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Hyposmotic Stress‐induced Cytoplasmic Calcium Increase Involves TRPV4 and Src Family Kinase Activation

Many cells respond to osmotic stress by releasing ATP that acts as an extracellular signal for purinergic receptors. Receptor‐triggered increases of calcium and activation of Src family tyrosine kinases (SFKs) have previously been observed in lens epithelium. Here we examined the effect of hyposmotic stress on cytoplasmic calcium [(Ca2+)i] response.