{'fraction_non_alphanumeric': 0.028985507246376812, 'fraction_numerical': 0.017391304347826087, 'mean_word_length': 5.225225225225225, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Permeability of gingival sulcular epithelium in the development of scorbutic gingivitis.

Young adult nonhuman primates were initially fed an ascorbic-acid free diet and, subsequently, a diet with a suboptimal level of the vitamin. Following approximately 12 weeks of this dietary regime, five of seven experimental animals of the gingival sulcular epithelium to 3H-inulin, but not to 14C-butyric acid or 14C-urea, had increased significantly (+86%, P less than 0.01) in the experimental group as compared to ad libitum or pair-fed controls. This defect in the intercellular permeability barrier of the sulcular epithelium could have contributed to the pathogenesis of scorbutic gingivitis.