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(In 88 at bats): .295 avg, 1 hr, 18 rbi, .353 obp and .420 slugging
(In 221 at bats): .268 avg, 1 hr, 22 rbi, .355 obp and .330 slugging
Jed is a solid major league ss. lugo SUCKS. He is a 9m dollar pinch runner. The problem is that Tito is so dumb. He is the opposite of Bill B. He'll play the veteran over the clearly more talented and reliable player.
I agree that Jed "appears" to be a solid short stop, i'm not ready to give him all-star status or anything, but he appears ready...and i'm not a big fan of Lugo at all. You're right that at this point he should be reduced to an unfortunate 9 million dollar pinch runner (by the way, anyone else think he is the goofiest "fast runner" you've ever seen run?).
I wouldn't call a manager who has produced two world series rings in the last four years in boston dumb, but we all have every right to question some of his moves. At times, his relationships with players run too deep and cause for a conflict of interest. (PS, i'll be updating Jed's numbers as we go)
Those Lugo/Lowrie comparisons are jarring. Let's see how Terry handles this once Julio recovers. My guess is we'll see Lugo at short, whether we like it or not.
SteveT I hope you're wrong, but I, like you, are nervous about this upcoming decision. One would think that the numbers alone would speak for themselves, but we all know Tito is not that kind of manager. It is hard to argue with Tito since he's done so much good here in Boston, but if Julio Lugo is the everyday short stop in a month, i'm going to be writting Tito a letter.
Not sure when Lugo is due back, hopefully its in Sept when the Sox can expand the roster and keep Lowrie up.
Lugo hasn't gotten it done in 2 seasons here, this year has been worse then last year as if that were possible. I don't buy the arguement that he's to expensieve to trade, you hear that on eei all the time. They print money at Fenway, trade him and eat his contract, lowrie is never going to be arod at shop, but he could be a solid player.
Lugo is a diggle


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|02 Apr 2013
Fitting a Sound card!
Not impressed with onboard audio on my ASUS M2N-SLI mobo, but alas do not know enough about about fitting a soundcard!, anyway apparently the mobo does not support win 7 64bit, strange really because that's what I have been running since win 7 hit the shops, also no firmware updates available.
Could I fit a
Asus Xonar DGX 5.1 PCI-Express Sound Card
sorry for the shouting (cut&paste)?, your thoughts most appreciated.
|My System Specs
|02 Apr 2013
Yep! I would go in one of the two small PCI-E x1 slots that are located above and below the blue PCI-E x16 slot.
|My System Specs
|02 Apr 2013
I'm impressed TwoCables, very much obliged.
|My System Specs
|05 Apr 2013
Just a quick update, purchased the Asus DGX Xonar audio card, why oh why did I tolerate the onboard audio system, anyway I am very pleased, also the card is a good price.
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Greetings Runners!
The warm weather is here, that means it is time to put on your running shoes and return to the outdoors for your work-outs.
ProEx Physical Therapy would like to offer you information on the most common injuries sustained while running, and provide some general running tips.
Understanding Common Running Injuries:
Heel and Foot Pain (Plantar Fasciitis)
- The plantar fascia is a ligament like connective tissue that is located on the underside of the foot. Plantar fasciitis is inflammation of the plantar fascia which can present as mild to severe pain in the bottom of the heel or foot. This usually occurs with either or a combination of over training, poor shoe stability or hyperpronation (excessive foot flattening when weight bearing on them). Common treatment for plantar fasciitis is stretching of the structures surrounding the foot and heel, modification of activity and shoe wear, and ice.
Bone Pain (Stress Fractures)
- A stress fracture can be classified as a hairline or complete break of the bone. This injury can be extremely painful, and will be exacerbated by impact activities. Typically in runners, stress fractures are seen in the lower leg bone (tibia) or the long bones of the foot (metatarsals). Appropriate treatment for a stress fracture is discontinuation of running activities and possible immobilization to let the bone heal.
Shin Splints (Medial Tibial Stress Syndrome)
- Inflammation and pain around the muscles and tendons on the inside of the shin bone (tibia) can be classified as medial tibial stress syndrome. Hyperpronation is also a common cause of this type of injury. Running activities should be modified, ice can be applied to the injured area, and a supportive arch can be placed in the footwear.
Achilles and Patella Tendonitis
- Two typical areas for tendonitis to occur are in the patella tendon (right blow your knee cap) and Achilles tendon (back of heel). Tendonitis is an inflammation of the tendon, which can become a chronic condition if not treated correctly. Pain and tenderness will be present in the affected tendon. Treatment should include modifying activity, stretching and applying ice to the injured area.
Managing Running Related injuries:
When in Doubt, Seek Medical Advice
Your Physician and local Physical Therapist can provide a thorough evaluation of your running injuries. If you are unsure of your aches and pains, we encourage you to seek out an appointment with a professional who can give you an appropriate diagnosis, and course of treatment. To learn more on how to manage an acute injury click here Acute Injury Management
Running Shoes: The Right Pair is Critical
With so many options for running shoes, it can be difficult to find the perfect pair. The most important items to consider when choosing sneakers are function and comfort. When a running sneaker has reached 500-600 miles of use, consider replacements. Most running shoe stores have employees that can be a good resource for you if they are knowledgeable on matching shoe models for different foot types. Just ask for help!
Pick the Right Running Surfaces
The perfect surface to run on should be flat, soft and safe. You may be able to find a path in your community specifically designed for running. Check out your local high school, many schools now have new "runner friendly" tracks. It is necessary to use caution when running hills as the impact will cause increase stress to your joints. If possible, avoid running on concrete and uneven surfaces.
Sports Medicine Approach to getting You Back to Running
After sustaining an injury, Physical Therapy is an option to aid in healing your injuries and return you back to running in a timely manner. Working with Sports Medicine professionals such as physical therapists, athletic trainers, and strength and conditioning specialists will assist you in resuming your desired level of running activity. Learn more about the Sports Medicine approach Sports Medicine


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"Mako Shark" was first published in 1961. The appearance of this experimental machine was inspired appeared two years earlier race car Chevrolet Sting Ray: many elements of the exterior clearly show the familial relationship between these machines, although this time the designers have decided to fully open all-wheel drive. But that was not the main thing.
It was said that all the same vice president of the center-style GM William Mitchell was not only a fanatical fan of racing, but also the habit of a fisherman. The idea to create a car-shark came to him after he caught near Florida mako shark. Shark, you see, has made a lasting impression on the designer, as all the exterior of the new machine in one way or another is reminiscent of marine predator. And especially – the color from almost white at the bottom of the color of the car goes into a dark blue, almost black "metallic" in the upper part of the body.
Unlike the prototype Sting Ray is decided as a platform to use is not experimental and conventional, standard chassis, which is constructed on the basis of production cars Chevrolet Corvette 1961 model year. His only slightly modified in view of the fact that the length of the hood of the "sharks" over the standard car has increased by 40 cm, and the front overhang was 112 cm instead of 81 cm.
Particular attention was paid to the ergonomics of the driver's seat. There used experimental seats with good lateral support, decorated in wood steering wheel, the position of which for the first time on the machines of the brand can be adjusted in both the vertical and horizontal planes.
If desired, this double roadster and can be fitted easily removable roof made of aluminum and plastic.
Since then, no changes in the structures is not provided, except for the replacement of engines. First, there was a 7-liter aluminum ZL-1 engine with a carburetor Holley, and then it was replaced by "charged" 500-horsepower engine with a special automatic transmission THM 400.
Now Mako Shark belongs to the design center of General Motors and often takes part in various exhibitions, which are held in the United States are vast.
Corvette XP 755 Shark Concept Car wallpapers for your PC, Iphone, Android Device or Tablet PC. If you want to download Corvette XP 755 Shark Concept Car High Quality wallpapers for your desktop, please download this wallpapers below and click «set as desktop background». You can share this wallpaper in social networks, we will be very grateful to you


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Wild boars (Sus scrofa) are indigenous in many countries in the world. These free-living swine are known reservoirs for a number of viruses, bacteria and parasites that are transmissible to domestic animals and humans. Changes of human habitation to suburban areas, increased use of lands for agricultural purposes, increased hunting activities and consumption of wild boar meat have increased the chances of exposure of wild boars to domestic animals and humans. Wild boars can act as reservoirs for many important infectious diseases in domestic animals, such as classical swine fever, brucellosis and trichinellosis, and in humans, diseases such as hepatitis E, tuberculosis, leptospirosis and trichinellosis. For examples, wild boars are reservoirs for hepatitis E virus, and cluster cases of hepatitis E have been reported in Japan of humans who consumed wild boar meat. In Canada, an outbreak of trichinellosis was linked to the consumption of wild boar meat. The incidence of tuberculosis owing to Mycobacterium bovis has increased in wild boars, thus posing a potential concern for infections in livestock and humans. It has also been documented that six hunters contracted Brucella suis infections from wild swine in Florida. This article discusses the prevalence and risk of infectious agents in wild boars and their potential transmission to livestock and humans.
