{'fraction_non_alphanumeric': 0.01634877384196185, 'fraction_numerical': 0.0, 'mean_word_length': 5.095435684647303, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Management of the asphyxiated full term infant.

In Britain, approximately one full term baby per thousand dies or is severely disabled as the result of birth asphyxia. It is arguably the most important avoidable cause of permanent neurological injury affecting the mature fetus/newborn. It is generally agreed that clinical signs of hypoxic-ischaemic encephalopathy (HIE) are the best markers for a diagnosis of intrapartum 'asphyxia'. Unfortunately the severity of HIE can only be diagnosed retrospectively after symptoms have developed. Early therapeutic intervention in the asphyxiated baby may be important to modify cerebral injury (see below) and therefore there remains a need to have early markers of asphyxia such as depressed Apgar scores, delay in establishing respiration, or evidence of significant metabolic acidosis on samples of cord blood. Although there appears to have been a fall in both the incidence of HIE and the number of children disabled by this condition in recent years,' there is little evidence that this has been due to improvement in postnatal management. In this paper I will consider the standard management of asphyxia, potentially useful new methods for treating the asphyxiated brain, and consider methods of deciding when to withdraw care. The brain of the full term infant responds to asphyxia in a very different manner to that of a premature baby and this paper only considers the management of asphyxia in full term infants.