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Histopathological Changes In Two Models Of Acute Kidney Injury

Two major challenges in management of Acute Kidney Injury, which cause and develop renal disturbances, are inflammation and oxidative stress. Acute Kidney Injury represents an important health problem, since an effective therapy to treat it is not available yet (1). During Acute Kidney Injury renal morphophysiology is affected as a consequence of renal ischemia/reperfusion (I/R) or nephrotoxicity (2, 3). The objective of this work was to identify renal histopathological changes in two experimental models of Acute Kidney Injury in Wistar rats.