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Hi all,
We have a group of around 15 sales staff that come into the office about once a month or so. Currently we provide them with ADSL in their home and a NetGear DG834 firewall. The ADSL should only be used for company business.
From the ADSL circuit, the sales staff connect their company owned laptop and VPN into our network.
I'm looking at other methods. I dislike VPN's due to the lack of security. I have no idea what the client device is or what state its in. Additionally it limits the end-users to just their laptop which isn't ideal.
We have just purchased a Juniper SA2500 SSL VPN device, which I want to implement as the remote access end-point for every remote connection we have. For example even Exchange OWA must go through this device.
The question is do we just continue with VPN's - albeit using SSL rather than PPTP? Issues like passwords, local document storage and management are something of an issue.
We also have a Citrix farm at our disposal. Used in our factory (for obvious reasons) it would also meet the needs of the sales team - but obviously only when they have an internet connection.
So this is my dilema - what do I do? Do we remove their laptops from the domain? Do we use full VPN tunnels still? Look at using Citrix? The tools at my disposal are:
- Outlook Web Access
- Juniper SA2500 SSL VPN device
- Citrix Presentation Server 4.5
- All sales staff have Windows XP (soon to be Vista) laptops
- All sales staff currently have company ADSL broadband at home
- All sales staff have 3g enabled Nokia or WM phones with unlimited data access
Any suggestions on the "optimium" remote access for these users by using the above tools would be great. The local HD's of the laptops will be encrypted - however there is obviously no backup at the moment other than when they come into the office they do a full sync.
They only really use ERP and Office at the moment. (Presentations, spreadsheets, email and our ERP Oracle app)
22 Replies
If you have a Citrix farm you could enable the WEB interface and use SSL to connect via browser. Works exactly like GoToMyPC and GoToMeeting which are two business units owned by Citrix and run on their technology.
This also gives you greater control of the application interface for your ERP and Office software so that you need only patch and update the server install.
Another benefit is tracking what the salespeople do to and with your data. Sales people have a tendacy to wander to competitors and this gives you a leg up on keeping your data with you rather than on the laptop you may not get back when they leave.
I currently have a General Contractor client and we run all their services this way so that we control the software standards, patches and upgrades and most of all, the data.
Thanks for the prompt reply.
Citrix could form a part of solution - but again it's dependant on the user have internet connectivity whenever and where ever they are working. The sales team are pretty demanding.... currently if the ADSL / local router goes down they can still work - just not get email / network drive. They can still access their documents as they are locally saved.
It's not a requirement at the moment (as I haven't put forward any suggestions to management), but by using Citrix it will obviously create a even bigger problem should the broadband go down.
I'm really after examples of how other people have remote access for their remote workers - is a single solution such as Citrix work? Is VPN the best solution? Are the remote machines part of the Windows domain? With our SSL VPN device - would just that be enough? (E.g. users can access file shares, apps and even a full Citrix desktop though just a web portal without any type of VPN)
Any more ideas very welcome!
The GC client I mentioned accesses the remote server from the field projects in job trailers using DSL lines much as you do. We dropped the VPN system due to speed issues on the various DSL circuits. As each DSL circuit is temporary based on the length of the project, it didn't make sense to contract for ADSL.
As a backup system, each Project Manager and Site Supervisor is equiped with a wireless modem from Verizon so that they can always connect to the server if the DSL is down or they are at a new project that doesn't have the circuit installed yet.
We also utilize remote sync to Exchange 2007 via their Motorola Q Smart phones so that they always have project updates and communications from everyone. In a pinch, they could run the browser interface to access the remote server, but the screen size and speed limitations makes that cumbersome.
We currently utilize Outlook WEB Access as well in the event they have Internet access while travelling and are testing Sharepoint services to make file sharing and access simpler as well.
If you must use VPN, then you may wish to look at a Cisco PIX system with client software so that you can connect on VPN via the wireless modems. I'm not sure what plans are like in your area, but my client pays a fixed $30 per month for each wireless modem with unlimited connectivity.
My personal belief is security over convenience, especially in this day and age of lost/stolen laptops and portable devices. My client would rather inconvenience a project manager than risk losing sesitive data. In your case, I imagine the sales people probably have sensitive client information, maybe even financial information, stored on the laptops and I'm not sure if you are already encrypting it or planning on encrypting it, but a lost encryption key can also lose a lot of data.
Thanks again for the quick reply!
Interesting stuff. Do your clients use terminals or a full-fat client? I love the idea of keeping all of the data at the head office with access via Citrix / OWA and push-email (plus possibly SharePoint. FYI - I've used SharePoint a lot and I strongly recommend it, but only on the basis that it will be highly used as it's not a cheap solution. But it is very good!)
I think the problem from our point of view is that the remote workers often do presentations. As such I can't expect connectivity all the time... So really I need to find a way for them to have key documents (e.g. presentations) stored locally in case the client the sales staff vist don't have public wifi or a internet PC nearby. This sort of takes me back to square one - which is a shame as I love the way you have implemented your remote system.
Thanks again.
I use a VPN over SSL solution (using a SonicWall Device) and terminal services and/or VNC, and to be honest it all works pretty well, but I do find things like sales presentations have to be stored locally on the laptop, because as you say, internet is not always available when required, but other than that it works for us
For those who require remote access, we install the Citrix client on their PC, and also iremote to create a sort of tunnel inwards. Our firewall is externally managed, and any remote access requests from a machine that's not been authorised (the user needs to login as a particular local user before they can successfully connect and login to Citrix). Once in, they can launch and run applications as if they were sitting right in the office - this runs well on a variety of PC's and laptops, using dialup, broadband (ADSL) where possible or AirCards (on company laptops).
Reva IT is an IT service provider.
i would look in to mobile thin clients, with builtin 3g card. as well as outlook mobile access on the phones.
or what we do, the user have external hdd at home which they sync to. and they sync with exchange & file servers when they connect to the network through are VPN using 3g or home broadband.
are sales team have hp 6710b laptops & we are looking into outlook mobile access at the moment.
we also use outlook web access.
We use Microsoft Terminal Server with RDP clients connecting to it. All remote users have verizon wireless broadband cards (either PCMCIA or USB) so they always have connection. All documents are stored on a file server, but they can be downloaded if required. Documents can be added and changed, but NOT deleted.
Since the remote users have dynamic IP's, we cannot limit access based on IP. We use fingerprint recognition on the remote laptop's or PC's and the users DO NOT know their passwords to any of the applications or logins. We setup the laptops or they bring in their desktops for setup. Fingerprint recognition software has provided us with security that we feel is appropriate for us.
This has worked well for us. We log everything we can and download the access logs into an in-house Alpha five database which then parses the logs and looks for abnormalities.
I don't know that there's a huge security difference b/t SSL/PPTP vs. Citrix vs. MS Terminal Svcs.
What might work well for you is to use Folder Redirects in GPO with Offline Files enabled (also in GPO). This lets their docs automatically sync every time they come in on secure VPN to a file server that you can then back up. But it also lets them "fly solo" on their files when their offline - the changes get synced again when they log back in, either IN the office our REMOTELY.
You can redirect/engage offline files on the Desktop, MyDocs, AppData and Start Menu.
This works for us perfectly.
Thanks for the replies everyone.
Don't think file-rediection will work as the links between the office and the remote workers will be 3G or 2Mbps ADSL at best. Whilst normally you could just get away with it these users have 50mb presentations files they work with! :-)
I'm planning on using the SA 2500 for Outlook webmail and web-proxy as well. This enables us to force all users to use the companies proxy server when using a company laptop - which should keep compliance and HR happy.
