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Uncoupling of monooxygenation and electron transport by fluorocarbons in liver microsomes.

Perfluoro-n-hexane in contrast to n-hexane is not hydroxylated by the microsomal monooxygenation system in liver microsomes of phenobarbital treated rats. However, the fluorocarbon forms an enzyme-substrate complex and, therefore, stimulates NADPH oxidation. Fluorocarbons can be regarded as dead-end inhibitors which uncouple electron transport from monooxygenation. This effect must be taken into consideration when fluorocarbons are used as oxygen carriers in liver perfusion.