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Blood Pressure and Catecholamine Responses to Sympathetic Stimulation in Normotensive and Hypertensive Subjects

JT has been established that individuals subjected to stress respond by increase in blood pressure, heart rate, and catecholamine excretion.1 It has been shown for many years that neurovascular reactivity to various stimuli, including chemical substances, is increased in primary hypertension.24 Some investigators have suggested that patients with labile hypertension have a higher blood pressure and catecholamine response to emotional stress than do normotensive subjects.5 However, such exaggerated catecholamine response to emotional stimulus has not been shown in patients with established hypertension.’ Many reports on the pattern of hemodynamic response to sympathetic stimulation among labile hypertensives, sustained hypertensives, and normotensives have given conflicting results.69 Furthermore, information about the concomitant changes in sympathetic activity as judged from plasma catecholamine concentration is missing. The development of sensitive radioenzymatic assays for accurate measurement of plasma catecholamines offers promise in this regard.’0 The present study was undertaken to examine blood pressure and heart rate