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A microanalytical study of bone mineral in a transgenic mouse model for brittle bone disease.

No single analytical technique will provide all the information necessary for the interpretation of a complex biological system. The mineralisation of bone is no exception, especially in a pathological situation. Brittle Bone disease, a genetically and biochemically heterogeneous group of disorders is characterised by the ease and frequency of bone fracture. Although numerous type I procollagen gene mutations have been detected, few reports exist of the mineral and its relationship with the altered collagen. In this study the calcium to phosphorus ratio in normal, human and or cortical bone has been compared with bone from normal mouse and a transgenic mouse model containing a shortened human procollagen gene. This mouse has exhibited many phenotypic features of human or (Cassella et ai, 1992).