{'fraction_non_alphanumeric': 0.016556291390728478, 'fraction_numerical': 0.005203405865657522, 'mean_word_length': 4.858725761772853, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Three unusual cases of parasitic infections.

Following EBV infection with or without infectious mononucleosis there is a VGA and an EBNA antibody response but only 50 to 70% of patients produce EA antibodies. In approximately 90% of patients the EBNA antibody response is delayed for a few weeks and occasionally up to 6 months. Therefore the absence of EBNA antibodies in the presence of VGA antibodies strongly suggests recent EBV infection. We have found that in the immunologically compromised host EBNA antibodies are often not produced or are lost (unpublished data). The presence of EBNA antibodies in the absence of VCA antibodies has never been reported to our knowledge. We therefore carefully ascertained that the antibodies detected were EBNA and not antinuclear antibodies. The two serum samples positive for EBNA antibodies gave negative results with EBV-negative human MOLT 4 and LM/ DM rhesus control cells. When we became aware of this case there was unfortunately no tissue available from the biopsy of the tumour for us to look for EBNA in the tumour cells. The serum of African patients with Burkitt's lymphoma usually contains EBNA, VCA and often EA antibodies in high titres.4 American patients with Burkitt's lymphoma occasionally do not have detectable serum EBV antibodies and many others have low titres of the antibodies.4A The possibility of Burkitt's tumours of different etiologies has therefore been considered. Antibodies to EBNA, the only antigen detectable in fresh tumour cells, were found in our patient's serum during the acute phase of the disease, in the absence of other antibodies to the virus. The correct interpretation of this rare finding will have to await the complete elucidation of the pathogenesis of Burkitt's lymphoma and of its relation to EBV. The possibility, however, that EBNA is a crossreacting cellular antigen must be considered. The gene coding for EBNA could also be part of the virus genome since in avian species a similar gene, the Sarc gene, is present both in the genome of the virus causing the sarcoma and in the normal cells of these species.7