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Trafficking and Cellular Distribution of Voltage‐Gated Sodium Channels

Electrical excitability in cells such as neurons and myocytes depends not only upon the expression of voltage‐gated sodium channels but also on their correct targeting within the plasma membrane. Placing sodium channels within a broader cell biological context is beginning to shed new light on a variety of important questions such as the integration of neuronal signaling. Mutations that affect sodium channel trafficking have been shown to underlie several life‐threatening conditions including cardiac arrhythmias, revealing an important clinical context to these studies.