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If you're looking for something to satisfy your sweet tooth, there's a new shop in downtown Altoona.
The Bakery, yes, that's its name, recently opened its doors.
The family-run shop sells sweets like sticky buns, cheesecakes, and breakfast sandwiches made on fresh bagels.
Owner Kurt Drost says there's one item folks are always coming in for.
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The Bakery also offers vegan and gluten free options for folks with restrictive diets.
Drost added that he's excited that the businesses in downtown Altoona are supportive of each other.
"It's really great to be a part of what seems to be the comeback of downtown Altoona, and we're supporting all of the other businesses that are already open or are getting ready to open, and we're excited for them and we're excited to be a part of it," Drost said.


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No matter how major or how minor, no one ever plans to be involved in a car accident. One minute you are driving down a residential street and the next someone runs a stop sign, causing a t-bone car or side-impact crash and multiple injuries to both the driver and passenger. In situations like this, the outlook may look bleak, but there is a light at the end of the tunnel.
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The Dexter boys' swimming and diving team dropped three close meets against a trio of the top swimming programs in the state on Saturday at Birmingham Groves in a non-league quad-meet. The Dreadnaughts lost 102-83 to Groves, 92-88 to University of Detroit Jesuit and 94-89 to Brother Rice.
Dexter had plenty of fast times and quality results.
In the medley relay, the Dexter team of Drew Kemp, Paul Schaefer, Zach Norris and Tanner Sharp took fourth in 1:43.03. The team of Stuart Bovich, Griffin Patel, Adam Hauser and Lucas Greatorex was ninth in 1:51.94.
In the 200 freestyle, the Dreadnaughts went two-three with Leo Varitek (1:48.82) and Luke Sayler (1:48.97). Joe Sharon was eighth in 1:57.22.
Norris went 2:03.48 to take third place in the IM. Hauser touched ninth in 2:13.24.
Sharp took third in the 50 free with a time 22.00. Kemp was eighth in 23.82.
Dexter sophomore Cole Warren took first in diving with a score of 250.60.
In the butterfly, Bovich was eighth in 1:00.48.
Sayler touched third in the 100 free in 49.43.
The Dreadnaughts had an impressive showing in the 500 free. Schaefer, a senior, took first place with a time of 4:53.93 while Varitek, a junior, was second in 5:03.62. Hauser was fifth in 5:16.53.
Dexter's 200 free relay team of Kemp, Sharp, Sayler and Varitek took second with a time of 1:31.98.
In the backstroke, Norris was second with a time of 56.04. Bovich was seventh in 1:00.25.
Sharp took second for the Dreadnaughts in the breaststroke with a time of 1:00.59. Schaefer was fourth in 1:03.55.
The Dreadnaughts were second in the 400 free relay as the team of Sayler, Varitek, Norrish and Schaefer touched in 3:22.73. The team of Hauser, Bartscht, Greatorex and Bovich were eighth in 3:49.98.
Dexter had no problems earlier in the week during a dual-meet with Adrian. The Dreads dunked the Maples 128-48.
The Dreadnaughts travel to Saline on Thursday for a quad-meet. The meet starts at 6 p.m.


text:
Ross Kemp Prepares for GIF Superstardom
Spice expands consciousness. Spice shuts up Ross Kemp.
Spice expands consciousness. Spice shuts up Ross Kemp.
The couple were reportedly arrested in July but their identities were only revealed this week since the Iranian government has not yet acknowledged the arrests.
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The entire industry is built on smuggling them into prison, he thinks.
After seven years in prison, Chelsea Manning was released early Wednesday morning from the Fort Leavenworth disciplinary barracks in Kansas.
Someone in government without a clue, shock.
How to get inked up on the inside.
Daniel Kelly, you've gone down in history.
Printing themselves out, stealing our jobs.
Cold turkey for convicts in Bullingdon Prison.
A flight of fancy indeed.
The US's overcrowded prison system is in a sad state, and it's not made any better by only having access to this incredibly basic 4.3-inch JP5mini tablet.
Sadly it crashed but it was a good idea and certainly better than trying to get a Galaxy Note 4 up your arse.
Man bought convincing-sounding domain to email prison staff from, saying he was up for bail. So they let him out. Criminal genius.


