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Chronic Hypoxia Attenuates cGMP‐dependent protein kinase (PKG)‐Mediated Vasorelaxation by Depressing BK Potassium Channel Activity in Cerebral Arteries

Chronic hypoxia attenuates the ability of PKG to induce vasorelaxation in serotonin‐contracted ovine carotids, and more so in fetal than adult arteries. The present investigation examines the hypothesis that alterations in the BK channel and other PKG kinase substrates contribute to hypoxic attenuation of PKG‐dependent vasorelaxation in term fetal and adult ovine cerebral arteries.