{'fraction_non_alphanumeric': 0.027979274611398965, 'fraction_numerical': 0.0051813471502590676, 'mean_word_length': 5.755244755244755, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Dry beriberi mimicking Guillain–Barre syndrome as the first presenting sign of thiamine deficiency

Sir, Thiamine deficiency can lead to Wernicke s encephalopathy (WE) characterized by the clinical triad of cerebellar dysfunction, oculomotor abnormalities, and cognitive impairment [1,2]. If untreated, WE can rapidly progress and cause irreversible neurological damage, then known as Korsakoff syndrome [3]. Rarely, thiamine deficiency presents with cardiac failure in wet Beriberi or as an acute painful neuropathy, also known as dry Beriberi, resulting in both proximal and distal weakness [4]. Herein, we describe a patient with thiamine deficiency who developed beriberi neuropathy mistaken for Guillain–Barre syndrome (GBS) 3 weeks prior to developing WE. We highlight that an acute motor neuropathy mimicking GBS can be the first presenting sign of thiamine deficiency and emphasize that early recognition of beriberi neuropathy is critical in preventing WE.