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Effect of gamma‐hydroxybutyric acid on tissue Na+,K+‐ATPase levels after experimental head trauma

Background:  A failure of the Na+,K+‐ATPase activity (which is essential for ion flux across the cell membranes) occurs in many pathological conditions and may lead to cell dysfunction or even cell death. By altering the concentration of specific opioid peptides, gamma‐hydroxybutyric acid (GHB) may change ion flux across cell membranes and produce the ‘channel arrest’ which we assumed will inhibit the failure of Na+,K+‐ATPase activity and therefore lead to energy conservation and cell protection. Therefore we planned this study to see the effects of GHB at two different doses on Na+,K+‐ATPase activity in an experimental head trauma model.