system
Given the following post, respond as if you're commenting on Reddit:
human
Since insulin regulates the production of ketones, what prevents Ketoacidosis when a Type 1 Diabetic attempts a ketogenic diet?

When a Type 1 Diabetic attempts to follow an extremely low carb diet they would have to take close to no insulin or else they would risk facing repeated low blood sugar levels which then would force them to eat carbohydrates to raise, making the diet no longer low carb. When insulin levels are cut too far down from a low carb diet, why doesn’t ketoacidosis occur? Do Type 1 Diabetics on keto produce more ketones because of this? And if so, does that mean that they can shred excess fat at a more effective rate than someone without the disease?
gpt
The metabolic pathway for ketone production is also regulated via homeostatic methods further down the biophysical pathway plus its not guaranteed that the pathway for ketone production is active. Fatty oxidation is primary, insulins effect on fatty oxidation is secondary or tertiary & only has a slight effect on the overall fatty oxidation process.