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PHYSIOLOGICAL ROLE OF PROSTAGLANDINS IN HUMAN PARTURITION

The ability of prostaglandin EI, £2 and F2a to stimulate the uterus to contract at any time of gestation and to interrupt pregnancy at any stage has led to a number of studies on their possible role in the physiological activation of the uterus during parturition. The hypothesis of an important role is supported by the fact that prostaglandin synthetase inhibitors can inhibit preterm labor and delay parturition. The marked rise in amniotic fluid levels of PGE2 and PGF2a during term labor and the rise in circulating levels of PGF metabolite (PGFM) during advanced labor also suggests a role for prostaglandins in the mechanism of labor. An important question is whether prostaglandins are involved in the initiation or maintenance of uterine contractions, or whether they are released as a consequence of labor and participate in other cellular functions.