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Acquired perforating dermatosis associated with sorafenib therapy

levels of Th2 chemokines may be related to the abnormal activation of Th2 cells and humoral immunity in the active stage of BP. Dahlman-Ghozlan K et al. reported that soluble isoforms of cellular adhesion molecules such as sE-selectin and soluble vascular cell adhesion molecule-1 were elevated in BP, and that the elevated sE-selectin levels correlated with the number of blisters. Interestingly, in our patient, the serum TARC/CCL17 level decreased before decreases in any of the three other parameters: eosinophil count, serum IgE level and anti-BP180 antibody level. Further studies using more longitudinal BP sera are needed to determine whether serum TARC/CCL17 can be used as a sensitive indicator of disease activity.