{'fraction_non_alphanumeric': 0.00625, 'fraction_numerical': 0.0, 'mean_word_length': 6.043956043956044, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Cognitive reserve for memory performance is mediated by preclinical frontotemporal and limbic amyloid deposition in subjects with normal cognition in early MCI

The neuroanatomic substrate for cognitive reserve has been poorly elucidated. The development of antemortem biomarkers such as amyloid‐PET afford the potential to begin to explore regional pathologic burden with overt cognitive performance as well as cognitive resilience in relation to neuroanatomic distributions of pathology. The present study sought to identify the relationship between regional amyloid burden and objective measures of cognitive reserve for memory function.