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Musanga cecropioides (Cecropiaceae) Attenuates Carbon Tetrachloride-Induced Non-Alcoholic Fatty Liver Disease in Wistar Rats

The incidence of non-alcoholic fatty liver disease (NAFLD) is on the rise globally largely due to increased obesity and change in lifestyle among adults and children resulting to accumulation of fat in the liver. NAFLD has also been implicated as a risk factor for cardiovascular diseases which is the most common mortality associated with patients and is increasingly the main reason for liver transplantation lately. 3 The presence of fatty liver or hepatic steatosis marks the onset of the disease and this could advance to steatohepatitis with subsequent inflammation of hepatocytes. Although, it has been reported that about 5 to 20% of patients with fatty liver progresses to non-alcoholic steatohepatitis (NASH) during the disease, but over 10 to 20% may advance to higher-grade fibrosis while less than 5% degenerates to cirrhosis of the liver. NAFLD-induced cirrhosis has also been reported to heighten the progression of liver disease to hepatocellular carcinoma. The parthogenesis of NAFLD remains unclear, however, documented evidence show type 2 diabetes mellitus, insulin resistance accumulation of fatty acids which can alter reactive oxygen species (ROS) has been implicated as risk factors.