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Sorry, European travelers. DHS poised to ban tablets, PCs
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Do you prefer the 'old school' way of things? If the answer is yes, you're not alone as many of our customers also do, and just want to sit down, go through everything and not get confused.com with all the 1000 of offers on the internet. If the truth be known, we love it as well!
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Politics PhDUniversity of Surrey
Program description
Politics PhD
The Department of Politics at the University of Surrey is a thriving space for research and public engagement on a range of political areas. We're home to roughly a dozen researchers working across politics and international relations.
We have a growing profile as a provider of outstanding practitioner and researcher training. There are frequent opportunities for discussion of ideas, presentation of work, and participation in the events organised within the Faculty and University. These events include research seminars, visits from regular external speakers, and opportunities to meet policy-makers and politicians. PhD students also benefit from a dedicated room for all research students with excellent IT facilities.
The Department has a strong focus on applied politics, and we are globally recognised for our work in European politics and on international intervention. This excellence is reflected in our two research centres: the Centre for Research on the European Matrix (CRonEM) and the Centre for International Intervention (Cii). We have a strong track record of securing funding from research councils and European funders.
Our doctoral programme promotes high-quality research in politics, international relations and policy studies. We encourage and support innovative thinking, and our postgraduate researchers play an important role at the heart of our thriving research environment.
Admission requirements
Applicants are expected to hold a good bachelors degree and preferably a masters degree from a UK university or overseas equivalent in a relevant discipline.
English language requirements
IELTS Academics: 6.5 or above (or equivalent) with 6.0 in each individual category.
View the other English language qualifications that we accept.
If you do not currently meet the level required for your programme, we offer intensive pre-sessional English language courses, designed to take you to the level of English ability and skill required for your studies here. The University of Surrey is also an IELTS test centre.
Does this course require proof of English proficiency?
The TOEFL® test is accepted by 10,000+ universities and higher education institutes in over 150 countries. Book your test today!Learn more
Program content
We welcome applications from students who would like to conduct research (leading to an MPhil or PhD degree) in any of our areas of expertise.
However, we are also interested in hearing about other areas in which you would like to conduct research, if they are broadly in line with the School's areas of research.
Research students within the Department are well supported, with training opportunities and regular supervision sessions.
For more information Visit University Website.
Tuition fees
The price for UK/EU fees is 4327 GBP.
The price for Overseas fees is 16500 GBP.
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About this institute
University of Surrey
The University of Surrey is a global community of ideas and people, dedicated to life-changing education and research. With a beautiful and vibrant campus, we provide exceptional teaching and practical learning to inspire and empower our students for personal and...
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They say you cannot buy Time. here is an exception. A blend of Armagnac 50 to 152 years old.
A fleeting opportunity. A timeless asset. $250 a bottle: the greatest value in the world of spirits.
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In 2006, Raj Peter Bhakta took a decrepit dairy farm and transformed it into WhistlePig, quickly growing America's most successful luxury whiskey company. With Bhakta Brandy, he's taking that farm-to-bottle ethos to the next level. With a mission of Revival for rural agriculture, exquisite spirits, and America itself, Bhakta is issuing an all-new niche in the spirits world. His tools include a Vermont Farm, a Florida ranch, and Armagnac chateau in France, and the oldest reserve of premium spirits in existence. This is just the beginning.
BHAKTA Brandy is rare, exquisite, and valuable. It draws from French Armagnac stocks dating from 1868-1970. Our 50 Year blend is older than any blended whiskey on the market, and notably superior in terms of small-scale craft distilling. With a unique Islay cask finish to balance sweet with smoke and salt, our brandy is the only one made for the whiskey-lover's palate. They say you cannot buy time. Here is an exception.
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Dissertation submitted to the Combined Faculties for the Natural Sciences and for Mathematics of the Ruperto-Carola University of Heidelberg, Germany for the degree of Doctor of Natural Sciences presented by Diplom-Pharmacy: Fang, Zhaoborn in: Hebei province, ChinaOral-examination: Tolerogenic dendritic cells in ret transgenic mouse model of spontaneous melanoma Referees: Prof. Dr. Viktor Umansky PD. Dr. Anne Regnier-Vigouroux The present work (1.10.2005-1.8.2008) was done in the research group led by Prof. Dr. Viktor Umansky, clinical cooperation unit for Dermato-Oncology, German Cancer Research Center (DKFZ) and University Hospital Mannheim. Publications during PhD work: Umansky, V., Abschuetz, O., Osen, W., Ramacher, M., Zhao, F., Kato, M., and Schadendorf, D. Melanoma specific memory T cells are functionally active in Ret transgenic mice without macroscopical tumors. Cancer Res. 2008. (in press) Zhao, F., Falk, C., Osen, W., Kato, M., Schadendorf, D., and Umansky, V. Activation of p38 MAPK drives dendritic cells to become tolerogenic during melanoma development in ret transgenic mouse model. 2008. (submitted) Work presention in conferences: Zhao, F., Osen, W., Schadendorf, D., and Umansky, V. Study of dendritic cells in Ret-transgenic mouse model of spontaneous melanoma. 2007. 14th International AEK Cancer Congress. Frankfurt, Germany Zhao, F., Osen, W., Schadendorf, D., and Umansky, V. Tolerogenic dendritic cells in Ret-transgenic mous melanoma. 2007. 5th Annual Meeting of the Association for Immunotherapy of Cancer (CIMT). Würzburg, Germany Zhao, F., Osen, Wansky, V. Tomelanoma development of Ret-transgenic mouse. 2007. 5th International Meeting on: 'Dendritic cell Vaccination and other Strategies to tip the Balance of the Immune System'. Bamberg, Germany. Zhao, F., Osen, W., Schadendorf, D., and Umansky, V. Up-regulated Stat3 and MAPK activities in DCs from a spontaneous melanoma mouse model: a role of tumor-derived factors in tolerogenic DC generation. 2008. 6th Annual Meeting of the Association for Immunotherapy of Cancer (CIMT). Mainz, Germany Index Index Summary.......................................................................................................................6 I. Introduction ..............................................................................................................8 1. Immune system and antitumor immune response .................................................... 8 1.1. Innate immunity ................................................................................................ 8 1.2. Adaptive immunity...........................................................................................11 1.3. Evidences of antitumor immunity in animal models and cancer patients . 