1. Introduction
Wild boars (Sus scrofa) are indigenous in many countries in the world. These free-living swine populations pose not only ecological concerns but infectious disease concerns as well. Wild boars harbour many important infectious agents that are transmissible to domestic pigs and other animal species including humans. Changes of human habitation to suburban areas owing to growing world populations, increased use of lands for agricultural purposes, and deforestation have all increased chances of contact exposure of wild boars to humans and domestic animals. In addition, recreational hunting of wild boars and consumption of wild boar meats in some regions of the world further provided ample opportunities for direct human contacts with wild boars, and thus created an ideal environment for the transmission of pathogens between wild boars and domestic swine, and between wild boars and humans (Gibbs 1997). In some regions, the wild boar populations are on the rise in part owing to the development of a commercial hunting industry (Acevedo et al. 2006), and this could further complicate the problem.
Wild boars also pose a problem when countries are trying to eradicate zoonotic diseases such as hepatitis E, tuberculosis, brucellosis and trichinellosis in humans and important livestock infectious diseases such as pseudorabies and porcine circovirus-associated diseases (PCVADs) in pigs. This is especially relevant for disease-free populations of animals and humans not exposed previously to the agents from wild boars and with no herd or population immunity, and thus highly susceptible to such infectious agents.
This review summarizes the potential risks posed by wild boars as sources of livestock (especially domestic swine) infections and as reservoirs for zoonoses. Specific examples of viral (hepatitis E), bacterial (tuberculosis) and parasitic (trichinellosis) zoonoses transmissible from wild boars to humans are also discussed.
2. Viruses in wild boars that are potentially transmissible to livestock and humans
Wild boars are reservoirs for a number of viruses that cause important diseases in livestock and humans (Gibbs 1997; Ruiz-Fons et al. 2008). Table 1 lists the viruses that are known to be prevalent in wild boars with potential for transmission to domestic animals and humans.
(a) Wild boars as reservoirs for livestock infections
Among the agriculturally important pathogens known to be prevalent in wild boars are classical swine fever virus (CSFV), pseudorabies virus (PRV) infection, African swine fever virus (ASFV), porcine circovirus type 2 (PCV2), porcine reproductive and respiratory syndrome virus (PRRSV) and porcine parvovirus (PPV) (table 1).
CSFV, a small single-strand positive-sense RNA virus, belongs to the genus pestivirus in the family Flaviviridae. CSFV infects both domestic swine and wild boars and can be transmitted from wild boars to domestic pigs and vice versa (Brugh et al. 1964). CSFV is highly contagious and swine are the only known reservoir. CSFV is transmitted primarily via the oronasal route by direct or indirect contact with infected domestic pigs or wild boars or via the oral route by ingestion of contaminated foodstuffs (Edwards et al. 2000). The clinical signs of disease associated with CSFV infection vary greatly with acute and/or chronic forms of the disease (Le Potier et al. 2006).
Several countries, including the USA, Australia, Canada and New Zealand are free of CSFV in domestic pig populations (Paton & Greiser-Wilke 2003). CSFV is circulating in some European countries, with prevalence rates ranging from less than 1 per cent in France (Albina et al. 2000), 31 per cent in Switzerland (Schnyder et al. 2002) and to up to 39 per cent in Croatia (Zupancic et al. 2002). CSFV infection reported in recent outbreaks in wild boars appeared to be subclinical with little mortality (Le Potier et al. 2006), although wild boars can serve as reservoirs for infection of domestic pigs. In some European countries, CSFV has been reintroduced periodically into domestic pigs via contact with infected wild boars (Le Potier et al. 2006).
PRV, an alphaherpesvirus, has a worldwide distribution and infects both wild boars and domestic swine (Ruiz-Fons et al. 2007, 2008). PRV infections in adult feral swine generally do not cause morbidity or mortality (Romero et al. 2001) but can result in latent infections. An important feature of PRV infection in wild or domestic swine is the establishment of latency in neuronal and non-neuronal cells, which has important implications for the spread of PRV. Exposure to PRV is high among wild boars and the seroprevalence rate ranges from 9 to 61 per cent in wild boars from different countries (table 1). The seroprevalence result was confirmed by the detection of PRV DNA in tissues of wild boars (Ruiz-Fons et al. 2007). The high prevalence of PRV in wild boars poses a concern for the health of both wild boars and other domestic animals and prospects for PRV control and eradication campaigns in swine. In the USA, a national pseudorabies eradication programme began in 1989 and, currently, domestic swine in the majority of states are free of PRV. However, the virus is well established in wild boar populations in the USA and other countries, and wild boars represent a potential reservoir of PRV for infection of domestic swine and native wildlife.
AFSV, an OIE (World Organization for Animal Health) List A virus, is a DNA virus in the family Asfarviridae. ASFV is highly resistant to inactivation by temperature and acidic pH and can persist in frozen or uncooked meats for weeks or months (Sanchez-Vizcaino 2006). ASFV is endemic in many African countries and also in Sardinia, Italy (Sanchez-Vizcaino 2006). In addition to domestic pigs, ASFV also infects European wild boars and African wild suids such as warthhogs, bush pigs and giant forest hog (Sanchez-Vizcaino 2006). Serological evidence of ASFV prevalence in wild boars has been reported in Italy and Spain where approximately 10 per cent of the wild boars tested (Pérez et al. 1998) had antibodies to ASFV but did not show clinical signs. European wild boars are not believed to play an important role as reservoirs for ASFV.
PCV2, the causative agent of PCVAD is a small single-stranded DNA virus with a circular genome (Ramamoorthy & Meng 2008). PCVAD is characterized by wasting, enlarged lymph nodes, jaundice and weight loss in affected weanling pigs, and lymphoid depletion is the hallmark microscopic lesion. Recently, several other complex syndromes, including reproductive failure, enteritis, pneumonia and necrotizing dermatitis, have also been linked to PCV2 infection. PCVAD is further complicated by co-infections with other bacterial and viral pathogens (Ramamoorthy & Meng 2008).
Evidence of PCV2 infection in wild boars has been reported in Hungary, Spain and Belgium (table 1). Approximately 50 per cent of wild boars tested in Spain were seropositive for PCV2 antibodies and approximately 21 per cent of wild boars from Hungary were positive for PCV2 DNA (Cságola et al. 2006). PCVAD was reported in Eurasian wild boars raised under free-range conditions (Vicente et al. 2004). Affected wild boars had pneumonia and enteritis and were cachectic. These data indicate that there is a high rate of PCV2 infection in wild boars, although the exact role of wild boars in PCV2 transmission to domestic pigs is not clear.
PRRSV, a single-strand positive-sense RNA virus in the family Arteriviridae, causes arguably the most economically significant disease facing the global swine industry today (Meng 2000a). Nursery pigs infected by PRRSV develop pneumonia and other respiratory diseases, whereas infected sows develop reproductive failures. PRRSV infection can subvert the pig immune system and cause persistent infection, which is one of the major obstacles for PRRSV control. The interactions between PRRSV and other swine pathogens produce clinically and economically significant disease syndromes, such as porcine respiratory disease complex. The role of wild boar in the transmission of PRRSV to domestic pigs is unclear. Antibodies to PRRSV have been detected in wild boar in some countries (table 1), although others failed to detect PRRSV antibodies in wild boars. There is lack of convincing evidence to suggest that wild boars serve as a reservoir for PRRSV.
PPV, a small DNA virus in the family Parvoviridae, is ubiquitous among swine throughout the world (Mengeling 2006). PPV is associated mainly with reproductive problems in gilts, sows and boars and causes only subclinical infections in other pigs. Large proportions of gilts are naturally infected with PPV before they conceive and develop an active immunity that probably lasts throughout their life.
PPV is highly prevalent in wild boars, with an incidence ranging from 14 to 17 per cent in Oklahoma and Tennessee, USA, to 57 per cent in Spain (table 1). The effect of PPV infection on the health status of wild boars appears to be minimal and subclinical, although it has been reported that PPV infection has a negative effect on the ovulation rate of female wild boars (Ruiz-Fons et al. 2006). It is possible that PPV is transmissible between wild boars and domestic pigs through direct contacts, although wild boar is not considered a significant reservoir for PPV transmission to domestic pigs (Ruiz-Fons et al. 2008).
(b) Wild boars as reservoirs for human infections
Antibodies to a number of zoonotic viruses have been detected in wild boar populations, including hepatitis E virus (HEV), swine influenza virus and Japanese encephalitis virus (table 1). Wild boars infected by these zoonotic viruses have the potential to transmit to humans in close contact. Direct evidence of boar to human transmission has been documented for HEV.
HEV, a single-strand positive-sense RNA virus, belongs to the family Hepeviridae. The disease caused by HEV, hepatitis E, is an important public health disease in many developing countries, even though the disease is also endemic in many industrialized countries (Meng 2000b, 2003; Meng & Halbur 2006). HEV is transmitted primarily through the faecal–oral route, and contaminated water or water supplies are the main sources of infection. The mortality caused by HEV is generally low (less than 1%), but 28 per cent was reported in infected pregnant women. There are at least four distinct genotypes of HEV worldwide. Genotypes 1 and 2 are associated with epidemics, whereas genotypes 3 and 4 cause sporadic cases of hepatitis E (Meng & Halbur 2006).
