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Mitochondrial Damage and Protein Modification in the Diabetic Kidney Mouse Model

A hallmark of diabetes is increased levels of oxidative stress, such as superoxide formation. Superoxide can have detrimental effects on the mitochondria by increasing reactive nitrogen species (RNS) and generating other reactive oxygen species (ROS). We hypothesize that diabetes induced oxidative stress leads to mitochondrial membrane permeability and protein modification in the diabetic mouse model.