{'fraction_non_alphanumeric': 0.04447852760736196, 'fraction_numerical': 0.012269938650306749, 'mean_word_length': 5.278846153846154, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Stromal Interaction Molecule 1 Disruption Protects Myocardial from Ischemia ‐ Reperfusion Injury

It has been reported that the increase in Stromal Interacting Molecule 1 (STIM1) expression is associated with myocardial complication. However, the role of vascular STIM1 in cardiac ischemia‐reperfusion (I/R) injury is unknown. Therefore, the central hypothesis of the present study is to determine the role of deleted STIM1 in endothelial cells or smooth muscle cells (SMC) in cardiac I/R injury. Thus, we performed cardiac I/R in control mice, mice lacking the STIM1 specifically in endothelial cells (Stim1−/−EC) or specifically in SMC (Stim1−/−SMC).