I love the idea of Citrix still - to the point that users can do their work online. I suppose we could "suggest" that they then mail or use the SA2500 to transfer the file/presentation to their workstation if they need to do a presentation at a clients site.
I have had quite a bit of experience with secure data and the troubles with information flow in the field. So I have a couple questions:
What data do you want them to have 100% access to?
How sensitive is the data?
Couple thoughts for you:
Citrix is nice but it does not allow offline capability, I dont like PPTP tunnels as they are error prone.
The best thing I have found is an SSL vpn though fortigate that I can serve up samba shares and terminal services through a single web page. For offline files I allow the users only to synch necessary files for offfline, and those are encrypted. That saves them in the situation where they need a presentation or some data but have no connection. It also helps me to sleep at night knowing that they are encrypted rather than just on their desktop.
Hi Adam, Thanks for the reply.
100% regarding the avaliability? Really it's going to be emails and presentations. The emails we can do via OWA, OMA and push/ActiveSync so that's a non-issue. However the presentations are semi-sensitive. Commerical sensitivity rather than any personal data. However I am already looking at encrypting their drives.
The problem is that due to their file sizes, (sometimes 50-100Mb each) syncronising simply isn't possible.... At least not automatically. Plus a single folder would just result in them storing all documents there as they think it will be like a backup. (but obviously it won't sync 2Gb of files! :-)
I'm thinking of having the clients run as Vista (or XP until I get a moment to upgrade them) and only as normal users. Access to applications other than office will be done through Citrix via the SA 2500 device. Emails are done through OWA/Push-Email, and all documents live on the network. If the users need a file for a meeting then they can use the SA 2500 to grab the file (e.g. a HTTP download) and re-upload later.
My final concern is regarding HTTP.
If a user goes to our https://remote.company.com/ portal and signs in, the SA2500 will allow them to browse through the portal. (e.g. in the same window) and redirect all traffic through a proxy I specify. This works great and semi-solves the problem.
However, the users will sometimes need to use wifi.... so I can't limit the HTTP traffic to just the remote portal.
Anyone know of any apps or Windows hacks where I can specify that the only websites a user can access are xxx.xxx.xxx.xxx (our portal) PLUS any local network URL's....? (e.g. if I user needs to pay to use a public wifi point they will need to HTTP to the relevant URL which is normally internal - however I don't want them to be able to surf the internet this way - they MUST use our HTTP proxy via our SA2500 portal...)
Thanks again,
Hmmmm, if I am understanding what you are asking for you want something that will sit on the stack and redirect all traffic to your network.
The only efficient way I can think of doing this is to shut down ALL ports on the box via GPO except for one non standard one like 8086. then have your portal answer on that port. Change their homepage to that and voila! network natzi's strike again :) This would even cause issues with your hacker wannabe TOR users ;)
hehe - I like your style! :-)
That would work fine - but there is one problem. The sales staff will be using public wifi points. Some of these points will proxy your data through to some payment page of some sort. (Generally I think they are hosted within the same subnet but not sure).
So I need to allow HTTP access to the local subnet, but nothing else. (Other than to remote.company.com which they can then browse from there)
Thanks again!
restricting to the local subnet will only work if the captive portal is on the local subnet. I have run my captive portal outside the local subnet on occation.
Even with tunnels you still run into these issues every once in a while where a router wont allow tunneling. (rare but happens).
Perhaps a better way, rather than locking them down is to do a big brother approach and put a web filter on their box that reports to their supervisor. There are a lot of these out there, rather inexpensive too for corporate ones.
Very true.
Was thinking that there are only a handful of "popular" wireless operators and I could ensure that they have access to them (e.g. *.spectrum.co.uk, *.btopenzone.com etc.) as well as remote.company.com.
Problem is expense. We've just shelled out £4k on the remote SSL device which IMHO is worth every penny - just the problem is the wifi!!!!
ideally I need a filter which will block all other than some keywords (being "spectrum", "openzone", "wireless", "gateway" etc.) and then allow our portal as well. This should allow access to the wifi gateways to pay / sign up and then block all traffic unless it routes through our gateway (which in turn routes via a leased line through a web filter in-between).
Hmmmm, interesting. Any suggestions on what this magical product maybe would be appreciated! :-)
well, arent most every portal for pay or authentication done via ssl? You could only let ssl (443) out.
Other than that check out these apps:
This one is the most promising but I have no experience with it.
This is a fully functional suite that I have heard good things about.
This is a cheap everyday one.
I use a SonicWall for web filtering. It will allow you to do content filtering and a number of other things. One of the nice features it has is blacklisting/whitelisting websites based on keywords that you specify.
Hi Steve-
I have also found recently that a lot of hotels and remote locations are beginning to block VPN access through them. I was having users complain that they were unable to connect and could not sync Outlook or access our file servers from the road. I implemented folder redirects of the MyDocs and allowed them to sync to server and work offline. I also configured Outlook to work over RPC which will allow their Outlook to connect without ever having to connect to any VPN if one is not available at the time.
We use Server 2008's RemoteApp functionality to run a propritary software package and Outlook 2007. For remote users (in our case, dispatchers), it runs like a normal application on thier desktop, but has the security and configurability of Remote Desktop.
It runs similar to Citrix, but since Remote Desktop is built into Windows XP and Windows Vista, it's easier to deploy. Just install the application on a server, configure terminal services and deploy the installation package it creates via group policy.
This doesn't address your issue with disconnected users (something we fight as well) but it does handle the 'no-vpn' and security requirements.
Ernest =)
Hi Steve
Have a look at this product it could provide the sales laptops with web management, IM management, AV, Antispyware, asset management and usage reporting. It's centrally managed across the web so no need for VPN. http://www.netintelligence.com/ni_enterprise.php
How about 3G cards for all the sales (if you still want to cover their home internet, NetGear makes a 3G home router that could share a card with their laptop). Use Citrix for database access and anything that needs to stay really secure. Use a SharePoint Site/Server for general file access. That will let them checkout large files to work on locally and upload changes made to the file when they check the file back in. That way all data is still backed up at your central location, collaboration will increase and you will be in the position to move to Microsofts VoIP solution in the future.


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If you want to reach one of the 7 Summits without any technical climbing or mountaineering required, then a Mt. Kilimanjaro climb is one to put on your list.
Mt. Kilimanjaro is the highest peak in Africa at 5895m above sea level and is known as one of the most accessible of the seven summits because it can be hiked by those with good fitness, determination and who can cope physically and mentally at high altitude.
Fitness for Kilimanjaro
Fitness is very important, and in general walking and hill training is a must for any multi-day hike. This is how we prepared this in the months leading up to departure as we found this to be the best way to replicate what we would be doing on our trip. This is also a good time to wear in the trekking boots you'll be wearing to ensure they're properly broken in before your trip. It is an extremely high-altitude experience as well, so this cannot be overlooked or taken lightly when deciding to commit to this trip.
A glimpse of our goal – Mt. Kilimanjaro 5895m above sea level.
Choosing your route
Acclimatisation is the key so choosing the right tour and pace is important – we did an extra night on the climb to help give us the most success at getting to the summit. We also chose to do the Machame Route as it's known for scenery and some rocky challenges, plus it helps avoid the crowded Marangu Route, which has huts.
We preferred to camp and with 3 alternative routes commercially offered with most trekking companies, the Machame Route ticked boxes for us.
Selecting the length of your trek
We chose a 7-day trek to give us an extra night on the mountain before our summit attempt. Looking back, this was the best move we made. Having that extra night on our trek meant we only had a half day of walking to base camp for our midnight summit attempt. Most other 6 day treks walk a whole day to get to Base Camp and then summit that night.
