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Intensive physical training reduces intermittent hypoxia‐related vascular dysfunction by affecting the mechanisms of smooth muscle from molecular to physiological scales

Obstructive sleep apnea syndrome (OSA) is a common clinical condition produced by episodes of repeated upper airways obstruction resulting in airflow blockade (apnea) during sleep. OSA is defined as a frequent chronic disease, which is highly prevalent in the population (affecting 9% of women and 24% of men) and which increases linearly with age. Chronic intermittent hypoxia (IH), the main pathogenic consequence related to OSA, is responsible for cardiovascular morbidity, including systemic arterial hypertension, cardiac infarct, atherosclerosis, and muscular remodeling in OSA patients.