14 2. Dendritic cells............................................................................................................. 15 3. Tolerogenic dendritic cells and tumor escape.......................................................... 17 3.1 Generation of tolerogenic dendritic cell ......................................................... 17 3.2. Tolerogenic DCs induce T-cell tolerance........................................................ 25 3.3. Tolerogenic DCs induce the differentiation of T 2 cells or Tregs................ 26 H 4. Malignant melanoma................................................................................................. 27 4.1. Epidemiology and diagnosis ........................................................................... 27 4.2. Melanoma therapies........................................................................................ 28 5. Ret transgenic spontaneous melanoma mouse model ............................................. 33 6. Aims of the study........................................................................................................ 34 II. Materials and methods .........................................................................................35 7. Mice and cell lines ...................................................................................................... 35 7.1. Ret transgenic spontaneous melanoma mouse model................................... 35 7.2. OT-I transgenic mice....................................................................................... 35 7.3. Cell lines ........................................................................................................... 35 8. Chemicals, buffers/medium, antibodies/cytokines.................................................. 36 8.1. Chemicals ......................................................................................................... 36 8.2. Buffers / medium ............................................................................................. 38 8.3. Medium ............................................................................................................ 40 2.4. Antibodies 41 2.5. Cytokines.......................................................................................................... 41 8.6. Other reagents ................................................................................................. 42 9. Reagent kits ................................................................................................................ 42 9.1 MACS kit 42 9.2. ELISA kit 42 9.3. 3,3',5,5' tetramethylbenzidine (TMB) substrate reagent kit....................... 42 9.4. Luminex cytokine assay.................................................................................. 43 9.5. Immunhistochemistry ..................................................................................... 43 10. Routine laboratory materials 43 10.1. Devices............................................................................................................ 43 10.2. Routine laboratory materials ....................................................................... 45 11. Softwares for data analysis...................................................................................... 46 12. Methods..................................................................................................................... 46 4 Index 12.1. Genotypization of ret transgenic mice ......................................................... 46 12.2. Preparation of single cell suspension from mouse organs ......................... 47 12.3. Flow cytometry .............................................................................................. 48 12.4. Bone marrow-derived DC culture ............................................................... 49 12.5. Isolation of DCs from spleen ........................................................................ 49 12.6. DC cytokine secretion ................................................................................... 50 12.7. T-cell priming and restimulation.................................................................. 50 12.8. ELISA............................................................................................................. 51 12.9. IFN-γ ELISPOT............................................................................................. 51 12.10. Reverse transcription (RT)-PCR 52 III. Results ..................................................................................................................54 13. Genotypization and tumor development of ret transgenic mice .......................... 54 14. Phenotype analysis of dendritic cells in lymphoid organs of ret transgenic mice55 14.1. Analysis of total DC numbers....................................................................... 55 14.2. Analysis of DC maturation status in spleen, BM and lymph nodes of ret transgenic mice ....................................................................................................... 57 15. Phenotype analysis of tumor infiltrating DCs ....................................................... 57 16. Functional assay of dendritic cells of ret transgenic mice .................................... 59 16.1. Cytokine profile of DCs ................................................................................ 59 16.2. T cell stimulation capacity of DCs ............................................................... 61 17. Mechanisms of tolerogenic DC generation in ret transgenic mice....................... 62 17.1. Generation of tolerogenic DCs from BM precursors of ret transgenic mice in vitro...................................................................................................................... 62 17.2. Production of immunosuppressive cytokines and growth factors in tumor bearing mice in vivo and by Ret melanoma cells in vitro .................................... 64 17.3. p38 MAPK is strongly activated in DCs of ret transgenic mice with macroscopical tumors ............................................................................................ 67 17.4. Inhibition of p38 MAPK activity normalized functions of DCs from tumor bearing ret transgenic mice.................................................................................... 68 IV. Discussion..............................................................................................................71 18. Reduction of DCs in lymphoid organs and accumulation of immature DCs in primary tumors of ret transgenic mice......................................................................... 71 19. Tolerogenic cytokine profile and impaired T cell stimulation.............................. 73 20. Activation of p38 MAPK drives DCs to display tolerogenic pattern during melanoma development................................................................................................. 75 21. Summary................................................................................................................... 