Ian and our guide, Baracka, cheering before we get to camp on day 2.
The tour guide company we used
We travelled with an amazing local crew of guides, porters and cooks through G Adventures. We had our tent and a sleeping mat provided so we needed to take our own down sleeping bag, liner (rated -4), water storage, purification tablets, clothing and a head torch.
Our guides also put our minds at ease (especially mine) about how to deal with things like headaches, stomach gas (some team members did get windy above 4000m!) and not overthinking it too early – anxiety about getting altitude sickness is common. It was reassuring to be in the hands of those who had trekked the route many times over and knew how to look after trekkers who might be feeling the effects of altitude.
I had been at higher altitudes only once before while trekking the Inca Trail in Peru (around 4200m) many years ago so I had not ventured much higher since and was hugely anxious about getting to the summit (in a conscious state). Looking back, I realise that 90% of this climb was psychological.
Pacing yourself
We chose a 7-day trek to give us an extra night on the mountain before our summit attempt and looking back this was the best move we made. Having that extra night included on our trek meant we only had a half day of walking to base camp for our midnight summit attempt. Most other 6 day treks walk a whole day to get to Base Camp and then summit that night.
The climb pace is agonisingly slow but for good reason! Our guides told us 'pole pole' (pronounced poly – poly) meaning 'slowly slowly' in Swahili, as we took our time on the trails. At times we felt like robots, but it's very important to take it easy and not push yourself too much.
Stopping for lunch along the way to recharge for the rest of the day.
Camping along the way
Camping is still a pretty comfy affair as there's a team of guides, porters and cooks. Most companies also offer a mess tent for serving multiple course meals. The porters carry the tents, your personal possessions, sleeping mats and camping gear – and will often manage to arrive before you and set the tents up by the time you reach camp.
It's phenomenal watching the porters pass, moving fast and sweating with huge loads despite the weights being strictly monitored before the trekking teams depart. With just our daypacks, we still were not able to keep up with porters carrying much heavier loads.
The first 5 days are slow going – acclimatisation is so important – you just need to remember to breathe! It's a great opportunity to bond with fellow trekkers and our guides. We soaked in the scenery as day by day it changed from rainforest on day 1 to bare rocky ground by day 5. Each night we chatted with trekkers from Spain, the UK and other parts of the world.
Here's us on day 3 – we stopped to rest at Lave Towers which is 4630m above sea level.
Altitude sickness
Both my hubby and I reacted differently to the altitude at different times on our trek. While he suffered on the 3rd day with fatigue and vomiting, on the 4th day I simply felt drunk and lightheaded and had to stop many times to remember to breathe deeply. It was just the effects of less oxygen and as it turned out I really just needed the extra rest. I love camping but I find it hard to sleep at high altitude, so I was not functioning well on a few hours of broken sleep each night.
Keeping clean and going to the toilet on the climb
The conditions make it tough to stay clean. There are no showers, and it can be very windy and dusty. Going to the toilet is something to get used to on the mountain, so good hygiene is paramount. Pack hand sanitiser, water purification tablets, more toilet paper than you would normally need, and wash your hands as often as you can as water is not available readily at all camps.
If you wear contact lenses, it is recommended not to wear them at night and switch to glasses where possible by day, if you can. This is not just for hygienic reasons but to maximise the oxygen supply to the eyes. I ended up wearing glasses for most of the trek. It was just easier. My hair was permanently tied up and under a beanie or Buff to keep it tidy and out of the way. Buffs are awesome for keeping your hair back, to cover your nose and mouth from dust, or as a scarf when it's chilly.
We did pack wet wipes to 'bathe' with each night in our tent, so we could wipe off a bit of sweat and grime in place of a shower. It was a mental pick me up to do this and nice to clean up at the end of a walking day before changing into our thermals and camp wear. You do get used to feeling unclean and smelling similarly after a few days – but it's comforting knowing everyone else is in the same position as you.
Climbing up Barranco Pass on day 4.
Dealing with sleep deprivation and the cold
The most challenging elements of the summit night are the sleep deprivation and the temperature. We got our "wake up" call at 11 pm even though we had not yet fallen asleep, and it was sleeting. We did not realise this at the time, but we would not see a bed again until 6 pm the next day.
The temperature was minus 15 degrees, so it's really important to pack layers, as I soon found out. I had 5 layers on top – including 2 thermals, a long-sleeved shirt, a polar fleece and Goretex jacket and 4 layers on my legs – 2 thermals, trekking pants and rainproof pants. My hands suffered the most, however, I had 2 pairs of gloves but this was not enough. I had to add a pair of socks and smack my hands together to get the circulation moving in the cold.
Delayering for a toilet stop was interesting! It took a lot longer to get the job done and did I say to pack loads of toilet paper? We ran out by summit night, and packed wet wipes instead which froze – making wiping a whole new challenge!
Getting through the climb
The moonlight climb was long and slow in darkness. We kept our heads down, breathed deeply and then breathed deeper again. We tried to eat and drink regularly but our water bottles eventually froze. I also made the mistake of looking up at one point in the darkness and all I could see was a zig zag lined of head torches above. It was going to be a long slog so I had to stay positive and focused!
I was yearning for the sun to rise. All I wanted was to see where I was after climbing all this way and to enjoy the views. But that would come.
The spectacular views on day four of the trek.
Reaching the summit of Kilimanjaro
Finally, after about 6 hours, the sun started to rise. We were nearly at Stella Point at 5685m. Reaching this felt like the hardest challenge yet. It was a struggle to do the last hour or so and push on to Uhuru Peak.
We reached Uhuru Peak around 8 am after a very slow trudge to the top. With tears of relief and elation, we had made it to 5895m! Our photo shoot was a quick one, it was really hard to get the oxygen in our lungs to stay up there long. We turned around and made a hasty descent back to Base Camp. The challenge was not quite over though, as we had to move on after a late breakfast back at camp. We then descended to High Camp arriving mid-afternoon. It had been a long time between sleep! Our final descent the following day had us at Mweka Gate by lunchtime – and just like that our trek finished.
This was by far the toughest climb I have ever done, challenging me physically and mentally. The fact I achieved it still surprises me sometimes when I reflect back on this trek.
The saying is true though if it doesn't challenge you it doesn't change you. And, that first shower at the end of the trek was a long one.
Here we are at the summit – 5895m above sea level!
Tips for travelling to Tanzania
- Getting to Tanzania is a bit long winded so allow some extra days in your itinerary before attempting the climb. Throw in a short safari if you can, as you'll be so close to the Serengeti and Ngorongoro Crater. This will also give you time to get over jet lag before you start, or if you lose your luggage and it needs to find its way to you.
- Carry your boots in your carry-on luggage. You can replace most gear if your luggage gets lost or delayed. But, the right pair of worn in boots are pretty much invaluable for a trek such as this.
- There are visas required for Australian passport holders so best to have these organised before you depart.
- Register on the Smart Traveller website and check for the latest official travel advice regarding Tanzania.
Vaccinations and health checks
- Don't forget to check with your doctor regarding vaccinations you may require, and altitude sickness prevention advice.
- Get an overall medical check-up prior to ensure that you're in the right health to attempt this climb.
Gear tips
- Get a decent headlamp – you will need it for most of the summit night climb! If you need help choosing a headlamp, check out our guide here.
- Hiking poles are invaluable especially for protecting your knees if they are prone to fatigue on multi-day hikes.
- Small thermos for the summit night is a good idea, so your water doesn't freeze.
- Ensure you have a high-quality sleeping bag that can cope with sub-zero temperatures. Down sleeping bags are super warm and well worth the investment for many years to come.
