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Characterization of the Cardiovascular Autonomic Dysfunction in an Experimental Model of Intermittent Acute Porphyria

Delta‐aminolevulinic acid (ALA) is an important endogenous prooxidant in the pathophysiology of porphyria. The ALA treatment may allow the elaboration of an experimental model for the study of neuroautonomic manifestations of porphyria. Porphyrias are enzymatic derangements congenital or acquired in the biochemical formation of the heme group. Individuals affected may manifest serious dysautonomic disorders such as cardiac arrhythmia, arterial blood pressure variation and neurogenic pain.