{'fraction_non_alphanumeric': 0.023668639053254437, 'fraction_numerical': 0.02485207100591716, 'mean_word_length': 4.64, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Sensitization for audiogenic seizures in two strains of mice and their F1 hybrids.

Acoustic sensitization or priming for audiogenic seizures was studied in 304 “seizure-resistant” mice of C57BL, BALB/c and their F1 hybirds. The effect of priming was found to be a function of strain, age of the first auditory exposure, and duration of exposure. In BALB/c mice priming at ages 21–28 days induced very high incidence of seizures; in this stran 2-min exposure was more effective than both 60 sec or 30 sec. The same parameters of priming induced very low seizure incidence in C57BL mice. A behavior-genetic analysis indicated that the genotype of each parental strain might contribute equally to the priming induced seizure risk of their F1 hybrids, but C57BL mice appeared to be dominant to the BALB/c for some components of audiogenic seizures.