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Multiple bacteria contribute to intraplaque T‐cell activation in atherosclerosis

Background  Infection with microorganisms is considered a pathogenic factor in atherogenesis. Several studies have shown the presence of a broad spectrum of bacterial species in atherosclerotic plaques, which could trigger local inflammation. Because T cells contribute to atherosclerotic plaque inflammation, we studied the responsiveness of human plaque derived T‐cell cultures to bacteria of different species.