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Flhip.com has released its latest restaurant openings report, providing restaurant vendors with a sampling of fresh sales and marketing leads that can be found on their website.
Flhip provides regularly updated lead lists, allowing vendors to check for updates at any time. To see leads in your sales area click here.
Philadelphia, PA – Aqimero
International celebrity chef, Richard Sandoval, will open his first Philadelphia restaurant in the Ritz-Carlton as the hotel moves through a $24 million renovation with the goal of opening in May 2016. The menu will revolve around steaks and sustainable seafood and feature a wood-burning grill and seating for 206 located at 10 Avenue of the Arts downtown.
Wichita, KS – Caesars Table
The popular two-year-old upscale buffet concept will open in its new location in May 2016 and maintain the mid-scale international cuisine it's known for during lunch, will open for breakfast and eventually dinner service as well. The new location will be 125 N. Market St and will offer catering to complement their on-site service.
Miami, FL – Laffa My Mediterranean Kitchen
The Owners of My Ceviche Restaurant will open their new fast-casual, build-your-own meal concept sometime in the first quarter of 2016 at 1250 S. Miami Avenue. The menu will offer seasonally changing eastern Mediterranean favorites with house-made hummus and pita bread serving as menu anchors along with their fresh juices.
San Jose, CA – Our House
185 Park Avenue in the San Jose financial district will be the home to the new casual operation that will first open for lunch and then breakfast in early March, opening is slated for mid-to-late January 2016. The menu will be seasonal with local, organic foods that are GMO-free.
Chicago, IL – Mora
The Plainfield, Chicago suburb will be the home of a new Asian-fusion concept featuring eastern-style barbecue and sushi. Located at 24108 W. Lockport St. it will be the most recent tenant of a historic local government building that also housed the town hall, jail, fire and police departments around 1900 to 1920 and will open mid-January 2016.
Houston, TX – The Brisket House
Operators of this Houston landmark will open their third unit at 3301 FM 1960 Road west in March 2016. The new unit will dwarf the two previous at just over 10,000 sq. ft. however, it will feature the same slow-smoked beef, pork, chicken and sausage that made them famous. Owners say to look for some new menu items once they are up and running.
For more information or to view the leads in your area, please visit www.flhip.com.
Ken Roberts


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Teaching or learning about Indigenous culture and history? See the following clips, produced during development of the exhibition Yiwarra Kuju: The Canning Stock Route.
Warning: Aboriginal people are advised that some of these videos show people who have passed away.
Martu elder Friday Jones leads Birriliburu artists and Canning Stock Route project team members to Jilakurru where they introduce themselves to the Country.
When Aboriginal people visit Country they've never been to before or have not visited for a long time, they introduce themselves. Martu people do so by throwing handfuls of sand into the water. Many waters are inhabited by powerful ancestral beings — and in this way they are introduced to people through their scent. Once properly introduced, the Country protects its visitors and Countrymen. Aboriginal people from other areas introduce themselves to important sites in similar ways, sometimes rubbing a stone on their skin and throwing it into the water.
Birriliburu artist Vera Anderson explains as Mangkaja artist Mervyn Street cuts a 'spear tree'. Then, Vera describes how old people used to make spearheads from stone, fixing them to their handles with spinifex glue.
Ngurra artists, Putuparri Tom Lawford and Kuji Rosie Goodgie, prepare a resin used to make glue and bush medicine. Kirti or jinalpa is a special kind of glue, made from the shiny black resin which ants gather from spinifex. Aboriginal people collect the resin from inside termite mounds, clean it of debris and soften it in the fire. Kirti can be used to attach blades to spear handles and, when it hardens, it forms a super-strong adhesive.
This resin has a beautiful fragrance. It is also used as bush medicine for cold-sickness and is placed on the fontanels (the gap in the skull) of newborn babies.
Vera Anderson draws stories in the sand for her granddaughters.
In April 2008 Martumili artists congregated at Kunawarritji (Well 33) for a major painting and weaving workshop. At this workshop Kunawarritji kids produced their own collaborative painting of Kunawarritji Country. It now belongs to the community's Rawa School.
Birriliburu artist Clifford Brooks describes how his family encountered the tracks of Len Beadell's bulldozer on the Talawanna track.
Artists from six remote art centres congregated at Well 36 for the largest painting workshop of the 2007 return to Country trip. Many paintings were produced at this workshop. The thrill of family reunion and the opportunity to return to Country after many years inspired and energised the artists involved in these special painting workshops.
Men from five art centres collaborated on a painting depicting the Country from wells 33 to 38. Jeffrey James, Helicopter Tjungurrayi, Tom Lawford, Patrick Tjungurrayi, Charlie Wallabi Tjungurrayi, Peter Tinker, Clifford Brooks and Richard Yukenbarri plan their painting of Canning Stock Route Country. Jeffrey James names each of the waters depicted on the finished canvas.
Martumili artists, and sisters, Muni Rita Simpson, Mantararr Rosie Williams and Jugarda Dulcie Gibbs, paint their story of the Seven Sisters at Kilykily, Well 36. The Seven Sisters travelled through this Country in the Dreamtime making many of the waters that would later become stock route wells.
About the clips
The above videos were produced for One Road, an eight-metre long multimedia interactive featured in Yiwarra Kuju: The Canning Stock Route, an exhibition developed by FORM, an arts and cultural organisation based in Western Australia, with the National Museum of Australia. One Road features around 150 videos made by Nicole Ma (Nicole Ma Productions) and emerging Aboriginal filmmakers Curtis Taylor (Martu Media), KJ Kenneth Martin (Kimberley Language Resource Centre), Morika Biljabu (Martumili Artists) and Clint Dixon (Goolarri Media). Most of the raw footage in these videos was shot between 2007 and 2008 on the FORM Canning Stock Route Project.

subset: commoncrawl


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Today I was out in gusty condition on both kites. I am big fun of NEO kites but today I noticed the depower of the SST is way better than NEO. I had to punch out the NEO were I would not have to on the SST due to a big jump of the wind speed. Sharing information for kite surfer who wants information. I think I am not buying any more NEO kites. I have been rinding NEO for 3 years and SST one year. I have a full quiver of both kites.
below is the closes wind meter


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Bill Roache trial: Police are probing sexual assault claims from three other women
CLEARED soap star Bill Roache faced fresh anguish last night after police revealed they were probing new allegations from three more women.
The actor, who has played Ken Barlow in Coronation Street for 53 years, was found not guilty of a string of sex charges on Thursday.
A jury cleared him of indecently assaulting four girls aged 12 to 16 and twice raping a fifth after a four-week trial at Preston Crown Court.
Afterwards angry Corrie co-stars claimed he was the victim of a post-Jimmy Savile witch-hunt.
But now Lancashire Police have announced that three more women contacted them about Roache while the trial was under way.
A spokesman said the allegations were being "reviewed" to decide if further action is required.
One of the claims is from a 64-year-old married woman who says she met the actor as a teen autograph hunter.
"One witness claimed Buswell, who played Ray Langton for 10 years, forced her to touch his manhood when she was 12"
Roache, 81, is due to meet Corrie bosses next week to discuss his return to the show. But the actor, who was quizzed by reporters yesterday as he took delivery of a new washing machine at his home in Wilmslow, Cheshire, said he would not be commenting on any aspect of his court ordeal.
Roache's management team also declined to comment on yesterday's development.
Lancashire Police also revealed that his former Street co-star Neville Buswell could face arrest over a sex assault allegation made in court during Roache's trial.
One witness claimed Buswell, who played Ray Langton for 10 years, forced her to touch his manhood for "about 10 seconds" in a dressing room at the Corrie studios when she was 12.
Buswell, 71, now lives in Las Vegas where he has worked as a casino croupier and financial adviser.
The Derbyshire-born actor, who was killed off from the soap in 2005, claims he has never met or heard of the girl who made the allegations, and strongly insists they are not true.


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But since this is Disney, and Emma Watson, and magic is real, we're going to say that this is a Potter reference.
So what about that reference? Does it add anything to the scene? Well, kind of. It connects Belle to Hermione in a subtle way. They're not that different of characters when you think about it, especially in the live-action version. Belle is now an inventor who's an outcast because of her curious mind and cleverness. Hermione, similarly, is initially branded as something of a nerd by her Hogwarts classmates. That designation disappears when she joins Harry in fighting Voldemort, just as Belle stops being an outsider compared to the Beast.
We'll allow that this creates an extremely tenuous connection between the two women. Sort of ethereal and taking on the shape of the connector. You know, kind of like a Patronus. Yeah, we just went there. Consider your mind blown.


