{'fraction_non_alphanumeric': 0.018741633199464525, 'fraction_numerical': 0.002677376171352075, 'mean_word_length': 5.990654205607477, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Brain and liver mitochondria isolated from diabeticGoto‐Kakizaki rats show different susceptibility to induced oxidative stress

Increased oxidative stress and changes in antioxidant capacity observed in both clinical and experimental diabetes mellitus have been implicated in the etiology of chronic diabetic complications. Many authors have shown that hyperglycemia leads to an increase in lipid peroxidation in diabetic patients and animals reflecting a rise in reactive oxygen species production. The aim of the study was to compare the susceptibility of mitochondria from brain and liver of Goto‐Kakizaki (12‐month‐old diabetic) rats (GK rats), a model of non‐insulin dependent diabetes mellitus, to oxidative stress and antioxidant defenses.