The discovery of swine HEV from pigs in the USA (Meng et al. 1997) lends credence to the zoonosis concept for HEV. Thus far, the viruses identified from pigs worldwide belong to either genotype 3 or 4, which both cause sporadic cases of acute hepatitis E (Meng & Halbur 2006). Genetic and phylogenetic analyses of the complete genomic sequences of swine HEV revealed that swine HEV is very closely related, or identical in some cases, to genotypes 3 and 4 of human HEV. Seroepidemiological studies demonstrated that swine HEV is ubiquitous in pigs in the midwestern USA (Meng et al. 1997). Similar findings were also reported in many other developing and industrialized countries, indicating that swine HEV infection in pigs is common worldwide (Meng 2003). The ubiquitous nature of swine HEV infection in pigs provides a source of virus for zoonotic human infections.
Swine HEV has been shown to cross species barriers and infect both rhesus monkey and chimpanzees (Meng et al. 1998). Conversely, genotypes 3 and 4 of human HEV have been shown to infect pigs under experimental conditions (Feagins et al. 2008). However, attempts to experimentally infect pigs with genotypes 1 and 2 of human HEV were unsuccessful. Hepatitis E is now considered as a zoonotic disease and pigs (and possibly other animal species) are reservoirs. Pig handlers, such as pig farmers and swine veterinarians, in both developing and industrialized countries are at increased risk of HEV infection. For example, swine veterinarians in the USA were 1.51 times (swine HEV antigen, p = 0.03) more likely to be positive for HEV antibodies than age- and geography-matched normal blood donors (Meng et al. 2002).
Sporadic and cluster cases of acute hepatitis E have been linked to the consumption of raw or undercooked pig livers. Approximately 2 per cent of pig livers sold in local grocery stores in Japan (Yazaki et al. 2003) and 11 per cent in the USA were tested positive for swine HEV RNA (Feagins et al. 2007). Contaminating virus in commercial pig livers sold in the grocery stores of the USA remains fully infectious. Most significantly, the genome sequences of viruses recovered from pig livers in grocery stores were closely related, or identical in a few cases, to the viruses recovered from human hepatitis E patients in Japan (Yazaki et al. 2003).
Wild boar populations in Japan, Germany, Italy, Spain and Australia are commonly infected by HEV, as demonstrated by the detection of HEV RNA and anti-HEV antibodies (table 1). A full-length genomic sequence of HEV from a boar was found to be 99.7 per cent identical to the virus from a wild deer hunted in the same forest and to four patients who consumed deer meat and contracted hepatitis E (Takahashi et al. 2004), suggesting a potential interspecies HEV transmission between boar and deer in wildlife. In Japan, a 53-year-old man developed severe hepatitis E after consumption of wild boar meat (Matsuda et al. 2003). Another patient, a 70-year-old man, who also ate the same wild boar meat developed hepatic coma and died of fulminant hepatic failure (Matsuda et al. 2003). In the three months prior to the onset of disease, neither patient had travelled to an HEV endemic area, but both patients ate uncooked wild boar liver on five occasions. This correlation provided strong direct evidence of HEV transmission from infected wild boar to humans.
3. Bacteria in wild boars that are potentially transmissible to livestock and humans
It has been clearly shown that wild boar can act as reservoirs for a long list of zoonotic bacterial agents. Infected boars have the potential to transmit such agents to livestock and to humans, who may come in contact with their discharges or consume infected meat. Major zoonotic bacterial agents that have been detected from wild boars by isolation and/or by specific antibodies are listed in table 2. Among the major bacterial agents of agricultural importance isolated from wild boars include Mycobacterium bovis (bTB), Brucella suis (BS), Brucella melitensis (BM), Brucella abortus (BA), Coxiella burnetii (Q fever), Yersinia pestis (plague) and Leptospira interrogans (Lepto). In this article, only those major zoonotic bacterial diseases transmissible to livestock and/or humans are discussed.
(a) Tuberculosis
Mycobacterium bovis (bTB), the causative agent of bovine tuberculosis, has the broadest host range compared with any other member of the genus Mycobacterium (Mycobacterium tuberculosis complex). Mycobacterium bovis infects cattle, other ruminants, humans and wildlife. The bacterium is transmitted mainly by aerosol or by consumption of infected meat, milk and their products. It typically causes a persistent infection with production of granulomas of the lymph nodes, lung, and may become systemic affecting most internal organs. It was originally thought that wild boars (wildlife) were the dead-end hosts for M. bovis.
During the last two decades, the role of wild boars as a dead-end host, spillover host, maintenance host and true wildlife reservoir of bTB has been extensively debated. Serraino et al. (1999) argued that wild boars were dead-end hosts, as mainly localized lesions were found in the lymph nodes of bTB-affected wild boars. They did not develop generalized lesions in the respiratory tract, which gave credence to the argument, although molecular typing showed that the same spoligotype affected both livestock and wild boar in Italy. Several recent publications from Spain demonstrate that intensively raised wild boars within the confines of fenced areas of wilderness can be a major source of bTB (Parra et al. 2006; Vicente et al. 2006). In the Mediterranean region of Spain, wild boar raised semiwild (with feeding and watering within fenced areas, so the pigs could be used as game animals by hunters) have created a unique epidemiological niche that has led to major disease outbreaks owing to M. bovis in the wildlife with potential exposure to domestic animals. The numbers of bTB-positive wild boar in Europe were close to 50 per cent (table 2), which is alarming. There is a clear indication that wild boar could be the source of bTB infection for domestic animals and humans, especially hunters and veterinarians. Zanella et al. (2008) reported that an outbreak of tuberculosis in wildlife in France, especially in heavily infected wild boars, was due to the same strain of M. bovis isolated from other wildlife and domestic cattle.
When bTB in domestic animals is controlled or eradicated, the incidence of infection in wildlife also decreases, suggesting that domestic animals could be the initial source of bTB infection to wild boars. If the populations of wildlife remain small and do not come in contact with domestic animals, they may act as maintenance hosts. However, when greater numbers of wild boars are raised in confined areas, they may suffer from severe bTB infections and become the source of infection to other hosts.
Examples of other wildlife serving as M. bovis reservoirs include ruminants such as African buffalo and Canadian bison, which were implicated in the epidemiology of bTB in South Africa and Canada, respectively (Nishi et al. 2006). The European badger was shown to be the source of M. bovis in the recent outbreaks of bovine tuberculosis in Ireland and UK (Delahay et al. 2002; Phillips et al. 2003) and the bushtail possum was found to be a major wildlife reservoir of bTB in New Zealand (Coleman & Cooke 2001). In the USA, the white-tailed deer has been shown to be one of the major sources of tuberculosis for domestic animals in disease-free states (Corner 2006).
(b) Brucellosis
Brucella suis is a Gram-negative coccobacilli and facultative intracellular pathogen that can cause zoonotic infections in pigs and other animals, including humans and wild boars, leading to abortions and infertility. Currently, eight species of Brucella are recognized that primarily affect a specific group of animals, including smooth B. melitensis (sheep and goats), B. suis (pigs), B. abortus (cattle), Brucella ceti (dolphins), Brucella pinnipedialis (seals), Brucella neotome (wood rat), rough Brucella canis (dogs) and Brucella ovis (sheep). Of the three major host-specific species of Brucella, B. suis is considered the second most pathogenic (only slightly less virulent than B. melitensis) in humans. Brucellosis is a chronic zoonotic disease resulting in 'undulant fever' in humans and abortion and/or infertility in affected animals.
Wood et al. (1976) showed that 18 percent of feral pigs in South Carolina were serologically positive for B. suis, and the increasing incidence with age suggested that the pathogen was being transmitted among the population. The serological results were further confirmed by isolation of B. suis biotype 1 from the lymph node of an affected feral pig. Approximately 20–30% of feral swine and wild boars have been shown to be Brucella-positive by serology in Italy, USA and Croatia (table 2). The high level of exposure suggests that there is an active transmission and thus it may pose a serious threat to nearby domestic swine and hunters. Cvetnic et al. (2003) isolated B. suis biovar 2 from 58 per cent of swine and 62.5 per cent of aborted piglets. These authors also demonstrated that approximately 23 per cent of wild boars examined in the study were seropositive for Brucella and that wild boars should therefore be considered as wildlife reservoirs of B. suis biovar 2 in Croatia.
Al Dahouk et al. (2005) found that 22 per cent of wild boar sera in Germany were seropositive. Ruiz-Fons et al. (2006) showed that approximately 10 per cent of young adults and 28 per cent of adult wild boars in open estates in southern Spain were seropositive for brucellosis. By comparison, in wild boars of fenced estates, the incidence of seropositivity was approximately 32 and 38 per cent in young adults and adults, respectively. In Japan, Watarai et al. (2006) analysed 115 wild boars from four prefectures and found that approximately 7.5 per cent were seropositive (table 2).