77 V. References: .............................................................................................................78 VI. Abbreviations:......................................................................................................96 Acknowledgement:.....................................................................................................99 5 Summary Summary To investigate mechanisms of the dendritic cell (DC) dysfunction during tumor progression, a transgenic murine model of spontaneous melanoma was used. Ret transgenic mice overexpress the human proto-oncogene ret in melanin containing cells and develop skin malignant melanoma which closely resembles human melanoma with respect to tumor genetics, histopathology and clinical development. + +Numbers of total DCs (MHCII CD11c cells) and mature DCs (DCs expressing CD40, CD80, or CD86) were found to be significantly decreased in the spleen, lymph nodes and bone marrow of tumor bearing mice as compared to ret transgenic tumor free or wild type mice (control groups). Moreover, tumors recruited more DCs during progression but the tumor infiltrating DCs were blocked at the immature stage. After stimulation in vitro, ex vivo isolated DCs or those generated from bone marrow precursors from tumor bearing mice produced significantly more IL-10 and less IL-12 than DCs from control mice, showing a tolerogenic cytokine pattern. DCs +from tumor bearing mice displayed also significantly less capacity to stimulate CD8 T cells to produce IFN-γ. Therefore, the phenotype and function of DCs in ret transgenic mice showed the characteristics of tolerogenic DCs. Melanoma-derived factors in ret transgenic mice were demonstrated to be involved in the acquisition of tolerogenic properties, since DCs generated from bone marrow precursors in medium supplemented with mouse melanoma cell conditioned medium produced significantly less IL-12. Moreover, when activity of VEGF, IL-6, or TGF-β was blocked with the respective neutralizing antibodies, IL-12 production by DCs was significantly upregulated. The p38 mitogen-activated protein kinase (MAPK) can be activated by different tumor-derived factors. A considerably elevated expression of the phosphorylated form of p38 MAPK was detected in DCs from tumor bearing mice. Suppression of p38 MAPK activity in DCs from tumor bearing mice in vitro was found to lead to normalization of their cytokine expression pattern and T-cell stimulation capacity. Taken together, constitutive activation of p38 MAPK is responsible for turning of DCs 6 Summary to display a tolerogenic profile in the process of melanoma development. We have demonstrated that suppression of the p38 MAPK activity in DCs from ret tumor bearing mice can reconstitute their impaired cytokine secretion pattern and ability to stimulate T cells suggesting thereby that such normalization of signaling pathways in DCs could represent an effective immunotherapeutic strategy in melanoma patients. 7 Introduction I. Introduction 1. Immune system and antitumor immune response The immune system defends host against infection. Serving as the first line of defense, innate immunity is essential for the control of common bacterial infections. However, it lacks the ability to recognize certain pathogens and to provide the specific protective immunity that prevents reinfection. In the adaptive immune response, lymphocytes, which express diverse antigen-specific receptors, enable the immune system to recognize any foreign antigen. Besides eliminating pathogens, the adaptive immune response can generate increased numbers of memory lymphocytes, which allow a more rapid and effective reaction upon reinfection. 1.1. Innate immunity Phagocytes The phagocytic cells of the immune system include macrophages, neutrophils, and dendritic cells (DCs). Macrophages are large phagocytic leukocytes, which are able to move outside of the vascular system by moving across the cell membrane of capillary vessels and entering the areas between cells in pursuit of invading pathogens. In tissues, organ-specific macrophages are differentiated from phagocytic cells present in the peripheral blood called monocytes. Macrophages are the most efficient phagocytes, and can phagocytose substantial numbers of bacteria or other cells or microbes. The binding of bacterial molecules to receptors on the surface of a macrophage triggers it to engulf and destroy the bacteria through the generation of a "respiratory burst", causing the release of reactive oxygen species. Pathogens also stimulate macrophages to produce chemokines, which summon other cells to the site of infection. Neutrophils eosinophils and basophils are known as granulocytes due to the 8 Introduction presence of granules in their cytoplasm, or as polymorphonuclear cells due to their distinctive lobed nuclei. Neutrophil granules contain a variety of toxic substances that kill or inhibit growth of bacteria and fungi. Similar to macrophages, neutrophils attack pathogens by activating a respiratory burst. DCs are phagocytic cells locate in tissues that are in contact with the external environment, mainly in the skin (where they are often called Langerhans cells), and in the inner mucosal lining of the nose, lungs, stomach and intestines. They are named for their resemblance to neuronal dendrites, but DCs are not connected to the nervous system. DCs are very important in the process of antigen presentation, and serve as a link between the innate and adaptive immune systems. Natural killer (NK) cells As a component of the innate immune system, NK cells attack host cells that have been infected by microbes, but do not directly attack invading microbes. For example, NK cells attack and destroy tumor and virus-infected cells through a process known as "missing-self" (1). This term describes cells with low level expression of major histocompatibility complex (MHC) class I, a situation that can arise in viral infections of host cells. They were named "natural killer" because of the initial notion that they do not require activation in order to kill cells that are "missing self." Mast cells Mast cells reside in the connective tissue and mucous membranes, and are intimately associated with defense against pathogens, wound healing, but are also often associated with allergy and anaphylaxis (2). When activated, mast cells rapidly release characteristic granules, rich in histamine and heparin, along with various hormonal mediators, and chemokines, or chemotactic cytokines into the environment. 9 Introduction Basophils and eosinophils Basophils and eosinophils are cells related to the neutrophils. When activated by a pathogen encounter, basophils releasing histamine is important in defense against parasites, and plays a role in allergic reactions such as asthma. Upon activation, eosinophils secrete a range of highly toxic proteins and free radicals that are highly effective in killing bacteria and parasites, but are also responsible for tissue damage occurring during allergic reactions. γδ T-cells Like other 'nonconventional' T-cell subsets bearing invariant T-cell receptors (TCRs) such as CD1d-restricted NK T cells, γδ T cells exhibit characteristics that place them at the border between innate and adaptive immunity. On one hand, γδ T cells may be considered as a component of adaptive immunity since they rearrange TCR genes to produce junctional diversity and develop a memory phenotype. On the other hand, they may function as a part of the innate immune system where a restricted T-cell or NK receptors may be used as a pattern recognition receptor. Complement The complement system is a biochemical cascade of the immune system that helps or "complements" the ability of antibodies to clear pathogens or mark them for destruction by other cells. The cascade is composed of many plasma proteins, synthesized in the liver, primarily by hepatocytes. The proteins work together to 1) trigger the recruitment of inflammatory cells; 2) "tag" pathogens for destruction by other cells via opsonizing the surface of the pathogen; 3) disrupt the plasma membrane of an infected cell, resulting in cytolysis of the infected cell, causing the death of the pathogen; 4) rid the body of neutralized antigen-antibody complexes. 10
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A long time ago, in a galaxy far away, I worked for a little Web start-up called Britannica.com Education. We had a live-work loft South of Market, we spent lots of money, we got stock options, and then the bubble burst.
