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The Role of the Corpus Callosum in the Establishment of Areas of Secondary Epileptiform Activity

The establishment of secondary epileptogenic foci through the direct application of penicillin to neocortex was studied in intact rats and in ones in which the corpus callosum was sectioned either before or after the penicillin application. Section of the corpus callosum before the creation of the focus prevented the ocurrence of a region of secondary (mirror) discharge, but low voltage, long latency activity could be observed in the contralateral hemisphere which was associated with each spike discharge in the primary hemisphere. Section of the corpus callosum after the bilateral application of penicillin temporarily disrupted the synchrony of spike discharges in the 2 hemispheres. The fact that synchrony of the epileptiform discharges did return was interpreted as indicating the presence of a subcortical mechanism independent of the callosal fibers.