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Biliary motility and abnormalities associated with cholesterol cholelithiasis

Gallbladder stasis has long been thought to contribute to cholesterol gallstone pathogenesis, but it has been difficult to define the exact role of impaired biliary motility and the mechanisms by which it evolves. Recent studies suggest that abnormal lipid composition of bile influences gallbladder contractility, perhaps by modifying the composition of gallbladder membranes so that transduction of smooth muscle regulatory signals is impaired.