The minimal infective dose of B. suis for humans is in the range of 10–100 colony forming units, and there are many cases of human infection owing to handling of infected feral and wild boars. Incidence of B. suis in wild boar populations has attained a very high level in southern Spain and pose a serious threat both to local domestic pigs and hunters and animal-care professionals. Starnes et al. (2004) reported two cases of brucellosis in members of a hunt club who had killed and prepared meat from wild boars.
(c) Leptospirosis
Domestic pigs infected by L. interrogans are known to develop a 'carrier state' with intermittent shedding of the bacterium after an outbreak. Carrier pigs can host the leptospiral organism in the anterior convoluted tubules of kidneys even in the presence of specific circulating antibodies. Ebani et al. (2003) examined 562 blood samples of wild boars from Italy and showed that approximately 6 per cent were positive. Deutz et al. (2003) found that approximately 10 per cent of hunters from southeastern Austrian federal states of Styria and Burgenland had leptospiral antibodies. Jansen et al. (2007) detected leptospiral antibodies in 18 per cent of wild boars from Berlin, suggesting that infected wild boars can be the source of infections to domestic animals and humans.
Other zoonotic bacterial agents that wild boars can serve as reservoirs for include C. burnetii, the causative agent of Q fever, Francisella tularensis, which causes tularaemia, and Y. pestis, the cause of bubonic and pneumonic plagues.
4. Parasites in wild boars that are potentially transmissible to livestock and humans
Wild boars are known to harbour two important parasites of humans in their edible tissues, and ingestion of raw or undercooked meat is the mode of infection. Freezing of wild boar meat is not effective protection against all species of Trichinella.
(a) Wild boars as reservoirs for important parasitic diseases of domesticated animals
(i) Trichinella spp.
Trichinella spiralis is perhaps the best-known nematode parasite of swine. There are presently eight named species of Trichinella based on the presence or absence of a capsule; mouse and pig infectivity; infectivity for birds and reptiles; resistance to freezing; and molecular characteristics (table 3). Three genotypes, viz. T6, T8 and T9, have been identified (Pozio & Murrell 2006). There are no published reports on clinical signs or disease in wild boar owing to Trichinella spp. infection. In one experimental study in wild boar, Tr. spiralis was found to be highly infective, Trichinella britovi, Trichinella nelsoni, Trichinella pseudospiralis (USA) and Tr. pseudospiralis (USSR) were moderately infective and Trichinella nativa, Trichinella murrelli, Trichinella pseudospiralis (Australia) and Trichinella T6 were poorly infective (Kapel 2001).
Trichinella spp. circulate in sylvatic and domestic cycles in many areas of the world. Infection of domestic livestock and companion animals is usually asymptomatic. Transmission in animals is by predation, cannibalism, scavenging or by intentional feeding of raw or undercooked meat. A major concern is the introduction of Trichinella spp. in domestic pigs, which then serve as a source of human and other animal infections. Infections in horses, which have been widely reported, usually occur when the animals have been fed uncooked or undercooked meat from infected animals (Murrell et al. 2004). Trichinella papuae and Trichinella zimbabwensis are infectious for reptiles and mammals (Pozio et al. 2004b) but not fish (Pozio & Rosa 2005). Trichinella pseudospiralis is infectious for birds and mammals but not reptiles (Pozio et al. 2004b). Trichinella papuae is common in wild pigs in Papua New Guinea, with a prevalence of approximately 11.5 per cent (Owen et al. 2005). The prevalence in farm-raised saltwater crocodiles owing to the feeding of wild boar meat in Papua New Guinea is approximately 21 per cent (Pozio et al. 2005). There is a single report of Tr. nelsoni in wild boar from Italy (Pozio et al. 1987), and both Tr. spiralis and Tr. britovi have been isolated from wild boar in Poland (Cabaj 2006) and Spain (Rodríguez et al. 2008). Antibodies to Trichinella spp. were found in 39 per cent of feral swine from coastal South Carolina, USA (Gresham et al. 2002). Trichinella murrelli is the species usually involved in the sylvatic cycle in temperate areas of North America (Pozio & La Rosa 2000).
This parasite is found in virtually all warm-blooded animals. Domestic and all other breeds of cat are the definitive host and excrete oocysts in their faeces. These oocysts sporulate and become infective in 1–2 days in the environment. Wild boar and other animals become infected by ingesting sporulated oocysts in the environment or by ingesting tissue cysts within the tissues of intermediate hosts. There are no reports of clinical toxoplasmosis in wild boar. Toxoplasma gondii can be transplacentally transmitted in domestic pigs and infections are usually asymptomatic. Young pigs are more susceptible to clinical disease than are older pigs (Dubey 1986).
Toxoplasma gondii was isolated from 2 per cent of wild boars in the Czech Republic, and a serological prevalence of 15 per cent was found in 124 wild boars (Hejlícek et al. 1997). The serological prevalence of T. gondii in wild boars was 5.6 per cent from Iriomote Island (Nogami et al. 1999) and 1.1 per cent from Amakusa Island, Japan (Shiibashi et al. 2004). Samples from 13 per cent of 135 wild hogs from California, USA, tested positive for antibodies to T. gondii (Clark et al. 1983). In South Carolina (USA), antibodies to T. gondii were found in 49 (Gresham et al. 2002) and 37 per cent of feral swine (Diderrich et al. 1996). Serum samples from 31 per cent of feral swine from the Great Smokey Mountains National Park, USA, were positive for antibodies to T. gondii (Diderrich et al. 1996). A study on Ossabaw Island, GA, USA, found a serological prevalence of only 0.9 per cent of 1264 wild boars and a serological prevalence of 18.2 per cent of 170 wild boars from mainland Georgia, USA (Dubey et al. 1997). The low serological prevalence on Ossabaw Island was attributed to a scarcity of domestic cats: only one domestic cat was known to be on the island and its whereabouts were not known (Dubey et al. 1997). It is well known that domestic pigs raised outdoors have a higher serological prevalence for T. gondii than do pigs raised indoors (van der Giessen et al. 2007). Exposure of wild boar to feline faeces and cannibalism are probably the reasons for an unusually high prevalence of T. gondii in wild boars.
(b) Wild boars as reservoirs for zoonotic parasitic infections in humans
(i) Trichinella spp.
Trichinosis is an emerging zoonotic disease in several European countries, and outbreaks after the consumption of wild boar meat are common (Serrano et al. 1989; Rodriguez-Osorio et al. 1999). In a survey from Spain, 75 per cent of 49 Trichinella outbreaks arose from the ingestion of wild boar meat (Rodríguez de las Parras et al. 2004). In Poland, it was estimated that 88 per cent of trichinosis cases during 2000–2005 were due to ingestion of wild boar (Gołab & Sadkowska-Todys 2006). Of special importance is the infection of wild boar with the highly freezing-resistant Tr. nativa in Spain (Pozio & Kapel 1999), and infection of wild boar with the moderately freezing-resistant Tr. britovi is common. Trichinella britovi has been reported from wild boar from Belgium (Schynts et al. 2006), Spain (Rodríguez de las Parras et al. 2004) and France (De Bruyne et al. 2006). The non-encapsulated Tr. pseudospiralis has been found in wild boar from Sweden (Pozio et al. 2004a), Texas, USA (Gamble et al. 2005) and Thailand (Jongwutiwes et al. 1998). An outbreak of Tr. pseudospiralis in Thailand involved 59 individuals and one died (Jongwutiwes et al. 1998). In Papua New Guinea where Tr. papuae is common in wild boar, 10 per cent of the population has antibodies to the parasite (Owen et al. 2005). Trichinella nelsoni is known to establish muscle infections in experimentally infected wild boar (Kapel et al. 2005). A single case of trichinellosis was reported from consumption of wild boar in the USA from 1991 to 2001 (Roy et al. 2003). Trichinella genotypes T6, T8 and T9 mainly exist in sylvatic cycles. Human infections with Trichinella T6 have been reported in individuals who consumed cougar or black bear meat, and symptoms were mild. No reports of human infection with genotypes T8 and T9 have been reported (Pozio & Murrell 2006).
Because freezing does not kill all species of Trichinella, proper cooking is the best means to prevent human infection. The killing of Trichinella larvae in meat is time and temperature dependent (Kotula et al. 1983). In a well controlled study, Kotula et al. (1983) found that larvae in 2 mm thick portions of homogenized pork retained their infectivity in samples heated to 55°C for 4 min, but were rendered non-infective after 6 min at 55°C. Larvae in samples heated to 49°C were infective after 5 h, but not after 6 h. One case of trichinosis has been reported in a person who ate bear hamburgers that had been cooked in a microwave oven, suggesting that some larvae may withstand microwave cooking (Nelson et al. 2003).
Most cases of toxoplasmosis in immunocompetent humans are subclinical. Congenital toxoplasmosis has long been recognized for the devastating effects it can have on the infected foetus (Jones et al. 2001), including hydrocephalus, blindness and mental retardation. Gauss et al. (2005) found that 38.4 per cent of 507 hunters who killed wild pigs in Spain were positive for antibodies to T. gondii, and Ruiz-Fons et al. (2006) found a prevalence of 36.3 per cent in hunters who killed wild boar sows. The relative importance of meat or oocysts as a source of human infection is unknown. An outbreak of unilateral chorioretinitis owing to T. gondii was reported in three patients from Korea who ingested raw liver and spleen from a wild pig (Choi et al. 1997). Although no other outbreaks have been reported from wild boar, the generally high serological prevalence of T. gondii in these animals indicates that consumption of raw or rare wild boar meats is likely to lead to an infection with T. gondii.
