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The effects of Receptor Interacting Protein 3 on necroptosis in RGC‐5 following elevated hydrostatic pressure

RIP3 has been found to induce necroptosis. Previous study have demonstrate necroptosis is an important neuronal death mode in retinal ischemia, and RIP3 may relate to necrosis in our observation. The lack of RIP3 inhibitor limited our study in vivo, so we first identify necroptosis in RGC‐5 following EHP, and then detect the protein level of RIP3, the percentage of necrosis using the pressure chamber via rip3 RNAi. Finally, we try to elaborate the mechanism of necroptosis.