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Toward personalized medicine for cardiovascular pharmacotherapy.

( A ) Role of some of the genetic loci associated with angiotensin-converting enzyme inhibitor discontinuation due to cough. ( B ) The authors hypothesized that genetic variants could partly share pathophysiological pathways, so that genetic susceptibility to ACEIAC could in part be driven by an innate cough sensitization, which progresses into cough after exposure to a trigger (e.g. ACEI, GERD, or allergens). Abbreviations: ACE, angiotensin-converting enzyme; ACEI, ACE inhibitor; ACEIAC: ACEI-associated cough; GERD, gastro-oesophageal re ﬂ ux disease; GWAS, genome-wide association study; NTR1, neurotensin receptor 1.