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TRAX Provides Neuroprotection for Huntington's Disease Via Modulating a Novel Subset of MicroRNAs

Huntington's disease (HD) is a neurodegenerative disease caused by CAG‐repeat expansions (>36) in exon 1 of HTT, which dysregulates multiple cellular machineries. Translin‐associated protein X (TRAX) is a scaffold protein with diverse functions, including suppressing the microRNA (miRNA)‐mediated silencing by degrading pre‐miRNA. To date, the role of TRAX in neurodegenerative diseases remains unknown.