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ALBANY — Albany police shot and killed a 20-year-old man as he was stabbing his mother in the face inside a city home late Friday night, Police Chief Robert Sears said Saturday.
Officers Elston Mackey and Derrick Ellis arrived at 61 Partridge St. at 11:30 p.m. Friday after getting a 911 call about someone with a weapon on the second floor. A woman could be heard screaming in the background, police said, but they gleaned little other information from the call.
When they arrived, a witness near the house told them that a woman who seemed to be injured was collapsed at the bottom of the stairs inside the house. The officers could see through the window a woman who was bleeding from her head, face and neck, Sears said. They called for an ambulance.
The door was locked and the house was difficult to gain entry to because of the thickness of the glass part of the door. As the officers were trying to get into the house, a man, later identified as 20-year-old Schuyler Lake, appeared at the top of the stairs with a knife in his hand. The officers ordered him to drop the knife as he descended the stairs but he ignored their commands and began stabbing the woman, Sears said.
Officer Mackey stuck his hand through the glass and fired off several rounds, hitting Lake, who died at the scene, Sears said.
The chief said the early stages of the investigations show the officers acted appropriately, calling the shooting "must and just."
"The actions of the officers saved that woman's life last night," Sears said at a 9 a.m. news conference on Saturday.
The 52-year-old stabbing victim is Lake's mother. Sears said a search of the home led to the discovery of another stabbing victim, Lake's 55-year-old uncle. Police have not released their names. Lake lived at the house, Sears said.
"There was no indication ... that anything like this would ever happen (at the home)," Sears said.
Lake is "not known to the Albany Police Department for having any criminal history or any other activity at all," he said.
The chief said police were wearing body cameras at the time but the recording would not be released now. An internal investigation is under way and the officers have been placed on administrative leave. Mackey and Ellis have both been with the department for two years.
Lake's uncle and mother were taken to the hospital and police were able to talk to the uncle before he was taken into treatment. Both were in critical but stable condition with a positive prognosis, Sears said. Mackey and Ellis were evaluated at local hospitals, a department policy after a police shooting, he said.
Sears declined to describe the knife Lake used and said he was waiting to be notified about an autopsy. Police are putting together a timeline of the events that led up to the shooting, he said. They spoke to witnesses on the street, he added.
Police remained at the home Saturday morning. A neighbor said her daughters heard the incident as it unfolded Friday night and said they thought they heard six gunshots. She added that she thought the people who lived at the home were new to the neighborhood. Another neighbor said he heard loud banging, likely from the officers trying to gain entry to the home, late Friday night.
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NATO applauds North Macedonia's contribution in Afghanistan
NATO applauds North Macedonia's contribution in Afghanistan
KABUL (Pajhwok): NATO Secretary General Jens Stoltenberg has commended the role of North Macedonia in Afghanistan's security forces training mission and promoting regional cooperation in Southeast Europe, according to a statement on Wednesday.
"North Macedonia already makes important contributions to the Alliance. You are part of NATO's training mission in Afghanistan, you promote regional cooperation in Southeast Europe and you are also implementing major reforms," Stoltenberg said while addressing a press conference along with North Macedonia Prime Minister Zoran Zaev.
"I encourage North Macedonia to continue on the path of reform. This is important, as you prepare for full membership. And build a better future for all citizens in your country," the secretary general said.
The North Macedonia would become the 30th member of NATO once all Allies complete the association protocol process, he said.
"Your country already has a seat at NATO's table. Last month I welcomed Minister Sekerinska to the meeting of NATO's Defence Ministers. And next month, Foreign Minister Dimitrov will join us in Washington to mark the founding of our Alliance seventy years ago," he added.
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After capitalism, what? (Random thoughts on Paul Mason's article)
by Jehu
I am reading an article by Paul Mason, "The end of capitalism has begun". This article can be placed in the context of several videos exploring the same theme by radical thinkers. There is, for instance, a video by Peter Hudis, "Alternatives to Capital"; and David Harvey gave a rambling lecture from 2013 along the same lines, "The End of Capitalism".
The three pieces are all of a type: speculation regarding the end of capitalism and of what might replace capitalism if it is at its end.
According to Mason, the end of capitalism is driven by three forces: First, capitalism has reduced the need for work, blurred the edges between work and free time and loosened the relationship between work and wages. Second, information is corroding the market's ability to form prices correctly because, while markets depend on scarcity, information is abundant. Third, Mason notes the spontaneous rise of collaborative production that is no longer determined by markets and managerial hierarchies.
To be sure, the end of capitalism has been predicted so often that any sane person would conclude the subject hardly bears serious examination. The end of capitalism is the "Who shot JFK?" of social commentary, periodically surfacing in society during crises. None of the above mentioned individuals are insane, however, yet they are willing to stand before audiences and speculate on what many may judge to be crackpot theories.
As persuasive as the above argument may appear, speculative discussions of what comes after capitalism are not just confined to the fringes of politics as in the case of Harvey, Hudis and Mason. As many have pointed out in the past few years, the most important bourgeois economics thinker of the 20th century, John Maynard Keynes, made no secret of his belief that the epoch of capitalism was rapidly drawing to a close. Keynes based his prediction not on some variation of a radical assertion that the poor would rise up against their oppressors, but that capitalism would essentially lose its economic rationale.
Accumulation, Keynes argued, lay behind not only capitalist relations of production, but behind even the laws and morality of bourgeois society. By 1930, Keynes argued, accumulation was being threatened by the fantastic increase in the productivity of labor facilitated by accumulation itself. Our success in developing the productivity of labor, he stated, was outrunning the new uses to which labor could be put. It did not take a genius of Keynes's sort to arrive at what may be for us a startling conclusion: the epoch of labor was passing away before our eyes. The "old Adam" in us was doomed and soon even labor itself would have to be rationed out like a scarce commodity during war-time.
What is most surprising about this is that no one challenged Keynes on this; in fact, his argument was commonplace in the 1920s and 1930s. Indeed, only a few years earlier, 1926, Henry Ford had shocked the country by unilaterally reducing the hours of his own labor force to eight while doubling their wages to five dollars a day. No one giving serious consideration to the subject could look at the industrial revolution and especially fordism and not conclude that a new epoch for society was just over the horizon.
Despite the fact that this sort of speculation was rife during Keynes's lifetime, labor plays no less a central role in our time than in his. In fact, today, labor and employment has become an obsession that drives all politics in every state in the world market. Every political measure offered by every political party is justified on the basis it will create jobs. If anything society has become more obsessed with labor today than it ever was in Keynes's day when jobs creation had not yet become the over-riding public goal.
So obsessed has society become with labor that, in their discussions of the subject of the future after capitalism, the three radical thinkers I mentioned, Mason, Harvey and Hudis, don't even speak of a future society after labor, but only a future society after a particular sort of labor: wage labor, i.e., capitalism. In fact, their separate discussions could easily be characterized as speculations on the future of labor after capitalism.
- What forms will labor take?
- How will labor be organized?
- What will labor produce?
- How will the product of labor be distributed?
The real subject of these sorts of discussions, which emerges explicitly in the hyperbolic discussions of automation in the mainstream media, is how do we save labor from a dying capitalism. In his article Paul Mason suggests it's time for utopian thinking. "At the heart of further change to come is information technology, new ways of working and the sharing economy." According to Mason, in the future there will still be labor, but this labor will use different, more advanced, means of production, be organized differently and its product will be distributed according to a different social logic than the market.
In the dystopian future set in the most recent Mad Max sequel, destitute crowds gather at the foot of a high cliff to receive not new, fantastic, and as yet unimagined consumer products created by modern industry, but a ration of water. In radical imaginings of the future after capitalism this scene most often appears: crowds of destitute people, bereft of all means to life, lining up to receive some ration of basic commodities.
The reality of our times is otherwise: people line up to be apportioned labor. They even risk their lives and the lives of their children, making dangerous journeys over very long distances not to get a handout in a refugee camp, but to get a job in the European Union and the United States. Here is the fundamental disconnect between reality and the radical discussion on post-capitalist society.
In the capitalist mode of production labor itself exists for labor alone, not for the enjoyment of society; production exists for production alone; capitalistic accumulation is its own end. Capital is self-expanding value — this every person who has ever opened Capital already knows — but value is itself only the expenditure of some definite quantity of living labor embodied in some use value. The self-expansion of value, therefore, is nothing more than wage labor reproducing itself as its own product on a still larger scale.
What sense does it make to speak of labor after labor has become its own aim? Once labor exists as its own end, how can labor have any future after this? And what sense does it make to speak of labor after capitalism when labor and its incessant expansion alone is the aim of capitalism?
According to Mason, "With the terrain changed, the old path beyond capitalism imagined by the left of the 20th century is lost." Yet, despite this changed terrain, the new world of labor after wage labor will still require politics, i.e., a state:
"But a different path has opened up. Collaborative production, using network technology to produce goods and services that only work when they are free, or shared, defines the route beyond the market system. It will need the state to create the framework – just as it created the framework for factory labour, sound currencies and free trade in the early 19th century. The postcapitalist sector is likely to coexist with the market sector for decades, but major change is happening."
In Mason's view, a society beyond capitalism will still coexist with capitalism for some period of time. Which is to say, a society where labor is an end in itself will still coexist with this new collaborative labor. How do we tell the difference between labor that exists as an end in itself (wage labor) and collaborative labor that does not? And why is the state necessary to mediate these two forms of labor or provide a framework for labor that does not exist as an end in itself? Which is to ask, why is the state necessary to provide a framework for labor that produces no value? What is it about Wikipedia, a collaborative project of volunteers, that requires a state? This question becomes that much more urgent once you realized the state itself is only labor in a particular form, surplus value. The whole of the state sector is simply the unproductive consumption of some previously expended value producing labor.
Here, Mason likens the present transition of society to the transition between feudalism and capitalism. But he neglects to mention that the state itself lived off the surplus labor produced in both modes of production. How is the state supposed to live off the voluntary labor expended on Wikipedia, since this labor does not take the form of a commodity and the labor expended on it produces no value? Without even realizing it, Mason is proposing that labor as an end in itself must dominate collaborative activity as long as the two coexist!
In fact, the state plays an altogether different role in this transition than the one Mason believes: it serves as an obstacle to the abolition of capitalism, a role it performs, at least in part, by placing severe artificial intellectual monopoly property rights on information.
It may be true, as Mason argues, that digitized information wants to be free, but that is hardly the problem with transition to a post-capitalist society. That information wants to be free is not very difficult for radicals to understand since, as a commodity, digitized information appears to be ephemeral, or unsubstantial, unmaterial and thus not really commodity at all in the sense we employ this term. Since Guttenberg introduced the press, the cost of the transfer or reproduction of information has collapsed to just a minute fraction of what it once was when reproduction of knowledge required days, weeks or even months of painstaking manual labor. Few of our generation realize information, knowledge itself, was once the most prized possession of the very wealthiest members of society. Today, it takes only a few minutes of googling to call up almost any knowledge mankind has acquired in the last 5,000 years.
The bigger problem, expressed in the converse form of extraordinary efforts by the monetary authorities of the world market to induce inflation, is that both commodities and labor want to be free as well. Society is thus faced with the very real prospect that the prices of everything it produces and labor power itself is headed toward zero. The trajectory of prices, deflation, effects not just capital alone, but labor and its wages as well. The insignificant cost of reproducing information is nothing compared to the advances in agriculture in the United States. Here, a task that once took the better part of the life time of almost all human beings — producing food — now engages the labor of less than one half of one percent of the population.
The thing that terrifies our radical thinkers today is that labor itself wants to be free, which implies, above all, that wages are going to zero. Nothing we do can prevent this from happening. And nobody knows what to do about it but to speculate on possible forms of labor beyond 'labor as an end in itself', i.e., capitalism.


