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Hypotheses for fat tissue supercompensation after exercise cessation.

Vol.14, No.1 | 2016 | hypothesis journal.com ABSTRACT General Adaptation Syndrome and the supercompensation model are frequently applied theoretical concepts in exercise science. Metabolic changes in response to an exercise stimulus can promote compensatory effects on different systems, including organic tissues and energetic substrates. It has already been established that specific exercise protocols can promote compensatory effects in muscles, bone mass, and encephalic mass. Curiously, fat tissue has shown the same pattern of compensatory effects when the stressor is liposuction, and to the best of the authors’ knowledge, yet there is no specific investigation about exercise-based stimulus and compensatory effects on fat. Our hypothesis indicates that since fat is an important tissue and the primary energetic substrate for moderate-intensity continuous exercise, this kind of exercise could promote a compensatory and adverse effect like an obesogenic factor, after recovery and detraining. A wide debate should begin about exercise recommendations and the prescription of exercise programs, especially when weight and fat loss are the main goal. This biological response to exercise should be investigated further to determine whether this compensatory effect influences fat oxidation and deposition.