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A comparative study of the effects of methyl iodide and cobalt chloride on the lipid metabolism in rabbits--with special reference to the content alteration of coenzyme A, carnitine and their derivatives in rabbit liver.

Following a single dose of methyl iodide1•`4) or repeated administration of cobalt chloride5•`7), the plasma triglyceride concentration in rabbit increases noticeably. Explanations for the mechanism on hypertriglyceridemia: elevations of plasma free fatty acid (FFA) concentration2), decrease of lipoprotein lipase activity in adipose tissue2,3), increased level of plasma glucose7) and insulin7), increased content of hepatic ƒ¿-glycerophosphate (ƒ¿-GP)8), and increased plasma triglyceride turnover rate9) have been reported. However, the mechanism concerned is still obscure. To gain further insight into factors related to hypertriglyceridemia, a comparative study was made on the effects of methyl iodide and cobalt chloride on lipid metabolism with special reference to the contents of CoA, carnitine and their derivatives to determine to what extent they might relate to triglyceride synthesis or fatty acid oxidation. Methyl iodide induced an accumulation of triglyceride in the liver2), while cobalt chloride did not6), but the latter did increase the level of plasma insulin7,8). Increase of plasma