{'fraction_non_alphanumeric': 0.01597051597051597, 'fraction_numerical': 0.0, 'mean_word_length': 5.848739495798319, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
The Utility of Vascular Dysfunction Studies in the Prediction and Prevention of Preeclampsia: A Historical Review

Preeclampsia is a major cause of maternal and perinatal mortality. Although preeclampsia may be caused by several factors, endothelial cell dysfunction has been proposed as the main pathophysiological cause. Dysfunctional endo- thelium in the uteroplacental circulation not only increases peripheral vascular resistance, but also affects generalized vasoconstriction via humoral factors released from the placenta. A standard method for predicting and preventing preeclampsia has yet to be developed; however, the analysis of a combination of biochemical markers, particularly mark- ers related to vascular dysfunction, may enhance our ability to predict and prevent preeclampsia in the near future.