Has this inspired you to climb one of the seven summits of the world? Which one do you think you'd be up for?
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For some reason God is really wanting me to be reminded of the brevity of life and the hope we can have on the other side of death.
Yesterday, I was given the opportunity to speak to a great group of local business leaders at the Goshen Noontime Kiwanis. I was asked a few weeks ago to talk about Easter. As I prayed and sought God on the nature of my talk, he kept saying, "Jim tell them about the hope that comes from the resurrection." So I prepared a message with that in mind.
As I shared it seemed like God really wanted this message to get out there. Even after I finished speaking, the room was pin drop quiet. It was as if God walked into the room and said hush. I left the meeting and headed out with my wife Anne, we talked about the warm gathering and the events of the hour. My wife is very in tune with God and she said, "Jim, that was just a powerful message of hope." As I shared I reminded this dear group that even if I were to pass away today I had hope in Jesus and my next breath would be in his presence. And that the Resurrection gives us hope that we can trust Jesus.
Later in that very day if not during that same hour a message came back to this group that one of their very own had died in a snorkeling accident in Belize. He was an active member. I marvel at the timeliness of God's word.
So today, as I was driving back to the office I was stopped by a funeral procession heading to the graveyard. In fact, the light turned green as I sat at the intersection so I became the first car at the end of this procession. Even though I was not in the funeral, cars along the way stopped and pulled off the side of the road for me. Because it looked like I was a part of this grieving group.
So maybe, God wants me to pass this on to you today. There is great hope beyond the grave when you have Jesus as your Lord and Savior. There is no reason to fear death in this life because Jesus is waiting on the other side. For some reason Jesus is reminding me of this again today. I have no reason to fear I know who holds my life in his hands.
Uncomfortably Human: Why Jesus Can Sympathize with You - [image: Uncomfortably Human] Jesus Christ is both fully God and fully man. When many of us read about the God-man in the Gospels, however, we see the God a...


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TOPSHAM — The town recently celebrated an extension to a bicycle-pedestrian trail that was years in the making.
The new trail runs from Town Hall at 100 Main St., across the street and around the Wright-Pierce office, then east through a forest to Community Drive, parallel to the U.S. Route 196 Coastal Connector.
Work on the approximately 0.75-mile segment, primarily completed last December, will continue this fall with landscaping to provide a buffer from the Coastal Connector and replacement of trees.
"It's just a better planting season; we don't want to be doing it mid-summer," Town Planner Rod Melanson said in an interview June 21, noting that the town is working with abutting landowners on the landscaping design, with assistance from Wright-Pierce.
"This was a very long process, getting this actually installed," Melanson said, referring to funding issues due to competing projects around the state, and noting that the project had five different Maine Department of Transportation managers during the life of the decade-long project.
Melanson recalled Perry Jordan, a resident of the Highlands retirement community who walked his Welsh corgi every day on Route 196 to the Tim Horton's restaurant, regardless of weather.
"He was so looking forward to this thing opening; he knew all about it," Melanson said of the new path.
Jordan died prior to the groundbreaking. His family donated a bench in his memory along the path.
Construction cost about $680,000, with 80 percent funded by the state, and about $50,000 in private donations.
The first segment of the four-phase project runs along Monument Place from the Topsham Fair Mall to Town Hall.
The town will extend the path in front of Town Hall by widening the sidewalk from 6 to 10 feet, and connecting to the existing path on Monument Place, Melanson explained. The rapid flashing beacons, located at the new crosswalk to the path, help pedestrians and bicyclists to cross Main Street much more safely, he added.
"This whole segment, from the Topsham Fair Mall all the way to Community Drive, will be one uninterrupted shared use path, at the end of the summer," Melanson said.
The next phase, for which preliminary design work has been completed, could start across the Coastal Connector at Village Drive and run to the Topsham Crossing neighborhood.
Planned future segments could include a bridge over the Coastal Connector and paths to the Androscoggin River Path and Mt. Ararat High School.
Alex Lear can be reached at 781-3661 ext. 113 or [email protected]. Follow him on Twitter: @learics.
Topsham held a ribbon-cutting ceremony earlier this month to commemorate the expansion of a bicycle-pedestrian trail in town. Town Manager Rich Roedner cuts the ribbon; to his right is John Rodrigue, project manager with the Maine Department of Transportation. Town Planner Rod Melanson is at far right.
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Minutes from the Federal Reserve's most recent Federal Open Market Committee meeting suggest key monetary policymakers spent their last gathering pulled in different directions, whipsawed back and forth between the reality of employment growth and the expectations of investors.
Still, at the time, most members thought tapering would end by the close of 2013.
"Most Fed officials still expected to begin tapering before year end and to halt it completely by mid-2014," analysts with Capital Economics said. However, economic data weakened leading up to the meeting, changing the mood a bit.
The minutes, which are from the Sept. 17-18, show a clear divide between FOMC members. A dominant majority decided not to taper the Fed's monthly purchases of mortgage-backed securities and Treasurys after experiencing months of rising mortgage rates and a general slowing in the housing economy as the market inched toward expectations of a Fed tapering in September.
The housing market's stall combined with improving – but hardly impressive – employment data prompted Fed officials to delay tapering once again. But the full board was not in agreement.
As always some members opposed the FOMC's decision not to announce some tapering of asset purchases. Other members pushed for a small tapering on the grounds that a failure to taper in September could send a message of pessimism to a market already anticipating some pullback in quantitative easing.
Another FOMC subgroup supported limited tapering, with the recommendation that the Fed carve back Treasury purchases first.
It's clear when reviewing the minutes that rising mortgage rates over the summer spooked FOMC members, causing them to turn cold on their QE clawback plans.
"Several participants judged that overall financial conditions had tightened notably over the past few months, as seen most importantly in the rise in mortgage rates," the FOMC minutes stated. "While acknowledging that it was too early to assess the effects of such an increase, they expressed concerns that tighter financial conditions might weigh on the recovery in the housing sector."
Other members of the FOMC pointed to higher equity prices and loosening lending standards as signs of improving financial conditions and justification for some tapering.
Housing remained a top concern, with the Fed cognizant of how MBS tapering could impact the overall housing sector.
Some members expressed concern that "an announcement of a reduction in asset purchases at this meeting might trigger an additional, unwarranted tightening of financial conditions, perhaps because markets would read such an announcement as signaling the committee's willingness, notwithstanding mixed recent data, to take an initial step toward exit from its highly accommodative policy."
The minutes also show a Fed fretting over how its communications play out in public.
"With many outside observers expecting a decision to reduce purchases at this meeting, some participants emphasized a need to clearly communicate the rationale behind any decision not to do so, in order to avoid conveying a message of pessimism regarding the economic outlook or to reinforce the distinction between decisions concerning the pace of purchases and those concerning the federal funds rate," the minutes stated.


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The Calcutta high court on Tuesday granted bail to former athlete Pinki Pramanik in a rape case.
Pinki was arrested on June 14 after a woman alleged that Pinki Pramanik was actually male and that he had raped her.
Pinki Pramanik's gender test report will be tabled in the court later in the day.
Pinki Pramanik, who won gold medal at the Doha Asian Games, was allegedly kept with male inmates in the jail resulting in protest by several human rights organizations.
Concerned over reports of ill treatment of former athlete, the National Commission for Women (NCW) had asked the Kolkata police for a report.
NCW officials said the authorities have been asked to reply in a week. In a statement, NCW said, "The commission has sought action taken report from the director general of prisons and commissioner of police, Kolkata regarding this matter within a week."


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Game of Thrones fans has been dying for any information on season six. Any and all information regarding the new season has been tightly under wraps…until now.
