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Beta‐Adrenergic Stimulation of Pig Myocytes with Decreased Cytosolic Free Magnesium Prolongs the Action Potential and Enhances Triggered Activity

Beta‐Adrenergic Stimulation and Repolarization. Introduction: Heart failure results in chronic beta‐adrenergic stimulation, repolarization lability, and arrhythmias associated with early afterdepolarizations (EADs) and delayed afterdepolarizations (DADs). Having described a significant reduction in intracellular free magnesium ([Mg2+]i) in experimental heart failure, we asked whether a reduction in [Mg2+]i would delay repolarization or facilitate EADs and/or DADs.