No freezing-resistant strains of T. gondii have been identified: freezing meat to −12°C will kill tissue cysts (Dubey 1996). Cooking is an effective means of killing tissue cysts. One controlled study demonstrated that T. gondii tissue cysts in 2 mm thick samples remained viable at 52°C for 9.5 min, but not at 58°C (Dubey et al. 1990). Tissue cysts in pork samples were generally rendered non-viable by heating to 61°C or higher temperature for 3.6 min (Dubey et al. 1990) and have been reported to survive 64°C for 3 min (Dubey et al. 1990). Cooking meat to an internal temperature of 67°C will kill tissue cysts (Dubey 1996).
One contribution of 12 to a Theme Issue 'Livestock diseases and zoonoses'.
- © 2009 The Royal Society


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Sienna Miller cast in "Live by Night"
Sienna Miller has been cast in the new Ben Affleck adaptation of 'Live by Night'.
Affleck, 42, is set to write, direct and star in the movie, which will also feature the likes of 'Guardians of the Galaxy' actress Zoe Saldana and Elle Fanning, who appeared in 'We Bought a Zoo'.
The eagerly-awaited movie is set in Prohibition-era Boston and focuses on the story of a son of a police officer who becomes involved in organised crime.
Miller is set to play the role of Emma, the love interest of a mob boss, according to Deadline, which also reported that Saldana will appear as Graciella Suarez, a Tampa resident.
Fanning, meanwhile, will play the role of Loretta Figgis, an aspiring actress who finds herself in danger.
Due to Affleck's involvement with 'Batman v Superman', the release date of 'Live by Night' has been pushed back, meaning the film will now open on October 7, 2016.
Last month, Affleck said he was unsurprised by the reaction to his casting as the new Batman.
The 'Argo' star revealed he expected there to be a mixed response among fans.
He explained: ''This is part and parcel of these movies now. There are a lot of active fans with a lot of opinions. To me, having been through a certain amount of that, it doesn't really. Everyone's entitled to their opinion.''


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Institute of Banking Personnel Selection (IBPS) has uploaded the lists of candidates provisionally allotted for IBPS PO III, IBPS Clerks III and IBPS Specialist Officers III across different states as a part of Common Recruitment Processes held for the year 2014-15. Use the below links to check your respective allotment result: Click here to see the […]Continue Reading... Comments Off on IBPS publishes list of selected candidates for PO III, Clerks III & Specialist Officers III recruitment process
IBPS has published final results for PO-III and Clerk-III exams today. IBPS PO-III Use the link to access PO-III result: http://ibps.sifyitest.com/cwepormar14/login.php?appid=llkdakadsffnahs For detailed allotment list: http://ibps.in/career_pdf/CWE_PO_MT_III_Allotment.pdf IBPS Clerk-III Use the link to access Clerk-III result: http://ibps.sifyitest.com/cweclkrmar14/login.php?appid=fnahllkdfsakads For detailed allotment list: http://ibps.in/career_pdf/CWE_CLERKS_III_Allotment.pdf Congrats to all the selected candidates!!! For those who were not selected, don't lose […]Continue Reading... Comments Off on IBPS published Final Result for PO III and Clerk III
IBPS has published a list of the all the candidates selected through IBPS PO II recruitment process. The list includes Roll Numbers and Marks of all the candidates selected in the IBPS PO II. IBPS PO II Process might be over now and allotment process of IBPS PO III should start soon. Check the below […]Continue Reading... Comments Off on IBPS published list of candidates selected in IBPS PO II
This has reference to the advertisement released by IBPS and published in Employment News dated 27th July- 2nd August 2013, regarding conduct of Common Recruitment Process CWE-PO/MT-III for recruitment of Probationary Officers/ Management Trainees in Participating Organisations. The tentative schedule of related events to follow is as listed herein under: Download of call letters for […]Continue Reading... Comments Off on IBPS Notification for Common Interview for CWE PO/MT-III
IBPS has declared the scores of IBPS PO III written test exam held in October and November this year. Also it has published the cut off scores for different sections and categories Please use the below link to check your result: http://ibpsresults.sifyitest.com/cwepothnov13/login.php?appid=sldkaadfsfahnlk IBPS has also uploaded the Sectional cut-off and cut-off for different categories… These […]Continue Reading... Comments Off on IBPS Published PO III Scores and Cut-off Marks


text: I was searching for fun India-themed craft projects… and came across this free download for a Taj Mahal paper model. Wouldn't it be fun to impress your friends with this little gem? So go and check it out right now – I know you probably needed something to do during Monday Night Football anyway!


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Tuesday, November 10, 2009
The Color of Stew
I have been so busy finalizing the new patterns and getting them off to be printed, sewing some beautiful clothing with the new fabrics for the girls which will be used for some print ads and just simply trying to keep up with laundry and this house. Normally I don't even include any housework in the list of things I am trying to get accomplished, so even mentioning that here means things are finally starting to slow a tiny little bit after what seems like months of non-stop non-stopness.
Today I insisted upon a slow stew. Which included a slow shop around the grocery with coffee in hand and a sleeping baby in the cart. (Can you think of a better way to shop?) After the ingredient chopping was complete and the bubbling was just beginning, I also insisted upon a slow walk around the yard with my camera. Everything I found through the lens plus the warm smell of beef stew that hit me once I came back in the door reminded me why I seldom feel the need to leave this house. Especially this time of year. But it also inspired me to get out the doors a bit more instead of just looking through them.
And such is the color of our stew.
If you're in search of warm and wonderful slow cooking to inspire your palette/palate, don't forget about the famous soup entry post from about this time last year. A soup cookbook in itself! (and for those of you that skim posts: No. The contest was over a year ago. But we'll come up with a new one here soon.)
Be back tomorrow to continue sharing the rest of the Little Folks fabric family.
g'nite, xo,Anna
53 comments:
Don't you just love embracing this time of year. The turn....the Fall! It's the best! The smells, the colors, hot food bubbling, fireplace cozy time, your post is so perfect!ReplyDelete
caroline- right! left it at home ;-PReplyDelete
You made me so hungry, I warmed up some leftover beef stew from the fridge-thanks for the beautiful pictures!ReplyDelete
I agree with Natalie above. It made me slow down and savor every single word you wrote. I'm home alone tonight. Can I come over and share your stew?ReplyDelete
I miss those trips to the store when my first born was litle. He would sit up front in the cart and we would have great conversations from the time he could sit upright in the cart. I was just htinking today about how much I just love being at my home. I am such a home body and fall really gets me ready to dig in deep and enjoy my time at the house. Your stew looks great and is such a nice cozy thing to make for fall.ReplyDelete
I'm glad you mentioned your soup blog entry post from last year, I was able to bookmark it so I can read thru to see which ones I'm interested in. So far the pumpkin w/curry is calling my name.ReplyDelete
No wonder your fabrics have such saturated colours. Inspiration is right at hand.ReplyDelete
I was just talking to my friend yesterday about stew... there's something about fall that just makes you want to make a stew. In fact, we made one Sunday, and we are still enjoying all the leftovers... stew is even better on Day 2!ReplyDelete
Stew is on my menu for tomorrow....hmmmm....thank you for the fallish inspiration! :)ReplyDelete
I totally hibernate this time of year! When it's cold, dark, and wet, I want the comforts of home -- and a great soup. Thanks for referencing the older post -- I was not a blogger or follower of blogs back then, and I always love trying new soup recipes.ReplyDelete
the stew looks fabulous and the fall colors are lovely - thanks for sharing the photos, especially for those of us who live where there are no fall colors!ReplyDelete
What a gorgeous garden. I love the treehouse and firepit.ReplyDelete
you can see the happy times.
Very beautiful pictures. And do I see a treehouse there? How great!ReplyDelete
Funny to read about take-away coffee. Something that sounds so American to me. You don't see that here. Coffee for me is something you sit with, together with someone. *grin* Love those cultural differences.
The stew looks delicious!