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Tips for Laguna Playhouse
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Parking is tricky. Very crowded area. Get there early to find parking.
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Ask for a lide and you can bring your beverage into the theater.
One of the best plays I've ever seen! Very funny...
Ditto great performances


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In a little olive oil fry the diced onion, diced garlic and diced celery until softened than add the white wine and bring to the boil. Allow simmering for 2 minutes and then add the mussels, cover and braise for about 6 minutes. Add the cream and quartered cherry tomatoes, simmer for 5 min remove from heat and add the chopped and fresh parsley. Cut 2 slices of baguette diagonally and grill. Chop finely the basil mix well with the mayonnaise and put some of the mixture onto each slice of toasted bread. Next, pan-fry the prawns with some garlic oil, wrap in Parma ham and put on 3 on each stick. Serve on the side with the mussel stew.
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Nearly 40,000 chickens were culled by the Bureau of Animal Industry (BAI) in an egg-producing farm in San Luis, Pampanga after detecting AH5N6, a strain of highly pathogenic avian influenza better known as the bird flu virus.
In its statement yesterday, the BAI said that their team have "humanely culled a total of 38,701 head of layers [egg-laying chickens] and disposed of them properly, employing the protocols under the Avian Influenza Protection Program of the Philippine Government."
Read: Duterte, former president Arroyo, eat chicken from bird flu-affected areas to show it's safe
DA-BAI attributed the emergence of bird flu in the farm from migratory birds flying into the area. The Environmental Department cited that some 150,000 migratory birds nest and breed annually in Central Luzon alone.
Bird flu can be passed on to humans and can be spread through the air, manure, and other types of contact.
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Pampanga, previously the epicenter of avian influenza in the country, was placed under a state of calamity in 2017 after suffering a bird flu outbreak, with the first cases appearing in San Luis.
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Coronavirus outbreak: Will fix price of COVID-19 treatment in private hospitals soon, says Haryana health minister Anil Vij
Haryana health minister Anil Vij said that he had discussed the modalities with senior officials of his department and a formal order would be issued soon.
Chandigarh: The Haryana government will soon issue an order fixing the cost of COVID-19 treatment in private hospitals, Health Minister Anil Vij said on Tuesday.
Vij said that he had discussed the modalities with senior officials of his department and a formal order would be issued soon.
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On the rise in the number of coronavirus cases in Haryana this month, Vij said that they were expecting infections to rise once borders were opened and more relaxations given leading to free movement of people.
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Asked if there is a possibility of reimposition of stricter measures like sealing of borders with Delhi if the cases continue to rise, Vij said, "We are keeping an eye, but as of now there is no such move."
Amid rising number of COVID-19 cases, the Haryana government had said 1,106 final-year MBBS students studying in government and private medical colleges would be deployed for effective management of the pandemic.
We have come up with a protocol for these students and it has been decided that they will not be put on hard duties. We will deploy them in carrying out surveys, maintaining containment zones, sample collection etc. We need more manpower in the hospitals and this step will help spare our doctors and other health professionals who otherwise have to manage these duties as well, said Vij.
He said that final-year B.Sc and M.Sc nursing students will also assist in effective management of the pandemic and the chief medical officers of districts will use their services accordingly.
Vij further said that over 600 doctors had been recruited recently, of whom 350 have already joined.
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30 Sep 2017 19:38
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Leadership And Collaboration The Key For Hamilton City Council's Climate Change Action Plan
Hamilton City Council's Climate Change Action Plan will include a city-wide partnership between businesses, organisations and community groups to tackle how our city responds to climate change.
Proposals for the Action Plan were outlined at today's Council meeting, including the formation of a Hamilton Climate Change Accord.
The Accord will see the Council partner with stakeholders across Hamilton to reduce carbon emissions generated within the city.
In developing the Action Plan, the Council commissioned a breakdown of the 982,284 tCO2e (tonnes of carbon dioxide equivalents) of emissions produced across Hamilton.
Land transport is the city's largest source of emissions with 62% (619,723 tCO2e), followed by energy used for industry and in homes (29%, 276,594 tCO2e).
The intent of the Accord is for the city to work together and share the load in addressing the challenges of climate change, especially those posed by transport and energy use.
