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α1‐Adrenergic effects and liver regeneration

The effects of several treatments involving α‐adrenergic mechanisms upon the early stages of rat liver regeneration were examined. Catecholamine concentrations in rat plasma were measured at various times after hepatectomy and were found to be elevated relative to those in plasma from sham‐operated rats. Surgical hepatic denervation or injection of an α1‐adrenergic receptor antagonist (prazosin) reduced incorporation of [3H] thymidine into liver DNA during the first 24 hr after partial hepatectomy. Chronic guanethidine injections (3 to 6 weeks) reduced liver catecholamine levels, but did not affect its ability to regenerate.