flows at 50, 75, 85%, and 25-75% of FVC were obtained from maximal expiratory volume curves by means of rapid thoracic compression with raised volume technique. Maximal flow at functional residual capacity was measured using rapid thoracic compression at tidal volume. Multiple regression analysis and generalized least squares (GLS) random-effects regression model were used to control for variables such as gender, weeks of gestation, age, birth weight, and tobacco smoke exposure. A sub-analysis was performed in infants born at 28+ weeks of gestation. Results BPD was associated to significantly lower flows (regression coefficients: -0.51, -0.54, -57, -0.53, and -0.82, respectively for FEF 50, FEF 75, FEF 85, FEF 25-75). This association was driven by males and maintained in the subgroup of infants born at 28+ weeks of gestation. Conclusion BPD is associated with an additional decrease of lung function during the first 2 years of life in infants born preterm. Pediatr Pulmonol. 2012; 47:674-681. © 2011 Wiley Periodicals, Inc.
PubMed | University of Lausanne, Instituto Of Ricovero E Cura A Carattere Scientifico, Telethon Institute of Genetics and Medicine, University of Modena and Reggio Emilia and 2 more.
Type: | Journal: Italian journal of pediatrics | Year: 2015
Multiple Sulfatase Deficiency (MSD; OMIM 272200) is a rare autosomal recessive inborn error of metabolism caused by mutations in the sulfatase modifying factor 1 gene, encoding the formylglycine-generating enzyme (FGE), and resulting in tissue accumulation of sulfatides, sulphated glycosaminoglycans, sphingolipids and steroid sulfates. Less than 50 cases have been published so far. We report a new case of MSD presenting in the newborn period with hypotonia, apnoea, cyanosis and rolling eyes, hepato-splenomegaly and deafness. This patient was compound heterozygous for two so far undescribed SUMF1 mutations (c.191C>A; p.S64X and c.818A>G; p.D273G).


text:
Sometimes I will admit I can become so tired of myself. Sick of myself in fact. I look in the mirror and see this weak, pathetic and needy woman looking back at me and I hate her.
The people that treat me the worse I treat the kindest and for what?
I apologise to the people I feel I have hurt because I can't carry that heaviness in my heart every day. The burden is too heavy and it poisons me every second of everyday that I do.
To the woman who I pushed away once I could tell she had, had enough of my grief. The same women who could no longer deal with a depressed friend. The woman who was in it for only the good times and only a quota of bad.
I apologised.
I told her sorry I showed her my depressive state. I apologised for bringing her down with my misery. Apologised for pushing her away.
She kindly carried on with her initial response and not only ignored my gesture but blocked me from her life all over again. Nice huh?
To the woman I helped give birth to her first born child. To the mother of the child I stayed awake hours caring for her colic child being gentle, sweet and patient as possible. To the woman whom I warned via her partner about people trying to harm her.
To the woman I would leave my own family for to accompany her on nights she needed or wanted me.
The same woman who called me a "terrible mother, terrible friend, terrible person and immature".
I apologised.
I told her I was sorry I hurt her by not following her every instruction. I was sorry that I assumed she trusted me just because I was caring for her new born child.
I apologised for being a terrible person.
To which she not only blocked me from her life in way of a reply but then tried to convince others including my own friends that I intentionally inflicted emotional trauma on her and I am a great deceiver. Then to add the "cherry on top" said she wished me the best of luck in life? At which I became confused at who was deceiving who.
To the distant friend who kicked me out of her home the weekend I came to stay. The same friend who did not care if I got hit by a car outside her home or if I walked miles in circles to try and find my way home from such a far away place that I did not know.
The same person who looked me in my eyes while I cried desperate tears and pleaded with to resolve our argument.
I apologised.
I apologised for not being a mind reader. I apologised for not doing what she had wanted, when she had wanted. I apologised for not explaining myself properly. I apologised for all our misunderstandings.
Why am I like this?
Why am I nice to people who clearly not only can't stand me, think I am worthless and treat me accordingly?
They say "kill em with kindness" but its killing me; not them.
I used to think the only option was to do the opposite and be more like them. Be nasty. Be bitchy.
I have it in me and yet I choose not to.
I am not them. They are not me.
At the end of the day I have to live with myself for the rest of my life. I want to be able to look back on my life and say I at least tried to be a good, loving and caring person.
I can only hope that being these things are rewarded.
I look at all these people now and see them getting on not a care in the world about what they did to me. Some I know for a fact still enjoy the fact they stepped on me and pray I am still broken as a result of what they did; yes some of them take pride in who they are and what they have done especially if it is horrible.
For all of those who supported me the other day in regards to my contemplation on Karma, I thank you again although I am still contemplating it.
I realise now Karma or not I am lucky in the sense that these people who treat me this way have given me the biggest gift of all by staying away because they are not the sort of people I ever want back in my life. Throwing apologies back in my face as if they are better, bigger and more maturer than me? Yet to me it seems they are the very opposite especially how they let their pride control them.
I may look weak.
Sometimes I may even feel weak especially for my apologising nature. However I have so many good friends, family, love and light that the reflection I see is only ever temporary like a small stone making a ripple in the sea.