I absolutely love this time of year! Especially with all that cooking up soups and baking going on. Not to mention all that color going on in the yard. My stepdaughter said the other day that she thought this time of year was gloomy and boring. I mentioned that this was a wonderful time of year, as one can be busy and create lovely things with thy hands. She asked if I would teach her to sew up some things. All the more reason to collect fabrics and get excited about sewing things up!ReplyDelete
um, a yellow picnic table?! ... fab! i recently garbage picked {did i just admit that?} a very similar red chair that needs to be stripped and re-painted. looking forward to that! i cannot wait for your *new* 'stuff' ... i hope - hope - hope somewhere in this business that there will be a splat mat pattern available?!?ReplyDelete
Your home is so beautiful! and you're right to enjoy it!ReplyDelete
I like to peep into your blog once in a while and your colorful world is fantastic! this made me order some of your fabrics a couple of days ago in a german e-shop and I'm looking forward to sew them into projects!!!
go on like this, your're just wonderful!
breath taking. thank you for sharing!ReplyDelete
The stew looks sumptuous. Mmmm. Almost enough to make me eat meet again. Thanks for the lovely pictures!ReplyDelete
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text: A Little Backround: In approximately 4-7 days, I will be kicked from my home, I plan on by then having at least a $100 a income coming from a mix of article writing and Ewhoring. My Skills are: Strong Article Writing Ability A Number of Tools This Forum $25 but I plan on article writing until I get the investment of $250 for my EWing method. A decently powerful arrays of methods and techniques to get me to my goal. Motivation Goals: -$100 a day starting in seven days. -Eventually raise to $500 a day for MORE than comfortable living. -Live in a comfortable apartment -Live a comfortable lifestyle and finish school. Immediate Goals: -Get $250 capital for the EW methods I have which Will DEFINITELY get me to my goal. -Find a good weekly paying EW network with lack of shaving and good support. Methods: -Article Writing for $3 per 300-350 word article or 100 words/$1. -Ewhoring method I saw some time ago with offline EWs to assist. DisAdvantages: I can only write for about 9 hours a day, and can write about 4-5 articles an hour, and I will only take a few micro breaks. So about 30-36 articles a day. -Lack of Time. -Lack of Support. -Lack of Capital But I will not quit. If I need $250/$3 an article = about 83 articles. I will pursue this to the fullest.I will post one-three times a day in here and record random thoughts to stay motivated. Onward?


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The Spa Medical Practice, Droitwich Medical Centre, Ombersley Street East , Droitwich Spa, Worcestershire, WR9 8RDTel: 01905 772389
Our practice provides the usual services which include:
From the age of 25 to 49 you will be offered a cervical smear every three years and from the age of 50 to 64 you will be recalled every five years. Invitations are sent from PCSE (Primary Care Support England) and the smear is taken by either Practice Nurse Caroline Gaynor or Angela Lapworth.
When you receive your invite please telephone the surgery on 772389 and option 2 to make a twenty minute appointment with the either of practice nurses mentioned above for a smear test.
The Health visitors no longer have a clinic at the practice but they offer Droitwich Baby and Child Health Clinics at two locations.
On a Wednesday at WANDS, Farmers Way, Droitwich, WR9 9BF between 09.30 and 11.30 and on a Friday at Droitwich Medical Centre, Ombersley Street East, WR9 8RD between 10.00 and 11.30am on the first floor.
To contact a health visitor ring 01905 681025
A full pre-conceptual, antenatal and postnatal maternity service is provided by the Droitwich Community and your doctor. Our midwife runs a clinic only on a Tuesday morning, so please book your first appointment with the midwife as soon as you know you are pregnant. Your Post-natal check will be done by with your usual GP at six to eight weeks.
To contact a midwife ring 01905 681042.
We offer comprehensive management of the above chronic diseases. It is essential for all our patients with a chronic disease to have regular monitoring checks with the Practice nurse. If you have any of the conditions listed above please make an appointment in your month of birth in the appropriate clinic.
Appointments are now available with our Clinical Pharmacist Mr Mohammed Jivraj.
Clinical Pharmacists are highly qualified healthcare professionals who train for many years to become specialists in medicines and how they work. They work directly with the General Practice Team, to ensure the safe and effective use of medicines to enable the best possible outcomes for patients.
Clinical Pharmacists can provide expert advice on medication. They can help you by making sure the medicines you are taking are right for you, reviewing your medicines and helping you with lifestyle changes to help manage your condition.
Our Clinical Pharmacist can also see patients for their annual Asthma and Hypertension reviews.
The practice offers a contraception service for patients of all ages, which is confidential and includes emergency contraception.
Please make an appointment with the practice nurse for follow up contraceptive checks. Unfortunately the practice does not insert or remove contraceptive implants.
Droitwich Sexual Health walk in service is from 16.10.2018 be a appointment only service.
Appointments will be available every Tuesday morning between 09.015 and 11.30 and will be available to book the day before.
To book an appointment please ring 01562 514512 on Mondays between 15.30 and 17.00
Regrettably, the practice is no longer funded by Worcestershire County Council to provide Quit Smoking Advice and GPs are unable to issue your prescriptions for your Nicotine Replacement Therapy medication. If you need help and support to quit smoking please refer to the County Council Website www.nhs.uk/smokefree.
The doctors and nurses at the surgery are aware of the difficulties faced by both patients and themselves when carrying out intimate examinations unsupervised in the consulting room.Please do not be afraid to request a chaperone to be present in the room - we will not be offended.
Practice staff
The Spa Medical Practice is the base for the town INR service. There are daily appointments in the clinic with Karen Read and Maria Walker or Beth Manton visit the housebound. The clinic provides monitoring and adjustment of the drug warfarin and provides patients the opportunity to learn about their medications, have their blood tests evaluated, and dose adjustment be made accordingly.
We run a town based ultrasound clinic offering a wide range of scans on a Tuesday and Thursday. The clinics are at either Spa Medical Practice or Corbett Medical Practice. Please note a referral is needed from your doctor to access this service. Please see the attached sheet for Ultrasound instructions. If you are diabetic please note you can eat up to three hours before your scan but please eat something light such as crackers, digestive biscuits, salad etc. Squash can be drunk and it is advisable to bring biscuits for after the scan.
The GPs run a minor surgery clinic. They are able to remove certain skin lesions under local anaesthetic. If you have a skin lesion you are worried about or would like removed then make an appointment with a GP first and they will be able to assess whether it could be removed in the minor surgery clinic. You will be out onto a waiting list and contacted with an appointment when one becomes available.
As of September 2017 the practice is no longer a minor injury unit. If you have an injury please advise reception and they will book you an appointment. If it is an injury that may need sutures or xray please go to your nearest Minor injury unit. In Droitwich this will be either the Princess of Wales hospital in Bromsgrove or Worcester Royal Hospital.
We offer a service to patients to enable direct appointment booking to a hospital of their choice following referral by the GP. This service is called Choose and Book.
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Electronic devices such as, but not limited to, recording devices, ipods, radios, CD players, and video games, are not permitted on campus and will be confiscated for safety purposes. Personal items such as cameras, designer sunglasses, designer purses, designer jackets, and/or other valuables should be left at home. If you bring them to school, they may be placed in the office until the end of the day. The school district does not provide insurance for lost, stolen, or damaged personal property on school premises. Repeated offenses will result in detention, assignment to the support room or suspension and/or requiring parents to pick up confiscated items. Confiscated items not picked up by the last day of school will be disposed of.
Students are allowed to carry cell phones on campus, but they are not allowed to have them turned on and in use on campus during the school day. Students who do not follow the rules will have their phones taken and held in the office. The student will be allowed to pick up the phone at the end of the school day on the first offense. Repeated offenses will require a parent to speak to the principal or the principal's designee.
Please help us keep our classrooms and school grounds clean by not bringing gum or candy to school. Gum, gum wrappers and candy wrappers, more often than not, end up on the classroom floor and/or school grounds, causing a litter problem on our campus. Therefore, students are not to bring either gum or candy to school. If a parent feels it is necessary for their student to eat a piece of candy at school, then they may pack a piece of candy and/or a candy bar in their student's lunch bag or lunch box. This item may be consumed in the cafeteria at lunch time only. All gum and other candy brought to school will be confiscated and will not be returned. Continued violations of this rule will result in disciplinary action as defiance of authority resulting in detention, community service, alternate placement, support room placement and/or suspension. It is our belief that both gum and candy serve only to distract from the educational process.
Restrooms are open for student use throughout the school day. During class time, students must obtain a hall pass from the teacher to use the restroom. Students using the restroom are to cooperate with staff members checking or supervising the area. Restrooms are not to be used for 'social gathering'.


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The party fled from the Ancient Black Dragon that was in the process of being crystalized, but soon realized they were not being pursued. After locating the recently slain young black dragon's small treasure hoard, they tried to exit the dwarven ruins and were set upon by another strange crystal creature and a pair of small dragons. After defeating them, the party made all haste to Whistledown to organize an evacuation via airship to the safety offered by Ruch. The evacuation plans were interrupted by a small force of Half-Dragons, but the party vanquished those in turn. All the gnomes were loaded onto a pair of airships and they took to the sky, but had to swing wide around the mountain as the Ancient Black Dragon had dragged itself out of its hiding place and onto the side of the mountain, along with a great swarm of its offspring.
The Ancient Black Dragon sent many of its (uncrystalized) offspring after the fleeing airships, forcing the party to man what meager weaponry the ships had to repel the waves of attacks. Despite a troubled start where Hazarex was briefly transformed into a wolf, the party brought down all the dragons that attacked the airships. No further pursuit followed, so several hours later in the early morning the inhabitants of Whistledown landed safely in Ruch.
