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Androgen receptor dampens tissue factor expression via NF-κ B and EGR 1

Background Prostate cancer is one of the leading causes of cancer death in men. Advanced prostate cancer is usually treated by androgen deprivation therapy (ADT), which aims at diminishing circulating testosterone to reduce cancer growth. There is growing evidence that ADT can increase the rate of venous thromboembolism (VTE) in prostate cancer patients. The tissue factor (TF) gene is one of the A cc ep te d A rt ic le This article is protected by copyright. All rights reserved. most important mediators of coagulation and VTE, but so far there is limited data on androgen receptor (AR) mediated TF gene expression.