text:
Sonoma State University environmental science major
Think about what career is best suited for you, and where you want to study. Request more information now.
Think about what career is best suited for you, and where you want to study. Request more information now.
Think about what career is best suited for you, and where you want to study. Request more information now.
Info concerning Sonoma State University environmental science major, and registering for discussion sections. Which college degrees give you the best chances of finding employment after graduation? Accredited engineering programs, combined with a brief internship or prior work-experience, offer a great return on your engineering education. Concentrating on Sonoma State University environmental science major, for example, you may gain an advantage in job recruiting on-campus. Check with the environmental engineering department for course registration deadlines, and environmental design program requirements. Please use the form above to request admissions info for Sonoma State University.
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Every day up to three impressive images are shown in the Editors' Selection on STRKNG, Instagram, Facebook, Twitter and other networks. Submit your most compelling work to benefit from this reach.
C'est la vie - © Samanta
- © Ángela Burón
Intimate - © jaioneamantegiestudio
arched - © Sandra Löwenherz
Laura Zalenga
- © Jessica Drew
Corinna Dumat


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My writing journey
My journey into writing has not been a very straight one. If I look back I can see it always appealed to me. I remember writing ghost stories when I was a child at home. They were usually inspired by the Stephen King films I had snuck in and watched while my dad was asleep. But when I started secondary school I was put in the lower set for English and I came to the conclusion I wasn't smart enough to be a writer. I remember struggling to write the a lower case "d" when I was very little. My hand kept wanting to do a lower case "b", and I got it eventually, but it took me longer than most other people in my class. I used to read The Beano and I thought that one of the characters was called 'Berly the Perly', not 'Beryl the Peryl', and I felt embarrassed for getting it wrong as I had transposed the "l" and the "y". These things were few and far between, but looking back at them, I can see my dyslexia and it wasn't picked up on in school, possibly because I could always read very well and I loved reading. I wonder if I had been taught in a different way if I would have had more confidence to write sooner? Being in a low set meant I wouldn't be able to do A-Level English and since I didn't know what I wanted to do for my career I left school at sixteen and got a job. I was fortunate to get a job in an art gallery and learned to be a picture framer. It was good to make something with my hands, but after a while framing wasn't enough creativity for me, and at twenty years old I decided to go back to school.
I painted as a hobby, but mainly spent my free time devouring stories whether they were in books or movies and thought I would like to tell stories through film. So I worked out what college course I needed to do in order to get onto a film degree, and chose a two year National Diploma in Multimedia. I made short films for as many of our assignments as I could, and got distinctions for my projects and was even student of the year - never before had I done so well in education. I was being taught by fantastic tutors in the right way for me to learn and I was being given the confidence that I could do these things. I have since read a great book on dyslexia called The Dyslexic Advantage, and I saw many similarities to how my own brain works. This book focused on the many positives of being dyslexic, and I think that part of my brain is what makes me good at telling stories and finding connections. It was also refreshing to read there are so many famous dyslexic writers, Agatha Christie, Anne Rice, Stephen King and Fannie Flag, just to name a few.
Making one of my college films:
I completed my ND and fortunately I happened to already live in the town which had one of the best film degrees in the UK. I think it was my sheer enthusiasm that won me a place on this competitive course. When I started I felt like everyone else on the course was much more experienced than me, but I kept up and the best piece of work I did in that first year was a written piece I chose to do on Nietzsche and Fight Club. I was told if I handed that in as my dissertation in my final year I would have got top marks. Towards the end of my first year at university, my now husband came back into my life and proposed. Soon after he was offered a job in San Francisco that he couldn't turn down, after much deliberating I decided to make the move to California.
During my first ten months in San Francisco I was unable to work so I spent that time teaching myself to cook and writing. I was working on an idea I had before I left England, and it became my first screenplay. As soon as I got my green card I found a job at a video game company working in the HR department, but again, not doing something creative was killing me inside. So I revisited the characters I had created in those first ten months and enrolled on the first of two 10-week screenwriting workshops. By the end of the second workshop, (our starting out number of thirteen people had gone down to just three) I had a fairly good first draft of a 95 page screenplay, which got into the quarterfinals in the Blue Cat screenwriting competition and felt like I was on the right track.
When my husband got laid off from his job in 2008 we looked at it as an opportunity to move somewhere new and less expensive so we could live on one salary and I could focus solely on writing, and that is what I have done ever since. I wrote three more screenplays, attended many writing seminars, my favorite being Story by Robert McKee. It was in L.A. that I had the first ideas for the Lucy Lick-Me-Not series and that is when I started to write children's stories and discovered I loved it.