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Latest News
Last day of Term is now Fri 20th July
The school governors have agreed to give the children an additional holiday day at the end of this term.
The last day of term for the children will now be...
Refugee Week
Last week at Harehills Primary School, as part of Refugee week, we were learning all about what it means to be a refugee and the challenges that refugees...
Year 6 Netball Team Gains 2nd Place in Leeds East Finals
Well done to our Y6 netball team who have gone onto the Leeds Netball Finals after coming 2nd place in the Leeds East Netball League.
The netballers made...
Harehills Debating Success
Congratulations to our year 6 girls Khadija, Aleesha, and Aisha who became Leeds 2018 Primary School Debating Competition Winners at the Leeds Civic Hall....
We are a Sun Safe School
Harehills Primary School is proud to have achieved Sun Safe School status as of 14th June 2018.
It is so important that all members of our Harehills...
Weekly Attendance Update June 4th 2018
Last week we had three classes acheive 100% attendance!
Well done to
- Sandhurst 2
- Elford 3
- Bexley 2
Darfield 2 and Conway 2 weren't far behind...
Exciting Opportunities
Would you like to work at Harehills Primary School?
We have two exciting vacancies for the following experienced educators:
- Assistant Head - Leader...
School closed for Staff Training Mon 4th June
School is closed to children today for staff training.
School will be open as normal tomorrow, Tuesday 5th June 2018.
- 1


