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Biological mechanisms at moderate levels of alcohol consumption that may affect coronary heart disease

The aims of the present article are to present a framework for understanding the ways in which various alcohol-related mechanisms might fit into current hypotheses of coronary artery disease (CAD) and to consider whether proposed alcohol-related mechanisms have a degree of validity conceptually, experimentally and epidemiologically. It does not attempt to review in detail the literature on either current concepts of CAD or alcohol-related mechanisms. which has been well reviewed elsewhere (Steinberg et al., 1989; Fuster et al., 1992a, b; Kreisberg and Segrest, 1993; Steinberg et al., 1991; Veenstra, 1992). In carrying out this task, it is important to