{'fraction_non_alphanumeric': 0.058954393770856504, 'fraction_numerical': 0.035595105672969966, 'mean_word_length': 4.8441558441558445, 'pattern_counts': {'":': 0, '<': 3, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Possible Regulation of Autologous Mixed Lymphocyte Reaction by OKM1+ NK Cells

T cells are stimulated by autologous non‐T cells and interleukin 2 (IL‐2) is produced in the conventional autologous mixed lymphocyte reaction (AMLR) in young healthy controls. The role of cells with natural killer (NK) cell markers (OKM1+ cells or Leu 7+ cells) in the AMLR was studied. There were significant inverse correlations between the percentage of input OKM1+ cells minus monocyte (OKM1+ NK cells) and either AMLR proliferation (γ = −0.9, P<0.001) or IL‐2 production (γ = −0.75, P<0.01) in the AMLR cultures after 7 days measured at 7 days. A statistically significant correlation was observed between the percentage of input Leu 7+ cells and AMLR proliferation (γ = −0.64, P<0.05), but not IL‐2 production. These results suggest that the AMLR is controlled by OKM1+ NK, perhaps acting through IL‐2 regulation.