{'fraction_non_alphanumeric': 0.03471295060080107, 'fraction_numerical': 0.01335113484646195, 'mean_word_length': 4.859375, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Prevalence of IgE mediated food allergy in children with inflammatory bowel disease

There has been a rapid increase in the number of children with both inflammatory bowel disease (IBD) and IgE mediated food allergy (FA) (1, 2). Similar factors have been implicated in their aetiology. Dysbiosis of the microbiome causes dysregulation of the immune system promoting the development of conditions such as IBD and FA (3). Abnormal intestinal barrier function is associated with both conditions (4, 5). The Th2 pathway is upregulated in both ulcerative colitis (UC|) and FA with Th1 cytokines playing more of a central role in the pathogenesis of Crohns disease (CD). In both IBD and FA, there are increased Th 17 cells and downregulation of Tregs (5).