{'fraction_non_alphanumeric': 0.02696078431372549, 'fraction_numerical': 0.00196078431372549, 'mean_word_length': 4.765536723163842, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
When Drinking Makes the Tremor Worse: A Task‐Specific Orolingual Tremor

The patient was a 49-year-old right-handed female homemaker. She reported a 6-month history of movements affecting the mouth and jaw when drinking. It was not present when eating. She had noticed that the movements were not precipitated by drinking with a straw, and she had taken to drinking in this fashion. Stress and caffeine were not exacerbating factors. The movements seemed to be reduced by drinking at least three glasses of wine, but she considered this intake excessive. There was no history of tremor or abnormal movement in the hands, arms, head, voice, or legs. There was no stiffness or slowness of the limbs nor walking difficulty. There was no reported disturbance of sleep and no history of headache or jaw pain suggestive of bruxism. She had played trumpet for 8 years while at high school. There was no history of oral trauma or recent dental procedure. There was no relevant past medical history. Her maternal grandmother had a tremor when holding items in her right hand, but no tremor at rest, and there was no family history of Parkinson’s disease or essential tremor. On examination, there was no visible tremor of the jaw, lips, head, or limbs at rest. A fine tremor of the jaw and lips became visible upon drinking from a cup (see Video). She could not voluntarily suppress the movement, but tended to take short sips of liquid, reducing the duration of the tremor. Cranial nerves, motor, sensory, cerebellar, and gait examination were also unremarkable. Reflexes were normal. Complete blood count, hepatic profile, and renal and thyroid function were unremarkable. Inflammatory markers were not increased. Serum manganese, urinary copper, and mercury were within normal limits. MRI brain with contrast showed a normal brain with minimal leukoaraiosis. There was no evidence of restricted diffusion or contrast enhancement. The patient was only mildly inconvenienced by the tremor and declined further treatment. Neurophysiological Findings