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Intestinal HuR deficiency exacerbates diet‐induced fatty liver disease

Nonalcoholic fatty liver disease (NAFLD) has become the most common chronic liver disease worldwide. However, no effective treatment is available due to the limited knowledge regarding the complex disease pathology. We and others have previously reported that impaired intestinal barrier function promotes the progression of various liver diseases, including NAFLD. Human antigen R (HuR) is a member of the Hu family of RNA‐binding proteins. It is reported that HuR is essential for maintaining gut epithelial integrity, and targeted deletion of HuR in the intestinal epithelium disrupts mucosal regeneration in mice. However, the role of intestinal HuR in NAFLD remains unclear and is the focus of this study.