{'fraction_non_alphanumeric': 0.03802281368821293, 'fraction_numerical': 0.012357414448669201, 'mean_word_length': 6.163265306122449, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Investigation of antibiotic resistance genes of beta-hemolytic and non-hemolytic streptococci with molecular methods

Streptococcus species continue to exist as important human pathogens. They produce and release virulence factors such as several resistance genes, toxins and adhesion molecules. The most common infections caused by them include acute otitis media, pharyngitis, respiratory tract infections (sinusitis), impetigo, erysipelas, endocarditis, meningitis, puerperal sepsis, arthritis, osteomyelitis, post-streptococcal glomerulonephritis and rheumatic fever (1,2). In addition to these infections, they are the pathogens causing severe neonatal infections characterized by maternal infections. They cause osteomyelitis, skin and soft tissue infections, infections associated with endocarditis and bacteremia (3,4). Every year, 4 million of people experience community-based pneumonia in the United States. Community-based pneumonia was reported to be caused by Streptococcus pneumonia in 40-60% and by other bacteria in 310% of people (5).