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Familial Hyperinsulinism.
Glaser B(1).In: Adam MP, Ardinger HH, Pagon RA, Wallace SE, Bean LJH, Stephens K, Amemiya A, editors. GeneReviews [Internet]. Seattle (WA): University of Washington,Seattle; 1993-2018.2003 Aug 19. (1)Endocrinology and Metabolism, Hadassah-Hebrew University Medical Center, Jerusalem, Israel CLINICAL CHARACTERISTICS: Familial hyperinsulinism (referred to as FHI in thisGeneReview) is characterized by hypoglycemia that ranges from severeneonatal-onset, difficult-to-manage disease to childhood-onset disease with mild symptoms and difficult-to-diagnose hypoglycemia. Neonatal-onset disease manifestswithin hours to two days after birth. Childhood-onset disease manifests duringthe first months or years of life. In the newborn period, presenting symptoms maybe nonspecific, including seizures, hypotonia, poor feeding, and apnea. In severecases, serum glucose concentrations are typically extremely low and thus easilyrecognized, whereas in milder cases, variable and mild hypoglycemia may make the diagnosis more difficult. Even within the same family, disease manifestations canrange from mild to severe. Individuals with autosomal recessive familialhyperinsulinism, caused by pathogenic variants in either ABCC8 or KCNJ11(FHI-KATP), tend to be large for gestational age and usually present with severe refractory hypoglycemia in the first 48 hours of life; affected infants usuallyrespond only partially to diet or medical management (i.e., diazoxide therapy)and thus may require pancreatic resection. Individuals with autosomal dominantFHI-KATP tend to be appropriate for gestational age at birth, to present atapproximately age one year (range: 2 days - 30 years), and to respond to diet anddiazoxide therapy. Exceptions to both of these generalities have been reported.FHI-GCK, caused by patho Continue reading >>
Location (UCSC)Chr 11: 17.39 17.48 MbChr 7: 46.1 46.18 Mb ATP-binding cassette transporter sub-family C member 8 is a proteinthat in humans is encoded by the ABCC8 gene.[5][6] ABCC8 orthologs [7]have been identified in all mammals for which complete genome data areavailable.The protein encoded by this gene is a member of the superfamily ofATP-binding cassette (ABC) transporters. ABC proteins transportvarious molecules across extra- and intra-cellular membranes. ABCgenes are divided into seven distinct subfamilies (ABC1, MDR/TAP, MRP,ALD, OABP, GCN20, White). This protein is a member of the MRPsubfamily which is involved in multi-drug resistance. This proteinfunctions as a modulator of ATP-sensitive potassium channels andinsulin release. Mutations and deficiencies in this protein have beenobserved in patients with hyperinsulinemic hypoglycemia of infancy, anautosomal recessive disorder of unregulated and high insulinsecretion. Mutations have also been associated withnon-insulin-dependent diabetes mellitus type II, an autosomal dominantdisease of defective insulin secretion. Alternative splicing of thisgene has been observed; however, the transcript variants have not beenfully described.[8] 1 See also2 References3 External links4 Further reading ATP-binding cassette transporterSulfonylurea receptor ^ a b c GRCh38: Ensembl release 89: ENSG00000006071 - Ensembl, May2017^ a b c GRCm38: Ensembl release 89: ENSMUSG00000040136 - Ensembl, May2017^ "Human PubMed Reference:".^ "Mouse PubMed Reference:".^ Glaser B, Chiu KC, Anker R, Nestorowicz A, Landau H, Ben-Bassat H,Shlomai Z, Kaiser N, Thornton PS, Stanley CA, et al. (Nov 1994)."Familial hyperinsulinism maps to chromosome 11p14-15.1, 30 cMcentromeric to the insulin gene". Nat Genet. 7 (2): 1858.doi:10.1038/ng0694-185. PMID792 Continue reading >>
Congenital Hyperinsulinism - Genetics Home Reference
What does it mean if a disorder seems to run in my family? What is the prognosis of a genetic condition? Congenital hyperinsulinism affects approximately 1 in 50,000 newborns. This condition is more common in certain populations, affecting up to 1 in 2,500 newborns. What information about a genetic condition can statistics provide? Why are some genetic conditions more common in particular ethnic groups? Congenital hyperinsulinism is caused by mutations in genes that regulate the release (secretion) of insulin, which is produced by beta cells in the . Insulin clears excess sugar (in the form of glucose) from the bloodstream by passing glucose into cells to be used as energy. congenital hyperinsulinism lead to over-secretion of insulin from beta cells. Normally, insulin is secreted in response to the amount of glucose in the bloodstream: when glucose levels rise, so does Congenital hyperinsulinism can have different inheritance patterns, usually depending on the form of the condition. At least two forms of the condition have been identified. The most common form is the diffuse form, which occurs when all of the beta cells in the pancreas secrete too much insulin. The focal form of congenital hyperinsulinism occurs when only some of the beta cells over-secrete insulin. congenital hyperinsulinism is inherited in an , which means both copies of the gene in each cell have mutations. The parents of an individual with an autosomal recessive condition each carry one copy of the mutated gene, but they typically do not show signs and symptoms of the condition. Less frequently, the diffuse form is inherited in an , which means one copy of the altered gene in each cell is sufficient to cause the disorder. congenital hyperinsulinism is more complex. For most genes, both copies are t Continue reading >>
Hyperinsulinemic Hypoglycemia, Familial, 2
for Hyperinsulinemic Hypoglycemia, Familial, 2 UniProtKB/Swiss-Prot : 71 Familial hyperinsulinemic hypoglycemia 2: Most common cause of persistent hypoglycemia in infancy. Unless early and aggressive intervention is undertaken, brain damage from recurrent episodes of hypoglycemia may occur. MalaCards based summary :Hyperinsulinemic Hypoglycemia, Familial, 2, also known as nesidioblastosis, is related to hypoglycemia and hyperinsulinemic hypoglycemia , and has symptoms including seizures, vomiting and diarrhea. An important gene associated with Hyperinsulinemic Hypoglycemia, Familial, 2 is KCNJ11 (Potassium Voltage-Gated Channel Subfamily J Member 11), and among its related pathways/superpathways are Integration of energy metabolism and Type II diabetes mellitus . Affiliated tissues include brain and pancreatic islet, and related phenotypes are endocrine/exocrine gland and homeostasis/metabolism Wikipedia : 72 Nesidioblastosis is a controversial medical term for hyperinsulinemic hypoglycemia attributed to... more... Continue reading >>
Familial Hyperinsulinism
Synonyms: FHI, Persistent Hyperinsulinemic Hypoglycemia of Infancy (PHHI) Familial hyperinsulinism (referred to as FHI in this GeneReview) is characterized by hypoglycemia that ranges from severe neonatal-onset, difficult-to-manage disease to childhood-onset disease with mild symptoms and difficult-to-diagnose hypoglycemia. Neonatal-onset disease manifests within hours to two days after birth. Childhood-onset disease manifests during the first months or years of life. In the newborn period, presenting symptoms may be nonspecific, including seizures, hypotonia, poor feeding, and apnea. In severe cases, serum glucose concentrations are typically extremely low and thus easily recognized, whereas in milder cases, variable and mild hypoglycemia may make the diagnosis more difficult. Even within the same family, disease manifestations can range from mild to severe. Individuals with autosomal recessive familial hyperinsulinism, caused by pathogenic variants in either ABCC8 or KCNJ11 (FHI-KATP), tend to be large for gestational age and usually present with severe refractory hypoglycemia in the first 48 hours of life; affected infants usually respond only partially to diet or medical management (i.e., diazoxide therapy) and thus may require pancreatic resection. Individuals with autosomal dominant FHI-KATP tend to be appropriate for gestational age at birth, to present at approximately age one year (range: 2 days - 30 years), and to respond to diet and diazoxide therapy. Exceptions to both of these generalities have been reported. FHI-GCK, caused by pathogenic variants in GCK, may be much milder than FHI-KATP; however, some persons have severe, diazoxide-unresponsive hypoglycemia. FHI-HADH, caused by pathogenic variants in HADH, tends to be relatively mild, although severe case Continue reading >>
Arup Genetics Test Menu
The test menu below is a complete list of ARUPs inherited disease testing. A patient history form is requested for many of these genetic tests. Click on the test code or test name to obtain a link to the patient history form for the desired test. DYPD 5-Fluorouracil toxicity5-FU toxicity5-FU toxicity5FU toxicityAdrucil (DPYD) Genotyping, 3 MutationsXeloda (capecitabine) (DPYD) Genotyping, 3 Mutations DPDUftoral (tegafur/uracil) (DPYD) Genotyping, 3 Mutations Acylcarnitine Quantitative Profile, Plasma ACYLCARN3-Hydroxy-3-Methylglutaryl-Coenzyme A Lyase Deficiency3-Hydroxy-3-Methylglutaryl-CoA Lyase DeficiencyHMG-CoA Lyase DeficiencyHMGCLHydroxymethylglutaryl-CoA lyase, mitochondrialHMG Lyase Deficiency3-Hydroxyacyl-Coenzyme A Dehydrogenase DeficiencyHADH DeficiencyHADHHydroxyacyl-coenzyme A dehydrogenase, mitochondrialSCHAD3-Methylglutaconic Aciduria Type 13-Methylglutaconyl-CoA Hydratase DeficiencyAUHMethylglutaconyl-CoA hydratase, mitochondrialMGA1MGA 1Acyl-CoA Dehydrogenase, Short/Branched Chain Deficiency2-Methyl Butyryl CoA Dehydrogenase DeficiencyAcyl-CoA Dehydrogenase, Short/Branched Chain DeficiencyACADSBShort/branched chain specific acyl-CoA dehydrogenase, mitochondrialSCADCarnitine Palmitoyltransferase IA DeficiencyCPT1A DeficiencyHepatic Carnitine Palmitoyltransferase 1 DeficiencyHepatic CPT1L-CPT 1 DeficiencyCPT1ACarnitine O-palmitoyltransferase 1, liver isoformCPT 1CPT 1ACPTICPT ICarnitine Palmitoyltransferase II DeficiencyCPT II DeficiencyCPT2Carnitine O-palmitoyltransferase 2, mitochondrialCPTIICPT 2Carnitine-Acylcarnitine Translocase DeficiencyCACT DeficiencySLC25A20Mitochondrial carnitine/acylcarnitine carrier proteinEthylmalonic EncephalopathyProtein ETHE1, mitochondrialETHE1EMAEthyl malonic acidEthylmalonic acidGA1Glutaricacidemia Type 1GAIGlutaricaci Continue reading >>
Congenital Hyperinsulinism
Research helps us better understand diseases and can lead to advances in diagnosis and treatment. This section provides resources to help you learn about medical research and ways to get involved. ClinicalTrials.gov lists trials that are related to Congenital hyperinsulinism. Click on the link to go to ClinicalTrials.gov to read descriptions of these studies. Please note:Studies listed on theClinicalTrials.govwebsite are listed for informational purposes only; being listed does not reflect an endorsement by GARD or the NIH. We strongly recommend that you talk with a trusted healthcare provider before choosing to participate in any clinical study. These resources provide more information about this condition or associated symptoms. The in-depth resources contain medical and scientific language that may be hard to understand. You may want to review these resources with a medical professional. Genetics Home Reference (GHR) contains information on Congenital hyperinsulinism. This website is maintained by the National Library of Medicine. GeneReviews provides current, expert-authored, peer-reviewed, full-text articles describing the application of genetic testing to the diagnosis, management, and genetic counseling of patients with specific inherited conditions. Medscape Reference provides information on this topic. You may need to register to view the medical textbook, but registration is free. The Monarch Initiative brings together data about this condition from humans and other species to help physicians and biomedical researchers. Monarchs tools are designed to make it easier to compare the signs and symptoms (phenotypes) of different diseases and discover common features. This initiative is a collaboration between several academic institutions across the world and is f Continue reading >>
Congenital Hyperinsulinism
"PHHI" redirects here. It is not to be confused with Wheeler Army Airfield . Insulin (which this condition creates in excess) Congenital hyperinsulinism is a medical term referring to a variety of congenital disorders in which hypoglycemia is caused by excessive insulin secretion. [1] [2] Congenital forms of hyperinsulinemic hypoglycemia can be transient or persistent, mild or severe. These conditions are present at birth and most become apparent in early infancy.Mild cases can be treated by frequent feedings, more severe cases can be controlled by medications that reduce insulin secretion or effects [1] [3] There are differenct types of congenital hyperinsulinism as indicated below: Hyperammonemic hyperinsulinism (glutamate dehydrogenase gain-of-function mutations) Beckwith-Wiedemann syndrome (thought to be due to hyperinsulinism but pathophysiology still uncertain: 11p15 mutation or IGF2 excess) Paternal SUR1 mutation with clonal loss of heterozygosity of 11p15 Paternal Kir6.23 mutation with clonal loss of heterozygosity of 11p15 Hypoglycemia in early infancy can cause jitteriness, lethargy , unresponsiveness, or seizures . [4] The most severe forms may cause macrosomia in utero, producing a large birth weight, often accompanied by enlargement of the heart ( cardiomegaly ) and liver . Milder hypoglycemia in infancy causes hunger every few hours, with increasing irritability or lethargy if feeding is delayed.Congenital hyperinsulinism often becomes apparent later via an inability to sleep through the night. Sometimes an unusual stress like an illness precipitates a severe hypoglycemic episode.Milder forms have occasionally been detected by investigation of family members of infants with severe forms. Adults with the mildest degrees of congenital hyperinsulinism may si Continue reading >>