The Chair of the Council's Environment Committee, Councillor Margaret Forsyth, said: "The Climate Change Accord is the key thing here. Not only can [the Council] be leading in what we're doing as an organisation, but by bringing stakeholders together, big and small, and working together as one … there is a leadership opportunity for us and a collaborative opportunity."
The Action Plan also proposes targets and actions for the Council as an organisation to reduce its carbon emissions.
Between July 2018 and June 2019, the Council generated 11,033 tCO2e, with the main contributors being fuel and natural gas (35.6%), electricity (30.8%) and biosolids to vermi-composting (22.2%).
The Action Plan seeks to reduce the Council's emissions by 50% by 2030 and to have net zero carbon emissions by 2050.
Within the Council's fuel and natural gas emissions, 65% is from the use of natural gas at the wastewater treatment plant to generate electricity at peak times, boilers to heat the pools at Waterworld and Gallagher Aquatic Centre, and for gas appliances at our various facilities.
"It's really important that we understand our baseline and our foundation – where we are starting from and what we are currently doing," said Cr Forsyth. "I often say, 'in order to go fast, you've got to go slow'. So, the time staff have spent gathering that baseline data is really important."
Options have been identified to achieve a 21% reduction, starting with those which address the main sources of emissions. These include:
- installing a biogas-fired engine at the city's wastewater treatment plant to reduce the natural gas used to generate electricity at peak times
- replacing the gas boilers at Waterworld with heaters that emit less carbon such as electric pumps
- improving the fuel efficiency of the vehicles and tools used by Council
- continuing the electricity efficiency programme for the city's street lights and Council buildings
- reducing the waste to landfill from Council sites.
A further 22% reduction in emissions by 2030 is anticipated to be achieved through the Government's commitment to reducing the use of coal and natural gas for electricity generation.
The remaining 7% of the 50% reduction target will come from projects identified between now and 2030.
The draft Climate Change Action Plan will be presented to the Environment Committee meeting in June 2020.
Click here to read the progress report on the Climate Change Action Plan as part of today's meeting agenda.


text:
100 greatest sales letters. Another big dairy market ice cream has seen stagnant sales. My dear girl i love you ever and ever without reserve. Benjamin franklin taylor swift and more. And the venerable kemps brand has been flipped between four corporate.
Watchmen is a science fiction american comic book limited series by the british creative team of writer alan moore artist dave gibbons and colorist john higginsit was published by dc comics in 1986 and 1987 and collected in a single volume edition in 1987. The 100 greatest headlines ever written whenever i need to write a drop dead kick butt stop em dead in their tracks type of headline i usually always first take a look at my list of the 100 greatest headlines. The 100 greatest pennsylvanians of all time ranked. Pennsylvania life culture.
The greatest sales letters of all time understand the rules to writing a great sales letter and you possess the secret to unlimited wealth. 19 complete website sales letter templates with fully customizable homepages graphics alternative headlines opt in offers guarantee pages and full website navigation plan. The greatest direct mail sales letters of all time. Nostalgiaavgnfan via youtube goldman sachs former executive greg smiths op ed piece in the nyt on wednesday caused more than a stir but hes not the only person to publicly make a fuss when.
Music film tv and political news coverage. Watchmen originated from a story proposal moore submitted to dc featuring superhero characters that the company had acquired from.


text:
Find the two most distant points
1813 Solvers
Pattern matching
1954 Solvers
Find Rotated Substring
102 Solvers
Circumscribed circles
301 Solvers
Derivative of polynomial
210 Solvers
old wine in a new bottle?
19 Solvers
resistor ladder
23 Solvers
Calories in a slice of pizza?
49 Solvers
polar inertia
35 Solvers
speed vs size
24 Solvers
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150 exhibitors from 19 countries discuss business with 2,800 professionals
On June 19th, the newly renovated International Fairgrounds of Casablanca opens its gates for the 5th edition of plast expo. Until June 22, 150 exhibitors from 19 countries present their innovations for the plastics and packaging industry. The organisers expect an audience of 2,800 professional trade visitors during the four days of the exhibition. plast expo is highlighted by a strong presence of Morocco as host country as well as Spain as Guest of Honor and further participation from Qatar, Italy, France, Turkey, Portugal and Germany. This business-to-business event with a clear focus on quality attendees is organised by the Moroccan Plastics Federation FMP, Forum 7 and their German partner fairtrade Messe from Heidelberg. The event is accompanied by a 3-day conference program.
"plast expo 2013 takes place against the backdrop of massive investments into the North-African plastics and packaging industry and a huge increase in demand for plastics and chemical products", says Nabil Saouaf, director of FMP. Saouaf further: "The participation of 150 exhibitors from 19 countries mirrors the importance of this international trade show for the whole MENA region."
By far the largest participation of course comes out of the host country Morocco. With 52 exhibitors Moroccan companies head the ranking. Spain has been appointed as Guest of Honor of plast expo 2013. On this occasion 12 Spanish exhibitors will present their innovations in plastics and packaging, in the frame of the Spanish pavilion organised by AVEP. Italy will also leave a heavy footprint at plast expo 2013 as 14 Italian exhibitors will be present on 150 sqm. The Italian group is supported by PROMOS of Milan. France is present with 14 exhibitors. For the very first time, 13 Qatari plastic product exporters join the Qatar Development Bank (QDB)"s export development agency Tasdeer at plast expo 2013. Qatar participates with an exclusive pavilion covering 144 sqm. Turkey participates with 7 leaders in plastics machinery and Germany and Portugal each with 6 companies.
