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The integrity of the FOG‐2 LXCXE pRb‐binding motif is required for small intestine homeostasis

What is the central question of this study? Do Fog2Rb−/Rb− mice present a defect of small intestine homeostasis? What is the main finding and its importance? The importance of interactions between FOG‐2 and pRb in adipose tissue physiology has previously been demonstrated. Here it is shown that this interaction is also intrinsic to small intestine homeostasis and exerts extrinsic control over mouse metabolism. Thus, this association is involved in maintaining small intestine morphology, and regulating crypt proliferation and lineage differentiation. It therefore affects mouse growth and adaptation to a high‐fat diet.