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Genetic and functional characterization of an antiserum to the lipid A‐specific triggering receptor on murine B lymphocytes

The inheritance of responsiveness to lipopolysaccharide (LPS), and of a marker recognized in LPS‐reactive cells by a heterologous antiserum, was studied in crosses between C3H/HeJ (nonresponder) and C3H/Tif (high responder) mice. F1 hybrid mice show codominant expression of these traits: (a) LPS‐reactive cells are only half as frequent in the hybrids as in the high responder parent; (b) the serologically defined marker is expressed in half as many cells in the hybrids as in the high responder parent. In backcross generations, both LPS responsiveness and this serological marker segregated into high, intermediate, and nonresponders.