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LYMPHOCYTIC RETICULOSIS AND THE SEZARY SYNDROME *

Inflammation of the skin has always been recognized as possessing a certain latent potential for malignancy. Thus the evolution of lymphoma, leukemia, Kaposi's sarcoma' and myeloma in patients with inflammatory skin disease has been repeatedly documented (I, 2). More recently the existence of certain chronic lymphocytic inflammatory states in the skin have been noted as possessing certain common features: lymphocytic proliferation, chronic course, response to x-ray and occasionally to penicillin and the potentiality for developing lymphoma despite varying morphologic clinical features that could include papules, nodules, plaques, atrophic macules, or erythroderma (3). This correlation has created a strong unifying concept which implies that delayed hypersensitivity reactions which are chronic and persistent produce proliferative changes and that malignancy is a natural consequence. The occurrence of lymphoma in the mice undergoing graft-host reaction studied by Schwartz (4) is