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Tuesday's program began with an introduction to BIOMIN by Herbert Kneissl, Division Marketing Officer at BIOMIN. He highlighted the firm's long history of academic partnership and research in the field of mycotoxins and gut health performance. Dr. Dieter Moll, Molecular Biology Research Team Leader, provided an overview of the structure and organization of the BIOMIN Research Center, the main platform for the firm's R&D process.
Attendees then toured BIOMIN Research Center which included visits to seven teams: analytical chemistry, fermentation, molecular biology, cell culture, microbiology, formulation and analytics. The afternoon featured a series of presentations on how mycotoxins and pathogens threaten animal performance along with innovative, natural strategies to mitigate their negative impacts.
Pedro Encarnação, Director Business Development, presented results of studies highlighting negative impacts of mycotoxins on aquaculture species. In general, cold water carnivorous species tend to have higher sensitivities to mycotoxins than warm water omnivorous ones. He highlighted that co-occurrence of mycotoxins is quite common. This poses additional risk to animals since in many cases the combined (synergistic) negative effects of two or more mycotoxins do more harm than each toxin alone. The available studies on various aquaculture species point to a wide range of harmful effects, including liver damage, impaired immune function, impaired performance, reduced protein quality, shorter storage time and higher mortality. While some of the impacts of mycotoxins on aquaculture species are known, he highlighted the need for further research on the topic.
Verena Starkl, Mycotoxin Technical Manager, pointed out that the inclusion of plant-derived matter in aqua feed introduces the risk of mycotoxin exposure to aquaculture. She highlighted the high toxicity of mycotoxins whose negative effects in animal can often be observed at low concentrations, as measured in parts per billion. Sharing the results of the latest BIOMIN Mycotoxin Survey, which reports on the occurrence of 6 major mycotoxins in agricultural commodities commonly used in feed, she revealed that 95% of finished fish feed samples tested positive for one or more mycotoxins. Turning to mitigation strategies, she explained how a successful mycotoxin risk management strategy relies upon different modes of action. Adsorption, or binding, can effectively combat aflatoxins, for example, while other mycotoxins cannot be adsorbed or absorption is considerably less effective. Mycotoxin detoxification through biotransformation uses enzymes to convert harmful mycotoxins into safe, non-toxic metabolites. Finally, bioprotection uses natural ingredients to support the liver and immune system function. The Mycofix® product line relies upon all three modes of action to deliver the most complete mycotoxin risk management available.
Prof. Loc Tran of Nong Lam University, Vietnam, presented the latest research on early mortality syndrome (EMS) in shrimp. Having identified the particular strain of Vibro bacteria responsible for the disease, there is currently no cure for the pathogen causes massive die-offs in shrimp ponds. While there is no single solution to EMS, some probiotic and phytogenic substances have reduced pathogen growth, quorum quenching and lower mortality rates. He recommended greater attention to biosecurity and improvements to broodstock procedures, reduced stocking density for ponds afflicted with EMS and a move to sustainable aquaculture.
Attila Kovács, Product Manager Acidifiers, addressed how to reduce the bacterial challenge in aquaculture using natural substances. Non-therapeutic use of antibiotics in livestock has prompted concerns about the threat of antibiotic-resistant superbugs that could threaten human health. The combination of acidifiers and phytogenic feed additives has shown enhanced effectiveness in combatting pathogens and supporting animal performance. Consisting of natural ingredients, separate in vivo trials involving shrimp and rainbow trout have demonstrated the beneficial effects of Biotronic® Top3 on pathogen control, feed hygiene and growth performance.
Rui Gonçalves, Technical Manager Aquaculture, emphasized the wide dispersion of academic research regarding yeast as an immunostimulant in aquaculture. Studies focus on various modes of action of single, combined or whole cell yeasts that, given their different scopes, are not easily compared. Furthermore, academic studies often address immune system function but fall short when it comes to examining effects on growth rates and final body weight. This oversight means that many yeast additives available involve an unnecessary trade-off between immune stimulation and animal performance. Consequently, caution is warranted in the selection of yeast additives that deliver both immune and performance benefits, such as Levabon® Aquagrow E.
Wednesday's program began with an on-campus welcome from Prof. Mansour El- Matbouli of the University of Veterinary Medicine in Vienna. Benedict Standen of Plymouth University in the UK showed how probiotics can modulate the gastrointestinal tract and presented the advantages of multi-species probiotics. He presented recent trial results showing performance improvement in tilapia using Aquastar® Growout and discussed several possible mechanisms for the positive outcome.
Barbara Weber, Scientist in Microbiology Research, explained the development of probiotics for aquaculture for both pond and feed application. She identified three ways that probiotics combat pathogens, namely: through antimicrobial activity that inhibits pathogen growth, through competitive exclusion that denies pathogens nutrients needed for survival and growth, and quorum quenching that cuts the communication signals among bacteria thus significantly reducing virulence and hence harm to animals.
Jutta Zwielehner, Product Manager Microbials, discussed how the objectives and probiotic strains used for feed and pond application differ. She outline the use and demonstrated success of the four products in the AquaStar® product line. Feed application aims to improve the intestinal microbial balance while water application promotes bioremediation that alters the microbial community in the rearing environment, degrades organic waste products and eliminates toxic substances and organic odors. Through numerous examples she showed how AquaStar® probiotics are efficient tools in pond management.
Trends in feed formulation and feed technology will center on meeting nutritional demands in the face of resource scarcity, according to Jorge Dias of SPAROS Lda. FAOStat projections indicate that farm-raised seafood will account for 62% of global consumption by 2030. He outlined the costs and challenges of producing high quality, healthy and safe food in an environmentally-sound manner. He highlighted several emerging ingredients in aqua feeds and related potential challenges.
Octavio Castro, Technical Manager Aquaculture, explained the use of phytogenic substances in addressing more efficient use of raw materials, supporting animal health and mitigating environmental impacts. He highlighted a recent trend in the reduction in use of premium materials such as fish meal and fish oil in feeds. He presented study results showing how phytogenics improve gut microflora, increase feed efficiency and adjust digestive secretion that results in better feed efficiency, fewer immunological challenges and lower environmental impact. Following the conference sessions, attendees took a guided tour of the fish trial facilities of the University of Veterinary Medicine in Vienna.


