{'fraction_non_alphanumeric': 0.02390438247011952, 'fraction_numerical': 0.0026560424966799467, 'mean_word_length': 5.283333333333333, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
The long‐term evolution of mycobacterial BCG and preformed immune complex BCG/anti‐BCG granulomas in rats

In previous studies it was found that infections with BCG or complexed BCG/anti‐BCG in rat skin produced granulomas that appeared to resolve at 8 months. In this follow‐up study 1 year later, it appeared that while lesions due to preformed complexes had resolved, those due to BCG alone had undergone a massive reactivation. Nevertheless despite the loss of CMI the infection was restricted. The bacilli, present in enormously increased numbers, were dead; the host macrophages were large and activated. Epithelioid cells and dendritic cells were common. The granuloma was confluent, with patchy necrosis and inconspicuous polymorph infiltration.