Towards the end of the online pre-registration for trade visitors it is already becoming apparent, that the exhibitors can expect a highly professional visitorship. Until now, more than 800 visitors from 29 countries announced their visit at this business-to-business event with a clear focus on quality attendees. And the majority amongst them are decision makers.
fairtrade Messe und Ausstellungs GmbH & Co KG
Seit ihrer Gründung 1991 zählt die fairtrade Messegesellschaft zu den führenden Veranstaltern internationaler Fachmessen in den aufstrebenden Märkten Osteuropas, des Nahen und Mittleren Ostens und Nordafrikas. Als inhaber-geführtes Unternehmen mit Sitz in Heidelberg verfügt fairtrade über ein weltweites Netz von Partnern und Repräsentanten. fairtrade ist ISO 9001:2008 zertifiziert. Als UFI-Mitglied organisiert fairtrade Messen nach den UFI Qualitätsnormen.
About fairtrade:
fairtrade ranks among the leading organizers of professional international trade fairs in the emerging markets of Eastern Europe, the Middle East, Asia and North Africa ever since its foundation in 1991. Headquartered in Heidelberg, Germany, fairtrade maintains a powerful network of agencies and partnerships throughout the world while focusing on achieving an ever higher degree of customer satisfaction, providing excellent service and generating valuable business contacts for exhibitors and trade visitors alike. ISO 9001:2008 certified and as a member of UFI The Global Association of the Exhibition Industry, fairtrade organizes events according to the UFI quality norms.
fairtrade Presse
Dominik Rzepka
Kurfuersten Anlage 36
69115 Heidelberg
+49 6221 456522
fairtrade Messe
Dominik Rzepka
Kurfürsten Anlage 36
69115 Heidelberg
+49 6221 456522


text:
Chapter 8
Herman Borman adjusted a knob below the green screen to widen the view. His wizened hands moved deftly over the controls as the limo grumbled onwards."Steven, do not be too fast. Let us be careful this time."
Borman wiped his chin of a thin line of spittle and resumed staring at the screen. The driver silently replied with an emphatic nod, and a quick sideways glance at Paul, sitting next to him. Still wearing a police uniform that served its purpose at the Schill incident, Borman could tell that Paul was decidedly unhappy. His preference to wear it again plainly gave away their arrival at the museum. That did not sit well with Borman, and things that didn't sit well with him eventually had a way of disappearing. Paul shot a nervous look over his shoulder and saw the old man huddled over his tracking device, face outlined in goblin green and seemingly indifferent to the world. He exhaled, trying not to make it too obvious, and did not notice Mr. Borman's sneer. Feigning an ignorance of Paul's backward glance, Borman knew that Paul was nervous. Borman spoke to the driver. "They are at the mine office. We do not want to spook them, so move ahead as quietly as you can. When we reach the office you and Paul will check the area. By this, I mean keep out of sight, and keep in touch with me. There may be more people involved. Once you have ascertained the situation, I will tell you what to do," And then, as if to himself, he continued in a whisper, which gradually increased in volume. "I want that medallion and I want the object Hedda has. I want both artifacts, and I do not care what it takes. Verstehen Sie mir? Are you understanding me?"
Steven and Paul nodded enthusiastically in an odd unison that could be comical under other circumstances. Borman had a habit of reverting to German when he became excited. They had been involved in similar activities for some time now. Tracking, hunting, retrieving. He had explained to them that he was a collector of rare artifacts, with plenty of money to finance retrieval operations. For this job, he was a bit more animated than usual, breaking with tradition to actively join in the hunt. They reached the outer gate.
Linda was the first to see it and tugged at Adam's arm. Just ahead, beyond a stand of overgrown forsythias, she made out the curved stone edges of a mine entrance at the base of a mound which blocked the view of the river beyond. The two approached, moving off the main trail and with an unspoken understanding more typical of a coordinated assault, adopted a flanking maneuver using the overgrowth as a cover. The remains of rusty iron rails protruded from the muddy trail and wound through the foliage into the bottom of a faded wooden wall inset a few feet into the mine entrance. Centered on the wall was a boarded up door announcing that the mine was closed, that there were dangers within and for any wayward trespassers to 'KEEP OUT'.
As they neared the blockaded entrance, Adam pointed at the base of the door. "Check out the footprints."
Between two nearly buried rails leading into the mine entrance, shoeprints in the dirt led to the door. Although such signs could be due a variety of curiosity seekers, one such print had a feature of singular interest. Its heel was visible while its toe disappeared under the wooden boards. This anomaly drew the two closer in for a more careful inspection. Adam gave the doorknob a twist and a pull, to no effect. While Linda felt around the edges of the door, Adam took a step back to take in a broader view. The wall consisted of unpainted planks, gray with years of oxidation. The coloring was uniform from top to bottom, except for a couple of small, nearly circular patches near the door. As Adam reached forward to press down on one of the marks, the board beneath gave in just a little. Linda joined him, pushing down on the other. A faint click sounded and the door swung outward along with its collage of warning placards. A plume of damp, cold air rolled over them. Instead of darkness within, they saw a string of light bulbs glaring along the roof of the tunnel and disappearing into the distance as they curved downward. Glistening walls and puddles caught between rails and rock debris scattered along the passageway completed the scene. They pulled the door closed behind them, careful to avoid making too much noise. Once inside the tunnel, they heard the sound of dripping, water seeping into the mine from countless cracks and crevices.
