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LPS‐Induced Occult Loss in Mice Requires FGL2

Problem  FGL2 prothrombinase is required for spontaneous abortion (resorptions) in the CBA × DBA/2 model, and for abortions in C57Bl/6 (B6) mice triggered by Salmonella enteritidis lipopolysaccharide (LPS). Unlike abortions, occult losses in B6 mice, which begin before gestation day 9.5 in mice, do not require the tumor necrosis factor‐α receptor type 1, and may be triggered by either Salmonella enteritidis or Escherichia coli LPS. Heterozygous matings of fgl2+/− × fgl2+/− B6 mice also have a high spontaneous occult loss of fgl2−/− and to a lesser extent, fgl2+/− embryos caused by hemorrhage between trophoblast and Reichert’s membrane. However, the frequency of such losses appears to vary among breeding periods and between laboratories.