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Producer: Tim Flagler | Tightline Productions
Tim Flagler ties a Crane Fly Larva on a Dai-Riki #285 size 12 hook. "For a fly that uses so few materials, and is so easy to tie," says Flagler, "it's amazingly effective." Crane fly larvae are common in streams all across the U.S. and are a tempting meal for trout, reaching up to more than 2 inches in length. Use this fly in a dropper rig with a Copper John and you've got a deadly combination.
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Theories of the Black Death
Theories of the Black Death are a variety of explanations that have been advanced to explain the nature and transmission of the Black Death (1347–51). A number of epidemiologists and since the 1980s have challenged the traditional view that the Black Death was caused by plague based on the type and spread of the disease. The confirmation in 2010 and 2011 that Yersinia pestis DNA was associated with a large number of plague sites has renewed focus on plague as the leading hypothesis, but has not yet led to a final resolution of all these questions.
Bubonic plague theory[edit]
Several possible causes have been advanced for the Black Death; the most prevalent is the bubonic plague theory.[2] Efficient transmission of Yersinia pestis is generally thought to occur only through the bites of fleas whose mid guts become obstructed by replicating Y. pestis several days after feeding on an infected host. This blockage results in starvation and aggressive feeding behaviour by fleas that repeatedly attempt to clear their blockage by regurgitation, resulting in thousands of plague bacteria being flushed into the feeding site, infecting the host. However, modelling of epizootic plague observed in prairie dogs, suggests that occasional reservoirs of infection such as an infectious carcass, rather than "blocked fleas" are a better explanation for the observed epizootic behaviour of the disease in nature.[3]
One hypothesis about the epidemiology—the appearance, spread, and especially disappearance—of plague from Europe is that the flea-bearing rodent reservoir of disease was eventually succeeded by another species. The black rat (Rattus rattus) was originally introduced from Asia to Europe by trade, but was subsequently displaced and succeeded throughout Europe by the bigger brown rat (Rattus norvegicus). The brown rat was not as prone to transmit the germ-bearing fleas to humans in large die-offs due to a different rat ecology.[4][5] The dynamic complexities of rat ecology, herd immunity in that reservoir, interaction with human ecology, secondary transmission routes between humans with or without fleas, human herd immunity, and changes in each might explain the eruption, dissemination, and re-eruptions of plague that continued for centuries until its unexplained disappearance.
Signs and symptoms of the three plagues[edit]
The three forms of plague brought an array of signs and symptoms to those infected. The septicaemic plague is a form of "blood poisoning", and pneumonic plague is an airborne plague that attacks the lungs before the rest of the body. The classic sign of bubonic plague was the appearance of buboes in the groin, the neck, and armpits, which oozed pus and bled. Most victims died within four to seven days after infection. When the plague reached Europe, it first struck port cities and then followed the trade routes, both by sea and land.
The bubonic plague was the most commonly seen form during the Black Death, with a mortality rate of thirty to seventy-five percent and symptoms including fever of 38–41 °C (101–105 °F), headaches, painful aching joints, nausea and vomiting, and a general feeling of malaise. Of those who contracted the bubonic plague, four out of five died within eight days.[6] Pneumonic plague was the second most commonly seen form during the Black Death, with a mortality rate of ninety to ninety-five percent. Symptoms included fever, cough, and blood-tinged sputum. As the disease progressed, sputum became free flowing and bright red and death occurred within 2 days. Septicemic plague was the least common of the three forms, with a mortality rate close to one hundred percent. Symptoms were high fevers and purple skin patches (purpura due to DIC). Both pneumonic and septicemic plague can be caused by flea bites when the lymph nodes are overwhelmed. In this case they are referred to as secondary forms of the disease.
David Herlihy[7] identifies from the records another potential sign of the plague: freckle-like spots and rashes. Sources from Viterbo, Italy refer to "the signs which are vulgarly called lenticulae", a word which bears resemblance to the Italian word for freckles, lentiggini. These are not the swellings of buboes, but rather "darkish points or pustules which covered large areas of the body".
Molecular evidence for Y. pestis[edit]
In 2000, Didier Raoult and others reported finding Y. pestis DNA by performing a "suicide PCR" on tooth pulp tissue from a fourteenth-century plague cemetery in Montpellier.[8] Drancourt and Raoult reported similar findings in a 2007 study.[9]
However, other researchers argued the study was flawed and cited contrary evidence. In 2003, Susan Scott of the University of Liverpool argued that there was no conclusive reason to believe the Montpellier teeth were from Black Death victims.[10] Also in 2003, a team led by Alan Cooper from Oxford University tested 121 teeth from sixty-six skeletons found in 14th century mass graves, including well-documented Black Death plague pits in East Smithfield and Spitalfields. Their results showed no genetic evidence for Y. pestis, and Cooper concluded that though "[w]e cannot rule out Yersinia as the cause of the Black Death ...right now there is no molecular evidence for it.".[10][11][12] Other researchers argued that those burial sites where Y. pestis could not be found had nothing to do with the Black Death in the first place [13]
In October 2010 the journal PLoS Pathogens published a paper Haensch et al. (2010)[14] by a multinational team that investigated the role of Yersinia pestis in the Black Death. The paper detailed the results of new surveys that combined ancient DNA analyses and protein-specific detection which were used to find DNA and protein signatures specific for Y. pestis in human skeletons from widely distributed mass graves in northern, central and southern Europe that were associated archaeologically with the Black Death and subsequent resurgences. The authors concluded that this research, together with prior analyses from the south of France and Germany
- "...ends the debate about the etiology of the Black Death, and unambiguously demonstrates that Y. pestis was the causative agent of the epidemic plague that devastated Europe during the Middle Ages."
Significantly, the study also identified two previously unknown but related clades (genetic branches) of the Y. pestis genome that were associated with distinct medieval mass graves. These were found to be ancestral to modern isolates of the modern Y. pestis strains Orientalis and Medievalis, suggesting that these variant strains (which are now presumed to be extinct) may have entered Europe in two distinct waves.
The presence of Y. pestis during the Black Death and its phylogenetic placement was definitely established in 2011 with the publication of a Y. pestis genome using new amplification techniques used on DNA extracts from teeth from over 100 samples from the East Smithfield burial site in London.[15][16][17]
Surveys of plague pit remains in France and England indicate that the first variant entered Europe through the port of Marseilles around November 1347 and spread through France over the next two years, eventually reaching England in the spring of 1349, where it spread through the country in three successive epidemics. However, surveys of plague pit remains from the Netherlands town of Bergen op Zoom showed that the Y. pestis genotype responsible for the pandemic that spread through the Low Countries from 1350 differed from that found in Britain and France, implying that Bergen op Zoom (and possibly other parts of the southern Netherlands) was not directly infected from England or France in AD 1349, suggesting that a second wave of plague infection, distinct from those in Britain and France, may have been carried to the Low Countries from Norway, the Hanseatic cities, or another site.[14]
Alternative explanations[edit]
Evidence against Y. pestis[edit]
Although Y. pestis as the causitive agent of plague is widely accepted, recent scientific and historical investigations have led some researchers to doubt the long-held belief that the Black Death was an epidemic of bubonic plague.
While Y. pestis was in some samples collected from grave sites in various burial sites across Europe, many skeletons did not yield the DNA. The fact that bubonic plague DNA was present does not necessarily mean that bubonic plague was the cause of death; the individual could possibly have survived a bout of bubonic plague and died of another disease agent. Y. pestis might well have been the cause of death, but since archaeologists have often specifically sought evidence of Y. pestis, other important information is potentially neglected. While rats were present in major sea ports across Europe, the evidence for rats in rural communities in Northern Europe is scanty. The fact that the epidemiology of the Black Death and its rapid spread does not match modern bubonic plague is an indication that all possibilities should be considered, even in the case that the epidemic was caused by bubonic plague [18]
In 1984, Graham Twigg published The Black Death: A Biological Reappraisal, where he argued that the climate and ecology of Europe and particularly England made it nearly impossible for rats and fleas to have transmitted bubonic plague. Combining information on the biology of Rattus rattus, Rattus norvegicus, and the common fleas Xenopsylla cheopis and Pulex irritans with modern studies of plague epidemiology, particularly in India, where the R. rattus is a native species and conditions are nearly ideal for plague to be spread, Twigg concludes that it would have been nearly impossible for Yersinia pestis to have been the causative agent of the plague, let alone its explosive spread across Europe. Twigg also shows that the common theory of entirely pneumonic spread does not hold up. He proposes, based on a reexamination of the evidence and symptoms, that the Black Death may actually have been an epidemic of pulmonary anthrax caused by Bacillus anthracis.
In 2002, Samuel K. Cohn published the controversial article, "The Black Death: End of the Paradigm".[19] In the article Cohn argues that the medieval and modern plagues were two distinct diseases differing in their symptoms, signs, and epidemiologies.[20] Cohn asserts that the agent causing the bubonic plague, Yersinia pestis, "was first cultured at Hong Kong in 1894." In turn, the medieval plague that struck Europe, according to Cohn, was not the bubonic plague carried by fleas on rats as traditionally viewed by scientists and historians alike.[21]
Cohn's argument that medieval plague was not rat-based is supported by his claims that the modern and medieval plagues occurred in different seasons (a claim supported in a 2009 article by Mark Welford and Brian Bossak[22]), had unparalleled cycles of recurrence, and varied in the manner in which immunity was acquired. The modern plague reaches its peak in seasons with high humidity and a temperature of between 50 °F (10 °C) and 78 °F (26 °C), as rats' fleas thrive in this climate.[23] In comparison, the Black Death is recorded as occurring in periods during which rats' fleas could not have survived, i.e. hot Mediterranean summers above 78 °F (26 °C).[21] In terms of recurrence, the Black Death on average did not resurface in an area for between five and fifteen years after it had occurred.[24] In contrast, modern plagues often recur in a given area yearly for an average of eight to forty years. Last, Cohn presents evidence displaying that individuals gained immunity to the Black Death, unlike the modern plague, during the fourteenth century. He states that in 1348, two-thirds of those suffering from plague died, in comparison to one-twentieth by 1382.[21] Statistics display that immunity to the modern plague has not been acquired in modern times.
In the Encyclopedia of Population, he points to five major weaknesses in this theory:
- very different transmission speeds — the Black Death was reported to have spread 385 km in 91 days (4.23 km/day) in 664, compared to 12–15 km a year for the modern bubonic plague, with the assistance of trains and cars
- difficulties with the attempt to explain the rapid spread of the Black Death by arguing that it was spread by the rare pneumonic form of the disease — in fact this form killed less than 0.3% of the infected population in its worst outbreak (Manchuria in 1911)
- different seasonality — the modern plague can only be sustained at temperatures between 10 and 26 °C and requires high humidity, while the Black Death occurred even in Norway in the middle of the winter and in the Mediterranean in the middle of hot dry summers
- very different death rates — in several places (including Florence in 1348) over 75% of the population appears to have died; in contrast the highest mortality for the modern bubonic plague was 3% in Bombay in 1903
- the cycles and trends of infection were very different between the diseases — humans did not develop resistance to the modern disease, but resistance to the Black Death rose sharply, so that eventually it became mainly a childhood disease
Cohn also points out that while the identification of the disease as having buboes relies on accounts of Boccaccio and others, they described buboes, abscesses, rashes and carbuncles occurring all over the body, the neck or behind the ears. In contrast, the modern disease rarely has more than one bubo, most commonly in the groin, and is not characterised by abscesses, rashes and carbuncles.[25] This difference, he argues, ties in with the fact that fleas caused the modern plague and not the Black Death. Since flea bites do not usually reach beyond a person's ankles, in the modern period the groin was the nearest lymph node that could be infected. As the neck and the armpit were often infected during the medieval plague, it appears less likely that these infections were caused by fleas on rats.[26]
Ebola-like virus[edit]
In 2001, Susan Scott and Christopher Duncan, respectively a demographer and zoologist from Liverpool University, proposed the theory that the Black Death might have been caused by an Ebola-like virus, not a bacterium. Their rationale was that this plague spread much faster and the incubation period was much longer than other confirmed Y. pestis–caused plagues. A longer period of incubation will allow carriers of the infection to travel farther and infect more people than a shorter one. When the primary vector is humans, as opposed to birds, this is of great importance. Epidemiological studies suggest the disease was transferred between humans (which happens rarely with Yersinia pestis and very rarely for Bacillus anthracis), and some genes that determine immunity to Ebola-like viruses are much more widespread in Europe than in other parts of the world. Their research and findings are thoroughly documented in Biology of Plagues.[27] More recently the researchers have published computer modeling[28] demonstrating how the Black Death has made around 10% of Europeans resistant to HIV.
In a similar vein, historian Norman Cantor, in In the Wake of the Plague: The Black Death and the World It Made (2001), suggests the Black Death might have been a combination of pandemics including a form of anthrax, a cattle murrain. He cites many forms of evidence including: reported disease symptoms not in keeping with the known effects of either bubonic or pneumonic plague, the discovery of anthrax spores in a plague pit in Scotland, and the fact that meat from infected cattle was known to have been sold in many rural English areas prior to the onset of the plague. The means of infection varied widely, with infection in the absence of living or recently dead humans in Sicily (which speaks against most viruses). Also, diseases with similar symptoms were generally not distinguished between in that period (see murrain above), at least not in the Christian world; Chinese and Muslim medical records can be expected to yield better information which however only pertains to the specific disease(s) which affected these areas.
Cutaneous anthrax infection in humans shows up as a boil-like skin lesion that eventually forms an ulcer with a black center (eschar), often beginning as an irritating and itchy skin lesion or blister that is dark and usually concentrated as a black dot. Cutaneous infections generally form within the site of spore penetration between two and five days after exposure. Without treatment about 20% of cutaneous skin infection cases progress to toxemia and death.[29] Respiratory infection in humans initially presents with cold or flu-like symptoms for several days, followed by severe (and often fatal) respiratory collapse. Historical mortality was 92%.[30] Gastrointestinal infection in humans is most often caused by eating anthrax-infected meat and is characterized by serious gastrointestinal difficulty, vomiting of blood, severe diarrhea, acute inflammation of the intestinal tract, and loss of appetite. After the bacteria invades the bowel system, it spreads through the bloodstream throughout the body, making more toxins on the way.[29]
Historians who believe that the Black Death was indeed caused by bubonic plague have put forth several counterarguments.
The uncharacteristically rapid spread of the plague could be due to respiratory droplet transmission, and low levels of immunity in the European population at that period. Historical examples of pandemics of other diseases in populations without previous exposure, such as smallpox and tuberculosis transmitted by aerosol amongst Native Americans, show that the first instance of an epidemic spreads faster and is far more virulent than later instances among the descendants of survivors, for whom natural selection has produced characteristics that are protective against the disease.
A 2012 report from the University of Bergen acknowledges that Y. pestis could have been the cause of the pandemic, but states that the epidemiology of the disease is different, most importantly the rapid spread and the lack of rats in Scandinavia and other parts of Northern Europe. R. rattus was present in Scandinavian cities and ports at the time of the Black Death but was not found in small, inland villages. Based on archaeological evidence from digs all over Norway, the black rat population was present in sea ports but remained static in the cold climate and would only have been sustained if ships continually brought black rats and that the rats would be unlikely to venture across open ground to remote villages. It argues that while healthy black rats are rarely seen, rats suffering from bubonic plague behave differently from healthy rats; where accounts from warmer climates mention rats falling from roofs and walls and piling high in the streets, Samuel Pepys, who described trifling observations and events of the London plague of 1665 in great detail, makes no mention of sick or dead rats, nor does Absalon Pederssøn in his diary, which contains detailed descriptions of a plague epidemic in Bergen in 1565. Ultimately, Hufthammer and Walløe offer the possibility of human fleas and lice in place of rats.[31]
University of Oslo researchers concluded that Y. pestis was likely carried over the Silk Road via fleas on giant gerbils from Central Asia during intermittent warm spells.[32][33]
Michael McCormick, a historian offering the idea that bubonic plague was indeed the source of the Black Death, explains how archaeological research has confirmed that the black or "ship" rat was indeed present in Roman and medieval Europe. Also, the DNA of Y. pestis has been identified in the teeth of the human victims, the same DNA which has been widely believed to have come from the infected rodents.[34] He does not deny the point that there exists a pneumonic expression of Y. pestis transmitted by human-to-human contact, but he states that this does not spread as easily as previous historians have imagined. The rat, according to him, is the only plausible agent of transmission that could have led to such a wide and quick spread of the plague. This is because of rats' proclivity to associate with humans and the ability of their blood to withstand very large concentrations of the bacillus.[35] When rats died, their fleas (which were infected with bacterial blood) found new hosts in the form of humans and animals. The Black Death tapered off in the eighteenth century, and according to McCormick, a rat-based theory of transmission could explain why this occurred. The plague(s) had killed a large portion of the human host population of Europe and dwindling cities meant that more people were isolated, and so geography and demography did not allow rats to have as much contact with Europeans. Greatly curtailed communication and transportation systems due to the drastic decline in human population also hindered the replenishment of devastated rat colonies.[36][clarification needed]
- ^ "Plague Backgrounder". Avma.org. Archived from the original on 2008-05-16. Retrieved 2008-11-03.
- ^ Drancourt,, M.; Houhamdi, L; Raoult, D. (April 2006). "Yersinia pestis as a telluric, human ectoparasite-borne organism". The Lancet Infectious Diseases. The Lancet. 6 (4): 234–41. doi:10.1016/S1473-3099(06)70438-8. PMID 16554248.
- ^ Webb,, Colleen T.; Christopher P. Brooks; K. L. Gage; Michael F. Antolin (7 April 2006). "Classic flea-borne transmission does not drive plague epizootics in prairie dogs" (PDF). Infectious Diseases. Proceedings of the National Academy of Sciences. Retrieved 2006-12-12.
- ^ Appleby, Andrew B (1980). "The Disappearance of the Plague: A Continuing Puzzle". Economic History Review. 33 (2): 161–173. doi:10.2307/2595837. PMID 11614424.
- ^ Slack, Paul (1981). "The Disappearance of the Plague: An Alternative View". Economic History Review. 34 (3): 469–476. doi:10.1111/j.1468-0289.1981.tb02081.x.
- ^ Rebecca Totaro, Suffering in Paradise: The Bubonic Plague in English Literature from More to Milton (Pittsburgh: Duquesne University Press: 2005), p. 26.
- ^ Herlihy, The Black Death and the Transformation of the West (1997) Harvard University Press: Cambridge, MA, p. 29.
- ^ Raoult, Didier; Aboudharam, Gérard; Crubézy, Eric; Larrouy, Georges; Ludes, Bertrand; Drancourt, Michel (7 November 2000). "Molecular identification by suicide PCR of Yersinia pestis as the agent of Medieval Black Death" (PDF). Proceedings of the National Academy of Sciences. 97 (23): 12800–12803. Bibcode:2000PNAS...9712800R. doi:10.1073/pnas.220225197. PMC 18844. PMID 11058154. Retrieved 2008-12-12.
- ^ Drancourt, Michel; Signoli, Michel; Dang, La Vu; Bizot, Bruno; Roux, Veronique; Tzortzis, Stefan; Raoult, Didier (February 2007). "Yersinia pestis Orientalis in remains of ancient plague patients". Emerging Infectious Diseases. 13 (2): 332–333. doi:10.3201/eid1302.060197. PMC 2725862. PMID 17479906.
- ^ a b MacKenzie, Debora (11 September 2003). "Case reopens on Black Death cause". New Scientist.
- ^ Gilbert, M. Thomas P.; et al. Absence of Y. pestis-specific DNA in human teeth from European excavations of putative plague victims (PDF). Society for General Microbiology – 153rd Meeting.
- ^ "Jury out on Black Death culprit". BBC News. 10 September 2003. Retrieved 2008-12-12.
- ^ Lavigne, F; Degeai, JP; Komorowski, JC; Guillet, S; Robert, V; Lahitte, P; Oppenheimer, C; Stoffel, M; Vidal, CM; Surono, Pratomo I; Wassmer, P; Hajdas, I; Hadmoko, DS; de Belizal, E (2013). "Source of the great A.D. 1257 mystery eruption unveiled, Samalas volcano, Rinjani Volcanic Complex, Indonesia". Proc. Natl. Acad. Sci. U.S.A. 110: 16742–7. Bibcode:2013PNAS..11016742L. doi:10.1073/pnas.1307520110. PMC 3801080. PMID 24082132.
- ^ a b Haensch, Stephanie; Bianucci, Raffaella; Signoli, Michel; Rajerison, Minoarisoa; Schultz, Michael; Kacki, Sacha; Vermunt, Marco; Weston, Darlene A.; Hurst, Derek; Achtman, Mark; Carniel, Elisabeth; Bramanti, Barbara (September 2010). Besansky, Nora J, ed. "Distinct Clones of Yersinia pestis Caused the Black Death". PLoS Pathogens. 6 (10): e1001134. doi:10.1371/journal.ppat.1001134. PMC 2951374. PMID 20949072. Retrieved 2010-11-16.
We confirm that Y. pestis caused the Black Death and later epidemics on the entire European continent over the course of four centuries. Furthermore, on the basis of 17 single nucleotide polymorphisms plus the absence of a deletion in glpD gene, our aDNA results identified two previously unknown but related clades of Y. pestis associated with distinct medieval mass graves. These findings suggest that plague was imported to Europe on two or more occasions, each following a distinct route. These two clades are ancestral to modern isolates of Y. pestis biovars Orientalis and Medievalis. Our results clarify the etiology of the Black Death and provide a paradigm for a detailed historical reconstruction of the infection routes followed by this disease.
- ^ McGrath, Matt (12 October 2011). "Black Death Genetic Code 'Built'". BBC World Service. Retrieved 12 October 2011.
- ^ Bos, Kirsten; Schuenemann, Verena J.; Golding, G. Brian; Burbano, Hernán A.; Waglechner, Nicholas; Coombes, Brian K.; McPhee, Joseph B.; Dewitte, Sharon N.; Meyer, Matthias; Schmedes, Sarah; Wood, James; Earn, David J. D.; Herring, D. Ann; Bauer, Peter; Poinar, Hendrik N.; Krause, Johannes (12 October 2011). "A draft genome of Yersinia pestis from victims of the Black Death". Nature. 478 (7370): 506–510. Bibcode:2011Natur.478..506B. doi:10.1038/nature10549. PMC 3690193. PMID 21993626.
- ^ Schuenemann, V.; Bos, K. (2011). DeWitte, S., Schmedes, S., Jamieson, J., Mittnik, A., Forrest, S., Coombes, B., Wood, J., Earn, D., White, W., Krause, J., & Poinar, H. "PNAS Plus: Targeted enrichment of ancient pathogens yielding the pPCP1 plasmid of Yersinia pestis from victims of the Black Death". Proceedings of the National Academy of Sciences. 108: E746–E752. Bibcode:2011PNAS..108E.746S. doi:10.1073/pnas.1105107108. PMC 3179067. PMID 21876176.
- ^ July 22, 2014. The Black Death: Where We Are Now
- ^ Cohn, Samuel. K. (2002). "The Black Death: End of a Paradigm". The American Historical Review. 107 (3): 703–738. doi:10.1086/532493. PMID 12132537
- ^ Cohn 2002, p. 703
- ^ a b c Cohn 2002
- ^ Welford, M.; Bossak, B. (December 2009). Carter, Dee A, ed. "Validation of Inverse Seasonal Peak Mortality in Medieval Plagues, Including the Black Death, In Comparison to Modern Yersinia pestis-Variant Diseases". PLoS ONE. 4 (12): e8401. Bibcode:2009PLoSO...4.8401W. doi:10.1371/journal.pone.0008401. PMC 2791870. PMID 20027294.
- ^ Welford & Bossak 2009, p. 725
- ^ Welford & Bossak 2009, p. 727
- ^ "Black Death". Encyclopedia of Population. 1. Macmillan Reference. 2003. pp. 98–101. ISBN 0-02-865677-6.
- ^ Samuel K. Cohn, The Black Death Transformed: Disease and Culture in Early Renaissance Europe (London: Edward Arnold Publishers, 2002), 81.
- ^ Scott, Susan and Duncan, Christopher. (2004). Return of the Black Death: The World's Greatest Serial Killer West Sussex; John Wiley and Sons. ISBN 0-470-09000-6.
- ^ Duncan, S. R.; Scott, S.; Duncan, C. J. (March 2005). "Reappraisal of the historical selective pressures for the CCR5-Delta32 mutation". Journal of Medical Genetics. 42 (3): 205–208. doi:10.1136/jmg.2004.025346. PMC 1736018. PMID 15744032.
- ^ a b "Anthrax Q & A: Signs and Symptoms". Emergency Preparedness and Response. Centers for Disease Control and Prevention. 2003. Archived from the original on 2007-04-05. Retrieved 2007-04-19.
- ^ Bravata DM, Holty JE, Liu H, McDonald KM, Olshen RA, Owens DK (2006), Systematic review: a century of inhalational anthrax cases from 1900 to 2005, Annals of Internal Medicine; 144(4): 270–80.
- ^ Hufthammer, Anne Karin and Walløe, Lars. 2012. Rats cannot have been intermediate hosts for Yersinia pestis during medieval plague epidemics in Northern Europe. Journal of Archaeological Science.
- ^ Black Death plague now blamed on giant gerbils, not rats
- ^ Gerbils, Not Rats, May Have Caused Bubonic Plague, Study Finds
- ^ McCormick, Michael (2003). "Rats, Communications, and Plague: Toward an Ecological History". Journal of Interdisciplinary History. 34: 25. doi:10.1162/002219503322645439.
- ^ McCormick 2003, p. 2
- ^ McCormick 2003, p. 23