The party immediately sought out the tiefling sisters and requested that Princess Faila come to them so they could all be informed of the that a Crystallized Ancient Black Dragon portended. Akta speculated that the reason the Crystalline Devil would corrupt the dragon was to gain enough power to kill Wee Jas and the Raven Queen in order to break the time loop cycle that was enabling the group to work against the crystal creatures' designs. The party was quick to realize that the nearest portal large enough to accommodate a creature of the dragon's size is the city gates of Ruch, making the dragon's next point of attack likely to be the city of Ruch.
Akta had some good news, though. She had discovered an analog to the dark power that the King of Highgore had infected them with; a glorious golden power wrought of their own reality, so therefore safe to use. The power was easily bestowed on everyone by a brief ritual. This only left the problem of the sheer might of an Ancient Black Dragon. Here the Princess Faila came through. She offered to let the group pilot the Royal Family's giant constructs if they were able to modify the mechs to be able to amplify the new golden power. Widget and Akta were then taken to the Royal Artificers' Guild to work on this problem, and the rest of the party left to their own devices.


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Hardest Hit: Pastor's Sons
Paul CoughlinPaul Coughlin's Weblog
- 2008 Jul 15
While offering individual instruction for the Christian Nice Guy problem throughout the
Let that sink in for a moment. Think about its ramifications.
In No More Christian Nice Guy, I wrote about subtle and overt forms of spiritual neglect from the pulpit to the pews when we are encouraged to emulate a gentle Jesus meek and milk who really did not exist. Now I'm going to reveal one of the most damaging forms of spiritual abuse that comes from the pews to the pulpit. And like most spiritual abuse, it isn't intention, though the result is sure real.
The pastor's sons I work with are almost always separated, divorced, or on the verge of divorce. Their wives or ex-wives complain that they just don't possess the kind of vigor or fire that they want from a husband. They sometimes complain that their husbands drain them of energy instead of invigorating them.
These men often have no definable self, a fact their wives point out, sometimes with disgust, when walking out the door. Our sermons encourage us to have self-control, but these men don't have a self to control in the first place. They are anchorless and are often too easily influenced by others.
Because they've been trained to be pleasant to everyone, they often over-yes and under-no others. Many think that it's simply wrong to tell others "no." And when they do, they lose sleep at night. Being human, having boundaries, feels unnatural and sinful to them.
They are resentful of how people have treated them and their families, and because they don't think they should experience or own negative feelings, they don't know what to do with them. They often denounce them as unchristian as opposed to being honest and working through them. As a result of poor treatment from others, they do not trust others very well, including their wives.
They are known by many, but not knowable in part because they possess personas, an assumed identity, but not discernable personalities. They feel that they have been forced to play rolls in life, to wear masks (one of the original meanings of "persona"), which is exhausting and depletes them of integrity and healthy self-confidence. Integrity makes us conspicuous, and it is always painful. It's important to realize that the word Jesus used for "hypocrite" while denouncing the Pharisees literally means "wearer of masks" (Matt. 23:13).
They know the right words to use in marriage—they know how to perform—but they don't know how to deeply love another person. This is what personas do: they are like holograms and holograms by nature are all surface, no substance.
They think that never showing indignation (which includes as part of its original meaning "much to grieve") or other forms of healthy anger toward anything or anyone and always remaining gentle are among the highest forms of spiritual maturity, even though Jesus wasn't always gentle and pleasant. More so, their spiritual training has them believing that it's wrong to not be gentle. But if this is true, then Jesus was wrong. Jesus sinned.
For example, one son of a pastor who asked me to help him overcome passivity in marriage told me how his mother's "gentle spirit" made her the perfect Christian woman.
"She was always so gentle," he said warmly. "She never got angry about anything. She was perfect!" he gushed like a child.
My inner Dr. Phil came out. "Perfect?!" I exclaimed. "In more than 25 years of ministry, she had to have seen wickedness and evil tearing people apart. She had to have seen divorce, adultery, child abuse, drug addiction, homicide and even suicide. And she never became indignant when she saw that kind of destruction, the way Jesus was indignant?!"
There is much to grieve in this life, and responding to life's destructive forces gently instead of with indignant power may well make us accomplices to these destructive forces. For many, gentleness is a disguise for being dispassionate spectators of life and a hiding place for fear and passivity.
The pastor's sons who ask for help are always expected to be happy, one of the most damaging myths in evangelicalism today. I call this the Happiness Mentality, and it needs to go away.
Here's an example from my own life that I write about in my upcoming book, Unleashing Courageous Faith. I went to a funeral of someone I loved and the minister said that we should not shed tears because he was with Jesus now. "This is not a day of mourning, but of celebration!" he said, with a level of enthusiasm that appeared fake to me. He didn't even seem to believe what he was saying. He spoke as if he was following a script.
"Celebration?!" I thought. "I loved this guy. I'm not going to celebrate his death, I'm going to weep his loss."
True to the Happiness Mentality that we slavishly idolize, this talker of spiritual matters did not allow for the spectrum of human life, love, and longing because this spectrum is not considered "spiritual." He didn't allow for both weeping and celebration. True to his spiritual training, he axed the negative stuff and gave us a plateful of spiritual dessert.
And instead of leading us toward a more loving and compassionate orientation toward life as it really is, he encouraged a very selfish approach toward those closest to the family man. Why express your condolences to his 12-year-old daughter who just lost her father when the spiritual leader just told you there's nothing really to cry about? She remains untouched and unloved. The Happiness Mentality in many ways is actually a cruel mentality.
This plastic world of our own making helps to make the Christian faith appear more and more irrelevant and ill.
Pastor's sons who find No More Christian Nice Guy indispensable have not been allowed to exercise a real will of their own. Instead, their wants, needs, desires and dreams have been subjected to the will of others. As a result, they are pretty much the ideal Christian child because they are tremendously pleasant to be around, but they later flounder in adult life.
Ultimately, these pastor's sons just don't feel safe in life, and if you gave them a shot of sodium pentothal—truth serum—my guess is most of them would say that it's sinful to be human.
A dear friend of our ministry in
Pray for the pastor's sons and daughters who you know.
Treat their parents with dignity when you disagree with them.
A friend of mine, Nate Larkin, Founder of Samson Society (www.samsonsociety.com) and author of Samson and the Pirate Monks: Calling Men to Authentic Brotherhood, is a pastor's son who knows the pressure they feel to live two very different lives. Here's what he recommends: "Let the kid be a kid. He's not a representative of the pastor, nor is he responsible for upholding his father's reputation. Let him make his mistakes. Give him the same grace you'd give any other kid in the church."
Second, "Don't compete with the kid for his father's attention. Too many pastor's kids grow up feeling that the congregation comes first in their father's affections. As a result, they become either resentful and rebellious or overly compliant and artificial in an effort to attract their father's attention."
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Paul Coughlin is the author of numerous books, including No More Christian Nice Guy and No More Jellyfish, Chickens or Wimps. He also co-authored a book for married couples with his wife Sandy, titled Married But Not Engaged. His articles appear in Focus on the Family magazine, and he as been interviewed by Dr. James Dobson, FamilyLife Radio, HomeWord, Newsweek, C-SPAN, The New York Times, and the 700 Club among others. Paul is founder of The Protectors, the faith-based answer to adolescent bullying, which provides curriculum for Sunday Schools, private schools, retreats, and individuals that trains people of faith to be sources of light in the theater of bullying.
Visit Paul's websites at: http://www.theprotectors.org, and http://www.paulcoughlin.net


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The Family Under the Bridge - by Natalie Savage Carlson
A Newbery Honor book, this is the delightfully warm and enjoyable story of an old Parisian named Armand, who relished his solitary life. Children, he said, were like starlings, and one was better off without them.
But the children who lived under the bridge recognized a true friend when they met one, even if the friend seemed a trifle unwilling at the start. And it did not take Armand very long to realize that he had gotten himself ready-made family; one that he loved with all his heart, and one for whom he would have to find a better home than the bridge.
Armand and the children's adventures around Paris -- complete with gypsies and a Santa Claus -- make a story which children will treasure.
About the Author:
Natalie Savage Carlson is fondly remembered as the author of the much-loved Orpheline series and Surprise in the Mountains. Born in Virginia, Ms. Carlson later lived in Oklahoma, Rhode Island, and the Pacific Northwest. She eventually settled in Florida prior to her death.
9-4 PST Monday-FridayEmail: email@example.com: 800.889.1978 or 805.542.9847Mail: 1306 Mill Street San Luis Obispo, CA 93401Warehouse: 805 Fiero Lane Suite F San Luis Obispo, CA 93401Copyright © 2009-2018 Beautiful Feet Books. All rights reserved.
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Research interests
My research is focused on predicting the spatial distribution of invasive freshwater fish. I am particularly interested in forecasting species distribution changes under global changes.
Invasive species, distribution modelling, climate change
Lauzeral C., Leprieur F., Beauchard O., Duron Q., Oberdorff, T and Brosse S. Identifying climatic niche shifts using coarse-grained occurrence data : a test with non-native freshwater fish. Global Ecology and Biogeography, 20 : 407-414
Lauzeral C., Grenouillet G. and Brosse S. Dealing with noisy absences to optimize species distribution models: an iterative ensemble modelling approach. Plos One 2012.


