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Localization of the T‐cell response to RSV infection is altered in infant mice

Respiratory syncytial virus (RSV) is a leading cause of lower respiratory tract infections worldwide, causing disproportionate morbidity and mortality in infants and children. Infants with stronger Th1 responses have less severe disease, yet little is known about the infant T‐cell response within the air space. Thus, we tested the hypothesis that RSV infected infant mice would have quantitative and qualitative deficiencies in CD4+ and CD8+ T‐cell populations isolated from the bronchoalveolar lavage when compared to adults and that local delivery of IFN‐γ would increase airway CD4+ Tbet+ and CD8+ Tbet+ T‐cell responses.