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WHAT can be achieved in 60 minutes? Drive from Worthing to Portsmouth, play a Chopin nocturne on the piano 12 times on a loop, or cook a slap-up meal, perhaps?
Sadly, one thing which I could not achieve in that time was getting through to Her Majesty's Revenue and Customs when phoning it this week.
My problems began at the start of my career, when I registered as self- employed before gaining my full-time job.
In many respects, those six months of freelance journalism were crucial in securing full-time employment at the Herald and Gazette series.
But for the hassle that short time has caused since, I would never go back to self-employment again.
And that is all because of my experience of HMRC.
One friend of mine told me they were kept waiting for 47 minutes and gave up. Can anyone beat the 'record' 60 minutes?Oli Poole, business reporter
In the early 2000s, historian and TV presenter Adam Hart-Davis fronted a series of advertisements for HMRC, with the catchy tagline 'tax doesn't have to be taxing'.
When the first advert aired, I was just 11 years of age and had no concept, or care, of what he was talking about.
But even now, having been through the system, I am still not convinced of the accuracy of those words.
For businesses across the Herald and Gazette area, dealing with HMRC will be a regular occurrence.
Business owners will be familiar with the self- assessment process and the importance of filling the forms in on time.
If you do not do so, you face a £100 fine, rising with interest the longer it is not paid.
Essentially, this is what happened to me – but having filed my accounts for the full period and 'ceased trading', I had no obligation to complete a self-assessment form again (to my great relief!).
Attempting to sort out this genuine error, I phoned HMRC, following the advice on the letter.
And after an hour of listening to its grating muzak, with no hint of an apology from the call handler for the length of my wait, he directed me to fill in a form.
Call centres are nearly always a nightmare across the board and HMRC is by no means alone in its long waiting times.
But with waiting times to it on the increase, according to the BBC earlier this year, it is clear I am not alone in experiencing issues.
One friend of mine told me they were kept waiting for 47 minutes and gave up. Can anyone beat the 'record' 60 minutes?


