{'fraction_non_alphanumeric': 0.013207547169811321, 'fraction_numerical': 0.0, 'mean_word_length': 5.896103896103896, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Autoimmune encephalitis in the age of neuronal surface antigens

Since anti-NMDA receptor (NMDAR) encephalitis was first described almost a decade ago, the family of disorders associated with antibodies against neuronal surface antigens is one of the most rapidly expanding categories of neurologic disease. With this growth comes a paradigm shift in neurology—autoimmune encephalitis has evolved from an often vaguely defined clinical syndrome to a group of precisely delineated disorders associated with specific autoantibodies.