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Enabling regeneration in a β‐catenin‐deficient liver: Unraveling a novel compensatory role of mTOR

Liver can regenerate to maintain its essential functions upon injury. Several signaling pathways including the mTORC1 and Wnt/β‐catenin pathways have been shown to independently stimulate liver regeneration (LR) to promote repair. However, how one pathway could compensate during deficiency of another is less well understood. Here, we report discovery and characterization of a compensatory interplay between the mTORC1 and the Wnt/β‐catenin pathway that enables LR following liver injury when Wnt/β‐catenin pathway is interrupted genetically.