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The mission of Calvary Lutheran Church is to lead people into a growing relationship with Jesus Christ.
For the sake of this mission, by the power of the Holy Spirit we will:
Live under the Bible's authority.
Invite all people to experience the love of Jesus.
Unite in outward-focused community.
Equip God's people to serve.
Work with local and global partners in Christ.
Give generously within and beyond Calvary.
Father in Heaven, Matthew 6:9, Romans 8:15-17
Continue the work you have begun in Calvary: Philippians 1:3-6, Matthew 16:18
Making of us a 1 Peter 2:4-5, Isaiah 64:8
Jesus-pursuing Philippians 2:5-11, Mark 1:16-18
Scripture-breathing John 15:7-8, Colossians 3:16
Outward-focused Luke 9:23-24, 2 Corinthians 5:14-20
Kingdom-seeking Mark 1:14-15, Matthew 6:33
Community Acts 2:42-47, Romans 12:1-13
Caring for one another across generations 1 Timothy 5:1-2, Deuteronomy 6:1-9
Making disciples who make disciples Matthew 28:18-20, 2 Timothy 2:2
Freeing those who are held captive, and Isaiah 61:1, Matthew 10:1
Proclaiming the Good News of Jesus Christ Acts 1:8, John 20:21
Through our words and actions Ephesians 2:10, Ephesians 5:19-20
By the power of the Holy Spirit.John 7:37-39, Joel 2:28-32
In the Name of Jesus, Amen.John 15:16, Acts 4:12


text:
You are here
External Funding
This page provides examples of places to find financial support from external sponsors. The examples shared here include tools such as databases and campus resources.
Campus Resources that Help with Finding Funding
Research Guides U-Mich Library
The Research Guides provided by the U-M Library are an excellent place to start to find support in learning how to search for select opportunities. Work with a subject librarian to learn how to access the rich and complex databases and library resources the University has have to support researchers.
Funding Opportunity Databases
Searchable third party databases (like SPIN and Pivot/COS) mine funding opportunities so you don't have to, and they can send new opportunities to you each week by email. The University of Michigan subscribes to these excellent resources and with your U-M login credentials you can create accounts.
- Pivot is a ProQuest product (formerly known as COS - Community of Science).
Pivot is an excellent resource for finding funding opportunities.
You can use Pivot to save a search and have it send you a weekly email alert when a match between your choice of keywords (research area) and funding programs are matched in the Pivot funding opportunities database. Please also see the Pivot Quick Start Guide.
- The SPIN database is an infoEd product. InfoEd offers an alert service called SMARTS. SMARTS will send you e-mail messages when there is a match between your choice of keywords and the funding programs in the SPIN funding opportunities database. Anyone with a "umich.edu" e-mail address can use SMARTS. Sign up for SMARTS (or create/update a profile).
Once you request a SMARTS profile, your request will be routed to the University of Michigan GENIUS Administrator: Constance Colthorp Amrine - firstname.lastname@example.com so that she can review your request and validate your profile. (For the department name choose the U-M department with which you are affiliated. If you do not see it listed you may choose our name (the option reads with our former name "Vice President Research - Research & Sponsored Projects (ORSP)").
External Resources
Sponsor Websites
View a few external sponsors' websites including various foundations, NIH, NSF, DOE, Michigan agencies and others.
External Sponsor Email Alerts
Get notified of sources of support from Foundations, NIH, NSF, DOE, Michigan agencies and others through regular email alerts from external sponsors.


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text:
Procedure for reporting a child's absence or tardiness:
Please call 451-7207 by 8:30 a.m.
If your child is absent or tardy for whatever reason, please provide the following information:
Child's nameGradeHomeroomReason for absence or tardiness (Please be specific).
Students arriving after 10:20 a.m. will be counted 1/2 day absent. Students leaving before 11:00 a.m. will be counted absent.


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on November 23, 2016
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In the wake of the subprime lending crisis, a state mortgage-broker trade group is taking steps to shore up the profession's image and separate itself from unethical brokers who contribute to borrowers' mortgage woes.
The Maryland Association of Mortgage Brokers, which represents about 800 of the state's 10,000 licensed brokers and loan originators, said yesterday that it has adopted a code of ethics and professional standards that members must follow to retain their membership.
"We've been very concerned for a number of years that within the large universe of mortgage brokers, there are a number of bad apples," said Thomas Shaner, the association's executive director.


text:
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text:
- Learn C#, a powerful modern language.
- Develop a positive attitude to problem solving.
- Gain an excellent general knowledge of game creation.
- Learn how object oriented programming works in practice.
- Transfer your knowledge to .NET, other languages, and more.
Learn how to create video games using Unity 3D, the free-to-use game development tool. We start super simple so you need no prior experience or Unity of coding! With our online tutorials, you'll be amazed what you can achieve.
Benefit from our world-class support from both other students, and Ben is on the forums regularly. Go on to build several games including a full 3D version of Pong with an online multiplayer scoreboard, and more.
You will have access to a course forum where you can discuss topics on a course-wide basis, or down to the individual video. Our thriving discussion forum will help you learn and share ideas with other students. Check out our reviews to see how people love this feature.
Unity 5: The first part of the course is taught in Unity 4. 6, as this is well established and stable. When you get to Bowlmaster we upgrade to Unity 5, and start using it's great new features. You can use Unity 5 from the start if you wish, with only minor code differences.
The course is project-based, so you will not just be learning dry programming concepts, but applying them immediately to real indie games as you go. All the project files will be included, as well as additional references and resources - you'll never get stuck. There are talking-head videos, powerful diagrams, quality screencasts and more.
Oh, and it's just bigger and better than the competition. See the length and the reviews.
For each demo game you build you will follow this process...
- Be challenged to build the entire game yourself.
- Be shown step-by step how to build it.
- Be challenged to apply, and re-apply your knowledge regularly.
You will get full lifetime access for a single one-off fee. The creators are qualified and experienced coders and avid gamers, so are able to explain complex concepts clearly, as well as entertain along the way. Most courses teach scripting using only C#.
You will learn C#, including Test Driven Development, a highly valuable skill. By the end of the course you'll be very confident in the basics of coding and game development, and hungry to learn more.


text:
Journal of Tropical Medicine
Volume 2011, Article ID 690286, 6 pages
Specific Viruses Detected in Nigerian Children in Association with Acute Respiratory Disease
1Viral Infections Unit, Department of Vaccination and Immune Protection, National Institute for Health and Welfare (THL), Helsinki, Finland
2Intestinal Viruses Unit, Department of Infectious Disease Surveillance and Control, National Institute for Health and Welfare (THL), Helsinki, Finland
3Department of Medical Microbiology and Parasitology, Obafemi Awolowo College of Health Science, Olabisi Onabanjo University, Shagamu, Ogun State, Nigeria
4Department of Virology, College of Medicine, University of Ibadan, Ibadan, Oyo State, Nigeria
Received 23 March 2011; Revised 14 May 2011; Accepted 6 July 2011
Academic Editor: Lawrence Okoror
Copyright © 2011 Oluwabukola M. Akinloye et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
How to Cite this Article
Oluwabukola M. Akinloye, Esa Rönkkö, Carita Savolainen-Kopra, et al., "Specific Viruses Detected in Nigerian Children in Association with Acute Respiratory Disease," Journal of Tropical Medicine, vol. 2011, Article ID 690286, 6 pages, 2011. https://doi.org/10.1155/2011/690286.


text:
Lynnwood, Co-Moderator: Lupus ForumSLE('00), Sjogren's Syndrome, SAD, Depression, Herpes Simplex 1Piroxicam, Plaquenil, Cellcept, Prednisone, Trazodone, Fosamax, Wellbrutrin SR, ValtrexLinks: DIAGNOSING LUPUS(4of11), LUPUS INFORMATION, LUPUS RESOURCES, Donate to HealingWell, Drug Interactions
Yes Vertigo does get worse with movment. Have you ever had an MRI done on your brain? I know a small number of people do get vertigo simply by having Lupus. Because Lupus can attack your brain anywhere it wants to.
As far as the plane goes of course every one is different but I have flown with ear infections and had no problem. You might try taking a decongestant and bring some cotton ball with you just in case. You have been mentioning this problem with vertigo for a while now. I would almost bet the farm it's not an inner ear problem and more a lupus brain/vertigo problem. Have you checked you meds to see if vertigo is a side effect of any of them? I don't know how you are fixed with insurance but if all possible you really need a brain scan especially if it's starting to effect your ability to drive.
In His Grip
AlwaysRosie "We can't control the waves, but we can learn how to surf!!"
Psalms 139
Co-Moderator - Lupus Forum
UCTD, Hashimotos, Inflammatory Bowel, Inflammatory Arthritis
Clickable Links: Lupus Resources Lupous.Org Lupus Criteria (4 of 11) Lupus Chapter Locator