Genetic Testing For Familial Hyperinsulinism | Blueprint Genetics
Label the sample tube with your patients name, date of birth and the date of sample collection. Note that we do not accept DNA samples isolated from formalin-fixed paraffin-embedded (FFPE) tissue. Familial hyperinsulinism (FHI) is characterized by hypoglycemia that can have an onset neonatally or later during childhood. The disease presentation can vary considerably even within one family. It can present as severe with a very low glucose concentration or with variable and milder hypoglycemia. The clinical utility of this panel for familial hyperinsulinism is 50-60%. Most of the patients with familial hyperinsulinism have a mutatedABCC8gene, while mutations inKCNJ11, GLUD1 andHFN4Ahave each been found in approximately 5% of patients. Congenital isolated hyperinsulinism is the most common cause of severe and persistent hypoglycemia in neonatal period. The prevalence has been estimated at 1:50,000 live births, with much higher numbers in certain more homogenous populations. Infants of diabetic mothers may present with a clinical picture identical to that of FHI and this panel has differential diagnostic power to diagnose cases with genetic causes of transient hypoglycemia in newborns. This panel also includes the Glycogen Storage Disorder Panel genes for differential diagnostic purposes, since hepatomegaly due to glycogen storage disorder might not be visible in the newborn period. Furthermore, the panel includes genes relevant in additional related phenotypes such as maturity onset diabetes of the young (MODY) or exercise-induced hyperinsulinism. Insulinoma and drug-induced hypoglycemia should also be considered in later-onset hyperinsulinism phenotypes. Genes in the Hypoglycemia, Hyperinsulinism and Ketone Metabolism Panel and their clinical significance * Some, or all, Continue reading >>
Permanent Neonatal Diabetes Mellitus
Initial Posting: February 8, 2008; Last Update: July 29, 2016. Permanent neonatal diabetes mellitus (PNDM) is characterized by the onset of hyperglycemia within the first six months of life (mean age: 7 weeks; range: birth to 26 weeks). The diabetes mellitus is associated with partial or complete insulin deficiency. Clinical manifestations at the time of diagnosis include intrauterine growth retardation, hyperglycemia, glycosuria, osmotic polyuria, severe dehydration, and failure to thrive. Therapy with insulin corrects the hyperglycemia and results in dramatic catch-up growth. The course of PNDM varies by genotype . Persistent hyperglycemia (plasma glucose concentration >150-200 mg/dL) in infants younger than age six months establishes the diagnosis of PNDM. Molecular testing is recommended: identification of pathogenic variant (s) in ABCC8 or KCNJ11 can guide treatment. Treatment of manifestations: Start rehydration and intravenous insulin infusion promptly after diagnosis. When the infant is stable and tolerating oral feedings begin subcutaneous insulin therapy. Children with pathogenic variants in ABCC8 or KCNJ11 can be treated with oral sulfonylureas; all others require long-term insulin therapy. High caloric intake is necessary for appropriate weight gain. Pancreatic enzyme replacement therapy is required for those with exocrine pancreatic insufficiency. Prevention of secondary complications: Aggressive treatment and frequent monitoring of blood glucose concentrations to avoid acute complications such as diabetic ketoacidosis and hypoglycemia and reduce the long-term complications of diabetes mellitus. Surveillance: Lifelong monitoring of blood glucose concentrations at least four times a day; periodic developmental evaluations. After age ten years, annual screen Continue reading >>
Congenital Hyperinsulinism: Current Trends In Diagnosis And Therapy
Orphanet Journal of Rare Diseases20116:63 Arnoux et al; licensee BioMed Central Ltd.2011 Congenital hyperinsulinism (HI) is an inappropriate insulin secretion by the pancreatic -cells secondary to various genetic disorders. The incidence is estimated at 1/50, 000 live births, but it may be as high as 1/2, 500 in countries with substantial consanguinity. Recurrent episodes of hyperinsulinemic hypoglycemia may expose to high risk of brain damage. Hypoglycemias are diagnosed because of seizures, a faint, or any other neurological symptom, in the neonatal period or later, usually within the first two years of life. After the neonatal period, the patient can present the typical clinical features of a hypoglycemia: pallor, sweat and tachycardia. HI is a heterogeneous disorder with two main clinically indistinguishable histopathological lesions: diffuse and focal. Atypical lesions are under characterization. Recessive ABCC8 mutations (encoding SUR1, subunit of a potassium channel) and, more rarely, recessive KCNJ11 (encoding Kir6.2, subunit of the same potassium channel) mutations, are responsible for most severe diazoxide-unresponsive HI. Focal HI, also diazoxide-unresponsive, is due to the combination of a paternally-inherited ABCC8 or KCNJ11 mutation and a paternal isodisomy of the 11p15 region, which is specific to the islets cells within the focal lesion. Genetics and 18F-fluoro-L-DOPA positron emission tomography (PET) help to diagnose diffuse or focal forms of HI. Hypoglycemias must be rapidly and intensively treated to prevent severe and irreversible brain damage. This includes a glucose load and/or a glucagon injection, at the time of hypoglycemia, to correct it. Then a treatment to prevent the recurrence of hypoglycemia must be set, which may include frequent and gl Continue reading >>
Approach To Hypoglycemia In Infants And Children
Approach to hypoglycemia in infants and children Approach to hypoglycemia in infants and children Approach to hypoglycemia in infants and children Correspondence to: Kajal Gandhi, DO, MPH. Section of Endocrinology, Nationwide Childrens Hospital, 700 Childrens Drive, Columbus, OH 43205, USA. Email: Abstract: Hypoglycemia is a heterogeneous disorder with many different possible etiologies, including hyperinsulinism, glycogen storage disorders, fatty acid disorders, hormonal deficiencies, and metabolic defects, among others. This condition affects newborns to adolescents, with various approaches to diagnosis and management. This paper will review current literature on the history of hypoglycemia, current discussion on the definition of hypoglycemia, as well as etiologies, diagnosis, and management. Keywords: Endocrinology; hypoglycemia; children Submitted Sep 25, 2017. Accepted for publication Oct 10, 2017. Hypoglycemia may be caused by various defective pathways in the production and metabolism of glucose, as well as defective pathways to maintain euglycemia. This review will discuss pertinent literature as it relates to the normal pathways for glucose utilization, history of hypoglycemia, select etiologies and approach to management. Throughout gestation, increasing glucose requirements by the growing fetus lead to transport of glucose through the placenta. This is performed by facilitated diffusion as the primary source of energy through the family of glucose transporter proteins (GLUT), primarily glucose transporter type 1 (GLUT1) through a concentration gradient which favors the fetus ( 1 - 3 ). Fetal glucose is mostly provided by maternal glucose supply ( 1 , 4 - 6 ), while fetal insulin functions primarily for growth ( 7 ). Although uterine glucose uptake is regula Continue reading >>
International Journal Of Endocrinology And Metabolism
International Journal of Endocrinology and Metabolism 1 Department of Pediatric Endocrinology, Mofid Childrens Hospital, Shahid Beheshti University of Medical Sciences, Tehran, IR Iran 2 Genetic Counseling Division, Mofid Childrens Hospital, Shahid Beheshti University of Medical Sciences, Tehran, IR Iran 3 Department of Medical Genetics, School of Medicine, Tehran University of Medical Sciences, Tehran, IR Iran 4 School of Medicine, Shahid Beheshti University of Medical Sciences, Tehran, IR Iran * Corresponding author: Susan Akbaroghli, Mofid Childrens Hospital, Tehran, IR Iran. Tel: +98-2122227033, Fax: +98-2122227033, E-mail: [email protected] International Journal of Endocrinology and Metabolism: October 2016, 14 (4) ; e37311.Published Online: September 10, 2016 Article Type: Case Report; Received : March 28, 2016; Revised : August 22, 2016; Accepted : September 5, 2016 DOI: To Cite : Alaei M R, Akbaroghli S, Keramatipour M, Alaei A. A Case Series: Congenital Hyperinsulinism, Int J Endocrinol Metab. 2016 ;14(4):e37311. doi: 10.5812/ijem.37311 . Introduction: Congenital hyperinsulinism is a rare inherited disease caused by mutations in genes responsible for -cells function in glucose hemostasis leading to profound and recurrent hypoglycemia. The incidence of the disease is about 1 in 50000 newborns. Mutations in at least 8 genes have been reported to cause congenital hyperinsulinism. Mutations in ABCC8 gene are the most common cause of the disease that account for approximately 40% of cases. Less frequently KCNJ11 gene mutations are responsible for the disease. Mutations in other genes such as HADH account for smaller fractions of cases. In nearly half of the cases the cause remains unknown. Case Presentation: During the period between 2005 and 2010, a total Continue reading >>
- International Textbook of Diabetes Mellitus, 4th Ed., Excerpt #82: Insulin Actions In Vivo: Glucose Metabolism Part 9 of 9
- CBD Oil And Diabetes - The Positive Effects Of CBD On Insulin And Metabolism
- Exercise and Glucose Metabolism in Persons with Diabetes Mellitus: Perspectives on the Role for Continuous Glucose Monitoring
Congenital Hyperinsulinism Sequencing Panel - Preventiongenetics
Congenital Hyperinsulinism Sequencing Panel We are happy to accommodate requests for single genes or a subset of these genes. The price will remain the list price. If desired, free reflex testing to remaining genes on panel is available. Alternatively, a single gene or subset of genes can also be ordered on our PGxome Custom Panel. Parental targeted testing for all probands with uncertain, likely pathogenic or pathogenic variants in the ABCC8 and KCNJ11 genes are free of charge. For ordering sequencing of targeted known variants, please proceed to our Targeted Variants landing page. The great majority oftests are completed within 20 days. In a cohort of 417 CHI patients studied at the Hyperinsulinism Center in The Childrens Hospital of Philadelphia (CHOP) (Snider et al. 2013), all nine genes were tested. Mutations were identified in 91% (272 of 298) of diazoxide-unresponsive probands (ABCC8, KCNJ11, and GCK), and in 47% (56 of 118) of diazoxide-responsive probands (ABCC8, KCNJ11, GLUD1, HADH, UCP2, HNF4A, and HNF1A). In another cohort of 300 CHI patients studied in United Kingdom (Kapoor et al. 2013), mutations were identified in 45.3% of patients (136/300) in eight tested genes (ABCC8, KCNJ11, GLUD1, GCK, HADH, SLC16A1, HNF4A and HNF1A).KATP (ABCC8 and KCNJ11) mutations were the most common genetic cause identified (109/300, 36.3%). Mutations in ABCC8/KCNJ11 were identified in 92 (87.6%) diazoxide-unresponsive patients (n=105). Among the diazoxide-responsive patients (n=183), mutations were identified in 41 patients (22.4%), including mutations in ABCC8/KCNJ11 (15), HNF4A (7), GLUD1 (16) and HADH (3). The great majority oftests are completed within 20 days. Clinical sensitivity for gross deletions and duplications in the HADH gene cannot be predicted as few patients h Continue reading >>
Congenital Hyperinsulinism
"PHHI" redirects here. It is not to be confused with Wheeler Army Airfield . Congenital hyperinsulinism ,CHI, is a medical term referring to a variety of congenital disorders in which hypoglycemia is caused by excessive insulin secretion. [4] [3] Congenital forms of hyperinsulinemic hypoglycemia can be transient or persistent, mild or severe. These conditions are present at birth and most become apparent in early infancy. Mild cases can be treated by frequent feedings, more severe cases can be controlled by medications that reduce insulin secretion or effects [4] [5] Insulin (which this condition creates in excess) Hypoglycemia in early infancy can cause jitteriness, lethargy , unresponsiveness, or seizures . [1] The most severe forms may cause macrosomia in utero, producing a large birth weight, often accompanied by abnormality of the pancreas. Milder hypoglycemia in infancy causes hunger every few hours, with increasing jitteriness or lethargy. Milder forms have occasionally been detected by investigation of family members of infants with severe forms, adults with the mildest degrees of congenital hyperinsulinism have a decreased tolerance for prolonged fasting. Other presentations are: [4] [5] [3] [6] The variable ages of presentations and courses suggest that some forms of congenital hyperinsulinism, especially those involving abnormalities of KATP channel function, can worsen or improve with time the potential harm from hyperinsulinemic hypoglycemia depends on the severity, and duration. Children who have recurrent hyperinsulinemic hypoglycemia in infancy can suffer harm to the brain [4] The cause of congenital hyperinsulinism has been linked to anomalies in nine different genes. [7] The diffuse form of this condition is inherited via the autosomal recessive manne Continue reading >>


