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Amelioration of the β‐cell dysfunction in diabetic APA hamsters by antioxidants and AGE inhibitor treatments

In our recent report, probucol treatment ameliorated glucose intolerance and increased the insulin‐positive area in the pancreas of streptozotocin (SZ)‐induced diabetic APA hamsters. The data suggested that the beneficial effects of probucol treatment on β‐cell function might result from its additive effect as an antioxidant. Here, we examined the antioxidant effects on the β‐cell function in SZ‐induced diabetic APA hamsters treated with three different agents, N‐acetyl‐L‐cysteine (NAC), aminoguanidine (AG) and pyridoxamine (PM).