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Anti World's Devin will dropped off a new 6 song EP called 'Labyrinth.' The new project from the Maryland rapper is produced by Shark with features Jimmy V and Leon'sWOLF.
You can stream 'Labyrinth' via Soundcloud below.
Way back in May 2017 videographer Max Beck linked up with Lil Peep in Berkeley, California to shoot the "Cry Alone"… Read more ›
After inking a fresh deal with Motown Records, Matt Ox returns with a new Soundcloud upload. The 14 year old rapper… Read more ›
6 Dogs blesses his fans today with a brand new track. On "Lifes Hard" the Georgia rapper links up with Blacky… Read more ›
Omen the 13th links up with Grigoryan the 1st for a brand new track. The Los Angeles rapper goes crazy over the… Read more ›
Casey Frey is one of the most uniquely talented artists I've ever met. You might know him from his viral… Read more ›
Ghostemane and crew ventured all the way to the middle of nowhere Minnesota, inviting 50 of his biggest fans, to… Read more ›


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CMSgt. Mark R. Stanton, USAF, Ret., 88, of New Baden died Dec. 1, 2017, at Friendship Manor in Nashville.
He was born June 4, 1929, in Pinckneyville, the son of Mark R. and Mabel, nee Ballheimer, Stanton. He married Norma Jean Daffron Oct. 22, 1949, in Mayfield, Ky., and she preceded him in death Jan. 10, 2003.
He is survived by a son, Mark R. (Lisa) Stanton III of Tamaroa; a daughter, Lori Fearing of Centralia; a son-in-law, Eric Gentz of New Baden; seven grandchildren, Mark R. (Haley) Stanton IV, Travis Stanton, Eric L. (Kayla) Gentz II, Erin (Cameron) Pettus, Melissa Wessel, Melinda Wessel and Dale Wessel Jr.; 11 great-grandchildren; one great-great-grandchild; three brothers, Richard Stanton and Robert (Janice) Stanton all of Granite City, and Roger (Kathy) Stanton of Robbins AFB, Ga.; and a sister-in-law, JoAnn Stanton of Granite City.
In addition to his parents and wife, he was preceded in death by a daughter, Tammy Gentz; a sister, Mabel Stanton; a brother, John Stanton; and brothers-in-law and sisters-in-law Tom C. Daffron, John Daffron, Ruth Sawyer, Ida Mae Rich, and Margie Stanton.
Following his retirement from the Air Force, Mr. Stanton worked for FKG Oil Co (MotoMart) and then was a manager at the Scott Credit Union Belleville branch. He was a member of First Baptist Church and American Legion Post 321 in New Baden. Mark loved to fish, hunt and spend time with his grandkids and great-grandkids.
Funeral services will be held at 11 a.m. Tuesday, Dec. 5, 2017, at Nordike Funeral Home in New Baden with Pastor Dave Manley officiating. Burial with full military honors will follow at Mueller Hill Cemetery in Pinckneyville.
Friends may call from 8 a.m. until time of service Tuesday, Dec. 5, 2017, at Nordike Funeral Home in New Baden.
Memorials may be made to Daffron Presswood Post 2504, Friendship Manor or American Legion Post 321. They will be received at the funeral home. Condolences may be sent to the family at nordikefuneralhome.com.


text: Emmanuelle Duez, founder of The Boson Project and WoMen'Up, gives a talk during the 4th edition of the Positive Economy Forum in le Havre. Emmanuelle Duez Founder, The Boson Project & Founder and Chairman, WoMen'Up BACKGROUND/ Academic and professional background Graduate in Law and of Sciences Po Paris, ESSEC Business School and Bocconi Universita. Has created WoMen'Up in 2011 and The Boson Project in 2013. Lieutenant, French National Marine since 2014. ACHIEVEMENT/ What is your project, initiative, start-up, company all about? Read more... www.positiveplanet.ngo AboutManifesto Our Publications Talks EventsLHFORUM/Positive Economy Forum 2015 Positive Economy Forum San Patrignano Positive Economy Forum Rostov-on-Don Positive Labs PartnersPositive Economy Forum partners Our Ecosystem


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Cloth diapers are soft and gentle against your baby's delicate skin. The breathability of a cotton diaper does wonders for the sensitive skin of babies. Instinctively, we choose only the softest clothing and blankets for our babies; cloth diapers are the same- they are the softest in diapering options. They just feel good to the touch!
Cloth diapers rely on the natural absorbency of cotton and are free of the many chemicals contained in disposable diapers. There are many studies on the effects of prolonged exposure to the chemicals in disposable diapers.
Cloth diapers are the epitome of 'green' because they are used again and again. Instead of entering a landfill after every use, they become great cleaning rags after they're done being great diapers. Most people want to make environmentally healthy and sustainable choices; with a diaper service, it's easy to be green!
Whether you wash your own diapers or use a service, cloth diapers are usually loads cheaper than disposables. And when you consider that lots of cloth diapering moms report faster potty training, the savings are multiplied.


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Based on responses of nearly 7,000 faculty members from 78 institutions, this COACHE report gives some additional clues and considerations one might entertain when job hunting for faculty positions.
COACHE, is a Harvard based collaboration of colleges and universities committed to gathering the peer diagnostic and comparative data academic administrators need to recruit, retain, and develop the cohort most critical to the long-term future of their institutions.
The core element of COACHE is an electronic survey designed and tested in focus groups and a rigorous pilot study with twelve sites. The COACHE Tenure-Track Job Satisfaction Survey provides participating institutions with a management tool that can be used as a powerful lever to improve the quality of work life for junior faculty.
I suspect you could determine, in advance, if an institution of interest to you is a participant - and ask for their findings and details of responses to the findings.


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George Lucas could be nearing the end of his primary task in preparing for the upcoming live-action Star Wars television series. The producer of the most popular science-fiction franchise in history has spent the last eight or nine months developing a story arc &#Array; spanning at least 100 episodes &#Array; for the live-action sequel that takes place after the events of Episode III: Revenge of the Sith.
According to IESB, Lucasfilm is now shopping around for writers to develop the actual episode screenplays. The studio is taking the task seriously, and may be looking to hire the talent behind some of today's most popular, well-written genre series, namely Battlestar Galactica, Lost, and Heroes. This certainly makes for good news; while the Star Wars theme will drum up huge interest initially, tightly written plots and dialog with realistic characters will be what keep audiences tuning in each week.
The report continues to suggest that each episode will cost between 2 and 4 million dollars to produce. The series as a whole will cater to teens and adults rather than to children.
It's expected that the series &#Array; still lacking a moniker other than Star Wars &#Array; will go into production next year and begin airing in 2009. Stay tuned for more news!


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Soaped oak w/black leather
The Shell Chair developed out of Michael Dreebens ongoing interest in traditional craft practices in North India - in this case saddle making. Exquisitely made saddles have been produced in India for centuries, and although some horse culture persists, it's waning as India becomes more technologically developed. The Shell Chairs attempt to incentivize this skilled trade by providing these artisans with a new market for their craft, and hopefully encouraging them to pass it on to younger generations. The shape of the chair is an homage to Scandinavian design of the mid-twentieth century and embraces some of its core values: it's clean of line and is intended to emphasize the leather forming and stitching, it's deeply comfortable, and it's durable – like the saddles it's derived from, it's crafted from thick, top-grain leather, and with reasonable care, will last for decades. Frame: black stained beechwood, dark stained oak, soaped oak and sirka grey oak. Chair top: premium brown saddle leather and black leather. Material: frame in beechwood or oak with top in premium brown saddle leather or black leather Dimensions: 66 cm or 74 cm Origin: handmade in Jaipur, India
For every one of the million of products we use to improve the quality of our lives, there are associated environmental, ethical and social consequences.


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Stupid rapture.
I hate to whine. But with the end of the world coming, I thought I could blow stuff off without consequence. I stopped paying bills. I told a few people to shove it (sorry about that, Mom) and did absolutely nothing by way of preparing for this week's column.
Seriously, is the world so out of control that you can't even take an 89-year-old millionaire Christian radio host at his word?
I was sure it was going to go down this time. I was sure because the whole world mocked the idea in one collected guffaw. "Oh, no!" two-thirds of you wrote in your Facebook status. "It's the rapture! I'm so scared!"
Funny people left shirts and ties scattered on their front lawns. One hilarious fellow left his pants and a pair of shoes on the floor beneath his toilet. Look, everybody! I ascended right off the can!
Your toilet joke here.
The rapture was the joke of the week. Even people who never joke about anything — those inexplicable folks who don't even think armpit noises are funny — found a way to get in on the mirth. Somebody, somewhere took the time to fill a dozen "inflatable companions" with helium and release them into the sky. Up they went, the entire population of the adult novelty store rising to their reward. Video of the glorious event hit YouTube and everybody laughed until their sides hurt.
Sheer hilarity. We are so confident about our place in the universe, we feel safe to openly ridicule dire prognostications like this one. And that, my yuck-yucking friends, is usually when bad things happen.
If life was a horror movie, Saturday would have been a bad day, indeed. The end would have come right in the middle of our rapture parties, in obeisance to every horror movie script written since the 1980s. Early evening Saturday is when the gorgeous teenage girl would have started tripping as she ran through the woods.
We're talking earthquakes, tidal waves and swarms of things that sting. The sinners among us would have watched from a world of fire as the righteous were raised up.
Which would have been cool, in its way, since the righteous apparently have to be naked to get where they're going.
But, no. Six p.m. came and 6:01 right after it. No sign of hellfire anywhere. No reward for the pious and punishment for … well, you. Saturday was just another rapture that failed to produce. Time to update your Facebook status to impart some version of "I told you so."
But I'd be willing to bet that plenty of perfectly normal people felt a stirring of relief when the end failed to come. They wouldn't admit as much aloud — What, me worry? — and to themselves, they could pass the feeling off as imagination or possibly, acid reflux.
But who could blame a person for worrying, if only in a vague way? Because, man, doesn't it feel like we're due for something big and bad? Haven't the signs been piling up to indicate something dark and dire is coming down the cosmic turnpike? I kind of lost count, but it seems to me we're pretty darn close to 40 straight days of rain.
Even ardent nonbelievers can get spooked if their inner Magic 8-ball quivers with signs of doom. If you don't believe in the Bible, maybe you believe in the Mayans. If you don't believe in the Mayans, maybe you believe in missiles. If you're human, you came with a sense of paranoia built right in.
The problem, of course, is that too many charlatans have fooled too many people for too damn long. With every Jim Jones, David Koresh or Harold Camping that comes and goes, we become more cynical, more sneering, more confident that we and we alone control our destiny. Yet predictions of doom still have the power to captivate us. The difference being that today, we flock to the World Wide Web instead of the church, to mock instead of to repent.
But, whatever; it's over. Rapture 2011 was good for a week of chatter, a few big parties and some absolutely hysterical sight gags. But bear in mind that Rapture 2011 was also small-time. It was one old man with a million dollars and a radio station instead of a sandwich board and a street corner.
You want to see a real end-of-the-world scare? Wait until next year. Wait until the solstice of 2012, and the big, black nothing that follows, a prediction not of one old crank with liver spots but an ancient civilization whose people, in their way, were far smarter than we are.
Of course, you will scoff every second of the way, your chest puffed out, your eyes laughing. And good for you, bad ass. I wouldn't want you to change your ways for anything. I just came to explain why I told you off last week and why I can't repay the money I owe you. I've got no dough left, you see, not a single dime.
A dozen inflatable companions don't come as cheap as you might think.
Mark LaFlamme is a Sun Journal staff writer. You can frighten him with your end-of-the-world prediction at [email protected]