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To find an appropriate position in business strategies to make use of Search Engine Marketing is more essential. It helps clients to shoot up their business through innovative and efficient methods. Crossing the border of the county, the business world has been globalized with the help of Internet. In world business scenario new challenges are inevitable and with properly utilizing these challenges can be converted into successful venture.
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Terms and Conditions ("Terms")
Last updated: (1/1/19)
Please read these Terms and Conditions ("Terms", "Terms and Conditions") carefully before using the https://ecrease.com website operated by Ecrease,LLC ("us", "we", or "our").
Your access to and use of the Service is conditioned on your acceptance of and compliance with these Terms. These Terms apply to all visitors, users and others who access or use the Service.
By accessing or using the Service you agree to be bound by these Terms. If you disagree with any part of the terms then you may not access the Service.
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Virus Wars iPhone App Review
iPhoneGlance Rating [rating=4] Virus Wars developed by dash and dot is highly addictive game. At first it appeared it was one of those games that issue you with a simple strategy, one that would have the user fight against the baddies (worms, Trojans and viruses). It's more like a digital tic-tac-toe. The app is simple and clean. "Each player can make 3 steps per 1 turn from his corner. Next step can be done in…Read More


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Currency: £ GBP | € EURO | $ USD
Release Date: 15/12/2014
Discs: 2
Reissue of the classic 1978 album.
Heavily influenced by the lighter side of the American R&B scene, the Stylistics, Chi-Lites, et al, The In Crowd created reggae with a decided urban flair, which proved popular on both sides of the Atlantic.
The band's sublime, sweet harmonies, bubbly rhythms, and arrangements edged with funk, blues, or urban flavours went down very easily.
Disc One
1. His Majesty Is Coming
2. Beg You a Two Cent
3. More Employment
4. Back a Yard
5. Let's Go to the Party
6. We Play Reggae
7. A Little Bit of Reggae
8. Slave Ship
9. You Facety Whitey
10. Born in Ethiopia
11. Power of the Spirit
12. Daughter Child
13. Reggae Groove
14. Life (12" Mix) (by Fil Callender)
15. Riding High (12" Mix)
16. No Money
Disc Two
1. Baby My Love (Feat. Jah Stitch)
2. Love Harmony (By Barry Bailey)
3. Marcus Garvey's Back In Town (12" Mix)
4. Dedicated To Jah
5. Man From New Guinea
6. Add A Little Light
7. Introducing Little Dread
8. Sweet Man (By Fil Callender)
9. Getting Cozy (Extended Mix)
10. Gamesta
11. Rasta Man No Evil
12. Everybody Wants To Be Loved
13. In My Arms
14. Rub A Dub Time (12" Mix) (By Fil Callender)
15. Give Us More Time


