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Acute cerebellar ataxia and concomitant infection

A c u T E cerebellar ataxia, a symptom complex of childhood, 1 begins abruptly and without evidence of acute infection of the nervous system. Ataxia is a constant feature and the hallmark of the syndrome. Nystagmus, ocular dysmetria, eye flutter, opsoclonus, dysarthria, tremor, hypotonia, convulsions, and altered sensorium may coexist. The cerebrospinal fluid protein may be elevated, and pleocytosis is present in one third of the cases. Recovery is usually rapid and complete although residual signs may persist. 2 More than one half of the patients have a history of antecedent systemic illness. During the fall of 1968, Mycoplasma pneumoniae infection was prevalent in Ontario. We encountered three children with acute ataxia following a respiratory infection, all of whom had serologic evidence of active M. pneumoniae infection. The salient clinical features of their illnesses are shown in Table I. This association seemed more than fortuitous.