GoT newcomer Elie Haddad (cast as a Dorathki) accidentally leaked a script page to his Instagram account. The picture has since been deleted, but fans were able to screenshot the image and decipher a few lines.
a new #goT actor ellie haddad accidentally shared a picture on instagram [now deleted] you can see some details pic.twitter.com/RKFhRgF9tL
— andee olivares (@o3flasho3) November 5, 2015
According to fans who were able to make out some of the show's script, the exchange takes place between two Dorathki. The pair appears to discuss the Khaleesi in typical and lewd Dorathki fashion. If you remember, Daenerys was taken away by the Dorathki at the end of season five. I wonder what the new Khalasar has in store for her.
Verrrry interesting.
Don't miss 10 things that are better than having a boyfriend and 9 times Mindy Kaling gave us outfit goals!
[Photo: HBO]


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How do you tell your story?
Social media, podcasts, opinion pieces, infographics - always make sure to match your content marketing activities to your audience and brand story.
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Special local artist's work on show at the Palms
The Palms Shopping Centre is partnering with local organisation SkillWise, which works with people with intellectual disabilities to provide personalised support and help with training, education and creative expression.
Q+A with Dr Troy Coyle
How do well-being and the heavy metals industry go together? Dr Troy Coyle, CEO of HERA, talks to Jack Tame on Q+A!
How to be more productive at work
Productivity hack: Structure your day to have specific time slots for doing work and spending time with your family.
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VR becomes critical tool for tackling NZ skill shortages
The Ministry of Social Development is putting its weight behind Joy Business Academy (JBA) to boost virtual reality use in industry training – particularly in construction.
Amazon Alexa can now teach English and te reo Māori
Amazon Alexa can now help teach English and te reo Māori, thanks to a new voice-activated Alexa skill.
Botany Town Centre unveils the first and largest stage of their $78 million development
On Thursday 2 May 2019 Botany Town Centre unveiled the first and largest stage of their $78 million development, the Pavilions Extension.
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Additionally, The Content Place recruits the services of leading experts in social media, web conversion, SEO and SEM, online PR and traditional and native advertising. These skills are brought to bear in the service of clients who need specialist relevant content produced for a range of 'owned' media and delivered in ways that will enhance their brands and the reputations of individual business leaders. Ask our team how we can help your content marketing programme.
Active Downtime: Getting Match-Fit For Business
In this article for his bi-monthly coach column for M2, Andrew Barnes writes of how his love for sailing has also benefitted the business side of his life.
Blue skies ahead: the 2016 fiduciary services forecast
Perpetual Guardians Personal Client Services Director John McFetridge explains what 2016 promises for fiduciary services in Asset Magazine.
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The Good Ship IPO
The seas of the market are constantly shifting. Whether the good ship IPO can set sail may depend heavily on the tides.
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How will you weather the ups and downs of the business cycle?
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Understanding some basic concepts may help you assess whether zero-coupon bonds have a place in your portfolio.
5 Smart Investing Strategies
Getting what you want out of your money may require the right game plan.


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12 September 2018, 22:20
In the 2nd quarter of 2018, no one fell victim to armed conflict in Kabardino-Balkaria
From April 1 to May 2018, in Kabardino-Balkaria, no victims to the armed conflict in Northern Caucasus were reported. These are the results of the calculations run by the "Caucasian Knot" based on its own materials and information from other open sources.
In the first quarter of 2018, no information about victims to the armed conflict involving law enforcers and militants in the territory of Kabardino-Balkaria was reported.
The "Caucasian Knot" notes that the provided statistics cannot reflect the number of victims with the 100 percent accuracy. Not all information is included in the news reports. It is not always possible to check the reliability of the statements voiced by the law enforcement bodies. Besides, new facts about the events of the reporting period become often known after the statistics had been already published on the "Caucasian Knot".
For the whole year of 2017, one person fell victim to the armed confrontation, he was killed.


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SVA Alumnus Jon Lung Wins Competition to Become a Host of 'MythBusters'
February 27, 2017
From CNET: "It's not easy filling the shoes of science rock stars Adam Savage and Jamie Hyneman, but two competitors have earned spots to host a new version of 'MythBusters.'
After a grueling competition in Science Channel reality show 'MythBusters: The Search,' 10 contestants were narrowed down to four finalists tasked with figuring out through scientific experiments whether it's possible to escape from prison using a rocket-propelled water heater.
'MythBusters: The Search' debuted in January with contestants asked to check myths running the gamut from finding a needle in a haystack to painting a room with explosives. The final episode aired Saturday.
Rescue diver and pilot Brian Louden of Spring, Texas, and product designer Jonathan Lung of Staten Island, New York, won and are the new official hosts of 'MythBusters.'…" (continue reading)


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I was raised by a pack of wild amoeba in a petri dish.. Couldn't stand the heat of the bunsen burner,so I struck out on my own... Now,looking to my right I can ascertain that the one who really belongs in this lab outpost is he that will always tempt the fates..And from another perspective.....
January 17, 2005
Bettie Page,the queen of pinups.This woman has been photographed more in her short career(1950-1957)than any other living fashion model! All kneel at the altar of the beautiful Bettie!
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"I can do all this through Him who strengthens me."
Philippians 4:13
Bethany Christian School has a carefully planned program that enhances the social, intellectual, physical, spiritual and emotional development of each child. We hope to maintain a balance of these areas so that each child can develop a positive attitude towards learning, and towards responsibility, self-discipline and self-esteem.
2. Intellectual – to provide a wide variety of activities within a routine where cooperative play is encouraged; to provide a well planned curriculum with areas of art, science, cooking, dramatic play, language and fine motor activities where each child will have the opportunity to talk about what is learned, heard, seen and done and begin sharing these experiences with their friends.
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4. Spiritual – to learn about prayer and that God loves you and hears your prayers. The threes and fours will have chapel once a week in the sanctuary.
5. Emotional – to provide a warm accepting environment for each child to work and play in; to help develop self-discipline in each child; to provide appropriate alternatives to angry and aggressive behavior and strengthen self- control.
Our Tuesday/Thursday classes attend Chapel on Tuesdays and our Monday/Wednesday/Friday classes attend Chapel on Wednesdays. Chapel is led by Bethany Christian Church's Senior Pastor, Don Tuttle in our sanctuary
Music is taught in all classes during circle time.
Spanish is taught in all classes during circle time.
Gross Motor Skills
Our gross motor teachers plan and implement daily activities that are developmentally appropriate for each classroom.
Soccer Shots
Soccer Shots will be hosting classes during our school day, once a week beginning in the fall semester. Keep an eye out for details to come!
Books Between Kids Book Drive at Bethany
Bethany Christian School will be hosting a book drive for Books Between Kids from September 16th through the 20th. Please bring new or gently used books at drop off or pick up to our drop-off bins in the gym.


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Eat Stop Eat
Genetic and metabolic factors in the development of obesity have received increasing attention since the identification of the protein product of the ob gene—leptin, and its receptor. Clearly there are cases among humans, as well as in strains of rodents, in which a single gene mutation has been identified as the direct and specific cause of obesity. That obesity is a genetic disorder is rarely disputed today. What remains under discussion is the relative magnitude of the contribution of genes to body weight compared to the contributions of excessive ingestion of calories due to environmental considerations. Excessive ingestion of food and reduced energy expenditure, both of which may be exacerbated by an enriched environment, are sometimes considered to be the primary culprits.
