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The Natriuretic Response to Acute K+ Intake

Diets rich in K+ are associated with unexplained protection against hypertension. Acute K+ intake induces natriuresis and kaliuresis in mammals linked to marked dephosphorylation of renal Na+/Cl‐‐cotransporters (NCC). Reduced activity of NCC increases Na+ delivery to the collecting ducts, hypothesized to increase driving force for ENaC‐dependent K+ secretion. Here we investigated K+ and Na+ excretion in mice adapted either to preserve or promote Na+ loss.