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Ultraviolet‐A1 (340–400 nm)‐mediated receptor and cytokine changes of transformed lymphocytes

Background: Ultraviolet‐A1 (340–400 nm) (UVA1) radiation causes singlet‐oxygen damage that depolarizes mitochondrial membranes triggering immediate apoptosis (T≤4 h), while it also causes oxidative damage to DNA inducing delayed apoptosis (T≥24 h). In this study, we examined some potential therapeutic endpoints associated with UVA1‐mediated immediate and delayed apoptosis, such as receptor and cytokine changes.