{'fraction_non_alphanumeric': 0.03761348897535668, 'fraction_numerical': 0.03372243839169909, 'mean_word_length': 5.327868852459017, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Effects of stroke on local cerebral metabolism and perfusion: Mapping by emission computed tomography of 18FDG and 13NH3

By means of emission computed tomography (ECT), we used 18F‐fluorodeoxyglucose (18FDG) and 13N‐ammonia (13NH3) as indicators of abnormalities in local cerebral glucose utilization (LCMRglc) and relative perfusion, respectively. The ECAT positron tomograph was used to scan normal control subjects and 10 stroke patients at various times during recovery. In normal subjects, mean CMRglc was 5.28 ± 0.76 mg per 100 gm tissue per minute (mean ± SD; N = 8). In patients with stroke, mean CMRglc in the contralateral hemisphere was moderately decreased during the first week, profoundly depressed in irreversible coma, and normal after clinical recovery.