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Bring yourself back to the days of sitting in your plush, elegant study smoking a cigar and drinking an aged whiskey that most people can't pronounce. With HomCom's aged leather armchair you can bring the 'good old days' back with an elegant, modern twist. The material of this chair beautifully emulates natural leather without worrying about the upkeep and maintenance that comes with old leather surfaces. The faux leather is textured and smooth like natural leather, but it can be easily wiped down with a damp cloth to reinvigorate its shine. Built on a sturdy pine wood frame, this chair is cushioned by plush medium density foam for lasting, comfortable sitting. The bucket design is ergonomically molded to support your back and sides. Finishing off this already exquisite design are brass nailhead accent studs, giving this already luxurious chair a pop of unique character.
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- Unique brass nailhead accents
- Durable pine wood frame
- Ergonomic curved back and armrests
- Easy clean surface; Just wipe with a damp cloth
- Plush medium density foam cushioning
- Color: Black
- Material: PU + Sponge + Pine
- Overall Size: 28" x 24" x 28"
- Seat Size: 19" x 19" x 3.5"
- Seat pad: 3" Thickness
- Legs Dimension: 7.9"x (1.4"~1.8") x (1.4"~1.8")
- 3.1" removable cushion


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We are excited about bringing you this update about our fall mission trip to Lima. Our focus has been on presenting conferences, as well as ministering in area churches, and our schedule is already almost full!
On Friday, November 4, we I will be presenting a Prayer Workshop, teaching participants how to use the Lord's Prayer as a six point outline for prayer.
We're going to do a one-evening marriage conference at the Evangelical Church of Christ of San Martin. Pastor Juan de Dios and his wife, Cristina, have been dear friends of ours for over fifteen years, and being in their church is like being at home.
Yesterday it was confirmed that Marsha will be a primary speaker at a women's conference. She had been scheduled to do one session, sharing her testimony. However, one of the primary speakers has had to cancel due to illness. It's a three day conference beginning on November 10, and the theme is the Holy Spirit's work in the lives of believers.
On Saturday, November 12, we will present our God's Blueprint for Marriage Workshop at Emanuel Bible Church of San Miguel. We preached at that church on a Sunday last year and were very well received, and we sold out of the Spanish version of our book. We're looking forward to being back with them this year for the marriage conference.
Additionally, we are preaching in several churches, and visiting old friends and making new ones.
Please continue to pray for us, that the anointing of God would be with us. And, be praying also for provision for the trip. Thank you so much for being a part of our work in Peru. God bless you all.
In His Love,
Jim and Marsha Dixon


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Those who could afford the rations were
not all pleased with the system, as Sir John
himself pointed out: "I've taken criticism all
my life but I did particularly in that period."
Vivienne Gardner remembers that her
mother, Bee Gilmore, was certainly one who
felt critical. She duly signed her family up for
their food rations at the Goody Shoppe (on
Reid Street) and soon wished for the coupons system implemented in England when
she realised she could not go to a different
store once the Goody Shoppe ran out.
Perhaps the deprivation many people feltmost acutely was bread, the result of a severeshortage of flour. As Bee Gilmore said, "AllI could get for three days was half a loaf ofmouldy bread!" In his book The Story ofBermuda and Her People, W.S. Zuill explainshow mouldy bread was deliberately sold todeter people from wanting to eat it. Sir Johnrecalled a ship mercifully coming in withflour when there was just one day's sup-ply left. Sometimes, as Ann Zuill Williamsrecounts, the flour wasn't exactly in pristinecondition as they would find weevils "minia-ture black beetles crawling through the bag."Butter and sugar were also rationed through-out the war and Ann Zuill Williams describeshow their new rations were delivered to theirhome, Orange Grove, in Smiths Parish everyFriday by horse and cart. Each member of thehousehold was allowed two ounces of eachwhich her mother, Christiana (Kitty) Zuill,would measure on the kitchen scales. She wouldthen use her empty Yardley face cream jars tostore each person's portion and if there wasenough left at the end of the week would useit to make fudge as a treat. Butter, Ann ZuillWilliams explains, easily went rancid so it wasdifficult to store. Sugar was easier, but VivienneGardiner remembers a rule that said no morethan one and a half pounds of sugar could bestored in a household. It was a strange rule, shesays, in that households were at the same timeadvised to lay in stores of food.
As is true in any time of shortage, a lotdepended on whom you knew. Any socialconnection with the American base washugely advantageous, as the Gilmores foundout when Vivienne's sister Beryl secured asecretarial position at the naval base. "Thiswas wonderful for our family since Beryl,who was a very pretty girl, had all the officerseager to help her in any way they could.Consequently, cases of wonderful food werebrought to our house, delivered by handsomeyoung men in Jeeps."Grace Rawlins's parents, Mary and JosephRawlins, were caretakers of South-lands on the south shore duringthe war when the property wastaken over by the American navy.
They lived in the main house where
the officers were billeted, while
the enlisted men camped in tents
along the shoreline. Mary Rawlins cooked for the officers which
meant that the Rawlins family ate
well. Young Grace, who was three
or four at the time, distinctly remembers steak and ice cream as
well as vegetables.
Bill Cooke remembers appreciating "an unexpected benefit"
of clerking the Bermuda Mineral Company's
trucks when they delivered soft drinks to the
canteens and messes of the military camps: a
small container of powdered saccharine to supplement his ration of sugar, which he describes
as "the most missed item of our allowance."
Sometimes pure luck came into play. As Vivienne Gardner recalls from reading a letter written by her mother, "when Will [Vivienne's father] went sailing, he once found a cask floating
out to sea that was 'evidently lost off a lifeboat.' It
was full of rations, tins of food and biscuits with
added vitamins for nourishment, malted milk
tablets and pork meat, all in perfect condition."
"...[the bags were] made of white cotton, often with a
flower print, so even if the flour was full of weevils, the
bags were good for making underpants and petticoats.
Germans, with the result there was no bran
or oats for the horses and cows. (The grass in
Bermuda did not have enough nourishment.)
As a result cows stopped producing milk and
the horses became ill as a result of starvation.
"They were in a desperate way." Vivienne
Gardner writes that "a law was enacted that no
horses were allowed to be driven faster than
a walk but as horses and carts were the only
mode of delivery many drivers disobeyed the
ruling." She remembers she would "bicycle
through town and scream at any driver who
was whipping his half-dead horse to make it
trot." Some horses had to be shot.
Wartime shortages also affected shoes and
clothing and for these items ration books
were supplied. As Ann Zuill Williams writes,
"For growing children it must have been
difficult because we outgrew our shoes faster
than the ration books allowed. Shoes were
passed from child to child." Ruth Thomas
recalls being in trouble for being hard on her
shoes and outgrowing them too fast. Many
a child did without. Where clothing was
concerned, people were very resourceful. For
example, the hundredweight bags in which
flour was shipped had more lasting use than
the flour itself. They were, according to The
Back Yard, "made of white cotton, often with
a flower print, so even if the flour was full
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Spammers hit the cloud
Spam increased globally in Q2 2011 as spam networks recover from take-down
Spammers are beginning to hit cloud services in Q2 2011, with Kaspersky Lab seeing several instances of emails containing links to the Google cloud services, which redirected users to advertising sites or phishing pages.
According to Kaspersky, users are less likely to suspect pages like these because they are located on popular resources and the connection is performed via the HTTPS protocol which supports encryption.
The share of spam in mail traffic averaging 82.5% in Q2 2011, an increase of 3.9 percentage points compared to Q1 and 0.3 percentage points higher than the average figure for 2010.
Spam networks that were taken down by internet security companies last year are slowly recovering, which accounts for the spam increase. Since the closures, spammers have had to apply a new set of tactics to keep them in business.
Q2 2011 saw an increase in the number of botnets, although they were quite small and none accounted for such large shares of spam traffic as Cutwail or Rustock did in the past.
According to Kaspersky Lab, this is because either the spammers still have not increased capacities to where they can send millions of spam emails daily or they are deliberately not risking everything on a single major botnet.
Sources of spam messages are more evenly spread out in Q2 2011, unlike in the past when three countries were regularly responsible for half of all world spam, zombie machines used to spread spam emails are now located in almost every country of the world.
Spammers' geographical expansion is now complete with no territories now left untouched by the botmasters.
In the Q2 2011, spam was sent almost exclusively from developing countries with India at the top of the list with an increase of 4.26 percentage points in spam sending, Brazil had increased by 3.14 percentage points and Indonesia was up 1.66 percentage points.
India's total contribution to spam volumes in Q2 was up by five percentage points compared to Q1 and totalled 14.06%.
Kaspersky Lab said that this is due to the presence of millions of unprotected, unpatched machines that can remain active in zombie networks for long periods of time, making India a happy hunting ground for botmasters.
The US also remains a comfortable place for the creators of botnets.
"Developing countries are attractive for botmasters due to the absence of effective anti-spam legislation and low IT security levels, while developed countries are of interest because of their fast, widely available Internet connections," said Darya Gudkova, head of Content Analysis & Research at Kaspersky Lab. "Therefore, it comes as no surprise that the US remains a very attractive bridgehead for cybercriminals creating botnets. The US may well have dropped out of the group of leading spam senders following the anti-botnet campaign by law enforcement agencies in autumn 2010, but after the command centers of a big botnet were closed, the cybercriminals started to create a new one almost straight away."
In February, the majority of malware sent to the US were banking Trojans, designed to steal users' financial data or to extort money from users, however, in March and April more than half of the Top 10 entries were Trojan downloaders for installing malware on a user's computer that added the infected machine to a botnet.
Emails containing malicious attachments continued to increase in Q2 2011: the average percentage of emails with malicious attachments increased by nearly 0.81 percentage points, reaching 3.86%.
Some of the worms detected not only collected email addresses and distributed themselves via mail traffic, some worms also installed other malicious programmes once they had penetrated a victim's computer.
Malicious attachments were found most frequently in email traffic received in Russia at 12.5% of emails, the US came second with 12.21%, an increase of 1.8 percentage points compared with Q1. Vietnam was third, accounting for 7.43% of all email antivirus detection activity an increase of +0.46 percentage points.


