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Involvement of HGF/SF–Met signaling in prostate adenocarcinoma cells: Evidence for alternative mechanisms leading to a metastatic phenotype in Pr‐14c

Hepatocyte growth factor/scatter factor (HGF/SF) facilitates intercellular communication between the epithelial carcinoma and its surrounding stromal tissue during metastatic invasion through interaction with its proto‐oncogenic receptor, Met, found on carcinoma cells. This study utilizes the C3(1)/Tag transgenic mouse prostate cancer cell line model in an attempt to characterize the interaction between HGF/SF and Met on the metastatic potential of prostate cancer.