{'fraction_non_alphanumeric': 0.02676659528907923, 'fraction_numerical': 0.01284796573875803, 'mean_word_length': 6.248062015503876, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Increased enzymuria in diabetic children without persistent minimal albuminuria

Summary: The urinary activity of the enzyme N‐acetyl‐β‐glucosaminidase is a useful, early marker of renal tubular damage in various conditions. Consequently, it is tempting to presume that the excretion of this enzyme might recognize subclinical renal damage long before elevation of urinary albumin excretion. the urinary N‐acetyl‐β‐glucosaminidase activity was therefore assessed in a group of 77 diabetic patients (37 girls and 40 boys, aged 2.9‐20 years) without persisting minimal albuminuria. Circulating glycosylated haemoglobin and fructosamine were significantly higher in the 24 patients with persistently increased urinary N‐acetyl‐β‐glucosaminidase compared with the remaining patients. It is concluded that raised urinary N‐ acetyl‐β‐glucosaminidase might be the earliest detectable abnormality of renal damage in chronic diabetes mellitus.