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QHSE Support - (Site Map) AIM > Statistical Charts > > The number of accidents by location is available via the stats and analysis ribbon.
This chart provides details of accidents by the locations added in the locations table. Again, this will no doubt become a management favourite as it instantly identifies those parts of the company that may warrant further investigation to help push accident figures downwards.
This chart provides details of accidents by locations and will no doubt become a management favourite as it instantly identifies which locations if targeted can most positively influence accidents statistics.
Pressing the Chart Preview brings up the the associated report for the users selected criteria.
Zoomed Extract
Note: the chart is the statistical relationship for accidents between the dates specified and locations. It will therefore vary depending on the time span requested.
On Safe Lines - Help file v8.4.01.04 : Copyright © 2020 On Safe Lines QHSE Software


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B.Sc. Media Graphics & Animation (Quick Enquiry)
Welcome to the creative world! This is the place where you can bring your imaginations into reality (pun intended)!
B.Sc. Media Graphics & Animation is the course as per the future needs. It helps you enter the vast digital field with a number of career opportunities. In short, it is your way out among all the boring, theory, non-productive basic courses like B. Com, B. A, or plain B. Sc. Even the glamorized fields like engineering and BCA are having a tough time in finding the right professional opportunities. This is where getting creative and walking hand-in-hand with the growing industry comes to your rescue.
Some Insights About the Course –
Designers' Institute is affiliated to Yashwantrao Chavan Maharashtra Open University, and so the B.Sc. Medial Graphics & Animation course degree will have certification from this reputed university. The certificate from a reputed university makes a lot of difference while applying for jobs and further career opportunities.
With B.Sc. Media Graphics & Animation degree course you shall become an all-around graphic and web designer. You shall be taken through all the parts and phases of educational level regarding graphics and animation. After completing the graphics and 3D animation course, you will be ready to enter the digital world and pursue any career you want on the digital screen.
Career Options After Completing VFX Animation Courses –
- Graphic Designer
- Web Designer
- Flash Designer
- Digital Artist
- Story Board Artist
- 2D and 3D Animator
- 3D Modeler
- VFX Artist
- Texturing Artist
- Rigging Artist
- Lighting & Rendering Artist
- Compositor
- Visualizer
- And many more upcoming positions
After completing the degree course of B.Sc. Media Graphics & Animation, you can enter any company with the digital expertise you have, as each company has a digital presence in today's world. You can even start your own brand with freelancing as your equipment.
Why Choose Designers' Institute for B.Sc. Media Graphics & Animation?
- YCMOU affiliated course with degree certification.
- Exposure to the field with seminars and industrial visits.
- Good infrastructure with quality teaching faculty.
- Flexible timings to suit your daily schedule.
- Direct entry to the professional field after completing the course.
- One of the best animation institute in Mumbai with reasonable animation courses fees.
- Located in the heart of Mumbai city, which makes it top search results 'best animation classes near me'.
Course Duration – 36 Months
Course Pattern – Yearly
Minimum Eligibility – Appeared 12th Examinations
Syllabus for the Three Years Graphics & Animation Degree Course –
- Introduction to Computers & Internet
- Drawing and Sketching
- Color Theory
- Typography
- Adobe Photoshop
- Adobe Illustrator
- Technical and Creative Writing
- Introduction to Multimedia and its Application
- Developing Presentations
- Design Principles
- Coral Draw
- Quark Express
- Introduction to Web Development
- Introduction to Flash
- Content Digitization
- Content Authoring on Web using Macromedia Dreamweaver
- Developing Dynamic Web Pages using Java and VB Scripts
- Video-Production Basics
- Story Boarding
- Visual Communication
- Audio-Editing: Sound Forge
- Video-Editing: Adobe Premier
- Advance Video Effects
- Animation Principles
- Introduction to Maya
- Character Set Up & Animation in Maya
- Advanced Maya
- Introduction to 3DS Max
- Advance 3DX Max
- Character Animation
- CG Film Making
- Project Work
- Environment Science


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18 ways your job is killing you
Photo: Flickr
If you're used to the hours of sedentary, stressful working conditions that come with your office job, you may want to know that this kind of working environment is killing you a lot faster than you think.
Aside from the stress that comes from tight deadlines, plenty of things you do every day in the workplace are slowly chipping away at you.
From the printer to your keyboard, the dangers presented in an office can have real effects on your physical well-being, just as mental strains can hurt you in the long-term.


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Arriva DAF Greenline Coaches
This page created 14th November 2005 by Ian Smith
Maidstone & District Plaxtons 2903-2910Despite the Government's interference in the bus and coach industry, making co-operation rather than competition illegal, Green Line managed to survive as a brand name, valued by several different operators of commuter services into London. This was particularly applicable to the Arriva group of companies. These included Maidstone and District, which had made its commuter services from Maidstone part of Green Line Associated Services from 1992, and had adopted Green Line branding in 1995.
In January 1998 Arriva Kent & Sussex (the current name for Maidstone & District) took delivery of eight DAF coaches with Plaxton bodywork for use on the Maidstone-London services. They received full Green Line livery and numbers 2903 to 2310.
2904 is seen on its intended duties, the 781 Greenline service from Maidstone to London, passing through New Cross in January 2005. At other times they can be found on Private Hires, such as 2903 passing Sevenoaks Station in July 2004.
Commuter's eye view of Greenline 2904, in a traffic jam on the A20 near Mottingham in August 2000. Some of the coaches have moved between the operating companies: 4055/2911 has variously worked Greenline for Arriva The Shires, Arriva Southend and Arriva Kent & Sussex. Here it is on a 797 Stevenage service approaching Victoria in October 2005.In August 2005 The Green Line services, along with the rest of Arriva Southern Counties coaching in the old M& D area, were hived off to New Enterprise. Services 781 and 784 continued to operate from the Maidstone depot. Most of the DAFs transferred to New Enterprise, with the exception of 2909, which transferred to Stevenage for the 797. Those that remained with New Enterprise were repainted into the company's white with red/navy blue ribbons. The "Green Line" services remained with New Enterprise until January 2009, when Chalkwell Services of Sittingbourne provided an alternative commuter service.
Arriva the Shires Plaxtons 4047-4056, 4064At the same time as Arriva took DAFs for Maidstone & District it bought an identical batch of ten Plaxton Premiere 320s for Luton & District, which was Arriva the Shires when they were delivered in November 1997. They were for Arriva's prestige airport route 757 (London (Victoria) - Luton Airport - Luton). The ten stayed with the 757 for two and a half years, then ten newer Plaxtons (see below) took over the prestige run and three of the Plaxtons (4047-4049) were cascaded onto Hemel Hempstead's commuter 758.
Two (4055 and 4056) went to Southend for their Green Line duties. The latter two moved on again in spring 2002 to Arriva Kent & Sussex for the Maidstone run, where they became 2911 and 2912. In 2005 they moved back north, but to Stevenage for the 797, regaining their AtS numbers.
Another four (4050-4053)were transferred from Luton-Dunstable to Hemel Hempstead in January 2006, after a batch of Alizees (4065-4069) arrived at Luton.
The other one, 4054, stayed at Luton as a spare and for contract work.
A further similar coach was acquired in November 2004 from Arriva Fox County (although it had a long history of other operators since new seven years previously). 4064 was used as a floating spare around the system, wherever needed.
4055 was going about its business on the 797 in Stevenage early one Sunday morning in June 2006. 4056 was in a queue of Green Line / Easyjet coaches loading up opposite Victoria Coach Station for a journey northwards in January 2008, ahead of Alizee 4059.
4055 joined other Green Line coaches at Wisley Airfield for Cobham's celebration of 80 years of Green Line in April 2010. It now wears a revised livery, with a bow-wave over the front wheel.This batch lasted with Arriva the Shires until 2011-12, for the most part (4056 went to New Enterprise in 2009, 4052 was written off after an accident in 2011)
Arriva the Shires Alizees 4057-4061, 4063Grey-Green, part of the Cowie/Arriva empire, had its own coaching fleet, including some DAFs with Van Hool Alizee bodywork, dating from spring 1995. Two were transferred to Arriva Cymru in April 1998. In August 1998 another two were transferred to Arriva the Shires for Green Line work, followed by three more in October and a sixth in April 1999. They went to Hemel Hempstead, where they donned Green Line livery with discrete 758 branding for the commuter 758.
Arriva the Shires' 4060 visited Showbus at Duxford in September 2005, as part of a Green Line 75 celebration. For some reason it was missing its Van Hool badge from the front.
4061 passes Victoria Station as it nears the end of its journey from Hemel Hempstead ready for the evening homeward surge in October 2005.The Van Hools lasted a relatively long time on front-line Green Line work. But by 2009 they were relatively long in the tooth, and once the big six-wheeler Acrons arrived on the Luton run these older coaches were pensioned off. Some went to other Arriva enterprises (4058,4059 to New Enterprise, 4057,4063 to Arriva Scotland West), while 4060 and 4061 became dedicated trainers. 4060 spent some time training in Green Line livery, which allowed it to be used as a spare when necessary. It was repainted in late 2010 in white with a dark blue skirt, for training and/or railway replacement work, but was scrapped in early 2011. 4061 went into training livery of white with grey skirt in 2009, and lasted until late 2011.
Arriva the Shires Plaxtons 4359, 4361-4369Another batch of ten Plaxton Premiere 320-bodied DAFs was bought by Arriva the Shires in July 2000. These went into service on the 757, replacing earlier similar Plaxtons.
Arriva the Shires' 4367 passes Victoria Station on 757 in October 2005 on a Luton Airport 757 service.Arriva took on responsibility for the Easyjet connection between Luton Airport and London, and in mid-2005 made a deal to offer a guaranteed proportion of seats on the 757 to Easyjet passengers at discounted fares. In conjunction with this 4361/3/5 were repainted in Easybus orange livery with prominent branding.
4366 went for scrap in 2006, after hitting a motorway gantry support in an horrific accident.
The batch stayed on the front line until the spring of 2009, when they were cascaded to Hemel Hempstead or Stevenage by the arrival of the six-wheeler Acrons. A variety of liveries appeared now, with the Easybus liveries disappearing from this batch. 4361 and 4365 went back into new bow-wave Green Line livery, along with 4359, 4367 and 4369; 4362, 4363 and 4368 went into schoolbus yellow; 4367 then was repainted again into interurban blue/blue.cream horn.
2011 saw four of the batch go into white with blue skirts for dual purpose training and/or railway replacement.
Arriva the Shires Van Hools 4065-4069At the end of 2005 six new Green Lines were received. These had Van Hool bodies with an attempt to cater for the wheelchair mobility market. The coaches retained the high floor needed for the underfloor luggage boot, but had a double front doorway. The second front door opened onto a wheelchair anchorage. This had to be booked in advance: when not booked the space was fitted with four seats. This arrangement resulted in a long front overhang.
The batch went into service on the 797 London-Stevenage service. One was damaged in a motorway accident in October 2007 and was impounded by police for two years. 4067 was then rebuilt and returned to service. Another Van Hool, given temporary fleet number 0412, was hired by Arriva for most of 2008.
4066 was at Cobham Museum's pre-hibernation day celebration of Green Line in October 2008.
Rebuilt 4067 joined other Green Line coaches at Wisley Airfield for Cobham's celebration of 80 years of Green Line in April 2010. The long front overhang and double front doorway are apparent.
National Express Van Hools 4070-4076Arriva Fox County operated National Express services between London and Derby (440), and London and Leeds (561), from depots in Derby and Leicester. These services used a fleet of Van Hool Alizees dating from 2003-5 in white National Express livery. In March 2010 a reorganisation of National Express services transferred these routes to Milton Keynes, together with the coaches. They became Arriva the Shires 4070-4076.
In January 2012 they became redundant with National Express. 4073 and 4074 went off to Wales, but the other five were repainted in Green Line livery and were transferred to Hemel Hempstead for second-line Green Line work.
Ian's Bus Stop Index DAF Greenline text histories photo refs


