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MicroRNA‐155 modulates P2R signaling and Th2 priming of dendritic cells during allergic airway inflammation in mice

Dendritic cells (DCs) are the professional antigen‐presenting cells (APCs) in the lung. They are known to be key players in the induction and maintenance of allergic asthma by cross‐linking innate and adaptive immune responses. MicroRNAs (miRNAs) are known to influence cell fate and function by translational suppression or induction of messenger RNA (mRNA) degradation. miR‐155 has been shown to be a crucial regulator of the immune system. However, its function in the pathogenesis of allergic airway inflammation (AAI) is not completely elucidated yet.