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Vascular hyporesponsiveness to angiotensin II in rats with CCl4‐induced liver cirrhosis

Background  Portal hypertension is triggered by vasodilation due to impaired contraction of extrahepatic vessels. Angiotensin II type 1 (AT1) receptor‐induced vasocontraction is mediated by G proteins and may be desensitized by recruitment of β‐arrestin‐2 to the receptor. In this study, we analysed the interaction of AT1 receptors with β‐arrestin‐2 in the context of vascular hypocontractility in rats with CCl4‐induced cirrhosis.