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Effects of aminoguanidine on γ‐aminobutyric acid accumulation pathways in fresh tea leaves under hypoxic conditions

Aminoguanidine (AG), a specific inhibitor of diamine oxidase (DAO), can be used to investigate the γ‐aminobutyric acid (GABA) accumulation pathways. The effect of AG on GABA accumulation pathways of tea fresh leaves remains to be explored. Our results indicated AG concentration did not significantly affect GAD activity, but high concentration solution made GABA‐T activity increase. AG (1 mm) maximally, but not entirely, blocked the polyamine degradation pathway, but had no effect on GAD and GABA‐T. The polyamine degradation pathway was responsible for 26.8% and 17.0% of GABA formation under hypoxic conditions involving immersion and immersion–vacuum, respectively, because DAO and PAO activity was not completely inhibited.