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Congenital Factor XIII Deficiency: A Patient Report and Review of the Literature

meshwork of hydrogen-bonded fibrin monomers into a covalently bonded polymer, thus imparting chemical and mechanical stability to the clot. Deficiency of this protein results in clot instability and subsequent characteristic clinical abnormalities including prolonged bleeding, poor wound healing, and, unlike other bleeding disorders, a high incidence of intracranial hemorrhage. The mode of inheritance appears to be autosomal recessive2 although sex-linked inheritance has been