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- Course Delivery
- Blended
- Total Credits
- 30
- In-State Tuition Per Credit
- $342.00
- Out of State Tuition Per Credit
- $445.00
Earn your degree from the University of Nebraska at Omaha
Get the same diploma as on campus students
The blended Graduate P-K12 School Librarian Graduate Endorsement program combines core graduate education courses with literacy, technology and school library courses for those candidates who wish to direct a PK-12 school library.
Coursework for this nationally-recognized program builds competencies in:
- Public services
- Technical services
- Leadership and management of school library media programs
The program is specifically designed around the four pillars of:
- Evaluating research to determine impact on classroom practice
- Using assessment to drive instructional decision making
- Considering culturally responsive teaching practices to teach to student strengths
- Developing personalized theoretical frameworks for effective teaching
Career Outlook:
The 21st Century school librarian is in high demand as this educators role in supporting PK-12 student achievement through instruction and programming is recognized and required across the United States. School librarians heighten a students research and inquiry skills through a diverse array of print and digital tools. According to the Bureau of Labor Statistics, the job outlook for librarians will grow 9% from 2016 to 2026.
About Faculty
University of Nebraska online courses are taught by expert faculty who embody the qualities resulting from research experience and professional/field experience. Students learn from faculty with a variety of backgrounds, many of whom are published researchers in their fields. NU faculty who teach online do so in a way that enables maximum learning and because technology provides students with maximum access to education.
To be accepted to this program, you must have:
A bachelor's degree
3.00 GPA or above on a 4.0 scale
Taken the TOEFL or IELTS
(Only required if English is not your native language)
Official transcripts from all previous schools
Valid teaching certificate
To apply to this program:
- Complete and submit the online application for admissions
- Pay the $45 non-refundable application fee
- Submit separate application to academic department
Application Deadlines
- Fall Semester
- August 25
- Spring Semester
- December 1
- Summer Semester
- May 1
Core Courses
|Course Name
|Special Methods in School Library Media
Course Number: TED 8006
This course is designed to provide experiences for school professionals to be orchestrators of the learning environment by enhancing the knowledge, skills, competencies, and dispositions necessary to create effective learning environments in their specific content area specialization. Content of the course is determined by the discipline area.
|Capstone Practicum
Course Number: TED 8520
This three credit hour supervised practicum is designed to give librarians an overview of the public services, technical services, and leadership and management of libraries and information agencies. Prereq: Minimum 24 hours of library science coursework and permission of advisor. (Application deadline February 1 for Fall practicum; October 1 for Spring practicum)
|Teaching & Learning in Digital Environments
Course Number: TED 8596
This course introduces technology and technical literacies required of educators and information specialists in 21st Century libraries and classrooms. Course topics include information literacy, instructional design in digital environments, Web page design and construction, social networking and learning, and academic integrity.
|Children's Literature and Education
Course Number: TED 8650
Explores story, poetry, drama, and informational materials for children. Examines current issues, recent materials, and the theoretical and research base of this field to develop meaningful and creative learning, literacy, and library experiences for children. Literacy resources and activities for students from preschool through grade six.
|Young Adult Literature and Education
Course Number: TED 8660
Explores story, poetry, drama, and informational materials for young adults. Examines current issues, recent materials, and the theoretical and research base of this field to develop meaningful and creative learning, literacy, and library experiences. Literacy resources and activities for students in middle and high school.
|Reference Resources and Services
Course Number: TED 8716
Candidates will demonstrate an understanding of the theories, concepts and activities integral to reference resources and services in 21st Century libraries and information agencies. Candidates will demonstrate an understanding of effective search strategies and efficient use of both print and digital resources, design and promote information literacy instruction that is developmentally appropriate, and understand the legal and ethical responsibilities integral to positive and proactive reference services for patrons and diverse learners.
|Cataloging and Classification
Course Number: TED 8746
Candidates will demonstrate a basic understanding of the theories, concepts and activities of descriptive and subject cataloging and classification procedures of books in 21st Century libraries and information agencies using the Anglo-American Cataloguing Rules, 2nd edition, the Library of Congress and Dewey Decimal.
|Managing Collections in Libraries & Information Agencies
Course Number: TED 8766
Candidates will demonstrate an understanding of the theories, concepts and activities integral to proactive collection management in 21st Century libraries and information agencies. Candidates will demonstrate an understanding of community analysis, collection analysis, and the ability to conduct critical evaluations of a diverse array of information resources.
|Leadership & Management in Libraries & Information Agencies
Course Number: TED 8806
Candidates will demonstrate an understanding of the theories, concepts and activities integral to leading and managing 21st Century libraries and information agencies. Candidates will demonstrate an understanding of leadership principles and management strategies that engage policies and procedures that support the personal, academic and professional needs for patrons and diverse learners.
Cost for Nebraska Residents
Per Credit Hour
- Tuition: $342.00
- Fees: $52.25
- Total: $394.25
3 Credit Hours
- Tuition: $1026.00
- Fees: $156.75
- Total: $1182.75
Cost for Out of State Residents
Per Credit Hour
- Tuition: $445.00
- Fees: $52.25
- Total: $497.25
3 Credit Hours
- Tuition: $1335.00
- Fees: $156.75
- Total: $1491.75
Questions about tuition? We can help.
What's it like to take a program online?
Quality learning experience
The University of Nebraska has offered distance education courses for more than 100 years so you can expect a quality, rigorous experience. Online courses are often highly interactive with faculty and students communicating through e-mail, discussion forums and chat groups. You'll have direct access to world-class faculty – researchers who are experts in their fields or practitioners with real-world experience. Also, you'll be part of a community of learners and can benefit from the perspectives of students from across the globe.
Online learning gives you the flexibility and freedom to attend your classes wherever is convenient to you. You can save time and money by being able to continue to work and by avoiding relocation or travel costs. You will be required to complete assignments in a certain timeframe, but in most instances, you can log in and complete coursework during the time of day that works best for you. You aren't tied to a specific class time.
Service you expect from a leading University
Online learners at the University of Nebraska have access to the same student services available to on-campus students. An academic adviser will guide you along your journey, library services are available to help you excel in your program and career services are available when you are ready to take your next step. These are just a few of the services in place to help you succeed.


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Hey You –
Daddy is taking off…I shouldn't say it that way. It is only temporary. I'm heading to Korea to cover the 2018 Winter Olympics. I'll be back in 24 days. You just do me a favor. Stay in Mommy. We can be patient. Don't need to have you showing up early like Mommy likes to do.
See you soon.


