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Tumor necrosis factor‐a antisense transfer remarkably improves hepatic graft viability

Abstract: Background: Cold ischemia/reperfusion injury of the hepatic graft, an unsolved problem in liver transplantations, is attributed to the release of inflammatory cytokines, especially the tumor necrosis factor‐ (TNF) α, from activated Kupffer cells (KC). Therefore, the specific inhibition of TNF‐α could improve the viability of the hepatic graft upon reperfusion.