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Seasonal variations in the incidence of photoparoxysmal response.

oidal bones as well as irregular bone destruction superiorly in the vault. There was also considerable encroachment on the left hemisphere with oedema ofthe white matter, compression of the left lateral ventricle and displacement of the ventricles to the right. After contrast an enhancing mass was demonstrated in the area of dysplastic bone (fig). At surgery a grey gelatinous tumour was found beneath the left parietal bone compressing the cerebral cortex, which was shown histologically to be a meningothelial meningioma infiltrating bone. There were no features of a sarcoma. The association between fibrous dysplasia and meningioma does not seem to be recognised and this may be the first report. The direct juxtaposition of the dysplastic bone and the tumour make it most likely that the link is causal. Neither previous craniectomy nor acrylic prostheses are known to be associated with meningioma and the patient had never been treated with radiotherapy. Although bone histology was not available in this case, the radiological appearances, the normal serum biochemistry and the age of onset in late childhood make fibrous dysplasia the only reasonable diagnosis. The bony changes could not be explained on the basis of the known hyperostosis which is associated with meningiomas both because many cranial bones were involved radiologically which were distant from the tumour and secondly no meningioma was present at his original craniectomy 22 years previously. The authors thank Dr G M Stern for permission to report this case.