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Fatty acids as metabolic mediators in innate immunity

Background  Increasing data support the hypothesis of a local and systemic crosstalk between adipocytes and monocytes mediated by fatty acids. The aim of this study was to characterize the immunomodulatory effects of a large panel of fatty acids on cytokines and chemokines in monocytic THP‐1 cells and primary human monocytes. We tested whether anti‐inflammatory fatty acids are able to inhibit the binding of lipopolysaccharide (LPS) to its receptor, toll‐like receptor/MD‐2 (TLR4/MD‐2).