{'fraction_non_alphanumeric': 0.037383177570093455, 'fraction_numerical': 0.007009345794392523, 'mean_word_length': 5.6953125, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
The Sialylation Pathway and Coronary Artery Disease.

Plasma levels of non–high-density (non-HDL) cholesterol are causally linked to the risk of coronary artery disease. Non-HDL cholesterol levels are a better predictor of cardiovascular risk than levels of low-density lipoprotein (LDL) cholesterol alone, because non-HDL cholesterol comprises all known cholesterol-containing lipoproteins that can cause atherosclerosis, including LDL, very-low-density lipoprotein, intermediate-density lipoprotein, chylomicrons and their remnants, and lipoprotein(a). Nioi et al. now report in the Journal important genetic evidence suggesting a causal association between ASGR1, a gene encoding the major subunit of the asialoglycoprotein receptor, and cardiovascular disease.1 Nioi and colleagues sequenced the genomes of 2636 Icelanders and identified variants . . .