{'fraction_non_alphanumeric': 0.01308139534883721, 'fraction_numerical': 0.005813953488372093, 'mean_word_length': 5.754901960784314, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Acute disseminated encephalomyelitis in a boy with elevated plasma levels of thrombin–antithrombin III complex

sidered an immune-mediated demyelinating central nervous system disorder.1 In multiple sclerosis, vascular endothelium is believed to be involved in the initiation of immune reactions2 and the level of thrombomodulin reportedly correlates with the disease severity.3 Endothelial cell damage and activation of the coagulation system have not been studied in relation to the course of relapsing ADEM. We describe a child with ADEM in whom the time course of hyperintensities in T2-weighted magnetic resonance images paralleled plasma levels of thrombin–antithrombin III complex.