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Expression of inducible nitric oxide synthase and nitric oxide‐modified proteins in Helicobacter pylori‐associated atrophic gastric mucosa

Background and Aim:  Induction of inducible nitric oxide synthase (iNOS) may be involved in carcinogenesis of the stomach, because nitric oxide (NO) derived from iNOS can exert DNA damage and post‐transcriptional modification of target proteins. In the present study, we investigated the correlation between endoscopic findings and iNOS mRNA expression/NO‐modified proteins in the gastric mucosa.