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An intense, chilling and compelling twist-a-minute psychological thriller.
When a young girl dies by hanging, terror strikes the hearts of the students and staff of a
Vancouver Island school. Mourning the loss of his mother,
RCMP Inspector Patrick Painter must push aside his grief to investigate the
child's dreadful death, only to find he's on a collision course, with the past
and present crashing into a deadly game of secrets, suicide and murder.
Do you believe in a Family Curse?
Book Excerpt
Tragedy strikes local family once again.
Thirteen year old Nathan Winsloe died yesterday at his Metchosin home. He was found hanging from a belt slung over the bar of a clothes closet. At his feet lay a bouquet of shamrocks.
While authorities refused comment, a source who asked not to be identified said suicide was suspected. A family friend added that the teenager was distraught over his mother's recent death. Vivian Winsloe (nee Brownlee) died March 17 of an apparent suicide.
"Suicide is a learned behaviour," said Clare Mully of the Greater Victoria Suicide Prevention Unit.
Nathan is survived by his father and two sisters. The investigation continues.
The Victoria Observer, March 22, 1978
Behold, I shew you a mystery; we shall not all sleep, but we shall all be changed.
Act One synopsis, Rigoletto, an Italian opera by Giuseppe Verdi, 1851
In a sixteenth century ducal palace, a female guest attracts the philandering duke's attention. Count Monterone storms in, denouncing the duke for seducing his daughter. When mocked by Rigoletto, a hunchbacked court jester, Monterone thunders a father's curse upon him. A widower with only one child, Rigoletto is appalled.
Later at his home, Rigoletto is accosted by a professional assassin who offers to kill the young man seen spying on the jester's cherished daughter, Gilda. Rigoletto dismisses him and then embraces his daughter. He cautions her about leaving the house and orders her chaperone to guard her. Gilda denies being followed from church and Rigoletto departs.
Gilda is stricken with remorse, having concealed her love for the man who has been following her.
The duke steps from his hiding place, pays off the chaperone, and, pretending to be a poor student, declares his passion for Gilda. Upon hearing footsteps, he is forced to flee. Gilda, singing Caro nome, slowly retires upstairs.
That evening, Rigoletto is tricked by courtiers into assisting in the kidnapping of his daughter. When he realizes she is gone, the jester laments that Monterone's curse has begun.
Tremble, old man, at your ruler's wrath that you provoked,
There is no remedy. This outburst was fatal for you.
The child was not schooled in the hangman's knot. It certainly wasn't the sort of instruction covered in Sister Benedict's grade five class. The Sisters of taught their fifty six charges to live in peace with God and offered what the Mother Superior called a diamond education: reading, writing, arithmetic and religion.
More's the pity, for it took her a long time to die. Gasping, kicking and scratching at the smooth leather belt that, in the end, was just tight enough.
* * * * *
The casket disappeared into the grave, sunlight stubbornly streaking off its small brass nameplate. No, Mom, no! Constable Patrick Painter's stomach lurched and he gulped for air. He tasted salt water, seaweed and cedar. For an instant, he fled Holy Trinity's graveyard by the sea and gratefully felt the nearby
Pacific Ocean's
cool, wet touch on his face.
Memories of beach bumming and cove hopping sliced his consciousness as the red Coast Guard vessels winked at him from
. As a child, he'd been terrified of
losing his mother. Every night he'd say, "See you in the morning,
Mom," a childish command, he now realized. A desperate effort to receive a
promise of not being left alone…with him. He'd wait as long as possible to shut
his eyes, rubbing the soft sheets, to avoid the risk of sleeping and her dying.
Patricia Bay
Each morning, he would awake with a start. Then he'd leap out of bed, dash into the hall and listen for her soft voice. Only when he heard it could he relax. Sometimes he thought his bladder would burst, but finally he'd hear her, then he would race into the bathroom.
The beeping of his pager startled the small clutch of mourners just as his father reached down to grab a handful of freshly dug dirt. What the—? Flushed, Constable Painter peered up under dark bangs and caught the priest's critical glance. A disap-proving clucking of tongues slipped its way round the circle of onlookers. Cursing inwardly again, he instinctively slapped his side and the short, nasal bursts stopped.
Patrick looked away, eyes skittering from the moss pocked tombstones, to the round earth mound, to the grave pit—Mom!—finally, up into his father's black and staring eyes. The younger Painter knew, with the last sudden, sublime finality that strikes a driver careering off a bridge, that the horror was true. He was alone with the man he loathed.
Leonard P. Painter opened a large hand and a clump of
Vancouver Island soil plopped onto
his wife's coffin. A long, ragged scar gleamed on his left cheek, contrasting
sharply with his ruddy complexion. The recently retired staff sergeant of the
Victoria Police Department turned carefully on his heel and tramped away.
Several mourners struggled to follow, shoulders hunched with grief.
Breathing deeply, Patrick waited while the others followed suit. A small, well-dressed man arrived breathless and late, offered condolences and an envelope, which Patrick shoved unthinkingly into his suit pocket.
Finally, only he and the priest remained. Patrick knew he had deliberately left the beeper on. What was he hoping for? That he'd have to leave and so avoid the nightmare? You freakin' wimp, Pat, you'd run from your own mother's funeral? You'd use the job excuse on her one last time? Despite himself, he snorted.
"Patrick?" A hand touched his shoulder. "Son, you all right?"
Painter blinked into the sun. "What? Oh, yeah. My mother's dead and if I were any better, I'd be twins."
"What?" The priest's sunken eyes stared. "Patrick, maybe we should—"
Marching off to his car phone, Constable Painter didn't hear the rest.
* * * * *
She struggled but couldn't grasp air. Voices of unattainable beauty rose round her like a shimmering fountain: lyric and mezzo sopranos rushed skyward, chased by a single clear treble. An astonishingly brilliant light enveloped her. Glowing faces and flowing robes darted across her path. Tempest Ivory inhaled profoundly, right from her toes; still no sound escaped her lips. She had to sing. She must!
The voices scaled atmospherically, forcing Tempest's hands over her ears. Ugly and sharp now, the bizarre harmonies jarred her. With a crackling whoosh, the light swallowed all the air. The unknown choir rushed at her like sheets of rain, their piercing shrieks pummelled her and she collapsed, trembling and gasping. Her soul shrivelled; she sobbed for her loss. A ringing crammed her skull and she woke with a gasping, heaving shudder.
Sunlight caressed her as she slowly began reattaching herself to her cool satin sheets, her cannon ball bed, her tiny cottage in pastoral North Saanich,
. The phone rang again. Her heart still
thudded, though her breath returned in frayed whispers. A ragged clank raised
the coppery hair on her freckled arms. She wasn't alone. Tempest bolted up,
started screaming, "Get outta my house!" and stumbled out of and
around her bed into her living room. British
A dark, hooded figure streaked from the galley kitchen into the narrow hall. Two strides to the front door. Too tortoise like to follow, Tempest unhinged her jaw, dug back into her spine and unleashed her only weapon. Her voice rose, effortlessly vaulting high C, saturating the tiny space, slamming the intruder's senses.
The figure stubbed its toe near the front door. With a surprised bellow, the prowler reached down, clutched the offending box and hobbled out into the dappled light.
Chest heaving in her lace nightgown, Tempest thumped barefoot onto her wooden porch. Just as her motorbike was thrown to the ground, she saluted the interloper with an angry, raised fist as he faded into the surrounding second growth coniferous forest.
Though incapable of physically describing her uninvited guest, Tempest could mentally replay the baritone's startled curse.
About Nicola:
Nicola pens mystery and inspirational novels, creates interactive books for the iPad, podcasts about genre writing (The Novel Experience), and teaches electronic publishing, when she's not playing Old-Timer's hockey, growing blossoms and bamboo or eating chocolate fudge.
Her first contemporary women's series, the Sisterhood of Shepherds, debuted with HEARTSONG in May 2014 (MantleRockPublishing). Nicola's swinging whodunit, TEED OFF! (republished in February 2014 by OakTreePress), features professional golfer and coroner Riley Quinn.
Her other novels include a psychological thriller (A HEMORRHAGING OF SOULS), six novels in The Church Choir Mysteries series and a multimedia online thriller, UNNATURALSTATES.
In addition, she has published three ebooks, YOUDUNIT WHODUNIT! HOW TO WRITE MYSTERIES, SELF-PUBLISH YOUR E-BOOK IN MINUTES! and TOP TEN GARDENING TIPS, as well as her first musical interactive children's book for the iPad, SAVING GRAPE-JELLY CHEEKS.
Nicola lives in a small seaside town on southern
. Vancouver
Island, British Columbia
To purchase Nicola's book:
Nicola Furlong is giving away an e-book copy of A Hemorrhaging of Souls. The giveaway is only available to
addresses. U.S.
To be entered in the book giveaway, leave a comment along with your email address. You may enter the book giveaway twice -- once on each spotlight post. (It's not too late to go back and leave a comment on yesterday's post.)
Off to read another great book!
Sandra M. Hart