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The salt‐sensitivity in C57Bl/6J mice is linked to increased renal protein expressions of SLC4A4 and SLC4A5

Renal regulation of sodium reabsorption largely depends on the function and protein expression of exchanger /transporters /channels for sodium and other ions along the nephron. Decreased protein expression of NCC mediated aldosterone‐escape, a reflection of pressure natriuresis phenomenon. However, the role of renal ion transporters in salt‐sensitive mice is not well understood. Therefore, we studied the effect of varying amounts of sodium intake on the blood pressure (BP) and renal expression of exchanger /transporters/channels for sodium and other ions in salt‐sensitive C57Bl/6J mice and salt‐resistant BALB/c mice.