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MR Imaging of Intracranial Tuberculosis

754 sal ganglia, the cerebellum, brainstem and in the ventricular system. These lesions begin as microgranulomata in an area of Tubercular cerebritis and gradually conglomerate to form a non-caseating tuberculoma. Subsequent central caseous necrosis develops in most cases. CT and MRI are the imaging techniques usually used in the anatomic localisation and charecterisation of such lesions. The tuberculoma is usually isodense or mildly hyperdense on non-enhanced CT. On contrast enhanced CT, the smaller lesions enhance uniformly and appear as disk lesions. The peripheral regions of the lesions enhance very well and the lesions appear as ring lesions (figure 1). Central calcification of the lesion is seen as “Target sign” in mature tuberculomas (figure 2). However, the CT features were found to be inadequate to make a definitive diagnosis in routine clinical practice. MRI due to superior contrast resolution has been explored for better charecterization of intracranial tuberculomas. The MR features of the individual tuberculoma depend on the maturity of the tuberculoma; whether the granuloma is non-caseating or caIntroduction