{'fraction_non_alphanumeric': 0.035114503816793895, 'fraction_numerical': 0.021374045801526718, 'mean_word_length': 5.368932038834951, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Deficit of Prepulse Inhibition in Mice Caused by Dietary n‐3 Fatty Acid Deficiencies.

Docosahexaenoic acid (DHA, 22:6n‐3) is the most abundant polyunsaturated fatty acid in the brain and plays an important role in learning and memory. It was shown that profound DHA deficiency led to modification in dopamine and serotonin systems. Prepulse inhibition is deficient in psychotic disorders such as schizophrenia and can be disrupted in animals by dopamine and serotonin agonist administration. DHA and eicosapentaenoic acid (20:5n‐3) are biosynthesized from α‐linolenic acid (LNA, 18:3n‐3), a shorter chain n‐3 fatty acid precursor or obtained with a diet.