{'fraction_non_alphanumeric': 0.026362038664323375, 'fraction_numerical': 0.0035149384885764497, 'mean_word_length': 5.705882352941177, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Activation of mast cells by immunoglobulin E‐receptor cross‐linkage, but not through adenosine receptors, induces A1 expression and promotes survival

Background Mast cells are a potent source of mediators that regulate the inflammatory response in allergy and asthma. Mast cells can be activated through different receptors, for example, via cross‐linkage of the high‐affinity IgE receptor (FcɛRI) and by adenosine acting on specific receptors. We have recently described mast cell survival of an IgE receptor activation by up‐regulation of the anti‐apoptotic gene A1.