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Effects of Antiepileptic Drugs on Epoxide Hydrolase and Glutathi‐one‐S‐Transferase Activities.

rons during developmental processes in localized portions of the brain. We suggest that there is a deficit of the gene(s) regulating, directly or indirectly, the proliferation or migration or both of the neurons in the hippocampus during the developmental stage in this new mutant. Such microdysgenesis in the central nervous system has also been found in the brains of patients with temporal lobe epilepsy, primary generalized epilepsy, and severe myoclonus epilepsy in infancy. It would be interesting to know whether microdysgenesis in our new epileptic mutant is related to epileptogenesis. Further genetic analysis involving inbreeding is required to clarify the contribution of the microdysgenesis in the hippocampus to the epileptogenesis in this mutant.