{'fraction_non_alphanumeric': 0.04847207586933614, 'fraction_numerical': 0.014752370916754479, 'mean_word_length': 4.9748427672955975, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
In Vitro Function of Peripheral Blood and Peritoneal B Lymphocytes from Capd Patientsa

Uremia is associated with immune function abnormalities ( 1-4). However, it is not clear whether a B lymphocyte defect exists (2,4). Stimulation of mononuclear cells (MNC's) from the peripheral blood of uremic patients with pokeweed mitogen (PWM) (5,6) or Staphylacaccus aureus Cowan strain I (SAC) ( 6,7) leads to low IgG secretion, while the response to Epstein-Barr virus is normal (7). Peritoneal MNC's from continuous ambulatory peritoneal dialysis (CAPD) patients fail to produce Ig in response to PWM (8). This could be due to selective B defects regarding different activation signals or to inhibitory cell effects in MNC's. In this study we tested the Ig secretion of peripheral blood-derived or peritoneal effluent-derived B cells from patients undergoing CAPD by co-culture with EL-4 thymoma cells in the presence ofT cell supernatant (T-SUP) (9,10).