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Mitochondrial fragmentation is a novel feature of adherent‐invasive E. coli evoked pathophysiology in enteric epithelia

Mitochondria are not distinct organelles; they exist as a dynamic network that constantly remodels via the processes of fission and fusion to meet the cells energy demands and allow recycling of damaged mitochondria. Mitochondrial dysfunction has been described in inflammatory bowel disease, where adherent‐invasive E. coli (AIEC) have been described as a pathobiont that can drive epithelial apoptosis. Little is known of microbial regulation of enterocytic mitochondrial dynamics and nothing of the potential of AIEC to perturb the critical metabolic function that mitochondria fulfill for the cell.