Linda folded her arms."Ooo, it's cold in here."
"Here, take this." Adam offered up his windbreaker which Linda gratefully accepted, wrapping it over her shoulders. He instinctively placed his arm around her waist and for a fleeting moment their eyes met, acknowledging their mutual anxiety and perhaps, more. They proceeded onward through the ragged circles of light, careful to step over the remnants of rails embedded in the floor. They felt the subtle tug of gravity with each step as the tunnel sloped downward. The rails once carried the coal cars or 'buggies' as they used to call them years ago. Rotting timbers running up either side of the wall threatened to crumble beneath their sagging crossbeams. Jagged outlines of blue-black rock reached out to them as they passed beneath hanging incandescents. The air they breathed carried a heavy dampness—a ghostly link to the miners who once worked the coal, perhaps to the miners who died here and were never recovered. There were several openings to either side of the tunnel, possibly marking entrances to side passageways. As they passed one such narrow excavation, something that might have once been a coal chute, they heard voices, distant and muffled, sounds that mixed with those of gurgling water. Intermittent splashes suggested vermin and other creatures of the dark scattering before them as they plodded onward, deeper into the mine. After a few minutes they could no longer make out the entrance, and instead, concentrated on what lay ahead.
Linda tugged Adam's elbow. "Do you hear that?"
They listened, trying to filter out the watery background sounds in the dank, half-lit tunnel. It was music, slow dance music—an old Sinatra, or perhaps a Nat King Cole croon, waxing and waning. The slow song, the tempo and undulating volume, mixed in with the dull echoes of trickling water and their own steps. As they neared the source, a subtle clicking brought up an image of a hand-wound record player to Adam. He recalled his father fussing with an conical megaphone contraption, probably an old Victrola, with a picture of an attentive dog pasted on the side. They picked up their pace, carefully placing each step to avoid announcing their arrival. It was almost as if the melancholy sounds emanating from the distance were calling out to them, drawing them nearer like sirens to rocky shoals. They reached what at first looked like a dead-end, a rock fall or cave-in. The broken rails continued on under a pile of coal and debris—a volcanic isle on a dark, shimmering ocean puddle. A second look made it clear that the tunnel turned to the left, and as they followed the curve, they found fresh timbers. A hundred yards or so ahead a sharp band of light from a side entrance stretched across the path. The crooning was louder.
Mona Lisa by Nat King Cole.
While staring ahead, Adam failed to notice a discarded bottle near his feet. His toe kicked it ahead, producing a loud and disquieting pinging as it tumbled across the floor. The noise may have been barely noticeable in the outside world, but here, in Nature's echo chamber, it was a clarion call. A moment later all the lights went out and the pair froze in their tracks. The music stopped. Seconds crawled by as they wondered what would happen next.
Then Adam ventured a call out. "Hedda, is that you?"
No response. Adam tried again. "It's us, Adam and Linda. We came down here to look for you."
A spotlight flashed on at the far end of the tunnel. A moment later, a familiar voice echoed through the tunnel. "Well, I guess ya found me. Hold on, I'll bring up the lights."
The overhead lights returned and Hedda emerged from the side shaft to greet them. "I thought I told ya to wait for me."
Linda said, "We started to get worried."
Hedda shook her head. "Well, I guess I'd be bringing ya here anyhow. Come with me, I got something to show ya."
They followed Hedda around the corner and into the shaft from where the music had been coming. The mine shaft led to a room with white-washed wooden walls and planked flooring. They felt warmth emanating from the room, cutting through the damp chill. There was a desk covered with notes and books, some chairs, cabinets, even a couple of easy chairs. In one corner Adam spotted a beaten up old desktop computer, and the grim lines of his mouth upturned into a smile, when in the opposite corner, he saw a bona fide Victrola.
"Have a seat. I think ya'll need to be sittin'," Hedda remarked as if to herself, and pointed to a pair of worn out and over-stuffed easy chairs. She slid onto one of the desk chairs. They all stared at one another for a few moments, as if waiting for something to happen.
Then Hedda sighed and turned her head to the paneled wall. "Ya'll can come out now."
A panel section swung open with a creak.. A white-haired man stepped through, stooped over and head bowed. As the door reseated itself behind him, he raised his head and smiled from ear to ear.
"Adam, my boy, what a pleasant surprise!"
Adam's neck hair stood at attention. Standing in front of him was a dead man, Dr. Ben Wujciak. The impossibility of it left Adam breathless. The man he saw before him looked exactly like the Ben Wujciak he remembered seeing back in New Jersey, perhaps with a bit more color and certainly more lively. He frantically looked to Linda, who returned only a blank stare, oblivious to the specter standing before them. With both hands clutching the arms of his chair, providing much needed support, Adam managed at last to cough out a response.
"But … but, how can this be? Are you really Dr. Ben Wujciak?"
The old man replied with a perplexed look. "Of course! Who else would I be?"
The VW in the parking lot.
Still dazed by the shock, Adam arose on trembling legs and walked over to Ben. He stared at his old family friend for a few seconds and then the two embraced. "My God. Ben, I thought you were dead. It's incredible that you're alive and here in this place."
Adam's eyes were misty as the pair unclenched. When he sat down, he unconsciously flashed the splayed-finger Vulcan greeting to his old friend.