Concerning obesity in humans, just as it may be difficult to get a genie back in a bottle, so it may be difficult to 'demodernize' the environment of humans, or to otherwise alter it so as to mitigate the 'New World syndrome' (25). This is confirmed by the modest success of most weight control programs, and suggests that the forces aligned against weight loss and weight loss maintenance are indeed powerful and poorly susceptible to the combination of environmental manipulations and volitional changes among humans with excess weight.
We have found the same to be true in non-human primates. For example, forced weight reduction by limitation of available calories provided to obese monkeys produces weight loss. However, the recidivism when the monkeys are returned to a non-restrained calorie regimen is 100%. This occurs despite the use of a high fiber, low fat chow diet which is not highly palatable. What might be the nature of these 'forces' that mitigate against significant weight loss for most middle-aged persons, and that promote weight regain or recidivism in most of those who have successfully lost weight? Are these 'forces' present in animals as well?
The strong familial aggregation of risk for obesity in humans provides evidence for a powerful familial component to the development of obesity. Some suggest that this is a combination of shared environment and shared genetic propensities. Both quantitative trait loci in rodent models and family linkage studies in humans have identified a number of chromosome areas which may carry obesity promoting genes. However, at this time these remain only promising regions, some of which may contain candidate genes for further study.
Figure 14.2 Longitudinal data from a single monkey (A-7), beginning at sexual maturity (young lean adult age 5) through the progressive development of obesity followed through age 17. Panels show sequential changes in body weight, % body fat determined by tritiated water dilution, fasting plasma glucose, fasting plasma insulin (and progressive hyperinsulinemia), M or glucose uptake rate during a euglycemic hyperinsulinemic clamp (a measure of whole body insulin sensitivity which declined over time) and K glucose or glucose disappearance rate during an intravenous glucose tolerance test (which showed a slow declining function)
Figure 14.2 Longitudinal data from a single monkey (A-7), beginning at sexual maturity (young lean adult age 5) through the progressive development of obesity followed through age 17. Panels show sequential changes in body weight, % body fat determined by tritiated water dilution, fasting plasma glucose, fasting plasma insulin (and progressive hyperinsulinemia), M or glucose uptake rate during a euglycemic hyperinsulinemic clamp (a measure of whole body insulin sensitivity which declined over time) and K glucose or glucose disappearance rate during an intravenous glucose tolerance test (which showed a slow declining function)
Single Gene Mutations in Animal Models
Lessons for human obesity can be learned from animal models of genetic obesity. First, single gene mutations can and do cause obesity in both rodent models and in humans—this is indisputable. In rodents such mutations have been identified in at least five genes, including the ob gene for the circulating adipose tissue-secreted factor leptin, the db gene for the receptor of leptin (and in rats, the fa gene), the agouti yellow (Ay/a) mutation which controls the production of melanin pigments controlling skin color in mice (with its human equivalent agouti signaling protein gene), the fat mutation in the car-boxipeptidase E gene which is a prohormone processing enzyme, and the tub mutation which is still under study to determine its function, but which may be a protein involved in insulin signaling (26). Other genes which have been implicated in body
Figure 14.3 Progressive changes in body weight, fasting plasma glucose, and fasting plasma insulin as monkeys progress from lean normal (phase 1) to heavier and older (phase 2), following which some monkeys continue through a series of phases reaching overt diabetes (phase 8) and severe diabetes (phase 9)(Redrawn from Hansen (101)). Others remain in phase 2 all of their lives. * P<0.05;| <0.01; = P< 0.001 (in comparison to phases 1 and 2)
Figure 14.3 Progressive changes in body weight, fasting plasma glucose, and fasting plasma insulin as monkeys progress from lean normal (phase 1) to heavier and older (phase 2), following which some monkeys continue through a series of phases reaching overt diabetes (phase 8) and severe diabetes (phase 9)(Redrawn from Hansen (101)). Others remain in phase 2 all of their lives. * P<0.05;| <0.01; = P< 0.001 (in comparison to phases 1 and 2)
weight regulation include the melanocortin-4 receptor (MC4-R) (27), and its other isoforms, melanin-concentrating hormone, receptors mediating leukocyte adhesion (deficiency in intercellular adhesion molecule-1 or in its receptor, leukocyte in-tegrin alpha M beta2 (Mac-1)), and the possible suppressor of obesity, the mahogany protein, which may be a signaling protein or receptor similar to the proteoglycan receptors. Although extensive efforts have been made to identify mutations in these and in other candidate genes for obesity in humans, to date, only a handful of individuals have been identified with mutations in any of the genes which have produced obesity in rodents.
Based on the observations of food intake and body weight in parabiotic obese and lean rodents, a circulating product of the adipose tissue had long been suspected of being involved in body weight regulation (28,29). Cross circulation studies in non-human primates, by contrast, did not support the idea that a circulating factor might be involved in feeding regulation in any dominant or major way (30). Interestingly, both studies now appear to be confirmed.
The specific genes and their mutations implicated in the early rat parabiosis studies were identified only a few years ago (31,32). The cloning of these genes, the identification of their mutations in obese rodents, and the identification of the circulating gene product (leptin) and its receptor (32) led to the hope that the genetic basis for human obesity might soon become clear. The ob genes in humans (33) and in monkeys (34) were cloned and sequenced. The deduced amino acid sequence of the human OB protein coding region was found to be 84% identical to that of mice, 83% identical to rats, and 91% identical to that of the rhesus monkey. The genes of many obese persons and monkeys have been searched for defects in the ob or leptin gene and its receptor. As noted, only a handful of patients and no monkeys have been identified with mutations in either of these genes (35-37).
Variations in the Circulating Product of the ob Gene, leptin
With the identification of the peptide released from adipose tissue, animal models as well as humans were examined for characteristics associated with variations in plasma leptin levels. Both monkeys and humans have been reported to show strong correlations between body weight and plasma lep-tin levels, and between body fat and leptin levels, as
shown in Figure 14.4 for rhesus monkeys (38). It was immediately noticed that while lean subjects generally have low leptin levels, not only was there an increase in leptin levels with increasing adiposity, but the variability increased greatly, such that obese subjects could be identified with normal to extremely elevated levels, e.g. 5- to 10-fold higher than normal. No explanation for these large variations has been established, since all plasma samples were obtained consistently under overnight fasted conditions. What is clear is that leptin is not released and does not circulate in a simple ratio to fat mass. This is in contrast to the observation that, in rats, the leptin-body fat ratio is a constant for a particular strain (39). Circulating levels are altered significantly by fasting and refeeding, and by many other factors.
The possibility that these large variations in circulating leptin levels might indicate important differences in the receptors for leptin has also been closely examined in rhesus monkeys. No leptin re
Figure 14.5 Expression of the long form of the leptin receptor (top panel) and the total (short plus long) leptin receptor in adipose tissue of monkeys ranging widely in body weight. Open circles are normal monkeys, open squares are obese hyperin-sulinemic, and solid circles are overtly diabetic monkeys. (Reproduced with permission from Hotta et al. (37))
Figure 14.5 Expression of the long form of the leptin receptor (top panel) and the total (short plus long) leptin receptor in adipose tissue of monkeys ranging widely in body weight. Open circles are normal monkeys, open squares are obese hyperin-sulinemic, and solid circles are overtly diabetic monkeys. (Reproduced with permission from Hotta et al. (37))
ceptor mutations were identified, and expression levels as determined by polymerase chain reaction (PCR) of the two principal isoforms of the receptor, the long form and the short form, were not associated with body weight as shown in Figure 14.5. There was also no association between expression of the forms of the leptin receptor and fasting plasma insulin, plasma glucose, or circulating plasma leptin levels.
