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Differential mechanisms of nitric oxide and cyclooxygenases in endothelium‐dependent relaxation of human internal mammary artery and saphenous vein as bypass grafts

Endothelial dysfunction plays a key role in vasculopathy of coronary artery bypass grafting (CABG). Both nitric oxide (NO) from eNOS and prostacyclin (PGI2) from cyclooxygenase (COX) mediate endothelium‐dependent relaxation. Internal mammary artery (IMA) has better compliance than saphenous vein (SA) in CABG with unknown mechanisms. This study compared the difference in endothelial relaxations of IMA and SV.