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The role of PKC‐B in the BKCa functionality of vascular smooth muscle of diabetic rats

Diabetes mellitus (DM) is a disease characterized by high blood glucose concentrations. Cardiovascular disorders associated with DM are the leading cause of high morbidity and mortality of this disease, where endothelial tissue as well as smooth muscle is damaged. For example, the contractility of the smooth muscle cells is increased partly due to a decrease in the activity of the calcium‐dependent high conductance potassium channels (BKCa), which along with other potassium conductance maintain membrane potential and vascular tone.