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A forum about Atari 16/32 computers and their clones. This forum is in no way affiliated with Atari Interactive.
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New Nursery School Building in the District Les Sablons, Le Mans, France
Customer Project
|Project Owner
City of Le Mans
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My name is osteosarcoma. I am a highly malignant tumor of mesenchymal origin and the second most common primary malignant bone tumor.I originate from mesenchymal cells. I have spindle shape and hyperchromatic nucleus. I have been formed when osteoblasts cells secreted malignant osteoid. I am like osteoblast (bone forming) cells, but I cannot make a strong matrix like them. You know that osteoblasts originate from immature mesenchymal stem cells. So my ancestors are from those. But I am already modified into osteosarcoma cell. I can occur at any bones found in the body. I am more frequent at sites of the most rapid bone growth; those are extremities of long bones near metaphyseal growth plates. From the sites that I frequently choose; distal and proximal end of a femur (42%), a proximal end of the tibia(19%), the proximal end of the humerus (10%), pelvis skull and jaw(85%) and pelvis( 8%). I can be appearing 1 to 3 million people per year. Of course, I am rare compared to other types of cancer. I can attack any age as primary osteosarcoma but mostly between 10-25yrs. I can also come at older individuals as secondary osteosarcoma. I affect males little bit more than females. I am most common in patients with Fraumeni syndrome, Rothmund-Thomson syndrome, Bloom and Werner syndromes. Exposing to Paget disease and ionizing radiation give favorable conditions for me to develop our colony.
Fig 1. Osteosarcoma genesis and osteogenesis
When I was changing to osteosarcoma, there was a mutation of MDM2 amplifications which led to p53 gene inactivation. Another controller gen which is called retinoblastoma (RB) genes was also altered. I become safe because there is no more checkpoint and control for me.But the normal bone cells have several checkpoints in their cell cycle. In the presence of RB mutations, I will occur at an incidence 500 times that of the normal population. Normal cells are always worrying
about G1/S checkpoint but I am not, because the p53 and Rb genes are already mutated (lost their function). I am preparing more DNA and for that I am also synthesizing mRNA and important proteins. During my replication time, I gained chromosome number 1 and lost chromosome 9, 10 and 13. Till now I am successfully dividing and I am forming other similar cells. In near future, I will have my own colony of cells and my own sarcoma tissues
As usual, I was preparing to divide but the proteins detected a damage in my DNA. The damage was too big to be repaired. Even if it is bigger damage I will not go to programmed cell death called apoptosis. Because this is not my character. My only goal is to survive and proliferate more. This is my unique behavior. Of course, I was not like this, I was following the normal path of the cell cycle and programmed cell death. This thing happened after I lost my DNA sequence that triggers cell death pathway.
Recently I got other mutations and my FOS and Jun proteins are up-regulated. Those proteins are components of activator protein1 complex (AP-1).AP-1 his regulator of cell transcription. Now I am getting additional energy because my transcription regulators are out of service. I can proliferate freely and destroy bone cortex and extend towards bone marrow cavity and soon outwards towards adjacent soft tissues.
I am now thinking why not I visit other organs. I have information lung, liver and also other bones are the safe place and more fertile to me.when I become more organized, I can migrate and live there. I am already resistant to programmed cell death (anoikis) and can exhibit anchorage-independent growth (AIG). Abnormal integrin? ??v6, Rho, ATPase e.g. Racl and Cdc42 upregulated p13 kinases which in turn inhibited proapoptotic factor Bad and this protected me from apoptosis even when I am not attached to any membrane. Now I have bigger size with the hypoxic and acidic environment. So I need supporting blood vessels to obtain oxygen and nutrients I have done angiogenesis for my sustained growth and further metastasis. My intrinsic conditions have to lead to stimulation of von Hippie Lindau protein. It releases hypoxia-inducible factor-1? (HIF-?)which further up-regulates several VEGF gene such as VEGF A through VEGF E. This further releases Nitric oxide (NO) from endothelial cells and results in vasodilation and increased vascular permeability. By doing those processes I got leaky and irregular vasculature.
I am starting a flight to the lung. I am using integrin proteins, which found on my surface, in attaching to matrix proteins (fibronectin).For my signal transduction and interaction with other cells, I use integrin along with ezrin protein, foal adhesion kinase (FAK), protein kinase C (PKC) and Rho GTPase. They also help me with my conformational changes. Besides to this matrix metalloproteinase (MMPs) are helping me in my sojourn through blood vessels and tissue renewal process. Now I have reached the lungs and I am growing, multiplying and consuming all nutrients. It is like as I heard.I like it.
Today, the patient could not resist me.He took me to hospital. Because I create severe pain and swelling. I was diagnosed with radiograph as high-grade sarcoma. Additionally, they did CT scan, RI, and biopsy and confirmed my stage. They are now discussing to fight against me.Yes, they have started chemotherapy (high dose methotrexate, doxorubicin, and cisplatin).i have been fighting against those drugs for the last three months and I am now getting old, starving and feeling weaker day by day. Oh,, those drugs become treble for me. Methotrexate is a dihydrofolate reductase inhibitor, it can block folic acid supply which is needed for my RNA and DNA synthesis. Doxorubicin which is also a drug that interacts with my DNA and inhibits biosynthesis of macromolecules. Cisplatin causes damage to my DNA. I have to create solutions because I am no longer able to make RNA efficiently. But I am struggling to continue my life. I got another mutation which is an alteration of reduced folate reductase (RFC) protein Leu291pro. This alteration unable carrier to transport methotrexate towards me.Besides to this, membrane pump-glycoproteins (P-gp) is also helping me with pumping out these drugs. Due to this, I start dividing and spreading slowly but steadily. Other worse situations are happening to me, they are using high energy rays (x-rays) to kill me. I am no longer able to grow. I am shrinking.
The worst thing has happened to me. Now, they are performing surgery and removing me as cancerous tissue. I was sending my daughters to afar to the lung. By myself, I am highly depressed and weak due to huge loss my colony. I try to grow and spread again by my reserve soldiers and by those immigrant members. They are still in dilemma to administer chemotherapeutic agent after surgery. The people are always struggling to combat me and other friends. If they post-surgery chemotherapy no more survival .I say goodbye I will not be there anymore.
Biomarkers in osteosarcoma, Colin Kong, M.S and Marc F. Hansen, Ph.D.
Biology and therapeutic advances in pedia
tric osteosarcoma, Nayssa Marina Et.al,The oncologist ,2003
Novel therapeutic strategy for osteosarcoma targeting osteoclast differenciation, bone reabsorbing activity , and apoptosis pathways.
The molecular pathogenesis of Osteosarcoma; A review, Matthew L. broadhead, sarcoma, 2011.
Current strategy for chemotherapy in Osteosarcoma, Dorothy carric, International Orthopedics, 2006
Osteosarcoma: A review of diagnosis, management and treatment strategies, David S. geller , MD, and Richard Gorlic MD
Switch from ?v?5 to ?v?6 integrin expression protects squamous cell carcinomas from anoikis, S.M. Janes, Journal of cell Biology,2006.
Analysis of molecular mechanisms of osteosarcoma using bioinformatics approach, Jaxon yang ,oncology letters,2016.


