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Clinical and pathological features of mitochondrial DNA deletion disease following antiretroviral treatment.

Clinical and Pathological Features of Mitochondrial DNA Deletion Disease Following Antiretroviral Treatment Certain nucleoside analog reverse transcriptase inhibitor (NRTI) antiretroviral drugs used to treat human immunodeficiency virus (HIV) infection lead to accelerated accumulation of somatic mitochondrial DNA (mtDNA) mutations.1 The clinical significance of this observation is unclear but a delayed-onset phenotype would be expected, perhaps years after the relevant drug exposure.