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Oz Gets Bloody in Red Brick Road
Warner Horizon is banking on the continued popularity of public domain with its plans to develop a fantasy series that promises a darker take on L. Frank Baum's beloved Oz novels.
According to The Hollywood Reporter, Red Brick Road is billed as "a continuation of the Wizard of Oz story but told in a Game of Thrones fashion, filled with politics, intrigue and violence." However, details are being kept under wraps.
The project was created by Rob Prior, who's worked as a concept and comic book artist. Red Brick Road has Mark Wolper and Roy Lee (Bates Motel) signed on to executive produce along with Adrian Askarieh who has a film called Leonardo da Vinci and the Soldiers of Forever set up at Warner Bros. with Lee.
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2010 Green Choice Campaign
If you love our work then tell the world!
You have an exciting opportunity to help make even more of a difference in our community. GreatNonprofits – a site like Amazon reviews – is conducting a campaign to find the top-rated environmental nonprofits.
Won't you help us participate in the campaign by posting a review of your experience with us? All reviews will be visible to potential donors and volunteers. It's easy and only takes 3 minutes!
Go to:www.greatnonprofits.org/reviews/upper-valley-land-trust-inc
Be sure to choose "Green Choice Campaign" from the drop down menu of campaigns in the review template. With your help, we can gain greater visibility in the community.


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Six bottoms. Two teams. This time, it's personal. Awe-inspiring, pulse-poundingly delicious brand new update from SpankRed3D…..
Though shouldn't that read Team Cherry Red? 😉
Only one bun blazing team can rise to the top in this tail-tanning, cherry-cheeked spanking competition.
Will it be the HotBots or the SpankRed team?? Visit SpankRed3D to get instant access to tons of exquisite, original and wicked hot spanking illustrations and artwork! 🙂
Check out what's streaming on-demand at CherryRedMovies. New users? Watch 10 minutes for free Instantly.


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Hi Seas Grand Slam Mono Leader 200-600 LB – 100 Yd Coils – back
Hi Seas Grand Slam Monofilament Leader – HI Seas advanced monofilament leader with properties that make it supple, yet extra tough. One leader that can be used for bottom fishing to deep dropping or offshore trolling for large gamefish. Unsurpassed for tying rigs of any kind. Superior knot strengths, Strong abrasion resistance Low stretch High strength. HI Seas Monofilament Leader Skeins make onboard leader making fast and easy! Available in Clear.
Sold individually … Stock sizes (250 Lb) (300 Lb) (400 Lb) (500 Lb) (600 Lb) ( 100Yds – Clear)
Returns Policy
You may return most new, unopened items within 30 days of delivery for a full refund. We'll also pay the return shipping costs if the return is a result of our error (you received an incorrect or defective item, etc.).
You should expect to receive your refund within four weeks of giving your package to the return shipper, however, in many cases you will receive a refund more quickly. This time period includes the transit time for us to receive your return from the shipper (5 to 10 business days), the time it takes us to process your return once we receive it (3 to 5 business days), and the time it takes your bank to process our refund request (5 to 10 business days).
If you need to return an item, simply login to your account, view the order using the 'Complete Orders' link under the My Account menu and click the Return Item(s) button. We'll notify you via e-mail of your refund once we've received and processed the returned item.
We can ship to virtually any address in the world. Note that there are restrictions on some products, and some products cannot be shipped to international destinations.
When you place an order, we will estimate shipping and delivery dates for you based on the availability of your items and the shipping options you choose. Depending on the shipping provider you choose, shipping date estimates may appear on the shipping quotes page.
Please also note that the shipping rates for many items we sell are weight-based. The weight of any such item can be found on its detail page. To reflect the policies of the shipping companies we use, all weights will be rounded up to the next full pound.


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Last year when pre-ordering was open for the Apple iPhone 4S around 600,000 took part and registered early. After millions more have sold, the iPhone 4S remains to be one of the World's most popular devices. This is demonstrated each week in the charts of the Official Smartphone Rankings. Today we feature what Zach O'Brien has to say about it.
Perfection minus the glass back. By Zach O'Brien, posted: May 21
As a student, I always want the best phone. My friend got the iPhone 4, and I had to have it. At that time, I had the Droid X2, which happened to flop and Verizon replaced it with a free iPhone 4S. So I got what I wanted. The transition from Android to iOS was quite different. When I went to iOS, I had a flawless experience with a few minor issues here and there (what operating system doesn't have 'em). But it felt good that my phone never randomly shut off on me randomly unlike prior Droids. I've had experience with all types of Android overlays: HTC Sense 3.5, Touchwiz, and Motoblur (whatever they call it these days). The only gripe I have is the durability, I love the sleekness of the device, but hate that it is easily breakable. Did you think a HS student would know so much about tech news? Overall, a 4.6/5
Display 5/5
Battery Life 4/5
Apps & Media Support 5/5
Reception & Call Quality 5/5
Design/Form Factor 4/5
Overall 4.6


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Back in front of my computer after a long while I can finally ask you what you thought of the last event (10th of April with Will Guthrie, John Edwards, Phil Durrant and all that)? Please send me your comments and impressions.
Also, I'd love to see pictures if you've got some so I can feel a bit less frustrated from missing it.
Now, I should have -and give- a break for a bit and will be back with new projects in two months. The first one is a night at Cafe Oto in Dalston where David Ryan and I will present works addressing - somehow - the relationship between improvisation and composition.
The other project is even more indeterminate but will involve dance, just to let you know that the research of the begining of the year hasn't been dropped out and we're building up a group of people interested in spending time on the idea of collaboration between music and dance.
Hope you're all well and merry.
See you before too long.
Tuesday, 13 May 2008
1 comment:
- David Papapostolou said...
Salut Olivier,
ça m'intéresse ton projet danse/musique, il vous manque du monde dans le "camp musique"?
Mon email:
david "petit point" papapostolou "petit arobase " laposte "petit point" net
Encore merci pour le Haggis dimanche :)
See you bientot
- 20 May 2008 at 06:54
Subscribe to: Post Comments (Atom)


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Effect of Lightweight Aggregate on Early-Age Cracking of Mass Concrete Schindler and Tankasala, Auburn University
Early-age cracking in mass concrete structures is a severe problem that can lead to long-term serviceability problems. Researchers at Auburn University conducted a study to evaluate the effects of using lightweight aggregates on the early-age cracking tendency of mass concrete.
A considerable amount of heat is generated by the hydration of cementitious materials. As a result, there is a significant temperature differential between the interior and exterior of mass concrete elements during the early stages of construction. These temperature differences can lead to thermal cracking at an early age. Effective control of early-age cracking can result in more durable concrete and extended service life of concrete structures. This study investigated the effects of lightweight aggregates on the concrete properties that influence early-age cracking tendencies in mass concrete, compared to control mixtures containing only normal weight aggregates. Concretes with water-cementitious materials ratios of 0.38 and 0.45 were evaluated.
The results of the study indicate that using lightweight aggregates in mass concrete can improve its resistance to early-age cracking. Although increasing the amount of lightweight aggregate in the concrete mixture results in an increase in the maximum concrete temperature, this will also reduce the modulus of elasticity, reduce the coefficient of thermal expansion, and reduce or eliminate autogenous shrinkage effects, all of which contribute to increasing resistance to early-age cracking.Download Report
Key Findings
- Concrete containing increased amounts of lightweight aggregates experienced higher concrete temperatures compared to the control concrete.
- The presence of lightweight aggregates delayed the time to cracking.
- Increasing the amount of lightweight aggregate in the concrete results in a greater reduction in modulus of elasticity, coefficient of thermal expansion, and autogenous shrinkage. These effects all contribute to greater resistance to early-age cracking.


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You can now get the #Samsung #GalaxyS5 for just $239.99 thanks to a deal running from eBay. The smartphone is Samsung's flagship offering from 2014 and is a very capable offering even today, given the kind of hardware it's packing underneath, with my favorite being water resistance.
As is the nature of these deals, there's no telling as to how long it will last, so make sure you hop on board before the seller changes his/her mind. This could also make a perfect gifting item if you're not looking to get a smartphone for yourself.
To jog your memory a little bit, the Galaxy S5 comes with a 5.1-inch Full HD display, a quad-core 2.5 GHz Snapdragon 801 SoC, a 16-megapixel rear camera, a 2-megapixel front camera, 2GB of RAM, 16GB of storage (expandable), a 2,800 mAh battery and Android 5.0 Lollipop.
Interested? Head over to the link below for more details on the device as well as the deal.
Get the unlocked Samsung Galaxy S5 for just $239.99 from eBay!