In 2015 I could feel my fifth screenplay needed more than my favored 90 pages and halfway through I attempted to write it as a novel and the story flowed much better. It was a little intimidating to leave behind the format of screenplays which I felt I had mastered and venture into new territory, but it was freeing as well. As I work on this novel I'm also preparing my first a young adult novel and I have many other children's stories, several of which are ready to be illustrated. Sometimes it feels like there is not enough time to do all these things, and I'd like to have them all out and published in the world now, but I don't want to rush my writing, I want it to be perfect. Every single word is chosen specifically, whether it's a children's rhyming story or a novel for adults. I'll rush the washing up. To be honest, I rush most of the things I don't enjoy - if you open some of my kitchen cupboards you may end up under a pile of precariously strewn Tupperware, if my hair looks crazy, so what? I do not consider myself a perfectionist, but when it comes to my writing, that's another story.
More soon,
A new home, a new season, a new project
For me the Gaelic calendar feels more natural for the seasons, especially when you see crocuses and snow drops sprouting in February. If you were following the Gaelic calendar, the spring months would be February, March and April, summer would be May, June, and July, autumn would be August, September and October, and winter would be November, December and January.
As I mentioned in previous posts I recently moved to the Pacific Northwest. It's the first relocation (out of many) that we have done with the seasons. It felt right to relocate and start fresh in the spring, planting real roots for the first time in a decade. I love that people used to plan their lives by the seasons - they had to. And having that natural rhythm to follow during this move has made it feel so much easier than all our previous relocations, which tended to happen in the autumn and made our lives hectic and busy at the time of year when things should be slowing down, and we would all like to be getting into a different rhythm for coming the winter months.
Springtime brings about images of renewal and new beginnings, and that is when we found our new home and began getting all the interior work done. We moved into our house in summer, and have been settling in to a more relaxed pace of life and enjoying the many flowers in our garden and ripe plums from our tree:
I'm very excited for the autumn and for being able to enjoy it properly now that we are settled into our new life. It will be our first real one in about eight years, and I can't wait to see the fall colors here and wear scarves and sweaters again. I'm looking forward to working in the garden and pruning it back for the winter and smelling that crispness in the air that is prevalent on most September mornings.
My back is still slowly getting more comfortable, physical therapy is helping and so I am back to writing a bit more and it feels good. I'm reworking a one-off children's story that is set around Halloween and features some new characters and a couple of seasonal monsters - more to come on that soon. For now Lucy Lick-Me-Not is taking a little break, but we'll update the FB page and website when there is more news on her return. Hopefully it won't be too long.
(Artwork by Bret Burkmar)
More soon,
A room with a view (at last!)
It's been just over two years since I started this blog and wrote about my little writing cupboard in my first post. After relocating up north I now have a writing room with an actual window (well two actual windows!) I can see many types of trees from both, and I love hearing and seeing all the different kinds of birds outside in our rural setting. I especially love to hear the roosters from the many friendly neighbors around me who raise chickens and have kindly welcomed us to the neighborhood by bringing over fresh eggs with yolks the color of sunshine. It is a stark difference to the sounds of busy city life I could hear from the dark cupboard I used to write in. The walk I take my girl, Murphy, on leads to the Puget Sound and on the way we even get to see a llama who is becoming less shy each time we pass him.
Some pics of the new writing room and our walk:
I will keep the blog name, Notes from a cozy cupboard, because that is where my blog started and the new writing room has a cozy feel to it as well.
More soon,


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I'm extremely filled with happiness.
I'm happy because I'm a mommy. And the constant kicks are reminders that I am a mommy to a cute little baby that will be born in about 20 weeks.
I love reading blogs. I mean what else am I gonna do to fill up the 40+ hours I am at work every week. I was reading through some blogs of women who got pregnant and then ended up losing there sweet precious little baby.
I pretty much started crying at work (darn pregnancy hormones) I seriously could see myself being in that situation.
And then just as I got really sad for these women my baby kicks me to remind me it's still there and it loves me. I don't know it just feels wonderful knowing that it's still there.
That's what I worry most. Is that I'll lose my baby. Every time we have an appointment I'm so anxious for it to make sure my little sweet baby is doing okay. And it's confirmed every week that it's still doing awesome.
Even after all the pain that I'm in and the constant tiredness and I'm just overjoyed that I'm a mommy.
So even if I get sad those little kicks are a reminder that everything is okay and will be okay.


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