Ben smiled and asked, "And who is this remarkably beautiful girl?"
No return greeting?
Too excited to press Ben further, Adam responded, "This is Linda. Dr. Linda Garcia. She's from the university. We're working on a DNA project."
Adam's mouth became dry and his tongue seemed tied in knots, unclear as to what to say next. He eased back in his chair and studied Ben. As he pondered, his smile faded to a thin, determined line. He needed to get past the normal niceties and drive the conversation toward enlightenment. He noticed Linda slowly shaking her head. The incongruity of the situation was sinking in.
Ben asked, "Is there a problem? It looks as if you two just saw a ghost."
Adam said, "Ghost is about right. Ben, do you remember my last office visit. Do you remember what I brought with me?"
The old man put both hands on his hips. "Well, it's been a long time."
"What was it that I showed you?"
With a shrug, as if explaining something very simple to a recalcitrant child, Ben answered, "Adam, my dear boy. I remember it quite well. It must be the reason you are here. It's the reason I am here." And after a very pregnant pause, he added, "It's the object you found in a lump of coal."
Adam slumped back into his chair. "But you're dead. You can't be here. How could you be alive?"
Ben cocked his head and responded, "Relax, Adam. I'm fine. Tell me why you think I'm dead."
Adam related the events of the past couple of days—the analysis of his medallion, the explosion, the note from Ben, and the fateful and extremely odd findings in Maplewood which eventually led him and Linda to Hedda and the museum. When Adam finished, Ben took a seat in one of the wooden desk chairs, clamped his hands across the back of his head, and appeared to be trying to absorb all the details of Adam's story, including those of his apparent death. His eyes closed and a deep silence settled amid them.
After a minute or so, Ben raised his head and said, "Adam, do you have the medallion with you?"
Ben's smile abruptly disappeared as at the same moment they all heard a thud in the tunnel, then another, and another. Hedda ran out of the room to investigate, returning moments later to shut off the tunnel lights. She brought her fingers to her lips and whispered, "There's someone trying to break in. They're at the front entrance."
The entry door caved in with a crash. Adam and Linda jumped. They were trapped in a dead-end mine.
Ben used a steady, calm voice. "Someone's followed you here, probably the gentlemen chasing you from the museum."
Hedda joined Ben as he rose from his chair and added, "No need to panic. Just follow us."
Ben led the group to the hidden door and swung it open.
"A secret passageway?" Adam inquired as they entered.
Ben turned his head. "Nothing so fancy. You're in what used to be the hospital section of the mine, and this is just another way out."
Rubbing his hands along the paneling, Ben continued," I put this panel in myself, so I suppose now it's a 'secret passageway', as you call it."
Hedda secured the door behind them and flicked on a light switch while Ben placed a heavy metal cross-brace into position against the closed panel. Unlike the old wood used at the entrance, the panel and wall were built with oversized beams that would be able to withstand any attempt at breaching. They found themselves in a small room with rough rock walls and an exit at the opposite end.
Ben tried calming the group. "If we can stay quiet, maybe we can wait here a bit and hear what these intruders have to say. It would be nice to know who they are, or at least what they're after."
It only took a few minutes before they heard the arrival of the uninvited duo.
"Paul, shine the light over here."
Desks and chairs were moved about.
"I found the switch. There's nobody here, Steve. Borman's gonna flip."
"You sure they came this way?"
"You saw the foot prints."
"Let's give this place a good overhaul, don't leave nothin' standin'. He'll be pissed off 'cause we came back empty-handed, but I don't want him jerkin' us around 'cause we weren't thorough."
The sounds beyond the door painted a vivid picture—furniture scraped across the uneven flooring, drawers were pulled and emptied, chairs fell over and cabinet doors slammed.
"Get a load of the record player."
"Leave that be."
"What about the computer?"
"Yeah, let's take it with us. They ain't here which means they're someplace else."
"There's nobody asleep in that little noggin' of yours."
"What's that?"
Computer equipment cables dragged across a table top. And from the doorway, "Paul, check out that chute up ahead. They could've gone up in there. Maybe there's another way out."
"Shit, Steve, you gotta be kiddin'. Nobody's gone up that chute. Besides, I'm gonna get my uniform dirty."
"Aw, shaddup. Check it out. Borman's not too happy with you, so you better show some initiative."
The voices faded. Hedda spoke up. "We know Borman. I know Borman. That asshole were Luke's boss long time ago. A few years ago he bought this mine. We made an arrangement to do some diggin' here. But that doesn't change anything. He's still an asshole."
Linda tilted her head and asked, "Did this guy know about the stuff Luke found?"
"Oh, he sure did. He were gonna take care of the find in the mornin'. The same mornin' the mines got flooded."
"Do you know if he got hold of the other metal pieces before the flood?" asked Adam.
"Don't rightly know, but I wouldn't be surprised if he did."
Adam turned to Ben. "Was there anything important on that computer they took? And, by the way, just what are you doing down here? If you really are Ben Wujciak, who did I see back in Maplewood?"
Adam could not let it go, and almost as an afterthought, and to no one in particular, he mumbled, "This is crazy."
Linda brought her arm around Adam's shoulders and they both looked to Ben with expectation dappled across their faces. The shadows brought on by the single light bulb directly above Ben added an eerie touch of drama, deepening Ben's eye sockets and chiseling his stubbled chin. When Ben spoke, the shadows danced upon his face as the most startling of revelations were about to be articulated, from the incredible to the inconceivable.