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Changes in coagulation‐fibrinolysis marker and neutrophil elastase following the use of tourniquet during total knee arthroplasty and the influence of neutrophil elastase on thromboembolism

Background:  To clarify in detail the mechanism underlying the development and exacerbation of deep venous thrombosis (DVT) and/or pulmonary thromboembolism (PTE), we focused on the following factors: the thrombin‐antithrombin III complex (TAT), D‐dimer and neutrophil elastase (NE). We basically investigated whether NE played an important role in the development of PTE I a mice model.