{'fraction_non_alphanumeric': 0.03576982892690513, 'fraction_numerical': 0.003110419906687403, 'mean_word_length': 6.318181818181818, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Rhinovirus infection‐induced alteration of tight junction and adherens junction components in human nasal epithelial cells

Manifestations of rhinovirus (RV) infections include mucus overproduction, increased vascular permeability, and secondary bacterial infection. These effects may reflect disrupted epithelial barrier functions, which are mainly regulated by intercellular junctions, referred to as tight junctions (TJs) and adherens junctions (AJs). The objective of this study was to investigate changes in the components of TJs (ZO‐1, occluding, and claudin‐1) and AJs (E‐cadherin) after RV infection in cultured nasal epithelial cells.