{'fraction_non_alphanumeric': 0.03492884864165589, 'fraction_numerical': 0.02457956015523933, 'mean_word_length': 5.910714285714286, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Diverse clinical outcomes of eosinophilic patients with T‐cell receptor gene rearrangements: The emerging diagnostic importance of molecular genetics testing

Abnormal clones of clusters of differentiation (CD)3−CD4+ or CD3+CD4−CD8− phenotypically abnormal lymphocytes have been identified in some patients who have the idiopathic hypereosinophilic syndrome. This report illustrates the disparate clinical courses of six eosinophilic patients with evidence of abnormal T‐cell clones based on the finding of a T‐cell receptor gene rearrangement. The data suggest that molecular genetics testing for T‐cell receptor gene rearrangements should be included in the routine work‐up of patients with idiopathic eosinophilia. Am. J. Hematol. 68:81–86, 2001. © 2001 Wiley‐Liss, Inc.