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Corynebacterium Xerosis Sepsis in a Pediatric Patient with Sickle Cell Disease (A Case Report)

ease (HbSS). Patients with sickle cell disease develop signs of splenic dysfunction as early as 3-4 months of age. This early loss of splenic function or functional aplenia is the result of sludging of sickle cells within the spleen, which impairs the circulation and ultimately yields multiple splenic infarcts and spleen death. The major observations of this immune deficit reported in the literature include the opsonic defect for Streptococcus pneumoniae, complement activation, impaired antibody responses, alteration of B and T lymphocytes and natural killer cells, phagocyte alterations, and cytokine production. The most common organism