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American tenor Stephen Costello will sing Dvořák's Stabat Mater with the Münchner Philharmoniker on the 12, 13 and 15 April. Marking Stephen's debut with the orchestra, Manfred Honeck conducts, with soprano Hanna-Elisabeth Müller, contralto Wiebke Lehmkuhl and bass Christof Fischesser completing the quartet of soloists. The three performances will take place at the Philharmonie am Gasteig.
Stephen has had an extremely busy season so far, with successes as the Duke in Rigoletto in both Toronto and Dresden, and will reprise the role at Deutsche Oper Berlin (alongside Simon Keenlyside and Albina Shagimuratova) after his concerts in Munich. Verdi's Duke is one of the roles on which Stephen has most decisively made his mark. In the Metropolitan Opera's Vegas staging of Rigoletto, the New York Times praised his "bright tenor that … was perfectly suited to [the] character." Likewise, at Houston Grand Opera, his Duke was hailed as "the sensation of the night" (Houston Chronicle), prompting Theater Jones to name him "one of the best lyric tenors in the business."
Read more about Münchner Philharmoniker's Stabat Mater here.


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Today I have had the day off work (I work every weekend so get my days off in the week) I have done a bit of shopping, cleaning and chilled out this afternoon - bliss, so when I woke up this morning I thought I would put the ELF Lock & Seal to the test.
"Increase the intensity of your makeup colors and lock it on instantly. Coat your eyeliner, shadow, lipsticks, blush, brow color and more for a sealed smudge-proof wear. The quick dry and easy to use formula keeps you makeup in place for hours. Say goodbye to makeup smudges and hello to makeup that stays put!"Sounds promising, inside you get a bottle of the product, a little dish to put the product in and mix in your chosen makeup and a brush.
Put a few drops of the Lock & Seal into the small dish provided. Dampen the brush thoroughly with the liquid and mix it with any powder or cream makeup, apply to the face and let it dry thoroughly.
And here is what it looked like after 10 hours.....
(I took both pictures in the same room with the same lighting even though the first pic looks slightly darker, I need a new camera).Overall the product did OK. The eyeshadow is still there but has lost it's sparkle but I think the colour would stay longer. I think this product would be great for a night out where you want vibrant eyes for a few hours, and for the price it does it's job well, but due to not being able to blend and already finding my perfect makeup setter (find out what here) I don't think I will be repurchasing it.


