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Further analysis of the resistance of a colony of rats to dextran.

Rats selectively bred for non-reactivity to ovomucoid and to clinical dextran (termed non-reactors) have a higher blood polymorphonuclear neutrophil count than do rats which react with an anaphylactoid reaction (termed reactors). This finding may explain why non-reactors do not exhibit the signs of arthritis when Freund’s adjuvant is injected into the hind paw. Nevertheless, the transfer of blood white cells and lymphocytes from spleen, thymus and lymph nodes of reactors fails to break down the resistance of non-reactors to dextran. Insulin likewise does not render non-reactor rats sensitive to dextran although it markedly potentiates the reaction in reactors. If new protein synthesis induced by insulin is involved in the potentiation, then non-reactors may lack the ability to synthesize this protein in the presence of insulin. However, insulin increases the severity of anaphylactic shock in both reactors and non-reactors when administered at the same time as the intravenous challenge of antigen.