Rare genetic defects in either the insulin molecule or the insulin receptor have been identified in humans and associated with insulin resistance and obesity (40). In non-human primates, which have circulating plasma insulin levels five-fold or more higher than in humans, no defects in either have been found to account for the apparent insulin resistance.
The insulin molecule in monkeys is identical to that of humans (41), and sequencing of the insulin receptor has shown it to be remarkably similar to that of humans (42). Molecular examination of the few amino acid substitutions in the monkey insulin receptor compared to that of humans failed to show why monkeys have such elevated insulin levels relative to humans (43).
The insulin receptor is expressed in two isoforms in humans and in monkeys. One form is expressed in higher proportions in obese, hyperinsulinemic prediabetic monkeys (and similarly in humans), and this proportion reverts to normal in diabetes (44,45). Whether the relative proportion of these isoforms plays a role in the progression of obesity to diabetes is unknown. The hyperinsulinemia of obese monkeys is not associated with any difference in food intake relative to similarly obese normo-insulinemic monkeys (46).
Many transgenic animal models of obesity are under study at this time. However, they will not be discussed in this review, which focuses upon spontaneous models of the human condition.
Mendelian Syndromes of Obesity not Identified in Animal Models
There is a large group of Mendelian syndromes in which obesity is a component, including Prader-Willi, Bardet-Biedl, Alstrom, Carpenter, Cohen, Wilson-Turner and others. However, these lack specific animal models. These genetic disorders are rare in humans, and family studies do not suggest that the genes responsible for these syndromes are involved in the common form(s) of human obesity. For more than 99% of all obese humans, and 100% of all obese non-human primates, the genetic basis of the obesity is unknown.
In animal models, adipose tissue has been the focus of numerous studies aimed at understanding its physiological and genetic regulation and the differential expression of various genes that might regulate adipose tissue mass. In non-human primates the possibility that the obese may differ in the expression of the genes that regulate adipocyte differentiation has been explored. The peroxisome prolif-
erator-activated receptor y (PPARy) has been the specific focus of much study since its identification as the receptor for the insulin sensitizer class of pharmaceutical agents, the thiazolidinediones (see below), and its very early expression during adipose cell differentiation. There are two isoforms of PPARy, (y 1 and y2), and both have been cloned and sequenced in the rhesus monkey (47). The latter is highly expressed in adipocytes, while PPARy 1 is expressed widely in many tissues. The ratio of the PPARy2 mRNA to total PPARy mRNA was significantly related to body weight and to fasting plasma insulin, while neither total PPAR y nor its two isoforms individually were related to obesity or to insulin levels (47). This is in contrast to the finding in rats of increased PPARy mRNA levels in the white adipose tissue of obese rats (48).
Other genes expressed during adipocyte differentiation include: CCAAT/enhancer binding protein a (C/EBP a), lipoprotein lipase (LPL), phos-phoenolpyruvate carboxykinase (PEPCK), and the glucose transporter gene (GLUT 4). As shown in Figure 14.6, in a study of normal weight, obese, and diabetic monkeys, none of the best known regulators of adipose differentiation were associated with body weight in any of the three groups, nor in the total group (47). They were, however, found to be associated with aging (49).
A newly identified adipocytokine, adiponectin (an adipose-specific protein abundantly expressed and released into the circulation), is paradoxically decreased in human and in monkey obesity. Its circulating levels are inversely correlated with plasma leptin levels. Adiponectin has been se-quenced in both humans and monkeys and found to have 98% identity (50). There was no relationship between body weight or obesity and adiponectin mRNA, suggesting posttranscriptional regulation by adiposity, a regulation which was disturbed when diabetes developed.
Genetic Susceptibility and Gene-Environment Interactions
Linkage studies and extensive candidate gene studies give presumptive evidence that multiple genes may be involved in the susceptibility to obesity in both humans and in animals, with each gene contributing in small measure to the propensity or sus-
Figure 14.6 Genes implicated in adipose cell differentiation were examined in normal (open circles), obese (open squares) and diabetic (solid circles) monkeys. The expression levels of PPARy (a), LPL (b), aP2 (c), CEBPa (d), Glut 4 (e), and PEPCK (f) mRNA measured by slot blot hybridization were not associated with body weight or total body fat, but, with the exception of aP2 and PEPCK, they were highly coordinately regulated with each other. (Redrawn from Hotta et al. (47))
ceptibility to develop obesity. This likelihood may contribute heavily to the difficulty in isolating the specific genetic contributions within a family or group where a single gene mutation is not known to be present.
Studies both in animals and in humans support the contention that individuals differ in their susceptibility to weight gain and to overt obesity under conditions in which the environment is facilitative or non-constraining. For example, a high fat diet fed to some strains of mice results in significantly more excess body fat than when fed to other strains; indeed there are strains that are resistant to the obesifying effects of a high fat diet (51). A high fat diet also induces obesity in adult rabbits (52). Similarly, in humans differences in susceptibility to weight gain have been identified (53,54).
Efforts are continuing to identify the genetic and molecular basis of obesity or of the 'obesities'. The likely outcome in the future is the identification of many genes, and, within those gene coding regions or in nearby regions that affect gene expression, many different mutations or variants which are responsible for the heavy genetic burden of obesity in humans and in non-human primates. Combinations of these genes are likely to increase susceptibility to weight gain and obesity when the environment is permissive or culpatory. The direct effects of these interacting genes may be to alter metabolic rate or nutrient partitioning, to alter lipid metabolism or adipose tissue function, to alter lean mass, to alter the hormonal milieu, and/or to regulate feeding behavior and appetite. Polygenic as well as major gene effects may be acting to produce the complex phenotype of obesity and its associated disorders in primates. The current state of this understanding for human obesity has been reviewed extensively (55). In addition, many of these genes are likely to interact with each other and with the environment for both humans and animals, thus further increasing the challenge for the future understanding of the mechanisms underlying the physiological basis of obesity.
It is clear that obesity results from an imbalance of energy input and energy output, and that in some animal models it can be induced by dietary methods. For example, the Psammomys obesus or Israeli desert gerbil or sand rat exhibits obesity only under nutritional conditions that this species does not see in the wild (14). Susceptibility of various animal models to nutritionally induced obesity appears to differ across strains and even within a single strain of rodents, and is a characteristic which some investigators have used in selective breeding (51). The mechanism which underlies this susceptibility to dietary obesity is unclear, but in the sand rat is suspected to relate to impaired activation of the insulin receptor and compromised tyrosine kinase activation, which interestingly, is reversible with calorie restriction (see below for further discussion of calorie restriction in obesity). High fat feeding produces many changes in metabolism, as illustrated by the recent finding of increased uncoupling protein 3 (UCP3) levels in brown adipose tissue and reduced skeletal muscle UCP3 in dietary obese rats (56). High fat diets in monkeys have produced significant weight gain, increased body fat, and increases in triglycerides and low density lipoprotein (LDL) and high density lipoprotein (HDL) cholesterol (57).
Viruses have been suspected of being involved in obesity in humans (58), and have been shown to be capable of producing obesity in rodents (59). Whether this is an important mechanism for the induction of obesity is, as yet, unclear.
