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The role of protein kinase C in myeloid cell apoptosis.

Unfortunately, we have just a correlation at present with no clear answer. It may well be that downstream elements of the bcl-2 signalling pathway are not functioning in this cell line. In this context it will be interesting to look at Bax levels since bcl-2 and bax are known to interact in the regulation of apoptosis. There may also be some other explanations as to why we see no correlation between bcl-2 expression and inhibition of apoptosis. One additional point of interest about HI,-60 cells is that they lack a functioning P.53 gene and this gene has been documented as being a promoter of apoptosis. With the lack of functioning p.53 and relatively high levels of bcl-2 one would expect HL-60 cells to be resistant to apoptosis; however, this is not the case! In conclusion, a number of genes have been identified as negative regulators of apoptosis and these include both bcl-2 and abl. However, the mechanism by which these cells exert their effects is still largely unknown.