{'fraction_non_alphanumeric': 0.012544802867383513, 'fraction_numerical': 0.010752688172043012, 'mean_word_length': 5.240223463687151, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Cerebral Angiography during Acetylcholine Chloride Induced Momentary Cardiac Arrest.: An Experimental Study in Dogs

Cerebral angiographic findings in momentary cardiac arrest were investigated in 16 dogs. Momentary cardiac arrest was induced utilizing rapid injection of acetylcholine chloride into the superior vena cava. The momentary cardiac arrest occurred 3 to 8 seconds after the injection, and lasted for 3 to 25 seconds. The duration of the cardiac arrest was proportional to the dose of the injection. The most striking angiographic finding during momentary cardiac arrest was the prolongation of the arterial phase in the Intracranial arteries during 6 or 12 second course of the study. Although the circulation was somewhat slow, good filling of the external carotid artery was always present. The angiographic findings were probably based on the rapid reduction of the arterial pressure below 30 mm Hg immediately after the arrest, which in turn, causes a marked reduction of cerebral blood flow. This momentary cerebral circulatory failure produces a stasis of the contrast medium in the arterial tree.