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HSP90/ENOS interactions contribute to maturational and chronic hypoxia‐induced changes in pulmonary vascular reactivity in newborn pigs

Hsp90 is a molecular chaperone involved in regulating eNOS signaling. Concomitant with enhanced NO production, the association of Hsp90 with eNOS increases upon stimulation with agonists such as Acetylcholine (ACh). The role of Hsp90/eNOS interactions in maturational changes in pulmonary vascular reactivity is unclear. Moreover, there are no data on Hsp90/eNOS interactions in newborns with chronic hypoxia‐induced pulmonary hypertension.