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Normal pregnancy following first‐trimester uterine rupture

recessive autosomal manner5. In particular, a single defect of a gene located on chromosome 5 or 11 has been postulated to cause the syndrome, after a reciprocal translocation was found in a girl having the syndrome5. The Klippel–Trenaunay–Weber syndrome has in general a benign course. In most cases the hemangiomatous lesions tend to regress in the neonatal period4. However, if the hemangiomata are very extensive, in utero heart failure and severe postnatal sequelae may occur, and if visceral organs are involved there is higher than normal morbidity due to internal bleeding5,6. Concerning prenatal counseling, parents should be informed about the inheritance pattern of the syndrome, particularly that most cases occur sporadically, that there is no known association with major chromosomal anomalies and that most cases have a benign postnatal course. If the image of multiple, dilated, fluid-filled intestinal loops is detected during prenatal ultrasound examination, Klippel–Trenaunay–Weber syndrome with intestinal involvement and in general hemangiomata of the intestine should be considered in the differential diagnosis, besides Hirschsprung’s disease and cystic fibrosis, and other less common causes of intestinal obstruction.