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Induction of hepatic peroxisomal beta-oxidation of fatty acids by dehydroepiandrosterone (DHEA) in male F-344 rats.

Pharmacological administration of dehydroepiandrostenone (DHEA, 300 mg/kg, p.o.) for 2 weeks significantly increased (P < 0.01) the activity of hepatic peroxisomal beta-oxidation of fatty acids in male F-344 rats. Similar effects were observed following 8 week administration of DHEA. These results are discussed in the light of oxidative stress-mediated hepatocarcinogenesis.