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Effect of naloxone on circadian rhythm in lung function

observations between ourselves and Dr Kay and his colleagues may be explicable by differences in the route of administration of monocrotaline and the size of the animals used. (ii) We find lung hyperplasia, right ventricular hypertrophy, and reductions in serotonin transport in the absence of changes in lung total ACE. (iii) If hyperplasia of an organ occurs, the expression of enzyme activities on a total organ basis is appropriate. (iv) We have observed right ventricular hypertrophy only after the development of lung hyperplasia. It is implied by Drs Keane and Kay that right ventricular hypertrophy may be obtained after monocrotaline administration in the absence of lung hyperplasia. This is an important statement, and should be appropriately documented (no data on organ weights are presented in their report), as it has a bearing on hypotheses concerning the inducement of hypertrophy.