{'fraction_non_alphanumeric': 0.025974025974025976, 'fraction_numerical': 0.005194805194805195, 'mean_word_length': 5.4581005586592175, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Acidemia of Cardiogenic Origin in Young Infants with Cyanotic Congenital Heart Abnormalities

* Formerly Associate Professor, Department of Pediatrics, Medical College of Georgia, Augusta, Georgia. Present Position, Director, Section on Pediatric Cardiology, Henry Ford Hospital, Detroit, Michigan 48202. METABOLIC acidemia of the newly born secondary to hypoxemia is a common lifethreatening event which, for its importance and relative ease of management, has received little comment. Particularly notable, however, are the reports of Gootman et al.1 and of Rudolph implicating the role of lactic acid in its genesis. Simply stated, the discrepancies between the supply of oxygen and the metabolic requirements of the cells for oxygen underlie the entire problem and form the bases for treatment. In infants with cyanotic heart disease, this complication is the most common cause of death, far surpassing heart failure and cyanosis, per se. This discussion considers two aspects of care, each equally important, and emphasizes the immediate or emergency treatment of these extremely ill infants. For a full understanding of this metabolic acidemia, how-