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Bipolar II disorder is associated with thinning of prefrontal and temporal cortices involved in affect regulation

The neurobiological substrate of bipolar II disorder (BD‐II) remains largely unknown. A few previous studies have found evidence for cerebral cortical thinning in mixed samples of BD‐II and bipolar I disorder patients; however, no study of cortical thickness or surface area has been limited to BD‐II. In the present study, we compared magnetic resonance imaging (MRI)‐based indices of cortical thickness and surface area between individuals with BD‐II and healthy controls.