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Epstein‐Barr virus infection and P53 expression in HIV‐related oral large B cell lymphoma

Head and neck non‐Hodgkin's lymphomas in HIV positive patients are highly related with Epstein‐Barr virus (EBV) infection. In general, viral agents can alter p53 protein levels by enhancing degradation of cellular p53 or by increasing its half‐life by viral protein‐p53 interaction. Moreover, it has been reported that modifications of p53 gene can modulate tumor cells' response to radio‐ and chemotherapy.