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Gene and protein expression of exercise pressor reflex‐related molecular receptors in dorsal root ganglion of spontaneous‐hypertensive and normotensive rats

The exercise pressor reflex (EPR), mediated by mechano‐ and metabosensitive skeletal muscle afferent feedback, is exaggerated in spontaneously hypertensive rats. This cardiovascular reflex dysfunction could be the result of disease‐related abnormalities in the expression and/or sensitivity of molecular receptors located on the terminal end of these muscle afferents, altered central integration of muscle afferent feedback, and/or altered end‐organ responsiveness to a given sympathetic activity.