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[Inhibition of the renin-angiotensin system in the ischemic kidney].

By blocking the renin-angiotensin-system with saralasin or salt-loading the postischemic damage of the renal microcirculation is diminished. This can be realized in a markedly improved oxygenation of renal tissue. As a result of the improved microcirculation 60 minutes after a 120-minutes ischemic damage one can find a better filtration rate, i.e. tubular function in treated kidneys.