Adipose Tissue Metabolism During the Development of Obesity
Although insulin sensitivity at the whole body level generally declines as obesity develops, as shown above for monkey A-7, it is seldom appreciated that this longitudinal change at the whole body level is not associated with a similar decline in the sensitivity of isolated adipocytes to insulin actions. Generally, it is believed that whole body insulin action, as measured by a euglycemic hyperinsulinemic clamp, is principally determined by glucose uptake into muscle, with a small contribution of adipose tissue and other organs. At the level of the
Figure 14.7 Insulin action on adipocytes obtained from the abdominal subcutaneous tissue of four groups of monkeys whose characteristics are differentiated in the top four panels: lean normal monkeys, obese normoinsulinemic monkeys, obese hyperin-sulinemic monkeys, and obese with overt type 2 diabetes. Differences in body weights, fasting plasma glucose, intravenous glucose tolerance, and fasting plasma insulin are shown in the top four panels. Panel 5 is the effect of insulin to increase glucose oxidation in isolated adipocytes and panel 6 is the effect of insulin to increase lipid synthesis, both in isolated subcutaneous abdominal adipocytes. (Redrawn with permission from Hansen et al. (60))
Figure 14.7 Insulin action on adipocytes obtained from the abdominal subcutaneous tissue of four groups of monkeys whose characteristics are differentiated in the top four panels: lean normal monkeys, obese normoinsulinemic monkeys, obese hyperin-sulinemic monkeys, and obese with overt type 2 diabetes. Differences in body weights, fasting plasma glucose, intravenous glucose tolerance, and fasting plasma insulin are shown in the top four panels. Panel 5 is the effect of insulin to increase glucose oxidation in isolated adipocytes and panel 6 is the effect of insulin to increase lipid synthesis, both in isolated subcutaneous abdominal adipocytes. (Redrawn with permission from Hansen et al. (60))
adipocyte, the action of insulin, as measured in biopsies obtained both cross-sectionally and longitudinally from rhesus monkeys during the development of obesity and progression to diabetes, shows increased ability of insulin to stimulate glucose oxidation and to stimulate lipid synthesis in obese animals compared to normals (60). The deterioration in insulin action at the adipocyte is a late event accompanying the development of diabetes, as shown in Figure 14.7.
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I lost over 60 pounds and 4+ inches off my waist without pills, strenuous exercise, or any of the things that the diet experts tell you to do...and I did it in less than 4 months! If you have the desire, and can read through my e-book , then this is for you! I could have easily made it a lot more difficult, with stacks of information that people will never read, but why?


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Our next to "last" day in Paris was the busiest. Our day started with breakfast at the hotel which consisted of nutella and croissants, breakfast meats and of course cheese. 🙂 After our Eiffel tower tour with Fat Tire Tours the day before, our tour guide told us about the church on the hill, known as Sacred Heart.
Paris: Eiffel Tower TourSeptember 27, 2015
The one thing I had planned to do in Paris, other than sit at cafes and people watch, was to go up the Eiffel Tower. I had read all about the long wait times and didn't want to be part of that. So, a week before our trip I began searching for our skip the line tickets. What I didn't realize was how busy Paris would be that first week in August.
While I searched and searched, I finally found Fat Tire Bike Tours with a spot for us. Yes, it was way more than buying the regular ticket, but we would have a tour guide and best of all, we'd get to skip the lines. On our way to Paris, our flight was delayed. I quickly emailed the company, who promptly responded and scheduled us on another tour date.
Three days in Paris: Day OneSeptember 7, 2015
This summer we had the opportunity to travel to Europe. You see, back in 2004, we visited Europe for the first time and made a promise to return in ten years. Fast forward to 2015, eleven years later, we made good on that promise with our summer vacation to Europe. On the travel log: Paris, Brussels and Amsterdam.
This was our first time visiting Paris and Brussels. We loved Amsterdam during our Honeymoon, but felt we didn't get enough of it the first time around. Ok, so on to Paris. We loved it. We wondered around the city, stopping for coffee, panini, and wine. It was fun. So much fun, that I can't wait to come back.
Day 1:
We were supposed to arrive in Paris around noon time. However, Delta changed our flight to go through Atlanta instead of Boston. This meant, we arrived in Paris about two hours later than originally planned. Not a big deal, except we had planned a tour for the Eiffel Tower through Fat Bike Tours. I immediately panicked. However, I notified the company via email and Facebook about our flight delay. Luckily, they were able to accommodate us on a different day. More about that later.
FYI- most restaurants in Paris do not serve food between 3 and 6 pm. However, they are open for drinks. So, after walking around for a while, we decided to indulge and start living like a Parisian. Our first dinner in Paris was amazing. It was in a tiny little restaurant off the main roads. Thanks to Yelp! we found Bistrot de L'oulette. I ordered the fish, Jim ordered the beef and we ordered duck for the girls.
After a full belly, it was time to explore Paris before bedtime. Remember, we just arrived a few hours later. We ventured over to the Palace de Voges area. Saw some soldiers with heavy machinery. I don't think the girls even noticed. We just kept walking by towards our hotel.
Stay tuned for my next post: Day two in Paris!


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Having spent the last two days, doing some very necessary job-hunting, attending interviews and generally being all sorts of responsible, I required a bit of a break. Two days is tough stuff, people. Look, I'm easing back into it, okay?
For my hard earned vacation, I took a trip to Osaka Castle. Osaka Castle was originally, a defensively built structure comprising mostly of granite. It was not, very successfully defensively built however, as it was breached and almost completely destroyed. Twice. Now, it is still a very impressive beautiful building, but it is a mostly concrete reconstruction. The added benefit of this is, that it does have an elevator that ascends its 8 floors, which I'm sure, was not part of the original blueprints. The Castle also houses a museum dedicated to the wars that had been fought in the Osaka area, primarily being the Osaka Summer Campaign and the Osaka Winter Campaign. One can only assume due to the flowers blossoming, and the foliage changing, that Spring and Autumn are just too pretty to fight during.
One of the suits of armour on display, that would have made a fearsome sight on the battlefield, was covered almost completely in real black bear fur. It was a very interesting garment to behold, I still couldn't help but feel sorry for the bear though.


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Two American soldiers died after militants attacked a coalition base in eastern Afghanistan, exploding a truck outside the gates and sparking a two-hour gun battle.
US forces called in air strikes to end the clash, killing more than 30 insurgents in Zerok district of Paktika province, said Hamidullah Zawak, the provincial governor's spokesman.
Backed by Afghan soldiers, US forces are involved in the biggest coalition offensive in Afghanistan since 2001. More than 4,000 US marines were airlifted last week into the southern province of Helmand which over-extended British troops have proved unable to secure in three years of tough combat.
Though the marines have had at least one other action with an estimated 30 or 40 Taliban, it is increasingly clear that the insurgents have fled or gone to ground rather than take on a heavily armed US force. British troops continue to push into the north of the province.


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Refrigerated carrier Wispak Transport has announced a new driver compensation program, that consists of bonuses for independent contractors as well as an increased per-mile pay schedule and complete benefits package for company drivers.
"It's not often that both contractors and company drivers find a place small enough to be treated with respect and big enough to pay them well," said director of transportation services John Hiscox. "But because we are a subsidiary of Emmak Foods, they get both."
Benefits for company drivers include life, health, disability and dental insurance, vacation and personal days, and a safety bonus program. In addition, all drivers get newer equipment, which include condo bunks.
Contractors using a Wispak Transport trailer now earn $0.91 per mile, drivers using their own trailer are paid $1.01 per mile. Contractors will also receive "drop" pay at $50.00 per stop, paid lumpers, a graduated per mile fuel surcharge, free fuel tax calculation and filing, and a safety bonus.


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If I were to say kitchen makeover, you'd instantly think "majorly expensive." And if this were some fancy pants site, you'd be right. But it's not and we totally get that not everyone is sitting on thousands of dollars to reno the kitchen at a moment's notice. But that doesn't mean you're stuck with a crappy, cookie-cutter kitchen. Here are 11 easy–and even better, inexpensive!–ways to make over your kitchen.
For more cool home ideas, check out functional furniture and decor, the best salt lamps and cool small space decorating tips.