{'fraction_non_alphanumeric': 0.015057573073516387, 'fraction_numerical': 0.0, 'mean_word_length': 5.608187134502924, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Interstitial emphysema: a study of physiologic factors involved in experimental induction of the lesion.

Perivascular interstitial emphysema was produced in anesthetized rabbits by the intratracheal application of air under increased pressure in the form of a square wave, five seconds in duration and ten times in succession. During pressurization, lung volume, tracheal air pressure, and pulmonary arterial pressure were measured simultaneously. In one group of animals, the thorax was taped to restrain expansion of the lungs. The magnitude of the applied tracheal pressure directly correlated with the frequency of the perivascular interstitial emphysema lesion in unbound animals. Thoracic binding limited increase in lung volume and decreased the incidence of perivascular interstitial emphysema. The alveolar-arterial pressure gradient did not correlate with occurrence of the lesion. It was postulated that unbalanced mechanical stresses occurred at the alveolar base because of a disproportionate change in pulmonary blood volume and air volume with positive lung inflation leading to tissue disruption and release o...