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Sensory nerve regeneration: an experimental study of skin grafts in the rabbit.

The reinnervation of skin following nerve injury is often incomplete, resulting in abnormal sensory function. It is thought that normal sensory perception is a product of a unique pattern of impulses initiated from cutaneous receptors able to transduce varieties and ranges of stimuli into impulse patterns (Melzack & Wall, 1962). These multifibre modulated patterns (Granit, 1955) may be modified by changes in the absolute and the relative number of myelinated and unmyelinated fibres with resulting sensory disorders. Sensory dissociation with loss of pain and temperature sensation has been shown both in patients with amyloidosis (Dyck & Lambert, 1969) and in a case of dysautonomia of Riley-Day (Aguayo, Nair & Bray, 1971) to be associated with a predominant loss of peripheral unmyelinated fibres. Following a nerve injury, sensory responses may be increased or diminished and it is not uncommon to find a raised sensory threshold in association with an unpleasant 'hyperpathic' response (Head & Sherren, 1905; Lourie & King, 1966; Ochoa, 1970).