{'fraction_non_alphanumeric': 0.03476946334089191, 'fraction_numerical': 0.018140589569160998, 'mean_word_length': 4.832599118942731, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
GASTRIC MOTILITY IN INFANTS WITH GASTROESOPHAGEAL REFLUX (GER)

Significantly delayed gastric emptying may be seen in 30 - 40% of infants with GER. The mechanism(s) responsible for this abnormality are presently unknown.Gastric motility was studied in 8 infants (ages: 2-7 mo) with GER and normal (N = 3) or delayed (N = 5) gastric emptying of formula as defined by Tc99m milk scans. Antral and fundal contractions and gastric pacesetter potentials were measured using perfused catheters with intragastric electrodes for 40 - 50 min following a formula meal of 5 cc/kg. Motility indices (M1) were calculated from pressure tracings. Results: 1) In both groups, gastric motor activity was insignificant following a formula meal. 2) Antral and fundal M1 (mmHg / sec / 5 min) were similar in all patients studied. 3) Gastric pacesetter potentials were observed a frequency of 3 cpm. 4) No gastric dysmotility was documented in either patient group.Conclusions: In infants with GER, formula is emptied as a liquid meal without significant gastric motor activity. Postprandial gastric motility is similar in patients with normal and delayed emptying; differences in motility patterns do not appear to be a mechanism for the delayed emptying. Other variables, such as position or caloric density of feedings may be more important.