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EECS associate professor Mehmet Fatih Yanik and members of his group the High-Throughput Neurotechnology Group in the Research Laboratory of Electronics (RLE) at MIT have reported the discovery of potential nerve regeneration compounds using the microchip technology they developed to carry out testing on live vertebrate specimens, in this case on tiny worms called C. elegans. Using the new technology, Yanik and his colleagues were able perform laser surgery, deliver drugs and image the resulting neuron regrowth in thousands of live animals. The technique and resulting identification of classes of potential compounds to treat spinal-cord damage as well as neurological diseases such as Alzheimer's or Parkinson's adds a new level of direct testing on tissue in live animals.
Yanik described this work to the MIT News Office saying, "Our technology helps researchers rapidly identify promising chemicals that can then be tested in mammals and perhaps even in humans. Using this technique, we have now identified one promising class of regeneration enhancers."
The paper, "Large-scale in vivo femtosecond laser neurosurgery screen reveals small-molecule enhancer of regeneration," now appears in the Oct. 11, 2010 online edition of the Proceedings of the National Academy of Sciences.
Lead authors of the paper are postdoctoral associate Chrysanthi Samara and EECS graduate students Christopher Rohde and Cody Gilleland, and collaborating chemists are Steve Haggarty and Stephanie Norton.
Development of the new technology and the regeneration screen was funded by the NIH Director's New Innovator Award Program, a Packard Fellowship in Science and Engineering, an Alfred Sloan Award in Neuroscience, an NSF Graduate Fellowship and a Merck Graduate Fellowship.
Read more:
Yanik Lab's high-throughput drug discovery technologies highlighted in The Economist, Nov. 29, 2010 "Testing drugs Mass-screening of drug candidates on tiny animals is now possible"
MIT News Office Oct. 12, 2010 article by Anne Trafton, "Nerve-cell regeneration quest is fast tracked. Microchip technology rapidly identifies compounds for regrowing nerves, in live animals."


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A cura di Giovanni Visco
Area 03 – Scienze chimicheTweet
SINTESIThe world is multivariate! In order to be significant, data coming from diagnostic on Environment as well as on Cultural Heritage must be multivariate. A cultural find born in the environment as row material, the last is subjected to degradation, deriving from the unavoidable interaction with the environment, handling by man to transfer a message and again alteration that, even if changes its initial face, at the same time tells its history and all those related to its contact with the environment and mankind; more, the analysis of the alteration products can guide to a correct Conservation procedure. Really, while all or some marks left by the past, depending on the mind line, could be maintained, new marks have to be avoided so, the present and future environmental conditions in which the artifact will be exposed must be carefully evaluated and eventually changed. Unfortunately the "Homo Sapiens" activity, directly by vandalic actions and indirectly through the environment pollution, negatively affects the conservation of Cultural Heritage artifacts.
Multivariate analysis and chemometry are integrated approaches necessary to the knowledge of the cultural item history, its constructive techniques, degradation, dating and origin (integrated approach to the knowledge). Really this is the zero number of our/your biannual Meeting dedicated to theorists, researchers, professors, experts, technicians, which use multivariate techniques in order to improve the knowledge of the environment and of the cultural artifacts. Every analytical technique/method is welcome if it is used in multivariate way in order to improve the knowledge of the cultural entity and of the living environment.
Principal Components 1 of the conference is to favour the meeting of researchers, experts, connoisseurs and competents on chemometry applied to Environment (environmetrics) and Cultural Heritage (CMA4CH) so I suggest all the participants to read careful presentation looking for cooperation in international projects.
Some researchers when I was doing publicity at the CMA4CH 2006 Meeting replied: «Very interesting but chemometry is not a topic research of mine!». But chemometry is today "under" any standard research, calibration, regression and data analysis, so, they can put some questions to themselves.What is chemometrics? Am I doing multivariate analysis? Am I using chemometry without knowing that I am using it? The index of the last book (Handbook of Chemometrics and Qualimetrics, Vol1 and Vol2) of the lamented Prof. D.L. Massart is often used as list, neither exhaustive, of what today the researchers intends for chemometrics. If you will find replies to your queries or the Meeting contacts will bring you some idea for new researches, we hope you will tell us them in the next edition on July 2008 (see at the end). (from Preface by Giovanni Visco)
Participant List: M. Abad, M. Anders, S. Atgin, P. Avino, A. Baklanov, P. Ballirano, M. Barazzutti, P. Barbieri, F. Bellanti, M. Bicchieri, M. Bobba, L. Bondioli, P. Bruno, R. Bucci, P. Busetto, M. Calderisi, R. Caminiti, L. Campanella, G. Capannesi, L.F. Capitan-Vallvey, G. Caracciolo, A. Carraro, G.R. Casale, M. Caselli, M. Castrucci, A. Cecchi, F. Cecere, R. Cesareo, R. Checa, M. Chr. Christensen, E. Ciceri, O. Colacicchi, M.P. Colombini, C. Coluzza, P.R. Dambruoso, B.E. Daresta, G. de Bruin, G. de Gennaro, M. de Vos, E. Delnevo, A.L. De Rosa, L. di Monte, M.I. Dias Portugal, G. Dogan, C. Dossi, P. Fermo, M. Ferretti, D. Ferro, E. Formigli, D. Frustace, N. Gabrielli, M.C. Gennaro, T. Georgiadis, G.E. Gigante, B. Giussani, E. Gregori, G. Guida, G. Güllü, P. Ielpo, A. Indaco, H. Khajehsharifi, R. Kirmaz, B. Knight, J. Kolar, M. Lasagni, M. Laurenzi Tabasso, D.A. Lichtblau, A. Livio, A. Macchia, A.L. Magrì, A.D. Magrì, A. Maiocchi, F. Malaspina, E. Manzano, E. Marengo, F. Marini, G. Martin, F. Modugno, D. Monticelli, M.F. Mousavi, M. Nardino, J. Palm, S. Palmieri, A. Panepinto, F.S. Panero, V. Paolillo, G. Pavese, G. Piantanida, C.M. Placentino, S. Plattner, P. Plossi, S. Polla, A. Pozzi, M.I. Prudêncio, R. Reale, E. Ribechini, S. Ridolfi, E. Robotti, L.R. Rodríguez-Simón, A. Rosada, D. Ruiu, F. Ruiz, C. Sadun, M.P. Sammartino, N. Selmani, A.M. Siani, H.E.B. Silva, M. Strlic, M. Tomassetti, T. Trafela, L. Trizio, G. Tuncel, G. Valentini, G. Vatankhah, G. Visco, E. Vuerich, A. Zararsiz, S. Zilitinkevich, T. Zoccarato.
|17 x 24
|data pubblicazione:
|Settembre 2006


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LMV98x-N are low-voltage, low-power operational amplifiers. LMV98x-N operate from 1.8-V
to 5-V supply voltages and have rail-to-rail input and output. LMV98x-N input common mode voltage
extends 200mV beyond the supplies which enables user enhanced functionality beyond the supply
voltage range. The output can swing rail-to-rail unloaded and within 105 mV from the rail with
600-Ω load at 1.8-V supply. LMV98x-N are optimized to work at 1.8 V, which makes them ideal for
portable two-cell battery powered systems and single cell Li-Ion systems.
LMV98x-N offer a shutdown pin that can be used to disable the device and reduce the
supply current. The device is in shutdown when the SHDN pin is low. The
output is high impedance in shutdown.
LMV98x-N exhibit excellent speed-power ratio, achieving 1.4-MHz gain bandwidth product at
1.8-V supply voltage with low supply current. LMV98x-N are capable of driving a 600-Ω load and up
to 1000-pF capacitive load with minimal ringing. LMV98x-N have a high DC gain of 101 dB, making
them suitable for low frequency applications.
LMV981-N is offered in space-saving, 6-pin DSBGA, SC70, and SOT-23 packages. The 6-pin DSBGA package
has only a 1.006 mm × 1.514 mm × 0.945 mm footprint. LMV982-N is offered in a space-saving, 10-pin VSSOP
package. These small packages are ideal solutions for area constrained PCBs and portable electronics such as
cellular phones and PDAs.


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It's been heartbreaking to see the images as a result of the recent hurricane. Or anytime you see pictures from a tropical storm, tornadoes or the massive destruction of earthquakes, it shakes us. It births a overwhelming feeling as the earth and all the familiar things in our lives are cut, ripped, moved, disappeared, destroyed, or worse yet just... lost. The devastation is stunning.
Living in Oklahoma seeing these kind of images are not new. We have had our share of disasters. But I must say...it's something I never get over. One question that pops loose when these catastrophic events happen is this one..."Where is God in the middle of the storm?"
He's right beside you.
We often question God as if these disasters have anything to do with His love for us.
Life weighs heavy on us, especially when we have to watch pain, loss and suffering and recovery. All we can do is click to make a donation...and don't feel like that it's enough. We even feel like our prayers aren't enough...matched with the amount of recovery that is needed.
How can you ever recover from that kind of force of nature wreaking havoc on your reality? In these instances...I am learning that just because our world is spinning out of control doesn't mean that God isn't still in control. Even when it looks like all will be lost and destroyed. These type of storms are certainly heartbreaking, but know that God sees and restores. He hasn't turned his back or rejected us. He hurts when we hurt. Help is on the way...and when the hand of compassion is extended...God works.
He speaks like into the broken places and brings healing through the hands of the firefighters, debri removers, the communities that pull together for a common cause. He brings miracles...too many to count. Do not be discouraged by the difficulties that seem overwhelming, impossible or when your circumstances are shouting over your common sense.
Now it's time to bring this home....what about your pain today?
God hasn't forgotten your pain, your loss, your hurts. He sees the destruction and the disasters. He sees your cracks and deep crevices that are filling with resentment and heartache. He knows and sees. He steps in and restores.
Ever notice with stories in the Bible that right before a miracle happens that something is broken. There is the feeding the 5000, Jesus lifted the pieces of bread to the heavens, broke the bread and then gave thanks?
When the wedding party was demanding more wine and the servants rushed to the back of the kitchen to get the best wine...there had to be some empty jars before there was a re-filling?
How about when Jesus when asked the tired and exhausted fishermen to cast their empty nets just once more on the other side of the boat? The disciples responded with groans...but obeyed. They were wrong about the sea being empty of fish that day.
God likes to work when nothing else will. God loves to work when things seem impossible. God does the impossible. God restores and brings miracles. We can take delight in that we can depend and totally trust Him for who He is.
God specializes in breaking so that only He can do the fixing.
And when things look overwhelming, destroyed, beyond repair, and always in a state of chaos, then...it's prime time for God to rescue.
"Jesus looked at them and said, "With man this is impossible,
but with God all things are possible." Matthew 19:26
So ask God to do the impossible in your current disaster.
Jesus met the disciples in the midst of their storm.
We can invite God into the middle of ours.
Jesus met the disciples in the midst of their storm.
We can invite God into the middle of ours.