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A low renal threshold for glucose in diabetic patients with a mutation in the hepatocyte nuclear factor‐1α (HNF‐1α) gene

One form of maturity‐onset diabetes of the young, Type 3 (MODY3), results from mutations in the gene coding for hepatocyte nuclear factor‐1α (HNF‐1α), a transcription factor first described in the liver. MODY3 is characterized by a defective glucose‐stimulated insulin secretion. Earlier observations of glycosuria with normal blood glucose levels in some MODY families suggest an additional renal manifestation of the respective genetic defect.