{'fraction_non_alphanumeric': 0.02827087442472058, 'fraction_numerical': 0.003287310979618672, 'mean_word_length': 4.9921259842519685, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
[Phonocardiographical studies in hypertensive subjects (author's transl)].

Phonocardiographical (PCG) studies were made to evaluate the relation of the PCG to blood pressure, ECG, chest X-ray finding and changes of fundi in hypertensive subjects. One hundred and thirty hypertensive patients and sixty normal adults were examined. Results were as follows: 1) There were abnormal second heart sounds such as, narrow splitting or ringing in most cases of hypertensive subjects with high both systolic and diastolic blood pressure. These phenomena seemed to be delay and accentuation of aortic component. 2) Ejection murmur was found in majority of constantly hypertensive cases. 3) The third and fourth sounds had no definite correlation to blood pressure. However, accentuation of the fourth sound was found only in subjects with severe myocardial changes in ECG. 4). Q-I time in hypertensive group was found to be prolonged as compared to normal. 5) In normal subjects, I-IIA interval was related closely to square root R--R. It was not clear in hypertensive group, and the most cases of this group showed that I-IIA interval was prolonged as compared to square root R--R. Prolonged I-IIA interval was seen in the group with only high voltage and no myocardial abnormality in ECG. In conclusion, characteristic phonocardiographical findings in hypertensive subjects were the prolonged mechanical contraction time involving isovolumic one and this time became shorter when myocardial damage was increased in its severity.