{'fraction_non_alphanumeric': 0.021996615905245348, 'fraction_numerical': 0.008460236886632826, 'mean_word_length': 5.7988505747126435, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Episodic hyperhidrosis as the only clinical manifestation of neuromyotonia

Hyperhidrosis and other autonomic symptoms are frequent in patients with peripheral nerve hyperexcitability syndromes, particularly when they are associated with involvement of the CNS (Morvan syndrome). In this clinical setting, hyperhidrosis is usually associated with prominent muscle hyperactivity, insomnia, or episodes of confusion and visual hallucinations.1 Around 5% of patients with peripheral nerve hyperexcitability and Morvan syndromes are associated with thymoma and more rarely other tumor types.2,3 Up to 35% of patients with these syndromes present raised titers of serum voltage-gated potassium channels (VGKC) antibodies3 and preliminary evidence suggests that the antibodies could be responsible for the clinical features.4

We report a patient with episodes of profuse sweating in absence of clinical neuromyotonia.

A 66-year-old man was evaluated for episodes of profuse sweating and malaise. Eighteen months earlier, the patient had received the diagnosis of polymyositis after a clinical picture of progressive anorexia, dysphagia, weight loss, and neck extensor weakness, with a …