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Nemo‐like kinase induces apoptosis and inhibits androgen receptor signaling in prostate cancer cells

The mitogen‐activated protein kinases (MAPKs) regulate cell growth, differentiation, and stress responses, and many critical signaling pathways are subject to cross‐regulation by MAPK signaling. Previous studies have yielded evidence of cross‐talk between the MAPK pathways and androgen receptor (AR) signaling, which plays a critical role in growth control of both normal prostate and prostate cancer (PCa). Objective of this study was to evaluate the expression of MAPK‐like protein nemo‐like kinase (NLK) in PCa and its effects on AR‐mediated transcription.