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Desktop integration
Triggering the right tab in Salesforce console from the desktop
Salesforce is providing lots of integration capabilities. Most of them are based on APIs that you can consume from the browser (ex : Chatter API) or from the server (SOAP or REST Api).
In some circumstances there could be a need to do browser side integration.
Let's imagine you have a dedicated CTI client deployed on your desktop. It is not running in the browser. Such tools can integrate with the browser by calling a Salesforce URL, pushing the context as parameters. On an incoming call, this
The browser technology prevent tabs from closing themself if it is not initated by a user. Try to access a custom URL that host a simple script doing window.close() , this will not close the tab.
The solution is to open the URL on the incoming call, always in the same targeted tab. How can you achieve this ? Imagine you are running Internet Explorer. Your CTI client on the desktop could run the following lines of code
var url = 'https://yourdomain.my.force.com/apex/yourVisualforcePage?param1=value1&phone=1234567890'; var shellApplication = new ActiveXObject('Shell.Application'); var windows = new Enumerator(shellApplication.Windows()); var w = null; while(!windows.atEnd()){w=windows.item();windows.moveNext()};// Retrieve the last window if(w)w.Navigate(url, 0, 'CTI');
The url has to be dynamically created to push the context as URL parameters. The last line of code will target the browser tab named 'CTI'.
But we still have a remaining technical tab visible in the browser. Not very user friendly.
Code for the Visualforce page that will be loaded into the iframe named 'CTI' :
<apex:page showHeader="false" standardStylesheets="false" docType="html-5.0" > <script type="text/javascript"> localStorage.setItem('JLA', window.location.search); </script> </apex:page>
This page is just writing the context into the localStorage. This will trigger the storage event on all other page
Code for the console component that will host the iframe page and run the popup
<apex:page> <apex:includeScript value="/support/console/34.0/integration.js"/> <script type="text/javascript" > var handle_storage = function (storageEvent) { /* StorageEvent { key; // name of the property set, changed etc. oldValue; // old value of property before change newValue; // new value of property after change url; // url of page that made the change storageArea; // localStorage or sessionStorage, depending on where the change happened. } */ if(storageEvent.key=='JLA' && storageEvent.newValue!=''){ //prevent triggering other events. IE9 triggers the event with empty value on the same window after removing the item var str = storageEvent.newValue; var objURL = {}; str.replace(new RegExp( "([^?=&]+)(=([^&]*))?", "g" ), function( $0, $1, $2, $3 ){objURL[ $1 ] = decodeURIComponent($3);} ); //If your context is in the 'phone' parameter, use fromobjURL["phone"] in a SOQL query to retrieve the targeted recordID //... imagine the variable 'targetURL' is built based on your soql result (removed from this code to make it shorter) var targetURL='/001g000000ia2Pc'; sforce.console.openPrimaryTab(undefined, targetURL, true, 'the Title'); localStorage.removeItem('JLA');//allow re-issuing same url for testing purpose } }; window.addEventListener("storage", handle_storage, false); </script><iframe name="CTI" width="5" height="5" frameborder="0" ></iframe> </apex:page>
Now you have a very simple, lightweight, browser technology in Salesforce that can react to desktop initiated events.


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Welcome to our site
We are a Japanese trading company.
A Japanese trading company that creates your living happily and now.
It is our big dream to make people around the world smile.
We will discover wonderful products sleeping around the world and continue to propose to our daily life.


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possible reposession ?greenspun.com : LUSENET : Repossession : One Thread
I have a joint mortgage with an ex partner who has never made any payment towards the mortgage, I took legal advice as he was also violent and my solicitor & counsel told me to stop paying mortgage, the property is now on the market but i'm worried in case it can be reposessed, what sholud i do ?
-- Tara O'Neill (email@example.com), July 03, 2003
The short answer is yes the mortgage lender can issue possession proceedings if the correct contractual instalment is not being paid.
Proceedings can normally be brought after one two or three missed payments. You will find the exact details in the Terms and Conditions of your mortgage.
Hope this helps Jane
-- jane (firstname.lastname@example.com), July 03, 2003.
I would contact your building society/bank immediately and make an appointment to see the manager so you can explain the situation to him/her, and hopefully come to some arrangement. Just leaving it will not help resolve the situation. Let us know how you get on. Good Luck.
-- M Amos (firstname.lastname@example.com), July 04, 2003.