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The hyaluronidase Hyal2 is a regulator of cell surface hyaluronan interactions

HA may increase cell motility, facilitate cancer cell invasion and induce angiogenesis. Many of these effects depend on HA binding to specific receptors, mostly CD44, on the cell surface and on the presence of a pericellular HA‐rich coat. We hypothesized that the ubiquitous glycosyl‐phosphatidylinositol‐anchored hyaluronidase Hyal2 could be one of the HA regulatory factors modifying cell response to HA.