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1Then Pilate had Jesus flogged with a lead–tipped whip.
2The soldiers made a crown of long, sharp thorns and put it on his head, and they put a royal purple robe on him.
3"Hail! King of the Jews!" they mocked, and they hit him with their fists.
4Pilate went outside again and said to the people, "I am going to bring him out to you now, but understand clearly that I find him not guilty."
5Then Jesus came out wearing the crown of thorns and the purple robe. And Pilate said, "Here is the man!"
6When they saw him, the leading priests and Temple guards began shouting, "Crucify! Crucify!" "You crucify him," Pilate said. "I find him not guilty."
7The Jewish leaders replied, "By our laws he ought to die because he called himself the Son of God."
8When Pilate heard this, he was more frightened than ever.
9He took Jesus back into the headquarters again and asked him, "Where are you from?" But Jesus gave no answer.
10"You won't talk to me?" Pilate demanded. "Don't you realize that I have the power to release you or to crucify you?"
11Then Jesus said, "You would have no power over me at all unless it were given to you from above. So the one who brought me to you has the greater sin."
12Then Pilate tried to release him, but the Jewish leaders told him, "If you release this man, you are not a friend of Caesar. Anyone who declares himself a king is a rebel against Caesar."
13When they said this, Pilate brought Jesus out to them again. Then Pilate sat down on the judgment seat on the platform that is called the Stone Pavement (in Hebrew, Gabbatha ).
14It was now about noon of the day of preparation for the Passover. And Pilate said to the people, "Here is your king!"
15"Away with him," they yelled. "Away with him––crucify him!" "What? Crucify your king?" Pilate asked. "We have no king but Caesar," the leading priests shouted back.
16Then Pilate gave Jesus to them to be crucified. So they took Jesus and led him away.
17Carrying the cross by himself, Jesus went to the place called Skull Hill (in Hebrew, Golgotha ).
18There they crucified him. There were two others crucified with him, one on either side, with Jesus between them.
19And Pilate posted a sign over him that read, "Jesus of Nazareth, the King of the Jews."
20The place where Jesus was crucified was near the city; and the sign was written in Hebrew, Latin, and Greek, so that many people could read it.
21Then the leading priests said to Pilate, "Change it from 'The King of the Jews' to 'He said, I am King of the Jews.'"
22Pilate replied, "What I have written, I have written. It stays exactly as it is."
23When the soldiers had crucified Jesus, they divided his clothes among the four of them. They also took his robe, but it was seamless, woven in one piece from the top.
24So they said, "Let's not tear it but throw dice to see who gets it." This fulfilled the Scripture that says, "They divided my clothes among themselves and threw dice for my robe." So that is what they did.
25Standing near the cross were Jesus' mother, and his mother's sister, Mary (the wife of Clopas), and Mary Magdalene.
26When Jesus saw his mother standing there beside the disciple he loved, he said to her, "Woman, he is your son."
27And he said to this disciple, "She is your mother." And from then on this disciple took her into his home.
28Jesus knew that everything was now finished, and to fulfill the Scriptures he said, "I am thirsty."
29A jar of sour wine was sitting there, so they soaked a sponge in it, put it on a hyssop branch, and held it up to his lips.
30When Jesus had tasted it, he said, "It is finished!" Then he bowed his head and gave up his spirit.
31The Jewish leaders didn't want the victims hanging there the next day, which was the Sabbath (and a very special Sabbath at that, because it was the Passover), so they asked Pilate to hasten their deaths by ordering that their legs be broken. Then their bodies could be taken down.
32So the soldiers came and broke the legs of the two men crucified with Jesus.
33But when they came to Jesus, they saw that he was dead already, so they didn't break his legs.
34One of the soldiers, however, pierced his side with a spear, and blood and water flowed out.
35This report is from an eyewitness giving an accurate account; it is presented so that you also can believe.
36These things happened in fulfillment of the Scriptures that say, "Not one of his bones will be broken,"
37and "They will look on him whom they pierced."
38Afterward Joseph of Arimathea, who had been a secret disciple of Jesus (because he feared the Jewish leaders), asked Pilate for permission to take Jesus' body down. When Pilate gave him permission, he came and took the body away.
39Nicodemus, the man who had come to Jesus at night, also came, bringing about seventy–five pounds of embalming ointment made from myrrh and aloes.
40Together they wrapped Jesus' body in a long linen cloth with the spices, as is the Jewish custom of burial.
41The place of crucifixion was near a garden, where there was a new tomb, never used before.
42And so, because it was the day of preparation before the Passover and since the tomb was close at hand, they laid Jesus there.


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Former General Secretary of the NPP, Kwadwo Owusu Afriyie has attributed the current economic challenges to the Supreme Court's ruling in the election petition that said President Mahama indeed won the 2012 presidential elections.
Mr. Owusu Afriyie affectionately called Sir John, said in an interview on the Point Blank segment on Eyewitness News, by affirming John Mahama winner of the 2012 election, the Supreme Court endorsed an "incompetent" government.
The NPP challenged the results of the 2012 elections at the Supreme Court alleging the results were rigged in favour John Dramani Mahama, but the Court ruled in favour of the President.
"They knew that it was not for them at the end and the conclusion of the matter they saw the judgment that was bequeathed to this country and we are all suffering as a result of that."
The economic challenges have compelled government to resort to the International Monetary Fund to seek support to turn the economy around.
Other pressure groups, including OccupyGhana, have also taken to the streets to protest against government's handling of the economy.
He insisted the NPP is not playing politics with the economic situation but insisted that "If you prophesy that the country is going to be in ruins, you are also in it and I am happy we are all {in it} together."
The NPP on Wednesday organized thousands of people to demonstrate against government's inability to manage the economic crises and also solve the energy crises which is collapsing more and more businesses.
The demonstration has been downplayed by some members of government and the Power Minister also described it as a "fitness exercise."
But President John Mahama has said he feels challenged to do more to stabilize the power crisis.
However, according to Sir John, the NDC government will pay dearly during the 2016 elections for the way it has managed the country.
"Ghanaians are fed up," he said and "will show them the exit if they don't listen." he said.
Source : citifmonline.com


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More than 20cm of snow recorded in Norfolk
PUBLISHED: 08:50 28 February 2018 | UPDATED: 09:17 28 February 2018
Archant Norfolk 2018
More than 20cm of snow has fallen in Norfolk causing traffic chaos and schools closures.
In Cringleford, one measurements topped 25cm.
In the west of Norwich some 22cm was recorded, while people in Hethersett and Tasburgh woke up to 21cm of the white stuff.
In Caister, near Great Yarmouth, 20cm was recorded, and in central Norwich, Thorpe Hamlet and Blundeston there were 18cm.
The University of East Anglia (UEA) had 17cm.
Meteorologist Dan Holley said temperatures plummeted to -9C overnight in Martham, but snow showers were expected to move off during the afternoon.
In other areas there were:
• 16cm in Costessey
• 15cm in Wymondham and Woodton
• 14cm in Beccles and Oulton Broad
• 13cm in Tacolneston, Reepham, and Aylsham
• 12cm in Horsford
• 11cm in Long Stratton and Dereham
• 10cm in Brundall
Mr Holley said although not official recordings, the depths - which were gave an indication.
He said: "It's probably of a similar magnitude to January 2013. I think it was pretty much expected, a couple of days ago the models were suggesting it would be a lot worse today. I know a lot of people where thought the worst was over."
He said the snow was expected to continue until lunchtime, but that freezing temperatures would continue, reaching -10C in some areas.
If you value what this story gives you, please consider supporting the Eastern Daily Press. Click the link in the yellow box below for details.


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Having caught up with three of Benegal's films in the past week—Ankur (1974), Kalyug (1981), and Trikal (1985), all of them available at Facets—I can see why he'd strike such an independent position. I haven't seen anything like them in my limited exposure to Indian movies (which includes a disproportionate number of Ray's): they're incisive, critical, and surprisingly sexual in their outlook. I look forward to seeing more of them.
Some context might be useful for Benegal's comment about Ray. Starting in the 1970s, Benegal became a leading figure of Parallel Cinema, a movement that sought a filmmaking style distinct from both Bollywood and Western models (the films still betray the influence of both, particularly the neorealist films of Roberto Rossellini). The movement would be inconceivable without Satyajit Ray's international success in the 50s; but by the time Benegal made Ankur, his first fiction feature, parallel filmmaking was thriving all over the world. To list a few major practitioners, there was Ousmane Sembene in Senegal, Lester James Peries in Sri Lanka, Lino Brocka in the Philippines, Ali Khamraev in Uzbekistan, and Yilmaz Güney in Turkey. That the last two of these directors only recently enjoyed their first Chicago retrospectives suggests that U.S. audiences (myself included) are still catching up to this exciting chapter of film history.
What unites these disparate filmmakers is a commitment to capturing native traditions, geography, and political attitudes—a proud sense of location that led to radically different kinds of films. Benegal made movies all over India, and based on the three I've seen, he seems to have altered his approach depending on where he set his story. Ankur takes place in the countryside of Benegal's home state Andhra Pradesh (on the southeastern coast); Kalyug in Bombay; and Trikal in Goa, a southwestern state that had been under Portuguese rule for centuries.
This pan-Indian perspective differs from that of Ray, who set almost all of his work in Bengal and whose perspective could be deemed universalist (which would explain why he remains the best-known Indian director in the U.S.). As most of the Indian films I've seen have come from Bengal or Maharashtra (i.e., Bollywood's state), Benegal's southern-set stories have been eye-opening. Trikal in particular provided insight into the legacy of Portuguese colonialism in India, something I hadn't considered before, in both social mores and the lay of the land. The architecture of Goa, which shows the entwined influence of Portuguese and Indian design, made me feel like I was seeing a landscape from Italo Calvino's Invisible Cities brought to life.
Kalyug and Trikal also feature love affairs that threaten to upset the established social order, which makes me suspect that destructive passion is a major theme in his work (I could be wrong, however; Benegal's directed roughly two dozen fiction features and almost twice as many documentaries). Surely the tension between individual liberty and cultural tradition is important to Benegal, who recently concluded a six-year term in the Rajya Sabha, the upper house of India's parliament. That Benegal is now a central part of Indian culture after holding a "parallel" position for decades suggests a fascinating career, one that deserves another Chicago retrospective.


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Part 5, and the Conclusion of this Series
Part 1 here, and Part 2 here, and Part 3 here, and Part 4 here.
Odum's Original 1973 article here: Energy, Ecology, and Economics
Saving the most important points for last:
During periods of expanding energy availabilities, activities that spur more growth and competition are successful in increasing the flow of energy beyond their cost. Institutio…
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Bus Driver Arrested in Death of Korean American Special Needs Student
John Kim, March 24, 2016, 11:23 a.m.
e driver of a school bus where a 19-year-old special needs student was left and died has been arrested, Whittier police said. Armando Abel Ramirez, 37, was booked on suspicion of dependent abuse Wednesday afternoon and is being held in lieu of $50,000 bail, jail records show.
In September, Hun Joon Lee was found slumped in the bus' aisle and pronounced dead after lifesaving efforts failed, according to Los Angeles County Coroner's Lt. David Smith. Lee regularly rode the bus to his home in Whittier, and Ramirez was the driver on Lee's final ride, police say.
The teen typically left school on the bus around 2:30 p.m. and arrived home at 4, said Brad White, a spokesman for the Whittier Police Department. When he didn't show up on Sept. 11, his mother called the school, which then called the bus company. A driver went out to the bus yard and found Lee slumped in the aisle of the parked bus, White said.
Police arrived at the Sierra Education Center to find several bus drivers attempting CPR on the young man. The officers took over the lifesaving effort, without success. Lee was pronounced dead about 10 minutes after police arrived.
Police are treating the death as suspicious because "nothing that we've learned so far would lead us to believe he had any medical conditions," White said. There were no signs of trauma on Lee's body, and no weapons were found at the scene, White said.
Lee attended the center, which has a transition program dedicated to adult students with special needs, said Valerie Martinez, Whittier Union High School District spokeswoman. Whittier Union High School District Supt. Sandra Thorstenson issued a written statement of condolence.
"Our hearts are with our student's parents and family — we're all grieving. We're making ourselves, our counseling services and our staff available to his family and to our students and staff who were close to him," she wrote.
Ramirez is scheduled to be arraigned Friday.
Source (.)